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Journal articles on the topic 'Toxicosis'

Author: Grafiati

Published: 28 May 2022

Last updated: 31 July 2025

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1

Amissah, Richard Quansah, Nadine A. Vogt, Chuyun Chen, Karolina Urban, and Jibran Khokhar. "Prevalence and characteristics of cannabis-induced toxicoses in pets: Results from a survey of veterinarians in North America." PLOS ONE 17, no. 4 (2022): e0261909. http://dx.doi.org/10.1371/journal.pone.0261909.

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Cannabis legalization in North America has coincided with an increase in reports of cannabis-induced toxicosis in pets, but the magnitude of this problem, as well as outcomes of these incidents remain unknown. Therefore, we examined the frequency, diagnostic criteria, clinical signs, and prognoses of cannabis toxicoses in pets in North America. We conducted an online survey between January, 2021 and April, 2021 targeting veterinarians practicing in Canada and the United States (US). Out of the 251 study participants, 191 practiced in Canada. Cannabis toxicosis was most commonly reported in dog

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2

Edmondson, A. J., B. B. Norman, and D. Suther. "Survey of state veterinarians and state veterinary diagnostic laboratories for selenium deficiency and toxicosis in animals." Journal of the American Veterinary Medical Association 202, no. 6 (1993): 865–72. http://dx.doi.org/10.2460/javma.1993.202.06.865.

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Summary: Surveys were performed in which the state veterinarian and the major state veterinary diagnostic laboratories in every state were asked about selenium (Se) deficiency and toxicosis in livestock and wildlife in their state. Selenium-deficiency diseases were diagnosed in 46 states and were reported to be an important livestock problem in regions of 37 states; deficiencies were diagnosed in wildlife in 10 states. Natural Se toxicosis was a rare problem, with only 7 states reporting naturally developing Se toxicosis. Oversupplementation with Se was reported as a cause of toxicosis in 15 s

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3

Villar, David, David Gomez-Beltrán, Scott Radke, Kent Schwartz, and Drew Magstadt. "Case report: Diagnostic findings of bovine neurological cases at the Iowa State University Veterinary Diagnostic Laboratory between January 2017 and May 2021." Bovine Practitioner 57, no. 1 (2023): 1–9. http://dx.doi.org/10.21423/bovine-vol57no1p1-9.

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This study reviews the Iowa State University Veterinary Diagnostic Laboratory (ISU-VDL) computer records of bovine neurological cases submitted from January 2017 to May 2021. There were 236 cases diagnosed as follows: listeriosis (83), polioencephalomalacia (44), thrombomeningoencephalitis (23), miscellaneous bacteria (22), lead toxicosis (21), nitrate toxicosis (18), urea toxicosis (9), nervous coccidiosis (5), hypomagnesemia (5), sodium toxicosis (3), rabies (1), herpesvirus type 6 (1), and epsilon toxin from Clostridium perfringens type D (1). Most histories did not provide specific descrip

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4

Fayyaz, Talha Bin, Zahida Memon, Mirza Tasawer Baig, Noor ul Ain, and Muhammad Saddam Aziz. "HEMATOLOGIC TOXICOSIS;." Professional Medical Journal 24, no. 05 (2017): 751–56. http://dx.doi.org/10.29309/tpmj/2017.24.05.1296.

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Objectives: To evaluate the protective effects of combination of extracts of Oleaeuropeae and Ginkgo biloba in response to hematological toxicosis induced by DOX. Designof study: Animal experimental study. Setting: Dow University of Health Sciences. Period: 2month. Materials & methods: The extracts of Olea europeae and Ginkgo biloba were preparedby using standard Soxhlet apparatus and then they were mixed together in 1:1 ratio. EighteenMale Wistar rats were incorporated in the study and divided in 3 groups: T* (control group: 2ml 0.9 % NS), TP0 (DOX group: 15 mg/kg/week) and TP1 (extract o

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5

Jones, Lynn A., and Sandra E. Morgan. "Gossypol Toxicosis." American Journal of Veterinary Research 49, no. 8 (1988): 1435. https://doi.org/10.2460/ajvr.1988.49.08.1435.

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6

Bates, Nicola. "Ibuprofen toxicosis." Companion Animal 21, no. 6 (2016): 346–50. http://dx.doi.org/10.12968/coan.2016.21.6.346.

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7

Blodgett, Dennis J. "Fescue Toxicosis." Veterinary Clinics of North America: Equine Practice 17, no. 3 (2001): 567–77. http://dx.doi.org/10.1016/s0749-0739(17)30052-4.

