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1

Meija-Feldmane, Anete. "Leachates of Thermally Modified Pine (Pinus sylvestris L.) Wood." Rural Sustainability Research 34, no. 329 (2015): 26–31. http://dx.doi.org/10.1515/plua-2015-0010.

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Abstract During the last decades, thermally modified wood has become an object of interest among the wood scientists as an environmentally friendly material, because nowadays environmental aspects of materials have become more and more significant. Leaching is one of the processes that occurs in outdoor use. The aim of this study was to evaluate concentration of potentially hazardous substances in leachates of thermally modified pine wood. Scots pine (Pinus sylvestris L.) wood was thermally modified using Wood Treatment Technology (WTT) company device at 170 °C for 1 hour (TMP170/1) and at 160
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2

Zahin, Maryam, Shin-je Ghim, Sujita Khanal, Gregory D. Bossart, Alfred B. Jenson, and Joongho Joh. "Molecular characterization of novel mucosotropic papillomaviruses from a Florida manatee (Trichechus manatus latirostris)." Journal of General Virology 96, no. 12 (2015): 3545–53. http://dx.doi.org/10.1099/jgv.0.000293.

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We isolated two new manatee papillomavirus (PV) types, TmPV3 and TmPV4, from a Florida manatee (Trichechus manatus latirostris). Two PV types were previously isolated from this species. TmPV1 is widely dispersed amongst manatees and a close-to-root PV; not much is known about TmPV2. The genomes of TmPV3 and TmPV4 were 7622 and 7771 bp in size, respectively. Both PVs had a genomic organization characteristic of all PVs, with one non-coding region and seven ORFs, including the E7 ORF that is absent in other cetacean PVs. Although these PVs were isolated from separate genital lesions of the same
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3

Asakura, Nobuaki, Chiharu Nakamura, and Ichiro Ohtsuka. "Homoeoallelic gene Ncc-tmp of Triticum timopheevii conferring compatibility with the cytoplasm of Aegilops squarrosa in the tetraploid wheat nuclear background." Genome 43, no. 3 (2000): 503–11. http://dx.doi.org/10.1139/g00-009.

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A nuclear gene, Ncc-tmp1A, of Triticum timopheevii is required for the nucleus-cytoplasm (NC) compatibility in tetraploid NC hybrids with the cytoplasm of Aegilops squarrosa. A euploid NC hybrid of T. durum was previously produced by introgressing the gene from chromosome 1A of T. timopheevii. To examine the possible presence of a functional homoeoallele in the G genome of T. timopheevii, segregation of seed viability was studied as a marker phenotype in BC1s involving the two types of NC hybrids, (Ae. squarrosa) - T. timopheevii and (Ae. squarrosa) - T. turgidum. The result of these test cros
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4

Wu, Chung-Pu, Sabrina Lusvarghi, Jyun-Cheng Wang, et al. "The Selective Class IIa Histone Deacetylase Inhibitor TMP195 Resensitizes ABCB1- and ABCG2-Overexpressing Multidrug-Resistant Cancer Cells to Cytotoxic Anticancer Drugs." International Journal of Molecular Sciences 21, no. 1 (2019): 238. http://dx.doi.org/10.3390/ijms21010238.

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Multidrug resistance caused by the overexpression of the ATP-binding cassette (ABC) proteins in cancer cells remains one of the most difficult challenges faced by drug developers and clinical scientists. The emergence of multidrug-resistant cancers has driven efforts from researchers to develop innovative strategies to improve therapeutic outcomes. Based on the drug repurposing approach, we discovered an additional action of TMP195, a potent and selective inhibitor of class IIa histone deacetylase. We reveal that in vitro TMP195 treatment significantly enhances drug-induced apoptosis and sensi
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5

Zhang, Wei, Yinjie Guan, George Bayliss, and Shougang Zhuang. "Class IIa HDAC inhibitor TMP195 alleviates lipopolysaccharide-induced acute kidney injury." American Journal of Physiology-Renal Physiology 319, no. 6 (2020): F1015—F1026. http://dx.doi.org/10.1152/ajprenal.00405.2020.

