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Journal articles on the topic 'Tissue folding'

Author: Grafiati
Published: 25 May 2024

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1

Zečić, Aleksandra, and Chadanat Noonin. "Whole-mount in situ hybridization: minimizing the folding problem of thin-sheet tissue-like crayfish haematopoietic tissue." Crustaceana 91, no. 1 (2018): 1–15. http://dx.doi.org/10.1163/15685403-00003745.

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Crayfish haematopoietic tissue (HPT) has a thin-sheet-like structure with a thickness of 100-160 μm and a width of approximately 1-2 cm. This structure makes HPT extremely easy to fold after removal from the animal. Therefore, it is difficult to handle the tissue without folding when processing for sectioning and histological study. The degree of tissue folding reflects the size of the tissue sections obtained, how complicated it is to interpret the location of each tissue section, and the accuracy of the interpretation of the location of a specific transcript. To facilitate the interpretation
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2

Inoue, Yasuhiro, Itsuki Tateo, and Taiji Adachi. "Epithelial tissue folding pattern in confined geometry." Biomechanics and Modeling in Mechanobiology 19, no. 3 (2019): 815–22. http://dx.doi.org/10.1007/s10237-019-01249-8.

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AbstractThe primordium of the exoskeleton of an insect is epithelial tissue with characteristic patterns of folds. As the insect develops from larva to pupa, the spreading of these folds produces the three-dimensional shape of the exoskeleton of the insect. It is known that the three-dimensional exoskeleton shape has already been encoded in characteristic patterns of folds in the primordium; however, a description of how the epithelial tissue forms with the characteristic patterns of folds remains elusive. The present paper suggests a possible mechanism for the formation of the folding pattern
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3

Hookway, Tracy A. "Engineering Biology by Controlling Tissue Folding." Trends in Biotechnology 36, no. 4 (2018): 341–43. http://dx.doi.org/10.1016/j.tibtech.2018.02.003.

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4

Allen, Simon, Hassan Y. Naim, and Neil J. Bulleid. "Intracellular Folding of Tissue-type Plasminogen Activator." Journal of Biological Chemistry 270, no. 9 (1995): 4797–804. http://dx.doi.org/10.1074/jbc.270.9.4797.

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5

Zartman, Jeremiah J., and Stanislav Y. Shvartsman. "Unit Operations of Tissue Development: Epithelial Folding." Annual Review of Chemical and Biomolecular Engineering 1, no. 1 (2010): 231–46. http://dx.doi.org/10.1146/annurev-chembioeng-073009-100919.

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6

Hiraiwa, Tetsuya, Fu-Lai Wen, Tatsuo Shibata, and Erina Kuranaga. "Mathematical Modeling of Tissue Folding and Asymmetric Tissue Flow during Epithelial Morphogenesis." Symmetry 11, no. 1 (2019): 113. http://dx.doi.org/10.3390/sym11010113.

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Recent studies have revealed that intrinsic, individual cell behavior can provide the driving force for deforming a two-dimensional cell sheet to a three-dimensional tissue without the need for external regulatory elements. However, whether intrinsic, individual cell behavior could actually generate the force to induce tissue deformation was unclear, because there was no experimental method with which to verify it in vivo. In such cases, mathematical modeling can be effective for verifying whether a locally generated force can propagate through an entire tissue and induce deformation. Moreover
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7

Chan, Hon Fai, Ruike Zhao, German A. Parada, et al. "Folding artificial mucosa with cell-laden hydrogels guided by mechanics models." Proceedings of the National Academy of Sciences 115, no. 29 (2018): 7503–8. http://dx.doi.org/10.1073/pnas.1802361115.

