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Journal articles on the topic 'Thoracic Aortic Aneurysms and Dissections'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

White, Rodney A., Carlos Donayre, Irwin Walot, James Lee, and George E. Kopchok. "Regression of a Descending Thoracoabdominal Aortic Dissection following Staged Deployment of Thoracic and Abdominal Aortic Endografts." Journal of Endovascular Therapy 9, no. 2_suppl (2002): II—92—II—97. http://dx.doi.org/10.1177/15266028020090s215.

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Purpose: To describe the successful endovascular repair and regression of an extensive descending thoracoabdominal aortic dissection associated with thoracic and abdominal aortic aneurysms. Case Report: An 83-year-old man presented with acute chest pain and shortness of breath. A descending thoracoabdominal aortic dissection that extended from near the left subclavian artery (LSA) to the right common iliac artery was found on computed tomography. Separate aneurysms in the thoracic and abdominal aorta were also identified. Staged endovascular procedures were undertaken to (1) close the single e

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2

Harky, Amer, Ka Siu Fan, and Ka Hay Fan. "The genetics and biomechanics of thoracic aortic diseases." Vascular Biology 1, no. 1 (2019): R13—R25. http://dx.doi.org/10.1530/vb-19-0027.

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Thoracic aortic aneurysms and aortic dissections (TAAD) are highly fatal emergencies within cardiothoracic surgery. With increasing age, thoracic aneurysms become more prevalent and pose an even greater threat when they develop into aortic dissections. Both diseases are multifactorial and are influenced by a multitude of physiological and biomechanical processes. Structural stability of aorta can be disrupted by genes, such as those for extracellular matrix and contractile protein, as well as telomere dysfunction, which leads to senescence of smooth muscle and endothelial cells. Biomechanical

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3

Cury, Marcelo, Fernanda Zeidan, and Armando C. Lobato. "Aortic Disease in the Young: Genetic Aneurysm Syndromes, Connective Tissue Disorders, and Familial Aortic Aneurysms and Dissections." International Journal of Vascular Medicine 2013 (2013): 1–7. http://dx.doi.org/10.1155/2013/267215.

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There are many genetic syndromes associated with the aortic aneurysmal disease which include Marfan syndrome (MFS), Ehlers-Danlos syndrome (EDS), Loeys-Dietz syndrome (LDS), familial thoracic aortic aneurysms and dissections (TAAD), bicuspid aortic valve disease (BAV), and autosomal dominant polycystic kidney disease (ADPKD). In the absence of familial history and other clinical findings, the proportion of thoracic and abdominal aortic aneurysms and dissections resulting from a genetic predisposition is still unknown. In this study, we propose the review of the current genetic knowledge in the

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4

Abdyldaev, I. Z., S. D. Chevgun, M. A. Aripov, et al. "Stent grafts implantation in patients with life-threatening aortic conditions – first experience in Kyrgyz Republic." Heart, Vessels and Transplantation 3, Issue 4 (2019): 208. http://dx.doi.org/10.24969/hvt.2019.156.

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Objective: Aortic diseases remain an acute and debatable problem. Among all the aortic pathologies, the most dangerous are dynamic aneurysmal expansion, traumatic dissections and / or aneurysms, pure aortic intima dissections. Contemporary and less invasive treatment method is the stent graft implantation into aorta. This report describes the first experience of thoracic endovascular aortic repair (TEVAR) and endovascular aneurysm repair (EVAR) in the Kyrgyz Republic in consecutive patients with life-threatening aortic conditions. Methods: Nine patients, who underwent TEVAR and EVAR, were pres

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5

Arroyave, Jose, Juan Manuel Carretero, and Domenico Gruosso. "Isolated aortic dilation without osteoarthritis: a case of SMAD3 mutation." Cardiology in the Young 28, no. 5 (2018): 765–67. http://dx.doi.org/10.1017/s1047951118000082.

