Relevant bibliographies by topics / Transformed growth

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Transformed growth'

Author: Grafiati
Published: 4 June 2021
Last updated: 8 February 2022

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Journal articles on the topic "Transformed growth"

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MORSE, PAIGE MARIE. "TRANSFORMED DSM TARGETS GROWTH." Chemical & Engineering News Archive 89, no. 11 (March 14, 2011): 24–27. http://dx.doi.org/10.1021/cen-v089n011.p024.

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Yang, Bo, Haguy Wolfenson, Vin Yee Chung, Naotaka Nakazawa, Shuaimin Liu, Junqiang Hu, Ruby Yun-Ju Huang, and Michael P. Sheetz. "Stopping transformed cancer cell growth by rigidity sensing." Nature Materials 19, no. 2 (October 28, 2019): 239–50. http://dx.doi.org/10.1038/s41563-019-0507-0.

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Macek, T., and K. Green. "Growth Studies Using Transformed Roots ofSolanum aviculareandS. nigrum." Planta Medica 59, S 1 (December 1993): A659. http://dx.doi.org/10.1055/s-2006-959923.

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Reed, M. L. E., and Bernard R. Glick. "Growth of canola (Brassica napus) in the presence of plant growth-promoting bacteria and either copper or polycyclic aromatic hydrocarbons." Canadian Journal of Microbiology 51, no. 12 (December 1, 2005): 1061–69. http://dx.doi.org/10.1139/w05-094.

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Growth of canola (Brassica napus) seeds treated with plant growth-promoting bacteria in copper-contaminated and polycyclic aromatic hydrocarbon (PAH)-contaminated soils was monitored. Pseudomonas asplenii AC, isolated from PAH-contaminated soil, was transformed to express a bacterial gene encoding 1-aminocyclopropane-1-carboxylate (ACC) deaminase, and both native and transformed bacteria were tested for growth promotion. Inoculation of seeds, grown in the presence of copper or creosote, with either native or transformed P. asplenii AC significantly increased root and shoot biomass. Native and transformed P. asplenii AC and transformed P. asplenii AC encapsulated in alginate were equally effective at promoting plant growth in copper-contaminated soils. In creosote-contaminated soils the native bacterium was the least effective, and the transformed encapsulated bacterium was the most effective in growth promotion.Key words: plant growth-promoting bacteria, phytoremediation, copper, polycyclic aromatic hydrocarbons, Brassica napus, ethylene, alginate encapsulation.
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Kuntz, Sabine, Silvia Rudloff, and Clemens Kunz. "Oligosaccharides from human milk influence growth-related characteristics of intestinally transformed and non-transformed intestinal cells." British Journal of Nutrition 99, no. 3 (March 2008): 462–71. http://dx.doi.org/10.1017/s0007114507824068.

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Human milk oligosaccharides (HMO) are considered to influence the composition of the gut microflora in breastfed infants. We investigated direct effects of milk HMO fractions or individual oligosaccharides on proliferation, differentiation and apoptosis in transformed human intestinal cells (HT-29 and Caco-2) and non-transformed small intestinal epithelial crypt cells of fetal origin (human intestinal epithelial cells; HIEC). We observed growth inhibition induced by neutral and acidic HMO fractions in HT-29, Caco-2 and HIEC cells in a dose dependent manner. However, the effects varied between cell lines, i.e. HT-29 and Caco-2 cells were more sensitive than HIEC cells. In HT-29, all 16 individual neutral and acidic oligosaccharides except from the two fucosyllactoses had an inhibitory effect on cell growth. Regarding the induction of differentiation in HT-29 and HIEC cells a threshold concentration was observed at 7·5 mg/ml for neutral and acidic HMO fractions. Among individual oligosaccharides, only sialyllactoses induced differentiation in HT-29 and HIEC cells; no effect neither of fractions nor of individual oligosaccharides was found in Caco-2 cells. A strong induction of apoptosis was only detected in HT-29 and HIEC cells for neutral oligosaccharide but not for acidic fractions. HMO were shown to induce growth inhibition in intestinal cells through two different mechanisms, by suppressing cell cycle progression through induction of differentiation and/or by influencing apoptosis. As the development and maturation of digestive and absorptive processes depend on differentiation our experiments show that oligosaccharides are effective at influencing various stages in gastrointestinal development in vitro.
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Burgess, Antony W. "Growth control mechanisms in normal and transformed intestinal cells." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 353, no. 1370 (June 29, 1998): 903–9. http://dx.doi.org/10.1098/rstb.1998.0254.

