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Books on the topic 'Thrombocytopenic Purpura'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2025

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1

Paul, Imbach, and Workshop on Immunoglobulin (1985 : :Lucerne, Switzerland), eds. Idiopathic thrombocytopenic purpura: Proceedings of a workshop. PharmaLibri, 1987.

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2

1941-, Kaplan Bernard S., Trompeter Richard S, and Moake Joel L. 1940-, eds. Hemolytic uremic syndrome and thrombotic thrombocytopenic purpura. Marcel Dekker, Inc., 1992.

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3

Handelsman, Harry. Protein A columns for immune thrombocytopenia. U.S. Dept. of Health and Human Services, Public Health Service, Agency for Health Care Policy and Research, 1991.

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4

Parker, James N., and Philip M. Parker. The official patient's sourcebook on immune thrombocytopenic purpura. Icon Health Publications, 2002.

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5

H, Waters A., Webster A. D. B, Sandoz Pharmaceuticals, Medi-Cine Communications International, and Royal Society of Medicine (Great Britain), eds. Intravenous immunoglobulins in immunodeficiency syndromes and idiopathic thrombocytopenic purpura. Royal Society of Medicine, 1985.

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6

Hewitt, John. Studies in antibody specificity against platelet glycoprotein IIb/IIIa in autoimmune thrombocytopenic purpura. University of Manchester, 1994.

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7

Parker, James N., and Philip M. Parker. Thrombotic thrombocytopenic purpura: A bibliography and dictionary for physicians, patients, and genome researchers [to Internet references]. ICON Health Publications, 2007.

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8

Scully, Marie A., and Spero R. Cataland. Fast Facts: Thrombotic Thrombocytopenic Purpura. Karger AG, S., 2020.

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9

Scully, Marie A., and Spero R. Cataland. Fast Facts: Thrombotic Thrombocytopenic Purpura. Karger AG, S., 2020.

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10

Lewis, David Charles. Canine idiopathic thrombocytopenic purpura pathogenesis and diagnosis. 1993.

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11

Thrombotic Thrombocytopenic Purpura: Causes, Diagnosis and Treatment. Nova Science Publishers, Incorporated, 2019.

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12

Hillam, Mason. Thrombotic Thrombocytopenic Purpura: Causes, Diagnosis and Treatment. Nova Science Publishers, Incorporated, 2019.

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13

Scully, Marie, John-Paul Westwood, Mari Thomas, and Matthew Stubbs. Fast Facts for Patients: Thrombotic Thrombocytopenic Purpura. Karger AG, S., 2022.

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14

Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Haemostasis and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0010.

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Assessing haemostasis - The coagulation system - Laboratory tests - Platelets - Bleeding - Bleeding: laboratory investigations - Bleeding: therapeutic products - von Willebrand disease - Haemophilia A and B - Rare congenital coagulation disorders - Congenital thrombocytopenias - Congenital platelet function defects - Congenital vascular disorders - Haemorrhagic disease of the newborn - Thrombocytopenia (acquired) - Specific thrombocytopenic syndromes - Disseminated intravascular coagulation - Liver disease - Renal disease - Acquired anticoagulants and inhibitors - Treatment of spontaneous FVII

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15

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, and Angela Theodoulou. Haemostasis and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0010_update_001.

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Assessing haemostasis - The coagulation system - Laboratory tests - Platelets - Bleeding - Bleeding: laboratory investigations - Bleeding: therapeutic products - von Willebrand disease - Haemophilia A and B - Rare congenital coagulation disorders - Congenital thrombocytopenias - Congenital platelet function defects - Congenital vascular disorders - Haemorrhagic disease of the newborn - Thrombocytopenia (acquired) - Specific thrombocytopenic syndromes - Disseminated intravascular coagulation - Liver disease - Renal disease - Acquired anticoagulants and inhibitors - Treatment of spontaneous FVII

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16

Publications, ICON Health. The Official Patient's Sourcebook on Immune Thrombocytopenic Purpura. Icon Health Publications, 2002.

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17

Tiessen, Karen. Vincristine infusion versus bolus in idiopathic thrombocytopenic purpura. 1985.