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8

Arrington, Kathryn A., Alfred M. Legendre, Gretchen Simpson Tabeling, and Donita L. Frazier. "Comparison of body surface area-based and weight-based dosage protocols for doxorubicin administration in dogs." American Journal of Veterinary Research 55, no. 11 (1994): 1587–92. http://dx.doi.org/10.2460/ajvr.1994.55.11.1587.

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Summary Pharmacokinetics and toxicity of a single dose of doxorubicin, at dosages of 30 mg/m2 of body surface area and 1 mg/kg of body weight, were compared in 17 dogs. Effects of doxorubicin on complete blood cell count, platelet count, and the dogs' clinical condition were evaluated for 14 days. Cluster analysis, on the basis of clinical signs of doxorubicin toxicosis at the 30- mg/m2 dosage, revealed that 6 of 7 small dogs (≤ 10 kg) became ill, whereas 7 of 10 large dogs (> 10 kg) remained clinically normal. Small dogs that received doxorubicin at a dosage of 30 mg/m2 had higher peak pla

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9

Singh, Shailendra Kumar, Rina Singh, Sonam Bedi, Arun Kumar Pandey, Alankar Tiwari, and Pradeep Kumar Rai. "Prevalence of T3 thyrotoxicosis and it’s management in new onset thyrotoxicosis patients: a prospective observational study." International Journal of Advances in Medicine 12, no. 1 (2024): 39–43. https://doi.org/10.18203/2349-3933.ijam20243812.

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Background: T3 toxicosis is a subtype of thyrotoxicosis where total triiodothyronine (TT3) level is high but total thyroxine (TT4) level is normal in presence of suppressed thyroid stimulating hormone (TSH). Accurate and rapid diagnosis is crucial in management of T3 toxicosis. Methods: In this prospective observational study from India, newly diagnosed thyrotoxicosis patients were enrolled. T3 toxicosis patients were diagnosed based on diagnostic criteria. T3 toxicosis patients were randomized into two arms, based on dose of carbimazole (CBZ) used for treatment. All patients are following up

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10

Bozynski, Chantelle C., Tim J. Evans, Young Kim Dae, et al. "Copper Toxicosis with Hemolysis and Hemoglobinuric Nephrosis in Three Adult Boer Goats." Journal of Veterinary Diagnostic Investigation 21, no. 3 (2009): 395–400. http://dx.doi.org/10.1177/104063870902100319.

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Acute and, particularly, chronic copper exposures, along with defects in hepatic copper metabolism, altered excretion of copper, and/or nutritional imbalances between copper and other trace elements, can lead to hepatic accumulation of copper and primary copper toxicosis. There is interspecies variation in susceptibility to copper toxicosis, with sheep being the species most likely to develop this condition. Adult dairy goats and Boer crosses are generally considered resistant to chronic copper toxicosis, especially the hemolytic stage of this disease. The current report is rather unusual in t

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11

Wolf, Jacob, Levi Hoffman, and Carl Southern. "Successful Management of Severe Manganese Toxicosis in Two Dogs." Journal of the American Animal Hospital Association 59, no. 3 (2023): 152–57. http://dx.doi.org/10.5326/jaaha-ms-7348.

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ABSTRACT Manganese is a common component of human joint supplements and may be a source of ingestion and subsequent toxicosis in dogs. Although hepatotoxicity secondary to manganese toxicosis has been reported in dogs before, no descriptions of successful management of manganese toxicosis has been reported in veterinary literature. A 5 yr old spayed female Shetland sheepdog and a 5 yr old female Shetland sheepdog were evaluated following accidental ingestion of a joint supplement. Consultation with a toxicologist revealed concern for manganese toxicosis resulting in hepatic injury. Both dogs d

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12

Stegelmeier, B. L., D. R. Gardner, L. F. James, K. E. Panter, and R. J. Molyneux. "The Toxic and Abortifacient Effects of Ponderosa Pine." Veterinary Pathology 33, no. 1 (1996): 22–28. http://dx.doi.org/10.1177/030098589603300103.

