Relevant bibliographies by topics / Transgenic mice. Mice

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Transgenic mice. Mice'

Author: Grafiati
Published: 4 June 2021
Last updated: 17 February 2022

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Journal articles on the topic "Transgenic mice. Mice"

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Ju Kim, H., K. i. Naruse, W. S. Choi, K. S. Im, C. S. Park, and D. I. Jin. "332 ENHANCEMENT OF GROWTH PERFORMANCE IN DOUBLE TRANSGENIC MICE WITH GROWTH HORMONE RECEPTOR AND IGF-1 RECEPTOR GENES." Reproduction, Fertility and Development 17, no. 2 (2005): 317. http://dx.doi.org/10.1071/rdv17n2ab332.

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The effect of amplifying growth-related receptor signaling, through overexpression of receptors, on growth regulation in animals was examined. Transgenic mice lines were produced by DNA microinjection using the metallothionein promoter ligated to either the growth hormone receptor (GHR) or IGF-1 receptor (IGF-1R) genes (3 GHR founders and 3 IGF-1R founders). Transgenic mouse lines were estimated to contain approximately 4 to 20 copies of transgenes per cell by Southern blot analysis. Founder mice of each transgenic line transmitted transgenes into F1 and F2 pups with Mendelian ratio. Double tr
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Babinet, C., D. Morello, and J. P. Renard. "Transgenic mice." Genome 31, no. 2 (1989): 938–49. http://dx.doi.org/10.1139/g89-165.

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Stable integration into the mouse genome of exogenous genetic information has become, over the past few years, a very potent approach for different aspects of biology. It is a common feature that the integrated exogenous gene (the transgene) is expressed properly both spatially and temporally. Constructing different lines of transgenic mice carrying various versions of a gene, therefore, permits cis acting DNA sequences involved in the specificity of expression to be defined, in the context of the developing animal. This in turn opens the way to a variety of experiments in which a given gene p
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Auerbach, Anna B. "Production of functional transgenic mice by DNA pronuclear microinjection." Acta Biochimica Polonica 51, no. 1 (2004): 9–31. http://dx.doi.org/10.18388/abp.2004_3593.

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Successful experiments involving the production of transgenic mice by pronuclear microinjection are currently limited by low efficiency of random transgene integration into the mouse genome. Furthermore, not all transgenic mice express integrated transgenes, or in other words are effective as functional transgenic mice expressing the desired product of the transgene, thus allowing accomplishment of the ultimate experimental goal--in vivo analysis of the function of the gene or gene network. The purpose of this review is to look at the current state of transgenic technology, utilizing a pronucl
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Hickman-Davis, Judy M., and Ian C. Davis. "Transgenic mice." Paediatric Respiratory Reviews 7, no. 1 (2006): 49–53. http://dx.doi.org/10.1016/j.prrv.2005.09.005.

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PALMITER, R., and R. BRINSTER. "Transgenic mice." Cell 41, no. 2 (1985): 343–45. http://dx.doi.org/10.1016/s0092-8674(85)80004-0.

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Friedman, Rick A., and Allen F. Ryan. "Transgenic Mice." Otolaryngologic Clinics of North America 25, no. 5 (1992): 1017–26. http://dx.doi.org/10.1016/s0030-6665(20)30922-1.

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Westphal, Heiner. "Transgenic mice." BioEssays 6, no. 2 (1987): 73–76. http://dx.doi.org/10.1002/bies.950060208.

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BABINET, CHARLES. "Transgenic Mice." Journal of the American Society of Nephrology 11, suppl 2 (2000): S88—S94. http://dx.doi.org/10.1681/asn.v11suppl_2s88.

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Abstract. Stable integration into the mouse genome of exogenous genetic information, i.e., the creation of transgenic mice, has become a privileged way of analyzing gene function in normal development and pathology. Both gene addition and gene replacement may be performed. This has allowed, in particular, the creation of mice in which precise mutations are introduced into a given gene. Furthermore, in recent years, strategies that induce the expression of a mutation in a given type of cell and/or at a given time in development have been developed. Thus, the transgenic methodology affords a uni
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Dent, L. A., M. Strath, A. L. Mellor, and C. J. Sanderson. "Eosinophilia in transgenic mice expressing interleukin 5." Journal of Experimental Medicine 172, no. 5 (1990): 1425–31. http://dx.doi.org/10.1084/jem.172.5.1425.

