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Journal articles on the topic 'Therapy induced'

Author: Grafiati

Published: 4 June 2021

Last updated: 10 February 2022

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1

Bobrova, Bobrova E. I., Vinogradskaya O. I. Vinogradskaya, Gubernatorova E. E. Gubernatorova, and Pavlova M. G. Pavlova. "Agranulocytosis induced by thyrostatics intake." Therapy 8_2020 (December 22, 2020): 162–67. http://dx.doi.org/10.18565/therapy.2020.8.162-167.

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Kumar, Niranjan. "Compare The Effectiveness of Modified Constraint Induced Movement Therapy (mCIMT) and Mirror Therapy (MT) in Stroke Patients Based on Severity." Physiotherapy and Occupational Therapy Journal 13, no. 1 (March 1, 2020): 29–40. http://dx.doi.org/10.21088/potj.0974.5777.13120.4.

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3

Kim, Min-Wook. "Constraint-induced movement therapy." Journal of the Korean Medical Association 56, no. 1 (2013): 38. http://dx.doi.org/10.5124/jkma.2013.56.1.38.

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Atay, Ahmet Engin, Elif Yorulmaz, Emel Gokmen, Bennur Esen Gullu, and Dede Sit. "Antihypertensive therapy-induced pancreatitis." Medical Journal of Goztepe Training and Research Hospital 28, no. 3 (May 16, 2014): 136–40. http://dx.doi.org/10.5222/j.goztepetrh.2013.136.

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Hayashida, Marina Zoega, Jhonatan Rafael Siqueira Pinheiro, Milvia Maria Simões e. Silva Enokihara, and Mônica Ribeiro de Azevedo Vasconcellos. "Biologic therapy-induced pemphigus." Anais Brasileiros de Dermatologia 92, no. 4 (August 2017): 591–93. http://dx.doi.org/10.1590/abd1806-4841.20176481.

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Zablotska, Lydia B., Anne H. Angevine, and Alfred I. Neugut. "Therapy-induced thoracic malignancies." Clinics in Chest Medicine 25, no. 1 (March 2004): 217–24. http://dx.doi.org/10.1016/s0272-5231(03)00124-2.

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van der Lee, Johanna H., Heleen Beckerman, Gustaaf J. Lankhorst, and Lex M. Bouter. "Constraint-induced movement therapy." Archives of Physical Medicine and Rehabilitation 80, no. 12 (December 1999): 1606. http://dx.doi.org/10.1016/s0003-9993(99)90339-8.

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Singanamala, Bhanudeep, Lokesh Saini, Priyanka Madaan, Paramjeet Singh, Pankaj C. Vaidya, and Jitendra Kumar Sahu. "Antitubercular therapy-induced psychosis." Neurology 93, no. 23 (December 2, 2019): 1012–13. http://dx.doi.org/10.1212/wnl.0000000000008578.

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Nair, Devi. "Antiretroviral therapy-induced hyperlipidaemia." International Journal of STD & AIDS 16, no. 1_suppl (October 2005): 2–13. http://dx.doi.org/10.1258/095646205774462805.

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Grotta, J. C. "Constraint-Induced Movement Therapy." Stroke 35, no. 11_suppl_1 (September 16, 2004): 2699–701. http://dx.doi.org/10.1161/01.str.0000143320.64953.c4.

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Behera, V., M. Randive, S. Sundaray, and M. S. N. Murty. "Antiretroviral therapy-induced lipodystrophy." Case Reports 2015, feb12 1 (February 12, 2015): bcr2014207090. http://dx.doi.org/10.1136/bcr-2014-207090.

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van der Lee, Johanna H., Heleen Beckerman, Gustaaf J. Lankhorst, and Lex M. Bouter. "Constraint-Induced Movement Therapy." Physical Therapy 80, no. 7 (July 1, 2000): 711–13. http://dx.doi.org/10.1093/ptj/80.7.711a.

