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Relevant bibliographies by topics / Tissue eosinophil

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Academic literature on the topic 'Tissue eosinophil'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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Journal articles on the topic "Tissue eosinophil"

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Cantarini, L., N. Volpi, P. Carbotti, et al. "Eosinophilia-associated muscle disorders: an immunohistological study with tissue localisation of major basic protein in distinct clinicopathological forms." Journal of Clinical Pathology 62, no. 5 (2009): 442–47. http://dx.doi.org/10.1136/jcp.2008.060616.

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Aims:(a) To evaluate tissue eosinophil density, location of eosinophil cytotoxic products, histopathological muscle changes and inflammatory cell types in different eosinophilia-associated myopathies that are clinicopathologically heterogeneous. (b) To determine the immunohistological range of tissue eosinophil density in non-eosinophilic inflammatory myopathies.Methods:Muscle biopsy specimens from seven patients with blood and/or tissue eosinophilia and clinicolaboratory myopathic signs (five chronic course myopathies, one subacute onset fasciitis/myositis, one acute myositis), and from 18 no

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Desreumaux, P., A. Janin, S. Dubucquoi, et al. "Synthesis of interleukin-5 by activated eosinophils in patients with eosinophilic heart diseases." Blood 82, no. 5 (1993): 1553–60. http://dx.doi.org/10.1182/blood.v82.5.1553.1553.

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Abstract Eosinophilic endomyocardial disease represents a major evolutive risk in chronic eosinophilia-associated disorders. Eosinophil granule proteins appear to be involved in cardiac injury, but the mechanisms leading to eosinophil infiltration and degranulation are not clear. Interleukin-5 (IL-5) has been recently shown to be produced by eosinophils and might play a role in both chemoattraction and degranulation of eosinophils. In four cases of eosinophilic diseases with severe cardiac failure, we evaluated the proportion of eosinophil phenotypes and the serum levels of eosinophil cationic

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Desreumaux, P., A. Janin, S. Dubucquoi, et al. "Synthesis of interleukin-5 by activated eosinophils in patients with eosinophilic heart diseases." Blood 82, no. 5 (1993): 1553–60. http://dx.doi.org/10.1182/blood.v82.5.1553.bloodjournal8251553.

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Eosinophilic endomyocardial disease represents a major evolutive risk in chronic eosinophilia-associated disorders. Eosinophil granule proteins appear to be involved in cardiac injury, but the mechanisms leading to eosinophil infiltration and degranulation are not clear. Interleukin-5 (IL-5) has been recently shown to be produced by eosinophils and might play a role in both chemoattraction and degranulation of eosinophils. In four cases of eosinophilic diseases with severe cardiac failure, we evaluated the proportion of eosinophil phenotypes and the serum levels of eosinophil cationic protein

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Simon, H. U., S. Yousefi, C. Schranz, A. Schapowal, C. Bachert, and K. Blaser. "Direct demonstration of delayed eosinophil apoptosis as a mechanism causing tissue eosinophilia." Journal of Immunology 158, no. 8 (1997): 3902–8. http://dx.doi.org/10.4049/jimmunol.158.8.3902.

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Abstract Nasal polyps, which often occur in association with allergic rhinitis and asthma, are characterized by a marked infiltration of eosinophils. Using a method for detecting eosinophils with DNA strand breaks, we found direct evidence for inhibition of eosinophil apoptosis in this model of tissue eosinophilia. By using Southern blot analysis linked to reverse transcription-PCR, we detected a mRNA signal specific for IL-5 in all nasal polyps. The identification of IL-5 as a major eosinophil survival factor was confirmed by ELISA measurements using tissue homogenates. Moreover, immunohistoc

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Van Hulst, Glenn, Fabrice Bureau, and Christophe J. Desmet. "Eosinophils as Drivers of Severe Eosinophilic Asthma: Endotypes or Plasticity?" International Journal of Molecular Sciences 22, no. 18 (2021): 10150. http://dx.doi.org/10.3390/ijms221810150.

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Asthma is now recognized as a heterogeneous disease, encompassing different phenotypes driven by distinct pathophysiological mechanisms called endotypes. Common phenotypes of asthma, referred to as eosinophilic asthma, are characterized by the presence of eosinophilia. Eosinophils are usually considered invariant, terminally differentiated effector cells and have become a primary therapeutic target in severe eosinophilic asthma (SEA) and other eosinophil-associated diseases (EADs). Biological treatments that target eosinophils reveal an unexpectedly complex role of eosinophils in asthma, inclu

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Radke, Amy L., Lauren E. Reynolds, Rossana C. N. Melo, Ann M. Dvorak, Peter F. Weller, and Lisa A. Spencer. "Mature human eosinophils express functional Notch ligands mediating eosinophil autocrine regulation." Blood 113, no. 13 (2009): 3092–101. http://dx.doi.org/10.1182/blood-2008-05-155937.