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AbstractAneurysm–osteoarthritis syndrome is a recently discovered inherited autosomal dominant connective tissue disease caused by SMAD3 mutations. Aneurysm–osteoarthritis syndrome is responsible for 2% of familial thoracic aortic aneurysms and dissections and is characterised by aneurysms, dissections, and tortuosity throughout the arterial tree in combination with osteoarthritis. Early-onset osteoarthritis is present in almost all patients. We present the case of a non-syndromic young boy with SMAD3 mutation isolated from the dilated aortic root and ascending aorta without osteoarthritis.

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Latt, Kyaw K., Alexey A. Moiseev, Natalya R. Chernaya, Konstantin N. Vasilyev, Nikolay L. Bayandin, and Viktor A. Stupin. "IMMEDIATE AND LONGTERM RESULTS OF ENDOVASCULAR STENT-GRAFTING IN DISSECTIONS AND ATHEROSCLEROTIC ANEURYSMS OF THORACIC AORTA." Atherothrombosis Journal, no. 2 (December 27, 2018): 135–40. http://dx.doi.org/10.21518/2307-1109-2018-2-135-140.

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Objective: to analyze the results of endovascular stent-graftingin dissections and atherosclerotic aneurysms of descending part of thoracic aorta.Material and method: in this analysis includes 28 patients with dissections and atherosclerotic aneurysms of descending part of thoracic aorta. Acute aortic dissection type 3 was occurred in 10 patients and two of them were complicated with rupture into the left pleural cavity. 10 patients with chronic aortic dissection type bunderwent operations and one of them was complicated with rupture into the left pleural cavity. Among 8 operated patients due

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7

Zotikov, A. E., D. I. Maryan, S. S. Ilyin, V. A. Kulbak, and A. V. Pokrovsky. "FEATURES OF DEBRANCHING TECHNIQUE FOR AORTIC DISSECTIONS AND ANEURYSMS IN THORATIC AND THORACO-ABDOMINAL AORTAS." Atherothrombosis Journal, no. 1 (June 29, 2019): 128–37. http://dx.doi.org/10.21518/2307-1109-2019-1-128-137.

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This article describes a hybrid surgical technique for high-risk patients with thoraco-abdominal aortic dissections and aneurysms. The technical aspects of the first stage of surgical treatment - visceral and brachiocephalic debranching are described in detail. 17 surgical interventions for chronic dissection and aneurysm in the thoracic and thoracoabdominal aortas were performed at the Vishnevsky National Medical Research Center of Surgery of Ministry of Health of the Russian Federation in the period from 2014 to 2019. In two cases, we observed postoperative pancreatitis after surgical treatm

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8

Samura, Makoto, Nobuya Zempo, Yoshitaka Ikeda, et al. "Single-stage thoracic and abdominal endovascular aneurysm repair for multilevel aortic disease." Vascular 22, no. 1 (2013): 55–60. http://dx.doi.org/10.1177/1708538112473965.

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This investigation evaluated the results of single-stage thoracic endovascular aneurysm repair (TEVAR) and endovascular aneurysm repair (EVAR) for multilevel aortic disease in a series of nine patients. The lesions repaired included thoracic and abdominal aortic aneurysms ( n = 7) and subacute type B dissections with abdominal aortic aneurysms ( n = 2). All procedures were successfully performed, and none of the patients experienced postoperative stroke or spinal cord ischemia. The median follow-up period for these patients was 18.9 months (range 1.7–31.4 months) and none of the patients exhib

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9

Anthony Lee, W. "Management of Aortic Aneurysms and Dissections with the Zenith TX2 Stent Graft." Vascular 14, no. 5 (2006): 282–89. http://dx.doi.org/10.2310/6670.2006.00052.

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Endovascular repair of thoracic aortic aneurysms offers a less invasive alternative to conventional surgery, similar to its counterpart in the abdominal aorta. Although its long-term durability and outcome remain relatively unknown, published midterm results from clinical trials suggest comparable rates of aneurysm-related mortality and significant reductions in early morbidity compared with open repair. The Zenith TX2 stent graft (Cook Endovascular, Bloomington, IN) is an investigational endovascular device that is designed to treat aneurysms and other degenerative pathologies of the thoracic

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10

Hostetler, Ellen M., Ellen S. Regalado, Dong-Chuan Guo, et al. "SMAD3 pathogenic variants: risk for thoracic aortic disease and associated complications from the Montalcino Aortic Consortium." Journal of Medical Genetics 56, no. 4 (2019): 252–60. http://dx.doi.org/10.1136/jmedgenet-2018-105583.