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Sepsis-associated acute kidney injury (SA-AKI) is associated with high mortality rates, but clinicians lack effective treatments except supportive care or renal replacement therapies. Recently, histone deacetylase (HDAC) inhibitors have been recognized as potential treatments for acute kidney injury and sepsis in animal models; however, the adverse effect generated by the use of pan inhibitors of HDACs may limit their application in people. In the present study, we explored the possible renoprotective effect of a selective class IIa HDAC inhibitor, TMP195, in a murine model of SA-AKI induced b
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6

Nagesh, Narayana, Varun K. Sharma, A. Ganesh Kumar, and Edwin A. Lewis. "Effect of Ionic Strength on Porphyrin Drugs Interaction with Quadruplex DNA Formed by the Promoter Region of C-myc and Bcl2 Oncogenes." Journal of Nucleic Acids 2010 (2010): 1–9. http://dx.doi.org/10.4061/2010/146418.

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C-myc and Bcl2 are well characterized oncogenes that are capable of forming G-quadruplex structures. Promoter regions of C-myc and Bcl2 forming G-quadruplex structures are chemically synthesized and G-quadruplex structure is formed in presence of 100 mM potassium ion. Three different porphyrin drugs, namely TMPyP2, TMPyP3, and TMPyP4 are allowed to interact with quadruplex DNA complex and the site and nature of interaction are studied. Drug interactions with quadruplex DNA were carried out in different potassium ionic strengths using fluorescence spectroscopy. It is found that fluorescence hyp
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7

Wang, HongBin, та Marcelo G. Kazanietz. "p23/Tmp21 Differentially Targets the Rac-GAP β2-Chimaerin and Protein Kinase C via Their C1 Domains". Molecular Biology of the Cell 21, № 8 (2010): 1398–408. http://dx.doi.org/10.1091/mbc.e09-08-0735.

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The C1 domains in protein kinase C (PKC) isozymes and other signaling molecules are responsible for binding the lipid second messenger diacylglycerol and phorbol esters, and for mediating translocation to membranes. Previous studies revealed that the C1 domain in α- and β-chimaerins, diacylglycerol-regulated Rac-GAPs, interacts with the endoplasmic reticulum/Golgi protein p23/Tmp21. Here, we found that p23/Tmp21 acts as a C1 domain-docking protein that mediates perinuclear translocation of β2-chimaerin. Glu227 and Leu248 in the β2-chimaerin C1 domain are crucial for binding p23/Tmp21 and perin
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8

Asare, Yaw, Thomas A. Campbell-James, Yury Bokov, et al. "Histone Deacetylase 9 Activates IKK to Regulate Atherosclerotic Plaque Vulnerability." Circulation Research 127, no. 6 (2020): 811–23. http://dx.doi.org/10.1161/circresaha.120.316743.

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Rationale: Arterial inflammation manifested as atherosclerosis is the leading cause of mortality worldwide. Genome-wide association studies have identified a prominent role of HDAC (histone deacetylase)-9 in atherosclerosis and its clinical complications including stroke and myocardial infarction. Objective: To determine the mechanisms linking HDAC9 to these vascular pathologies and explore its therapeutic potential for atheroprotection. Methods and Results: We studied the effects of Hdac9 on features of plaque vulnerability using bone marrow reconstitution experiments and pharmacological targ
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9

Kishk, Safaa M., Rania M. Kishk, Asmaa S. A. Yassen, et al. "Molecular Insights into Human Transmembrane Protease Serine-2 (TMPS2) Inhibitors against SARS-CoV2: Homology Modelling, Molecular Dynamics, and Docking Studies." Molecules 25, no. 21 (2020): 5007. http://dx.doi.org/10.3390/molecules25215007.