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Experiments in vitro suggest that although interleukin 5 (IL-5) stimulates the late stages of eosinophil differentiation, other cytokines are required for the generation of eosinophil progenitor cells. In this study transgenic mice constitutively expressing the IL-5 gene were established using a genomic fragment of the IL-5 gene coupled to the dominant control region from the gene encoding human CD2. Four independent eosinophilic transgenic lines have thus far been established, two of which with 8 and 49 transgene copies, are described in detail. These mice appeared macroscopically normal apar
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Sigmund, C. D., C. A. Jones, H. J. Jacob, et al. "Pathophysiology of vascular smooth muscle in renin promoter-T-antigen transgenic mice." American Journal of Physiology-Renal Physiology 260, no. 2 (1991): F249—F257. http://dx.doi.org/10.1152/ajprenal.1991.260.2.f249.

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The pathophysiological consequence of targeted production of SV-40 T-antigen to renin-expressing cells in the kidney of transgenic mice is reported. A histopathologic analysis of the kidney from adult transgenic mice (12–16 wk old) revealed the presence of severe vascular lesions manifested by marked atypical hyperplasia of vascular smooth muscle. The levels of plasma renin, kidney renin, and kidney renin mRNA were examined in 6- and 9-wk-old transgenic mice and were found to be significantly lower than their age-matched non-transgenic littermates and were nonresponsive to captopril treatment.
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Dissertations / Theses on the topic "Transgenic mice. Mice"

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Simard, Marie-Chantal. "Nef pathogenesis in transgenic mice." Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=103182.

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In order to study the functions of SIV Nef in vivo, in a small animal model, transgenic (Tg) mice expressing the SIVmac239 nef gene, under the control of the human CD4 gene promoter (CD4C) were generated. The transgene was found to be expressed in the same cells targeted by the virus, in vivo. These CD4C/SHIV-nef SIV Tg mice develop a severe AIDS-like disease, including premature death, failure to thrive/weight loss, wasting, thymic atrophy, exhibit an especially low number of peripheral CD8+ T cells as well as low number of peripheral CD4+ T cells, diarrhea, splenomegaly, kidney (interstitial
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Husbands, Sandra D. "Tolerance and immunity in transgenic mice." Thesis, University College London (University of London), 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.303680.

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Oghumu, Steve Onyeka. "Generation and Characterization of CXCR3 Bicistronic Reporter Mice and CXCR3 Transgenic Mice." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1274927351.

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Malmström, Vivianne. "Arthritis susceptibility and tolerance in collagen transgenic mice." Lund : Dept. of Cell and Molecular Biology, Section for Medical Inflammation Research, Lund University, 1997. http://catalog.hathitrust.org/api/volumes/oclc/38986502.html.

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Gratao, Ana Angelica. "Impaired Fertility in Transgenic Mice Overexpressing Betacellulin." Diss., lmu, 2007. http://nbn-resolving.de/urn:nbn:de:bvb:19-65751.

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Gratao, Ana Angélica. "Impaired fertility in transgenic mice overexpressing betacellulin." [S.l.] : [s.n.], 2007. http://edoc.ub.uni-muenchen.de/archive/00006575.

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Felmer, R. "Genetic manipulation of fat in transgenic mice." Thesis, University of Edinburgh, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.650832.

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The present dissertation describes the use of a novel system to achieve specific cell ablation in fat tissue. The method is based on the use of <i>E.coli</i> nitroreductase (NTR) enzyme that activates certain nitro compounds into cytotoxic DNA interstrand cross-linking agents. This system was assessed first <i>in vitro,</i> in a preadipocyte cell line (3T3L1). Clones of cells that expressed NTR were successfully killed after treatment with CB1954. It was confirmed that the mechanism of cell killing involved is apoptosis and the presence of a cell-permeable metabolite that is released to the me
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Cox, April. "Effects of hyperoxia in alzheimers transgenic mice." Scholar Commons, 2005. http://scholarcommons.usf.edu/etd/2836.

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An association between major surgery in the elderly and precipitation of Alzheimers disease (AD) has been reported. Hyperoxia (100%) oxygen is commonly administered after surgery to increase the oxygen content of blood. However, hyperoxia is a potent cerebral vasoconstrictor and generator of free radicals, as is [beta]amyloid (A[beta];). This study was aimed at examining behavioral, neuropathological, and neurochemical effects of hyperoxia treatments in APPsw transgenic mice (Tg+), which have elevated brain A[beta]; levels by 3-4 months of age but are not yet cognitively-impaired. At 3 months
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Derrett-Smith, E. C. "Systemic sclerosis vasculopathy : exploration in transgenic mice." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1383812/.