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The surfaces of many hollow or tubular tissues/organs in our respiratory, gastrointestinal, and urogenital tracts are covered by mucosa with folded patterns. The patterns are induced by mechanical instability of the mucosa under compression due to constrained growth. Recapitulating this folding process in vitro will facilitate the understanding and engineering of mucosa in various tissues/organs. However, scant attention has been paid to address the challenge of reproducing mucosal folding. Here we mimic the mucosal folding process using a cell-laden hydrogel film attached to a prestretched to
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8

Ko, Clint S., Vardges Tserunyan, and Adam C. Martin. "Microtubules promote intercellular contractile force transmission during tissue folding." Journal of Cell Biology 218, no. 8 (2019): 2726–42. http://dx.doi.org/10.1083/jcb.201902011.

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During development, forces transmitted between cells are critical for sculpting epithelial tissues. Actomyosin contractility in the middle of the cell apex (medioapical) can change cell shape (e.g., apical constriction) but can also result in force transmission between cells via attachments to adherens junctions. How actomyosin networks maintain attachments to adherens junctions under tension is poorly understood. Here, we discovered that microtubules promote actomyosin intercellular attachments in epithelia during Drosophila melanogaster mesoderm invagination. First, we used live imaging to s
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9

Codd, S. L., R. K. Lambert, M. R. Alley, and R. J. Pack. "Tensile stiffness of ovine tracheal wall." Journal of Applied Physiology 76, no. 6 (1994): 2627–35. http://dx.doi.org/10.1152/jappl.1994.76.6.2627.

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The epithelial folding that occurs during bronchoconstriction requires that the pressure on the muscle side of the folding membrane be greater than that on the lumen side. The pressure required for a given level of folding depends on the elastic properties of the tissue and on the geometry of the folding. To quantify the elastic properties, uniaxial tensile stiffness of the tracheal inner wall of nine sheep was measured in two directions: parallel to the tracheal axis and circumferentially. The tissue showed anisotropic behavior, being approximately three times stiffer longitudinally than circ
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10

Tozluoǧlu, Melda, and Yanlan Mao. "On folding morphogenesis, a mechanical problem." Philosophical Transactions of the Royal Society B: Biological Sciences 375, no. 1809 (2020): 20190564. http://dx.doi.org/10.1098/rstb.2019.0564.

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Tissue folding is a fundamental process that sculpts a simple flat epithelium into a complex three-dimensional organ structure. Whether it is the folding of the brain, or the looping of the gut, it has become clear that to generate an invagination or a fold of any form, mechanical asymmetries must exist in the epithelium. These mechanical asymmetries can be generated locally, involving just the invaginating cells and their immediate neighbours, or on a more global tissue-wide scale. Here, we review the different mechanical mechanisms that epithelia have adopted to generate folds, and how the u
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11

Mason, Frank M., Shicong Xie, Claudia G. Vasquez, Michael Tworoger, and Adam C. Martin. "RhoA GTPase inhibition organizes contraction during epithelial morphogenesis." Journal of Cell Biology 214, no. 5 (2016): 603–17. http://dx.doi.org/10.1083/jcb.201603077.

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During morphogenesis, contraction of the actomyosin cytoskeleton within individual cells drives cell shape changes that fold tissues. Coordination of cytoskeletal contractility is mediated by regulating RhoA GTPase activity. Guanine nucleotide exchange factors (GEFs) activate and GTPase-activating proteins (GAPs) inhibit RhoA activity. Most studies of tissue folding, including apical constriction, have focused on how RhoA is activated by GEFs to promote cell contractility, with little investigation as to how GAPs may be important. Here, we identify a critical role for a RhoA GAP, Cumberland GA
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12

Johnson, Travis K., Karyn A. Moore, James C. Whisstock, and Coral G. Warr. "Maternal Torso-Like Coordinates Tissue Folding During Drosophila Gastrulation." Genetics 206, no. 3 (2017): 1459–68. http://dx.doi.org/10.1534/genetics.117.200576.

