Relevant bibliographies by topics / TLR- 3

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'TLR- 3'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "TLR- 3"

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Pineda, Antonio, S. Leticia Verdin-Terán, Ausencio Camacho, and Leticia Moreno-Fierros. "Expression of Toll-like Receptor TLR-2, TLR-3, TLR-4 and TLR-9 Is Increased in Placentas from Patients with Preeclampsia." Archives of Medical Research 42, no. 5 (July 2011): 382–91. http://dx.doi.org/10.1016/j.arcmed.2011.08.003.

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Szczepański, Mirosław, Witold Szyfter, Renata Jenek, Maciej Wróbel, Iwona Mozer Lisewska, and Jan Żeromski. "Toll-like receptors 2, 3 and 4 (TLR-2, TLR-3 and TLR-4) are expressed in the microenvironment of human acquired cholesteatoma." European Archives of Oto-Rhino-Laryngology 263, no. 7 (March 15, 2006): 603–7. http://dx.doi.org/10.1007/s00405-006-0030-1.

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TAMAKI, YASUNOBU, YUYA TAKAKUBO, TOMOYUKI HIRAYAMA, YRJÖ T. KONTTINEN, STUART B. GOODMAN, MITSUNORI YAMAKAWA, and MICHIAKI TAKAGI. "Expression of Toll-like Receptors and Their Signaling Pathways in Rheumatoid Synovitis." Journal of Rheumatology 38, no. 5 (February 15, 2011): 810–20. http://dx.doi.org/10.3899/jrheum.100732.

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Objective.Toll-like receptors (TLR) recognizing endogenous and exogenous danger signals could play a role in rheumatoid arthritis (RA). Our aim was to describe the presence, localization, and extent of expression of TLR and their adapters.Methods.TLR 1, 2, 3, 4, 5, 6, and 9 receptors, and myeloid differentiation primary response protein 88, Toll/interleukin receptor (TIR) domain-containing adapter protein MyD88 adapter-like, and TIR domain-containing adapter-inducing interferon/TIR-containing adapter molecule-1 adapters were analyzed in RA (n = 10) and osteoarthritis (OA; n = 5) samples using
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van Tongeren, Joost, Korneliusz Golebski, Danielle Van Egmond, Esther J. de Groot, Wytske J. Fokkens, and Cornelis M. van Drunen. "Synergy between TLR-2 and TLR-3 signaling in primary human nasal epithelial cells." Immunobiology 220, no. 4 (April 2015): 445–51. http://dx.doi.org/10.1016/j.imbio.2014.11.004.

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Pryimenko, Nataliia O., Tetiana M. Kotelevska, Tetiana I. Koval, Vadym A. Bodnar, Liudmyla M. Syzova, and Stanislav S. Rudenko. "EFFICACY OF SPECIFIC PREVENTION OF INFLUENZA IN INDIVIDUALS WITH POLYMORPHISMS ARG753GLN OF TLR-2, LEU412PHE OF TLR-3, ASP299GLY OF TLR-4 GENES." Wiadomości Lekarskie 73, no. 9 (2020): 1944–49. http://dx.doi.org/10.36740/wlek202009209.

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The aim: Is to study the efficacy of influenza vaccination for individuals with polymorphism Arg753Gln of TLR-2 gene, Leu412Phe of TLR-3 gene, and Asp299Gly of TLR-4 gene. Materials and methods: 66 people with mutant genotypes and normal distribution of alleles of TLR-2, TLR-3, TLR-4 genes, aged 18-63, were inoculated with anti-influenza vaccine. The genotyping of Arg753Gln polymorphic site of TLR-2, Asp299Gly of TLR-4, and Leu412Phe of TLR-3 gene was carried out by polymerase chain reaction with oligonucleotide primers usage. The immunological efficacy of vaccination was evaluated by seroconv
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Qi, Chen, Xu Xiaofeng, and Wang Xiaoguang. "Effects of Toll-Like Receptors 3 and 4 in the Osteogenesis of Stem Cells." Stem Cells International 2014 (2014): 1–7. http://dx.doi.org/10.1155/2014/917168.

