Relevant bibliographies by topics / TP53 polymorphism

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers
  5. Reports

Academic literature on the topic 'TP53 polymorphism'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "TP53 polymorphism"

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Dabre, Soayebo, Abdou Azaque Zoure, Touwendpoulimdé Isabelle Kiendrébéogo, et al. "Involvement of p.R72P and PIN3 Ins16bp (TP53) Polymorphisms and the I157T (CHEK2) Mutation in Breast Cancer Occurrence in Burkina Faso." Asian Pacific Journal of Cancer Biology 8, no. 2 (2023): 135–45. http://dx.doi.org/10.31557/apjcb.2023.8.2.135-145.

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Introduction: The TP53 and CHEK2 genes have been described as breast cancer susceptibility genes and some of their polymorphisms have been associated with an increased risk of breast cancer in certain populations.Aim: The objective of this study was to investigate the p.R72P and PIN3 Ins16bp (TP53) polymorphisms and the I157T (CHEK2) mutation developping of breast cancer. Methods: This case-control study had enrolled 144 participants including 65 cases (breast cancer patients) and 79 controls (women without breast abnormalities) in the city of Ouagadougou in Burkina Faso. The DNA was extracted
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Nishizuka, Satoshi, Yukimi Ohmori, Takeshi Iwaya, and Keisuke Koeda. "Recurrence risk evaluation in stage IB gastric cancer with TP53 codon 72 polymorphism." Journal of Clinical Oncology 37, no. 15_suppl (2019): 4044. http://dx.doi.org/10.1200/jco.2019.37.15_suppl.4044.

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4044 Background: Post-operative adjuvant chemotherapy is not currently indicated for Stage IB gastric cancer. However, about 10% of these patients experience recurrence and metastasis. Our previous study on a panel of gastric cancer cell lines indicated that TP53 codon 72 polymorphisms may affect the degree of biological malignancy. Hence, we hypothesized that the TP53 codon 72 polymorphisms may have been associated with post-operative survival without adjuvant chemotherapy. In this study, we investigated the risk of recurrence after treatment of Stage IB gastric cancer patients carrying the T
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Diakite, Brehima, Yaya Kassogue, Mamoudou Maiga, et al. "Abstract 7326: TP53 p.Pro47Ser polymorphism in breast cancer susceptibility in Mali." Cancer Research 84, no. 6_Supplement (2024): 7326. http://dx.doi.org/10.1158/1538-7445.am2024-7326.

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Abstract Background: The aim of this study was to assess the potential association of p.Pro47Ser polymorphisms of TP53 with the risk of developing breast cancer in the Malian population. Methods: We analyzed DNA samples from 72 breast cancer patients and 121 unrelated healthy, age-matched individuals to examine the presence of the polymorphisms. The genotyping of TP53 polymorphism was performed using PCR-RFLP and HRM (high-resolution melting) methods. Results: The genotypic frequencies of PP, PS, and SS were found to be 63.9% PP, 30.6% PS, and 5.6% SS in breast cancer patients, compared to 70.
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Liao, Fan, Li Yuan, Jinhong Zhu, Wei Chen, Yemu Zhao, and Jing He. "Association of TP53 rs1042522 C>G Polymorphism with Glioma Risk in Chinese Children." BioMed Research International 2022 (August 13, 2022): 1–6. http://dx.doi.org/10.1155/2022/2712808.

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Glioma is the most common intracranial malignancy. TP53 is a crucial tumor suppressor gene that plays an essential regulatory role in cell growth, apoptosis, and DNA repair. The TP53 rs1042522 C>G polymorphism has been reported to be strongly associated with various tumor risks. To assess the TP53 rs1042522 C>G polymorphism with glioma risk in Chinese children, we determined the genotypes of the TP53 rs1042522 C>G polymorphism in 171 glioma patients and 228 cancer-free controls by Taqman assay. We assessed the association of the polymorphism with glioma risk using odds ratio (OR) and
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Moe, Svein Erik, Fredrik A. Erland, Siren Fromreide, et al. "The TP53 Codon 72 Arginine Polymorphism Is Found with Increased TP53 Somatic Mutations in HPV(−) and in an Increased Percentage among HPV(+) Norwegian HNSCC Patients." Biomedicines 11, no. 7 (2023): 1838. http://dx.doi.org/10.3390/biomedicines11071838.