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Ponderosa pine needles cause abortion and a poorly described toxicosis when eaten by cattle. In previous trials, the abortifacient compound of pine needles was identified as isocupressic acid. At abortifacient doses, isocupressic acid caused no other toxicosis. However, other pine needle fractions, similar in composition to several commercially available rosin products, caused no abortion but were very toxic. The purpose of this study was to describe the toxicoses of ponderosa pine, compare its toxicity with other rosin and related pine products, and identify the toxin. Four groups of three pr

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13

Rumbeiha, Wilson K., W. Emmett Braselton, Raymond F. Nachreiner, and Kent R. Refsal. "The Postmortem Diagnosis of Cholecalciferol Toxicosis: A Novel Approach and Differentiation from Ethylene Glycol Toxicosis." Journal of Veterinary Diagnostic Investigation 12, no. 5 (2000): 426–32. http://dx.doi.org/10.1177/104063870001200506.

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The objectives of this study were to develop a novel approach to postmortem diagnosis of cholecalciferol (CCF) toxicosis in dogs using kidney, bile, and urine samples, and to differentiate CCF from ethylene glycol (EG) toxicosis. To achieve these objectives, specimens collected from 2 previous laboratory studies in which dogs were given a single oral toxic dose of CCF (8.0 mg/kg) were used. For EG toxicosis, historical data from the previous 13 years (1985–1998) were reviewed and confirmed cases of EG toxicosis were selected. The historical data were used to compare trace mineral concentration

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14

Paul, A. J., W. J. Tranquilli, R. L. Seward, K. S. Todd, and J. A. DiPietro. "Clinical observations in Collies given ivermectin orally." American Journal of Veterinary Research 48, no. 4 (1987): 684–85. https://doi.org/10.2460/ajvr.1987.48.04.684.

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SUMMARY An oral liquid form of ivermectin was administered to 14 purebred Collies (12 rough coated, 2 smooth coated). All Collies were given ivermectin at dosages of 100 and then 200 μg/kg of body weight. Three of the dogs developed mild clinical signs of toxicosis (salivation, vomiting, confusion, ataxia, and tremors) with the 100 μg/kg dosage. After the 200 μg/kg dosage, 7 dogs (including 1 smooth-coated Collie) developed severe toxicosis (seizurelike activity, recumbency, nonresponsiveness, and coma). Because dogs that developed severe toxicosis were not retreated, only the 7 remaining dogs

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15

STERNLIEB, IRMIN. "Hepatic copper toxicosis." Journal of Gastroenterology and Hepatology 4, no. 2 (1989): 175–81. http://dx.doi.org/10.1111/j.1440-1746.1989.tb00823.x.

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16

Müller, T., W. Müller, and H. Feichtinger. "Idiopathic copper toxicosis." American Journal of Clinical Nutrition 67, no. 5 (1998): 1082S—1086S. http://dx.doi.org/10.1093/ajcn/67.5.1082s.

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17

Bates, Nicola. "Anticoagulant rodenticide toxicosis." Companion Animal 21, no. 8 (2016): 466–71. http://dx.doi.org/10.12968/coan.2016.21.8.466.

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18

Bates, Nicola. "Vitamin D toxicosis." Companion Animal 22, no. 12 (2017): 700–706. http://dx.doi.org/10.12968/coan.2017.22.12.700.

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19

Narchi, Hassib. "Idiopathic copper toxicosis." Journal of Pediatrics 137, no. 3 (2000): 440. http://dx.doi.org/10.1067/mpd.2000.106444.

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20

Neiger, Regg, Nicole Nelson, Dale Miskimins, Jim Caster, and Larry Caster. "Bovine Arsenic Toxicosis." Journal of Veterinary Diagnostic Investigation 16, no. 5 (2004): 436–38. http://dx.doi.org/10.1177/104063870401600512.

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21

Arisaka, Osamu, Atsuto Hosaka, and Keijiro Yabuta. "Free triiodothyronine toxicosis." Journal of Pediatrics 125, no. 2 (1994): 330–31. http://dx.doi.org/10.1016/s0022-3476(94)70224-1.

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22

Vela, B. Sylvia, and Richard I. Dorin. "Factitious triiodothyronine toxicosis." American Journal of Medicine 90, no. 1 (1991): 132–34. http://dx.doi.org/10.1016/0002-9343(91)90520-8.

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23

Johnson, Larry R. "Tricyclic Antidepressant Toxicosis." Veterinary Clinics of North America: Small Animal Practice 20, no. 2 (1990): 393–403. http://dx.doi.org/10.1016/s0195-5616(90)50034-0.

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24

Sirajli, Ulviyya, Mahbuba Azizova, Farida Gurbanova, Gubakhanim Hajizade, and Gulnar Babayeva. "TOXICOSIS OF PREGNANT." SCIENTIFIC WORK 17, no. 4 (2023): 289–93. http://dx.doi.org/10.36719/2663-4619/89/289-293.

