Log in

Relevant bibliographies by topics / TNFRSF10A

Contents

Journal articles
Dissertations / Theses
Book chapters
Conference papers

Academic literature on the topic 'TNFRSF10A'

Author: Grafiati

Published: 4 June 2021

Last updated: 14 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'TNFRSF10A.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "TNFRSF10A"

1

Teocchi, Marcelo Ananias, and Lília D’Souza-Li. "Apoptosis through Death Receptors in Temporal Lobe Epilepsy-Associated Hippocampal Sclerosis." Mediators of Inflammation 2016 (2016): 1–12. http://dx.doi.org/10.1155/2016/8290562.

Full text

Abstract:

Seizure models have demonstrated that neuroinflammation and neurodegeneration are preponderant characteristics of epilepsy. Considering the lack of clinical studies, our aim is to investigate the extrinsic pathway of apoptosis in pharmacoresistant temporal lobe epilepsy (TLE) associated with hippocampal sclerosis (HS) patients, TLE(HS). By a specific death receptor-mediated apoptosis array plate, 31 upregulated targets were revealed in the sclerotic hippocampus from TLE(HS) patients. Amongst them are the encoding genes for ligands (FASLG,TNF,andTNFSF10) and death receptors (FAS,TNFRSF1A,TNFRSF

APA, Harvard, Vancouver, ISO, and other styles

2

Carnicer, Maria J., Adriana Lasa, Elena Serrano, et al. "Epigenetic-Based Treatment Induces Apoptosis in Leukemic Cell Lines." Blood 108, no. 11 (2006): 4382. http://dx.doi.org/10.1182/blood.v108.11.4382.4382.

Full text

Abstract:

Abstract Pharmacological treatment of cancer cells with demethylating agents and histone deacetylase inhibitors synergistically reactivates the transcription of previously silenced genes. The aim of this study was to investigate the antileukemic properties of a DNA mehtyltransferase inhibitor 5-aza-2′-deoxycytidine (5-Aza-dC),an histone deacetylase inhibitor Trichostatin A (TSA) and trans-retinoc acid (ATRA), alone or in combination. The effects of these drugs on apoptosis, cell cycle progression, cell-survival pathways and restoration of proliferation-associated genes silenced by aberrant epi

APA, Harvard, Vancouver, ISO, and other styles

3

Seirafian, Sepehr, Virginie Prod’homme, Daniel Sugrue, et al. "Human cytomegalovirus suppresses Fas expression and function." Journal of General Virology 95, no. 4 (2014): 933–39. http://dx.doi.org/10.1099/vir.0.058313-0.

Full text

Abstract:

Human cytomegalovirus (HCMV) is known to evade extrinsic pro-apoptotic pathways not only by downregulating cell surface expression of the death receptors TNFR1, TRAIL receptor 1 (TNFRSF10A) and TRAIL receptor 2 (TNFRSF10B), but also by impeding downstream signalling events. Fas (CD95/APO-1/TNFRSF6) also plays a prominent role in apoptotic clearance of virus-infected cells, so its fate in HCMV-infected cells needs to be addressed. Here, we show that cell surface expression of Fas was suppressed in HCMV-infected fibroblasts from 24 h onwards through the late phase of productive infection, and wa

APA, Harvard, Vancouver, ISO, and other styles

4

Lee, Kuei-Fang, Julia Tzu-Ya Weng, Paul Wei-Che Hsu, et al. "Gene Expression Profiling of Biological Pathway Alterations by Radiation Exposure." BioMed Research International 2014 (2014): 1–9. http://dx.doi.org/10.1155/2014/834087.

Full text

Abstract:

Though damage caused by radiation has been the focus of rigorous research, the mechanisms through which radiation exerts harmful effects on cells are complex and not well-understood. In particular, the influence of low dose radiation exposure on the regulation of genes and pathways remains unclear. In an attempt to investigate the molecular alterations induced by varying doses of radiation, a genome-wide expression analysis was conducted. Peripheral blood mononuclear cells were collected from five participants and each sample was subjected to 0.5 Gy, 1 Gy, 2.5 Gy, and 5 Gy of cobalt 60 radiati

APA, Harvard, Vancouver, ISO, and other styles

5

Li, Tianliang, Ling Su, Yuanjiu Lei, Xianfang Liu, Yajing Zhang, and Xiangguo Liu. "DDIT3 and KAT2A Proteins Regulate TNFRSF10A and TNFRSF10B Expression in Endoplasmic Reticulum Stress-mediated Apoptosis in Human Lung Cancer Cells." Journal of Biological Chemistry 290, no. 17 (2015): 11108–18. http://dx.doi.org/10.1074/jbc.m115.645333.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Xiao, Zheng, Kechao Nie, Tong Han, et al. "Development and Validation of a TNF Family-Based Signature for Predicting Prognosis, Tumor Immune Characteristics, and Immunotherapy Response in Colorectal Cancer Patients." Journal of Immunology Research 2021 (September 9, 2021): 1–16. http://dx.doi.org/10.1155/2021/6439975.

