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Relevant bibliographies by topics / Thoracic Aortic Aneurysms and Dissections / Dissertations / Theses

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Dissertations / Theses on the topic 'Thoracic Aortic Aneurysms and Dissections'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Hannuksela, Matias. "Familial thoracic aortic aneurysms and dissections : studies on genotype and phenotype." Doctoral thesis, Umeå universitet, Anestesiologi och intensivvård, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-134028.

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Background: Thoracic aortic aneurysms and dissections (TAAD) have a genetic component with an estimated 20-25% of the patients having a positive family history. An aneurysm often precedes a dissection. Acute aortic dissections are associated with high mortality and morbidity, even when operated on. Complications due to prophylactic surgery are considerably fewer. Therefore, patients at risk for dissection should be identified, followed-up and evaluated for prophylactic intervention. Aims: 1. To establish reference values for ascending (AoA) and descending aortic (AoD) diameters measured by com

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2

Qing, Kaixiong, and 庆开雄. "Thoracic endovascular stent graft repair (TEVAR) for treating type B aortic dissections (TBAD) : a hemodynamic and morphologic perspective." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/197128.

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TEVAR has been used extensively to treat TBAD. The principle of treatment involve placement of a stent graft in the true lumen to cover the primary tear, thereby excluding the false lumen. Success depends on a combination of factors: reduction of false lumen pressure and perfusion, thrombosis of the false lumen, and remodeling of the aorta leading to eventual healing. The long-term goals are to prevent continuous growth of the false lumen, reinterventions, and aneurysm rupture. The success of TEVAR depends on a combination of factors, including the blood flow and pressure in the two aortic lum

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3

Winnerkvist, Anders. "Management of thoracoabdominal aortic aneurysms and dissections : with emphasis on spinal cord protection in aneurysm repair and non-surgical treatment of type-B dissection /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-768-5/.

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4

Bergeron, Sarah Elizabeth. "Biochemical basis of human disease-causing actin mutations." Diss., University of Iowa, 2011. https://ir.uiowa.edu/etd/2822.

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Actin isoform specific mutations have been identified as causes for various human diseases. These include twelve missense mutations in γ-nonmuscle actin leading to early onset autosomal dominate nonsyndromic hearing loss and twenty two missense mutations in α-smooth muscle actin leading to thoracic aortic aneurysms and dissections (TAAD). The molecular mechanisms leading to these human pathologies are mainly unknown, principally due to the inability to isolate pure mutant γ-nonmuscle actin and α-smooth muscle actin in quantities required for biochemical analysis. To begin to address these prob

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5

Kathiravel, Ushanthine [Verfasser]. "Molecular diagnosis and pathogenesis of Marfan syndrome and related heritable diseases associated with thoracic aortic aneurysms and dissections / Ushanthine Kathiravel." Hannover : Technische Informationsbibliothek und Universitätsbibliothek Hannover (TIB), 2014. http://d-nb.info/1058241354/34.

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6

Koch, Christopher D. "PROTEOGLYCAN DYSREGULATION IN THORACIC AORTIC ANEURYSM AND DISSECTION." Cleveland State University / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=csu1556890106847896.

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7

Huusko, T. (Tuija). "Genetic and molecular background of ascending aortic aneurysms." Doctoral thesis, Oulun yliopisto, 2013. http://urn.fi/urn:isbn:9789526201269.

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Abstract Thoracic aortic aneurysms (TAAs) are a significant source of morbidity and mortality. Classical risk factors for TAAs are hypertension, atherosclerosis, male gender, smoking, age, high body mass index, family history and chronic obstructive pulmonary disease. In addition, in certain cases of TAAs, i.e., ascending aortic aneurysms (AscAA), genetic factors are highly prominent. Matrix metalloproteinases are in a major role in the destruction of the aortic wall and the imbalance between matrix metalloproteinases, and their inhibitors are involved in the formation of aneurysms. In additio

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8

El, Bitar Sandy. "Genetic causes of cardiovascular diseases : special focus on familial hypercholesterolemia and thoracic aortic aneurysms." Thesis, Sorbonne Paris Cité, 2018. https://theses.md.univ-paris-diderot.fr/EL_BITAR_Sandy_2_va_20180403.pdf.

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L'identification de nouveaux acteurs impliqués dans les maladies cardiovasculaires (MCV) permet une meilleure compréhension de leur physiopathologie. L'objectif de ces Études de thèse est de déterminer les causes génétiques des MCV et leurs facteurs de risque, en particulier de hypercholestérolémie familiale (FH) et des anévrismes de l'aorte thoracique (TAA).La première partie portait sur l’étude des causes génétiques de la FH chez des proposants français. Grâce à une approche de séquençage de l'exome, nous avons identifié les variations p.(Arg50Gln) et p.(Ala3396Thr) dans une région de líAPOB

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9

Edvinsson, Marie. "Chlamydophila pneumoniae in Cardiovascular Diseases : Clinical and Experimental Studies." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-8667.

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10

Borghi, Alessandro. "Computational analysis of flow and stress patterns in thoracic aortic aneurysms : a patient specific study." Thesis, Imperial College London, 2007. http://hdl.handle.net/10044/1/7600.

