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Journal articles on the topic 'Transciptomic analysis'

Author: Grafiati

Published: 4 June 2021

Last updated: 9 February 2022

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1

Kim, Kwang-Ho, and Dae-Weon Lee. "Transciptomic Analysis of Larval Fat Body of Plutella xylostella under Low Temperature." Korean Journal of Environmental Agriculture 38, no. 4 (2019): 296–306. http://dx.doi.org/10.5338/kjea.2019.38.4.40.

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Orekhov, A., Y. Oishi, N. Nikiforov, et al. "Transciptome analysis revealed inflammatory genes responsible for foam cell formation." Atherosclerosis 275 (August 2018): e116. http://dx.doi.org/10.1016/j.atherosclerosis.2018.06.329.

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3

Wiwanitkit, Somsri, and Viroj Wiwanitkit. "Nuclear-expressed sequence Tag (NEST) analysis of Zika virus nonstructural protein 1: Transciptomics approach." Annals of Tropical Medicine and Public Health 10, no. 4 (2017): 1081. http://dx.doi.org/10.4103/1755-6783.196696.

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4

Cui, Jun, Shikai Liu, Bing Zhang, et al. "Transciptome Analysis of the Gill and Swimbladder of Takifugu rubripes by RNA-Seq." PLoS ONE 9, no. 1 (2014): e85505. http://dx.doi.org/10.1371/journal.pone.0085505.

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Hotinski, Anya K., Karen M. Lower, and Bryone J. Kuss. "Somatic MDC1 Mutation in Putative Pre-Leukaemic Stem Cell of a Biclonal Case of Chronic Lymphocytic Leukaemia." Blood 132, Supplement 1 (2018): 5534. http://dx.doi.org/10.1182/blood-2018-99-115704.

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Abstract Chronic lymphocytic leukaemia (CLL) is an inherently heterogeneous disease in which founding lesions and cell of origin remain a subject of debate. This case of truly biclonal CLL provides evidence of the presence of a pre-leukaemic stem cell, supporting other lines of evidence that pathogenic genetic and epigenetic lesions in CLL arise early in haematopoiesis. An individual with trisomy 12 containing CLL, present at a frequency of 30%, was identified. Two distinct CD5/19-positive CLL clones were separated by flow cytometry, based on bimodal expression of CD49d. Each leukaemic clone w

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Babenko, Vladislav V., Rustam H. Ziganshin, Christoph Weise, et al. "Novel Bradykinin-Potentiating Peptides and Three-Finger Toxins from Viper Venom: Combined NGS Venom Gland Transcriptomics and Quantitative Venom Proteomics of the Azemiops feae Viper." Biomedicines 8, no. 8 (2020): 249. http://dx.doi.org/10.3390/biomedicines8080249.

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Feae’s viper Azemipos feae belongs to the Azemiopinae subfamily of the Viperidae family. The effects of Viperidae venoms are mostly coagulopathic with limited neurotoxicity manifested by phospholipases A2. From A. feae venom, we have earlier isolated azemiopsin, a novel neurotoxin inhibiting the nicotinic acetylcholine receptor. To characterize other A. feae toxins, we applied label-free quantitative proteomics, which revealed 120 unique proteins, the most abundant being serine proteinases and phospholipases A2. In total, toxins representing 14 families were identified, among which bradykinin-

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Branca, Enrica, Giovanna Carrà, Isabella Russo, Massimo Terzolo, Angelo Guerrasio, and Alessandro Morotti. "In-Silico Transcriptome Analyses of Hemostasis Triggers in Inflamed Vs Normal Mucosa of IBD Patients." Blood 136, Supplement 1 (2020): 19–20. http://dx.doi.org/10.1182/blood-2020-136706.

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Background:inflammatory bowel disease (IBD) is characterized by chronic inflammation associated with an increased tendency to thrombosis and thromboembolic complications. The underlying mechanisms of VTE are not yet fully understood. Several studies reported different expressions of circulating procoagulant factors or fibrinolysis inhibitors in IBD. Others linked podoplanin overexpression with thrombosis, hypercoagulability, and increased risk of VTE. Here, we aimed to identify, among genes able to trigger thrombosis, those aberrantly expressed in IBD samples as compared with matched normal mu

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Ribeiro, Edna, Mariana Delgadinho, and Miguel Brito. "Environmentally Relevant Concentrations of Bisphenol A Interact with Doxorubicin Transcriptional Effects in Human Cell Lines." Toxics 7, no. 3 (2019): 43. http://dx.doi.org/10.3390/toxics7030043.