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Background: Somatic TP53 mutations are frequent in head and neck squamous cell carcinoma (HNSCC) and are important pathogenic factors. Objective: To study TP53 mutations relative to the presence of human papillomavirus (HPV) in tumors in HNSCC patients. Methods: Using a custom-made next-generation sequencing (NGS) panel on formalin-fixed, paraffin-embedded tumor tissue, we analyzed somatic TP53 mutations and the TP53 single-nucleotide polymorphism (SNP) codon 72 (P72R; rs1042522) (proline → arginine) from 104 patients with HNSCC. Results: Only 2 of 44 patients with HPV-positive (HPV(+)) HNSCC
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Deben, Christophe, Jolien Van den Bossche, Nele Van Der Steen, et al. "Deep sequencing of the TP53 gene reveals a potential risk allele for non–small cell lung cancer and supports the negative prognostic value of TP53 variants." Tumor Biology 39, no. 2 (2017): 101042831769432. http://dx.doi.org/10.1177/1010428317694327.

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The TP53 gene remains the most frequently altered gene in human cancer, of which variants are associated with cancer risk, therapy resistance, and poor prognosis in several tumor types. To determine the true prognostic value of TP53 variants in non–small cell lung cancer, this study conducted further research, particularly focusing on subtype and tumor stage. Therefore, we determined the TP53 status of 97 non–small cell lung cancer adenocarcinoma patients using next generation deep sequencing technology and defined the prognostic value of frequently occurring single nucleotide polymorphisms an
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Iordanishvili, Saba, Tornike Metreveli, Elene Lipartia, et al. "The HPV-TP53-MALAT1 Axis: Unravelling interactions in cervical cancer development." PLOS ONE 18, no. 10 (2023): e0291725. http://dx.doi.org/10.1371/journal.pone.0291725.

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Introduction Cervical cancer, primarily driven by Human Papillomavirus (HPV) infection, stands as a substantial global health challenge. The TP53 gene’s, Arg72Pro polymorphism has emerged as a noteworthy player in cervical cancer development, particularly among individuals harboring high-risk (HR) HPV types. Additionally, long non-coding RNAs (lncRNAs), exemplified by metastasis-associated lung adenocarcinoma transcript 1 (MALAT1), exert critical roles in cancer biology. This study delves into unravelling the intricate connections linking HPV infection, TP53 Arg72Pro polymorphism, and MALAT1 e
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Usategui-Martín, Ricardo, Nadia Galindo-Cabello, Salvador Pastor-Idoate, et al. "A Missense Variant in TP53 Could Be a Genetic Biomarker Associated with Bone Tissue Alterations." International Journal of Molecular Sciences 25, no. 3 (2024): 1395. http://dx.doi.org/10.3390/ijms25031395.

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Metabolic bone diseases cover a broad spectrum of disorders that share alterations in bone metabolism that lead to a defective skeleton, which is associated with increasing morbidity, disability, and mortality. There is a close connection between the etiology of metabolic bone diseases and genetic factors, with TP53 being one of the genes associated therewith. The single nucleotide polymorphism (SNP) Arg72Pro of TP53 is a genetic factor associated with several pathologies, including cancer, stroke, and osteoporosis. Here, we aim to analyze the influence of the TP53 Arg72Pro SNP on bone mass in
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F. Fahmy, Nahed, Nagwa S. Ahmed, Ghada Galal, et al. "Study the Role of Tp53 in the Development of Hepatocellular Carcinoma in HCV Infected Patients." Egyptian Journal of Medical Microbiology EJMM29, no. 4 (2020): 165–72. http://dx.doi.org/10.51429/ejmm29421.