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Vascular disease in systemic sclerosis (SSc) leads to significant morbidity and mortality. No robust animal model of vasculopathy in SSc has been described. The hypothesis underpinning work described in this thesis is that a primary defect in TGFβ signalling is sufficient to generate the fibrotic, vascular and inflammatory phenotype of this condition. This is explored using a novel transgenic mouse model of SSc (TβRIIΔk-fib). The transgenic mouse strain TβRIIΔk-fib carries a kinase-deficient type II TGFβ receptor which is expressed under the control of a fibroblast specific promoter leading to
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Calver, Andrew Robert. "Oligodendrocyte population dynamics : insights from transgenic mice." Thesis, University College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322239.

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Books on the topic "Transgenic mice. Mice"

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Tinsley, Jonathon Mark. Human papillomavirus transgenic mice. University of Birmingham, 1991.

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Egorov, I. K. Transgenic Mice and Mutants in MHC Research. Springer Berlin Heidelberg, 1990.

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Egorov, Igor K., and Chella S. David, eds. Transgenic Mice and Mutants in MHC Research. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-75442-5.

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Transgenic mouse methods and protocols. 2nd ed. Humana Press, 2011.

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Silver, Lee M. Mouse genetics: Concepts and applications. Oxford University Press, 1995.

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Haddad, George E. Expression of HLA class II genes in transgenic mice. National Library of Canada, 1994.

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Herrup, Karl. Transgenic and ES cell chimeric mice as tools for the study of the nervous system. Published by Elsevier for the Foundation for the Study of the Nervous System, 1995.

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Ferrick, David A. Transgenic mice as a in vivo model for self reactivity. R.G. Landes Co., 1994.

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What's wrong with my mouse?: Behavioral phenotyping of transgenic and knockout mice. 2nd ed. Wiley-Interscience, 2007.

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Gingrich, Jeffrey R. Characterization of a neural-specific inducible genetic system in transgenic mice. National Library of Canada = Bibliothèque nationale du Canada, 1999.

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Book chapters on the topic "Transgenic mice. Mice"

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Lo, Lilian H., and Vincent W. Keng. "Transgenic Mice." In Encyclopedia of Gerontology and Population Aging. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-69892-2_967-1.

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Chen, X., Y. Matsuura, and J. F. Kearney. "CD5 Transgenic Mice." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-642-79275-5_25.

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Diack, Abigail B., Rona Wilson, Enrico Cancellotti, Barry Bradford, Matthew Bishop, and Jean C. Manson. "Transgenic Mice Modelling." In Prions and Diseases. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-5338-3_10.

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Lee, Myung-Shik, and Nora Sarvetnick. "IL-10 Transgenic Mice." In Interleukin-10. Springer Berlin Heidelberg, 1995. http://dx.doi.org/10.1007/978-3-662-22038-2_15.

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Hanahan, Douglas. "Oncogenesis in Transgenic Mice." In Oncogenes and Growth Control. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-73325-3_48.

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Scherrmann, Jean-Michel, Kim Wolff, Christine A. Franco, et al. "Ataxin-3 Transgenic Mice." In Encyclopedia of Psychopharmacology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_659.

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Götz, Jürgen, Markus Tolnay, Robi Barmettler, et al. "Human Tau Transgenic Mice." In Advances in Experimental Medicine and Biology. Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1249-3_6.

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Wagner, E. F. "Oncogenes and Transgenic Mice." In Growth Factors, Differentiation Factors, and Cytokines. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74856-1_27.

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Jaenisch, Rudolf, Douglas Gray, Tetsuo Noda, and Hans Weiher. "Mutations in Transgenic Mice." In Vectors as Tools for the Study of Normal and Abnormal Growth and Differentiation. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74197-5_6.

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Feng, Guoping, Jing Lu, and Jimmy Gross. "Generation of Transgenic Mice." In Pain Research. Humana Press, 2004. http://dx.doi.org/10.1385/1-59259-770-x:155.

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Conference papers on the topic "Transgenic mice. Mice"

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Keyes, Joseph T., Stacy Borowicz, Urs Utzinger, Mohamad Azhar, and Jonathan P. Vande Geest. "Quantification of the Biomechanical Differences in Wild-Type and Heterozygous TGF Beta2 Knockout Mice." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19482.

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The use of transgenic mice is an incredibly powerful tool in understanding the underlying etiology of disease. To understand the usefulness of specific transgenic mice, the systems of interest should be characterized. We have created TGFβ2-deficient mouse fetuses that develop widespread aortic and coronary artery aneurysms [1]. Several studies have pointed to a strong connection between elevated TGFβ signaling and aortic aneurysm [2]. In situ hybridization has shown that Tgfb2 and Tgfb3 are major ligands expressed in the aortic medial wall. Further reduction of TGFβ signaling by combining TGFβ
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Welsh, John P., Josef Turecek, and Eric E. Turner. "Evaluating cerebellar functions using optogenetic transgenic mice." In SPIE BiOS, edited by Samarendra K. Mohanty and Nitish V. Thakor. SPIE, 2013. http://dx.doi.org/10.1117/12.2010204.