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13

Heer, Natalie C., Pearson W. Miller, Soline Chanet, Norbert Stoop, Jörn Dunkel, and Adam C. Martin. "Actomyosin-based tissue folding requires a multicellular myosin gradient." Journal of Cell Science 130, no. 11 (2017): e1.2-e1.2. http://dx.doi.org/10.1242/jcs.206243.

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14

Gracia, Mélanie, Corinne Benassayag, and Magali Suzanne. "Is Epithelio-Mesenchymal transition actively involved in tissue folding?" Mechanisms of Development 145 (July 2017): S98. http://dx.doi.org/10.1016/j.mod.2017.04.253.

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15

Hughes, Alex J., Hikaru Miyazaki, Maxwell C. Coyle, et al. "Engineered Tissue Folding by Mechanical Compaction of the Mesenchyme." Developmental Cell 44, no. 2 (2018): 165–78. http://dx.doi.org/10.1016/j.devcel.2017.12.004.

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16

Heer, Natalie C., Pearson W. Miller, Soline Chanet, Norbert Stoop, Jörn Dunkel, and Adam C. Martin. "Actomyosin-based tissue folding requires a multicellular myosin gradient." Development 144, no. 10 (2017): 1876–86. http://dx.doi.org/10.1242/dev.146761.

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17

Byers, Peter H. "Folding defects in fibrillar collagens." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 356, no. 1406 (2001): 151–58. http://dx.doi.org/10.1098/rstb.2000.0760.

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Fibrillar collagens have a long triple helix in which glycine is in every third position for more than 1000 amino acids. The three chains of these molecules are assembled with specificity into several different molecules that have tissue–specific distribution. Mutations that alter folding of either the carboxy–terminal globular peptides that direct chain association, or of the regions of the triple helix that are important for nucleation, or of the bulk of the triple helix, all result in identifiable genetic disorders in which the phenotype reflects the region of expression of the genes and th
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18

Braakman, I., H. Hoover-Litty, K. R. Wagner, and A. Helenius. "Folding of influenza hemagglutinin in the endoplasmic reticulum." Journal of Cell Biology 114, no. 3 (1991): 401–11. http://dx.doi.org/10.1083/jcb.114.3.401.

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The folding of influenza hemagglutinin (HA0) in the ER was analyzed in tissue culture cells by following the formation of intrachain disulfides after short (1 min) radioactive pulses. While some disulfide bonds were already formed on the nascent chains, the subunits acquired their final disulfide composition and antigenic epitopes posttranslationally. Two posttranslational folding intermediates were identified. In CHO cells constitutively expressing HA0, mature HA0 subunits were formed with a half time of 3 min and their folding reached completion at 22 min. The rate of folding was highly depe
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19

Grigoryev, Sergei A. "Higher-order folding of heterochromatin: Protein bridges span the nucleosome arrays." Biochemistry and Cell Biology 79, no. 3 (2001): 227–41. http://dx.doi.org/10.1139/o01-030.

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In interphase eukaryotic nuclei, chromatin is divided into two morphologically distinct types known as heterochromatin and euchromatin. It has been long suggested that the two types of chromatin differ at the level of higher-order folding. Recent studies have revealed the features of chromatin 3D architecture that distinguish the higher-order folding of repressed and active chromatin and have identified chromosomal proteins and their modifications associated with these structural transitions. This review discusses the molecular and structural determinants of chromatin higher-order folding in r
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20

Yoshino, Kunihiko, Kohei Abe, Koyu Suzuki, et al. "A Novel Technique of Endoscopic Vein Harvesting With Preserved Perivascular Tissue." Innovations: Technology and Techniques in Cardiothoracic and Vascular Surgery 15, no. 5 (2020): 475–77. http://dx.doi.org/10.1177/1556984520948139.