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The cells populating the intestinal crypts are part of a dynamic tissue system which involves the self–renewal of stem cells, a commitment to proliferation, lineage–specific differentiation, movement and cell death. Our knowledge of these processes is limited, but even now there are important clues to the nature of the regulatory systems, and these clues are leading to a better understanding of intestinal cancers. Few intestinal–specific markers have been described; however, homeobox genes such as cdx–2 appear to be important for morphogenic events in the intestine. There are several intestinal cell surface proteins such as the A33 antigen which have been used as targets for immunotherapy. Many regulatory cytokines (lymphokines or growth factors) influence intestinal development: enteroglucagon, IL–2, FGF, EGF family members. In conjunction with cell–cell contact and/or ECM, these cytokines lead to specific differentiation signals. Although the tissue distribution of mitogens such as EGF, TGFα, amphiregulin, betacellulin, HB–EGF and cripto have been studied in detail, the physiological roles of these proteins have been difficult to determine. Clearly, these mitogens and the corresponding receptors are involved in the maintenance and progression of the tumorigenic state. The interactions between mitogenic, tumour suppressor and oncogenic systems are complex, but the tumorigenic effects of multiple lesions in intestinal carcinomas involve synergistic actions from lesions in these different systems. Together, the truncation of apc and activation of the ras oncogene are sufficient to induce colon tumorigenesis. If we are to improve cancer therapy, it is imperative that we discover the biological significance of these interactions, in particular the effects on cell division, movement and survival.
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Rizzino, Angie, and Eric Ruff. "Fibroblast growth factor induces the soft agar growth of two non-transformed celllines." In Vitro Cellular & Developmental Biology 22, no. 12 (December 1986): 749–55. http://dx.doi.org/10.1007/bf02621092.

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Massagué, J. "Transforming growth factor-beta modulates the high-affinity receptors for epidermal growth factor and transforming growth factor-alpha." Journal of Cell Biology 100, no. 5 (May 1, 1985): 1508–14. http://dx.doi.org/10.1083/jcb.100.5.1508.

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The epidermal growth factor (EGF) receptor mediates the induction of a transformed phenotype in normal rat kidney (NRK) cells by transforming growth factors (TGFs). The ability of EGF and its analogue TGF-alpha to induce the transformed phenotype in NRK cells is greatly potentiated by TGF-beta, a polypeptide that does not interact directly with binding sites for EGF or TGF-alpha. Our evidence indicates that TGF-beta purified from retrovirally transformed rat embryo cells and human platelets induces a rapid (t 1/2 = 0.3 h) decrease in the binding of EGF and TGF-alpha to high-affinity cell surface receptors in NRK cells. No change due to TGF-beta was observed in the binding of EGF or TGF-alpha to lower affinity sites also present in NRK cells. The effect of TGF-beta on EGF/TGF-alpha receptors was observed at concentrations (0.5-20 pM) similar to those at which TGF-beta is active in promoting proliferation of NRK cells in monolayer culture and semisolid medium. Affinity labeling of NRK cells and membranes by cross-linking with receptor-bound 125I-TGF-alpha and 125I-EGF indicated that both factors interact with a common 170-kD receptor structure. Treatment of cells with TGF-beta decreased the intensity of affinity-labeling of this receptor structure. These data suggest that the 170 kD high-affinity receptors for EGF and TGF-alpha in NRK cells are a target for rapid modulation by TGF-beta.
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Nomura, Takahiro, Kazuo Ryoyama, Gensaku Okada, Sadaya Matano, Shinobu Nakamura, and Tadanori Kameyama. "Non-transformed, but notras/myc-transformed, Serum-free Mouse Embryo Cells Recover from Growth Suppression by Azatyrosine." Japanese Journal of Cancer Research 83, no. 8 (August 1992): 851–58. http://dx.doi.org/10.1111/j.1349-7006.1992.tb01990.x.

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Holzer, Claudia, Peter Maier, and Gerhard Zbinden. "Comparison of Exogenous Growth Stimuli for Chemically Transformed Cells: Growth Factors, Serum and Cocultures." Pathobiology 54, no. 5-6 (1986): 237–44. http://dx.doi.org/10.1159/000163362.

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Dissertations / Theses on the topic "Transformed growth"

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Abraham, Samuel D. M. "Activation of multiple hemopoietic growth factor genes in Abelson virus transformed myeloid cells." Thesis, University of British Columbia, 1988. http://hdl.handle.net/2429/27786.

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The stringent requirement for hemopoietic growth factors (HGF) in the induction of hemopoiesis in vitro has raised questions as to their possible role(s) in leukemogenesis. Several recent clinical studies have shown aberrant cell growth factor gene activation in patient derived leukemic cells. Assessment of growth factor activity is often based on in vitro bioactivity assays of conditioned media or body fluids. The specificity of this type of endpoint is, however, open to question due to the overlap in biological activities of many HGFs. In assessing the role of growth factor gene expression in a murine myeloid leukemia model I have used a sensitive RNA detection procedure coupled with a vector-probe system that enables the synthesis of uniformly labelled radioactive DNA probes to detect unambiguously the expression of particular growth factor genes. The Abelson murine leukemia virus (A-MuLV) derived myeloid transformants used in this study had previously been shown to produce a multi-lineage colony stimulating activity (CSA). While these A-MuLV transformants were shown to produce GM-CSF, it seemed likely that the multi-lineage CSA was due to another factor. In addition to confirming the expression of GM-CSF mRNA, I was able to show that the cells of all four A-MuLV transformed lines tested also expressed interleukin-3 mRNA. This finding was strongly corroborated by bio-activity data obtained using the CM from the A-MuLV myeloid transformants. Additional preliminary analysis by bioactivity assays have also shown the possible presence of interleukin-6 (IL-6) and a recently described pre-B cell factor suggesting perhaps a common mechanism underlying the activation of these various growth factor genes.
Medicine, Faculty of
Medical Genetics, Department of
Graduate
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Roberts, April M. "Steroid hormone treatments alter growth characteristics in transformed human ovarian cell lines." Virtual Press, 2003. http://liblink.bsu.edu/uhtbin/catkey/1265095.