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Abstract Eosinophil chemotaxis and survival within tissues are key components in the development of tissue eosinophilia and subsequent effector responses. In this study, we demonstrate a novel mechanism of eosinophil autoregulation affecting migration and survival mediated through Notch signaling. We show for the first time that human blood eosinophils express Notch receptors and Notch ligands, expressions of which are influenced by the presence of eosinophil-activating granulocyte-macrophage colony-stimulating factor (GM-CSF). Evidence of Notch receptor activation and subsequent transcription

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Masterson, Joanne C., Calies Menard-Katcher, Leigha D. Larsen, Glenn T. Furuta, and Lisa A. Spencer. "Heterogeneity of Intestinal Tissue Eosinophils: Potential Considerations for Next-Generation Eosinophil-Targeting Strategies." Cells 10, no. 2 (2021): 426. http://dx.doi.org/10.3390/cells10020426.

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Eosinophils are implicated in the pathophysiology of a spectrum of eosinophil-associated diseases, including gastrointestinal eosinophilic diseases (EGIDs). Biologics that target the IL-5 pathway and are intended to ablate eosinophils have proved beneficial in severe eosinophilic asthma and may offer promise in treating some endotypes of EGIDs. However, destructive effector functions of eosinophils are only one side of the coin; eosinophils also play important roles in immune and tissue homeostasis. A growing body of data suggest tissue eosinophils represent a plastic and heterogeneous populat

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Saraiva, Ana Laura, and Fátima Carneiro. "New Insights Into the Role of Tissue Eosinophils in the Progression of Colorectal Cancer: A Literature Review." Acta Médica Portuguesa 31, no. 6 (2018): 329. http://dx.doi.org/10.20344/amp.10112.

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Introduction: Amongst the inflammatory cells implicated in the immune surveillance of colorectal cancer, a growing body of evidence suggests a role for eosinophils in carcinogenesis. We aimed to review the value of tumour-associated tissue eosinophilia (TATE) in the prognosis of colorectal cancer emphasizing the identification and measurement of tissue-infiltrating eosinophils and their association with the clinicopathological features of the disease.Material and Methods: We used PubMed and Web of Science search engines to retrieve studies that looked at the association between tissue eosinoph

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Rothenberg, Marc E., James A. MacLean, Eric Pearlman, Andrew D. Luster, and Philip Leder. "Targeted Disruption of the Chemokine Eotaxin Partially Reduces Antigen-induced Tissue Eosinophilia." Journal of Experimental Medicine 185, no. 4 (1997): 785–90. http://dx.doi.org/10.1084/jem.185.4.785.

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The chemokines are a large group of chemotactic cytokines that regulate leukocyte trafficking and have recently been shown to inhibit human immunodeficiency virus entry into cells. Eotaxin is a C–C chemokine implicated in the recruitment of eosinophils in a variety of inflammatory disorders and, unlike all other eosinophil chemoattractants, is eosinophil specific. However, given the large number of chemoattractants that have activities on eosinophils, it is unclear whether eotaxin has an important role in vivo. Furthermore, it remains unclear why there is constitutive expression of eotaxin in

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Manohar, Murli, Alok K. Verma, Sathisha Upparahalli Venkateshaiah, and Anil Mishra. "Role of eosinophils in the initiation and progression of pancreatitis pathogenesis." American Journal of Physiology-Gastrointestinal and Liver Physiology 314, no. 2 (2018): G211—G222. http://dx.doi.org/10.1152/ajpgi.00210.2017.

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Eosinophilic pancreatitis (EP) is reported in humans; however, the etiology and role of eosinophils in EP pathogenesis are poorly understood and not well explored. Therefore, it is interesting to examine the role of eosinophils in the initiation and progression of pancreatitis pathogenesis. Accordingly, we performed anti-major basic protein immunostaining, chloroacetate esterase, and Masson’s trichrome analyses to detect eosinophils, mast cells, and collagen in the tissue sections of mouse and human pancreas. Induced eosinophils accumulation and degranulation were observed in the tissue sectio

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Dissertations / Theses on the topic "Tissue eosinophil"

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Zagai, Ulrika. "Platelets and eosinophils in lung tissue remodelling /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-841-X/.

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Zillmann, Christine. "Eosinophile Granulozyten als Träger von intravaskulärem tissue factor (TF)." Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-151254.

