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Relevant bibliographies by topics / Thyroid stimulating immunoglobulin

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Academic literature on the topic 'Thyroid stimulating immunoglobulin'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Thyroid stimulating immunoglobulin"

1

Zhou, Yulin, Mengxi Zhou, Yicheng Qi, Weiqing Wang, Xinxin Chen, and Shu Wang. "The prognostic value of thyroid-stimulating immunoglobulin in the management of Graves’ disease." Therapeutic Advances in Endocrinology and Metabolism 12 (January 2021): 204201882110449. http://dx.doi.org/10.1177/20420188211044943.

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Background: The bioassay of thyroid-stimulating immunoglobulin was reported to have a similar performance to the commonly used thyroid-stimulating hormone binding inhibition assay, also known as thyroid receptor antibody assay. The normal reference range of thyroid receptor antibody levels indicates the withdrawal of anti-thyroid drugs in the recent clinical guidelines. Methods: A prospective, longitudinal observational study was conducted to evaluate the prognostic value of thyroid-stimulating immunoglobulin in patients with Graves’ disease. Results: A total of 77 patients with Graves’ diseas

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Tamai, Hajime, Kanji Kasagi, Osamu Mizuno, et al. "Thyroid-stimulating antibody and thyrotropin-binding inhibitory immunoglobulin activity in hypothyroid patients who subsequently developed thyrotoxicosis." Acta Endocrinologica 122, no. 4 (1990): 499–504. http://dx.doi.org/10.1530/acta.0.1220499.

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Abstract. Although abnormal thyroid-stimulating and -blocking antibodies have been demonstrated in hyperthyroid and hypothyroid patients with autoimmune thyroid disorders, a direct correlation is not always observed. Thyroid-stimulating antibody, thyrotropinbinding inhibitory immunoglobulin, and thyroid-stimulating blocking antibody levels were determined in three hypothyroid patients who subsequently developed hyperthyroidism. Thyroid-stimulating antibodies levels were normal in one, elevated in another, and unmeasured in the third hypothyroid patient, but became elevated in all patients with

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3

Jiang, Nai-Siang, Virgil F. Fairbanks, and Ian D. Hay. "Assay for Thyroid Stimulating Immunoglobulin." Mayo Clinic Proceedings 61, no. 9 (1986): 753–55. http://dx.doi.org/10.1016/s0025-6196(12)62778-5.

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4

Woeber, K. A. "Relationship between thyroid stimulating hormone and thyroid stimulating immunoglobulin in Graves’ hyperthyroidism." Journal of Endocrinological Investigation 34, no. 3 (2010): 222–24. http://dx.doi.org/10.1007/bf03347070.

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OCHI, YUKIO, TAKEHIRO INUI, TSUYOSHI KOUKI, KEI YAMASHIRO, TAKASHI HACHIYA, and YOSHIHIRO KAJITA. "Thyroid Stimulating Immunoglobulin(TSI) in Graves' Disease." Endocrine Journal 45, no. 6 (1998): 701–8. http://dx.doi.org/10.1507/endocrj.45.701.

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6

ANDO, Michiyasu, Kazuyuki YAMAUCHI, Hiroshi TANAKA, et al. "Thyroid Stimulating Immunoglobulin Bioassay Using Cultured Normal Human Thyroid Cells." Folia Endocrinologica Japonica 61, no. 8 (1985): 847–58. http://dx.doi.org/10.1507/endocrine1927.61.8_847.

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7

Berardo, Jeronimo, Leon A. Fogelfeld, Dan V. Mihailescu, Maria E. Padilla Sorto, and Donald W. Trepashko. "PSAT275 A Rare Case of Thyroid-Stimulating Immunoglobulin-Induced Thyrotoxicosis in Poorly Differentiated Metastatic Thyroid Cancer." Journal of the Endocrine Society 6, Supplement_1 (2022): A884. http://dx.doi.org/10.1210/jendso/bvac150.1830.