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Background: Hepatocellular carcinoma (HCC) is considered as the primary malignancy of the liver that usually occurs on top of chronic viral hepatitis. The TP53 (tumor protein 53) is a tumor suppressor gene which is the key in tumor development and progression and the single nucleotide polymorphism (SNP) of the p53 gene codon 72 (p53Arg/Pro) changes the structure of the protein. Objectives: to determine the association of the TP53 Arg72Pro polymorphism with the risk of HCC development in Hepatitis C virus (HCV) infected Egyptian patients. Methodology: This is a case control study conducted in S
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Maiga, Mamoudou, Brehima Diakite, Yaya Kassogue, et al. "Abstract 7328: Relationship between PIN3 16bp duplication of TP53 and HPV in HIV women in Mali." Cancer Research 84, no. 6_Supplement (2024): 7328. http://dx.doi.org/10.1158/1538-7445.am2024-7328.

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Abstract Background: TP53 polymorphisms can influence the immune response to HPV, affecting TP53's role in cell cycle regulation and apoptosis, crucial for controlling HPV infection and preventing cancer. Objective: Explore the link between TP53 PIN3 16-bp duplication and HPV infection in HIV-positive Malian women. Methods: This study explored the link between PIN3 16-bp duplication polymorphism of TP53 and HPV infection in 50 HIV-positive women in Mali. Data included surveys, self-sampled vaginal swabs, and blood collection. HPV typing used GeneXpert, and PIN3 genotyping utilized AS-PCR. Resu
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Dissertations / Theses on the topic "TP53 polymorphism"

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Silva, Ana Manoela Maria da. "ANÁLISE DO POLIMORFISMO DOS GENES TP53 E CYP1A1m1 EM BIÓPSIA DE ENDOMÉTRIO DE PACIENTES COM ENDOMETRIOSE." Pontifícia Universidade Católica de Goiás, 2012. http://localhost:8080/tede/handle/tede/2343.

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Made available in DSpace on 2016-08-10T10:38:32Z (GMT). No. of bitstreams: 1 ANA MANOELA MARIA DA SILVA.pdf: 819143 bytes, checksum: e4c543c7bf8fe8063cf665a20af885e5 (MD5) Previous issue date: 2012-06-25<br>Endometriosis is a benign gynecological disease estrogen dependent that affects women at reproductive age. It is characterized by the presence of endometrial tissue outside the uterine cavity designated as ectopic endometrium, involving chronic inflammation in the pelvic cavity. The four stages of endometriosis are classified as: minimal (stage I), mild (stage II), moderat
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Almeida, Priscilla Silva Rosa de. "POLIMORFISMO DO GENE TP53 EM SARCOMAS DE PARTES MOLES NO ADULTO." Pontifícia Universidade Católica de Goiás, 2008. http://localhost:8080/tede/handle/tede/2412.

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Made available in DSpace on 2016-08-10T10:39:18Z (GMT). No. of bitstreams: 1 PRISCILLA SILVA ROSA DE ALMEIDA.pdf: 3720637 bytes, checksum: b39a9c071d90058d47a3a8c7aec2c7f3 (MD5) Previous issue date: 2008-07-21<br>Soft tissue sarcomas (STS) are tumors with mesodermical origin, comprising about 1% of all adult neoplasms. Because of its effect on the p53 protein coding sequence, and its association with an increased risk for some cancer types, TP53 codon 72 polymorphism has been investigated in several studies. TP53 codon 72 codes for either Arginine (p53Arg), or Proline (p53Pro) at the p53 pro
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Melo, Márcia Portela de. "Análise do polimorfismo R72P do gene TP53 em paciente com carcinoma de mama ductal invasor." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2008. http://hdl.handle.net/10183/13063.

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Introdução: O câncer de mama é a neoplasia mais freqüente e também a principal causa de morte por câncer entre as mulheres. O gene TP53 é polimórfico no códon 72 da proteína que ele codifica, podendo conter arginina (CGC) ou prolina (CCC) nesta posição. Este polimorfismo pode estar envolvido na suscetibilidade e predisposição ao câncer e apresenta uma distribuição étnica e geográfica bastante variável. O genótipo homozigoto para arginina parece ser um fator de risco e prognóstico significativo para o câncer de mama. Métodos: Extraído DNA a partir do sangue periférico de 76 pacientes consecutiv
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Cortezzi, Sylvia Sanches. "Papilomavírus humano e polimorfismo do gene TP53 no carcinoma espinocelular de cabeça e pescoço /." São José do Rio Preto : [s.n.], 2002. http://hdl.handle.net/11449/92507.