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Kamioka, Yuji, Kenta Sumiyama, Rei Mizuno, and Michiyuki Matsuda. "Live imaging of transgenic mice expressing FRET biosensors." In 2013 35th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2013. http://dx.doi.org/10.1109/embc.2013.6609453.

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Sakai, Mizu, Tetsuya Kubota, Mayuka Isaka, et al. "Lung Injury Model Using Human MUC1 Transgenic Mice." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a5987.

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Laufer, J. G., J. O. Cleary, E. Z. Zhang, M. F. Lythgoe, and P. C. Beard. "Photoacoustic imaging of vascular networks in transgenic mice." In BiOS, edited by Alexander A. Oraevsky and Lihong V. Wang. SPIE, 2010. http://dx.doi.org/10.1117/12.842204.

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Liu, C., M. Kronenberg, X. Jiang, D. Rowe, and M. Hadjiargyrou. "Characterization of Mustn1PRO-GFPtpz transgenic mice." In 2007 IEEE 33rd Annual Northeast Bioengineering Conference. IEEE, 2007. http://dx.doi.org/10.1109/nebc.2007.4413256.

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Luo, Xue-Gang, Ting-Ting Qin, Zhong-Shuai Xin, Dao-Zhu Huangfu, and Tao Xi. "Construction of SMYD3 liver-specific expression transgene for the eestablishment of transgenic mice." In 2010 International Conference on Bioinformatics and Biomedical Technology. IEEE, 2010. http://dx.doi.org/10.1109/icbbt.2010.5478970.

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Irungbam, K., M. Roderfeld, Y. Churin, et al. "Abcb4-knockout reduces hepatic lipid steatosis in HBs transgenic mice." In 36. Jahrestagung der Deutschen Arbeitsgemeinschaft zum Studium der Leber. Georg Thieme Verlag KG, 2020. http://dx.doi.org/10.1055/s-0039-3402182.

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Gibson, Katelin A., Merit L. Goodman, and Christy R. Hagan. "Abstract 5228: Mammary gland tumors in progesterone receptor transgenic mice." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-5228.

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Ooi, Chen Yen, and Naomi C. Chesler. "The Role of Collagen in Pulmonary Hypertension-Induced Large Artery Stiffening." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-192951.

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Hypoxic pulmonary hypertension (HPH) leads to stiffening of large pulmonary arteries, which affects right ventricular afterload. We hypothesized that vascular collagen accumulation is the major cause of large pulmonary artery (PA) stiffening in HPH. We tested this hypothesis with transgenic mice that produce collagen type I resistant to degradation (Col1a1R/R) and wild type littermate controls (Col1a1+/+) exposed to hypoxia and allowed to recover. Pressure-diameter testing on left PAs demonstrated that stiffness in control mice increased with hypoxia and decreased with recovery (p &lt; 0.05).
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Reports on the topic "Transgenic mice. Mice"

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Segall, Jeffrey. Analysis of Metastasis in Transgenic Mice. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada384861.

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Segall, Jeffrey E. Analysis of Metastasis in Transgenic Mice. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada403427.

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Kast, W. M. Prostate Cancer Immunotherapy Development in Prostate Specific Antigen Transgenic Mice. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada395836.

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Shankar, Deepa. Analysis of Multistep Mammary Tumorigenesis in WNT-1 Transgenic Mice. Defense Technical Information Center, 1995. http://dx.doi.org/10.21236/ada302270.

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Windle, Jolene J. Mechanisms of Ras Control of Mammary Tumor Properties in Transgenic Mice. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada403384.

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Windle, Jolene J. Mechanisms of Ras Control of Mammary Tumor Properties in Transgenic Mice. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada392893.

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Mao, Muling. Development and Characterization of Transgenic Mice with Mammary Gland Specific Expression of the Tumor Suppressor. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada404605.

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Cadieux, Chantal. Mammary Gland Tumor Development in Transgenic Mice Overexpressing Different Isoforms of the CDP/Cux Transcription Factor. Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada506317.

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Cadieux, Chantal. Mammary Gland Tumor Development in Transgenic Mice Overexpressing Different Isoforms of the CDP/Cux Transcription Factor. Defense Technical Information Center, 2007. http://dx.doi.org/10.21236/ada469226.

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Cuevas, Bruce. Training Entitled Development and Characterization of Transgenic Mice With Mammary Gland Specific Expression of the Tumor Suppressor. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada390697.

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