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BackgroundPathogenic variants in SMAD3 cause thoracic aortic aneurysms and dissections, along with aneurysms and rupture of other arteries. Here, we examined differences in clinical presentation of aortic events (dissection or surgical repair of an aneurysm) with respect to age and variant type in an international cohort of individuals with SMAD3 variants.MethodsAortic status and events, vital status and clinical features were abstracted through retrospective review of medical records of 212 individuals with 51 unique SMAD3 variants, including haploinsufficiency (HI) and missense substitutions

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11

Guo, Dongchuan, Sumera Hasham, Shao-Qing Kuang, et al. "Familial Thoracic Aortic Aneurysms and Dissections." Circulation 103, no. 20 (2001): 2461–68. http://dx.doi.org/10.1161/01.cir.103.20.2461.

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12

Guo, Dong-Chuan, Ellen S. Regalado, Charles Minn, et al. "Familial Thoracic Aortic Aneurysms and Dissections." Circulation: Cardiovascular Genetics 4, no. 1 (2011): 36–42. http://dx.doi.org/10.1161/circgenetics.110.958066.

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13

Prahlow, Joseph A., Jeffrey J. Barnard, and Dianna M. Milewicz. "Familial Thoracic Aortic Aneurysms and Dissections." Journal of Forensic Sciences 43, no. 6 (1998): 14395J. http://dx.doi.org/10.1520/jfs14395j.

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14

Pisano, Calogera, Carmela Rita Balistreri, Alessandro Ricasoli, and Giovanni Ruvolo. "Cardiovascular Disease in Ageing: An Overview on Thoracic Aortic Aneurysm as an Emerging Inflammatory Disease." Mediators of Inflammation 2017 (2017): 1–8. http://dx.doi.org/10.1155/2017/1274034.

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Medial degeneration associated with thoracic aortic aneurysm and acute aortic dissection was originally described by Erdheim as a noninflammatory lesion related to the loss of smooth muscle cells and elastic fibre fragmentation in the media. Recent evidences propose the strong role of a chronic immune/inflammatory process in aneurysm evocation and progression. The coexistence of inflammatory cells with markers of apoptotic vascular cell death in the media of ascending aorta with aneurysms and type A dissections raises the possibility that activated T cells and macrophages may contribute to the

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15

Peterson, Joshua C., Lambertus J. Wisse, Valerie Wirokromo, et al. "Disturbed nitric oxide signalling gives rise to congenital bicuspid aortic valve and aortopathy." Disease Models & Mechanisms 13, no. 9 (2020): dmm044990. http://dx.doi.org/10.1242/dmm.044990.

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ABSTRACTPatients with a congenital bicuspid aortic valve (BAV), a valve with two instead of three aortic leaflets, have an increased risk of developing thoracic aneurysms and aortic dissection. The mechanisms underlying BAV-associated aortopathy are poorly understood. This study examined BAV-associated aortopathy in Nos3−/− mice, a model with congenital BAV formation. A combination of histological examination and in vivo ultrasound imaging was used to investigate aortic dilation and dissections in Nos3−/− mice. Moreover, cell lineage analysis and single-cell RNA sequencing were used to observe

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16

Bhandari, Rohan, Rajani D. Aatre, and Yogendra Kanthi. "Diagnostic approach and management of genetic aortopathies." Vascular Medicine 25, no. 1 (2020): 63–77. http://dx.doi.org/10.1177/1358863x19886361.