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The no-touch saphenous vein harvesting technique is considered to be the ideal procedure to achieve the best quality of vein, whereas the endoscopic vein harvesting (EVH) technique is considered to be ideal for decreasing wound complications. We developed a new technique of EVH with perivascular tissue preservation. This procedure was performed by dissecting the immediate anterior and posterior perivascular connective tissues of the saphenous vein followed by cutting approximately 1 cm laterally from the saphenous vein with the use of a harvester (MAQUET Getinge Group, Getinge AB, Göteborg, Sw
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21

Yevick, Hannah G., Pearson W. Miller, Jörn Dunkel, and Adam C. Martin. "Structural Redundancy in Supracellular Actomyosin Networks Enables Robust Tissue Folding." Developmental Cell 50, no. 5 (2019): 586–98. http://dx.doi.org/10.1016/j.devcel.2019.06.015.

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22

Del-Valle-Anton, Lucia, and Víctor Borrell. "Folding brains: from development to disease modeling." Physiological Reviews 102, no. 2 (2022): 511–50. http://dx.doi.org/10.1152/physrev.00016.2021.

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The human brain is characterized by the large size and intricate folding of its cerebral cortex, which are fundamental for our higher cognitive function and frequently altered in pathological dysfunction. Cortex folding is not unique to humans, nor even to primates, but is common across mammals. Cortical growth and folding are the result of complex developmental processes that involve neural stem and progenitor cells and their cellular lineages, the migration and differentiation of neurons, and the genetic programs that regulate and fine-tune these processes. All these factors combined generat
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23

Kim, Jae-Young, Elizabeth A. Fogarty, Franklin J. Lu, et al. "Twin-Arginine Translocation of Active Human Tissue Plasminogen Activator in Escherichia coli." Applied and Environmental Microbiology 71, no. 12 (2005): 8451–59. http://dx.doi.org/10.1128/aem.71.12.8451-8459.2005.

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ABSTRACT When eukaryotic proteins with multiple disulfide bonds are expressed at high levels in Escherichia coli, the efficiency of thiol oxidation and isomerization is typically not sufficient to yield soluble products with native structures. Even when such proteins are secreted into the oxidizing periplasm or expressed in the cytoplasm of cells carrying mutations in the major intracellular disulfide bond reduction systems (e.g., trxB gor mutants), correct folding can be problematic unless a folding modulator is simultaneously coexpressed. In the present study we explored whether the bacteria
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24

John, Alphy, and Matteo Rauzi. "A two-tier junctional mechanism drives simultaneous tissue folding and extension." Developmental Cell 56, no. 10 (2021): 1469–83. http://dx.doi.org/10.1016/j.devcel.2021.04.003.

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25

Jung, Sae-Young, Dae-Young Kang, Hyun-Seung Shin, and Jung-Chul Park. "3D analysis of soft tissue around implant after flap folding suture." Journal of Dental Rehabilitation and Applied Science 37, no. 3 (2021): 130–37. http://dx.doi.org/10.14368/jdras.2021.37.3.130.

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26

Visetsouk, Mike R., Elizabeth J. Falat, Ryan J. Garde, Jennifer L. Wendlick, and Jennifer H. Gutzman. "Basal epithelial tissue folding is mediated by differential regulation of microtubules." Development 145, no. 22 (2018): dev167031. http://dx.doi.org/10.1242/dev.167031.

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27

Kim, Hosung, Benoit Caldairou, Ji-Wook Hwang, et al. "Accurate cortical tissue classification on MRI by modeling cortical folding patterns." Human Brain Mapping 36, no. 9 (2015): 3563–74. http://dx.doi.org/10.1002/hbm.22862.

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28

Sala, Ambre J., Laura C. Bott, and Richard I. Morimoto. "Shaping proteostasis at the cellular, tissue, and organismal level." Journal of Cell Biology 216, no. 5 (2017): 1231–41. http://dx.doi.org/10.1083/jcb.201612111.