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Resumo: O carcinoma espinocelular de cabeça e pescoço é uma neoplasia cujo principal fator de risco é o consumo de tabaco e/ou álcool. As infecções pelo HPV têm sido observadas nesse grupo de tumores. Os HPVs de alto risco podem induzir alterações calulares pela interação de suas proteínas E6 e E7 com as proteínas p53 e a pRb, respectivamente. As oncoproteínas são insuficientes para o desenvolvimento do fenótipo maligno, sugerindo a presença de outros cofatores, como a susceptibilidade genética. O gene TP53 possui um polimorfismo que resulta na presença de uma prolina ou de uma arginina na pos
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Fagundes, Simone Souza. "RELAÇÃO DO POLIMORFISMO DO GENE TP53 NO CÓDON 72 COM CÂNCER DE MAMA: UMA ATUALIZAÇÃO DE METANÁLISE (2002-2015)." Pontifícia Universidade Católica de Goiás, 2016. http://tede2.pucgoias.edu.br:8080/handle/tede/3524.

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Submitted by admin tede (tede@pucgoias.edu.br) on 2016-10-07T13:26:52Z No. of bitstreams: 1 SIMONE SOUZA FAGUNDES.pdf: 1864838 bytes, checksum: 28db816101558781ede4b2fb42be7695 (MD5)<br>Made available in DSpace on 2016-10-07T13:26:52Z (GMT). No. of bitstreams: 1 SIMONE SOUZA FAGUNDES.pdf: 1864838 bytes, checksum: 28db816101558781ede4b2fb42be7695 (MD5) Previous issue date: 2016-06-27<br>Breast cancer is the most frequent in the world and Brazilian women, except for cases of skin cancer nonmelanoma. It is a complex disease that has no single cause, results from the interaction of multiple ri
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Cortezzi, Sylvia Sanches [UNESP]. "Papilomavírus humano e polimorfismo do gene TP53 no carcinoma espinocelular de cabeça e pescoço." Universidade Estadual Paulista (UNESP), 2002. http://hdl.handle.net/11449/92507.

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Made available in DSpace on 2014-06-11T19:26:04Z (GMT). No. of bitstreams: 0 Previous issue date: 2002-08-23Bitstream added on 2014-06-13T20:54:02Z : No. of bitstreams: 1 cortezzi_ss_me_sjrp.pdf: 512438 bytes, checksum: c98927f964472197d57903b9dbd21cb1 (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>O carcinoma espinocelular de cabeça e pescoço é uma neoplasia cujo principal fator de risco é o consumo de tabaco e/ou álcool. As infecções pelo HPV têm sido observadas nesse grupo de tumores. Os HPVs de alto risco podem induzir alterações calulares pela interação de sua
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Ribeiro, Eliane da Silva. "IMPORTÂNCIA DO HPV NO CARCINOMA DO COLO UTERINO E A ASSOCIAÇÃO COM O POLIMORFISMO R72P DO GENE TP53 E A RADIOTERAPIA." Pontifícia Universidade Católica de Goiás, 2012. http://localhost:8080/tede/handle/tede/2364.

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Made available in DSpace on 2016-08-10T10:38:42Z (GMT). No. of bitstreams: 1 ELIANE DA SILVA RIBEIRO.pdf: 1086550 bytes, checksum: fdf51565bf5bda0fc2d8aacfc69c4ef8 (MD5) Previous issue date: 2012-06-29<br>Persistent Human Papillomavirus (HPV) high risk, is the most important risk factor for development of cervical cancer. However, only high risk HPV types are associated with cancer. This ability carcinogenic due to interaction between HPV E6 protein with p53 protein, considered the main event of this process of tumor development. The present study evaluated the genotypes of HPV; Sequencing o
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Lima, Junior Mario Maciel de 1974. "Avaliação antropométrica, prostatica e polimorfismos de TP53 e GSTP1 em populações do estremo setentrional amazônico." [s.n.], 2012. http://repositorio.unicamp.br/jspui/handle/REPOSIP/310299.