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Abstract We report case of a rare of hyperthyroidism secondary to functional metastatic thyroid carcinoma, which is an infrequent phenomenon that can be challenging to diagnose and treat, as low thyroid-stimulating hormone (TSH) levels can suppress the development and growth of differentiated thyroid carcinoma cells. Previous studies have reported that hyperfunctioning thyroid carcinoma may present as autonomous functioning thyroid nodules (AFTN) within the thyroid gland, or as functioning lesions in metastatic foci, which is our case. Functional thyroid carcinomas are capable of absorbing iod

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8

Baek, Han-Sang, and Dong-Jun Lim. "Interpretation of Thyroid Autoantibodies in Hyperthyroidism." Korean Journal of Medicine 98, no. 3 (2023): 132–36. http://dx.doi.org/10.3904/kjm.2023.98.3.132.

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Thyrotoxicosis is a clinical state with a variety of various etiologies that results from excess thyroid hormones, including hyperthyroidism and thyroiditis. Graves' disease (GD) is a well-known autoimmune thyroid disease that causes hyperthyroidism, and its pathogenesis is mainly driven by the thyroid-stimulating hormone receptor antibody (TSHRAb), which is highly specific for GD. Measuring the TSHRAb is a fast and accurate diagnostic tool for GD and has been used to monitor disease activity and the treatment response. However, conventional TSH-binding inhibitory immunoglobulin (TBII) does no

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9

Nishikawa, Mitsushige, Masayoshi Yoshimura, Nagaoki Toyoda, et al. "Correlation of orbital muscle changes evaluated by magnetic resonance imaging and thyroid-stimulating antibody in patients with Graves' ophthalmopathy." Acta Endocrinologica 129, no. 3 (1993): 213–19. http://dx.doi.org/10.1530/acta.0.1290213.

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To evaluate the relationship between eye changes and autoantibody to the thyrotropin receptor in patients with Graves' disease, we evaluated the eye changes using magnetic resonance imaging and the results were correlated with thyroid-stimulating antibody, thyrotropin binding inhibitor immunoglobulin and thyroid growth activity. Subjects were 15 patients with Graves' disease who had Graves' ophthalmopathy, including exophthalmos and other signs and symptoms, and nine patients without ophthalmopathy; all were maintained in a euthyroid state by antithyroid drugs. The thyrotropin-binding inhibito

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Goretzki, Peter E., Michael West, Rainer Koob, Christine Koller, K. Joseph, and Hans-Dietrich Röher. "Adenylate cyclase stimulation and [3H]thymidine incorporation in human thyroid tissues and thyrocyte cultures: The effect of IgG preparation from patients with different thyroid disorders." Acta Endocrinologica 116, no. 1_Suppl (1987): S281—S287. http://dx.doi.org/10.1530/acta.0.114s281.

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Abstract. Primary cell cultures of normal and adenomatous human thyroid tissues were incubated with TSH or ammonium sulphate precipited IGG fractions (1 mg/ml) of sera from patients with different thyroid diseases (Graves' disease: active n = 7 in remission n = 12; thyroid autonomy n = 39; simple euthyroid goitre n = 15) and were compared to controls (n = 26). [3H]thymidine incorporation in primary thyrocyte cultures demonstrated a typical bell shape curve after incubation with EGF and TSH with a maximal effect at 10–100 μIU/ml. This effect, however, was inconsistent and positive only in 2 of

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Book chapters on the topic "Thyroid stimulating immunoglobulin"

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Wenzel, B. E., M. Dwenger, T. Mansky, U. Engel, V. Bay, and P. C. Scriba. "Thyrotrophin and Growth Promoting Immunoglobulin(TGI) of FRTL-5 Cells Have No Growth Stimulating Activity on Human Thyroid Epithelial Cell Cultures." In Thyroid Autoimmunity. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-0945-1_60.

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Schatz, H., I. Ludwig, F. Wiss, and P. E. Goretzki. "Evidence For Intrathyroidal Production of Thyroid Growth-Stimulating Immunoglobulins." In Thyroid Autoimmunity. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-0945-1_58.

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Bech, K., U. Feldt-Rasmussen, H. Bliddal, et al. "Thyroid Stimulating Immunoglobulins without Evidence of in Vivo Thyroid Stimulation in Some Non-Thyroid Autoimmune Diseases." In Thyroid Autoimmunity. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-0945-1_52.

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Filetti, Sebastiano, Giuseppe Damante, Daniela Foti, Rosaria Catalfamo, and Riccardo Vigneri. "The Effect of Thyroid Stimulating Immunoglobulins (TSI) on Thyroid Camp: Comparison with TSH Activity." In Thyroid Autoimmunity. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-0945-1_53.