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The severe acute respiratory syndrome coronavirus 2 (SARS-CoV2), which caused novel corona virus disease-2019 (COVID-19) pandemic, necessitated a global demand for studies related to genes and enzymes of SARS-CoV2. SARS-CoV2 infection depends on the host cell Angiotensin-Converting Enzyme-2 (ACE2) and Transmembrane Serine Protease-2 (TMPRSS2), where the virus uses ACE2 for entry and TMPRSS2 for S protein priming. The TMPRSS2 gene encodes a Transmembrane Protease Serine-2 protein (TMPS2) that belongs to the serine protease family. There is no crystal structure available for TMPS2, therefore, a
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10

Liu, Shengchun, Si Zhang, Kelley Bromley-Brits, et al. "Transcriptional Regulation of TMP21 by NFAT." Molecular Neurodegeneration 6, no. 1 (2011): 21. http://dx.doi.org/10.1186/1750-1326-6-21.

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11

Nagesh, Narayana, and Arumugam Ganesh Kumar. "Interaction of TMPyP4, TMPyP3, and TMPyP2 with Intramolecular G-Quadruplex Formed by Promoter Region of Bcl2 and KRAS NHPPE." ISRN Biophysics 2012 (February 7, 2012): 1–12. http://dx.doi.org/10.5402/2012/786596.

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Oncogenes are rich in guanine and capable of forming quadruplex structure. Promoter regions oncogenes such as Bcl2 and KRAS NHPPE are rich in guanine content and they can form quadruplex structures. Alterations in the mode and nature of molecular binding to DNA, certainly has effect on the posttranscriptional activities. Recent experiments indicate that structure of quadruplex complex and ligand has predominant role on ligand-quadruplex DNA interaction. In order to understand the nature of each ligand interaction with quadruplex DNA, Bcl2, KRAS NHPPE quadruplex DNA interaction with three porph
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12

Choi, Sun-Il, Seung-Wan Jee, Youn-Kyung Her, et al. "Overexpression of TMP21 Could Induce not only Downregulation of TrkA/ERK Phosphorylation but also Upregulation of p75NTR/RhoA Expression on NGF Receptor Signaling Pathway." Journal of Life Science 21, no. 8 (2011): 1134–41. http://dx.doi.org/10.5352/jls.2011.21.8.1134.

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13

Pardossi-Piquard, Raphaëlle, Christopher Böhm, Fusheng Chen та ін. "TMP21 Transmembrane Domain Regulates γ-Secretase Cleavage". Journal of Biological Chemistry 284, № 42 (2009): 28634–41. http://dx.doi.org/10.1074/jbc.m109.059345.

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14

Hörer, Jürgen, Robert Blum, Peter Feick, Wolfgang Nastainczyk, and Irene Schulz. "A Comparative Study of Rat and Human Tmp21 (p23) Reveals the Pseudogene-Like Features of Human Tmp21-I1." DNA Sequence 10, no. 2 (1999): 121–26. http://dx.doi.org/10.3109/10425179909008429.

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15

Cho, Eun-Kyung, and Young-Ju Choi. "Antioxidant and Anti-Inflammatory Activities of Extracts of Traditional Medicinal Plants Mixtures 1 and 2." Journal of Life Science 21, no. 4 (2011): 596–603. http://dx.doi.org/10.5352/jls.2011.21.4.596.

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16

Poublanc, Julien, Adrian Crawley, Larissa Mcketton, Kevin Sam, and David J. Mikulis. "Abstract TMP112: Evidence of Diffuse Cortical Vascular Dysfunction in Patients With White Matter Hyperintensities." Stroke 50, Suppl_1 (2019). http://dx.doi.org/10.1161/str.50.suppl_1.tmp112.

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Dakay, Katarina, Andrew D. Chang, Morgan Hemendinger, et al. "Abstract TMP112: Left Atrial Enlargement is Associated With Anticoagulation Failure in Patients With Acute Ischemic Stroke and Atrial Fibrillation." Stroke 49, Suppl_1 (2018). http://dx.doi.org/10.1161/str.49.suppl_1.tmp112.

