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Journal articles on the topic 'Thrombose – Diagnostic'

Author: Grafiati

Published: 4 June 2021

Last updated: 6 February 2022

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1

Delsart, D., G. Girard, N. Moulin, K. Rivron-Guillot, and H. Décousus. "Thrombose veineuse : diagnostic et traitement." EMC - Médecine d 'urgence 2, no. 1 (January 2007): 1–14. http://dx.doi.org/10.1016/s1959-5182(07)73324-x.

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2

Chacar, H. R., R. K. Hejeily, and A. Aouad. "Thrombose du sinus caverneux retard au diagnostic et complications." Archives de Pédiatrie 6 (January 1999): S540. http://dx.doi.org/10.1016/s0929-693x(99)81640-6.

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3

Maufus, M., A. Delluc, G. Le Gal, P. Quehe, D. Mottier, and L. Bressollette. "Thrombose veineuse profonde en cocarde, un nouveau diagnostic échographique." Journal des Maladies Vasculaires 34 (September 2009): S28—S29. http://dx.doi.org/10.1016/j.jmv.2009.07.037.

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4

Huret, B., S. Boulanger, and M. Bure. "Diagnostic fortuit d’une thrombose veineuse profonde en échoendoscopie bronchique." Revue des Maladies Respiratoires 36, no. 4 (April 2019): 561–62. http://dx.doi.org/10.1016/j.rmr.2018.11.003.

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5

Barbar, S. D., M. Rondeau, F. Lange, and J. C. Weber. "La thrombose de la veine azygos, un surprenant diagnostic histologique." La Revue de Médecine Interne 24 (December 2003): 476s—477s. http://dx.doi.org/10.1016/s0248-8663(03)80590-8.

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6

Courdurie, A., J. G. Fuzibet, C. Kannengiesser, E. Lainey, A. Renneville, P. Rohrlich, and T. Cluzeau. "Thrombose intra médullaire conduisant au diagnostic d’un syndrome des télomères courts." La Revue de Médecine Interne 38 (June 2017): A124—A125. http://dx.doi.org/10.1016/j.revmed.2017.03.151.

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7

Elias, A., M. Maufus, M. Elias, M. A. Sevestre, and G. Pernod. "Diagnostic de la récidive de thrombose veineuse profonde des membres inférieurs." JMV-Journal de Médecine Vasculaire 42, no. 2 (March 2017): 85. http://dx.doi.org/10.1016/j.jdmv.2017.01.072.

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8

Ouarhlent, Yamina, Hamida Laiadhi, Rabeh Chafai, and Habiba Zidani. "Thrombosis revealing malignant haemopathies. Report of 2 cases." Batna Journal of Medical Sciences (BJMS) 3, no. 2 (December 31, 2016): 132–33. http://dx.doi.org/10.48087/bjmscr.2016.3218.

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Le risque de survenue de thromboses est augmenté dans les affections hématologiques, en particulier les hémopathies malignes et ce au moment du diagnostic et pendant le traitement par chimiothérapie. Mais aussi, les thromboses peuvent précéder de plusieurs mois la survenue d’hémopathies malignes et par conséquent, ces complications thrombotiques peuvent compromettre le pronostic vital. Donc dans l’enquête étiologique des thromboses, il est judicieux de penser à une hémopathie maligne sous-jacente et de ce fait nous rapportons deux observations qui illustrent la survenue de thromboses dans les hémopathies malignes.

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9

Noël-Savina, Élise, Gilles Quéré, Sylvie Gouva, Gilles Robinet, and Renaud Descourt. "Infection et thrombose sur chambre implantable : diagnostic et prise en charge thérapeutique." Bulletin du Cancer 98, no. 9 (September 2011): 1107–18. http://dx.doi.org/10.1684/bdc.2011.1403.

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10

Constans, J., C. Boutinet, R. Salmi, J. C. Saby, P. Baudouin, M. L. Nelzy, F. Sampoux, et al. "Scores de prédiction du diagnostic de thrombose veineuse profondeen medecine de ville." La Revue de Médecine Interne 22 (December 2001): 458s—459s. http://dx.doi.org/10.1016/s0248-8663(01)80083-7.

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11

Fornecker, C., C. Griffon, V. Knauer, and D. StEphan. "Le syndrome de morel-lavallée, un diagnostic différentiel inhabituel de thrombose veineuse profonde." Journal des Maladies Vasculaires 31, no. 2 (May 2006): 98–100. http://dx.doi.org/10.1016/s0398-0499(06)76527-1.

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12

Zaouini, H., T. Guertin, C. Oppenheim, and J. F. Méder. "Interets des nouvelles sequences IRM pour le diagnostic de la thrombose veineuse cerebrale." Journal de Radiologie 89, no. 10 (October 2008): 1509. http://dx.doi.org/10.1016/s0221-0363(08)76635-6.

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13

Merminod, T., and P. de Moerloose. "Diagnostic de la thrombose veineuse profonde des membres inferieurs : performances des tests diagnostiques." Annales de Cardiologie et d'Angéiologie 51, no. 3 (2002): 135–38. http://dx.doi.org/10.1016/s0003-3928(02)00085-9.