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18

Lotta, Luca Andrea. Pathophysiology of Thrombotic Thrombocytopenic Purpura: The Two-Hit Paradigm. Amsterdam University Press, 2012.

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19

Lotta, Luca Andrea. Pathophysiology of Thrombotic Thrombocytopenic Purpura: The Two-Hit Paradigm. Amsterdam University Press, 2012.

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20

Noris, Marina, and Tim Goodship. The patient with haemolytic uraemic syndrome/thrombotic thrombocytopenic purpura. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0174.

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The patient who presents with microangiopathic haemolytic anaemia, thrombocytopenia, and evidence of acute kidney injury presents a diagnostic and management challenge. Haemolytic uraemic syndrome (HUS) and thrombotic thrombocytopenic purpura (TTP) are two of the conditions that frequently present with this triad. They are characterized by low platelet count with normal or near-normal coagulation tests, anaemia, and signs of intravascular red cell fragmentation on blood films, and high LDH levels.HUS associated with shiga-like toxins produced usually by E.coli (typically O157 strains) may occu

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21

Peabody, Francisco. Coloring Book - You Will Get Better - Immune Thrombocytopenic Purpura. Independently Published, 2021.

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22

Lotta, Luca Andrea. Pathophysiology of Thrombotic Thrombocytopenic Purpura: The Two-Hit Paradigm. Amsterdam University Press, 2012.

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23

Karpin, Jessenia. Coloring Book - You Will Get Better - Idiopathic Thrombocytopenic Purpura. Independently Published, 2021.

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24

Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Haematological emergencies. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0013.

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Septic shock/neutropenic fever - Acute transfusion reactions - Delayed transfusion reaction - Post-transfusion purpura - Hypercalcaemia - Hyperviscosity - Disseminated intravascular coagulation - Overdosage of thrombolytic therapy - Heparin overdosage - Heparin-induced thrombocytopenia - Warfarin overdosage - Massive blood transfusion - Paraparesis/spinal collapse - Leucostasis - Thrombotic thrombocytopenic purpura - Sickle crisis

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25

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, and Hassan Al-Sader. Haematological emergencies. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0013_update_001.

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Septic shock/neutropenic fever - Acute transfusion reactions - Delayed transfusion reaction - Post-transfusion purpura - Hypercalcaemia - Hyperviscosity - Disseminated intravascular coagulation - Overdosage of thrombolytic therapy - Heparin overdosage - Heparin-induced thrombocytopenia - Warfarin overdosage - Massive blood transfusion - Paraparesis/spinal collapse - Leucostasis - Thrombotic thrombocytopenic purpura - Sickle crisis

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26

Scully, M. A., S. R. Cataland, M. Stubbs, and M. Thomas. Fast Facts for Patients : Thrombotic Thrombocytopenic Purpura: Prompt Action Saves Lives. Karger AG, S., 2022.

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27

Oland, Marty. Coloring Book - You Will Get Better - Thrombocytopenic Purpura: Heal While Coloring. Independently Published, 2021.

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28

Carton, James. Haematopathology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199591633.003.0014.

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Iron deficiency anaemia 256Anaemia of chronic disease 257Megaloblastic anaemias 258Hereditary spherocytosis 259Glucose-6-phosphate dehydrogenase deficiency 260Thalassaemias 261Sickle cell disorders 262Idiopathic thrombocytopenic purpura 264Thrombotic thrombocytopenic purpura 265von Willebrand disease 266Haemophilia 267Thrombophilia 268Acute B-lymphoblastic leukaemia 269...

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29

Kesteven, Patrick. Haematology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0061.

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This chapter describes the common haematological abnormalities seen in rheumatic conditions with special reference to laboratory findings and diagnostic traps. The chapter is organized into sections dealing with red cell abnormalities (and the distinction between anaemia of chronic disease and iron deficiency); white cell abnormalities (neutrophilia and neutropenia); platelets (thrombocytosis, idiopathic thrombocytopenic purpura and thrombotic thrombocytopenic purpura); and finally coagulation abnormalities (lupus anticoagulant).