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Objective. To investigate the effects of Toll-like receptors in stem cell osteogenesis.Methods. Bone marrow mesenchymal stem cells (BMSCs) were divided into the blank group, the TLR-3 activated group, and the TLR-4 activated group. After 10 days’ osteogenic-promoting culture, expression of type I collagen and osteocalcin was determined by Western blot. Osteoblasts (OBs) were also divided into three groups mentioned above. Alkaline phosphatase (ALP) and alizarin red staining were performed after 10 days’ ossification-inducing culture. The expression ofβ-catenin was investigated by Western blot.
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López-Haber, Cynthia, Roni Levin-Konigsberg, Yueyao Zhu, Jing Bi-Karchin, Tamas Balla, Sergio Grinstein, Michael S. Marks та Adriana R. Mantegazza. "Phosphatidylinositol-4-kinase IIα licenses phagosomes for TLR4 signaling and MHC-II presentation in dendritic cells". Proceedings of the National Academy of Sciences 117, № 45 (27 жовтня 2020): 28251–62. http://dx.doi.org/10.1073/pnas.2001948117.

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Toll-like receptor (TLR) recruitment to phagosomes in dendritic cells (DCs) and downstream TLR signaling are essential to initiate antimicrobial immune responses. However, the mechanisms underlying TLR localization to phagosomes are poorly characterized. We show herein that phosphatidylinositol-4-kinase IIα (PI4KIIα) plays a key role in initiating phagosomal TLR4 responses in murine DCs by generating a phosphatidylinositol-4-phosphate (PtdIns4P) platform conducive to the binding of the TLR sorting adaptor Toll-IL1 receptor (TIR) domain-containing adaptor protein (TIRAP). PI4KIIα is recruited t
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O'Neill, Luke A. J. "TLR-7 and antiviral immunity." Trends in Immunology 23, no. 5 (May 2002): 234. http://dx.doi.org/10.1016/s1471-4906(02)02199-3.

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Wonder, Robyn, Steliana Penzkofer, and Evelyn G. Hazen. "Cardiotoxicity of anthracycline: Novel approach through down regulation of TLR-3 via TRAF/MAPK signaling pathway." American Journal of BioMedicine 2, no. 3 (June 2, 2015): 423–32. http://dx.doi.org/10.18081/2333-5106/015-02/413-422.

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Cardiotoxicity is one of the most important complications doxorubicin (DOX) and its pathomechanisms are not completely elucidated. We hypothesize that signaling via toll-like receptor (TLR)-3, a receptor that is activated upon binding of double-stranded nucleotides, might play a crucial role in the pathogenesis of cardiac-toxicity following DOX treatment. Male adult C57BL6 wild-type mice and TLR-3 knock-out (-/-) mice were subjected to 20 mg/kg; administered intraperitoneally. TLR-3 down-stream signaling was activated in WT mice lead to strong pro-inflammatory response with significant monocyt
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Wonder, Robyn, Steliana Penzkofer, and Evelyn G. Hazen. "Cardiotoxicity of anthracycline: Novel approach through down regulation of TLR-3 via TRAF/MAPK signaling pathway." American Journal of BioMedicine 3, no. 2 (June 2, 2015): 423–32. http://dx.doi.org/10.18081/2333-5106/015-02/423-432.

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Cardiotoxicity is one of the most important complications doxorubicin (DOX) and its pathomechanisms are not completely elucidated. We hypothesize that signaling via toll-like receptor (TLR)-3, a receptor that is activated upon binding of double-stranded nucleotides, might play a crucial role in the pathogenesis of cardiac-toxicity following DOX treatment. Male adult C57BL6 wild-type mice and TLR-3 knock-out (-/-) mice were subjected to 20 mg/kg; administered intraperitoneally. TLR-3 down-stream signaling was activated in WT mice lead to strong pro-inflammatory response with significant monocyt
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Dissertations / Theses on the topic "TLR- 3"

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Guillot, Loic. "Rôle des "toll-like receptor" (TLR) 3 et TLR4 dans l'immunité innée de la muqueuse pulmonaire." Paris 6, 2004. http://www.theses.fr/2004PA066147.

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Hamann, Timothy [Verfasser]. "Interaktion von TLR-3 stimuliertem retinalen Pigmentepithel und retinaler Mikroglia vor dem Hintergrund der altersabhängigen Makuladegeneration / Timothy Hamann." Kiel : Universitätsbibliothek Kiel, 2017. http://d-nb.info/114495519X/34.

