Relevant bibliographies by topics / Tmem30a

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
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Academic literature on the topic 'Tmem30a'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Tmem30a"

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Yang, Yeming, Wenjing Liu, Kuanxiang Sun, Li Jiang, and Xianjun Zhu. "Tmem30a deficiency leads to retinal rod bipolar cell degeneration." Journal of Neurochemistry 148, no. 3 (2019): 400–412. http://dx.doi.org/10.1111/jnc.14643.

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Zhang, Shanshan, Wenjing Liu, Yeming Yang, et al. "TMEM30A deficiency in endothelial cells impairs cell proliferation and angiogenesis." Journal of Cell Science 132, no. 7 (2019): jcs225052. http://dx.doi.org/10.1242/jcs.225052.

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Chen, Rui, Erin Brady, and Thomas M. McIntyre. "Human TMEM30a Promotes Uptake of Antitumor and Bioactive Choline Phospholipids into Mammalian Cells." Journal of Immunology 186, no. 5 (2011): 3215–25. http://dx.doi.org/10.4049/jimmunol.1002710.

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Yang, Fan, Yumin Huang, Xianda Chen, et al. "Deletion of a flippase subunit Tmem30a in hematopoietic cells impairs mouse fetal liver erythropoiesis." Haematologica 104, no. 10 (2019): 1984–94. http://dx.doi.org/10.3324/haematol.2018.203992.

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Wang, Jiao, Qian Wang, Dongfang Lu, et al. "A biosystems approach to identify the molecular signaling mechanisms of TMEM30A during tumor migration." PLOS ONE 12, no. 6 (2017): e0179900. http://dx.doi.org/10.1371/journal.pone.0179900.

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Yang, Yeming, Kuanxiang Sun, Wenjing Liu та ін. "The phosphatidylserine flippase β-subunit Tmem30a is essential for normal insulin maturation and secretion". Molecular Therapy 29, № 9 (2021): 2854–72. http://dx.doi.org/10.1016/j.ymthe.2021.04.026.

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Liu, Leiming, Lingling Zhang, Lin Zhang, et al. "Hepatic Tmem30a Deficiency Causes Intrahepatic Cholestasis by Impairing Expression and Localization of Bile Salt Transporters." American Journal of Pathology 187, no. 12 (2017): 2775–87. http://dx.doi.org/10.1016/j.ajpath.2017.08.011.

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Ennishi, Daisuke, Shannon Healy, Ali Bashashati, et al. "TMEM30A loss-of-function mutations drive lymphomagenesis and confer therapeutically exploitable vulnerability in B-cell lymphoma." Nature Medicine 26, no. 4 (2020): 577–88. http://dx.doi.org/10.1038/s41591-020-0757-z.

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Li, Ning, Yeming Yang, Cailing Liang, et al. "Tmem30a Plays Critical Roles in Ensuring the Survival of Hematopoietic Cells and Leukemia Cells in Mice." American Journal of Pathology 188, no. 6 (2018): 1457–68. http://dx.doi.org/10.1016/j.ajpath.2018.02.015.

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Sun, Kuanxiang, Wanli Tian, Xiao Li, Wenjing Liu, Yeming Yang, and Xianjun Zhu. "Disease Mutation Study Identifies Critical Residues for Phosphatidylserine Flippase ATP11A." BioMed Research International 2020 (June 2, 2020): 1–9. http://dx.doi.org/10.1155/2020/7342817.

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Phosphatidylserine flippase (P4-ATPase) transports PS from the outer to the inner leaflet of the lipid bilayer in the membrane to maintain PS asymmetry, which is important for biological activities of the cell. ATP11A is expressed in multiple tissues and plays a role in myotube formation. However, the detailed cellular function of ATP11A remains elusive. Mutation analysis revealed that I91, L308, and E897 residues in ATP8A2 are important for flippase activity. In order to investigate the roles of these corresponding amino acid residues in ATP11A protein, we assessed the expression and cellular
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Dissertations / Theses on the topic "Tmem30a"

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Gego, Audrey. "Identification de protéines hépatocytaires impliquées dans l'infection hépatique de plasmodium par une approche d'interférence à ARN à grande échelle." Paris 6, 2009. http://www.theses.fr/2009PA066263.