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Liu, Chi 1963. "Transforming growth factors produced by normal and neoplastically transformed rat liver epithelial cells in culture." Thesis, McGill University, 1987. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=63768.

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SIPES, NANCY JO. "GROWTH REGULATION OF HUMAN MELANOMA: FACTORS INVOLVED IN THE EXPRESSION OF THE TRANSFORMED PHENOTYPE (SOFT AGAR, GROWTH FACTORS, PLATELETS, ENDOTHELIAL CELLS, PARACRINE)." Diss., The University of Arizona, 1986. http://hdl.handle.net/10150/183788.

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Cellular transformation is accomplished in vitro through the concerted action of growth factors and oncogenes. This association has demonstrated that malignant growth results from aberrations in pathways that normally operate to control proliferation. Activation of genes that code for growth factors, their receptors, and/or molecules essential in the transduction of signals from the cell surface to the nucleus are all potential mechanisms by which tumor cells could establish a selective growth advantage over normal cells. This dissertation addresses the question of what oncogenic mechanisms are important in the development and progression of human melanoma. These studies show that melanoma growth is regulated by endogenous substances produced by the melanoma cells themselves (autocrine stimulation), as well as by exogenous substances supplied by neighboring cells and platelets (paracrine stimulation). These factors work to drive the expression of the transformed phenotype for melanoma as evidenced by induction of serum-free soft agar growth. Human platelets were found to the the richest source of paracrine growth promoters. The factor from human platelets was characterized and partially purified. Melanoma cells respond to this 60,000 molecular weight, disulfide-bond-containing protein in colony formation assays. In addition, the protein has endothelial cell growth factor activity. Purified fractions which promoted optimal colony formation for human melanoma cells also maximally stimulated monolayer growth of bovine aortic endothelial cells, while melanocytes were nonresponsive. This implies that melanoma cells are expressing receptors for a protein which plays no known or apparent role in the normal growth of melanocytes. Melanoma cells are sensitive to growth regulatory molecules of autocrine and paracrine nature. This dissertation provides clues to the genetic lesions which have occurred in these melanoma cells to influence their proliferation. The aberrations appear to reside in those genes important in growth factor pathways at the level of endogenous production and misguided response to exogenous factors through receptor expression. We can not hope to fully inhibit the proliferation of tumor cells until we identify and understand those forces which drive their growth. These studies have increased our knowledge of those signals which stimulate melanoma cellular proliferation, and thus provide insight into important therapeutic targets.
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Taglienti, Cherie A. "Signal Transduction by the EGF Receptor in Normal and Transformed Cells: a Dissertation." eScholarship@UMMS, 1996. http://escholarship.umassmed.edu/gsbs_diss/314.

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Much research has been conducted and significant progress has been made in understanding how signals are transmitted in response to extracellular stimuli. Continued research is necessary to elucidate how specificity in signaling from the cell surface is achieved and to understand how disruption of such signaling pathways leads to disease. The question of specificity is important since many of the same proteins can be activated by different stimuli in a variety of signaling pathways. One means of achieving specificity would be for different cell types to express different sets of proteins that interact with "common" proteins such as GRB2, Shc, and Ras. There were two goals to my dissertation research. The first goal was to study disease which results from perturbation of the EGF receptor. The second was to explore the possibility that signals generated in response to EGF or other stimuli might be transmitted through MAP kinase proteins other than ERKs 1 and 2. Chapter II of this thesis centers around further analyzing the disease potential of erbB in chickens. I have conducted oncogenicity tests with an erbB oncogene that contains no mutations, only the characteristic N-terminal truncation. My studies have revealed that truncation of the EGF receptor (erbB) can induce tumorigenesis of the kidney as well as the erythroblastosis originally associated with erbB. Previous studies have only found kidney tumors in association with mutated forms of the erbB oncogene. Furthermore, I have demonstrated that mutational removal of a negative regulatory serine phosphorylation site increases the number of wing web tumors caused by the erbB oncogene. Chapter III describes the identification of the Ser/Thr protein kinase p56 KKIAMRE and characterization of p56 KKIAMRE and p42 KKIALRE. These kinases have homology to the MAP kinase group and contain the conserved Thr-Xaa-Tyr dual phosphorylation site. I have demonstrated that both kinases can be activated by treatment of cells with EGF. Interestingly, phosphorylation of Thr and Tyr in the Thr-Xaa-Tyr motif is not necessary for EGF stimulated activity.
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Mißbach, Sven. "Untersuchungen zu standortunabhängigen Eigenschaften des Wachstums für verschiedene Baumarten." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-175061.