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The proteostasis network (PN) regulates protein synthesis, folding, transport, and degradation to maintain proteome integrity and limit the accumulation of protein aggregates, a hallmark of aging and degenerative diseases. In multicellular organisms, the PN is regulated at the cellular, tissue, and systemic level to ensure organismal health and longevity. Here we review these three layers of PN regulation and examine how they collectively maintain cellular homeostasis, achieve cell type-specific proteomes, and coordinate proteostasis across tissues. A precise understanding of these layers of c
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29

Jodoin, Jeanne N., and Adam C. Martin. "Abl suppresses cell extrusion and intercalation during epithelium folding." Molecular Biology of the Cell 27, no. 18 (2016): 2822–32. http://dx.doi.org/10.1091/mbc.e16-05-0336.

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Tissue morphogenesis requires control over cell shape changes and rearrangements. In the Drosophila mesoderm, linked epithelial cells apically constrict, without cell extrusion or intercalation, to fold the epithelium into a tube that will then undergo epithelial-to-mesenchymal transition (EMT). Apical constriction drives tissue folding or cell extrusion in different contexts, but the mechanisms that dictate the specific outcomes are poorly understood. Using live imaging, we found that Abelson (Abl) tyrosine kinase depletion causes apically constricting cells to undergo aberrant basal cell ext
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30

Martin, Adam C. "The Physical Mechanisms of Drosophila Gastrulation: Mesoderm and Endoderm Invagination." Genetics 214, no. 3 (2020): 543–60. http://dx.doi.org/10.1534/genetics.119.301292.

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A critical juncture in early development is the partitioning of cells that will adopt different fates into three germ layers: the ectoderm, the mesoderm, and the endoderm. This step is achieved through the internalization of specified cells from the outermost surface layer, through a process called gastrulation. In Drosophila, gastrulation is achieved through cell shape changes (i.e., apical constriction) that change tissue curvature and lead to the folding of a surface epithelium. Folding of embryonic tissue results in mesoderm and endoderm invagination, not as individual cells, but as collec
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31

Wen, Fu-Lai, Chun Wai Kwan, Yu-Chiun Wang, and Tatsuo Shibata. "Autonomous epithelial folding induced by an intracellular mechano–polarity feedback loop." PLOS Computational Biology 17, no. 12 (2021): e1009614. http://dx.doi.org/10.1371/journal.pcbi.1009614.

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Epithelial tissues form folded structures during embryonic development and organogenesis. Whereas substantial efforts have been devoted to identifying mechanical and biochemical mechanisms that induce folding, whether and how their interplay synergistically shapes epithelial folds remains poorly understood. Here we propose a mechano–biochemical model for dorsal fold formation in the early Drosophila embryo, an epithelial folding event induced by shifts of cell polarity. Based on experimentally observed apical domain homeostasis, we couple cell mechanics to polarity and find that mechanical cha
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32

ONOE, Hiroaki, and Shoji TAKEUCHI. "713 Cell Origami : The Construction of 3D Cellular Tissue by Origami Folding." Proceedings of the Dynamics & Design Conference 2012 (2012): _713–1_—_713–3_. http://dx.doi.org/10.1299/jsmedmc.2012._713-1_.

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33

WATANABE, Masahiro, Mamoru UEDA, Ryosuke KUBOTA, Rio MIN, Katsuya TANAKA, and Toshihiko TAKENOBU. "A case of folding of the retrodiscal tissue of the temporomandibular joint." Japanese Journal of Oral and Maxillofacial Surgery 69, no. 12 (2023): 562–66. http://dx.doi.org/10.5794/jjoms.69.562.

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34

Denzel, Angela, Maurizio Molinari, Cesar Trigueros, et al. "Early Postnatal Death and Motor Disorders in Mice Congenitally Deficient in Calnexin Expression." Molecular and Cellular Biology 22, no. 21 (2002): 7398–404. http://dx.doi.org/10.1128/mcb.22.21.7398-7404.2002.