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BAROLD, S. SERGE, ANDREAS KUCHER, DAVID J. McENEANEY, and ROLAND X. STROOBANDT. "Alternans-Induced ICD Therapy." Pacing and Clinical Electrophysiology 38, no. 9 (February 6, 2015): 1109–13. http://dx.doi.org/10.1111/pace.12564.

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Martin, Olga A., and Robin L. Anderson. "Editorial: Therapy-induced metastasis." Clinical & Experimental Metastasis 35, no. 4 (April 2018): 219–21. http://dx.doi.org/10.1007/s10585-018-9914-x.

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15

Almasri, Hussam A., Ashraf Tawfiq Soliman, Vincenzo Desanctis, Arwa E. Alsaud, Ruoa Alhashimy, Mona M. Babikir, Rita Wafik Ahmad, et al. "Iron Therapy Induced Leukopenia." Blood 136, Supplement 1 (November 5, 2020): 39–40. http://dx.doi.org/10.1182/blood-2020-135951.

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Introduction Iron deficiency anemia (IDA) is the most common cause of anemia in both developed and developing countries, particularly affecting females in the child bearing age and children. The treatment of IDA is a major health goal, it consists of treating the underlying cause and iron supplements. Iron replacement comes in form of oral or intravenous, there are certain side effects of this therapy including constipation and allergy. Leukopenia as a side effect of iron therapy is under reported in the literature as only sporadic cases were prescribed. We conducted a study to clarify this issue and to check for its clinical significance. Objective: To assess the relationship between iron therapy (intravenous) and leukopenia, neutropenia or lymphocytopenia, and its impact on patient's clinical settings. Materials and Methods We retrospectively reviewed the electronic medical records of patients attended Haematology clinic for iron deficiency anemia and treated with intravenous iron (ferric carboxymaltose or iron saccharide) over 2 years in Hamad Medical Corporation, Doha/Qatar. Adult female patients with IDA cases who received IV iron were included. anemia due to other nutrients deficienciesa nd conditions (including other medications) that may alter WBCs count were excluded.Age, Ethnicity, BMI, Complete blood count and iron studies data were collected before and after treatment with IV iron therapy. Infection occurrence at the time of IDA and leukopenia, the use of antibiotics and infection related complications were also collected. Leukopenia was defined as WBCs count to be less than 4000/microlitre, Neutropenia as ANC less than 1500/microlitre and lymphocytopenia as lymphocytes less than 1000/mocrolitre. Statistical analysis was done using mean , SD and t test. Results After iron therapy, out of 1567 case of iron deficiency anemia, 30 cases (1.914%) have leukopenia,15 cases (0.957%) have neutropenia and 12 cases (0.765%) have lymphocytopenia. All had normal readings before treatment. 2 patients (6.66%) had infection, 1 had upper respiratory tract infection and 1 urinary tract infection, the latter was treated with antibiotics, none reported infection related complications Discussion Leukocytopenia is defined as low WBCs circulating in the blood and this can be caused by low neutrophils count, low lymphocytes count, other WBCs components or combined. Some previous reported cases generated the idea of a possible connection between iron supplement therapy and leukopenia, Brito-Babapulle et al reported a case of fatal bone marrow suppression linked to ferric carboxymaltose therapy in a patient with IDA. The pathophysiology is not well understood but thought to be a toxic effect of iron on bone marrow and it can affect all cell lineages. Our findings suggest possible iron replacement side effect as there was significant drop of the WBCs count after treating IDA patients with IV iron, however this association was not common. There was no life threatening or serious infections in the affected patients, which can suggest that most of these cases are mild and transient. More studies are needed to address this issue, particularly on larger scales. Patient education also may be appreciated before treatment with IV iron. Conclusions: Leukopenia in form of neutropenia or lymphocytopenia maybe a side effect of IV iron therapy. Clinical significance is limited in view of current literature further studies needed to elaborate more in this important adverse event. Disclosures No relevant conflicts of interest to declare.

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16

Sayed, Asmaa Osama, Hebatallah Mohamed Kamal, and Nagham Mohamed Kamal Khalil. "CONSTRAINT INDUCED MOVEMENT THERAPY AND REAL-WORLD ARM USE IN CHILDREN WITH HEMIPARESIS." International Journal of Physiotherapy and Research 5, no. 3 (June 11, 2017): 2095–100. http://dx.doi.org/10.16965/ijpr.2017.149.