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14

Sidibé, S., M. Touré, L. Diakité, A. Kéita, M. Kané, and I. Traoré. "Apport de l’echographie et de l’echographie Doppler dans le diagnostic de la thrombose veineuse." Journal de Radiologie 86, no. 10 (October 2005): 1448. http://dx.doi.org/10.1016/s0221-0363(05)75853-4.

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15

Grare, F., I. Barazer, O. Falguières, B. Simorre, O. Millot, E. Oziol, P. Berdagué, and J. L. Reny. "Suspicion de thrombose veineuse profonde ou d'embolie pulmonaire: intérêt d'un organigramme d'aide au diagnostic." Journal des Maladies Vasculaires 32 (March 2007): 34. http://dx.doi.org/10.1016/j.jmv.2007.01.086.

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16

Condat, Bertrand, Laurent Dugué, David Zanditenas, Jean-Marc Gornet, Soufiéne Chouaïb, Marie-Pierre Hauuy, Véronique Collot, et al. "Difficultés du diagnostic d’appendicite aiguë en cas de thrombose portale aiguë : apport du scanner." Gastroentérologie Clinique et Biologique 29, no. 12 (December 2005): 1291–93. http://dx.doi.org/10.1016/s0399-8320(05)82225-0.

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17

Tiganas, D., R. Durant, F. Raschilas, H. Blain, F. Tigoulet, N. Mitermite, P. Hemmi, and C. Jeandel. "Intérêt du score de probabilité clinique dans le diagnostic de thrombose veineuse profonde en gériatrie." La Revue de Médecine Interne 26, no. 12 (December 2005): 931–37. http://dx.doi.org/10.1016/j.revmed.2005.08.010.

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18

Danse, E., and Y. Horsmans. "Thrombose portale calcifiée dans le suivi d’un shunt porto-cave: aspect échographique et diagnostic différentiel." Journal de Radiologie 87, no. 11 (November 2006): 1696–99. http://dx.doi.org/10.1016/s0221-0363(06)74149-x.

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19

Bosson, J. L., A. Guyard, C. Rolland, and G. Pernod. "Diagnostic différentiel devant une suspicion de thrombose veineuse. Contribution de l’écho-doppler des membres inférieurs." Journal des Maladies Vasculaires 38, no. 2 (March 2013): 108–9. http://dx.doi.org/10.1016/j.jmv.2012.12.080.

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20

Tiganas, D., R. Durant, F. Raschilas, H. Blain, P. Hemmi, and C. Jeandel. "Intérêt du score de probabilité clinique dans le diagnostic de thrombose veineuse profonde en gériatrie." La Revue de Médecine Interne 24 (December 2003): 425s. http://dx.doi.org/10.1016/s0248-8663(03)80432-0.

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21

Delluc, A., L. Bressollette, B. Guias, K. Lacut, C. Delluc, G. Le Gal, and D. Mottier. "Prévalence et localisation des cancers lors du diagnostic de thrombose veineuse profonde symptomatique des membres inférieurs." La Revue de Médecine Interne 29 (June 2008): S25—S26. http://dx.doi.org/10.1016/j.revmed.2008.03.044.

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22

Vin, F. "Diagnostic étiologique sur une série de 70 patients présentant une thrombose veineuse profonde des membres inférieurs." Journal des Maladies Vasculaires 34, no. 2 (March 2009): 143. http://dx.doi.org/10.1016/j.jmv.2008.12.046.

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23

Risse, Jessie, Alix Martin-Bertaux, Alain Nicolas, Pierre Olivier, and Denis Wahl. "Tomographie par émission de positons au 18-fluorodésoxyglucose et diagnostic de récidive de thrombose veineuse profonde." La Presse Médicale 45, no. 2 (February 2016): 271–73. http://dx.doi.org/10.1016/j.lpm.2015.11.005.

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24

Boog, G., F. Moussaly, and M. P. Quere. "Diagnostic echographique en salle de naissance d'une thrombose veineuse renale du foetus a l'occasion d'une souffrance foetale." European Journal of Ultrasound 7 (February 1998): S32. http://dx.doi.org/10.1016/s0929-8266(97)80229-7.

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25

Zouaoui, W., A. Ben Hassine, N. Falfoul-Borsali, F. Boussama, C. Chammakhi-Jemli, H. Mzabi, S. Sehili-Briki, and M. H. Daghfous. "CV43 Apport de l’imagerie dans le diagnostic et le bilan d’extension d’une thrombose veineuse du membre superieur." Journal de Radiologie 87, no. 10 (October 2006): 1456. http://dx.doi.org/10.1016/s0221-0363(06)87575-x.

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26

Leblanc, C., F. Douane, G. Roussey, O. Multon, M. A. Pistorius, J. Connault, and M. Artifoni. "Diagnostic anténatal à 33 semaines d’aménorrhées d’une thrombose de la veine cave inférieure et de la veine rénale droite." Journal des Maladies Vasculaires 41, no. 2 (March 2016): 145. http://dx.doi.org/10.1016/j.jmv.2015.12.154.

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27

Boltshauser and Landau. "Aknetherapie mit Folgen – Pseudotumor cerebri durch Hypervitaminose A." Praxis 91, no. 1 (January 1, 2002): 23–26. http://dx.doi.org/10.1024/0369-8394.91.1.23.