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Orientadores: Laura Sterian Ward, Ubirajara Ferreira<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-21T18:25:14Z (GMT). No. of bitstreams: 1 LimaJunior_MarioMacielde_D.pdf: 2397268 bytes, checksum: 63d55e4a5572d5d7f06e615be4d98384 (MD5) Previous issue date: 2012<br>Resumo: As doenças da glândula prostática em geral e a incidência de câncer da próstata, em particular, mostram disparidades acentuadas entre os diferentes países e etnias. De fato, etnia, idade e história familiar são os mais fortes fatores de risco co
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Achatz, Maria Isabel Alves de Souza Waddington. "Modificadores de penetrância de mutações germinativas no gene TP53 em famílias brasileiras com diagnóstico clínico da síndrome de Li-Fraumeni e Li-Fraumeni like: impacto dos polimorfismos intragênicos do TP53 e de genes." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/5/5155/tde-29012009-172419/.

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A síndrome de Li-Fraumeni (LFS) e sua variante like (LFL) são associadas a mutações germinativas no gene TP53 e predispõe ao alto risco para múltiplos tumores em idade jovem. Analisamos 91 famílias LFS/LFL do sul/sudeste do Brasil para mutações germinativas e haplótipos de TP53 (PIN2, PIN3 e PEX4) e MDM2 (309T-G). A mutação R337H ocorreu em 44,4% das famílias avaliadas. Em 750 controles da região a freqüência populacional da mutação foi 0,3%. A genotipagem de oito indivíduos não relacionados R337H-positivos para 29 TAG SNPs intragênicos demonstrou o mesmo haplótipo raro estabelecendo efeito fu
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Portari, Elyzabeth Avvad. "Estudo de polimorfismos nos genes TP53 e p21(WAF1) e do perfil imunohistoquímico das proteínas p53, p21(WAF1), p16(INK4a) e ciclina D1 pela técnica de Tissue Microarray (TMA) e sua importância para o desenvolvimento e/ou severidade das neoplasias cervicais." Universidade do Estado do Rio de Janeiro, 2012. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=9228.

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O câncer de colo do útero é o terceiro tipo de câncer mais frequente em mulheres no mundo, e a infecção persistente pelo papilomavirus humano (HPV) oncogênico é condição necessária, mas não suficiente para seu desenvolvimento. As oncoproteínas virais E6 e E7 interferem direta ou indiretamente na ação de várias proteínas celulares. Entretanto, as variantes proteicas, resultantes de polimorfismos genéticos, podem apresentar comportamento distinto mediante a infecção pelo HPV. O objetivo deste estudo foi avaliar possíveis associações entre polimorfismos nos genes TP53 (p53 PIN3, p53 72C>G) e p21
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Book chapters on the topic "TP53 polymorphism"

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V., Evgeny, Nadezhda V., Nicolay V., et al. "TP53 Gene Polymorphisms in Cancer Risk: The Modulating Effect of Ageing, Ethnicity and TP53 Somatic Abnormalities." In Tumor Suppressor Genes. InTech, 2012. http://dx.doi.org/10.5772/27768.

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Conference papers on the topic "TP53 polymorphism"

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Santos, Igor Lopes dos, Nathalia Amaral Nogueira, Luciana Corrêa Amador, et al. "ANALYSIS OF THE R337H VARIANT IN THE TP53 GENE IN A GROUP OF PREMENOPAUSAL WOMEN WITH BREAST CANCER FROM THE CENTRALWESTERN REGION OF BRAZIL: A PILOT STUDY." In Brazilian Breast Cancer Symposium 2022. Mastology, 2022. http://dx.doi.org/10.29289/259453942022v32s2053.