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Towler, Melissa J. "Effects of inoculum density, carbon concentration, and feeding scheme on the growth of transformed roots of Artemisia annua in a modified nutrient mist bioreactor." Digital WPI, 2005. https://digitalcommons.wpi.edu/etd-dissertations/269.

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Previous work has shown that despite the lack of oxygen limitation, transformed roots of Artemisia annua had lower biomass productivity in a nutrient mist bioreactor than in a liquid-phase bubble column reactor where the roots demonstrated metabolic signs of oxygen stress. Mathematical modeling suggested that the roots were too sparsely packed to capture mist particles efficiently and to achieve high growth rates. In this study, higher packing fractions were tested, and the growth rate increased significantly. Similarly, higher sucrose concentrations increased the growth rate. Growth kinetics for 2, 4, and 6 days showed an unexpected decrease or stationary growth rate after only 4 days for both 3% and 5% sucrose feeds. Residual media analyses indicated that carbon was not exhausted, nor were other major nutrients including phosphate. Increasing the misting frequency such that the total amount of carbon delivered from a 3% sucrose feed was equivalent to that delivered in a 5% sucrose feed showed that growth was affected by the modified cycle. These studies showed that both the concentration of carbon source and alteration of misting frequency can significantly increase growth rates of hairy roots in mist reactors.
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Zinn, Jennifer Nicole. "Studying modulation of tumor necrosis factor-Ã and transforming growth factor-ß by norepineprhine in murine alveolar macrophages, murine splenocytes and transformed cell populations." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp01/MQ33833.pdf.

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Shoji(kawata), Sanae. "p21Waf1/Cip1/Sdi1 functions to prevent apoptosis as well as stimulate growth in cells transformed or immortalized by human T-cell leukemia virus type 1-encoded Tax." Kyoto University, 2003. http://hdl.handle.net/2433/148476.

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Hafermann, Juliane O. [Verfasser], and Martin [Akademischer Betreuer] Löchelt. "The effect of APOBEC3A-mediated mutagenesis on tumour growth of immortalised, transformed cells and on survival of head and neck squamous cell carcinoma patients / Juliane O. Hafermann ; Betreuer: Martin Löchelt." Heidelberg : Universitätsbibliothek Heidelberg, 2019. http://d-nb.info/1177043645/34.

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Books on the topic "Transformed growth"

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Redeemed!: Embracing a transformed life. Springfield, Mo: My Healthy Church, 2012.

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Fanny, Lederlin, ed. La force de l'immatériel: Pour transformer l'économie. Paris: Presses Universitaires de France, 2011.

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Greenway, Jeffrey E. Make room to grow: Transform the church without killing the congregation. Nashville, TN: Abingdon Press, 2007.

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Randelli, Filippo, and Francesco Dini, eds. Oltre la globalizzazione: le proposte della Geografia economica. Florence: Firenze University Press, 2012. http://dx.doi.org/10.36253/978-88-6655-307-6.

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In 1980 Froebel, Heinrichs and Kreye published the English-language The New International Division of Labour, trying to highlight the consequences of market reorganization after the crisis of the mid 1970s, which was soon to transform into so-called globalization. A third of a century later, the "fantastic adventure" of market integration seems to have been crystallized by the 2007-2008 crisis, opening a further period of great instability. But the geography of wealth production has transformed radically and appears unrecognizable to the early-80s scholar. In a framework of great social, political and cultural change, China, a country at the time defined as an "economic dwarf", is the second largest economy on the planet and has become its "factory". The standardizing concept of "Third World" having vanished, some former colonial economies have undertaken rapid growth processes, while others have ruinously accentuated their underdevelopment. The traditionally advanced regions, then defined as "industrial", have opened out into trajectories defined, vice versa, as "post-industrial", some consolidating their competitive edge and others sparking lengthy declines.
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Matthews, Rose Peggy, ed. Read for your life: 11 ways to transform your life with books. Deerfield Beach, Fla: Health Communications, 2007.

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Leerssen, Joep. National Thought in Europe. NL Amsterdam: Amsterdam University Press, 2018. http://dx.doi.org/10.5117/9789462989542.

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Bringing together sources from many countries and many centuries, this study critically analyses the growth of national thought and of nationalism — from medieval ethnic prejudice to the Romantic belief in a nation’s ‘soul’. The belief and ideology of the nation’s cultural individuality emerged from a Europe-wide exchange of ideas, often articulated in literature and belles lettres. In the last two centuries, these ideas have transformed the map of Europe and the relations between people and government. In tracing the modern European nation-state, cross-nationally and historically, as the outcome of a cultural self-invention, Leerssen also provides a surprising perspective on Europe’s contemporary identity politics. National Thought in Europe has been brought up to date in this new, third edition.
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More light, less heat: How dialogue can transform Christian conflicts into growth. San Francisco, Calif: Jossey-Bass, 1999.

