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Books on the topic 'Thrombose – Diagnostic'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 February 2022

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1

Ansell, Jack E. Handbook of hemostasis and thrombosis: A diagnostic and therapeutic approach. Little, Brown, 1986.

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2

Lockwood, Charles J. Thrombosis, thrombophilia and thromboembolism. American College of Obstetricians and Gynecologists, 2007.

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3

Ogston, Derek. Venous thrombosis: Causation and prediction. Wiley, 1987.

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4

Handbook of hemostasis and thrombosis: A diagnostic and therapeutic approach. Little, Brown, 1986.

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5

International, Symposium on Experimental and Clinical Aspects of Cerebral Sinus Thrombosis (1987 Nymphenburg Munich Germany). Cerebral sinus thrombosis: Experimental and clinical aspects. Plenum Press, 1990.

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6

W, Hoyer Leon, and Drohan William, eds. Recombinant technology in hemostasis and thrombosis. Plenum Press, 1991.

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7

Tinkler, Kerry. Setting up, piloting, implementing and reviewing a GP direct access service for the diagnosis of lower limb deep vein thrombosis. University of Portsmouth, 2004.

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8

MD, Spencer Fredrick A., ed. Dx/Rx. Jones and Bartlett Publishers, 2005.

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9

Bergan, John J. Management of venous disorders. Landes Bioscience, 2000.

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10

Marques, Marisa B. Quick guide to hemostasis. American Association for Clinical Chemistry, 2015.

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11

European, Thrombosis Research Organization Workshop on Disseminated Intravascular Coagulation (1984 Stockholm Sweden). European Thrombosis Research Organization Workshop on Disseminated Intravascular Coagulation: Definition, experimental investigations, laboratory diagnosis and treatment of DIC and similar proteolytic states : Stockholm, September, 1984. Published for Medisinsk fysiologisk forenings forlag, Oslo by Blackwell Scientific Publication, 1985.

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12

European Thrombosis Research Organisation. Workshop. Monoclonal antibodies and human blood platelets: Proceedings of the European Thrombosis Research Organisation Workshop, under the patronage of the Institut national de la santé et de la recherche médicale, held in Lyon (France), 26-27 September 1985. Edited by McGregor John Louis and Institut national de la santé et de la recherche médicale (France). Elsevier Science Publishers, 1986.

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13

M, Samama Meyer, and Mauriat Françoise, eds. Hémorragies et thromboses: Du diagnostic au traitement. Masson, 2004.

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14

Beek, Edwin J. R. van., Büller H. R, and Oudkerk Matthijs, eds. Deep vein thrombosis and pulmonary embolism. J. Wiley, 2009.

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15

Deep vein thrombosis and pulmonary embolism. J. Wiley, 2009.

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16

1935-, Hirsh Jack, ed. Venous thrombosis and pulmonary embolism: Diagnostic methods. Churchill Livingstone, 1987.

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17

Hillam, Mason. Thrombotic Thrombocytopenic Purpura: Causes, Diagnosis and Treatment. Nova Science Publishers, Incorporated, 2019.

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18

(Editor), A. Baethmann, K. Einhäupl (Editor), and O. Kempski (Editor), eds. Cerebral Sinus Thrombosis. Springer, 1991.

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19

Hvas, Anne-Mette, Erik L. Grove, and Steen Dalby Kristensen. Biomarkers of coagulation and thrombosis. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0038.

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Coagulation is evaluated by conventional coagulation analyses, often supplemented by point-of-care tests. Recently, a number of point-of-care tests for evaluation of platelet function and the efficacy of antiplatelet therapy has been investigated. Thrombophilia contributes to the risk of thrombosis, and a battery of complex assays is required to identify all thrombophilias. Disseminated intravascular coagulation is characterized by microthrombosis and clinical bleeding. A scoring system for overt disseminated intravascular coagulation provides a five-step diagnostic algorithm. The cornerstone

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20

Hvas, Anne-Mette, Erik L. Grove, and Steen Dalby Kristensen. Biomarkers of coagulation and thrombosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0038_update_001.