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Afin de mettre en évidence des protéines de l’hôte nécessaires au développement des formes intra-hépatiques de Plasmodium chez l’Homme, nous avons utilisé comme outil l’ARN interférence, pour atténuer l’expression de 6080 gènes de l’hôte dans le modèle d’infection HepG2-A16/hCD81EGFP / P. Yoelii. Après deux étapes de sélection successives, vinqt-sept gènes ont été retenus. L’invalidation de ces gènes par ARNi induit une diminution de l’infection d’au moins 30% par rapport aux contrôles sans altérer la viabilité des cellules hôtes. Nous en avons étudié cinq d’entre eux : ALDH18A1, LIPG, TMEM30A
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Adomaviciene, Aiste. "TMEM16A channels : molecular physiology and pharmacological regulation." Thesis, University of Manchester, 2012. https://www.research.manchester.ac.uk/portal/en/theses/tmem16a-channels-molecular-physiology-and-pharmacological-regulation(681d1c72-3207-41f5-bd78-c6af0a6ccdf3).html.

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Calcium-activated chloride channels (CaCCs) are a class of the ligand-gated channels involved in numerous cellular functions. In vascular smooth muscle, these ion channels couple agonist-induced calcium-release from the sarcoplasmic reticulum to membrane depolarisation and vasoconstriction. For this reason, CaCCs have been suggested as a potential molecular target to treat a range of vascular disorders. These ion channels, however, have not been yet explored as a drug target because their molecular identity has been elusive and their pharmacology has been restricted to compounds with low poten
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Brookfield, Rebecca. "The pharmacology and cardiovascular function of TMEM16A channels." Thesis, University of Manchester, 2015. https://www.research.manchester.ac.uk/portal/en/theses/the-pharmacology-and-cardiovascular-function-of-tmem16a-channels(bdc16466-cecd-4343-9d40-b20bc647d70f).html.

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Calcium-activated chloride channels (CaCCs) are ubiquitously expressed in a plethora of cell types and, consequently, are involved in numerous cellular processes as diverse as epithelial secretion, regulation of cardiac excitability and smooth muscle contraction. Current pharmacology of CaCCs is limited to compounds with low potency and poor selectivity. The lack of knowledge surrounding the molecular identity of the CaCC has greatly hindered the development of more specific drugs and has impaired our understanding of the channel physiology and biophysics. The recent discovery that the TMEM16A
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Suzuki, Takayuki. "Functional Swapping between Transmembrane Proteins TMEM16A and TMEM16F." Kyoto University, 2014. http://hdl.handle.net/2433/188693.

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Page, Henry Askew. "An investigation into the role of TMEM16A in the coronary vasculature." Thesis, St George's, University of London, 2017. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.754073.

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Introduction: Failure of coronary blood vessels to adequately supply cardiac myocytes with oxygen underlies ischaemic heart disease and ultimately myocardial infarction. Therefore, it is important to determine the factors that regulate coronary blood flow. Activation of Ca2+-activated chloride channels depolarises vascular smooth muscle cells sufficiently to cause Ca2+ influx through voltage-dependent channels and contraction of the cell. TMEM16A is the main molecular candidate for these channels. Hypothesis: TMEM16A is expressed in rat coronary arteries where it regulates vascular smooth musc
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Ayoub, Christine. "Analyse de TMEM16A, un gène surexprimé dans les cancers des voies-aéro-digestives supérieures." Strasbourg, 2009. http://www.theses.fr/2009STRA6279.