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Dixon, Jim. What would Jesus ask?: 10 questions that will transform your life. Ventura, California: Regal, 2013.

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Schwarz, Christian A. Paradigm shift in the church: How natural church development can transform theological thinking. Carol Stream, IL: ChurchSmart Resources, 1999.

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InGenius: Unleash your creativity to transform obstacles into opportunities. New York: HarperOne, 2012.

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Book chapters on the topic "Transformed growth"

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Aceña, Pablo Martín, and Elena Martínez Ruiz. "The Golden Age of Spanish Capitalism: Economic Growth without Political Freedom." In Spain Transformed, 30–46. London: Palgrave Macmillan UK, 2007. http://dx.doi.org/10.1057/9780230592643_2.

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Athota, Vidya S. "Conclusion: Building Transformed Growth Minded Organizations." In Mind over Matter and Artificial Intelligence, 57–60. Singapore: Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-0482-9_6.

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Fitz, T. A., M. M. Marr, T. L. Walden, W. A. Schmidt, and C. A. Winkel. "Gap Junctions in Transformed Rat Granulosa Cells." In Growth Factors and the Ovary, 245–50. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5688-2_27.

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Inman, Wendelyn H., and Graham Carpenter. "The Receptor for Epidermal Growth Factor." In Development and Recognition of the Transformed Cell, 111–21. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1925-2_8.

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Yamamoto, Tadashi, and Kumao Toyoshima. "Two erbB-Related Protooncogenes Encoding Growth Factor Receptors." In Development and Recognition of the Transformed Cell, 93–110. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1925-2_7.

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Fahrlander, Paul D., and Kenneth B. Marcu. "Regulation of c-myc Expression in Normal and Transformed Mammalian Cells." In Oncogenes and Growth Control, 264–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-73325-3_36.

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Heldin, C. H., and B. Westermark. "Autocrine Stimulation of Growth of Normal and Transformed Cells." In Growth Factors, Differentiation Factors, and Cytokines, 267–78. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74856-1_20.

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Pitts, John D. "Appendix: Growth Properties of Normal and Transformed Cells." In Ciba Foundation Symposium - Growth Control in Cell Cultures, 261–66. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470719817.app1.

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Walker, Cheryl, Edilberto Bermudez, Wendy Stewart, and Jeff Everitt. "Growth Factor and Growth Factor Receptor Expression in Transformed Rat Mesothelial Cells." In Mechanisms in Fibre Carcinogenesis, 377–83. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-1363-2_31.

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Heldin, Carl-Henrik, and Bengt Westermark. "Role of PDGF-Like Growth Factors in Autocrine Stimulation of Growth of Normal and Transformed Cells." In Oncogenes and Growth Control, 43–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-73325-3_6.

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Conference papers on the topic "Transformed growth"

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Serizawa, Ryosuke, Masanori Kikuchi, and Shinya Yamada. "Evaluation of Subsurface Crack Propagation Under Fatigue." In ASME 2014 Pressure Vessels and Piping Conference. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/pvp2014-28159.

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There is a proximity rule to calculate residual fatigue life of components in nuclear power plants. It is easy to evaluate soundness of a structure member by using this rule. If a subsurface crack is located near free surface of the structure, this real subsurface crack is transformed to surface crack. The condition to transform subsurface crack to surface crack is defined by relationship between crack size and the distance from crack tip to free surface. However, many organizations proposed proximity rules which differ from each other. It is advisable to verify which rule is preferable in these rules by experiment, but it is difficult to introduce subsurface crack at an optional position. Therefore, numerical simulation is needed for this purpose. Especially, S-version FEM is very useful for as much as model of subsurface crack is independent of global structure, and crack growth is easily simulated. Both subsurface and transformed surface crack growths are simulated by S-FEM. Subsurface crack grows toward free surface. When subsurface crack tip was touched to free surface, this crack was converted into surface crack by using stress intensity factor calculated at this time. In this way, crack growth behavior from subsurface to surface flaw is represented. By comparing the crack growth rate of surface to subsurface flaw with that of surface flaw transformed by each proximity rule, proximity rules can be verified by numerical simulation. Authors had proposed the proximity rule at the ASME PVP 2013 conference [1]. However, new rule was proposed by numerical simulation only under cyclic tensile load. In addition, only two proximity rules were studied at the last conference. In this study, the number of proximity rules is increased, and this problem is simulated under other loading condition such as cyclic bending load. New proximity rule gives reasonable and conservative results in numerical simulation.
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Hasegawa, Kunio, Yinsheng Li, Katsumasa Miyazaki, and Koichi Saito. "Comparison of Experiment and Calculation on Fatigue Crack Growth for Transformed Surface Flaw." In ASME 2012 Pressure Vessels and Piping Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/pvp2012-78688.