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17

Parasher, Ishan. "Vitamin D status among Pulmonary Tuberculosis Drug Induced Hepatotoxicity of Anti-tubercular Therapy." Journal of Medical Science And clinical Research 05, no. 05 (May 17, 2017): 21984–90. http://dx.doi.org/10.18535/jmscr/v5i5.119.

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18

Roberts, Pamela S., Jane A. Vegher, Michael Gilewski, Aimee Bender, and Richard V. Riggs. "Client-Centered Occupational Therapy Using Constraint-Induced Therapy." Journal of Stroke and Cerebrovascular Diseases 14, no. 3 (May 2005): 115–21. http://dx.doi.org/10.1016/j.jstrokecerebrovasdis.2005.01.002.

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19

Sadowska, Anna M., Pol Specenier, Paul Germonpre, and Marc Peeters. "Antineoplastic therapy-induced pulmonary toxicity." Expert Review of Anticancer Therapy 13, no. 8 (August 2013): 997–1006. http://dx.doi.org/10.1586/14737140.2013.817684.

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20

&NA;. "ADRs induced by fluoxetine therapy." Reactions Weekly &NA;, no. 324 (October 1990): 8. http://dx.doi.org/10.2165/00128415-199003240-00043.

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&NA;. "Diarrhoea induced by cisplatin therapy." Reactions Weekly &NA;, no. 740 (February 1999): 4. http://dx.doi.org/10.2165/00128415-199907400-00005.

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22

Sparano, Joseph A., David L. Brown, and Antonio C. Wolff. "Predicting Cancer Therapy???Induced Cardiotoxicity." Drug Safety 25, no. 5 (2002): 301–11. http://dx.doi.org/10.2165/00002018-200225050-00001.

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23

Greven, Margaret A., and Loh-Shan B. Leung. "Photodynamic Therapy-Induced Macular Hole." Ophthalmic Surgery, Lasers and Imaging Retina 49, no. 4 (April 1, 2018): 270–72. http://dx.doi.org/10.3928/23258160-20180329-10.

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24

Rittmannsberger, H., and F. Leblhuber. "Asterixis induced by carbamazepine therapy." Biological Psychiatry 32, no. 4 (August 1992): 364–68. http://dx.doi.org/10.1016/0006-3223(92)90040-7.

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25

Ravenel, James G., H. Page McAdams, John F. Plankeel, Kelly J. Butnor, and Thomas A. Sporn. "Sarcoidosis Induced by Interferon Therapy." American Journal of Roentgenology 177, no. 1 (July 2001): 199–201. http://dx.doi.org/10.2214/ajr.177.1.1770199.

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26

Nayati, Jasir T., and Alan R. Hirsch. "136 CerefolinNAC Therapy-Induced Dizziness." CNS Spectrums 23, no. 1 (February 2018): 85–86. http://dx.doi.org/10.1017/s1092852918000329.