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Le syndrome de la pseudotumeur cérébrale (PTC) consiste en une élévation anormale de la pression intracrânienne dont les causes probablement multifactorielles ne sont souvent pas élucidées. Nous décrivons le cas d'une jeune fille de 15 ans souffrant d'un syndrome PTC après un traitement prolongé à l'Arovit® (vitamin A) pour cause d'acné. Le diagnostic a été posé après qu'une élévation anormale de la pression intracrânienne ait été vérifiée et qu'un processus expansif intracrânien ainsi qu'une thrombose veineuse cérébrale aient été exclus. Une concentration plasmatique élevée de vitamine A confirme l'hypothèse que l'hypervitaminose A est à l'origine du syndrome de PTC chez cette patiente. Le traitement a consisté en des ponctions lombaires répétées et à l'administration d'acétazolamide (Diamox®). Le syndrome de PTC suite à une hypervitaminose A est une complication grave, pouvant conduire à une altération irréversible de la fonction visuelle. Les patients sous traitement aux rétinoïdes doivent faire l'objet d'un suivi médical.

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28

Katus, H. A., and J. Ruef. "Venöse Thrombose und Thrombophilie." Hämostaseologie 23, no. 04 (2003): 186–98. http://dx.doi.org/10.1055/s-0037-1619596.

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ZusammenfassungIn den Industrieländern wird die Mortalitätsstatistik von kardiovaskulären Erkrankungen angeführt, wovon wesentliche Anteile auf venöse Thrombose und Thromboembolie fallen. Ursache einer Thrombose können temporäre Faktoren oder eine permanente thrombophile Diathese sein. Neue Erkenntnisse über die Pathophysiologie der Thrombose sowie neue diagnostische Methoden erleichtern die Erstellung eines Risikoprofils bezüglich venöser Thrombosen. Im Folgenden sollen verschiedene Thrombophilierisikofaktoren hinsichtlich ihrer Bedeutung und Diagnostik beschrieben und diskutiert werden. Weiterhin soll ausgeführt werden, welche prophylaktischen und therapeutischen Maßnahmen bei Thrombophiliekonstellation oder manifester Thrombose vorzunehmen sind. Hierbei wird auf zurzeit zugelassene, aber auch auf vor der Zulassung stehende neue Antikoagulanzien eingegangen (Pentasaccharid, Melagatran), deren Verwendung eine zunehmend patientenfreundliche und kosteneffiziente Prophylaxe und Behandlung venöser thromboembolischer Komplikationen verspricht.

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29

Rudofsky, G. "Thrombosediagnostik mit Venenverschlußplethysmographie, Doppler und Lichtreflexionsrheographie." Hämostaseologie 13, no. 03 (May 1993): 112–15. http://dx.doi.org/10.1055/s-0038-1655222.

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ZusammenfassungFunktionelle Untersuchungen bei tiefer Venenthrombose mit Hilfe von Venenverschlußplethysmographie, Doppler und Lichtreflexionsrheographie werden durch die Lage und Ausdehnung der Thrombose bestimmt. Im Vergleich zur Phlebographie erlaubt die Lichtreflexionsrheographie lediglich eine orientierende Diagnostik, eine Thrombose kann mit dieser Methode nicht ausgeschlossen werden. Verwendet man Doppler-Sonographie und Venenverschlußplethysmographie, so läßt sich die Sicherheit der Diagnose erheblich steigern. Dennoch können alte und weniger ausgedehnte Thrombosen mit diesen Methoden nicht ausreichend diagnostiziert werden, so daß eine sichere Diagnose oder eine Ausschlußdiagnostik nicht gegeben ist.

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30

Stefano, Valerio De, and Ida Martinelli. "Rare thromboses of cerebral, splanchnic and upper-extremity veins." Thrombosis and Haemostasis 103, no. 06 (2010): 1136–44. http://dx.doi.org/10.1160/th09-12-0873.

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SummaryVenous thrombosis typically involves the lower extremity circulation. Rarely, it can occur in the cerebral or splanchnic veins and these are the most frightening manifestations because of their high mortality rate. A third site of rare venous thrombosis is the deep system of the upper extremities that, as for the lower extremity, can be complicated by pulmonary embolism and post-thrombotic syndrome. The authors conducted a narrative review focused on clinical manifestations, risk factors, and treatment of rare venous thromboses. Local risk factors such as infections or cancer are frequent in thrombosis of cerebral or portal veins. Upper extremity deep-vein thrombosis is mostly due to local risk factors (catheter- or effort-related). Common systemic risk factors for rare venous thromboses are inherited thrombophilia and oral contraceptive use; chronic myeloproliferative neoplasms are closely associated with splanchnic vein thrombosis. In the acute phase rare venous thromboses should be treated conventionally with low-molecular-weight heparin. Use of local or systemic fibrinolysis should be considered in the case of clinical deterioration in spite of adequate anticoagulation. Anticoagulation with vitamin K-antagonists is recommended for 3–6 months after a first episode of rare venous thrombosis. Indefinite anticoagulation is recommended for Budd-Chiari syndrome, recurrent thrombosis or unprovoked thrombosis and permanent risk factors. In conclusion, the progresses made in the last couple of decades in diagnostic imaging and the broadened knowledge of thrombophilic abnormalities improved the recognition of rare venous thromboses and the understanding of pathogenic mechanisms. However, the recommendations for treatment mainly derive from observational studies.