Full text

Abstract:

In this study, a comprehensive analysis of TNF family members in colorectal cancer (CRC) was conducted and a TNF family-based signature (TFS) was generated to predict prognosis and immunotherapy response. Using the expression data of 516 CRC patients from The Cancer Genome Atlas (TCGA) database, TNF family members were screened to construct a TFS by using the univariate Cox proportional hazards regression and the least absolute shrinkage and selection operator- (LASSO-) Cox proportional hazards regression method. The TFS was then validated in a meta-Gene Expression Omnibus (GEO) cohort ( n = 1

APA, Harvard, Vancouver, ISO, and other styles

7

Rinaldi, Andrea, Ekaterina Chigrinova, Ivo Kwee, et al. "SNP-Arrays Provide New Insights Into the Pathogenesis of Richter Syndrome (RS)." Blood 118, no. 21 (2011): 263. http://dx.doi.org/10.1182/blood.v118.21.263.263.

Full text

Abstract:

Abstract Abstract 263 RS represents the development of a diffuse large B-cell lymphoma (DLBCL) in the context of chronic lymphocytic leukemia (CLL). The pathogenesis of RS is still largely unknown. Analysis of RS has been often focused on the study of lesions previously identified in de novo DLBCL. However, we have previously shown that RS lacks many of the typical genetic lesions shown in DLBCL (Rossi et al, Blood 2011). Here, we have applied an unbiased, genome-wide approach, searching for DNA copy number alterations in a large series of RS, comparing them with de novo DLBCL, CLL-phase of RS

APA, Harvard, Vancouver, ISO, and other styles

8

Kohlhammer, Holger, Carsten Schwaenen, Swen Wessendorf, et al. "Genomic DNA-chip hybridization in t(11;14)-positive mantle cell lymphomas shows a high frequency of aberrations and allows a refined characterization of consensus regions." Blood 104, no. 3 (2004): 795–801. http://dx.doi.org/10.1182/blood-2003-12-4175.

Full text

Abstract:

AbstractTumor samples of 53 patients with t(11;14)-positive mantle cell lymphomas (MCLs) were analyzed by matrix-based comparative genomic hybridization (matrix-CGH) using a dedicated DNA array. In 49 cases, genomic aberrations were identified. In comparison to chromosomal CGH, a 50% higher number of aberrations was found and the high specificity of matrix-CGH was demonstrated by fluorescence in situ hybridization (FISH) analyses. The 11q gains and 13q34 deletions, which have not been described as frequent genomic aberrations in MCL, were identified by matrix-CGH in 15 and 26 cases, respective

APA, Harvard, Vancouver, ISO, and other styles

9

Liu, Yang, Yong Zhang, Phong Quang, et al. "Deregulation of TNFRSF18 (GITR) Through Promoter CpG Island Methylation Induces Tumor Proliferation in Multiple Myeloma." Blood 118, no. 21 (2011): 2424. http://dx.doi.org/10.1182/blood.v118.21.2424.2424.

Full text

Abstract:

Abstract Abstract 2424 Introduction Tumor necrosis factor receptor super families (TNFRSFs) play an important role in activation of lymphocyte and cell apoptosis. However the function of TNFRSFs in multiple myeloma (MM) remains unknown. Loss of function mutation of Fas antigen (TNFRSF6) was identified in MM cells, thus suggesting the possible role of TNFRSFs in regulating MM pathogenesis. We therefore investigated the epigenetic mechanisms that may mediate inactivation of TNFRSFs and its functional role in MM. Methods Dchip software was utilized for analyzing gene expression dataset. DNA was e

APA, Harvard, Vancouver, ISO, and other styles

10

Dufva, Olli, Khalid Saeed, Sara Gandolfi, et al. "CRISPR Screens Identify Mechanisms of Natural Killer Cell Evasion across Blood Cancers." Blood 134, Supplement_1 (2019): 3597. http://dx.doi.org/10.1182/blood-2019-129837.