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30

Glasper, Edward Alan, Gillian McEwing, and Jim Richardson, eds. Haematology and immunity problems. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780198569572.003.0017.

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The blood 564Immune protection in children 566Anaemia 568Sickle cell disease (SCD) 570Management of sickle cell disease 572Haemophilia 574Henoch-Schnölein purpura 576Idiopathic thrombocytopenic purpura (ITP) 578Thalassaemia 580Thrombocytopenia 582HIV infection 584Primary deficiencies of the innate immune system in children ...

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31

Publications, ICON Health. The Official Patient's Sourcebook on Idiopathic Thrombocytopenic Purpura: A Directory for the Internet Age. Icon Health Publications, 2005.

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32

Publications, ICON Health. Idiopathic Thrombocytopenic Purpura - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References. ICON Health Publications, 2004.

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33

Fridbinstons, Pit Alex. Causes of Thrombocytopenia: Enlarged Spleen, Anemia, Leukemia, Pregnancy, Heavy Alcohol Consumption, Viral Infections, Chemotherapy Medications, Hemolytic Uremic Syndrome, Thrombotic Thrombocytopenic Purpura. Independently Published, 2021.

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34

Carton, James. Haematopathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0015.

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This chapter discusses haematopathology, including iron deficiency anaemia, anaemia of chronic disease, megaloblastic anaemias, hereditary spherocytosis, glucose-6-phosphate dehydrogenase deficiency, thalassaemias, sickle-cell disorders, idiopathic thrombocytopenic purpura (ITP), thrombotic thrombocytopenic purpura (TTP), von Willebrand disease, haemophilia, thrombophilia, acute B-lymphoblastic leukaemia, acute myeloid leukaemias, chronic lymphocytic leukaemia (CLL), chronic myelogenous leukaemia, polycythaemia vera (PV), essential thrombocythaemia (ET), primary myelofibrosis (PMF), myelodyspl

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35

Provan, Drew, Trevor Baglin, Inderjeet Dokal, and Johannes de Vos. Paediatric haematology. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199683307.003.0012.

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Blood counts in children - Red cell transfusion and blood component therapy—special considerations in neonates and children - Polycythaemia in newborn and childhood - Neonatal anaemia - Anaemia of prematurity - Haemolytic anaemia in the neonate - Congenital red cell defects - Acquired red cell defects - Haemolytic disease of the newborn - Hyperbilirubinaemia - Neonatal haemostasis - Neonatal alloimmune thrombocytopenia - Congenital dyserythropoietic anaemias - Congenital red cell aplasia - Acquired red cell aplasia - Fanconi anaemia - Rare congenital marrow failure syndromes - Neutropenia in c

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36

Provan, Drew, Trevor Baglin, Inderjeet Dokal, Johannes de Vos, and Angela Theodoulou. Paediatric haematology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199683307.003.0012_update_001.

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Blood counts in children - Red cell transfusion and blood component therapy—special considerations in neonates and children - Polycythaemia in newborn and childhood - Neonatal anaemia - Anaemia of prematurity - Haemolytic anaemia in the neonate - Congenital red cell defects - Acquired red cell defects - Haemolytic disease of the newborn - Hyperbilirubinaemia - Neonatal haemostasis - Neonatal alloimmune thrombocytopenia - Congenital dyserythropoietic anaemias - Congenital red cell aplasia - Acquired red cell aplasia - Fanconi anaemia - Rare congenital marrow failure syndromes - Neutropenia in c

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37

Misbah, Siraj. Immunological support. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0301.

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Until the 1980s, the use of immunoglobulin as a therapeutic agent was confined to replacement therapy in patients with primary or secondary antibody deficiency. Its role as an effective immunomodulator was discovered serendipitously, when IV immunoglobulin (IVIg) was shown to consistently increase the platelet count in a child with antibody deficiency and immune thrombocytopenic purpura. Since then, the use of high-dose IVIg as an immunomodulator has become established as an important therapeutic option in many immune-mediated diseases. This chapter reviews the therapeutic indications for immu

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38

Elham, Bayat. Neurologic Manifestations of Hematological Disease. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0193.