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18

Ma, Li, Yu Chen, and Yuanli Zhao. "Abstract TMP112: Subsequent Hemorrhage in Children With Untreated Brain Arteriovenous Malformation: Higher Risk With Unbalanced Inflow and Outflow Angioarchitecture." Stroke 48, suppl_1 (2017). http://dx.doi.org/10.1161/str.48.suppl_1.tmp112.

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Instruction: Children with brain arteriovenous malformations (bAVM) are at risk of life-threatening hemorrhage in their early lives. Our aim was to analyze various angiographic features of bAVM in conjunction with other morphological risk factors to predict the risk of subsequent hemorrhage during follow-up in children. Methods: We identified all consecutive children admitted to our institution for bAVMs between July 2009 and September 2015. Children with at least 1 month of treatment-free follow-up after diagnosis were included in further analysis. The effects of bAVM features on hemorrhagic
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19

Guilliams, Kristin, Melanie Fields, Niket Gupta, et al. "Abstract TMP112: Increased Large Vessel Diameter is Associated With Increased Cerebral Metabolic Stress in Children With Sickle Cell Anemia." Stroke 51, Suppl_1 (2020). http://dx.doi.org/10.1161/str.51.suppl_1.tmp112.

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Background: Children with sickle cell anemia (SCA) invoke compensatory mechanisms to meet cerebral oxygen demand in the setting of low oxygen carrying capacity. Whereas cerebral blood flow (CBF) is largely regulated by cerebral arterioles as one compensatory mechanism, we sought to determine if the cross-sectional area of the large in-flow arteries was also increased. Methods: Children and young adults with SCA underwent 3 Tesla magnetic resonance angiography (MRA) and imaging (MRI) with pseudocontinuous arterial spin labeling (pCASL) and asymmetric spin echo sequence to measure CBF and oxygen
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20

He, Hongpeng, Xiang Liu, Yue Liu, et al. "Human Papillomavirus E6/E7 and Long Noncoding RNA TMPOP2 Mutually Upregulated Gene Expression in Cervical Cancer Cells." Journal of Virology 93, no. 8 (2019). http://dx.doi.org/10.1128/jvi.01808-18.

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ABSTRACT TMPOP2 was previously suggested to be an oncogenic long noncoding RNA which is excessively expressed in cervical cancer cells and inhibits E-cadherin gene expression by recruiting transcription repressor EZH2 to the gene promoter. So far, the function and regulation of TMPOP2 in cervical cancer remain largely unknown. Herein, we found that TMPOP2 expression was correlated with human papillomavirus 16/18 (HPV16/18) E6 and E7 in cervical cancer cell lines CaSki and HeLa. Tumor suppressor p53, which is targeted for degradation by HPV16/18, was demonstrated to associate with two p53 respo
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Romero, Jose R., Serkalem Demissie, Alexa Beiser, et al. "Abstract TMP115: Carotid Atherosclerosis Progression and Hippocampal Volume." Stroke 50, Suppl_1 (2019). http://dx.doi.org/10.1161/str.50.suppl_1.tmp115.

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22

Lau, Susanna K. P., George C. S. Lo, Franklin W. N. Chow, et al. "Novel Partitivirus Enhances Virulence of and Causes Aberrant Gene Expression inTalaromyces marneffei." mBio 9, no. 3 (2018). http://dx.doi.org/10.1128/mbio.00947-18.

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ABSTRACTTalaromyces marneffeiis the most important thermal dimorphic fungus causing systemic mycosis in Southeast Asia. We report the discovery of a novel partitivirus,Talaromyces marneffeipartitivirus-1 (TmPV1). TmPV1 was detected in 7 (12.7%) of 55 clinicalT. marneffeiisolates. Complete genome sequencing of the seven TmPV1 isolates revealed two double-stranded RNA (dsRNA) segments encoding RNA-dependent RNA polymerase (RdRp) and capsid protein, respectively. Phylogenetic analysis showed that TmPV1 occupied a distinct clade among the members of the genusGammapartitivirus. Transmission electro
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23

Felling, Ryan J., Mubeen F. Rafay, Timothy Bernard, et al. "Abstract TMP102: Predicting Recovery and Outcome After Pediatric Stroke." Stroke 49, Suppl_1 (2018). http://dx.doi.org/10.1161/str.49.suppl_1.tmp102.