Full text

Abstract:

Natural killer (NK) cells have been suggested to control progression and relapse in several hematological malignancies. Enhancing NK cell reactivity represents an attractive approach to improve treatment responses. However, mechanisms enabling evasion of hematologic cancer cells from NK cells are incompletely understood. To identify cancer cell-intrinsic factors enabling resistance to NK cell cytotoxicity, we conducted genome-wide CRISPR screens in a range of hematological malignancies. Cas9-expressing cancer cells from diverse hematological malignancies, including acute and chronic myeloid le

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "TNFRSF10A"

1

Hosoda, Yoshikatsu. "Genome-wide association analyses identify two susceptibility loci for pachychoroid disease central serous chorioretinopathy." Kyoto University, 2020. http://hdl.handle.net/2433/253180.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Rebelo, V. Susana Lopes. "Structural and signalling properties of TRAPS-associated TNFRSF1A mutants." Thesis, University of Nottingham, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.437070.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Nedjai, Belinda. "Genetic and functional studies of TNFRSF1A mutations in inflammatory disorders." Thesis, Queen Mary, University of London, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.515237.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Ishii, Kei. "The Trp53-Trp53inp1-Tnfrsf10b Pathway Regulates the Radiation Response of Mouse Spermatogonial Stem Cells." Kyoto University, 2015. http://hdl.handle.net/2433/195971.

Full text

Abstract:

Kei Ishii, Masamichi Ishiai, Hiroko Morimoto, Mito Kanatsu-Shinohara, Ohtsura Niwa, Minoru Takata, Takashi Shinohara, The Trp53-Trp53inp1-Tnfrsf10b Pathway Regulates the Radiation Response of Mouse Spermatogonial Stem Cells, Stem Cell Reports, Volume 3, Issue 4, 14 October 2014, Pages 676-689, ISSN 2213-6711<br>Kyoto University (京都大学)<br>0048<br>新制・課程博士<br>博士(医学)<br>甲第18685号<br>医博第3957号<br>新制||医||1007(附属図書館)<br>31618<br>京都大学大学院医学研究科医学専攻<br>(主査)教授斎藤通紀, 教授藤田潤, 教授近藤玄<br>学位規則第4条第1項該当

APA, Harvard, Vancouver, ISO, and other styles

5

Sazzini, Marco <1980&gt. "TNFRSF13B Genetic variability an anthropological - evolutionary approach to Biomedical Research." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2009. http://amsdottorato.unibo.it/1692/.

Full text

Abstract:

In the recent years TNFRSF13B coding variants have been implicated by clinical genetics studies in Common Variable Immunodeficiency (CVID), the most common clinically relevant primary immunodeficiency in individuals of European ancestry, but their functional effects in relation to the development of the disease have not been entirely established. To examine the potential contribution of such variants to CVID, the more comprehensive perspective of an evolutionary approach was applied in this study, underling the belief that evolutionary genetics methods can play a role in dissecting the origin,

APA, Harvard, Vancouver, ISO, and other styles

6

Siebert, Stefan. "Functional characterisation of tumour necrosis factor receptor superfamily 1A (TNFRSF1A) mutations that cause systemic inflammation." Thesis, Cardiff University, 2005. http://orca.cf.ac.uk/55610/.

Full text

Abstract:

This work describes a study into the effects of clinically relevant TNF receptor superfamily 1A (TNFRSF1A) mutations associated with the TNF receptor-associated periodic syndrome (TRAPS). TRAPS is a dominantly inherited autoinflammatory disorder characterised by episodes of systemic inflammation. The first part focuses on a novel TNFRSF1A mutation (C43S) from a patient with TRAPS. A primary dermal fibroblast line was established from the patient and used to study the functional effects of this TRAPS mutation. Experiments revealed that the C43S TNFRSF1A mutation resulted in reduced activation o

APA, Harvard, Vancouver, ISO, and other styles

7

Schön, Simon. "TNFRSF19 als Zielgen des Wnt/β-Catenin-Signalweges im kolorektalen Karzinon". Diss., Ludwig-Maximilians-Universität München, 2014. http://nbn-resolving.de/urn:nbn:de:bvb:19-172145.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Okur, Anne Maria Sophie [Verfasser], Jörg T. [Akademischer Betreuer] Epplen, and Claus G. [Akademischer Betreuer] Haase. "Zur Bedeutung von Variationen in den \(\it TNFRSF1A\)- und \(\it TNFAIP3\)-Genen bei Multipler Sklerose / Anne Maria Sophie Okur. Gutachter: Jörg T. Epplen ; Claus G. Haase." Bochum : Ruhr-Universität Bochum, 2015. http://d-nb.info/1079843612/34.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Fichtner, Miriam Franziska Laura [Verfasser], and Edgar [Akademischer Betreuer] Meinl. "Features of the two isoforms of TACI/TNFRSF13B in soluble and membrane-bound form / Miriam Franziska Laura Fichtner ; Betreuer: Edgar Meinl." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2018. http://d-nb.info/1200353234/34.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Schön, Simon [Verfasser], та Frank [Akademischer Betreuer] Kolligs. "TNFRSF19 als Zielgen des Wnt/β-Catenin-Signalweges im kolorektalen Karzinon : Untersuchungen zu Expression, Regulation und Funktion / Simon Schön. Betreuer: Frank Kolligs". München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2014. http://d-nb.info/1055907610/34.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "TNFRSF10A"