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31

Žigon, Polona, Saša Čučnik, Aleš Ambrožič, Tanja Kveder, Snežna Sodin Šemrl, Blaž Rozman, and Borut Božič. "Detection of Antiphosphatidylserine/Prothrombin Antibodies and Their Potential Diagnostic Value." Clinical and Developmental Immunology 2013 (2013): 1–8. http://dx.doi.org/10.1155/2013/724592.

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Antiprothrombin antibodies, measured with phosphatidylserine/prothrombin complex (aPS/PT) ELISA, have been reported to be associated with antiphospholipid syndrome (APS). They are currently being evaluated as a potential classification criterion for this autoimmune disease, characterized by thromboses and obstetric complications. Given the present lack of clinically useful tests for the accurate diagnosis of APS, we aimed to evaluatein-houseand commercial assays for determination of aPS/PT as a potential serological marker for APS. We screened 156 patients with systemic autoimmune diseases for antibodies against PS/PT,β2-glycoprotein I, cardiolipin and for lupus anticoagulant activity. We demonstrated a high degree of concordance between the concentrations of aPS/PT measured with thein-houseand commercial assays. Both assays performed comparably relating to the clinical manifestations of APS, such as arterial and venous thromboses and obstetric complications. IgG aPS/PT represented the strongest independent risk factor for the presence of obstetric complications, among all tested aPL. Both IgG and IgM aPS/PT were associated with venous thrombosis, but not with arterial thrombosis. Most importantly, the association between the presence of IgG/IgM aPS/PT and lupus anticoagulant activity was highly significant. Taken together, aPS/PT antibodies detected with thein-houseor commercial ELISA represent a promising serological marker for APS and its subsets.

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32

Labropoulos, N., M. Bishawi, A. Gasparis, A. Tassiopoulos, and S. Gupta. "Great saphenous vein stump thrombosis after harvesting for coronary artery bypass graft surgery." Phlebology: The Journal of Venous Disease 29, no. 4 (October 3, 2012): 215–19. http://dx.doi.org/10.1258/phleb.2012.012094.

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Objectives To determine the rate of superficial venous thrombosis in patients undergoing great saphenous vein (GSV) harvesting for coronary artery bypass surgery (CABG). Methods Post-CABG patients with suspected lower-extremity thrombosis underwent duplex scanning. Thrombus in the saphenofemoral junction stump was noted, and thrombus extension and associated complications collected. Results Out of 2335 patients who underwent CABG in five years, 98 patients presented with signs and symptoms of lower-extremity thrombosis. Thrombosis was present in 19 (19.4%) of these patients, 15 of which had a thrombus in the GSV. Five patients had significant signs and symptoms of pulmonary embolism (PE). On objective diagnostic imaging, three of them had a PE. Conclusion Patients undergoing great saphenous vein harvesting for CABG are at an increased risk of developing superficial vein thrombosis especially at the saphenous stump. Given the increased risk of deep vein thrombosis and PE, further studies investigating this topic are warranted.

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33

Kayes, Oliver, Nina Patrick, and Anup Sengupta. "A peculiar case of bilateral, spontaneous thromboses of the pampiniform plexi." Annals of The Royal College of Surgeons of England 92, no. 7 (October 2010): e22-e23. http://dx.doi.org/10.1308/147870810x12822015504400.

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Acute thrombosis of the pampiniform plexus is an uncommon clinical problem which causes a diagnostic conundrum in men presenting urgently to medical personnel. We present an unusual case of a young man who presented with metachronous thromboses of his pampiniform plexi and review the literature to explore potential aetiologies and therapeutic strategies.

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34

Tan, Melanie, Gerben C. Mol, Cornelis J. van Rooden, Frederikus A. Klok, Robin E. Westerbeek, Antonio Iglesias del Sol, Marcel A. van de Ree, Albert de Roos, and Menno V. Huisman. "Magnetic resonance direct thrombus imaging differentiates acute recurrent ipsilateral deep vein thrombosis from residual thrombosis." Blood 124, no. 4 (July 24, 2014): 623–27. http://dx.doi.org/10.1182/blood-2014-04-566380.

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Key Points Diagnostic management of ipsilateral recurrent DVT of the leg is complicated because residual DVT is common and mimics acute DVT on CUS. MRDTI is able to reproducibly distinguish acute ipsilateral recurrent DVT from 6-month-old chronic residual thrombi in the leg veins.

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35

Nemani, Lalita, and Satish Killi. "Right Atrial Thrombus Mimicking Myxoma." Indian Journal of Cardiovascular Disease in Women WINCARS 02, no. 04 (December 2017): 086–90. http://dx.doi.org/10.1055/s-0038-1622964.

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AbstractRight atrial mass could be a tumor, thrombus, or vegetation, and it often poses a diagnostic dilemma. Accurate diagnosis is crucial to planning the correct management strategy. However, despite the advanced and sophisticated diagnostic modalities available, differentiating intracardiac masses could still be challenging. Clinical presentation leads to the appropriate conduit of investigations, and histopathology is confirmatory. When the diagnostic dilemma persists even after all efforts, clinical scenario should be strongly reconsidered, especially in unique clinical settings before concluding a diagnosis. In this article, the authors describe the case of a young woman with history of peripheral arterial thrombosis, who underwent surgical excision of a right atrial mass diagnosed as right atrial myxoma. She later presented with massive pulmonary embolism and deep vein thrombosis of the lower limbs. In view of her extensive thrombotic history, her entire case was reviewed including the histopathology slides, and the diagnosis of right atrial thrombus was considered and confirmed. On further workup for thrombotic state, she was found to have protein C deficiency.