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If a subsurface flaw is located near a component surface, the subsurface flaw is transformed to a surface flaw in accordance with a flaw-to-surface proximity rule. The re-characterization process from subsurface to surface flaw is adopted in all fitness-for-service (FFS) codes. However, the criteria of the re-characterizations are different among the FFS codes. Cyclic tensile experiment was conducted on a carbon steel flat plate with a subsurface flaw at ambient temperature. The objective of this paper is to compare the experiment and calculation of fatigue crack growth behavior for a subsurface flaw and the transformed surface flaw, and to confirm the flaw-to-surface proximity rule defined by ASME Code Section XI and JSME S NA1 Code.
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Hasegawa, Kunio, Yinsheng Li, Katsumasa Miyazaki, and Koichi Saito. "Fatigue Crack Growth for Subsurface Flaws Near Component Surface and Proximity Rules." In ASME 2013 Pressure Vessels and Piping Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/pvp2013-97559.

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If a subsurface flaw is located near a component surface, the subsurface flaw is transformed to a surface flaw in accordance with a flaw-to-surface proximity rule. The re-characterization process from subsurface to surface flaw is adopted in all fitness-for-service (FFS) codes. However, the criteria of the re-characterizations are different among the FFS codes. Cyclic tensile experiment was conducted on a carbon steel flat plate with a subsurface flaw at ambient temperature. The objective of this paper is to compare the experiment and calculation of fatigue crack growth behavior for a subsurface flaw and the transformed surface flaw, and to describe the validity of the flaw-to-surface proximity rule defined by ASME Code Section XI, JSME S NA1 Code and other codes.
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Yoo, Byungseok, Darryll Pines, and Ashish S. Purekar. "Guided Lamb Wave Interrogation of a Curved Composite Plate [0/90] Using the Hilbert-Huang Transform Approach." In ASME 2008 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. ASMEDC, 2008. http://dx.doi.org/10.1115/smasis2008-591.

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This paper discusses a study of the Hilbert-Huang transform approach as a structural health monitoring tool for a curved composite panel with a [90/0/0/90]sym lay-up sequence. The Hilbert amplitude, phase, and frequency coupled with empirical mode decomposition (EMD) process are defined as the Hilbert-Huang transform (HHT) and used to evaluate structural damage in the curved composite panel as the level of damage increases. Piezoelectric (PZT) sheets are used as an actuator and receivers, akin to the pitch-catch method to excite the guided Lamb waves and to obtain the vibratory response of the curved composite panel. In this paper, a new damage metric using the Hilbert-Huang transformed energy and amplitude in conjunction with the Hilbert-Huang transformed phase are proposed and investigated as a new structural damage index. Results show that these damage metrics are capable of tracking progressive damage growth in the form of loss of torque load.
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Rystad, J. "Turning Decline to Growth for North Sea Countries – How Cost Reduction has Transformed a Declining Basin to a Growth Basin." In 80th EAGE Conference and Exhibition 2018. Netherlands: EAGE Publications BV, 2018. http://dx.doi.org/10.3997/2214-4609.201800817.

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HUERGO, M. A. C., M. A. PASQUALE, A. E. BOLZÁN, A. J. ARVIA, and P. H. GONZÁLEZ. "PHENOMENOLOGICAL STUDY OF THE GROWTH RATE OF TRANSFORMED VERO CELLS, CHANGES IN THE GROWTH MODE AND FRACTAL BEHAVIOUR OF COLONY CONTOURS." In International Symposium on Mathematical and Computational Biology. WORLD SCIENTIFIC, 2010. http://dx.doi.org/10.1142/9789814304900_0003.

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Baxevanis, Theocharis, Dimitris Lagoudas, and Chad Landis. "Mode I Steady Crack-Growth in Superelastic Shape Memory Alloys." In ASME 2012 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/smasis2012-7934.

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A numerical analysis of quasi-static, steady state crack growth in superelastic Shape Memory Alloys (SMAs) under small-scale transformation conditions is carried out for plane strain, mode I loading. Crack growth is assumed to proceed at a critical level of the crack-tip energy release rate. Finite-element results concerning the mechanical fields near the advancing crack tip are presented and the ratio of the far-field applied energy release rate to the crack-tip energy release rate is obtained for a range of thermomechanical parameters. A substantial fracture toughening is observed associated with closure stresses placed on the crack tip by the transformed material left behind in the wake of the advancing crack tip.
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Keith, Meggan E., Erika LaPorta, and JoEllen Welsh. "Abstract A52: Mechanisms of 1,25‐dihdyroxyvitamin D‐mediated growth arrest and apoptosis in transformed mammary cells." In Abstracts: First AACR International Conference on Frontiers in Basic Cancer Research--Oct 8–11, 2009; Boston MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.fbcr09-a52.