1

Gonçalves, João, Helena Soares, Norman L. Eberhardt, et al. "TNFRSF12A." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_101376.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Nedjai, Belinda, Niamh Quillinan, Robert J. Coughlan, et al. "Lessons from Anti-TNF Biologics: Infliximab Failure in a TRAPS Family with the T50M Mutation in TNFRSF1A." In Advances in Experimental Medicine and Biology. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-6612-4_43.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

"TNFRSF12A." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_103881.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

"TNFRSF13a." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_103882.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

"TNFRSF13b." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_103883.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

"TNFRSF13c." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_103884.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

"TNFRSF17." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_103885.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

"Tumor Necrosis Factor Receptor Superfamily Member 11B (TNFRSF11B)." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_104014.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Obici, L., S. Marciano, G. Palladini, et al. "TNFRSF1A Mutations in Italian Patients Affected by Apparently Sporadic Periodic Fever Syndrome." In Amyloid and Amyloidosis. CRC Press, 2004. http://dx.doi.org/10.1201/9781420037494-64.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Palladini, G., L. Obici, G. Merlini, et al. "TNFRSF1A Mutations in Italian Patients Affected by Apparently Sporadic Periodic Fever Syndrome." In Amyloid and Amyloidosis. CRC Press, 2004. http://dx.doi.org/10.1201/9781420037494.ch61.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "TNFRSF10A"

1

Sun, Donglei, Hongyan Gou, Dandan Wang, and Jun Yu. "IDDF2021-ABS-0076 lncRNA TNFRSF10A-AS1 mediates tumorigenesis of gastric cancer by directly binding to MPZL1 and associates with patient outcomes." In Abstracts of the International Digestive Disease Forum (IDDF), Hong Kong, 4–5 September 2021. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2021. http://dx.doi.org/10.1136/gutjnl-2021-iddf.32.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Abo, Ryan P., Marina Parry, Sushila H. Rigas, Angela Cox, and Nicola J. Camp. "Abstract 2844: Association of genetic variants in TNFRSF10B and breast cancer." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2844.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Schabath, Matthew B., Anna R. Giuliano, Zachary Thompson, et al. "Abstract 4506: TNFRSF10B polymorphisms and haplotypes predicts survival in non-small cell lung cancer patients." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-4506.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Dhruv, Harshil D., Serdar Tuncali, Jean Kloss, et al. "Abstract 2621: TNFRSF19 (TROY) promotes glioma cell survival signaling and therapeutic resistance." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-2621.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Salloum, Darin. "Abstract PR03: HVEM (TNFRSF14) tumor suppressor in immune therapies of follicular lymphoma." In Abstracts: Second CRI-CIMT-EATI-AACR International Cancer Immunotherapy Conference: Translating Science into Survival; September 25-28, 2016; New York, NY. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/2326-6066.imm2016-pr03.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Tejera, Paula, Lin Li, Yang Zhao, et al. "POPDC3 And TNFRSF11A Genetic Variants Are Associated With The Development Of Acute Respiratory Distress Syndrome From Pulmonary Sources." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4913.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Dutton-Regester, Ken, and Nicholas K. Hayward. "Abstract 4226: TNFRSF14 is a cell surface marker of MITF expression in melanoma." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4226.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Tchatchou, Sandrine, Lucine Bosnoyan-Collins, Dushanthi Pinnaduwage, Shelley B. Bull, and Irene L. Andrulis. "Abstract B110: Characterization of the role of TRAF2 and TNFRSF14 in breast cancer." In Abstracts: AACR Special Conference on Advances in Breast Cancer Research: Genetics, Biology, and Clinical Applications - October 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1557-3125.advbc-b110.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Dutton-Regester, Ken, and Nicholas K. Hayward. "Abstract 4226: TNFRSF14 is a cell surface marker of MITF expression in melanoma." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4226.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Bonella, Francesco, Hung Tran, Dirk Theegarten, Josune Guzman, and Ulrich Costabel. "Determination of a single nucleotide polymorphism (SNP) of the TNFalpha-R1 region (TNFRSF1A) in patients with lung sarcoidosis: Preliminary results." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa3321.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!