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36

Ng, Carol, and Gloria Ting Wong. "Round Ligament Varicosity Thrombosis Presenting as an Irreducible Inguinal Mass in a Postpartum Woman." Journal of Clinical Imaging Science 9 (June 14, 2019): 28. http://dx.doi.org/10.25259/jcis-19-2019.

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Round ligament varicosities occur almost exclusively in pregnant and postpartum women and present similarly to inguinal hernias clinically, thus posing diagnostic challenges to clinicians. The distinction of the two conditions is important as round ligament varicosities do not require surgery while inguinal hernias do. Rarely, round ligament varicosites may be complicated by thrombosis or hemorrhage. In this case report, we present a rare case of round ligament varicosity (RLV) which has been complicated by thrombosis. The radiological features on ultrasound and contrast computed tomography of a thrombosed RLV are presented.

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37

Ng, Arnold C. T., David R. Holmes, Michael J. Mack, Victoria Delgado, Raj Makkar, Philipp Blanke, Jonathon A. Leipsic, Martin B. Leon, and Jeroen J. Bax. "Leaflet immobility and thrombosis in transcatheter aortic valve replacement." European Heart Journal 41, no. 33 (September 1, 2020): 3184–97. http://dx.doi.org/10.1093/eurheartj/ehaa542.

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Abstract Transcatheter aortic valve replacement (TAVR) has grown exponentially worldwide in the last decade. Due to the higher bleeding risks associated with oral anticoagulation and in patients undergoing TAVR, antiplatelet therapy is currently considered first-line antithrombotic treatment after TAVR. Recent studies suggest that some patients can develop subclinical transcatheter heart valve (THV) thrombosis after the procedure, whereby thrombus forms on the leaflets that can be a precursor to leaflet dysfunction. Compared with echocardiography, multidetector computed tomography is more sensitive at detecting THV thrombosis. Transcatheter heart valve thrombosis can occur while on dual antiplatelet therapy with aspirin and thienopyridine but significantly less with anticoagulation. This review summarizes the incidence and diagnostic criteria for THV thrombosis and discusses the pathophysiological mechanisms that may lead to thrombus formation, its natural history, potential clinical implications and treatment for these patients.

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38

Dharmarajah, B., V. Sounderajah, SP Rowland, ELS Leen, and AH Davies. "Aging techniques for deep vein thrombosis: a systematic review." Phlebology: The Journal of Venous Disease 30, no. 2 (March 25, 2014): 77–84. http://dx.doi.org/10.1177/0268355514528691.

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Deep vein thrombosis is common with an incidence of 1 in 1000. Acute thrombus removal for extensive proximal deep vein thrombosis using catheter-directed techniques highlights the need for accurate assessment of thrombus age. This systematic review summarises experimental and clinical evidence of imaging techniques for aging deep vein thrombosis. Ultrasound elastography and magnetic resonance imaging were highlighted as the most studied imaging modalities. Elastography was shown to distinguish between acute and chronic clots, despite demonstrating difficulty in accurate aging of clots older than 10 days in rat models. Elastography is noted as a feasible adjunct to current first-line imaging for deep vein thrombosis using duplex ultrasonography. Combinations of magnetic resonance imaging techniques can identify acute, sub-acute and chronic thrombi using endogenous contrast agents and provide objective standardisation of the diagnostic process, with reduced onus upon operator dependency. Further validation is required of these novel imaging techniques prior to clinical implementation for deep vein thrombosis aging.

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39

Khalifeh, Ali, Tamara Khashab, Michael Huffner, Zahra N. Rezvani, Justin Kwan, and Shahab Toursavadkohi. "Radial neuropathy following arterial line removal: A rare complication from a routine ICU procedure." SAGE Open Medical Case Reports 6 (January 1, 2018): 2050313X1876074. http://dx.doi.org/10.1177/2050313x18760740.

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Radial artery thrombosis is a rare complication of cannulation. There are no reported cases of acute thrombosis and severe acute neuropathy in the setting of cannula discontinuation. We report a case of acute radial nerve mono-neuropathy following thrombosis after radial arterial line removal. The thrombus was immediately evident on exam and diagnostic imaging after cannula discontinuation. The patient was consented and promptly taken to OR for immediate repair. Mild radial neuropathy persisted despite immediate repair. Immediate recognition of signs and symptoms is essential for diagnosis and management, especially in the high-risk population.

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40

Ranard, Lauren S., David J. Engel, Ajay J. Kirtane, and Amirali Masoumi. "Coronary and cerebral thrombosis in a young patient after mild COVID-19 illness: a case report." European Heart Journal - Case Reports 4, no. 5 (October 1, 2020): 1–5. http://dx.doi.org/10.1093/ehjcr/ytaa270.