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A wide sprectum of hematologic disorders affect the central and peripheral nervous system. These disorders include porphyria, thrombotic thrombocytopenic purpura-hemolytic uremic syndromes, sickle cell disease, plasma cell dyscrasias, monoclonal gammopathy, primary systemic amyloidosis, primary systemic amyloidosis, Waldonstrom’s macroglobulinemia, myeloproliferative syndromes, cryoglobulinemia, and polycythemia vera. Some, like porphyria, cause both central and peripheral nervous system manifestations including sensory/motor peripheral neuropathy, dysautonomia, pain, seizures, and abdominal p

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39

Sternbach, Marion. Apheresis in the ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0268.

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This chapter describes therapeutic plasma exchange, as well as cytapheresis for hyperleukocytosis and essential thrombocythemia, as well as harvesting haematological stem cells (HSC) for transplantation. Instrumentation and techniques are mostly density centrifugation, much less column adsorption for antibodies or membrane filtration for noxious molecules. Pathophysiology of apheresis is dealt with in great detail with emphasis on prevention and treatment of side effects, much more critical in the intensive care unit (ICU) setting. Main manifestations are: hypocalcaemia due to chelation by ant

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40

Dragun, Duska, and Björn Hegner. Acute kidney injury in pregnancy. Edited by Norbert Lameire. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0250_update_001.

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Any kind of acute renal deterioration that occurs in young women may, besides typical pregnancy-related disorders, account for pregnancy-related acute kidney injury (PR-AKI). Incidence of PR-AKI is continuously decreasing, yet still represents a significant cause of fetomaternal morbidity and mortality. Hyperemesis gravidarum causing volume depletion and septic shock with renal cortical necrosis upon septic abortion are major causes of PR-AKI during early pregnancy. Pre-eclampsia and bleeding complications associated with placental abruption or other causes of obstetric haemorrhage are respons

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41

Wiles, Kate, and Catherine Nelson-Piercy. Pre-eclampsia and related disorders. Edited by Norbert Lameire and Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0296_update_001.

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Pre-eclampsia is a pregnancy-specific condition diagnosed by new-onset hypertension and proteinuria after 20 weeks’ gestation. The incidence of pre-eclampsia means that it is both the most prevalent cause of acute kidney injury (AKI) in pregnancy and the commonest glomerular disease in the world. This chapter outlines the diagnosis and management of pre-eclampsia. Particular emphasis is given to the post-partum disease course as this is when a specialist nephrology opinion may be sought. HELLP (haemolysis, elevated liver enzymes, low platelets) syndrome is a variant of severe pre-eclampsia. Th

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42

Kothari, Jaimal, and Marie Scully. Thrombocytopenia in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0272.

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Thrombocytopenia is an extremely common finding in patients in intensive care. This can lead to serious morbidity and mortality from bleeding, and also uncertainty with regards to how to assess this risk in the wider context of the underlying pathology, which may be multifactorial, when specific blood products should be transfused, and the associated risks of carrying out interventional procedures. Haemodilution, platelet consumption, increased platelet destruction, reduced production, and increased sequestration are the main mechanisms that account for thrombocytopenia, and in the ICU setting

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43

Mannucci, Pier Mannuccio. Bleeding and haemostasis disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0070.

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The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, an

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44

Mannucci, Pier Mannuccio. Bleeding and haemostasis disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199687039.003.0070_update_001.

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The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, an

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45

Mannucci, Pier Mannuccio. Bleeding and haemostasis disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199687039.003.0070_update_002.

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The main cause of haemostasis defects and related bleeding complications in patients with acute coronary syndromes admitted to the intensive cardiac care unit is the use of multiple antithrombotic drugs, alone or concomitantly with invasive procedures such as percutaneous coronary intervention with stent deployment and coronary artery bypass surgery. These drugs, that act upon several components of haemostasis (platelet function, coagulation, fibrinolysis), are associated with bleeding complications, particularly in elderly patients (more so in women than in men), those who are underweight, an

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