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AbstractStudy ObjectiveCerefolinNAC (CFLN-NAC) contains L-methylfolate (6 mg), methylcobalamin (2 mg), and N-acetylcysteine [NAC] (600 mg) [Pamlab 2017]. Dizziness and lightheadedness have not heretofore been described with use of CFLN-NAC.MethodsCase Study: A 64 year old right-handed female was started on CFLN-NAC for smell and taste issues. Over a three day period, she experienced a gradual increase in dizziness. This was a non-vertiginous lightheadedness, so severe that she was unable to walk, and would lie down the entire day to alleviate the dizziness. It was associated with nausea, but without any vomiting or falls. The dizziness would come and go, last for several hours, and was 9/10 in severity.She admits to a past history of epochs of vertigo. The vertigo occurred three times with nausea and vomiting 13 years, 11 years, and 4 years prior to presentation. She also developed a constant, bilateral, high-pitched tinnitus 14 years prior to presentation, which obstructs her hearing. It is level 3/10 in intensity during the night and in the quiet. There were no alleviating or aggravating factors. Acupuncture was without effect, and she denies any ear pain. After ceasing CFLN-NAC for three days, a gradual reduction of dizziness to baseline ensued.ResultsAbnormalities in Physical Examination: General: Decreased blink frequency and hypokinesia. Cranial Nerve III, IV, VI: Saccadization of horizontal eye movements. Motor Examination: Tone: 1+ cogwheel rigidity in both upper extremities, left more than right. Drift Test: Bilateral Abductor Digiti Minimi sign with cerebellar spooning. Reflexes: Absent quadriceps femoris and Achilles bilaterally. Positive Hoffman reflex bilaterally. Neuropsychiatric Testing: Go-No-Go Test: 6/6 (normal). Animal Fluency Test: 19 (normal). Reliable Digit Span: 10 (normal). Clock Drawing Test: 4 (normal). Center for Neurologic Study Lability Scale: 10 (normal). Other: Audiometry and Fiberoptic Endoscopy: normal. MRI of the brain with and without contrast was normal.ConclusionNone of the individual components in CFLN-NAC have been reported to precipitate dizziness [Pamlab 2017]. The non-vertiginous nature of the dizziness makes it unlikely to be due to vestibular involvement, raising the spectre of this drug having an impact on the autonomic nervous system. While a nocebo effect could be in action, this is unlikely since dizziness was not presented as a potential side effect on initiation of the medication. In addition, methylcobalamin can cause hyperviscosity syndrome, but due to an absence of visual disturbances and altered mental status, it is also improbable. The relatively rapid onset with initiation and resolution upon discontinuation of this medication strongly suggests that it is not a coincidence, rather an origin for the dizziness. Those who are treated with CFLN-NAC should be queried as to new onset dizziness. For those already dizzy, one should consider other treatment options.Funding AcknowledgementsSmell and Taste Treatment and Research Foundation

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Nayati, Jasir T., Fizah S. Chaudhry, Tajinder Parhar, Ather M. Ali, and Alan R. Hirsch. "24 CerefolinNAC Therapy-Induced Dysgeusia." CNS Spectrums 24, no. 1 (February 2019): 186. http://dx.doi.org/10.1017/s109285291900018x.

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AbstractIntroductionCerefolinNAC (CFLN-NAC) is a prescription medical food reported to help with mild to moderate cognitive impairment [Pamlab 2017]. It contains L-methylfolate calcium (6mg), methylcobalamin (2mg), Schizochytrium (90.3mg), and N-acetylcysteine (NAC) (600mg) [Pamlab 2017]. However, dysgeusia secondary to CFLN-NAC therapy has not heretofore been described.MethodsA 64 year-old female presented with an eight year history of progressively decreased ability to smell and taste of unknown origin. CFLN-NAC was prescribed off-label to treat her hypogeusia and hyposmia. Three days after treatment initiation, her taste sensations gradually returned and she was able to describe food as bitter, salty, sour and sweet. Also, she was able to decipher the taste of different nuts, such as almonds, macadamia, pecans, and peanuts at baseline. However, her taste sensations became distorted and she was unable to distinguish specific foods. She reported that most food tasted bland, but she was still able to sense textures of various foods describing them as, “crunchy, but without taste.” She denied any oral pain, xerostomia, hot flashes, and psychological distress. CFLN-NAC was continued for three months and her hypogeusia improved from 20% to 80%. Her dysgeusia persisted, but remitted once CFLN-NAC was discontinued.ResultsAbnormalities in physical examination: General: scalloped tongue, decreased blink frequency, and hypokinesia. Cranial Nerve (CN) Examination: Olfaction (CN I) Testing: Alcohol Sniff Test: 8 (hyposmia). Pocket Smell Test: 2 (hyposmia). Olfactometer Identification Test: Left: 5 (anosmia); Right: 12 (hyposmia). CN III, IV, VI: saccadization on horizontal eye movement. Motor Examination: hypokinetic movements and 1+ cogwheel rigidity in bilateral upper extremities. Drift Test: bilateral abductor digiti minimi signs with cerebellar spooning. Reflexes: absent patellar and Achilles bilaterally. Hoffman’s Reflex: present bilaterally. Other: Magnetic resonance imaging (MRI) of the brain with contrast was unremarkable.ConclusionWhen treating taste impairments, vitamins and minerals have been found to enhance the effect of non-injured nerves, but they do not repair damaged nerves. The presence of a scalloped tongue may suggest nerve injury of unknown proportion, and can either diminish or alter taste. CFLN-NAC may have enhanced the gustatory stimulus of the non-injured nerves. This transient increase could have either caused her dysgeusia or possibly unmasked the dysgeusia secondary to a scalloped tongue. Notable impairments found in her exam evince Parkinson’s disease as a possible etiology, but structural abnormalities were not seen on brain MRI, making this unlikely. Conversely, the relatively rapid resolution after terminating CFLN-NAC strongly suggests that this is not merely a coincidence, but rather an origin. Those initiated on CFLN-NAC should be queried for new onset of dysgeusia and warrant other treatment options.Funding Acknowledgements: Smell & Taste Treatment and Research Foundation