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Abstract Background COVID-19 has spread worldwide and has caused significant morbidity and mortality. Myocardial injury and thrombo-embolism are known complications for those with severe forms of disease. The incidence and risk factors for these complications for those patients who are asymptomatic or with mild forms of COVID-19 is unknown. Case summary In this report we describe the case of a 35-year-old man with no past cardiac history who presented with chest pain and a high-sensitivity troponin level of 386 ng/L in the context of an unspecified mild viral illness 1 month previously. Diagnostic evaluation revealed a new cardiomyopathy, left ventricular thrombus, and mid right coronary artery thrombosis. The coronary thrombosis was treated with thrombectomy. SARS-CoV-2 antibodies returned positive. He initially did well post-procedure; however, prior to discharge, he developed a second arterial thrombo-embolism event, a middle cerebral artery stroke. He was treated with thrombectomy and remains hospitalized. Discussion Recognition that mild COVID-19 can be complicated by subsequent cardiac injury and/or coagulopathy is important. As more people recover from this viral illness and return to normal activity levels, discussion among cardiac experts has begun regarding screening for occult myocardial injury in those who plan to resume competitive athletic activity. This case highlights the need for investigation regarding (i) the duration of thrombophilia after recovery from illness; (ii) the population that should receive thromboprophylaxis; and (iii) the duration of thromboprophylaxis therapy for COVID-19.

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Zubarev, A. R., N. V. Krivosheeva, I. V. Rychkova, A. K. Demidova, and S. E. Nikolskiy. "RECENT DIAGNOSTIC OPPORTUNITIES IN ULTRASOUND DIAGNOSTICS OF LOWER EXTREMITIES DEEP VEINS THROMBOSIS." Russian Electronic Journal of Radiology 6, no. 2 (2016): 44–53. http://dx.doi.org/10.21569/2222-7415-2016-6-2-44-53.

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Mannhalter, Ch. "Molekularbiologie und Hämostase." Hämostaseologie 28, no. 05 (2008): 272–88. http://dx.doi.org/10.1055/s-0037-1617176.

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ZusammenfassungFür die Diagnostik von Erkrankungen spielen molekularbiologische Methoden bei schweren angeborenen Krankheiten (z. B. Hämophilie A oder B) eine wichtige Rolle. Auch zur Diagnostik polygenetischer Erkrankungen (z. B. venöse und arterielle Thrombosen) sind sie unentbehrlich. Neben der Analyse der zwei häufigsten genetischen Defekte (Inversion im Intron 22 und Intron 1) im Faktor-VIII-Gen als Ursache der schweren Hämophilie A wurde in den vergangenen Jahren die Sequenzierung des Faktor-VIII-Gens in mehreren Zentren eingeführt und wird nun in der Hämophilie- und Überträgerinnen- Diagnostik eingesetzt. Bei Patienten mit Thrombophilie trägt der Nachweis von Mutationen im Protein-C- und Protein-S-Gen zur Verbesserung der Diagnostik bei bekanntem familiären Protein-C- bzw. -S-Mangel bei. Die Analysen der Arg506Gln-Mutation im Faktor-V-Gen (Faktor-V-Leiden) und die 20210G>A-Mutation im Prothrombin-Gen, die das Risiko für venöse Thrombose beeinflussen, können potenziell helfen, das individuelle Risiko für eine Thrombose bzw. Rezidivthrombose besser einzuschätzen. Allerdings führt die unkritische Untersuchung genetischer Ursachen der Thrombose zu keinem wesentlichen Informationsgewinn hinsichtlich Behandlung und Beratung der Patienten und kostet Zeit und Geld. Daher sollen immer nur jene Tests durchgeführt werden, die medizinische bzw. therapeutische Konsequenzen nach sich ziehen. Trotz der Bedeutung der molekulargenetischen Diagnostik sind die Einsatzmöglichkeiten der Mutationsdiagnostik im klinischen Alltag eines Gerinnungslabors begrenzt. Große Studien haben gezeigt, dass eine Mutation nicht bei jedem Menschen die gleiche Auswirkung hat, da endogene und exogene modulierende Faktoren den Phänotyp beeinflussen. Da sehr wenig über modulierende Faktoren bekannt ist, ist es häufig schwierig, die Auswirkung einer Mutation in ihrer Tragweite zu bewerten. Es ist daher von außerordentlicher Wichtigkeit, die Forschung voranzutreiben, um Gen- Gen- und Gen-Umwelt-Interaktionen zu verstehen, damit in Zukunft eine zuverlässige Interpretation der Mutationsergebnisse möglich wird.

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Tølbøll Sørensen, Anne Louise, Magalie Rolland, Jacob Hartmann, Zitta Barrella Harboe, Casper Roed, Tomas Ø. Jensen, Lilian Kolte, et al. "A case of thrombocytopenia and multiple thromboses after vaccination with ChAdOx1 nCoV-19 against SARS-CoV-2." Blood Advances 5, no. 12 (June 17, 2021): 2569–74. http://dx.doi.org/10.1182/bloodadvances.2021004904.