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Biffi, Alessandro, Christopher D. Anderson, Alison M. Ayres, Steven M. Greenberg, Jonathan Rosand, and Anand Viswanathan. "Abstract TMP113: Long-term Cognitive Decline After Intracerebral Hemorrhage." Stroke 47, suppl_1 (2016). http://dx.doi.org/10.1161/str.47.suppl_1.tmp113.

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Introduction: Cerebral small vessel disease (CSVD) is the leading cause of Vascular Cognitive Impairment (VCI). Although Intracerebral Hemorrhage (ICH) is the most severe manifestation of CSVD, VCI among ICH survivors has been the subject of limited investigations. Hypothesis: We sought to test these hypotheses: 1) long-term cognitive decline rates are substantial even among initially non-demented ICH survivors; 2) underlying CSVD plays a substantial role in determining post-ICH long-term cognitive decline. Methods: We enrolled survivors of primary ICH, with no evidence of dementia 6 months po
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Uchiyama, Shinichiro, Takao Hoshino, Leila Sissani, et al. "Abstract TMP119: The Second Report of Japanese Sub-Analysis of TIAregistry.org." Stroke 49, Suppl_1 (2018). http://dx.doi.org/10.1161/str.49.suppl_1.tmp119.

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Chen, Wanqiu, Yi Guo, Kristine Jun, Mamta Wankhede, Hua Su, and William L. Young. "Abstract TMP118: Alk1 Deficiency Impairs Mural Cell Recruitment During Brain Angiogenesis." Stroke 44, suppl_1 (2013). http://dx.doi.org/10.1161/str.44.suppl_1.atmp118.

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Background and Purpose: VEGF stimulation induced dysplasia vessel formation in the Alk1 -deficient brain. The dysplastic vessels had less mural cell coverage associated with increased extravasation of blood contents. Platelet-derived growth factor B (PDGFB) signaling is important in mural cell recruitment during angiogenesis. Notch signaling is upstream of PDGFB. We hypothesized that Alk1 deletion impairs pericyte recruitment through downregulation of DLL4/PDGFB signaling. Thalidomide can increase PDGFB expression and was used to test our hypothesis in vivo. Methods: Human brain microvascular
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Zafar, Atif, Jeffrey Nelson, Charles E. McCulloch, et al. "Abstract TMP116: Intracranial Hemorrhage Rate in Familial Cerebral Cavernous Malformation Patients." Stroke 51, Suppl_1 (2020). http://dx.doi.org/10.1161/str.51.suppl_1.tmp116.

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Background: Familial cerebral cavernous malformations (FCCM) is an autosomal dominant disease caused by mutations in three genes: CCM1 , CCM2 , or CCM3 . FCCM patients typically present with multiple brain CCMs that can increase in number and size over time, and can cause intracranial hemorrhage (ICH) and other disabling symptoms, e.g., seizures, headaches, and deficits. Rates and predictors of ICH events in FCCM are not well described. The purpose of this study was to estimate ICH event rates and predictors in a cohort of FCCM patients followed prospectively. Methods: We collected baseline an
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Murthy, Santosh, Alexander E. Merkler, Setareh S. Omran, et al. "Abstract TMP110: Outcomes After Intracerebral Hemorrhage in Patients With Arteriovenous Malformations." Stroke 48, suppl_1 (2017). http://dx.doi.org/10.1161/str.48.suppl_1.tmp110.

