Relevant bibliographies by topics / Thermogenic gene program

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  2. Dissertations / Theses

Academic literature on the topic 'Thermogenic gene program'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Thermogenic gene program"

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Yi, Danielle, Hai P. Nguyen, and Hei Sook Sul. "Epigenetic dynamics of the thermogenic gene program of adipocytes." Biochemical Journal 477, no. 6 (2020): 1137–48. http://dx.doi.org/10.1042/bcj20190599.

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Brown adipose tissue (BAT) is a metabolically beneficial organ capable of burning fat by dissipating chemical energy into heat, thereby increasing energy expenditure. Moreover, subcutaneous white adipose tissue can undergo so-called browning/beiging. The recent recognition of the presence of brown or beige adipocytes in human adults has attracted much attention to elucidate the molecular mechanism underlying the thermogenic adipose program. Many key transcriptional regulators critical for the thermogenic gene program centering on activating the UCP1 promoter, have been discovered. Thermogenic
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Li, Siming, Lin Mi, Lei Yu, et al. "Zbtb7b engages the long noncoding RNA Blnc1 to drive brown and beige fat development and thermogenesis." Proceedings of the National Academy of Sciences 114, no. 34 (2017): E7111—E7120. http://dx.doi.org/10.1073/pnas.1703494114.

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Brown and beige adipocytes convert chemical energy into heat through uncoupled respiration to defend against cold stress. Beyond thermogenesis, brown and beige fats engage other metabolic tissues via secreted factors to influence systemic energy metabolism. How the protein and long noncoding RNA (lncRNA) regulatory networks act in concert to regulate key aspects of thermogenic adipocyte biology remains largely unknown. Here we developed a genome-wide functional screen to interrogate the transcription factors and cofactors in thermogenic gene activation and identified zinc finger and BTB domain
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Hoang, Anh Cuong, Haidong Yu, and Tamás Röszer. "Transcriptional Landscaping Identifies a Beige Adipocyte Depot in the Newborn Mouse." Cells 10, no. 9 (2021): 2368. http://dx.doi.org/10.3390/cells10092368.

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The present study sought to identify gene networks that are hallmarks of the developing inguinal subcutaneous adipose tissue (iWAT) and the interscapular brown adipose tissue (BAT) in the mouse. RNA profiling revealed that the iWAT of postnatal (P) day 6 mice expressed thermogenic and lipid catabolism transcripts, along with the abundance of transcripts associated with the beige adipogenesis program. This was an unexpected finding, as thermogenic BAT was believed to be the only site of nonshivering thermogenesis in the young mouse. However, the transcriptional landscape of BAT in P6 mice sugge
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Shao, Mengle, Jeff Ishibashi, Christine M. Kusminski, et al. "Zfp423 Maintains White Adipocyte Identity through Suppression of the Beige Cell Thermogenic Gene Program." Cell Metabolism 23, no. 6 (2016): 1167–84. http://dx.doi.org/10.1016/j.cmet.2016.04.023.

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Armengol, Jordi, Josep A. Villena, Elayne Hondares та ін. "Pref-1 in brown adipose tissue: specific involvement in brown adipocyte differentiation and regulatory role of C/EBPδ". Biochemical Journal 443, № 3 (2012): 799–810. http://dx.doi.org/10.1042/bj20111714.

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Pref-1 (pre-adipocyte factor-1) is known to play a central role in regulating white adipocyte differentiation, but the role of Pref-1 in BAT (brown adipose tissue) has not been analysed. In the present study we found that Pref-1 expression is high in fetal BAT and declines progressively after birth. However, Pref-1-null mice showed unaltered fetal development of BAT, but exhibited signs of over-activation of BAT thermogenesis in the post-natal period. In C/EBP (CCAAT/enhancer-binding protein) α-null mice, a rodent model of impaired fetal BAT differentiation, Pref-1 was dramatically overexpress
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Stadion, M., K. Schwerbel, A. Graja, et al. "Interferon activated gene 202b (Ifi202b) promotes Zfp432 expression and suppresses the thermogenic gene program resulting in obesity and insulin resistance." Diabetologie und Stoffwechsel 12, S 01 (2017): S1—S84. http://dx.doi.org/10.1055/s-0037-1601654.