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The worldwide production of synthetic chemicals, including endocrine disruptor chemicals (EDCs), such as Bisphenol A (BPA) has increased significantly in the last two decades. Human exposure to BPA, particularly through ingestion, is continuous and ubiquitous. Although, considered a weak environmental estrogen, BPA can induce divergent biological responses through several signaling pathways, including carcinogenesis in hormone-responsive organs. However, and despite the continuous increase of tumor cell-resistance to therapeutic drugs, such as doxorubicin (DOX), information regarding BPA drug

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9

de Jong, Madelon M. E., Zoltan Kellermayer, Natalie Papazian, et al. "Single Cell Transcriptomic Analysis of the Multiple Myeloma Bone Marrow Identifies a Unique Inflammatory Stromal Cell Population Associated with TNF Signaling." Blood 134, Supplement_1 (2019): 690. http://dx.doi.org/10.1182/blood-2019-123012.

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Background: In multiple myeloma, tumor cell survival, disease progression and therapy response are influenced by signals derived from the non-malignant bone marrow niche. This notwithstanding, a detailed in-vivo definition of the cells that define the multiple myeloma niche is lacking. Mesenchymal stromal cells are important niche constituents. Recent progress made with single cell transciptomics suggests that mesenchymal stromal cells are a dynamic population of cells that can exist as several subsets with functionally distinct activation and differentiation profiles. Aim: To identify mesench

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10

Visani, Giuseppe, Francesco Di Raimondo, Felicetto Ferrara, et al. "Low-Dose Lenalidomide Plus Low Dose Cytarabine Induce Complete Remission That Can Be Predicted By Genetic Profiling In Very Elderly Acute Myeloid Leukemia Patients." Blood 122, no. 21 (2013): 496. http://dx.doi.org/10.1182/blood.v122.21.496.496.

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Abstract Outcome for older patients with acute myeloid leukemia (AML) is extremely poor. Intensive induction chemotherapy is often unsuitable. In this phase II study we tested, for the first time, the efficacy of a novel combination therapy with low-dose lenalidomide plus low-dose cytarabine. Further, based on the hypothesis that genetic features might influence treatment response, we aimed at identifying a possible biomarker by studying the global gene expression profiles (GEP). We designed a prospective phase II study to assess the efficacy of the concomitant administration of low-dose lenal

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11

Park, Christopher Y., Wendy W. Pang, Elizabeth Price, John A. Pluvinage, Stanley L. Schrier, and Irving L. Weissman. "Hematopoietic Stem Cells Are the Disease-Initiating Cells in the Myelodysplastic Syndromes." Blood 118, no. 21 (2011): 789. http://dx.doi.org/10.1182/blood.v118.21.789.789.

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Abstract Abstract 789 Myelodysplastic syndrome (MDS) is a disorder of ineffective hematopoiesis presumed to originate from self-renewing clonal hematopoietic stem cells (HSC). Previous work has shown that immunophenotypic HSC from MDS patients harbor characteristic clonal cytogenetic abnormalities such as del(5q) at high levels, strongly suggesting that the HSC is the MDS-initiating cell (Tehranchi R., et al., NEJM, 363:11;1025-37, 2010); however, these studies did not examine other cytogenetic subtypes of MDS, nor did they functionally evaluate the HSC from these patients for their ability to

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12

Poulos, Michael Gustave, Michael Gutkin, Christopher Y. Park, and Jason M. Butler. "Rejuvenation of Aged Vascular Niches to Enhance Hematopoietic Function." Blood 126, no. 23 (2015): 781. http://dx.doi.org/10.1182/blood.v126.23.781.781.

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Abstract The molecular mechanisms regulating the aging of hematopoietic stem cells (HSCs) are poorly understood. To date, most studies describing age-related alterations have focused on HSC-intrinsic alterations, showing that the absolute number of immunophenotypically defined HSCs increases with age but that aged HSCs exhibit decreased long-term reconstitution potential, self-renewing capacity, altered transciptomes, cell-cycling and responses to cellular stress and DNA damage. Furthermore, old HSCs exhibit a significant myeloid bias at the expense of lymphopoiesis, which is thought to predis

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13

Sun, Shengming, Fujun Xuan, Hongtuo Fu, Jian Zhu, Xianping Ge, and Zhimin Gu. "Transciptomic and histological analysis of hepatopancreas, muscle and gill tissues of oriental river prawn (Macrobrachium nipponense) in response to chronic hypoxia." BMC Genomics 16, no. 1 (2015). http://dx.doi.org/10.1186/s12864-015-1701-3.

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14

Yuan, Chen, Jingya Xu, Qianqian Chen, et al. "C-terminal domain phosphatase-like 1 (CPL1) is involved in floral transition in Arabidopsis." BMC Genomics 22, no. 1 (2021). http://dx.doi.org/10.1186/s12864-021-07966-8.