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Background: Few population-level data exist regarding functional outcomes after intracerebral hemorrhage (ICH) caused by a ruptured cerebral arteriovenous malformation (AVM). Our aim was to compare outcomes after ICH from AVM rupture versus other causes of ICH. Methods: We performed a retrospective population-based study using data from the Nationwide Inpatient Sample (NIS), a nationally representative survey of hospitalizations across the U.S. We used previously validated International Classification of Diseases, 9 th Revision, Clinical Modification ( ICD-9-CM ) diagnosis codes to identify IC
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Nakai, Ryosuke, Hiroyuki Kusada, Fumihiro Sassa, et al. "Draft Genome Sequence of Novel Filterable Rhodospirillales Bacterium Strain TMPK1, Isolated from Soil." Microbiology Resource Announcements 10, no. 28 (2021). http://dx.doi.org/10.1128/mra.00393-21.

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We report the draft genome sequence of a novel Rhodospirillales bacterium strain, TMPK1, isolated from a micropore-filtered soil suspension. This strain has a genome of 4,249,070 bp, comprising 4,151 protein-coding sequences. The genome sequence data further suggest that strain TMPK1 is an alphaproteobacterium capable of carotenoid production.
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Jickling, Glen, Bradley P. Ander, Xinhua Zhan, et al. "Abstract TMP117: Relationship of Cerebral White Matter Hyperintensity Progression and Leukocyte Activation." Stroke 50, Suppl_1 (2019). http://dx.doi.org/10.1161/str.50.suppl_1.tmp117.

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31

Shimizu, Tomohisa, Kenji Sugiu, Koji Tokunaga, et al. "Abstract TMP119: Stereotactic Radiosurgery For Dural Arteriovenous Fistula Reflactory To Endovascular Treatment." Stroke 44, suppl_1 (2013). http://dx.doi.org/10.1161/str.44.suppl_1.atmp119.

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Background: Although almost all dural arteriovenous fistulas (DAVFs) can be highly curable using endovascular treatment, some of them are reflactory. We evaluated the efficacy of stereotactic radiosurgery for DAVFs reflactory to endovascular treatment. Methods: In 178 patients with DAVFs treated by endovascular embolization in our institution, 12 patients were reflactory and treated by CyberKnife (CK) radiosurgery. Patients background, efficacy of CyberKnife radiosurgery and complication were analyzed. Results: 5 men and 7 women underwent CyberKnife radiosurgery. Ages ranged from 21 to 74 year
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Kirton, Adam, Janice Yajure, Ion Leah, Jamie Roe, Steve Scott, and Sean Dukelow. "Abstract TMP102: Robotic Assessment Of Proprioceptive Dysfunction In Children With Perinatal Stroke." Stroke 44, suppl_1 (2013). http://dx.doi.org/10.1161/str.44.suppl_1.atmp102.

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Objective: Perinatal stroke causes most hemiplegic cerebral palsy. Sensory dysfunction has been ignored and objective measurement tools are limited. Robotic technology can quantify complex sensory function in adult stroke but has not been applied to kids. Methods: Children from the Alberta Perinatal Stroke Project had MR confirmed unilateral perinatal stroke and upper extremity functional deficit. A bilateral exoskeletal robot (KINARM) capable of testing planar upper limb movements in an augmented reality environment was employed. Primary robotic outcomes were 2 dimensional variability, shift,
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Carrion-Penagos, Julian, Sean P. Polster, Hussein A. Zeineddine, et al. "Abstract TMP117: Subclinical Imaging Changes in Cerebral Cavernous Angiomas During Prospective Surveillance." Stroke 51, Suppl_1 (2020). http://dx.doi.org/10.1161/str.51.suppl_1.tmp117.

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Introduction: Studies on the natural clinical course of Cavernous Angioma (CA) have reported a <0.5% annual risk of symptomatic hemorrhage (SH). CAs, however, may undergo subclinical changes that can reflect lesional activity. We assess the frequency of asymptomatic hemorrhage or growth (AH) in CAs, and new lesion formation (NLF) during systematic longitudinal follow-up using magnetic resonance imaging (MRI), and correlate with prior SH. Methods: Patients with CA diagnosis were prospectively enrolled in IRB approved database from 09/2009 to 02/2019 (N=327). Imaging and clinical follow-up we
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Mayasi, Yunis M., Johana Helenius, and Nils Henninger. "Abstract TMP116: White Matter Hyperintensity Lesion Patterns in Patients With Cardioembolic Strokes." Stroke 48, suppl_1 (2017). http://dx.doi.org/10.1161/str.48.suppl_1.tmp116.