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ABSTRACT Calnexin is a ubiquitously expressed type I membrane protein which is exclusively localized in the endoplasmic reticulum (ER). In mammalian cells, calnexin functions as a chaperone molecule and plays a key role in glycoprotein folding and quality control within the ER by interacting with folding intermediates via their monoglucosylated glycans. In order to gain more insight into the physiological roles of calnexin, we have generated calnexin gene-deficient mice. Despite its profound involvement in protein folding, calnexin is not essential for mammalian-cell viability in vivo: calnexi
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35

Sriharsha, Tirumalasetty, Raghav Raj J., Sudha Venkatesan, et al. "A rare presentation of amyloid goiter with renal amyloidosis in a young female." International Journal of Advances in Medicine 10, no. 4 (2023): 304–6. http://dx.doi.org/10.18203/2349-3933.ijam20230706.

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Amyloidosis is a rare systemic disorder caused by abnormal folding of normal soluble proteins leading to fibril formation in one or more body organs, systems or soft tissues. Amyloid goiter is characterized by deposits of amyloid protein in the thyroid tissue. Amyloid infiltration of thyroid gland with development of secondary goiter is rare. Here we report a case of 36-year-old female presented with progressive painless swelling over neck. Thyroid profile was normal. Ultrasound neck showed enlarged bilateral thyroid gland and isthmus. Fine needle aspiration cytology suggestive of subacute thy
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36

Oldmixon, E. H., and F. G. Hoppin. "Alveolar septal folding and lung inflation history." Journal of Applied Physiology 71, no. 6 (1991): 2369–79. http://dx.doi.org/10.1152/jappl.1991.71.6.2369.

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On the basis of microscopic appearance of excised lungs, it has been thought that alveolar septa may fold and unfold during deflation and inflation. We suspected that this appearance might depend heavily on the inflation history of the lung preparation. We therefore studied, by light and electron microscopy, dog, rabbit, and rat lungs fixed over a range of inflation pressures and after a variety of inflation histories. Septal folding, as suggested by the configurations of the air spaces, by the placement of the fine and coarse connective tissue elements, and by the pattern of infolding of alve
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37

Matus, Soledad, Vicente Valenzuela, Danilo B. Medinas, and Claudio Hetz. "ER Dysfunction and Protein Folding Stress in ALS." International Journal of Cell Biology 2013 (2013): 1–12. http://dx.doi.org/10.1155/2013/674751.

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Amyotrophic lateral sclerosis (ALS) is the most frequent paralytic disease in adults. Most ALS cases are considered sporadic with no clear genetic component. The disruption of protein homeostasis due to chronic stress responses at the endoplasmic reticulum (ER) and the accumulation of abnormal protein inclusions are extensively described in ALS mouse models and patient-derived tissue. Recent studies using pharmacological and genetic manipulation of the unfolded protein response (UPR), an adaptive reaction against ER stress, have demonstrated a complex involvement of the pathway in experimental
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38

Long, Katherine R., and Wieland B. Huttner. "How the extracellular matrix shapes neural development." Open Biology 9, no. 1 (2019): 180216. http://dx.doi.org/10.1098/rsob.180216.

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During development, both cells and tissues must acquire the correct shape to allow their proper function. This is especially relevant in the nervous system, where the shape of individual cell processes, such as the axons and dendrites, and the shape of entire tissues, such as the folding of the neocortex, are highly specialized. While many aspects of neural development have been uncovered, there are still several open questions concerning the mechanisms governing cell and tissue shape. In this review, we discuss the role of the extracellular matrix (ECM) in these processes. In particular, we c
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Terada, Kazutoyo, Masaki Kanazawa, Bernd Bukau, and Masataka Mori. "The Human DnaJ Homologue dj2 Facilitates Mitochondrial Protein Import and Luciferase Refolding." Journal of Cell Biology 139, no. 5 (1997): 1089–95. http://dx.doi.org/10.1083/jcb.139.5.1089.