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Le gène TMEM16A a été isolé à l’issu d’un crible à l’aide de « Differential Display » et « microarrays » visant à identifier des gènes dont l’expression est altérée dans des cellules cancéreuses des VADS. TMEM16A est fortement exprimé dans les cancers des VADS et dans plusieurs autres types de cancers. De plus, la protéine est localisée préférentiellement à la surface des cellules situées au niveau des fronts d’invasion. TMEM16A se situe dans le locus CCND1-EMS1 de la région génomique 11q13 souvent amplifiée dans les cancers humains. L’analyse bioinformatique montre que TMEM16A possède plusieu
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Jin, Xin. "Regulation of Ca2+ activated Cl- channel ANO1 (TMEM16A) by different Ca2+ sources in sensory neurons." Thesis, University of Leeds, 2015. http://etheses.whiterose.ac.uk/9246/.

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Proteins of anoctamin (TMEM16) family are the candidate subunits for Ca2+- activated Cl- channels (CaCC). In recent years, studies have shown that anoctamin-1 (TMEM16A or ANO1) plays important physiological roles in processes including epithelial fluid secretion, muscle contraction and olfactory transduction. How the Ca2+ regulates the activity of ANO1 in different tissue is still not clear. In this study, I showed that the excitatory CaCC in nociceptors (small-diameter sensory neurons that are responsible for transmission of painful stimuli) was activated by the release of Ca2+ from the 1, 4,
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Forst, Maik der [Verfasser], and Heimo [Akademischer Betreuer] Ehmke. "Die Relevanz des vaskulären TMEM16A für die Blutdruckregulation bei kreislaufwirksamen Bedingungen / Maik der Forst ; Betreuer: Heimo Ehmke." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2017. http://d-nb.info/1137624949/34.

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Brochier, Camille. "Analyse des transcriptomes du cerveau de souris : mise en évidence de patrons régionaux d'expression conservés chez l'homme et altérés dans des modèles de maladies neurodégénératives." Phd thesis, Université Paris Sud - Paris XI, 2007. http://tel.archives-ouvertes.fr/tel-00361207.

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L'analyse à grande échelle de l'expression des gènes dans le cerveau est une approche puissante et relativement nouvelle, susceptible d'identifier des gènes candidats pour l'analyse des fonctions cérébrales. Nous avons utilisé la méthode Serial Analysis of Gene Expression pour établir un profil d'expression quantitatif de 11 régions du cerveau de souris, dont plusieurs régions corticales, le noyau accumbens, le striatum, le thalamus, la substance noire et l'aire tegmentale ventrale. Plus d'un million d'étiquettes SAGE ont été générées, permettant la détection de transcrits peu abondants. La co
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Heinze, Christoph [Verfasser], Christian A. [Akademischer Betreuer] Hübner, Aria [Akademischer Betreuer] Baniahmad, and Heimo [Akademischer Betreuer] Ehmke. "Die Bedeutung des calciumaktivierten Chloridkanals TMEM16A in vaskulären glatten Muskelzellen / Christoph Heinze. Gutachter: Christian A. Hübner ; Aria Baniahmad ; Heimo Ehmke." Jena : Thüringer Universitäts- und Landesbibliothek Jena, 2015. http://d-nb.info/1076038409/34.

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Books on the topic "Tmem30a"

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Hoi Hellenes tes Voulgarias: Hena historiko tmema tou periphereiakou Hellenismou (Hidryma Meleton Chersonesou tou Haimou). Panellenia Omospondia Syllogon Anatolikes Romylias, 1999.

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Evgenios Vulgaris und die neugriechische Aufklärung in Leipzig: Beiträge der Konferenz an der Universität Leipzig, Institut für Klassische Philologie, Abteilung Byzantinische und Neugriechische Philologie, vom 16.-18. Oktober 1996 = O Eugenios Voulgares : kai o Neoellenikos Diaphotismos ste Lipsia : Anakoinoseis Synedriou sto Institouto Klasikes Philologias (Tmema Byzantines kai Neoellenikes Philologias) tou Panepistemiou Lipsias, 16-18 Oktobriou 1996. Leipziger Universitätsverlag, 2003.

