Relevant bibliographies by topics / Transcriptional autoregulation

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Transcriptional autoregulation'

Author: Grafiati
Published: 6 September 2023
Last updated: 31 July 2025

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Journal articles on the topic "Transcriptional autoregulation"

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De Siervi, Adriana, Paola De Luca, Jung S. Byun, et al. "Transcriptional Autoregulation by BRCA1." Cancer Research 70, no. 2 (2010): 532–42. http://dx.doi.org/10.1158/0008-5472.can-09-1477.

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Crews, Stephen T., and Joseph C. Pearson. "Transcriptional autoregulation in development." Current Biology 19, no. 6 (2009): R241—R246. http://dx.doi.org/10.1016/j.cub.2009.01.015.

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Hearing, P., and T. Shenk. "Sequence-independent autoregulation of the adenovirus type 5 E1A transcription unit." Molecular and Cellular Biology 5, no. 11 (1985): 3214–21. http://dx.doi.org/10.1128/mcb.5.11.3214-3221.1985.

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The adenovirus E1A gene is known to be autoregulated at the level of transcription. Autoregulation was found to be mediated by products of the E1A 13S mRNA, which induced a fivefold increase in E1A transcription rate. Deletion analysis suggested that the autoregulation did not require any specific sequence in the E1A transcriptional control region. This conclusion was reinforced by the demonstration that a cellular alpha-globin gene substituted for the E1A gene on the adenovirus chromosome was also positively regulated by E1A gene products.
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Hearing, P., and T. Shenk. "Sequence-independent autoregulation of the adenovirus type 5 E1A transcription unit." Molecular and Cellular Biology 5, no. 11 (1985): 3214–21. http://dx.doi.org/10.1128/mcb.5.11.3214.

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The adenovirus E1A gene is known to be autoregulated at the level of transcription. Autoregulation was found to be mediated by products of the E1A 13S mRNA, which induced a fivefold increase in E1A transcription rate. Deletion analysis suggested that the autoregulation did not require any specific sequence in the E1A transcriptional control region. This conclusion was reinforced by the demonstration that a cellular alpha-globin gene substituted for the E1A gene on the adenovirus chromosome was also positively regulated by E1A gene products.
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Hobert, Oliver. "Maintaining a memory by transcriptional autoregulation." Current Biology 21, no. 4 (2011): R146—R147. http://dx.doi.org/10.1016/j.cub.2011.01.005.

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MAGENHEIM, Judith, Rachel HERTZ, Ina BERMAN, Janna NOUSBECK та Jacob BAR-TANA. "Negative autoregulation of HNF-4α gene expression by HNF-4α1". Biochemical Journal 388, № 1 (2005): 325–32. http://dx.doi.org/10.1042/bj20041802.

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HNF-4α (hepatocyte nuclear factor-4α) is required for tissue-specific expression of many of the hepatic, pancreatic, enteric and renal traits. Heterozygous HNF-4α mutants are inflicted by MODY-1 (maturity onset diabetes of the young type-1). HNF-4α expression is reported here to be negatively autoregulated by HNF-4α1 and to be activated by dominant-negative HNF-4α1. Deletion and chromatin immunoprecipitation analysis indicated that negative autoregulation by HNF-4α1 was mediated by its association with the TATA-less HNF-4α core promoter enriched in Sp1, but lacking DR-1 response elements. Also
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Sassone-Corsi, Paolo, John C. Sisson, and Inder M. Verma. "Transcriptional autoregulation of the proto-oncogene fos." Nature 334, no. 6180 (1988): 314–19. http://dx.doi.org/10.1038/334314a0.

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Delahodde, A., T. Delaveau, and C. Jacq. "Positive autoregulation of the yeast transcription factor Pdr3p, which is involved in control of drug resistance." Molecular and Cellular Biology 15, no. 8 (1995): 4043–51. http://dx.doi.org/10.1128/mcb.15.8.4043.

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Simultaneous resistance to an array of drugs with different cytotoxic activities is a property of Saccharomyces cerevisiae, in which the protein Pdr3p has recently been shown to play a role as a transcriptional regulator. We provide evidence that the yeast PDR3 gene, which encodes a zinc finger transcription factor implicated in certain drug resistance phenomena, is under positive autoregulation by Pdr3p. DNase I footprinting analyses using bacterially expressed Pdr3p showed specific recognition by this protein of at least two upstream activating sequences in the PDR3 promoter. The use of lacZ
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Irvine, K. D., J. Botas, S. Jha, R. S. Mann, and D. S. Hogness. "Negative autoregulation by Ultrabithorax controls the level and pattern of its expression." Development 117, no. 1 (1993): 387–99. http://dx.doi.org/10.1242/dev.117.1.387.