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Desnous, Béatrice. "Effets de l'administration intra-hippocampique et intra-péritonéale de l'acide polyinosinique polycytidylique, agoniste des récepteurs TLR-3, sur l'épileptogenèse." Sorbonne Paris Cité, 2015. http://www.theses.fr/2015USPCC289.

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Notre approche expérimentale a pour but d'évaluer le rôle de l'inflammation virale dans l'épileptogenèse. Nous avons choisi de modéliser l'inflammation virale par injection d'un agoniste TLR-3, acide polyinosinique polycytidylique (PIC) soit intra-péritonéale, soit intra-hippocampique, Chaque modélisation inflammatoire était couplée à un modèle d'épileptogenèse électrique le kindling rapide. L'ensemble de notre travail expérimental était réalisé chez le rat Wistar à P14 et à P75. Notre travail a montré que l'injection I. H, de PIC facilitait l'épileptogenèse du cerveau immature et adulte et in
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Endoh, Yasumi Medical Sciences Faculty of Medicine UNSW. "New mechanisms modulating S100A8 gene expression." Publisher:University of New South Wales. Medical Sciences, 2008. http://handle.unsw.edu.au/1959.4/42942.

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S100A8 is a highly-expressed calcium-binding protein in neutrophils and activated macrophages, and has proposed roles in myeloid cell differentiation and host defense. Functions of S100A8 are not fully understood, partly because of difficulties in generating S100A8 knockout mice. Attempts to silence S100A8 gene expression in activated macrophages and fibroblasts using RNA interference (RNAi) technology were unsuccessful. Despite establishing validated small interfering RNA (siRNA) systems, enzymaticallysynthesized siRNA targeted to S100A8 suppressed mRNA levels by only 40% in fibroblasts activ
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Fallah, Mosoka Papa. "ROLE OF PI3K-AKT PATHWAY IN THE AGE ASSOCIATED DECLINE IN TLR MEDIATED ACTIVATION OF INNATE AND ADAPTIVE IMMUNE RESPONSES." UKnowledge, 2011. http://uknowledge.uky.edu/gradschool_diss/205.

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Immunosenescence results in reduced immune response to infections with Streptococcus pneumoniae as well as to pneumococcal polysaccharide vaccines. The antibody response to the capsular polysaccharide (CPS) provides protection against S. pneumoniae infection. CPS immunoresponse is T cell independent and needs the macrophage-derived cytokines such as IL-12, IL-6 and IL-1β to elicit an antibody response. We showed a cytokine dysregulation, i.e. a decrease in IL-12, IL-6 and TNF-α but an increase in IL-10, in the aged (18-24 months old comparable to >65 years in human) compared to young adult mou
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Campbell, Sara J. "Mechanisms of Moraxella catarrhalis Induced Immune Signaling in the Pulmonary Epithelium." University of Toledo Health Science Campus / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=mco1268141520.

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Salisbury, Richard L. Jr. "TCDD represses 3'IghRR activation through an AhR-dependent shift in the NF-κB/Rel protein complexes binding to κB motifs within the hs1,2 and hs4 enhancers". Wright State University / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=wright1401136335.

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Kuzemtseva, Liudmila. "Distribución tisular de los receptores Toll-like (TLR) 3, 7 y 9 en el cerdo y efecto in vitro de la infección por el virus de síndrome respiratorio y reproductivo porcino en su regulación en macrófagos alveolares porcinos." Doctoral thesis, Universitat Autònoma de Barcelona, 2012. http://hdl.handle.net/10803/284490.

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Los receptores Toll-like (TLRs), en particular los que se encuentran en vesículas intracelulares de origen endosomal (TLR3, TLR7 y TLR9), están involucrados en la respuesta innata antivírica. La unión a sus respectivos ligandos conduce a la activación de cascadas intracelulares que resultan en una producción de citoquinas pro-inflamatorias (TNF-α) y antivirales (interferones de tipo I). Existe muy poca información sobre la distribución celular y tisular de estos receptores en la especie porcina así como su regulación en condiciones normales o de infección. En el primer estudio de la presente
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Liljeroos, M. (Mari). "Toll-like receptor 2 (TLR2) and TLR4 signaling in the innate response against bacterial components." Doctoral thesis, University of Oulu, 2008. http://urn.fi/urn:isbn:9789514288111.