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Background: Among patients with a cardioembolic stroke, atrial fibrillation (AF) is a common comorbidity that is associated with a worse outcome. Mounting data indicates that AF may relate to stroke pathogenesis through mechanisms other than cerebral thromboembolic complications. We sought to identify different WMH patterns between patients with AF vs. non-AF related embolic ischemic strokes. Methods: We retrospectively analyzed consecutive patients with acute anterior circulation embolic ischemic stroke included in an academic medical center’s stroke registry. The stroke etiology was determin
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35

Vaughan, Jessica, Michael Taylor, James L. Leach, Paul Horn, Sudhakar Vadivelu, and Todd Abruzzo. "Abstract TMP113: Non-invasive Estimate of Intracranial Arterial Luminal Diameter in Children." Stroke 51, Suppl_1 (2020). http://dx.doi.org/10.1161/str.51.suppl_1.tmp113.

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Background: Growth of the intracranial arteries facilitates the rapid increase in cerebral growth in the first 2 decades of life. In the age of cerebral endovascular treatment, many questions arise in the pediatric cohort regarding the safety and efficacy of these therapeutic approaches. Small arterial diameter may influence treatment selection in the very young, but to date little normative data is available to define vascular growth in infants and children. Methods: Subjects in this single-institution, retrospective cohort study were identified by MRI brain imaging keyword query as “normal”
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Dunbar, Mary J., Hely Shah, Siddharth Shinde, Joseph Vayalumkal, Otto G. Vanderkooi, and Adam Kirton. "Abstract TMP104: Stroke in Paediatric Bacterial Meningitis." Stroke 49, Suppl_1 (2018). http://dx.doi.org/10.1161/str.49.suppl_1.tmp104.

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Nirwane, Abhijit, Benjamin Nguyen, Jeffrey Miner та Yao Yao. "Abstract TMP102: Loss of Laminin-α5 From Mural Cells Attenuates Ischemia-reperfusion Injury". Stroke 50, Suppl_1 (2019). http://dx.doi.org/10.1161/str.50.suppl_1.tmp102.

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Chantler, Paul D., Stephen D. Brooks, Kayla W. Branyan, et al. "Abstract TMP115: Nox-2 and Cerebrovascular Function in Metabolic Syndrome After a Stroke." Stroke 48, suppl_1 (2017). http://dx.doi.org/10.1161/str.48.suppl_1.tmp115.

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Introduction: The aim here is to elucidate the mechanisms involved in post-stroke cerebrovascular impairments in metabolic syndrome (MetS) that pre-dispose obese patients to more severe post-stroke deficits. Methods: 1 hr middle cerebral artery occlusion was performed in 17 week old lean (LZR) and obese Zucker (OZR: MetS model) rats. At 24 hr or 15 days post-stroke, the ipsilateral middle cerebral artery (MCA) was cannulated in an ex-vivo microvessel preparation to examine MCA reactivity. Cerebrovascular microvessel density (MVD) was examined 15 days post-stroke via IHC. To examine a potential
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Levine, Deborah A., Alden L. Gross, Emily M. Briceño, et al. "Abstract TMP110: Design of the BP-COG Study of Blood Pressure Over the Life Course and Later-Life Cognition in Blacks, Hispanics, and Whites: A Pooled Cohort Analysis of ARIC, CARDIA, CHS, FOS, MESA, and NOMAS." Stroke 50, Suppl_1 (2019). http://dx.doi.org/10.1161/str.50.suppl_1.tmp110.

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Pasi, Marco, Lansing Sugita, Li Xiong, et al. "Abstract TMP111: Prediction of Cognitive Impairment after Intracerebral Hemorrhage using MRI Small Vessel Disease Score." Stroke 50, Suppl_1 (2019). http://dx.doi.org/10.1161/str.50.suppl_1.tmp111.