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The Drosophila homeotic gene Ultrabithorax (Ubx) encodes transcriptional regulatory proteins (UBX) that specify thoracic and abdominal segmental identities. Ubx autoregulation was examined by manipulating UBX levels, both genetically and with an inducible transgene, and monitoring the effect of these manipulations on the expression of Ubx and Ubx-lacZ reporter genes. Positive autoregulation by Ubx is restricted to the visceral mesoderm, while in other tissues Ubx negatively autoregulates. In some cases, negative autoregulation stabilizes UBX levels, while in others it modulates the spatial and
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Conacci-Sorrell, Maralice, Inbal Simcha, Tamar Ben-Yedidia, Janna Blechman, Pierre Savagner, and Avri Ben-Ze'ev. "Autoregulation of E-cadherin expression by cadherin–cadherin interactions." Journal of Cell Biology 163, no. 4 (2003): 847–57. http://dx.doi.org/10.1083/jcb.200308162.

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Transcriptional repression of E-cadherin, characteristic of epithelial to mesenchymal transition, is often found also during tumor cell invasion. At metastases, migratory fibroblasts sometimes revert to an epithelial phenotype, by a process involving regulation of the E-cadherin–β-catenin complex. We investigated the molecular basis of this regulation, using human colon cancer cells with aberrantly activated β-catenin signaling. Sparse cultures mimicked invasive tumor cells, displaying low levels of E-cadherin due to transcriptional repression of E-cadherin by Slug. Slug was induced by β-caten
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Dissertations / Theses on the topic "Transcriptional autoregulation"

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Zhu, Cong. "GENE REGULATORY NETWORKS OF AGL15 A PLANT MADS TRANSCRIPTION FACTOR." UKnowledge, 2005. http://uknowledge.uky.edu/gradschool_diss/446.

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Plant embryogenesis is an intriguing developmental process that is controlled by many genes. AGAMOUS Like 15 (AGL15) is a MADS-domain transcriptional regulator that accumulates preferentially during this stage. However, at the onset of this work it was unknown which genes are regulated by AGL15 or how AGL15 is regulated. This dissertation is part of the ongoing effort to understand the biological roles of AGL15. To decipher how AGL15 functions during plant development, a chromatin immunoprecipitation (ChIP) approach was adapted to obtain DNA fragments that are directly bound by AGL15 in vivo.
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Unoarumhi, Yvette Ochuwa. "Evolution of a Bacterial Global Regulator- Lrp." University of Toledo Health Science Campus / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=mco1461859521.

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Ouk, Tan-Sothéa. "Rôle de la protéine LMP1 dans la balance survie/apoptose des cellules infectées par le virus d'Epstein-Barr." Limoges, 2008. https://aurore.unilim.fr/theses/nxfile/default/e0773f8b-990b-46fb-983c-9e953f8732d5/blobholder:0/2008LIMO4028.pdf.

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Le virus d’Epstein-Barr (EBV) est un herpèsvirus humain qui infecte 90% de la population, mais l’infection est, le plus souvent, asymptomatique. Après infection, le virus établit une latence virale caractérisée par l’expression restreinte de gènes viraux, appelés gènes de latence permettant au virus de persister tout au long de la vie. On définit trois latences virales (I, II et III) selon le profil d’expression des gènes de latence codant les antigènes EBNAs (-1, -2, -3 et -LP) et les protéines membranaires LMPs (-1 et -2). L���immortalisation in vitro des lymphocytes B aboutit à des lignées
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Vernié, Tatiana. "Analyse fonctionnelle d'EFD, un régulateur transcriptionnel de la nodulation au cours de l'interaction symbiotique entre Medicago truncatula et Sinorhizobium meliloti." Toulouse 3, 2008. http://thesesups.ups-tlse.fr/222/.

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Les Légumineuses sont capables d'établir une interaction symbiotique avec des bactéries de la rhizosphère, appelées Rhizobia. Cette interaction implique deux processus strictement contrôlés par la plante : une infection bactérienne et la formation d'un nouvel organe : le nodule, dans lequel l'azote atmosphérique est réduit. Les mécanismes de ces contrôles restent peu connus. A partir d'études transcriptomiques, nous avons sélectionné un régulateur potentiel, EFD (Ethylene response Factor required for nodule Differentiation), codant pour un facteur de transcription de type ERF. Le profil d'expr
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Wu, Tian-Yu, and 吳天宇. "Autoregulation of gbsR, identification of GbsR-binding sites, and transcriptional regulation of opuB and opuC operons in Bacillus subtilis." Thesis, 2013. http://ndltd.ncl.edu.tw/handle/47042651578494871587.