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25

Huang, Yan, Jing Ge, Yongjie Wang, Sarah Shelby, Palika Morse та Ken Coffey. "283 Quercetin Antagonizes Tall Fescue Toxicosis-Induced Cardiovascular Toxicity in Lamb (Ovis Aries) via Modulating PGC-1α Mediated Mitochondrial Biogenesis". Journal of Animal Science 100, Supplement_3 (2022): 131–35. http://dx.doi.org/10.1093/jas/skac247.250.

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Abstract Quercetin is a natural flavonoid with a higher antioxidant capacity and anti-inflammatory. Tall fescue [Schedonorus arundinaceus (Schreb.) Dumort; E+] is perennial cool-season grass for grazing livestock in North America. Fescue infected with the endophyte (Neotyphodium coenophialum) produces chemicals toxic to livestock. However, the impact of quercetin on alleviating fescue toxicosis has received only limited attention. The objective of this experiment was to investigate the mitigation effect of quercetin on fescue toxicosis-induced cardiotoxicity of lambs. A total number of 24 Dorp

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26

Ogilvie, Gregory K., Ralph C. Richardson, C. R. Curtis, et al. "Acute and short-term toxicoses associated with the administration of doxorubicin to dogs with malignant tumors." Journal of the American Veterinary Medical Association 195, no. 11 (1989): 1584–87. https://doi.org/10.2460/javma.1989.195.11.1584.

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Summary One hundred eighty-five dogs with histologically confirmed, measurable malignant tumors were used in a study to determine the toxicity of the anthracycline antitumor antibiotic, doxorubicin, which was administered once or twice (at a 21-day interval) at the rate of 30 mg/m2 of body surface area, iv. During this study, 7 dogs died as a direct result of doxorubicin-induced toxicosis and 16 died as a direct result of the malignant neoplastic disease. Each dog was evaluated for signs of toxicosis for 3 weeks after the last dose was administered (15 dogs received 1 dose, 170 dogs received 2

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27

Ogilvie, Gregory K., Antony S. Moore, Joyce E. Obradovich, et al. "Toxicoses and efficacy associated with administration of mitoxantrone to cats with malignant tumors." Journal of the American Veterinary Medical Association 202, no. 11 (1993): 1839–44. http://dx.doi.org/10.2460/javma.1993.202.11.1839.

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Summary: Eighty-seven cats with histologically confirmed malignant tumors were used in a prospective study to determine the toxicity of mitoxantrone, a dihydroxyquinone derivative of anthracene, which was administered at 21-day intervals at dosages ranging from 2.5 to 6.5 mg/m2 of body surface, iv. Eleven of these cats were treated concurrently with radiation but were evaluated separately. Each cat was evaluated for signs of toxicosis for 3 weeks after each dose was administered or until the cat developed progressive disease, or until the cat's quality of life diminished to an unacceptable lev

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28

Ogilvie, Gregory K., Joyce E. Obradovich, Robyn E. Elmslie, et al. "Toxicoses associated with administration of mitoxantrone to dogs with malignant tumors." Journal of the American Veterinary Medical Association 198, no. 9 (1991): 1613–17. http://dx.doi.org/10.2460/javma.1991.198.09.1613.

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Summary One hundred twenty-nine dogs with histologically confirmed malignant tumors were used in a prospective study to determine the toxicity of the new dihydroxyquinone derivative of anthracene, mitoxantrone, which was administered iv at 21-day intervals at dosages ranging from 2.5 to 5 mg/m2 body surface area. Each dog was evaluated for signs of toxicosis for 3 weeks after each dose was administered or until the dog died, whichever came first. The number of dogs in each evaluation period were as follows: 1 dose (n = 129), 2 doses (n = 82), 3 doses (n = 43), 4 doses (n = 26), 5 doses (n = 19

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29

Blythe, L. L., A. M. Craig, C. Estill, and C. Cebra. "Clinical manifestations of tall fescue (Festuca arundinacea) and perennial ryegrass (lolium perenne) toxicosis in Oregon and Japan." NZGA: Research and Practice Series 13 (January 1, 2007): 369–72. http://dx.doi.org/10.33584/rps.13.2006.3170.