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Aortic aneurysms were the primary cause of nearly 10,000 deaths in 2014 according to data from the Centers for Disease Control and may involve segments of the thoracic or abdominal aorta. Thoracic aortic aneurysms and dissections are more commonly associated with an underlying genetic etiology. In the past several decades, in parallel with the burst of new genome sequencing technologies, a number of genetic aortopathies have been identified. These have provided important insights into the molecular mechanisms of aneurysmal disease, but pose challenges in clinical practice as there are limited

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17

Valaika, Arūnas, Gediminas Norkūnas, Gintaras Kalinauskas, et al. "Extra-anatomic thoracic aortic bypass operations." Medicina 44, no. 5 (2008): 373. http://dx.doi.org/10.3390/medicina44050048.

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Objective. When the patient condition contraindicates major surgery for descending thoracic aneurysms, the surgeon should consider using an ascending aorta to abdominal aorta bypass graft, leaving the diseased segment undisturbed. Our experience with eight patients is presented. Material and methods. Between 1988 and 2008, eight patients were treated for the following indications: reoperation for coarctation (two patients), complicated descending aortic aneurysms (five patients), and posttraumatic descending aorta dissection (one patient). The mean age of the patients was 44±8 years (range, 27

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18

Baril, Donald T., Jae S. Cho, Rabih A. Chaer, and Michel S. Makaroun. "Thoracic Aortic Aneurysms and Dissections: Endovascular Treatment." Mount Sinai Journal of Medicine: A Journal of Translational and Personalized Medicine 77, no. 3 (2010): 256–69. http://dx.doi.org/10.1002/msj.20178.

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19

Strecker, T., A. Agaimy, and M. Weyand. "PS160 Inflammatory Thoracic Aortic Aneurysms and Dissections." Global Heart 11, no. 2 (2016): e40. http://dx.doi.org/10.1016/j.gheart.2016.03.137.

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20

Raunsø, Jakob, Rebecca J. Song, Ramachandran S. Vasan, et al. "Familial Clustering of Aortic Size, Aneurysms, and Dissections in the Community." Circulation 142, no. 10 (2020): 920–28. http://dx.doi.org/10.1161/circulationaha.120.045990.

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Background: Ruptured aortic aneurysm and aortic dissections are potentially preventable disorders associated with high mortality. Screening of individuals at risk may translate into elective surgical interventions and lowered mortality. It is uncertain if the risk of aortic dilation of varying degrees aggregates within families. Methods: We investigated the risk of having thoracic and abdominal aortic sizes in the highest quartile (measured by computed tomography scans and indexed for body size) if at least 1 parent did so in the Framingham Heart Study cohorts, and estimated the incidence rate

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21

Milewicz, Dianna M., and Francesco Ramirez. "Therapies for Thoracic Aortic Aneurysms and Acute Aortic Dissections." Arteriosclerosis, Thrombosis, and Vascular Biology 39, no. 2 (2019): 126–36. http://dx.doi.org/10.1161/atvbaha.118.310956.

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22

Pannu, Hariyadarshi, Van Tran-Fadulu, and Dianna M. Milewicz. "Genetic basis of thoracic aortic aneurysms and aortic dissections." American Journal of Medical Genetics Part C: Seminars in Medical Genetics 139C, no. 1 (2005): 10–16. http://dx.doi.org/10.1002/ajmg.c.30069.

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23

Qato, Khalil, Allan Conway, Eileen Lu, Nhan Nguyen Tran, Gary Giangola, and Alfio Carroccio. "Outcomes of Thoracic Endovascular Aneurysm Repair (TEVAR) in Patients With Connective Tissue Disorders." Vascular and Endovascular Surgery 54, no. 8 (2020): 676–80. http://dx.doi.org/10.1177/1538574420945070.