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Koncarevic, Alan, Shingo Kajimura, Milton Cornwall-Brady та ін. "A Novel Therapeutic Approach to Treating Obesity through Modulation of TGFβ Signaling". Endocrinology 153, № 7 (2012): 3133–46. http://dx.doi.org/10.1210/en.2012-1016.

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Obesity results from disproportionately high energy intake relative to energy expenditure. Many therapeutic strategies have focused on the intake side of the equation, including pharmaceutical targeting of appetite and digestion. An alternative approach is to increase energy expenditure through physical activity or adaptive thermogenesis. A pharmacological way to increase muscle mass and hence exercise capacity is through inhibition of the activin receptor type IIB (ActRIIB). Muscle mass and strength is regulated, at least in part, by growth factors that signal via ActRIIB. Administration of a
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8

Dinas, Petros C., Argyro Krase, Eleni Nintou, et al. "Thermogenic Capacity of Human White-Fat: The Actual Picture†." Proceedings 25, no. 1 (2019): 2. http://dx.doi.org/10.3390/proceedings2019025002.

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AIM: Cold exposure and exercise may increase thermogenic capacity of white adipose tissue (WAT), which could subsequently enhance energy expenditure and body weight loss. We aimed to identify possible alterations in uncoupling protein 1 (UCP1)—the main biomarker of thermogenic activation—in human WAT due to both cold exposure and exercise, as well as the link between environmental temperature and thermogenic capacity of human WAT. MATERIAL & METHOD: We conducted four human experimental studies and two systematic reviews and meta-analyses—PROSPERO registration CRD42019120116, CRD42019120213
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Mao, Xiaoxiang, Dandan Huang, Caijun Rao, et al. "Enoyl coenzyme A hydratase 1 combats obesity and related metabolic disorders by promoting adipose tissue browning." American Journal of Physiology-Endocrinology and Metabolism 318, no. 3 (2020): E318—E329. http://dx.doi.org/10.1152/ajpendo.00424.2019.

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Browning of white adipose tissue (WAT) has been recognized as an important strategy for the treatment of obesity, insulin resistance, and diabetes. Enoyl coenzyme A hydratase 1 (ECH1) is a widely known enzyme involved in lipid metabolism. However, whether and how ECH1 is implicated in browning of WAT remain obscure. Adeno-associated, virus-mediated genetic engineering of ECH1 in adipose tissue was used in investigations in mouse models of obesity induced by a high-fat diet (HFD) or browning induced by cold exposure. Metabolic parameters showed that ECH1 overexpression decreased weight gain and
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Xiong, Yan, Feng Yue, Zhihao Jia, et al. "A novel brown adipocyte-enriched long non-coding RNA that is required for brown adipocyte differentiation and sufficient to drive thermogenic gene program in white adipocytes." Biochimica et Biophysica Acta (BBA) - Molecular and Cell Biology of Lipids 1863, no. 4 (2018): 409–19. http://dx.doi.org/10.1016/j.bbalip.2018.01.008.

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Dissertations / Theses on the topic "Thermogenic gene program"

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Nam, Minwoo. "Role of Energy Metabolism in the Thermogenic Gene Program." eScholarship@UMMS, 2017. http://escholarship.umassmed.edu/gsbs_diss/886.

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In murine and human brown adipose tissue (BAT), mitochondria are powerful generators of heat. Emerging evidence has suggested that the actions of mitochondria extend beyond this conventional biochemical role. In mouse BAT and cultured brown adipocytes, impaired mitochondrial respiratory capacity is accompanied by attenuated expression of Ucp1, a key thermogenic gene, implying a mitochondrial retrograde signaling. However, few have investigated this association in the context of mitochondria-nucleus communication. Using mice with adipose-specific ablation of LRPPRC, a regulator of respiratory c
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