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11

Larsson, Annika. "Medin amyloid - a matter close to the heart : Studies on medin amyloid formation and involvement in aortic pathology." Doctoral thesis, Uppsala universitet, Institutionen för genetik och patologi, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-9275.

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Amyloidoses are a group of protein misfolding diseases characterized by deposits of insoluble fibrillar protein aggregates. Medin amyloid, which is the focus of this thesis, appears in the media of the thoracic aorta in nearly all individuals over 50 years. The fibrils are derived from a 50 amino acid residue fragment of the precursor protein lactadherin. How medin amyloid arises is unknown, but in paper I we demonstrated, with immunohistochemical and in vitro binding experiments, that both lactadherin and medin interact with elastin, implying that the elastic fibre is central in amyloid forma

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12

Olsson, Christian. "Thoracic Aortic Surgery : Epidemiology, Outcomes, and Prevention of Cerebral Complications." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-6899.

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13

Saito, Naritatsu. "Feasibility of the Inoue single-branched stent-graft implantation for thoracic aortic aneurysm or dissection involving the left subclavian artery : short- to medium-term results in 17 patients." Kyoto University, 2007. http://hdl.handle.net/2433/135736.

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14

Vapnik, Joshua. "Anatomy determines etiology in thoracic aortic aneurysm." Thesis, 2015. https://hdl.handle.net/2144/16190.

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BACKGROUND: It is well established that thoracic aortic aneurysms (TAA) and abdominal aortic aneurysms (AAA) have different risk factors, clinical features, and genetic influences. Differences between and amongst subtypes of TAAs have received less attention. Despite observations of divergent clinical outcomes between ascending thoracic aortic aneurysms (ATAAs) and descending thoracic aortic aneurysms (DTAAs), etiologic factors determining the anatomic distribution of these aneurysms are not well understood. METHODS: From 3,247 patients registered in an institutional Thoracic Aortic Center

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15

Chiang, Yu-Lin Fan, and 范姜郁琳. "Application of Next Generation Sequencing to Establish the Genetic Testing Platform for Thoracic Aortic Aneurysm and Dissection Syndrome." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/92030430283610040366.

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碩士<br>國立臺灣大學<br>分子醫學研究所<br>105<br>Thoracic Aortic Aneurysm and Dissection syndrome (TAAD) can be divided into two main categories according to clinical features: (1) Syndromic, often accompanied by multiple groups of organ damage, including Marfan syndrome (MFS), Loeys-Dietz syndrome (LDS), Ehlers-Danlos syndrome (EDS), Aneurysms-Osteoarthritis syndrome (AOS), and transforming growth factor-β signaling related diseases; (2) Non-syndromic, only in the aortic disease, generally divided into familial TAAD and sporadic TAAD. However, the diversity of clinical features or symptoms are not clear, an

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16

Dias, Lara Romana Ribeiro. "Gender differences on mortality and re-interventions after TEVAR for intact thoracic aortic aneurysms." Dissertação, 2019. https://hdl.handle.net/10216/119785.

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17

Dias, Lara Romana Ribeiro. "Gender differences on mortality and re-interventions after TEVAR for intact thoracic aortic aneurysms." Master's thesis, 2019. https://hdl.handle.net/10216/119785.

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18

Wu, I.-Hui, and 吳毅暉. "Analysis of Clinical Geometry and Biomarkers of Thoracic and Abdominal Aortic Stent Grafts in the Treatment of Aortic Aneurysms." Thesis, 2015. http://ndltd.ncl.edu.tw/handle/35030960317440813961.

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博士<br>國立臺灣大學<br>臨床醫學研究所<br>103<br>This thesis mainly focuses on analysis of clinical geometry of thoracic and abdominal aortic stent grafts in the treatment of aortic aneurysms. Including (1) The Signiﬁcance of Endograft Geometry on the Incidence of Intraprosthetic Thrombus Deposits after Abdominal Endovascular Grafting (2) Outcomes following endovascular or open repair for ruptured abdominal aortic aneurysm in a Chinese population (3) One-stage Hybrid Repair to Thoracoabdominal Aortic Aneurysm (4) Crossover Chimney Technique to Preserve the Internal Iliac Artery During Endovascular Repair of

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19

Baraki, Hassina [Verfasser]. "The frozen elephant trunk technique for treatment of thoracic aortic aneurysms / vorgelegt von Hassina Baraki." 2008. http://d-nb.info/988993805/34.

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20

Babu, Anju R. "Role of Microstructure in the Mechanics of Soft Matter." Thesis, 2015. http://etd.iisc.ernet.in/2005/3826.

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Materials which exhibit non-linear mechanical behaviors under large deformations are generally classified as “soft matter”. Elastomers represent an important class of soft materials which have wide commercial applications and isotropic non-linear behavior. In contrast, biological materials have anisotropic responses due to their heterogeneous and composite architectures. The underlying microstructure determines the arterial macroscopic behavior and is represented through constitutive models to describe the stress-strain relationships. Mechanical characterization and development of constitutive

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