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Objectives: Thoracic endovascular aortic repair (TEVAR) remains controversial in patients with connective tissue disorders given the concern for durability. We report on the largest series to date on outcomes of patients with thoracic aortic disease and connective tissue disorders treated with TEVAR. Methods: The Vascular Quality Initiative registry identified 12 207 patients treated with TEVAR from January 2010 to December 2018, including 102 with Marfans, Ehlers-Danlos, or Loey-Dietz syndrome. Outcomes were analyzed per the Society for Vascular Surgery reporting standards. Results: Median ag

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24

Gramatiuk, S., Yu Ivanova, V. Kravchenko, and O. Lohvinenko. "Activity of matrix proteinases and their inhibitors in patients with thoracic aortic dissection." Bukovinian Medical Herald 24, no. 4 (96) (2020): 19–26. http://dx.doi.org/10.24061/2413-0737.xxiv.4.96.2020.98.

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Purpose. To study changes in profibrogenic mediators in patients with thoracic aortic dissection.Material and methods. The study was carried out in 66 patients with dissection of the ascending aneurysm and the aortic arch, which occurred against the background of atherosclerosis and hypertension, aged 48 to 73 years. According to the objectives of the study, the patients were divided into three groups: the first – 32 patients with acute dissections, the second – 34 patients with subacute and chronic dissections, the third – 30 patients with aneurysms of the thoracic and abdominal aorta, who we

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25

Shahrrava, Anahita, Sunnan Moinuddin, Prajwal Boddu, and Rohan Shah. "A Case of Glucocorticoid Remediable Aldosteronism and Thoracoabdominal Aneurysms." Case Reports in Endocrinology 2016 (2016): 1–4. http://dx.doi.org/10.1155/2016/2017571.

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Glucocorticoid remediable aldosteronism (GRA) is rare familial form of primary aldosteronism characterized by a normalization of hypertension with the administration of glucocorticoids. We present a case of GRA and thoracoabdominal aneurysm complicated by multiple aortic dissections requiring complex surgical and endovascular repairs. Registry studies have shown a high rate of intracranial aneurysms in GRA patients with high case fatality rates. The association of thoracoabdominal aneurysms with GRA has not been described, thus far, in literature. Studies have shown that high tissue aldosteron

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Gordeev, M. L., V. E. Uspenskiy, A. Y. Bakanov, et al. "Aortic arch reconstruction in surgical treatment of ascending aortic aneurysms and dissections." Patologiya krovoobrashcheniya i kardiokhirurgiya 20, no. 4 (2016): 45. http://dx.doi.org/10.21688/1681-3472-2016-4-45-57.

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<p><strong>Aim.</strong> The study focused on the analysis of short-term results of aortic arch reconstruction in patients undergoing open heart surgery for ascending aortic aneurysms and dissections, comparison of intra-operative brain protection methods and verification of predictors of complications.<br /><strong>Methods.</strong> 84 patients (mean age 55.5 ± 11.5 years, 72.6 % (61) males) with ascending aortic aneurysms and Stanford type A ascending aortic and arch dissections underwent surgery over a period from January, 2013, to March, 2015. Patients w

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27

Hasham, Sumera N., Dong-chuan Guo, and Dianna M. Milewicz. "Genetic basis of thoracic aortic aneurysms and dissections." Current Opinion in Cardiology 17, no. 6 (2002): 677–83. http://dx.doi.org/10.1097/00001573-200211000-00015.

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28

Regalado, E. S., D. C. Guo, R. L. P. Santos-Cortez, et al. "PathogenicFBN1variants in familial thoracic aortic aneurysms and dissections." Clinical Genetics 89, no. 6 (2016): 719–23. http://dx.doi.org/10.1111/cge.12702.

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29

Caspar, Sylvan M., Nicolo Dubacher, and Gabor Matyas. "More Genes for Thoracic Aortic Aneurysms and Dissections." Journal of the American College of Cardiology 73, no. 4 (2019): 528–29. http://dx.doi.org/10.1016/j.jacc.2018.09.094.

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30

Suh, Jong Hui, Jeong-Seob Yoon, Hwan Wook Kim, and Keon Hyon Jo. "Adventitial Fibroblast Abormality in Thoracic Aortic Aneurysms and Aortic Dissections." Korean Journal of Thoracic and Cardiovascular Surgery 44, no. 6 (2011): 406–12. http://dx.doi.org/10.5090/kjtcs.2011.44.6.406.