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Kanthou, C., C. Parker, D. E. Huber, P. Stroobant, V. V. Kakkar, N. Pringle, and W. Richardson. "PLATELET-DERIVED GROWTH FACTORA-CHAIN GENE ACTIVATION AND GROWTH FACTOR PRODUCTION BY HUMAN AORTIC SMOOTH MUSCLE CELLS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643751.

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The many contributory factors leading to the development of cardiovascular disease are currently thought to induce a common pathological change involving smooth muscle cells, which migrate from the vessel wall, proliferate,accumulate at the sites of endothelial cell damage, and then secrete connective tissue proteins and lipids which contribute to the plaque which results in the occlusion of the vessel. According to the recently modified hypothesis of Ross (1), a key event in the development of atheroma may be the abnormal release of a number of growth modulatory polypeptides,including platelet-derived growth factor (PDGF), which can potentially originate from platelets, endothelial cells, monocytes or macrophages, and smooth muscle cells themselves.We have isolated smooth muscle cell lines from 25 samples of human aorta, using digestion with collagenase and elastase. With DNA synthesis and Northern blot techniques, we examined them for both the production of PDGF-like proteins, and for the possible activation of the PDGF A-chain and B-chain genes. Severallines secreted a growth factor and were stillviable after culture for 57 days in serum-free medium. Parallel experiments using Northernblot analysis revealed the activation of the PDGF A-chain gene in all lines examined with no detectable B-chain gene transcripts.These data raise the possibility that vascular damage may activate the gene encoding the A-chain of PDGF in adjacent smooth muscle cells. Such cells might then become capable ofautonomous growth, in an analogous manner tocells transformed by Simian Sarcoma Virus, whose sis oncogene encodes the B-chain of PDGF.
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Hasegawa, Kunio, Yinsheng Li, Valery Lacroix, and Bohumir Strnadel. "Re-Characterization of Subsurface Flaw to Surface Flaw Based on Equivalent Fatigue Crack Growth Rate." In ASME 2015 Pressure Vessels and Piping Conference. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/pvp2015-45946.

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A subsurface flaw located near a component surface is transformed to a surface flaw in accordance with a flaw-to-surface proximity rule. The re-characterization process from subsurface to surface flaw is adopted in all fitness-for-service (FFS) codes. However, the criteria of the re-characterizations are different among the FFS codes. In addition, the proximity factors in the rules are defined by constant values, irrespective of flaw aspect ratios. This paper describes the stress intensity factor interaction between the subsurface flaw and component free surface, and proposes a proximity factor from the point of view of fatigue crack growth rates.
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Reports on the topic "Transformed growth"

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Jagannathan, Shanti, and Dorothy Geronimo. Reaping the Benefits of Industry 4.0 through Skills Development in High-Growth Industries in Southeast Asia: Insights from Cambodia, Indonesia, the Philippines, and Viet Nam. Asian Development Bank, January 2021. http://dx.doi.org/10.22617/spr200328.

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This synthesis report explores the implications of the Fourth Industrial Revolution (4IR) on the future of the job market in Southeast Asia. It is part of the series of reports that assesses how jobs, tasks, and skills are being transformed in industries with high relevance to 4IR technologies in Cambodia, Indonesia, the Philippines, and Viet Nam. The series provides recommendations to strengthen policies, skills, and training as well as new approaches, strategies, and actions to enhance the readiness of each country’s workforce for 4IR.
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Jagannathan, Shanti, and Dorothy Geronimo. Reaping the Benefits of Industry 4.0 through Skills Development in Viet Nam. Asian Development Bank, January 2021. http://dx.doi.org/10.22617/spr200308.

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This report explores the implications of the Fourth Industrial Revolution (41R) on the future of the job market in Viet Nam. It assesses how jobs, tasks, and skills are being transformed, particularly in logistics and agro-processing. These two industries are important for the country’s employment, economic growth, and international competitiveness and are also highly relevant for 4IR technologies. The report is part of series developed from an Asian Development Bank study on trends in skills demand in Cambodia, Indonesia, the Philippines, and Viet Nam.
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Jagannathan, Shanti, and Dorothy Geronimo. Reaping the Benefits of Industry 4.0 through Skills Development in Indonesia. Asian Development Bank, January 2021. http://dx.doi.org/10.22617/spr200327.

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This report explores the implications of the Fourth Industrial Revolution (4IR) on the future of the job market in Indonesia. It assesses how jobs, tasks, and skills are being transformed in food and beverage as well as in automotive manufacturing. These two industries have high relevance to 4IR technologies and both are important to Indonesia’s national employment, economic growth, and international competitiveness. They are also likely to benefit considerably from the transformational effect of 4IR, if there is adequate investment in jobs, skills, and training. The report is part of series developed from an Asian Development Bank study on trends in skills demand in Cambodia, Indonesia, the Philippines, and Viet Nam.
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Jagannathan, Shanti, and Dorothy Geronimo. Reaping the Benefits of Industry 4.0 through Skills Development in the Philippines. Asian Development Bank, January 2021. http://dx.doi.org/10.22617/spr200326.