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Objective: The aim of this study was to investigate the frequency of the R337H variant in the TP53 gene in a group of premenopausal women with breast cancer from the central-western region of Brazil and its possible associations with clinical, pathological, and prognostic aspects. Methods: The research comprised a pilot study of 36 patients with breast carcinomas diagnosed before the age of 50, selected from the records of the Laboratory of Immunohistochemistry, Department of Pathology, Hospital Araújo Jorge, in Goiânia (GO). DNA extraction was performed with QIAamp DNA FFPE Advanced (Qiagen,
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Rivera, Bianca L., Ricardo López, Roger Vázquez, Yarilis Castro, and Adriana Báez. "Abstract 1558: TP53 codon 72 polymorphism association with prognosis in Puerto Rican head and neck squamous cell carcinoma patients." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-1558.

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Falk, Ingrid Jakobsen, Kerstin Willander, Roza Chaireti, et al. "Abstract 3518: TP53 mutations and MDM2 single nucleotide polymorphism 309T-G predicts outcome and treatment resistance in acute myeloid leukemia." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-3518.

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Shiraishi, Kouya, Hideo Kunitoh, Chiharu Tanai, et al. "Abstract C109: Association of TP53 gene polymorphism with response to platinum-based doublet chemotherapy in non-small cell lung cancer patients." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Nov 12-16, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1535-7163.targ-11-c109.

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NOGAMI, P. Y., M. N. S. SILVA, I. B. COSTA, et al. "TP53 GENE PRO72ARG(RS1042522) SINGLE NUCLEOTIDE POLYMORPHISM IS ASSOCIATED WITH INCREASED RISK OF METASTASIS IN GASTRIC ADENOCARCINOMA IN THE AMAZON POPULATION." In ONCOLOGY 2023 International Symposium. Even3, 2025. https://doi.org/10.29327/oncology-2023-international-symposium.1084768.

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Samuel, Nardin, Ana Novokmet, Thomas J. Hudson, and David Malkin. "Abstract 16: Impact of germline TP53 mutations and polymorphisms in women with premenopausal breast cancer." In Abstracts: AACR Special Conference: Cancer Susceptibility and Cancer Susceptibility Syndromes; January 29-February 1, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.cansusc14-16.

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Jadhav, Trafina, Jesus Salazar-Gonzalez, Shantel Hebert-Magee, et al. "Abstract 2776: Codon 72 and Intron-3 polymorphisms in TP53 are risk factors for breast cancer." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-2776.

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Kuddus, Ruhul H., Asmahan A. El Ezzi, Mohammed A. El-Saidi, Scott Baker, and Wissam Zaidan. "Abstract 4821: Association of polymorphisms in TP53, CXCL2, MDM2, MDM4 and BCL2 genes and proliferative prostate diseases among Lebanese men." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4821.

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Souza, Cristabelle Madona N. de, Jeremy Chien, and Jill Madden. "Abstract B41: Studying the effect of germline polymorphisms on somatic hotspot mutations in TP53 for the treatment of high-grade serous ovarian carcinoma." In Abstracts: AACR Special Conference: Addressing Critical Questions in Ovarian Cancer Research and Treatment; October 1-4, 2017; Pittsburgh, PA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1557-3265.ovca17-b41.

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Reports on the topic "TP53 polymorphism"

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Jafrin, Sarah, Md Abdul Aziz, and Mohammad Safiqul Islam. Extended Investigation on the connection of TP73 G4C14-A4T14 Polymorphism with different Cancer Types – An Updated Meta-analysis with 56 Case-control Studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.1.0070.

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Review question / Objective: TP73 G4C14-A4T14 variant has been suspected of elevating the risk of cancer for many years. The available evidence was unsatisfactory and could not provide a reliable conclusion. Therefore, we performed this meta-analysis to re-evaluate the previous findings and illustrate the actual role of TP73 G4C14-A4T14 variant on cancer development. Condition being studied: The association of the G4C14-A4T14 variant with cancer risk was studied. Information sources: PubMed, Google Scholar, EMBASE, Cochrane Library, and Web of Science, CNKI.
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