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There are multiple vehicles for endophyte toxicosis in animals including exposure from pasture, straw residues and seed screenings. This report discusses the clinical cases typically seen with tall fescue and perennial ryegrass toxicosis in Oregon and Japan. Case I involves a herd of 330 Black Angus cattle. Before the March calving season the owner wished to increase the protein content of the feed ration by feeding pellets made of seed screenings and grass hay. Forty two animals were lost to tall fescue toxicosis and dry gangrene of the feet and legs. Case II involves 1300 beef cows in Easter

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30

Thatcher, C. D., J. B. Meldrum, S. E. Wikse, and W. D. Whittier. "Arsenic toxicosis and suspected chromium toxicosis in a herd of cattle." Journal of the American Veterinary Medical Association 187, no. 2 (1985): 179–82. https://doi.org/10.2460/javma.1985.187.02.179.

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31

Hyun, Changbaig, and Lucio J. Filippich. "Inherited copper toxicosis with emphasis on copper toxicosis in Bedlington terriers." Journal of Experimental Animal Science 43, no. 1 (2004): 39–64. http://dx.doi.org/10.1016/j.jeas.2004.01.003.

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32

Tater, Kathy C., Sharon Gwaltney-Brant, and Tina Wismer. "Topical Minoxidil Exposures and Toxicoses in Dogs and Cats: 211 Cases (2001–2019)." Journal of the American Animal Hospital Association 57, no. 5 (2021): 225–31. http://dx.doi.org/10.5326/jaaha-ms-7154.

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ABSTRACT Topical minoxidil is a medication for hair loss, initially available in the United States by prescription only and available since 1996 as an over-the-counter product. To determine the epidemiology of minoxidil exposures and toxicoses in dogs and cats, 211 dog and cat cases with topical minoxidil exposure were identified from the American Society for the Prevention of Cruelty to Animals Animal Poison Control Center database. In 87 cases with clinical signs of toxicosis (62 cats, 25 dogs), case narratives were reviewed and coded for exposure-related circumstances. Unintentional deliver

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33

Andrae, J. G., and C. A. Roberts. "Transferring endophyte technology to North American farmers." NZGA: Research and Practice Series 13 (January 1, 2007): 233–36. http://dx.doi.org/10.33584/rps.13.2006.3066.

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Bacon and others discovered the tall fescue endophyte in 1977. Since that time, North American researchers have developed several endophyte-based technologies to reduce fescue toxicosis. The most recent has been the release of MaxQ, a novel endophyte that enhances persistence but does not cause fescue toxicosis. Despite its advantages, MaxQ has been adopted slowly across the fescue belt of the US. This paper presents possible social and managerial reasons for this slow adoption. It closes by offering some possible solutions. Keywords: MaxQ, novel endophyte, tall fescue toxicosis, pasture renov

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34

Poole, Rebecca K., and Daniel H. Poole. "Impact of Ergot Alkaloids on Female Reproduction in Domestic Livestock Species." Toxins 11, no. 6 (2019): 364. http://dx.doi.org/10.3390/toxins11060364.

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Fescue toxicosis is a multifaceted syndrome that elicits many negative effects on livestock consuming ergot alkaloids produced by endophyte-infected tall fescue. The economic losses associated with fescue toxicosis are primarily due to reproductive failure including altered cyclicity, suppressed hormone secretion, reduced pregnancy rates, agalactia, and reduced offspring birth weights. For decades, a multitude of research has investigated the physiological and cellular mechanisms of these reproductive failures associated with fescue toxicosis. This review will summarize the various effects of

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35

Fazekas, Bela, Eva Ivanics, Istvan Hajtos, and Robert Glavits. "Diazinon Toxicosis in Geese." Open Toxinology Journal 1, no. 1 (2008): 5–8. http://dx.doi.org/10.2174/1875414700801010005.

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36

Aleman, Monica, K. Gary Magdesian, Tracy S. Peterson, and Francis D. Galey. "Salinomycin toxicosis in horses." Journal of the American Veterinary Medical Association 230, no. 12 (2007): 1822–26. http://dx.doi.org/10.2460/javma.230.12.1822.

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37

Bates, Nicola. "Molluscicide toxicosis in dogs." Companion Animal 27, no. 5 (2022): 1–8. http://dx.doi.org/10.12968/coan.2021.0080.

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Slug and snails are important garden pests and intermediate hosts of lungworm, and various products are available for their control. Metaldehyde has long been used in molluscicide products and poisoning in dogs is common, having rapid-onset and prolonged seizure activity. However, these products are to be banned for outdoor use in the UK in spring 2022. This should eventually reduce the incidence of metaldehyde poisoning in pets. Methiocarb, a carbamate pesticide, was also used in slug baits but was banned in 2015. Cases still occasionally occur, characterised by rapid-onset gastrointestinal,

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38

Bates, Nicola, Zoe Tizzard, and Nick Edwards. "Hops toxicosis in dogs." Journal of Veterinary Emergency and Critical Care 32, no. 2 (2021): 274. http://dx.doi.org/10.1111/vec.13161.