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31

Lepore, Vincenzo, Lars Lönn, Martin Delle, et al. "Endograft Therapy for Diseases of the Descending Thoracic Aorta: Results in 43 High-Risk Patients." Journal of Endovascular Therapy 9, no. 6 (2002): 829–37. http://dx.doi.org/10.1177/152660280200900617.

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Purpose: To report an initial experience with endovascular stent-graft implantation for diseases of the descending thoracic aorta in high-risk patients. Methods: Forty-three patients (28 men; mean age 67 years, range 17–82) with 16 descending thoracic aortic dissections, 14 aneurysms, 7 contained ruptures, 3 mycotic aneurysms, 2 posttraumatic pseudoaneurysms, and an aneurysm of an anomalous right subclavian artery were treated between June 1999 and July 2001. Twenty-three (53%) patients were treated emergently. Results: There were no conversions to open repair, but 3 (7%) patients died during

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Boran, Mertay, Ali İhsan Parlar, and Ertay Boran. "Giant Aortic Pseudoaneurysm with Stanford Type A Aortic Dissection after Aortic Valve Replacement." Case Reports in Medicine 2012 (2012): 1–3. http://dx.doi.org/10.1155/2012/473732.

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Giant pseudoaneurysm of the ascending aorta is a rare but dreadful complication occurring several months or years after aortic surgery. Thoracic aortic aneurysms tend to be asymptomatic and were previously often diagnosed only after a complication such as dissection or rupture. We present a rare case of giant ascending aneurysm with Stanford type A aortic dissection occurring 6 years after aortic valve replacement and also illustrate the potential dimensions the ascending aorta may reach by a pseudoaneurysm and dissection after AVR.

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33

Manapat, Adrian E., Jorge M. Garcia, Joseph B. Barril, Gary A. Lopez, and Diomedes A. Talavera. "Repair of Thoracic Aortic Aneurysm and Dissection Using Deep-Hypothermic Circulatory Arrest." Asian Cardiovascular and Thoracic Annals 6, no. 2 (1998): 95–100. http://dx.doi.org/10.1177/021849239800600205.

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From October 1989 to September 1997, 14 patients underwent repair of a thoracic aortic aneurysm or dissection using deep-hypothermic circulatory arrest. There were 10 males and 4 females with a mean age of 58 years (range, 43 to 82 years). The diagnoses included one ascending aortic aneurysm, one ascending aortic and arch aneurysm, 2 aortic arch and descending aortic aneurysms, 4 descending aortic aneurysms, 2 chronic aortic dissections of type A and 4 of type B. The involved aortic segment was replaced with a woven Dacron tube graft in 11 patients and repaired with a patch of woven Dacron in

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34

Takeda, Norifumi, Hironori Hara, Takayuki Fujiwara, Tsubasa Kanaya, Sonoko Maemura та Issei Komuro. "TGF-β Signaling-Related Genes and Thoracic Aortic Aneurysms and Dissections". International Journal of Molecular Sciences 19, № 7 (2018): 2125. http://dx.doi.org/10.3390/ijms19072125.

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Transforming growth factor-β (TGF)-β signaling plays a crucial role in the development and maintenance of various organs, including the vasculature. Accordingly, the mutations in TGF-β signaling pathway-related genes cause heritable disorders of the connective tissue, such as Marfan syndrome (MFS), Loeys-Dietz syndrome (LDS), and Shprintzen-Goldberg syndrome (SGS), and these syndromes may affect skeletal, ocular, pulmonary, and cardiovascular systems. Aortic root aneurysms are common problems that can result in aortic dissection or rupture, which is the leading cause of sudden death in the nat

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35

Bhave, Shreyas A., Dongchuan Guo, Stoyan N. Angelov, et al. "Human SMAD4 Genomic Variants Identified in Individuals with Heritable and Early-Onset Thoracic Aortic Disease." Cardiogenetics 11, no. 3 (2021): 132–38. http://dx.doi.org/10.3390/cardiogenetics11030015.