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DnaJ homologues function in cooperation with hsp70 family members in various cellular processes including intracellular protein trafficking and folding. Three human DnaJ homologues present in the cytosol have been identified: dj1 (hsp40/hdj-1), dj2 (HSDJ/hdj-2), and neuronal tissue-specific hsj1. dj1 is thought to be engaged in folding of nascent polypeptides, whereas functions of the other DnaJ homologues remain to be elucidated. To investigate roles of dj2 and dj1, we developed a system of chaperone depletion from and readdition to rabbit reticulocyte lysates. Using this system, we found tha
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40

Shirshakova, Maria, Elena Morozova, Daria Sokolova, Svetlana Pervykh, and Lyailya Kayumova. "Cosmetic Syndrome Correction with Calcium Hydroxylapatite-Based Filler in Patients with Connective Tissue Dysplasia." Dermatology Research and Practice 2021 (April 14, 2021): 1–7. http://dx.doi.org/10.1155/2021/6673058.

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Undifferentiated connective tissue dysplasia is one of the most common diseases of nowadays, which does not fit into the group of hereditary syndromes. This condition is diagnosed in 20–50% of the population at any age. The study aimed to correct the facial soft tissues of patients with undifferentiated connective tissue dysplasia through the cosmetic procedure of calcium hydroxylapatite injection. In 2018, a 36-year-old patient addressed the beauty salon with signs of undifferentiated connective tissue dysplasia, such as severe asymmetry of the face, infraorbital and nasolabial sulci, and thi
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Eritano, Anthony S., Claire L. Bromley, Antonio Bolea Albero, et al. "Tissue-Scale Mechanical Coupling Reduces Morphogenetic Noise to Ensure Precision during Epithelial Folding." Developmental Cell 53, no. 2 (2020): 212–28. http://dx.doi.org/10.1016/j.devcel.2020.02.012.

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42

Ionov, Leonid, Svetlana Zakharchenko, and Georgi Stoychev. "Soft Microorigami: Stimuli-Responsive Self-Folding Polymer Films." Advances in Science and Technology 77 (September 2012): 348–53. http://dx.doi.org/10.4028/www.scientific.net/ast.77.348.

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Asymmetry is intrinsic to natural systems and is widely used by living organisms for efficient adaptation, mimicry and movement. Polymer bilayers are the example of synthetic asymmetric systems, which are able to generate macroscopic motion and fold by forming different 3D objects such as tubes and capsules. Similar to bimetal films, the polymer bilayer consist of two substances with different swelling properties. One polymer is non-swellable and hydrophobic. Another polymer is water-swellable hydrogel. The folding, which might occur in response to temperature or pH, is caused by swelling of t
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43

Meng, Xiang-Sen, Li-Chuan Zhou, Lei Liu, et al. "Deformable hard tissue with high fatigue resistance in the hinge of bivalve Cristaria plicata." Science 380, no. 6651 (2023): 1252–57. http://dx.doi.org/10.1126/science.ade2038.

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The hinge of bivalve shells can sustain hundreds of thousands of repeating opening-and-closing valve motions throughout their lifetime. We studied the hierarchical design of the mineralized tissue in the hinge of the bivalve Cristaria plicata , which endows the tissue with deformability and fatigue resistance and consequently underlies the repeating motion capability. This folding fan–shaped tissue consists of radially aligned, brittle aragonite nanowires embedded in a resilient matrix and can translate external radial loads to circumferential deformation. The hard-soft complex microstructure
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44

Karbaschi, Mohammad Reza, Brett Williams, Acram Taji, and Sagadevan G. Mundree. "Tripogon loliiformis elicits a rapid physiological and structural response to dehydration for desiccation tolerance." Functional Plant Biology 43, no. 7 (2016): 643. http://dx.doi.org/10.1071/fp15213.