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Coagulation is evaluated by conventional coagulation analyses, often supplemented by point-of-care tests. Recently, a number of point-of-care tests for evaluation of platelet function and the efficacy of antiplatelet therapy has been investigated. Thrombophilia contributes to the risk of thrombosis, and a battery of complex assays is required to identify all thrombophilias. Disseminated intravascular coagulation is characterized by microthrombosis and clinical bleeding. A scoring system for overt disseminated intravascular coagulation provides a five-step diagnostic algorithm. The cornerstone

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21

Deep Vein Thrombosis: Symptoms, Diagnosis and Treatments. Nova Science Pub Inc, 2012.

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22

Andrew N. Nicolaides MS FRCS FRCSE and David S. Sumner MD. Investigation of Patients with Deep Vein Thrombosis and Chronic Venous Insufficiency. Med-Orion Publishing Co Ltd, 1991.

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23

Nicolaides, Andrew N. Investigation of patients with deep vein thrombosis and chronic venous insufficiency. Med-Orion, 1991.

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24

Pichler, Michael R., and Robert D. Brown. Cerebral Venous Thrombosis. Edited by Emma Ciafaloni, Cheryl Bushnell, and Loralei L. Thornburg. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0017.

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Thrombosis of cortical veins and/or dural venous sinuses (CVT) is a rare but potentially devastating condition. CVT is more common in women and is strongly associated with pregnancy and the postpartum period, likely due to numerous procoagulant changes during this time. CVT can cause a wide range of symptoms depending on location of thrombosis. Clinical manifestations can include headache, cranial nerve deficits, seizures, and venous infarction with associated focal neurologic deficits. Severe cases may progress to coma and death, emphasizing the importance of early diagnosis and treatment. Th

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25

Hirsh, Jack. Venous Thrombosis and Pulmonary Embolism: Diagnostic Methods (Methods in Haematology). Churchill Livingstone, 1987.

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26

Markus, Hugh, Anthony Pereira, and Geoffrey Cloud. Cerebral venous thrombosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198737889.003.0012.

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Most stroke results from arterial disease but venous occlusion can also cause stroke, and other neurological complications. This condition is uncommon and needs a high index of suspicion if it is not to be missed. The clinical presentations are varied and can mimic other neurological conditions. The diagnosis is important because with appropriate treatment the prognosis can be much better than for arterial infarction.

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27

Jørgen, Jespersen, Bertina Rogier M, and Haverkate F. 1931-, eds. Laboratory techniques in thrombosis: A manual. 2nd ed. Kluwer Academic Publishers, 1999.

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28

Schrader, Ernst-August. Die Klinik der Arteriellen Thrombosen im Beckenbereich: "Pathogenese, Untersuchungsmethoden Diagnostik Und Therapie". Springer, 2012.

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29

Ogston, Derek. Venous Thrombosis: Causation and Prediction (Wiley Medical Publications). John Wiley & Sons, 1988.

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30

V, Caso, Agnelli Giancarlo, and Paciaroni M, eds. Handbook on cerebral venous thrombosis. Karger, 2008.

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31

(Editor), V. Caso, and G. Agnelli (Editor), eds. Handbook on Cerebral Venous Thrombosis (Frontiers of Neurology and Neuroscience). S Karger Pub, 2007.

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32

Hirsh, Jack, and Jeff Ginsberg. Critical Decisions in Thrombosis and Hemostasis (Critical Decisions). BC Decker Inc., 1998.

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33

Current concepts of thrombosis: Prevalent trends for diagnosis and management. Saunders, 1998.

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34

Laboratory Techniques in Thrombosis - A Manual: Second Revised Edition of the Ecat Assay Procedures. 2nd ed. Springer, 1999.

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35

The antiphospholipid syndrome II: Autoimmune thrombosis. Elsevier, 2002.

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36

E, Preston F., Kitchen Steve Dr, and Olson John David 1944-, eds. Quality in laboratory hemostasis and thrombosis. Wiley-Blackwell, 2009.

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37

Olson, John D., Steve Kitchen, F. Eric Preston, and Frits R. Rosendaal. Quality in Laboratory Hemostasis and Thrombosis. Wiley & Sons, Incorporated, John, 2011.

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38

Quality in Laboratory Hemostasis and Thrombosis. Wiley-Blackwell, 2013.

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39

Olson, John D., Steve Kitchen, and F. Eric Preston. Quality in Laboratory Hemostasis and Thrombosis. Wiley & Sons, Incorporated, John, 2013.