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McFadden, J. P., A. V. Powles, and M. Walker. "Rosacea induced by PUVA therapy." British Journal of Dermatology 121, no. 3 (September 1989): 413. http://dx.doi.org/10.1111/j.1365-2133.1989.tb01440.x.

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Toivonen, Lauri K., and Sinikka Pohjola-Sintonen. "Vasodilator Therapy-induced Pacemaker Syndrome." Chest 91, no. 6 (June 1987): 919–20. http://dx.doi.org/10.1378/chest.91.6.919.

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30

Ewald, Jonathan A., Joshua A. Desotelle, George Wilding, and David F. Jarrard. "Therapy-Induced Senescence in Cancer." JNCI: Journal of the National Cancer Institute 102, no. 20 (October 20, 2010): 1536–46. http://dx.doi.org/10.1093/jnci/djq364.

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31

Schoepe, Stefanie, Heike Schacke, Ekkehard May, and Khusru Asadullah. "Glucocorticoid therapy-induced skin atrophy." Experimental Dermatology 15, no. 6 (June 2006): 406–20. http://dx.doi.org/10.1111/j.0906-6705.2006.00435.x.

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32

Newman, W. Christopher, and Edward A. Monaco. "Radiosurgery-Induced Phenotype-Targeted Therapy." Neurosurgery 77, no. 2 (August 2015): N21—N23. http://dx.doi.org/10.1227/01.neu.0000467299.41257.2a.

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Noda, Yoshihiro, Zafiris J. Daskalakis, Paul B. Fitzgerald, Jonathan Downar, Tarek K. Rajji, and Daniel M. Blumberger. "Magnetic Seizure Therapy-induced Mania." Journal of ECT 31, no. 1 (March 2015): e4-e6. http://dx.doi.org/10.1097/yct.0000000000000145.

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Fink, Max. "Induced Seizures as Psychiatric Therapy." Journal of ECT 20, no. 3 (September 2004): 133–36. http://dx.doi.org/10.1097/00124509-200409000-00001.

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35

Mahler, Ronald. "Pigmentation Induced by Quinidine Therapy." Archives of Dermatology 122, no. 9 (September 1, 1986): 1062. http://dx.doi.org/10.1001/archderm.1986.01660210112031.

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Mahler, R. "Pigmentation induced by quinidine therapy." Archives of Dermatology 122, no. 9 (September 1, 1986): 1062–64. http://dx.doi.org/10.1001/archderm.122.9.1062.

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37

Aycock, Dawn M., Sarah Blanton, Patricia C. Clark, and Steven L. Wolf. "What Is Constraint-Induced Therapy?" Rehabilitation Nursing 29, no. 4 (July 8, 2004): 114–15. http://dx.doi.org/10.1002/j.2048-7940.2004.tb00326.x.