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Resurrection plants can withstand extreme dehydration to an air-dry state and then recover upon receiving water. Tripogon loliiformis (F.Muell.) C.E.Hubb. is a largely uncharacterised native Australian desiccation-tolerant grass that resurrects from the desiccated state within 72 h. Using a combination of structural and physiological techniques the structural and physiological features that enable T. loliiformis to tolerate desiccation were investigated. These features include: (i) a myriad of structural changes such as leaf folding, cell wall folding and vacuole fragmentation that mitigate de
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Kim, P. S., O. Y. Kwon, and P. Arvan. "An endoplasmic reticulum storage disease causing congenital goiter with hypothyroidism." Journal of Cell Biology 133, no. 3 (1996): 517–27. http://dx.doi.org/10.1083/jcb.133.3.517.

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In humans, deficient thyroglobulin (Tg, the thyroid prohormone) is an important cause of congenital hypothyroid goiter; further, homozygous mice expressing two cog/cog alleles (linked to the Tg locus) exhibit the same phenotype. Tg mutations might affect multiple different steps in thyroid hormone synthesis; however, the microscopic and biochemical phenotype tends to involve enlargement of the thyroid ER and accumulation of protein bands of M(r) < 100. To explore further the cell biology of this autosomal recessive illness, we have examined the folding and intracellular transport of new
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Liu, Zihan, Jorge Alemán-Báez, Richard G. F. Visser, and Guusje Bonnema. "Cabbage (Brassica oleracea var. capitata) Development in Time: How Differential Parenchyma Tissue Growth Affects Leafy Head Formation." Plants 13, no. 5 (2024): 656. http://dx.doi.org/10.3390/plants13050656.

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This study aims to categorize the morphological changes during cabbage (B. oleracea ssp. capitata) development, seedling, rosette, folding, and heading, and to elucidate the cellular mechanisms of the leaf curvature, essential for the formation of the leafy head. We followed the growth of two cabbage cultivars with distinct head shapes (round and pointed) and one non-heading collard cultivar; we phenotyped the size and volume of the whole plant as well as the size, shape, and curvature of the leaves during growth. By integrating these phenotypic data, we determined the four vegetative stages f
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Fal, Kateryna, Niklas Korsbo, Juan Alonso-Serra, et al. "Tissue folding at the organ–meristem boundary results in nuclear compression and chromatin compaction." Proceedings of the National Academy of Sciences 118, no. 8 (2021): e2017859118. http://dx.doi.org/10.1073/pnas.2017859118.

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Artificial mechanical perturbations affect chromatin in animal cells in culture. Whether this is also relevant to growing tissues in living organisms remains debated. In plants, aerial organ emergence occurs through localized outgrowth at the periphery of the shoot apical meristem, which also contains a stem cell niche. Interestingly, organ outgrowth has been proposed to generate compression in the saddle-shaped organ–meristem boundary domain. Yet whether such growth-induced mechanical stress affects chromatin in plant tissues is unknown. Here, by imaging the nuclear envelope in vivo over time
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Naglieri, Valentina. "Bio Focus: Nanopatterned self-folding origami may open up new possibilities in tissue engineering." MRS Bulletin 41, no. 11 (2016): 840. http://dx.doi.org/10.1557/mrs.2016.253.

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Wiréhn, Jimmy, Karin Carlsson, Anna Herland, et al. "Activity, Folding, Misfolding, and Aggregationin Vitroof the Naturally Occurring Human Tissue Factor Mutant R200W†." Biochemistry 44, no. 18 (2005): 6755–63. http://dx.doi.org/10.1021/bi047388l.

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Vasiev, Iskandar, Andrew I. M. Greer, Ali Z. Khokhar, John Stormonth-Darling, K. Elizabeth Tanner, and Nikolaj Gadegaard. "Self-folding nano- and micropatterned hydrogel tissue engineering scaffolds by single step photolithographic process." Microelectronic Engineering 108 (August 2013): 76–81. http://dx.doi.org/10.1016/j.mee.2013.04.003.

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