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Recently, reports of severe thromboses, thrombocytopenia, and hemorrhage in persons vaccinated with the chimpanzee adenovirus-vectored vaccine (ChAdOx1 nCoV-19, AZD1222, Vaxzevria; Oxford/AstraZeneca) against severe acute respiratory syndrome coronavirus 2 have emerged. We describe an otherwise healthy 30-year-old woman who developed thrombocytopenia, ecchymosis, portal vein thrombosis, and cerebral venous sinus thrombosis the second week after she received the ChAdOx1 nCoV-19 vaccine. Extensive diagnostic workup for thrombosis predispositions showed heterozygosity for the prothrombin mutation, but no evidence of myeloproliferative neoplasia or infectious or autoimmune diseases. Her only temporary risk factor was long-term use of oral contraceptive pills (OCPs). Although both the prothrombin mutation and use of OCPs predispose to portal and cerebral vein thrombosis, the occurrence of multiple thromboses within a short time and the associated pattern of thrombocytopenia and consumption coagulopathy are highly unusual. A maximum 4T heparin-induced thrombocytopenia (HIT) score and a positive immunoassay for anti-platelet factor 4/heparin antibodies identified autoimmune HIT as a potential pathogenic mechanism. Although causality has not been established, our case emphasizes the importance of clinical awareness. Further studies of this potentially new clinical entity have suggested that it should be regarded as a vaccine-induced immune thrombotic thrombocytopenia.

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DIACONU, Camelia C., Mădălina ILIE, and Mihaela Adela IANCU. "Upper extremity deep venous thrombosis: risk factors, diagnosis, treatment." Romanian Journal of Medical Practice 11, no. 1 (March 31, 2016): 28–32. http://dx.doi.org/10.37897/rjmp.2016.1.5.

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Upper extremity deep venous thrombosis is a condition with increasing prevalence, with high risk of morbidity and mortality, due to embolic complications. In the majority of the cases, thrombosis involves more than one venous segment, most frequently being affected the subclavian vein, followed by internal jugular vein, brachiocephalic vein and basilic vein. Upper extremity deep venous thrombosis in patients without risk factors for thrombosis is called primary deep venous thrombosis and includes idiopathic thrombosis and effort thrombosis. Deep venous thrombosis of upper extremity is called secondary when there are known risk factors and it is encountered mainly in older patients, with many comorbidities. The positive diagnosis is established only after paraclinical and imaging investigations, ultrasonography being the most useful diagnostic method. The most important complication, with high risk of death, is pulmonary embolism. Treatment consists in anticoagulant therapy, for preventing thrombosis extension and pulmonary embolism.

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Sayko, O., A. Bohdan, and J. Malankevich. "Chronic headache as a clinical manifestation of cerebral venous sinus thrombosis." INTERNATIONAL NEUROLOGICAL JOURNAL 17, no. 1 (April 13, 2021): 50–54. http://dx.doi.org/10.22141/2224-0713.17.1.2021.226919.

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Patients with cerebral venous thrombosis (CVT) pre-sent with a range of symptoms, which varies widely from headache to coma. Chronic occlusion of venous dural sinuses leads to the initiation of compensation processes, such as vascular collatera-lization, recanalization of the organized thrombus. Consequently, their efficacy determines disease course and severity. The purpose of our study was to highlight the clinical challenges while managing a patient with subcompensated CVT. Taking into account the fact that mild severity is observed in 20 % of cases, eventually, about a quarter of patients presenting with the headache of unclear etio-logy becomes unnoticed and results in low quality of life. To analyze the disease outcome and prognosis, we accurately studied compensation mechanisms, which occur in a patient due to cerebral venous sinus thrombosis, notably venous collateral circulation and thrombus recanalization. However, the most important “pitfall” of chronic dural sinuses occlusion, when not appropriately treated, is the fact that hemodynamic compensation also leads to a high risk of cerebrovascular events, which may lead to death or disabi-lity. The patient with cerebral venous thrombosis of the transverse and sigmoid sinuses manifested migraine-like headache with a vegetative issue and signs of increased intracranial pressure. Due to the absence of anticoagulant therapy, venous drainage through collateral pathways and recanalization of thrombosed sinuses were insufficient to maintain a cerebral blood supply, resulting in diffuse cerebral edema, secondary brain injury. After an accurate survey, diagnostic procedures, prescribing of individualized treatment, and long-term follow-up with correction of therapy the patient experienced regression of CVT symptoms. Cerebral MRI venography is an important tool for the diagnosis and prediction of the prognosis of this condition. Thus, patients with a long history of untreatable headache and signs of intracranial hypertension should undergo this procedure. To consider intracranial vascular events and outcomes, the venous hemodynamics should be evaluated. Moreover, it is often necessary to examine cerebral veins and sinuses, when hemorrhagic stroke does not correspond to cerebral arterial territories and has unclear etiology.

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Abel, Gregory A., Daniel J. DeAngelo, Jean M. Connors, Lynette M. Sholl, Ronald P. McCaffrey, and Janina A. Longtine. "JAK2 V617F in Patients with Idiopathic Thromboses in Common Locations." Blood 110, no. 11 (November 16, 2007): 1634. http://dx.doi.org/10.1182/blood.v110.11.1634.1634.