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Zillmann, Christine [Verfasser], and Bernd [Akademischer Betreuer] Engelmann. "Eosinophile Granulozyten als Träger von intravaskulärem tissue factor (TF) : Bedeutung bei Gerinnungsprozessen und Signaltransduktion / Christine Zillmann. Betreuer: Bernd Engelmann." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2012. http://d-nb.info/1029662061/34.

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Titz, Tiago de Oliveira. "Avaliação fenotípica e funcional dos eosinófilos da dermatite atópica do adulto." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/5/5133/tde-20052015-121525/.

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Introdução: A dermatite atópica (DA) é uma doença cutânea inflamatória de caráter crônico, recidivante, em que o prurido intenso e a xerose cutânea são frequentes. A etiopatogenia da DA é multifatorial, envolvendo fatores genéticos, ambientais e imunológicos. Eosinófilos são leucócitos polimorfonucleares multifuncionais que estão implicados na patogênese de diversos processos inflamatórios, incluindo a DA. Além da produção e secreção de diversas proteínas presentes nos grânulos citoplasmáticos, os eosinófilos também apresentam potencial para secretar metaloproteinases, enzimas proteolíticas qu

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Bertges, Klaus Ruback. "Avaliação do remodelamento tecidual em biópsias de pacientes portadores de esofagite eosinofílica." Universidade Federal de Juiz de Fora (UFJF), 2018. https://repositorio.ufjf.br/jspui/handle/ufjf/6721.

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Silva, Patricia Angeli da. "Efeitos da inibição crônica das óxido nítrico sintases na mecânica de tecido periférico, no recrutamento eosinofílico e no remodelamento da matriz extracelular induzida por inflamação crônica pulmonar." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/5/5160/tde-25032009-140404/.

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INTRODUÇÃO: A importância da resposta mecânica do parênquima pulmonar na fisiopatologia da asma tem sido recentemente reconhecida. O óxido nítrico é um mediador que controla o tônus muscular liso das vias aéreas, porém este efeito no parênquima pulmonar periférico ainda não foi previamente investigado. Nossa hipótese é que a inibição crônica das óxido nítrico sintases por meio do tratamento com L-NAME (falso substrato para todas as óxido nítrico sintases) pode modular a mecânica do parênquima pulmonar, o recrutamento eosinofílico e o remodelamento da matriz extracelular em modelo de inflamação

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Saraiva, Ana Laura da Silva Santos. "New Insights into the Role of Tissue Eosinophils in the Progression of Colorectal Cancer - A literature review." Master's thesis, 2018. https://hdl.handle.net/10216/112138.

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Saraiva, Ana Laura da Silva Santos. "New Insights into the Role of Tissue Eosinophils in the Progression of Colorectal Cancer - A literature review." Dissertação, 2018. https://repositorio-aberto.up.pt/handle/10216/112138.

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Books on the topic "Tissue eosinophil"

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Barsoum, Rashad S. Schistosomiasis. Edited by Neil Sheerin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0181_update_001.

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AbstractSchistosomes are blood flukes that parasitize humans, apes, cattle, and other animals. In these definitive hosts they are bisexual, and lay eggs which are shed to fresh water where they complete an asexual cycle in different snails, ending in the release of cercariae which infect the definitive hosts to complete the life cycle.Seven of over 100 species of schistosomes are human pathogens, causing disease in different organs depending on the parasite species. Racial and genetic factors are involved in susceptibility, severity, and sequelae of infection.Morbidity is induced by the host’s

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Crawshaw, Anjali. Sarcoidosis and other granulomatous lung disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0138.

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Sarcoidosis is a multisystem disorder characterized by the formation of non-caseating granulomas in many tissues. A granuloma is an organized aggregate of immune cells; it forms in response to an antigenic stimulus. It contains abnormal macrophages (epithelioid histiocytes, which often fuse to form multinucleated giant cells), lymphocytes, neutrophils, eosinophils, fibroblasts, and collagen.

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Herrington, William G., Aron Chakera, and Christopher A. O’Callaghan. Interstitial renal disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0160.

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Tubulointerstitial renal diseases affect the renal tubules and/or the supporting interstitial tissue around them. The glomeruli are typically spared in early disease. Acute interstitial nephritis is characterized by an inflammatory infiltrate (often containing eosinophils). Chronic tubulointerstitial nephritis (TIN) is characterized by extensive tubular atrophy and interstitial fibrosis. The processes are clinically distinct but a prolonged acute interstitial nephritis will develop into chronic disease. This chapter looks at the etiology of interstitial renal disease, as well as its symptoms a

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Jayne, David. Treatment of ANCA-associated vasculitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0132.