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Abstract Toll-like receptors (TLRs) are transmembrane proteins involved in the recognition of specific microbial structures and thus the activation of signaling cascades of innate immunity. Regulation of the innate immune response is a complex biological process involving the combined synergistic and antagonistic effects of distinct signaling mediators. Although TLR signaling has been widely studied in recent years, there remain many unexplored unique features of each TLR signaling pathway. The present study evaluated the activation and regulation of TLR4 and TLR2 signaling with the aim of bet
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Rao, Bhalchandra Shantikumar. "Diverse Biological Functions For 3'-5' Nucleotide Addition Reactions: tRNA Repair to tRNAHis Identity." The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1397425994.

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Books on the topic "TLR- 3"

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Zuffi, Marco, ed. Societas herpetologica italica. Atti del V congresso nazionale, Calci (Pisa), 29 settembre-3 ottobre 2004. Florence: Firenze University Press, 2006. http://dx.doi.org/10.36253/88-8453-420-8.

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This book contains the proceedings of the Congress, thus making available the extensive range of research performed and in progress. The contributions, in synthetic form, illustrate the methods and results, with the support of data, tables and bibliographies, of 35 studies carried out by research teams of biologists from the universities of Roma-Sapienza, Roma Tor Vergata, Florence, Naples, Turin, Milan, Palermo, Genoa, Pisa, Padua, Pavia, Perugia and Calabria, as well as various museums, study centres, reserves and nature parks.
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Bauer, Stefan, and Gunther Hartmann, eds. Toll-Like Receptors (TLRs) and Innate Immunity. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-72167-3.

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Klein, Michael, H. D. Schulte, and E. Gams, eds. TMLR Management of Coronary Artery Diseases. Berlin, Heidelberg: Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-642-72134-2.

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Mercuri, Louis G., ed. Temporomandibular Joint Total Joint Replacement – TMJ TJR. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-21389-7.

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Ait Ameur, Yamine, and Klaus-Dieter Schewe, eds. Abstract State Machines, Alloy, B, TLA, VDM, and Z. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-43652-3.

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Micheau, Olivier, ed. TRAIL, Fas Ligand, TNF and TLR3 in Cancer. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56805-8.

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Butler, Michael, Klaus-Dieter Schewe, Atif Mashkoor, and Miklos Biro, eds. Abstract State Machines, Alloy, B, TLA, VDM, and Z. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-33600-8.

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Butler, Michael, Alexander Raschke, Thai Son Hoang, and Klaus Reichl, eds. Abstract State Machines, Alloy, B, TLA, VDM, and Z. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91271-4.

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Congreso Nacional de Historia del Papel en España (5th 2003 Sarrià de Ter, Spain). Actas del V Congreso Nacional de Historia del Papel en España: Sarrià de Ter, Girona, 2, 3 y 4 de octubre de 2003. Girona: CCG Ediciones, 2003.

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Bi, Yunchen. Study of the Calcium Regulation Mechanism of TCR Activation Using Nanodisc and NMR Technologies. Berlin, Heidelberg: Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-54618-5.

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Book chapters on the topic "TLR- 3"

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Stasi, Alessandra, Rossana Franzin, Giuseppe Stefano Netti, Elena Ranieri, Loreto Gesualdo, Giovanni Stallone, and Giuseppe Castellano. "TLR-4 Signaling in Pericytes." In Stem Cell Biology and Regenerative Medicine, 165–87. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-62129-2_7.

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Neagu, Monica, and Carolina Constantin. "Signal Transduction in Immune Cells and Protein Kinases." In Advances in Experimental Medicine and Biology, 133–49. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-49844-3_5.

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AbstractImmune response relies upon several intracellular signaling events. Among the protein kinases involved in these pathways, members of the protein kinase C (PKC) family are prominent molecules because they have the capacity to acutely and reversibly modulate effector protein functions, controlling both spatial distribution and dynamic properties of the signals. Different PKC isoforms are involved in distinct signaling pathways, with selective functions in a cell-specific manner.In innate system, Toll-like receptor signaling is the main molecular event triggering effector functions. Various isoforms of PKC can be common to different TLRs, while some of them are specific for a certain type of TLR. Protein kinases involvement in innate immune cells are presented within the chapter emphasizing their coordination in many aspects of immune cell function and, as important players in immune regulation.In adaptive immunity T-cell receptor and B-cell receptor signaling are the main intracellular pathways involved in seminal immune specific cellular events. Activation through TCR and BCR can have common intracellular pathways while others can be specific for the type of receptor involved or for the specific function triggered. Various PKC isoforms involvement in TCR and BCR Intracellular signaling will be presented as positive and negative regulators of the immune response events triggered in adaptive immunity.
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Alpuente, María, Demis Ballis, Javier Espert, and Daniel Romero. "Model-Checking Web Applications with Web-TLR." In Automated Technology for Verification and Analysis, 341–46. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-15643-4_25.