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碩士<br>國立陽明大學<br>生化暨分子生物研究所<br>101<br>The soil bacterium Bacillus subtilis can use glycine betaine, which is one of the most important osmoprotectants in nature, to cope with the environmental osmotic stress. Choline oxidation by GbsB (choline dehydrogenase) and GbsA (glycine betaine aldehyde dehydrogenase) is the only known pathway for the synthesis of glycine betaine in B. subtilis. Choline cannot be synthesized by B. subtilis cells, but can be imported from the environment by osmoinducible ABC transporters OpuB and OpuC. GbsR is a choline-sensing regulator that negatively controls transcript
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Dar, Roy David. "Adaptation and Stochasticity of Natural Complex Systems." 2011. http://trace.tennessee.edu/utk_graddiss/959.

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The methods that fueled the microscale revolution (top-down design/fabrication, combined with application of forces large enough to overpower stochasticity) constitute an approach that will not scale down to nanoscale systems. In contrast, in nanotechnology, we strive to embrace nature’s quite different paradigms to create functional systems, such as self-assembly to create structures, exploiting stochasticity, rather than overwhelming it, in order to create deterministic, yet highly adaptable, behavior. Nature’s approach, through billions of years of evolutionary development, has achieved se
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Chandra, Soumyanetra. "Probing Protein Sequence-Function Relationships using Deep Mutational Scanning." Thesis, 2021. https://etd.iisc.ac.in/handle/2005/5662.

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Deep Mutational Scanning (DMS) approaches help elucidate sequence-function-phenotype relationships in proteins, which ultimately improves our understanding of residue (or nucleotide)-specific contributions to protein function and organismal fitness. Such comprehensive knowledge finds use in reliable prediction of consequences of mutations, as well as in protein design and engineering. We have investigated the molecular mechanisms of how mutations at surface exposed sites, away from the active sites in a model protein, CcdB produce drastic defects on the protein’s activity and organismal phenot
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Books on the topic "Transcriptional autoregulation"

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Gill, Robert James Montgomery. Characterization of the human RB1 promoter and of elements involved in transcriptional autoregulation. National Library of Canada = Bibliothèque nationale du Canada, 1993.

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Book chapters on the topic "Transcriptional autoregulation"

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Draper, David E. "Mechanisms of Ribosomal Protein Translational Autoregulation." In Post-Transcriptional Control of Gene Expression. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75139-4_28.

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Oei, Shiao Li, Herbert Herzog, Monica Hirsch-Kauffmann, Rainer Schneider, Bernhard Auer, and Manfred Schweiger. "Transcriptional regulation and autoregulation of the human gene for ADP-ribosyltransferase." In ADP-Ribosylation: Metabolic Effects and Regulatory Functions. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4615-2614-8_13.

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Bateman, Erik. "Autoregulation of Eukaryotic Transcription Factors." In Progress in Nucleic Acid Research and Molecular Biology. Elsevier, 1998. http://dx.doi.org/10.1016/s0079-6603(08)60892-2.

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Kaufmann Kerstin, Nagasaki Masao, and Jáuregui Ruy. "Modelling the Molecular Interactions in the Flower Developmental Network of Arabidopsis thaliana." In Studies in Health Technology and Informatics. IOS Press, 2011. https://doi.org/10.3233/978-1-60750-704-8-279.

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We present a dynamical model of the gene network controlling flower development in Arabidopsis thaliana. The network is centered at the regulation of the floral organ identity genes (AP1, AP2, AP3, PI and AG) and ends with the transcription factor complexes responsible for differentiation of floral organs. We built and simulated the regulatory interactions that determine organ specificity using an extension of hybrid Petri nets as implemented in Cell Illustrator. The network topology is characterized by two main features: (1) the presence of multiple autoregulatory feedback loops requiring the
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Conference papers on the topic "Transcriptional autoregulation"

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Herlinger, Alice Laschuk, Min Gao, Ren-Chin Wu, Tian-Li Wang, Leticia B. A. Rangel, and Ie-Ming Shih. "Abstract A80: NAC1 attenuates BCL6 negative autoregulation and functions as a BCL6 coactivator of FOXQ1 transcription in ovarian cancer (OVCA)." In Abstracts: AACR Special Conference: Advances in Ovarian Cancer Research: Exploiting Vulnerabilities; October 17-20, 2015; Orlando, FL. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3265.ovca15-a80.

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