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The goals of treatment in anti-neutrophil cytoplasm antibody (ANCA) vasculitis are to stop vasculitic activity, to prevent vasculitis returning, and to address longer-term comorbidities caused by tissue damage, drug toxicity, and increased cardiovascular and malignancy risk. Cyclophosphamide and high-dose glucocorticoids remain the standard induction therapy with alternative immunosuppressives, such as methotrexate or azathioprine, to prevent relapse. Refractory disease resulting from a failure of induction or remission maintenance therapy requires alternative agents and rituximab has been par

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Bowman, Dwight D. Zoonotic hookworm infections. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0069.

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Hookworms on occasion cause creeping lesions in the superficial layers of the human skin that have been designated as cutaneous larva migrans for the purpose of contrasting the condition with visceral larva migrans. Currently, the disease is presenting most commonly to physicians specializing in tropical or travel medicine in patients who have just visited a tropical beach and are presenting with serpiginous tracks in their skin. The serpiginous tracts can persist for week, and are often pruritic, may be associated with accompanying bulla, and can rarely lead to secondary sequelae. The larval

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Book chapters on the topic "Tissue eosinophil"

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Gachanja, Naomi N., David A. Dorward, Adriano G. Rossi, and Christopher D. Lucas. "Assays of Eosinophil Apoptosis and Phagocytic Uptake." In Methods in Molecular Biology. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1095-4_10.

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AbstractEosinophil apoptosis (programmed cell death) plays an important role in several inflammatory and allergic conditions. Apoptosis triggers various mechanisms including activation of cysteine-aspartic proteases (caspases) and is characterized by morphological and biochemical changes. These include cellular condensation, nuclear fragmentation, increased mitochondrial permeability with loss of membrane potential, and exposure of phosphatidylserine on the cell membrane. A greater understanding of apoptotic mechanisms, subsequent phagocytosis (efferocytosis), and regulation of these processes

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Jahnsen, Frode, Trond S. Halstensen, and Per Brandtzaeg. "Immunostaining with Monoclonal Antibodies to Eosinophil Cationic Protein (EG1 and EG2) does not Distinguish between Resting and Activated Eosinophils in Formalin-Fixed Tissue Specimens." In Advances in Experimental Medicine and Biology. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-1941-6_59.

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De Schepper, A. M. A., and H. R. M. Degryse. "Eosinophilic Granuloma." In Magnetic Resonance Imaging of Bone and Soft Tissue Tumors and Their Mimics. Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0997-7_11.

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Olbrich, Courtney L., Leigha D. Larsen, and Lisa A. Spencer. "Assessing Phenotypic Heterogeneity in Intestinal Tissue Eosinophils." In Methods in Molecular Biology. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1095-4_19.

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Zhu, Xiang, and Nives Zimmermann. "Detection of Eosinophils in Tissue Sections by Immunohistochemistry." In Methods in Molecular Biology. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-2364-0_14.

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Krstić, Radivoj V. "Loose Connective Tissue. Wandering Cells: Eosinophilic Granulocyte." In General Histology of the Mammal. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70420-8_73.

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Sugiura, H., M. Omoto, and M. Uehara. "Modulatory Factors for Tissue Eosinophilia in Atopic Eczema." In New Trends in Allergy IV. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60419-5_36.

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Mai, Eric, Ajinkya R. Limkar, Caroline M. Percopo, and Helene F. Rosenberg. "Generation of Mouse Eosinophils in Tissue Culture from Unselected Bone Marrow Progenitors." In Methods in Molecular Biology. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1095-4_4.

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Wagner, N., and G. Dannecker. "Systemischer Lupus erythematodes und seltene rheumatische Erkrankungen (Mixed connective tissue disease, Sjögren-Syndrom, eosinophile Fasziitis)." In Pädiatrie. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-76460-1_88.

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Percopo, Caroline M., Ajinkya R. Limkar, Albert C. Sek, and Helene F. Rosenberg. "Detection of Mouse Eosinophils in Tissue by Flow Cytometry and Isolation by Fluorescence-Activated Cell Sorting (FACS)." In Methods in Molecular Biology. Springer US, 2021. http://dx.doi.org/10.1007/978-1-0716-1095-4_5.

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Conference papers on the topic "Tissue eosinophil"

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Parker, KA, C. Protheroe, JJ Lee, KO Leslie, and LJ Wesselius. "Survival in Acute Lung Injury Is Predicted by Eosinophil Degranulation in Lung Tissue." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3889.