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Martín, Óscar, Alberto Verdejo, and Narciso Martí-Oliet. "Model Checking TLR* Guarantee Formulas on Infinite Systems." In Specification, Algebra, and Software, 129–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-54624-2_7.

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Balistreri, Carmela Rita, Giuseppina Candore, and Calogero Caruso. "Role of TLR Polymorphisms in Aging and Age-Related Diseases." In Handbook of Immunosenescence, 1091–107. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-99375-1_34.

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Balistreri, Carmela Rita, Giuseppina Candore, and Calogero Caruso. "Role of TLR Polymorphisms in Aging and Age-Related Diseases." In Handbook of Immunosenescence, 1–18. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-64597-1_34-1.

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von Stebut, Esther. "Dendritische Zellen 2008: Verschiedene DC Subtypen, TLR-Profile und neue Zytokine." In Fortschritte der praktischen Dermatologie und Venerologie, 3–6. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-77148-7_1.

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Compte, Nathalie, Thierry Pepersack, and Stanislas Goriely. "Frailty in Old Age Is Associated with Altered Cytokine Production in Response to TLR Ligation." In Handbook of Immunosenescence, 2417–34. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-99375-1_152.

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Compte, Nathalie, Thierry Pepersack, and Stanislas Goriely. "Frailty in Old Age is Associated with Altered Cytokine Production in Response to TLR Ligation." In Handbook of Immunosenescence, 1–18. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-64597-1_152-1.

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Hornung, Veit, Winfried Barchet, Martin Schlee, and Gunther Hartmann. "RNA Recognition via TLR7 and TLR8." In Toll-Like Receptors (TLRs) and Innate Immunity, 71–86. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-72167-3_4.

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Conference papers on the topic "TLR- 3"

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Zhang, Wenqiu, Hyunjoon Kim, Vidhi Khanna, David M. Ferguson, Thomas Griffith, and Jayanth Panyam. "Abstract 4985: TLR agonists for anticancer immunotherapy." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4985.

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Zhang, Wenqiu, Hyunjoon Kim, Vidhi Khanna, David M. Ferguson, Thomas Griffith, and Jayanth Panyam. "Abstract 4985: TLR agonists for anticancer immunotherapy." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4985.

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Stolberg-Stolberg, J., A. Böttcher, M. Sambale, J. Sherwood, MJ Raschke, T. Pap, and J. Bertrand. "Inhibition des TLR-3 Signalwegs schützt vor post-traumatischer Arthrose." In Deutscher Kongress für Orthopädie und Unfallchirurgie. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0040-1717815.

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Takeoka, Tomohira, Hirotsugu Nagase, Yasuhiro Miyazaki, Tsuyoshi Takahashi, Yukinori Kurokawa, Tomoki Makino, Makoto Yamasaki, et al. "Abstract A169: NY-ESO-1 protein cancer vaccine with TLR 3 and 4 agonists." In Abstracts: CRI-CIMT-EATI-AACR Inaugural International Cancer Immunotherapy Conference: Translating Science into Survival; September 16-19, 2015; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr15-a169.

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Suresh, Madathilparambil V., Matthew D. Bender, Bi Yu, David Machado-Aranda, Beth B. Moore, and Krishnan Raghavendran. "Toll Like Receptor-3 (TLR-3) Is Required For Acute Inflammatory Response And Injury In Mice Following Lung Contusion." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1326.

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Aryee, Ken-Edwin, Lisa Burzenski, Dale Greiner, Giles F. Whalen, Leonard Shultz, James Keck, and Michael Brehm. "Abstract 1522: NovelNOD-scid IL2rgnull(NSG)mice for preclinical evaluation of TLR agonists in cancer immunotherapy." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1522.