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Thoracic aortic aneurysms (TAAs) that progress to acute thoracic aortic dissections (TADs) are life-threatening vascular events that have been associated with altered transforming growth factor (TGF) β signaling. In addition to TAA, multiple genetic vascular disorders, including hereditary hemorrhagic telangiectasia (HHT), involve altered TGFβ signaling and vascular malformations. Due to the importance of TGFβ, genomic variant databases have been curated for activin receptor-like kinase 1 (ALK1) and endoglin (ENG). This case report details seven variants in SMAD4 that are associated with eithe

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Ohki, Takao, Yuji Kanaoka, and Koji Maeda. "Endovascular Treatment of Various Aortic Pathologies: Review of the Latest Data and Technologies." International Journal of Angiology 27, no. 02 (2018): 081–91. http://dx.doi.org/10.1055/s-0038-1645881.

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AbstractThe technologies and innovations applicable to endovascular treatment for complex aortic pathologies have progressed rapidly over the last two decades. Although the initial outcomes of an endovascular aortic repair have been excellent, as long-term data became available, complications including endoleaks, endograft migration, and endograft infection have become apparent and are of concern. Previously, the indication for endovascular therapy was restricted to descending thoracic aortic aneurysms and abdominal aortic aneurysms. However, its indication has expanded along with the improvem

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37

Kacani, Andi, Saimir Kuci, Arber Aliu, Alfred Ibrahimi, Aferdita Veseli, and Arben Baboci. "A Huge Aneurism of Ascending Thoracic Aorta." Albanian Journal of Trauma and Emergency Surgery 5, no. 2 (2021): 883–86. http://dx.doi.org/10.32391/ajtes.v5i2.239.

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Giant Ascending Aorta Aneurysm (AAA) is a rare condition, because of early diagnosis incidence appears to be increasing as a result of routine screening, increased clinical awareness, and improved imaging modalities. The etiology of aneurysms involving the aortic root and ascending aorta can be genetically triggered, degenerative or atherosclerotic, inflammatory, or can result from infectious diseases. According to many studies for ascending aortic aneurysms larger than 6 cm the risk of rupture, dissection, or deaths was 15.6, making it a large life-threatening aneurysm. We present the c

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Lee, Jacqueline J., Justin B. Dimick, David M. Williams, et al. "Existence of abdominal aortic aneurysms in patients with thoracic aortic dissections." Journal of Vascular Surgery 38, no. 4 (2003): 671–75. http://dx.doi.org/10.1016/s0741-5214(03)00727-4.

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39

Fann, James I., and D. Craig Miller. "ENDOVASCULAR TREATMENT OF DESCENDING THORACIC AORTIC ANEURYSMS AND DISSECTIONS." Surgical Clinics of North America 79, no. 3 (1999): 551–74. http://dx.doi.org/10.1016/s0039-6109(05)70024-4.

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40

Khanna, NN. "Endovascular Repair of Thoracic and Abdominal Aortic Aneurysms / Dissections." Apollo Medicine 8, no. 3 (2011): 217–27. http://dx.doi.org/10.1016/s0976-0016(11)60077-x.

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41

Kuang, Shao-Qing, Olga Medina-Martinez, Dong-chuan Guo, et al. "FOXE3 mutations predispose to thoracic aortic aneurysms and dissections." Journal of Clinical Investigation 126, no. 3 (2016): 948–61. http://dx.doi.org/10.1172/jci83778.

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42

Guo, Dong-chuan, Ellen S. Regalado, Limin Gong, et al. "LOX Mutations Predispose to Thoracic Aortic Aneurysms and Dissections." Circulation Research 118, no. 6 (2016): 928–34. http://dx.doi.org/10.1161/circresaha.115.307130.

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43

Faggion Vinholo, Thais, Mohammad A. Zafar, Bulat A. Ziganshin, and John A. Elefteriades. "Nonsyndromic Thoracic Aortic Aneurysms and Dissections—Is Screening Possible?" Seminars in Thoracic and Cardiovascular Surgery 31, no. 4 (2019): 628–34. http://dx.doi.org/10.1053/j.semtcvs.2019.05.035.