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Sivkova, T. N., and S. V. Volkov. "PATHOANATOMICAL CHANGES IN THE NASAL WAYS OF VULPES VULPES L., 1758 INFECTED BY CAPILLARIA (EUCOLEUS) BOEHMI SUPPERER, 1953." In THEORY AND PRACTICE OF PARASITIC DISEASE CONTROL. VNIIP – FSC VIEV, 2024. http://dx.doi.org/10.31016/978-5-6050437-8-2.2024.25.363-367.

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The infection by the nasal nematode Capillaria (Eucoleus) boehmi Supperer, 1953 has been recorded in foxes in North America, Europe, and in some regions of Russia including the Perm Kray. However, to date, a description of pathoanatomical changes in wild foxes has not been presented in scientific literature, which determines the relevance of this study. We carried out dissection of the carcasses of foxes killed by hunters or in road accidents. The nasal cavities were opened. Tissue samples were placed in a 10% neutral formaldehyde solution. The histological preparations were prepared in the UN

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Southworth, Thomas, Umme Kolsum, Andrew Higham, et al. "Analysis of Immunoglobulin A and M gene expression and plasma cells in bronchial tissue from COPD patients with high blood eosinophil counts." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.oa4924.

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Turato, Graziella, Umberto Semenzato, Nerea Torrecilla, et al. "Blood eosinophils do not reflect lung tissue eosinophils in COPD." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa1014.

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Bonato, Matteo, Erica Bazzan, Deborah Snijders, et al. "Biomarkers to identify tissue eosinophils in wheezing children." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4097.

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Chibana, K., Y. Horigane, N. Uchida, et al. "Investigation of Clinical Factors Corelated with Airway Tissue Eosinophils." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a1288.

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Jogdand, Prajakta, Michiko Mori, Anders Bergqvist, Andrew Walls, Caroline Sanden, and Jonas Erjefält. "Mapping of eosinophil and basophils in COPD lung tissues." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa384.

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de Toledo, Renata Silva, Luís Henrique Sales Oliveira, and Pâmela Camila Pereira. "Respiratory muscle training in asthmatic patient: Case study." In IV Seven International Congress of Health. Seven Congress, 2024. http://dx.doi.org/10.56238/homeivsevenhealth-086.

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Asthma is a chronic inflammatory condition of the airways characterized by hyperresponsiveness and variable airflow obstruction. It often begins in childhood and can be triggered by several external factors such as allergens and pollutants. Inflammation involves mediators such as eosinophils, mast cells and cytokines, resulting in airway injury and tissue remodeling. During asthma attacks, bronchial obstruction occurs with lung hyperinflation and changes in ventilatory mechanics. Treatment includes approaches such as Respiratory Muscle Training (RMT), which improves lung function and quality o

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Pavlenko, S. G., A. A. Evglevsky, A. A. Evglevsky, and D. S. Berdysh. "HEMOSTATIC AND REPARATIVE PROPERTIES OF APPLE AND CITRUS PECTIN POWDERS." In NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. LLC Institute Information Technologies, 2023. http://dx.doi.org/10.47501/978-5-6044060-3-8.64-71.

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The article presents data on the hemostatic and repair properties of apple and citrus pectin powders in an experiment on laboratory animals. Stopping bleeding from wounds of the kidney and liver with the help of pectin powders occurs within 10 seconds with the formation of a strong fixed clot. Mor-phological data indicate an acceleration of tissue repair of the parenchymal organ in the absence of an eosinophilic reaction.

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Cabrera-López, Carlos, Alejandra Sánchez-Santos, Angelina Lemes-Castellano, et al. "Different subtypes of eosinophils between asthma and COPD in bronchial tissue." In ERS International Congress 2023 abstracts. European Respiratory Society, 2023. http://dx.doi.org/10.1183/13993003.congress-2023.pa2976.

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Reports on the topic "Tissue eosinophil"

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Viksna, Ludmila, Oksana Kolesova, Aleksandrs Kolesovs, Ieva Vanaga, and Seda Arutjunana. Clinical characteristics of COVID-19 patients (Latvia, Spring 2020). Rīga Stradiņš University, 2020. http://dx.doi.org/10.25143/fk2/hnmlhh.

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Data include following variables: Demographics, epidemiological history, comorbidities, diagnosis, complications, and symptoms on admission to the hospital. Also, body’s temperature and SpO2. Blood cells: white cells count (WBC), neutrophils (Neu), lymphocytes (Ly), eosinophils (Eo) and monocytes (Mo), percentages of segmented and banded neutrophils, erythrocytes (RBC), platelet count (PLT), hemoglobin (Hb), and hematocrit (HCT); Inflammatory indicators: erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP); Tissue damage indicators: alanine aminotransferase (ALT), lactate dehydrog

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