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Halpern, Alfredo, and Geraldo Medeiros-Neti. "The Significance of Immunoglobulins Related to Stimulation of Thyroid Growth in Patients with Endemic Goiter." In Thyroid Autoimmunity. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-0945-1_55.

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Schatz, H., H. Stracke, R. G. Bretzel, H. Müller, and S. F. Grebe. "Thyroid growth stimulating immunoglobulins: Clinical significance in euthyroid goitre and relationship to thyrotropin-binding inhibiting antibodies." In Thyrotropin, edited by G. Leb, A. Passath, O. Eber, and H. Höfler. De Gruyter, 1987. http://dx.doi.org/10.1515/9783110867398-041.

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7

Molnár, Ildikó. "Deiodinase Enzymes and Their Activities in Graves’ Hyperthyroidism." In Graves' Disease [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.97007.

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The origin of hyperthyroidism in Graves’ disease was displayed demonstrating the complexity of the processes. The role of stimulating TSH receptor antibodies is the one factor for the production of increased thyroidal T3 and T4. The T3 and T4 formation in colloid-embedded thyroglobulin and the activities of thyroidal deiodinases [type 1 (DIO1) and type 2 (DIO2)] play a crucial role in that. The findings of different authors were summarized with respect to highlighting the role of tissue-specific deiodinase activities. Apart from the results of experimental studies, the clinical results were br

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Paluchamy, Thenmozhi. "Graves’ Disease." In Graves' Disease [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96418.

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Graves’ disease is an autoimmune disease characterized by hyperthyroidism due to circulating autoantibodies. Graves’ disease was originally known as “exophthalmic goiter” but is now named after Sir Robert Graves, an Irish doctor who first described the condition in 1835. A number of conditions can cause hyperthyroidism, but Graves’ disease is the most common, affecting around 1 in 200 people. It most often affects women under the age of 40, but it is also found in men. It affects an estimated 2–3 percent of the world’s population. Thyroid-stimulating immunoglobulin (TSIs) binds to and activate

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Maria Panaitescu, Anca. "Pregnancy and Graves’ Disease." In Graves' Disease [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96245.

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Graves’ disease (GD) is one of the most common autoimmune conditions in women of reproductive age. The disorder is characterized by the presence of pathogenic immunoglobulins that bind the TSH receptors (TRAbs) and stimulate the production of thyroid hormones leading to hyperthyroidism (the occurrence of inhibiting or neutral antibodies being rare). Affected individuals can be treated by radioiodine therapy, surgical removal of the gland or by antithyroid drugs (ATDs). Thyroid stimulating immunoglobulins may persist for years after medical treatment, radioiodine therapy or surgical removal of

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Butler, Gary, and Jeremy Kirk. "Thyroid gland disorders." In Paediatric Endocrinology and Diabetes. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198786337.003.0009.

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• The thyroid gland produces all of the T<sub>4</sub> and 20% of T<sub>3</sub>. • Congenital hypothyroidism is caused by: ◦ anatomical defects: agenesis/dysgenesis, ectopic, sublingual ◦ inborn errors of thyroid hormone metabolism ◦ secondary (pituitary thyroid-stimulating hormone (TSH)) or tertiary (hypothalamic thyrotropin-releasing hormone) deficiency ◦ iodine deficiency (commonest cause worldwide of hypothyroidism, patients are usually euthyroid). • Genetic causes are rare. • In most countries worldwide, newborn TSH screening is performed at 0–5 days of age. Treatment with l-thyroxine is (

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Conference papers on the topic "Thyroid stimulating immunoglobulin"

1

Carvalho, Ana Cláudia Pires, Stella Angelis Trivellato, Guilherme Jardini Drumond Anastacio, et al. "Lambert-Eaton syndrome without an identified neoplasm." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.504.

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Introduction: Lambert-Eaton syndrome occurs due to the attack of autoantibodies to voltage-gated calcium channels in the presynaptic terminal of the neuromuscular junction and is usually paraneoplastic. Objectives: Describe the case of a patient with weakness which was investigated for neoplasm. Design and setting: Case report Methods: Analysis of medical record, photographic record of the diagnostic methods and literature review. Case description: Woman, 60 years old, diabetic, hypertensive and ex-smoker, with proximal weakness in the lower limbs for 4 months with paresthesia in the extremiti

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