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44

Takeda, Norifumi, and Issei Komuro. "Genetic basis of hereditary thoracic aortic aneurysms and dissections." Journal of Cardiology 74, no. 2 (2019): 136–43. http://dx.doi.org/10.1016/j.jjcc.2019.03.014.

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Orend, K. H., R. Scharrer-Pamler, X. Kapfer, F. Liewald, J. Görich, and L. Sunder-Plassmann. "Endovascular treatment of thoracic aortic aneurysms, dissections, and ruptures." Gefässchirurgie 6, no. 3 (2001): 136–44. http://dx.doi.org/10.1007/s007720100140.

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Gokalp, Orhan, Levent Yılık, Ismail Yurekli, Serkan Gur, Zehra Ilke Akyildiz, and Ali Gurbuz. "Dissecting Thoracic Aortic Aneurysms." Annals of Thoracic Surgery 94, no. 4 (2012): 1377–78. http://dx.doi.org/10.1016/j.athoracsur.2012.03.098.

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PANNU, H., N. AVIDAN, V. TRAN-FADULU, and D. M. MILEWICZ. "Genetic Basis of Thoracic Aortic Aneurysms and Dissections: Potential Relevance to Abdominal Aortic Aneurysms." Annals of the New York Academy of Sciences 1085, no. 1 (2006): 242–55. http://dx.doi.org/10.1196/annals.1383.024.

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Schütz, Wolfram, Albrecht Gauss, Rainer Meierhenrich, Reinhard Pamler, and Johannes Görich. "Transesophageal Echocardiographic Guidance of Thoracic Aortic Stent-Graft Implantation." Journal of Endovascular Therapy 9, no. 2_suppl (2002): II—14—II—19. http://dx.doi.org/10.1177/15266028020090s203.

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Purpose: To evaluate the efficacy of intraoperative transesophageal echocardiography (TEE) as an adjunctive measure in guiding the implantation of endoluminal stent-grafts in the thoracic aorta. Methods: TEE was used in 21 of 30 patients (27 men; median age 70 years; range 19–77) undergoing implantation of Excluder or Talent stent-grafts for management of 11 type B aortic dissections, 7 thoracic aortic aneurysms, 2 traumatic thoracic aortic ruptures, and an aortic coarctation. We evaluated the ability of TEE to provide evidence of (1) correct placement of the guidewire within the true lumen, (

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Shirshova, Elena V., O. Y. Annenkova, E. V. Ekusheva, and V. N. Petrov. "The chest pain in neurological practice. Non deesset..." Journal of Clinical Practice 10, no. 2 (2019): 91–96. http://dx.doi.org/10.17816/clinpract10291-96.

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Chest pain can be a “mask” of a life-threatening condition, which the practitioner must remember. One of such life-threatening condition is aortic aneurysm, which diagnosis presents significant difficulties in routine clinical practice. Clinical manifestations of thoracic aortic aneurysms are extremely variable and non-specific and are mainly depends on the size of the aneurysmal sac, its localization and extent, as well as the etiology of the disease. Here we present a clinical case report of a 48 y.o. patient who died because of the acute cardiac tamponade as a complication of dissecting tho

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Zafer İşcan, Hakkı, and Ertekin Utku Ünal. "Surgeon-modified fenestrated stent graft deployment in type B aortic dissection." Turkish Journal of Thoracic and Cardiovascular Surgery 29, no. 2 (2021): 285–89. http://dx.doi.org/10.5606/tgkdc.dergisi.2021.20641.

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The treatment of aortic dissections and aneurysms may be challenging for vascular surgeons. Currently, thoracic endovascular aortic repair is usually the first treatment option for descending aortic pathologies. Left subclavian artery coverage during this procedure is often required to achieve a sufficient proximal landing zone. Most surgeons agree that the left subclavian artery can be selectively covered, but revascularization is preferred to reduce the risk of neurological or ischemic complications. The chimney method, hybrid operations with extra-anatomic bypass, back table or in situ fene

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