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39

Linnett, P.-J. "Permethrin toxicosis in cats." Australian Veterinary Journal 86, no. 1-2 (2008): 32–35. http://dx.doi.org/10.1111/j.1751-0813.2007.00198.x.

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40

Gunn, G., A. Rafferty, G. Rafferty, et al. "Additional algal toxicosis hazard." Veterinary Record 129, no. 17 (1991): 391. http://dx.doi.org/10.1136/vr.129.17.391-b.

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Bates, Nicola. "Chocolate toxicosis in pets." Veterinary Nurse 15, no. 8 (2024): 320–25. http://dx.doi.org/10.12968/vetn.2024.0046.

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Chocolate ingestion is very common in pets, particularly dogs, but has also been reported in other animals including cats and rabbits. Most cases occur around Christmas and Easter, when large amounts of chocolate are often present in the home in the form of chocolate bars, snacks, confectionery, Easter eggs and chocolate figurines. Chocolate-covered raisins, nuts and coffee beans present an additional toxicological hazard in dogs. The type of chocolate is defined in law by the percentage of cocoa solids, with dark (plain) chocolate containing more than milk chocolate. The main toxic component

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42

Lloyd, W. Eugene, Yoshimitsu Maede, and Mutsumi Inaba. "Methionine toxicosis in cats." American Journal of Veterinary Research 48, no. 7 (1987): 1172. https://doi.org/10.2460/ajvr.1987.48.07.1172.

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Maede, Yoshimitsu, Takuo Hoshino, Mutsumi Inaba, and Shigeo Namioka. "Methionine toxicosis in cats." American Journal of Veterinary Research 48, no. 2 (1987): 289–92. https://doi.org/10.2460/ajvr.1987.48.02.289.

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SUMMARY Cats given dl-methionine (1 g/kg of body weight/day) developed severe hemolytic anemia with marked increase of methemoglobin (MetHb) concentration and Heinz-body formation at treatment-day 6 to 10. Cats fed 0.5 g of me-thionine/kg for 52 days had a moderate Heinz-body hemolytic anemia with methemoglobinemia at treatment days 17 to 31, but thereafter recovered from the anemia despite continuation of methionine feeding, indicating an adaptation of the cats. In vitro, significant (P < 0.01) increases of MetHb concentration and Heinz-body formation were observed when rbc were incubated

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Casteel, Stan W., Gary D. Osweiler, William O. Cook, George Daniels, and Robert Kadlec. "Selenium toxicosis in swine." Journal of the American Veterinary Medical Association 186, no. 10 (1985): 1084–85. https://doi.org/10.2460/javma.1985.186.10.1084.

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SUMMARY Selenium toxicosis was diagnosed as the cause of fatal paralytic disease in a group of feeder pigs. Lumbar poliomyelomalacia and coronary band necrosis were the principal lesions. High selenium concentrations were detected in liver and kidney. Excessive selenium was traced to the premix added to the complete ration.

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45

Wiwanitkit, Viroj. "Lead toxicosis: some concerns." Veterinary Clinical Pathology 39, no. 1 (2010): 3. http://dx.doi.org/10.1111/j.1939-165x.2010.00218.x.

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46

Schmitz, D. G. "Cantharidin Toxicosis in Horses." Journal of Veterinary Internal Medicine 3, no. 4 (1989): 208–15. http://dx.doi.org/10.1111/j.1939-1676.1989.tb00859.x.

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47

Bates, Nicola. "Zinc toxicosis in dogs." Companion Animal 24, no. 2 (2019): 102–7. http://dx.doi.org/10.12968/coan.2019.24.2.102.

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48

Bates, Nicola. "Xylitol toxicosis in dogs." Companion Animal 24, no. 4 (2019): 182–87. http://dx.doi.org/10.12968/coan.2019.24.4.182.

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49

Plumlee, Konnie H. "Toxicosis from agricultural chemicals." Clinical Techniques in Equine Practice 1, no. 2 (2002): 94–97. http://dx.doi.org/10.1053/ctep.2002.34238.

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Casteel, Stan W. "Metal Toxicosis in Horses." Veterinary Clinics of North America: Equine Practice 17, no. 3 (2001): 517–27. http://dx.doi.org/10.1016/s0749-0739(17)30049-4.

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