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Abstract Background: Polycythemia vera and essential thrombocytosis, two of the myeloproliferative disorders (MPD) often characterized by the recurrent somatic missense mutation JAK2 V617F, are associated with an increased risk of arterial and venous thrombosis. Although recent anecdotal reports have suggested the possibility of latent MPDs by finding JAK2 mutations in patients with abdominal venous thromboses (AVT), the usefulness of routine JAK2 V617F testing has not been assessed in a cohort of general hospital patients with idiopathic thromboses in common locations. Methods: To assess the utility of JAK2 V617F testing in patients undergoing thrombophilia workup, we identified all patients with negative molecular diagnostic tests for the Factor V Leiden (FVL) mutation and the prothrombin gene mutation (G20210A) over the course of one year at the Brigham and Women’s Hospital. We then determined, by medical record review, the nature of the thrombotic events for which they were being evaluated, and tested their nucleated white blood cell DNA for the JAK2 V617F mutation using the amplification refractory mutation system (ARMS)-PCR assay (which has a lower limit of detection of 5% mutant alleles). Results: From Februrary 2006 to February 2007, 111 patients had negative tests for both FVL and G20210A. Of these, our chart review revealed that 65 had documented thromboses without a history of malignancy or MPD. None of these were found to have the JAK2 V617F mutation. Laboratory hypercoaguability studies for thrombosis by type (from medical record review) were as follows: Protein C deficiency (%) Protein S deficiency (%) Anti-thrombin III deficiency (%) Antiphospholipid Abs (%) Elevated homocysteine (%) JAK2 V617 mutation (%) None (%) CVA/Stroke (n=10) 0 20 0 10 0 0 70 DVT/PE (n=45) 11 20 2 4 13 0 60 AVT (n=6) 33 33 0 0 17 0 33 Other Arterial Thrombosis (n=4) 0 25 0 0 25 0 50 In terms of clinical risk factors, 27.6% of patients with documented thromboses had had surgery within the prior month, 26.2% were current smokers, and 31.4% of female patients were either pregnant or taking oral contraceptives. 29.2% of all patients had no clinical or laboratory thrombophilic risk factors. Conclusions: About two-thirds of our general hospital patients with thrombosis and negative tests for FVL and G20210A had another underlying thrombophilic risk factor. Adding JAK2 V617F mutation testing as a routine component of the thrombophilia diagnostic workup is not recommended, as it is unlikely to reveal evidence of a putative latent MPD.

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Kiderlen, M. J. "Diagnostik und Therapie der Thrombose." Phlebologie 42, no. 05 (May 2013): 283–86. http://dx.doi.org/10.12687/phleb2168-5-2013.

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Guest, Will, Dipanka Sarma, and Thomas Marotta. "Partial thrombosis of an anterior communicating artery aneurysm prior to endovascular coiling, with intra-procedural distal thrombus embolization." Interventional Neuroradiology 23, no. 6 (September 24, 2017): 589–93. http://dx.doi.org/10.1177/1591019917733124.

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Thromboembolic stroke from migration of thrombus formed in non-giant intracranial aneurysms is a recognized but rare event. We describe a case of partial thrombosis of a 7 mm anterior communicating artery aneurysm, which embolized to the right callosomarginal artery in the brief time interval between two sequential diagnostic angiograms performed as part of elective endovascular coiling, and before any instrumentation had been advanced into the intracranial circulation. To our knowledge, this is the first reported case of aneurysmal thrombus embolization observed angiographically in near real time.

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Bosch, Floris T. M., Marcello Di Nisio, Harry R. Büller, and Nick van Es. "Diagnostic and Therapeutic Management of Upper Extremity Deep Vein Thrombosis." Journal of Clinical Medicine 9, no. 7 (July 1, 2020): 2069. http://dx.doi.org/10.3390/jcm9072069.

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Upper extremity deep vein thrombosis (UEDVT) accounts for 5% of all deep vein thromboses (DVTs). UEDVT may be complicated by post thrombotic syndrome and pulmonary embolism, and early recognition and prompt start of anticoagulant treatment are key. Primary UEDVT, also known as Paget-von Schrötter syndrome, is associated with repeated or sudden physical activity of the upper arm and venous outflow obstruction due to anatomical variations. Secondary UEDVT is often associated with malignancy or use of intravenous devices, such as central venous catheters or pacemaker leads. Although the diagnosis and treatment of UEDVT have many similarities with DVT of the lower extremities, knowledge of specific aspects regarding UEDVT is important to guide optimal management. In this review, we will discuss the epidemiology, diagnosis, and treatment of UEDVT based on the current literature.

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Oliveira, Nelson, Emanuel Dias, Ricardo Lima, Fernando Oliveira, and Isabel Cássio. "Primary Iliac Venous Leiomyosarcoma: A Rare Cause of Deep Vein Thrombosis in a Young Patient." Case Reports in Medicine 2011 (2011): 1–5. http://dx.doi.org/10.1155/2011/123041.

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Introduction. Primary venous tumours are a rare cause of deep vein thrombosis. The authors present a case where the definitive diagnosis was delayed by inconclusive complementary imaging.Clinical Case. A thirty-seven-year-old female presented with an iliofemoral venous thrombosis of the right lower limb. The patient had presented with an episode of femoral-popliteal vein thrombosis five months before and was currently under anticoagulation.Phlegmasia alba dolensinstalled progressively, as thrombus rapidly extended to the inferior vena cava despite systemic thrombolysis and anticoagulation. Diagnostic imaging failed to identify the underlying aetiology of the deep vein thrombosis. The definitive diagnosis of primary venous leiomyosarcoma was reached by a subcutaneous abdominal wall nodule biopsy.Conclusion. Primary venous leiomyosarcoma of the iliac vein is a rare cause of deep vein thrombosis, which must be considered in young patients with recurrent or refractory to treatment deep vein thrombosis.

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