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Simpkins, Alexis N., Amir Sarayani, Nandakumar Nagaraja, et al. "Abstract TMP113: Vascular Dementia Amongst Patients Included in the National Inpatient Sample Database 2006-2014 is not on the Decline." Stroke 50, Suppl_1 (2019). http://dx.doi.org/10.1161/str.50.suppl_1.tmp113.

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Kudrin, Bertina, Emily Goldmann, Eric T. Roberts, Nina S. Parikh, Noa Appleton, and Bernadette Boden-Albala. "Abstract TMP114: Exploring the Role of Cognitive Function in Secondary Stroke Prevention: Evidence from the Discharge Educational Strategies for Reduction of Vascular Events (DESERVE) Study." Stroke 50, Suppl_1 (2019). http://dx.doi.org/10.1161/str.50.suppl_1.tmp114.

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Cushman, Mary, Peter W. Callas, Jennifer J. Manly, et al. "Abstract TMP116: Higher Lipoprotein(a) is a Risk Factor for Cognitive Impairment in Black but Not White Americans: The Reasons for Geographic and Racial Differences in Stroke (REGARDS) Cohort." Stroke 50, Suppl_1 (2019). http://dx.doi.org/10.1161/str.50.suppl_1.tmp116.

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Bryan, Robert M., James W. Nelson, Bojun Zhang, and David J. Durgan. "Abstract TMP118: The Gut Microbiota Can Protect Against the Onset of Symptoms in an Animal Model of Cerebral Small Vessel Disease." Stroke 50, Suppl_1 (2019). http://dx.doi.org/10.1161/str.50.suppl_1.tmp118.

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Tomek, Ales, Petr Janský, Anna Olšerovã, et al. "Abstract TMP110: The Correlation of Through Plasmatic Concentration of Dabigatran and CES1 Genotype With Major Bleeding in Stroke Patients." Stroke 49, Suppl_1 (2018). http://dx.doi.org/10.1161/str.49.suppl_1.tmp110.

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Sun, Jie, Emily Goldmann, Nina S. Parikh, Noa Appleton, and Bernadette Boden-Albala. "Abstract TMP111: Physical Activity among Stroke Patients in the Discharge Educational Strategies for Reduction of Vascular Events (DESERVE) Study." Stroke 49, Suppl_1 (2018). http://dx.doi.org/10.1161/str.49.suppl_1.tmp111.

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Nakajima, Makoto, Yasuteru Ito, Tomohiro Takita, et al. "Abstract TMP113: Do Preceding Direct Oral Anticoagulants Make Stroke Milder in Patients With Atrial Fibrillation? -K-PLUS Registry." Stroke 49, Suppl_1 (2018). http://dx.doi.org/10.1161/str.49.suppl_1.tmp113.

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Wang, Anxin, Xia Meng, Yongjun Wang, and Yilong Wang. "Abstract TMP114: Oxidized Low Density Lipoprotein Predicts Recurrent Stroke in Patients With Minor Stroke or Transient Ischemic Attack." Stroke 49, Suppl_1 (2018). http://dx.doi.org/10.1161/str.49.suppl_1.tmp114.

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Wen, Chi Pang, Chung Y. Hsu, June Han Lee, Po Jung Lu, and Min Kuang Tsai. "Abstract TMP115: Identifying Overlooked Risk Factors For Stroke From A Prediction Model Constructed From Half A Million Adults In Taiwan." Stroke 49, Suppl_1 (2018). http://dx.doi.org/10.1161/str.49.suppl_1.tmp115.

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Stein, Laura K., Gurmeen Kaur, John Liang, Stanley Tuhrim, Stanley Tuhrim, and Mandip S. Dhamoon. "Abstract TMP116: Predictors of Readmissions Following Percutaneous Patent Foramen Ovale Closure in a Nationally Representative Dataset." Stroke 49, Suppl_1 (2018). http://dx.doi.org/10.1161/str.49.suppl_1.tmp116.

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