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Bradfield, Jason S., and Kalyanam Shivkumar. "Cardiac Resynchronization Therapy–Induced Proarrhythmia." Circulation: Arrhythmia and Electrophysiology 7, no. 6 (December 2014): 1000–1002. http://dx.doi.org/10.1161/circep.114.002390.

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39

Safa, G., and V. Paumier. "Psoriasis induced by dupilumab therapy." Clinical and Experimental Dermatology 44, no. 3 (January 17, 2019): e49-e50. http://dx.doi.org/10.1111/ced.13901.

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40

Walker, Ulrich A. "Antiretroviral therapy-induced liver alterations." Current Opinion in HIV and AIDS 2, no. 4 (July 2007): 293–98. http://dx.doi.org/10.1097/coh.0b013e328122dbaa.

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41

Steiger, M. J., and N. P. Quinn. "Hormone replacement therapy induced chorea." BMJ 302, no. 6779 (March 30, 1991): 762. http://dx.doi.org/10.1136/bmj.302.6779.762.

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Fleisch, Martin, and Bernhard Meier. "Radiation Therapy–Induced Cardiac Injury." Circulation 96, no. 7 (October 7, 1997): 2462–63. http://dx.doi.org/10.1161/01.cir.96.7.2462.

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Warren, Stephen M., and James W. May. "Lipodystrophy Induced by Antiretroviral Therapy." New England Journal of Medicine 352, no. 1 (January 6, 2005): 63. http://dx.doi.org/10.1056/nejmicm040628.

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Soy, M., H. Ozer, A. Canataroglu, D. Gumurdulu, and E. Erken. "Vasculitis Induced by Zafirlukast Therapy." Clinical Rheumatology 21, no. 4 (August 2002): 328–29. http://dx.doi.org/10.1007/s100670200086.

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Benoff, Lane J., and Paul Schweitzer. "Radiation therapy-induced cardiac injury." American Heart Journal 129, no. 6 (June 1995): 1193–96. http://dx.doi.org/10.1016/0002-8703(95)90403-4.

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Yanık, B., C. Turkay, and H. Atalar. "Hepatotoxicity induced by alendronate therapy." Osteoporosis International 18, no. 6 (January 17, 2007): 829–31. http://dx.doi.org/10.1007/s00198-007-0323-2.

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Levy, Antonin, Antoine Hollebecque, Céline Bourgier, Yohann Loriot, Joël Guigay, Caroline Robert, Suzette Delaloge, et al. "Targeted therapy-induced radiation recall." European Journal of Cancer 49, no. 7 (May 2013): 1662–68. http://dx.doi.org/10.1016/j.ejca.2012.12.009.

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Larramendi, C. H., J. L. Garcia-Abujeta, M. Llombart, and R. Sanchez. "Intravenous Immunoglobulin Therapy-Induced Pompholyx." Journal of Allergy and Clinical Immunology 117, no. 2 (February 2006): S231. http://dx.doi.org/10.1016/j.jaci.2005.12.913.

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Nikitin, Nikitin E. Yu, Shakhnova E. A. Shakhnova, Osipova A. V. Osipova, Vorykhanov A. V. Vorykhanov, Uyanaeva M. A. Uyanaeva, Melkonyan G. G. Melkonyan, Drozdov V. N. Drozdov, and Shikh E. V. Shikh. "Drug-induced acute kidney injury among medical inpatients of therapeutic profile." Therapy 6_2021 (August 23, 2021): 18–23. http://dx.doi.org/10.18565/therapy.2021.6.18-23.

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Wu, Ching-yi, Ya-fen Chang, Keh-chung Lin, Hsieh-ching Chen, Chia-ling Chen, and Wei-hsien Hong. "KINEMATIC ANALYSIS OF BIMANUAL MOVEMENT AFTER CONSTRAINT-INDUCED THERAPY VS. BILATERAL MOVEMENT THERAPY IN PATIENTS WITH STROKE(2B1 Orthopaedic & Rehabilitation Biomechanics IV)." Proceedings of the Asian Pacific Conference on Biomechanics : emerging science and technology in biomechanics 2007.3 (2007): S142. http://dx.doi.org/10.1299/jsmeapbio.2007.3.s142.

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