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This report explores the implications of the Fourth Industrial Revolution (4IR) on the future of the job market in the Philippines. It assesses how jobs, tasks, and skills are being transformed in the information technology-business process outsourcing industry and electronics manufacturing industry. These two industries have high relevance to 4IR technologies and are important to the country’s employment, growth, and international competitiveness. They are likely to benefit from the transformational effect of 4IR, if there is adequate investment on jobs, skills, and training. The report is part of series developed from an Asian Development Bank study on trends in skills demand in Cambodia, Indonesia, the Philippines, and Viet Nam.
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Nin Pratt, Alejandro, and Héctor Valdés Conroy. After the Boom: Agriculture in Latin America and the Caribbean. Inter-American Development Bank, December 2020. http://dx.doi.org/10.18235/0002955.

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The convergence of a favorable macroeconomic environment and high prices of primary commodities between 2000 and 2011 contributed to the best performance of agriculture in Latin America and the Caribbean (LAC) since the 1980s, with steady growth of total factor productivity (TFP) and output per worker and a reduction in the use of input per worker. The end of the upward phase of the commodity cycle in 2011 together with less favorable external markets and a deterioration of the policy environment in several countries, motivates us to revisit the situation of agriculture in LAC in recent years to analyze how these changes have affected its performance. This study applies a framework that uses index numbers together with data envelopment analysis (DEA) to estimate levels of productivity and efficiency, incorporating technical change together with technical (TE) and environmental efficiency (EE) into the decomposition of TFP. The EE index adjusts the TFP measure for pollution, treating GHG emissions as a by-product of the desired crop or livestock outputs. TFP and efficiency of crop and livestock sub-sectors was calculated for 24 LAC countries from 2000 to 2016. Our results show that the period of fast agricultural growth in LAC, driven by technical change and resource reallocation, transformed agriculture in the region leaving it in a better position to cope with the more unfavorable regional macroeconomic environment and the less dynamic global markets observed after 2011.
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Dinh, Long N. LiOH corrosion growth and thermal stability investigated by diffuse reflectance infrared Fourier Transform (DRIFT) spectroscopy. Office of Scientific and Technical Information (OSTI), March 2019. http://dx.doi.org/10.2172/1544959.

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Jones, Emily, Beatriz Kira, Anna Sands, and Danilo B. Garrido Alves. The UK and Digital Trade: Which way forward? Blavatnik School of Government, February 2021. http://dx.doi.org/10.35489/bsg-wp-2021/038.

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The internet and digital technologies are upending global trade. Industries and supply chains are being transformed, and the movement of data across borders is now central to the operation of the global economy. Provisions in trade agreements address many aspects of the digital economy – from cross-border data flows, to the protection of citizens’ personal data, and the regulation of the internet and new technologies like artificial intelligence and algorithmic decision-making. The UK government has identified digital trade as a priority in its Global Britain strategy and one of the main sources of economic growth to recover from the pandemic. It wants the UK to play a leading role in setting the international standards and regulations that govern the global digital economy. The regulation of digital trade is a fast-evolving and contentious issue, and the US, European Union (EU), and China have adopted different approaches. Now that the UK has left the EU, it will need to navigate across multiple and often conflicting digital realms. The UK needs to decide which policy objectives it will prioritise, how to regulate the digital economy domestically, and how best to achieve its priorities when negotiating international trade agreements. There is an urgent need to develop a robust, evidence-based approach to the UK’s digital trade strategy that takes into account the perspectives of businesses, workers, and citizens, as well as the approaches of other countries in the global economy. This working paper aims to inform UK policy debates by assessing the state of play in digital trade globally. The authors present a detailed analysis of five policy areas that are central to discussions on digital trade for the UK: cross-border data flows and privacy; internet access and content regulation; intellectual property and innovation; e-commerce (including trade facilitation and consumer protection); and taxation (customs duties on e-commerce and digital services taxes). In each of these areas the authors compare and contrast the approaches taken by the US, EU and China, discuss the public policy implications, and examine the choices facing the UK.
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Asian Development Outlook 2021 Update: Transforming Agriculture in Asia. Asian Development Bank, September 2021. http://dx.doi.org/10.22617/fls210352-3.

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This report forecasts growth in developing Asia of 7.1% in 2021 and 5.4% in 2022 in an uneven recovery caused by divergent growth paths. Its theme chapter explores sustainable agriculture. Growth forecasts are revised up for East Asia and Central Asia from the projections made in April, but down for South Asia, Southeast Asia, and the Pacific. This reflects differences in vaccination progress and control of domestic COVID-19 outbreaks but also other factors, including rising commodity prices and depressed tourism. Inflation is expected to remain under control. The main risks to the economic outlook come from the COVID-19 pandemic, including the emergence of new variants, slower-than-expected vaccine rollouts, and waning vaccine effectiveness. Sustainable food production and agricultural systems that are resilient to climate change will be crucial for developing Asia. To transform agriculture in the region, its economies must tackle challenges from changing consumer demand, changing demographics, and a changing and more fragile environment.
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