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Abstract Background RNA polymerase II plays critical roles in transcription in eukaryotic organisms. C-terminal Domain Phosphatase-like 1 (CPL1) regulates the phosphorylation state of the C-terminal domain of RNA polymerase II subunit B1, which is critical in determining RNA polymerase II activity. CPL1 plays an important role in miRNA biogenesis, plant growth and stress responses. Although cpl1 mutant showes delayed-flowering phenotype, the molecular mechanism behind CPL1’s role in floral transition is still unknown. Results To study the role of CPL1 during the floral transition, we first tes

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15

Yan, Xiujuan, Xin He, Yunxia Zhao, Jingxia Gao, and Xuemei Wang. "Transciptome and Metabolomics Analysis of Pepper (Yin Chuan Cavel ) Male Sterility Lind." Molecular Plant Breeding, 2020. http://dx.doi.org/10.5376/mpb.2020.11.0024.

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16

Wu, Zhi-Gang, Wu Jiang, Nitin Mantri, Xiao-Qing Bao, Song-Lin Chen, and Zheng-Ming Tao. "Transciptome analysis reveals flavonoid biosynthesis regulation and simple sequence repeats in yam (Dioscorea alata L.) tubers." BMC Genomics 16, no. 1 (2015). http://dx.doi.org/10.1186/s12864-015-1547-8.

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17

Spitler, Kathryn M., Jessica M. Ponce, Duane D. Hall, and Chad E. Grueter. "Abstract 278: Cardiac Med1 is Necessary for Postnatal Survival in Mice." Circulation Research 117, suppl_1 (2015). http://dx.doi.org/10.1161/res.117.suppl_1.278.

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Alterations in gene transcription are commonly associated with cardiovascular disease pathogenesis characterized by cardiomyocyte hypertrophy. The phenotypic responses result in diminished cardiac contractility, ventricular dilation, fibrosis and ultimately sudden death. The mediator complex is a crucial facilitator of gene transcription; however, few studies have investigated the role of mediator in cardiovascular disease initiation and progression. A key subunit of the Mediator complex, MED1, interacts with nuclear receptors to target gene-specific transcription. To determine the role of MED

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18

Hofeld, Benjamin C., Venkata K. Puppala, Sudhi Tyagi, et al. "Lactobacillus plantarum 299v probiotic supplementation in men with stable coronary artery disease suppresses systemic inflammation." Scientific Reports 11, no. 1 (2021). http://dx.doi.org/10.1038/s41598-021-83252-7.

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AbstractRecent trials demonstrate that systemic anti-inflammatory therapy reduces cardiovascular events in coronary artery disease (CAD) patients. We recently demonstrated Lactobacillus plantarum 299v (Lp299v) supplementation improved vascular endothelial function in men with stable CAD. Whether this favorable effect is in part due to anti-inflammatory action remains unknown. Testing this hypothesis, we exposed plasma obtained before and after Lp299v supplementation from these subjects to a healthy donor’s PBMCs and measured differences in the PBMC transciptome, performed gene ontological anal

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19

Liu, Zhigang, Tian Yuan, Xiaoshuang Dai, Lin Shi, and Xuebo Liu. "Intermittent Fasting Alleviates Diabetes-induced Cognitive Decline via Gut Microbiota-metabolites-brain Axis (OR32-04-19)." Current Developments in Nutrition 3, Supplement_1 (2019). http://dx.doi.org/10.1093/cdn/nzz052.or32-04-19.

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Abstract Objectives Cognitive decline is one of severe type 2 diabetes complications. Intermittent fasting (IF) is a promising dietary intervention for T2D risk reduction, but its protective effect and mechanism on diabetic cognitive dysfunction remain elusive. Gut microbiota plays a vital role interphasing diet and host physiology and pathology and highly affected by the dietary composition and patterns. It has been reported that the microbiota homeostasis is essential for maintenance of gut health and for modulating cognitive function. We hypothesized that gut microbiota might play a pivotal

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20

Zeineddine, Hussein, Romuald Girard, Yan Li, et al. "Abstract 121: Transcriptome Profiling of Laser-Capture Microdissected Endothelial Cells From Human Cerebral Cavernous Malformations." Stroke 48, suppl_1 (2017). http://dx.doi.org/10.1161/str.48.suppl_1.121.

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Introduction: Cerebral cavernous malformations (CCMs) are common lesions causing stroke and epilepsy. Mechanistic studies in cultured endothelial cells (ECs) and in murine models have correlated CCM gene loss with signaling aberrations related to MEKK3-KLF2/4, Rho/ROCK and angiogenesis activity, and disruption of junctional proteins and EC-mesenchymal transition. Other studies demonstrated a robust antigen driven clonally expanded B-cell immune response, and pathogenetic B-T cell interactions. We hypothesized that the transcriptome of endothelial ECs from human CCM lesions reflects these postu

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