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40

Olson, John D., Steve Kitchen, F. Eric Preston, and Frits R. Rosendaal. Quality in Laboratory Hemostasis and Thrombosis. Wiley & Sons, Incorporated, John, 2011.

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41

Alchi, Bassam, and David Jayne. The patient with antiphospholipid syndrome with or without lupus. Edited by Giuseppe Remuzzi. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0164.

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Antiphospholipid syndrome (APS) is an autoimmune disorder characterized by recurrent arterial or venous thrombosis and/or pregnancy loss, accompanied by laboratory evidence of antiphospholipid antibodies (aPL), namely anticardiolipin antibodies (aCL), lupus anticoagulant (LA), and antibodies directed against beta-2 glycoprotein 1 (β‎‎‎2GP1). APS may occur as a ‘primary’ form, ‘antiphospholipid syndrome,’ without any known systemic disease or may occur in the context of systemic lupus erythematosus (SLE), ‘SLE-related APS’. APS may affect any organ system and displays a broad spectrum of thromb

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42

Alikhan, Raza. Prothrombotic conditions. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0285.

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The term thrombophilia is used to describe an individual who has a tendency to develop thrombosis. Arterial thrombosis is usually linked with classical risk factors such as age, smoking, hypertension, hyperlipidaemia, or diabetes; a thrombophilia assessment and workup is not usually considered in cases of arterial thrombosis. A clinically useful approach to the diagnosis and management of a patient with a venous thrombotic process is to categorize the disorder as either a primary (inherited) or secondary (acquired) hypercoagulable state. This topic addresses the diagnosis and management of pro

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43

Chan, Chee M., and Andrew F. Shorr. Prevention and management of thrombosis in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0271.

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Venous thromboembolism (VTE) represents a significant challenge in the care of critically-ill patients. Because of a combination of factors including comorbidities, their acute illness, and medical interventions, patients in the intensive care unit (ICU) face a heightened risk for VTE. In addition, because of their impaired physiological reserves, critically-ill subjects will not tolerate events, such as pulmonary emboli (PEs), well. A number of recent studies better describe the epidemiology and outcomes related to VTE acquired in the ICU. New research also explores optimal approaches for VTE

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44

(Editor), James T. Robertson, and Thaddeus S., Jr. Nowak (Editor), eds. Frontiers in Cerebrovascular Disease: Mechanisms, Diagnosis & Treatment. Futura Publishing Company, 1998.

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45

Chong, Ji Y., and Michael P. Lerario. Driving Is a Headache. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190495541.003.0022.

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Headache and hemorrhagic infarcts in patients who are hypercoagulable (including pregnancy and puerperium) should prompt an evaluation for dural venous sinus thrombosis. Intracranial venous thrombosis is a rare cause of stroke. Diagnosis is made with venous imaging such as magnetic resonance venography. Investigation for underlying prothrombotic state is needed. Treatment is typically with anticoagulation, despite hemorrhage on imaging.

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46

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Pulmonary embolism. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.1697_update_004.

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47

1931-, Robertson James T., Nowak Thaddeus S, and Princeton Conference on Cerebrovascular Disease (20th : 1996 : Memphis, Tenn.), eds. Frontiers in cerebrovascular disease: Mechanisms, diagnosis, and treatment. Futura Pub. Co., 1998.

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48

Turmel-Rodrigues, Luc, and Claude J. Renaud. Diagnostic and Interventional Radiology of Arteriovenous Accesses for Hemodialysis. Springer, 2012.

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49

Erlinge, David, and Göran Olivecrona. Diagnosis and management of ST-elevation of myocardial infarction. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0147.

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ST-elevation myocardial infarction (STEMI) is generally caused by a ruptured plaque that triggers local thrombus formation, which occludes the coronary artery. STEMI should be diagnosed rapidly, based on the combination of ST-segment elevation and symptoms of acute myocardial infarction. The main treatment objective is myocardial tissue reperfusion as quickly as possible. The preferred method of reperfusion is primary percutaneous coronary interventionif transport time is below 2 hours, and thrombolysis if longer STEMI patients with acute onset cardiogenic shock should be evaluated by echocard

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50

(Editor), G. Agnelli, and Harry R. Buller (Editor), eds. The Diagnosis of Deep Vein Thrombosis and Pulmonary Embolism (Proceedings of a Symposium, Mexico City, April 1994). S. Karger AG (Switzerland), 1995.

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