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Relevant bibliographies by topics / Thyroxine Sodium

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Journal articles
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Academic literature on the topic 'Thyroxine Sodium'

Author: Grafiati

Published: 5 June 2025

Last updated: 2 August 2025

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Journal articles on the topic "Thyroxine Sodium"

1

&NA;. "Reduced thyroxine absorption with sodium polystyrene sulfonae." Inpharma Weekly &NA;, no. 889 (1993): 25. http://dx.doi.org/10.2165/00128413-199308890-00063.

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Renuka, Keshri, Praveen Arshi, Kaushav Rajni, and Jha Abhilasha. "Outcome of Treating Primary Hypothyroidism in Correction of Mildly Elevated Serum Prolactin Level in Menstrual Disorders." International Journal of Pharmaceutical and Clinical Research 15, no. 5 (2023): 1397–99. https://doi.org/10.5281/zenodo.12599057.

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<strong>Introduction: </strong>Association of hyperprolactinemia with primary hypothyroidism is well known in the literature however, marked elevation of serum prolactin in subclinical hypothyroidism (SCH) has not been commonly reported. Elevated levels of serum thyroid stimulating hormone (TSH) and serum prolactin are seen in different menstrual irregularities. <strong>Method: </strong>In our study, we have assessed serum prolactin and serum TSH levels in females suffering from menstrual disorders and implication of treatment of primary hypothyroidism on serum prolactin level.&

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3

Shakir, K. M. Mohamed, David Turton, Brian S. Aprill, Almond J. Drake, and Radm John F. Eisold. "Anemia: A Cause of Intolerance to Thyroxine Sodium." Mayo Clinic Proceedings 75, no. 2 (2000): 189–92. http://dx.doi.org/10.4065/75.2.189.

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4

El-khateeb, S. Z. "Spectrophotometric Determination of Thyroxine Sodium with p-Benzoquinone." Analytical Letters 22, no. 10 (1989): 2223–32. http://dx.doi.org/10.1080/00032718908051251.

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Shakir, K. M. Mohamed, David Turton, Brian S. Aprill, Almond J. Drake, and Radm John F. Eisold. "Anemia: A Cause of Intolerance to Thyroxine Sodium." Mayo Clinic Proceedings 75, no. 2 (2000): 189–92. http://dx.doi.org/10.1016/s0025-6196(11)64193-1.

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6

Clauss, W., B. Hoffmann, R. Krattenmacher, and W. Van Driessche. "Current-noise analysis of Na absorption in the embryonic coprodeum: stimulation by aldosterone and thyroxine." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 265, no. 5 (1993): R1100—R1108. http://dx.doi.org/10.1152/ajpregu.1993.265.5.r1100.

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The mechanism and regulation of sodium transport in the embryonic coprodeum of chicken were investigated with isolated epithelia in vitro by electrophysiological techniques. Electrogenic sodium transport (INa) was measured in Ussing chambers by the short-circuit current (Isc) technique and identified by the diuretic amiloride or by removal of sodium from the apical medium. Apical sodium channels and the kinetics of amiloride binding were investigated by current-noise analysis. Isc and INa were measured under control conditions and under the influence of in vitro incubation with aldosterone and

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7

Munera, Y., F. C. Hugues, C. Le Jeunne, and J. F. Pays. "Drug points: Interaction of thyroxine sodium with antimalarial drugs." BMJ 314, no. 7094 (1997): 1593. http://dx.doi.org/10.1136/bmj.314.7094.1593.

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8

Sergalieva, Mariyam U., Alexandra A. Tsybizova, Lyudmila A. Andreeva, Nikolay F. Myasoedov, Olga A. Bashkina, and Marina A. Samotrueva. "Effect of glyprolins on white blood cell parameters and phagocytic activity of neutrophils in conditions of experimental hyperthyroidism." Bulletin of the Russian Military Medical Academy 24, no. 1 (2022): 55–60. http://dx.doi.org/10.17816/brmma88670.

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This study investigated the effects of glyproline neuropeptide compounds (selank and Pro-Gly-Pro) on the white blood cell count and phagocytic activity of neutrophils in 40 nonlinear white male rats aged 68 months with experimental hyperthyroidism. Experimental hyperthyroidism was simulated by intragastric administration of L-thyroxine sodium salt pentahydrate at a dose of 150 g/kg/day for 21 days. All animals were equally divided into four groups: healthy rats (control), rats treated with L-thyroxine sodium pentahydrate (hyperthyroidism), rats treated with selank, and rats treated with Pro-Gl

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9

Michael, U. F., J. L. Logan, and L. A. Meeks. "The beneficial effects of thyroxine on nephrotoxic acute renal failure in the rat." Journal of the American Society of Nephrology 1, no. 11 (1991): 1236–40. http://dx.doi.org/10.1681/asn.v1111236.

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We were able to confirm previous studies demonstrating that administration of thyroxine is capable of ameliorating the severity of acute nephrotoxic renal failure in the rat. Nephrotoxic acute renal failure was induced by the subcutaneous injection of potassium dichromate (6.25 mg/kg) into Sprague-Dawley rats. Twenty-four hours after this injection, rats received an intraperitoneal injection of either thyroxine (80 micrograms/kg body wt) or normal saline. Forty-eight hours after the potassium dichromate injection, renal clearance studies were performed. Inulin clearance was significantly highe

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10

Ljunggren, J. G., and B. Persson. "SUBCUTANEOUS ADMINISTRATION OF SODIUM L-THYROXINE TO PATIENTS WITH HYPOTHYROIDISM." Acta Medica Scandinavica 197, no. 1-6 (2009): 471–72. http://dx.doi.org/10.1111/j.0954-6820.1975.tb04954.x.

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Dissertations / Theses on the topic "Thyroxine Sodium"

1

Hibelot, Catherine. "Etude comparative de deux traitements substitutifs de l'hypothyroidie LT3 et LT4 : répercussions osseuses au bout de un an." Bordeaux 2, 1992. http://www.theses.fr/1992BOR2M032.

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Book chapters on the topic "Thyroxine Sodium"

1

Toft, Anthony. "Treatment of hypothyroidism." In Oxford Textbook of Endocrinology and Diabetes. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235292.003.3293.

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Treatment of primary hypothyroidism is usually both gratifying and simple and, in most cases, lifelong. Thyroxine, as l-thyroxine sodium, is the therapy of choice and is available in the UK as tablets of 25, 50, and 100 μ‎g. A greater variety of tablet strength is marketed in other parts of Europe and North America. Thyroxine has a half-life of some 7 days and should be given as a single daily dose which improves compliance. Thyroxine, taken at bedtime, is associated with higher thyroid hormone concentrations and lower thyroid-stimulating hormone (TSH) concentrations compared to the same dose

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2

Nygaard, Birte. "Treatment of Hypothyroidism." In Oxford Textbook of Endocrinology and Diabetes 3e, edited by John A. H. Wass, Wiebke Arlt, and Robert K. Semple. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198870197.003.0072.

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The goals for treatment of primary hypothyroidism are to restore euthyroidism with normalization of serum thyroid-stimulating hormone (TSH) and fT<sub>4</sub> concentrations and to eliminate symptoms. Thyroxine, as levothyroxine sodium (L-T<sub>4</sub>), is the therapy of choice, and its use in most patients’ treatment is straightforward. In a minor patient group, persistent symptoms are present although thyroid function parameters are normalized. In these patients, it is pertinent to search for alternative explanations for the symptoms other than thyroid disease. A possible effect of a combin

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