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S, Henrich Gunther, and Universitat Leipzig. Abteilung Byzantinische und Neugriechische Philologie., eds. Evgenios Vulgaris und die neugriechische Aufklarung in Leipzig: Beitrage der Konferenz an der Universitat Leipzig, Institut fur Klassische Philologie, Abteilung Byzantinische und Neugriechische Philologie, vom 16.-18. Oktober 1996 = O Eugenios Voulgares kai o Neoellenikos Diaphotismos ste Lipsia = Anakoinoseis Synedriou sto Institouto Klasikes Philologias (Tmema Byzantines kai Neoellenikes Philologias) tou Panepistemiou Lipsias, 16-18 Oktobriou 1996. Leipziger Universitatsverlag, 2003.

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Book chapters on the topic "Tmem30a"

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Mizuta, Ken, Masahide Sakabe, Satoshi Somekawa, Yoshihiko Saito, and Osamu Nakagawa. "TMEM100: A Novel Intracellular Transmembrane Protein Essential for Vascular Development and Cardiac Morphogenesis." In Etiology and Morphogenesis of Congenital Heart Disease. Springer Japan, 2016. http://dx.doi.org/10.1007/978-4-431-54628-3_21.

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Diodato, Daria, Federica Invernizzi, Eleonora Lamantea, et al. "Common and Novel TMEM70 Mutations in a Cohort of Italian Patients with Mitochondrial Encephalocardiomyopathy." In JIMD Reports. Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/8904_2014_300.

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Conference papers on the topic "Tmem30a"

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Kondo, M., K. Hara, M. Tsuji, A. Kurokawa, K. Takeyama, and E. Tagaya. "TMEM16A Inhibitors Decrease TMEM16A Expression and Goblet Cell Metaplasia in IL-13-Treated Guinea Pig Trachea In Vivo." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2186.

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Bai, Xiaoyun, and Di Xia. "Abstract 2112: Structural characterization of transmembrane protein TMEM205." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-2112.

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Bai, Xiaoyun, and Di Xia. "Abstract 2112: Structural characterization of transmembrane protein TMEM205." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-2112.

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Cao, G., B. Deeney, E. Chung та ін. "TMEM16A Modulates TGFβ1-Induced MLC2 Phosphorylation in Human Airway Smooth Muscle". У American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1255.

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Danielsson, J., A. Kuforiji, Y. Zhang, and C. W. Emala. "TMEM16A Agonism Contracts Airway Smooth Muscle in In Vivo and Ex Vivo Models." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a2839.

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Danielsson, J., G. T. Yocum, D. B. Xu, and C. W. Emala. "TMEM16A siRNA Knockdown Increases Expression of PPARG Coactivator 1 Alpha in Airway Smooth Muscle." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1250.

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Dai, Z., Y. Zhao, and J. Dai. "Single-Cell Transcriptomes Identify Endothelial TMEM100 Playing a Pathogenic Role in Pulmonary Arterial Hypertension." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a7863.

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Schäfer, J., H. Janssen, A. Bicker, et al. "Loss of Sirtuin6 induces expression of the cancer-related transmembrane protein TMEM45A in HepG2 cells." In 36. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0039-3402222.

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Yang, L., S.-J. Yu, and Z.-M. Shao. "Abstract P2-01-16: CRISPR-Cas9 screen identifies TMEM106A as a suppressor of breast cancer metastasis." In Abstracts: 2018 San Antonio Breast Cancer Symposium; December 4-8, 2018; San Antonio, Texas. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-p2-01-16.

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Wu, A., A. Kuforiji, C. W. Emala та J. Danielsson. "The TMEM16A Antagonist Benzbromarone Decreases β2-Adrenergic Receptor Desensitization in Human Airway Smooth Muscle In Vitro". У American Thoracic Society 2021 International Conference, May 14-19, 2021 - San Diego, CA. American Thoracic Society, 2021. http://dx.doi.org/10.1164/ajrccm-conference.2021.203.1_meetingabstracts.a4502.

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Reports on the topic "Tmem30a"

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Armani, Gonzalo R., María Cruz Tubio, and Hernán D. Eiroa. Mutación en el Gen TMEM70: una Forma de Aciduria 3-Metilglutacónica con Fenotipo Variable. Presentación de Dos Casos Clínicos. Siicsalud.com, 2018. http://dx.doi.org/10.21840/siic/157864.

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