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Aryee, Ken-Edwin, Lisa Burzenski, Dale Greiner, Giles F. Whalen, Leonard Shultz, James Keck, and Michael Brehm. "Abstract 1522: NovelNOD-scid IL2rgnull(NSG)mice for preclinical evaluation of TLR agonists in cancer immunotherapy." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1522.

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Haria, Dhwani, Helena Kiefel, Yuliya Katlinskaya, Sunit Jain, Thomas Weinmaier, Shoko Iwai, Todd DeSantis, Toshi Takeuchi, Karim Dabbagh, and Kareem Graham. "Abstract 1490: Novel microbiome-derived peptides activate the host innate immune system by regulation of TLR signaling." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1490.

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Haria, Dhwani, Helena Kiefel, Yuliya Katlinskaya, Sunit Jain, Thomas Weinmaier, Shoko Iwai, Todd DeSantis, Toshi Takeuchi, Karim Dabbagh, and Kareem Graham. "Abstract 1490: Novel microbiome-derived peptides activate the host innate immune system by regulation of TLR signaling." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1490.

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Sa, Y. A. P. J., N. S. B. Ribeiro, T. P. T. Ferreira, M. Hohmann, M. S. Espindola, J. C. Alves-Filho, M. A. Martins, C. M. Hogaboam, and P. M. Silva. "Role of Toll-Like Receptor (TLR)3 in Lung Fibrosis Triggered by Silica Particles in Mice." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a2642.

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Reports on the topic "TLR- 3"

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Struckmeyer, R. NRC TLD Direct Radiation Monitoring Network. Progress report, July--September 1993: Volume 13, No. 3. Office of Scientific and Technical Information (OSTI), November 1993. http://dx.doi.org/10.2172/10108304.

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Struckmeyer, R. NRC TLD direct radiation monitoring network: Progress report, July--September 1997. Volume 17, Number 3. Office of Scientific and Technical Information (OSTI), January 1998. http://dx.doi.org/10.2172/569100.

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Struckmeyer, R. NRC TLD direct radiation monitoring network: Volume 15, No. 3. Progress report, July--September 1995. Office of Scientific and Technical Information (OSTI), December 1995. http://dx.doi.org/10.2172/193651.

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Witt, D. C. Am/Cm TTR testing -- 3/8-inch glass beads evaluation in CIM5[Technical Task Request]. Office of Scientific and Technical Information (OSTI), January 2000. http://dx.doi.org/10.2172/750865.

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Dresner, L. The superfluid diffusion equation S(T)(@T/@t) = nabla ter dot (K(T)( nabla T) sup 1/3 ). Office of Scientific and Technical Information (OSTI), June 1990. http://dx.doi.org/10.2172/6702252.

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J. Cunningham and J. Shank. Guidelines for Electromagnetic Interference Testing of Power Plant Equipment: Revision 3 to TR-102323. Office of Scientific and Technical Information (OSTI), November 2004. http://dx.doi.org/10.2172/837279.

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Auerbach, E. A 3/2 LAMBDA BUMP TO CORRECT AGS ORBIT DISTORTION AT THE K17 GAMMA-TR QUAD. Office of Scientific and Technical Information (OSTI), May 1994. http://dx.doi.org/10.2172/1151302.

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Fisk, B. E., and D. A. Timian. In-Tank Precipitation Facility (ITP) and H-Tank Farm (HTF) geotechnical report, WSRC-TR-95-0057, Revision 0, Volume 3. Office of Scientific and Technical Information (OSTI), June 1995. http://dx.doi.org/10.2172/125432.

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Talbot, A. F., J. R. Swesey, and L. G. Magill. Turbine Fuels from Tar Sands Bitumen and Heavy Oil. Volume 2. Phase 3. Process Design Specifications for a Turbine Fuel Refinery Charging San Ardo Heavy Crude Oil. Fort Belvoir, VA: Defense Technical Information Center, September 1987. http://dx.doi.org/10.21236/ada190120.

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Miller, D., P. L. Richards, W. Y. Lee, N. Newman, S. M. Garrison, and J. S. Martens. Infrared phonon structure in epitaxial films of Tl sub 2 Ca sub 2 Ba sub 2 Cu sub 3 O sub 10 at low temperatures. Office of Scientific and Technical Information (OSTI), February 1992. http://dx.doi.org/10.2172/5001494.

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