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Dissertations / Theses on the topic 'Transgenic muscle'

Author: Grafiati

Published: 4 June 2021

Last updated: 16 February 2022

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1

Ning, Jie. "Estrogen receptor [alpha] and [beta] knock-out effects on skeletal muscle in mature female and male mice, and aromatase knock-out effects on skeletal muscle in mature male mice." Diss., Columbia, Mo. : University of Missouri-Columbia, 2007. http://hdl.handle.net/10355/6273.

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Thesis (M.S.)--University of Missouri-Columbia, 2007.<br>"August 2007" The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Vita. Includes bibliographical references.

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2

Chen, Paula Renee. "Muscle Fiber Hyperplasia in Leg Muscle of Transgenic Quail Overexpressing anAlternative Splicing Variant of Myostatin." The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1462207424.

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3

Sjuve, Rolf. "Function of contractile and cytoskeletal proteins in smooth muscle effects of hypertrophy and age and of desmin removal in a transgenic animal /." Lund : Dept. of Physiology and Neuroscience, Lund University, 1998. http://books.google.com/books?id=ccFqAAAAMAAJ.

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4

Chalothorn, Dan. "Cellular trafficking properties and physiological functions of the [alpha]1-adrenoceptor subtypes." Lexington, Ky. : [University of Kentucky Libraries], 2003. http://lib.uky.edu/ETD/ukypham2003d00083/Chalothorn.pdf.

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Thesis (Ph. D.)--University of Kentucky, 2003.<br>Title from document title page. Document formatted into pages; contains x, 192p. : ill. Includes abstract. Includes bibliographical references (p. 165-189).

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5

Sonner, Martha Jean. "Investigating Anatomical and Molecular Aspects of Proprioceptive Sensory Neuron Diversity Using a Transgenic Mouse Model." Wright State University / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=wright1420817202.

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6

Bogomolovas, Julius, Jennifer R. Fleming, Brian R. Anderson, et al. "Exploration of pathomechanisms triggered by a single-nucleotide polymorphism in titin's I-band: the cardiomyopathy-linked mutation T2580I." ROYAL SOC, 2016. http://hdl.handle.net/10150/621990.

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Missense single-nucleotide polymorphisms (mSNPs) in titin are emerging as a main causative factor of heart failure. However, distinguishing between benign and disease-causing mSNPs is a substantial challenge. Here, we research the question of whether a single mSNP in a generic domain of titin can affect heart function as a whole and, if so, how. For this, we studied the mSNP T2850I, seemingly linked to arrhythmogenic right ventricular cardiomyopathy (ARVC). We used structural biology, computational simulations and transgenic muscle in vivo methods to track the effect of the mutation from the m

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7

Brittsan, Angela Gail. "TRANSGENIC APPROACHES TO ELUCIDATE THE ROLE OF PHOSPHOLAMBAN IN BASAL CONTRACTILITY AND DURING BETA-ADRENERGIC STIMULATION OF THE HEART." University of Cincinnati / OhioLINK, 2000. http://rave.ohiolink.edu/etdc/view?acc_num=ucin960908353.

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8

Sanford, Jamie Lynn. "Analysis of the cell junction proteins CASK and claudin-5 in skeletal and cardiac muscle." Connect to this title online, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1117553681.

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Thesis (Ph. D.)--Ohio State University, 2005.<br>Title from first page of PDF file. Document formatted into pages; contains xv, 188 p.; also includes graphics (some col.) Includes bibliographical references (p. 166-188). Available online via OhioLINK's ETD Center

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9

Pritchard, Tracy J. "Expression and function of the Na+-K +ATPase a-isoforms in smooth muscle: evidence from transgenic mice /." Cincinnati, Ohio : University of Cincinnati, 2007. http://www.ohiolink.edu/etd/view.cgi?ucin1186672962.

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Thesis (Ph. D. )--University of Cincinnati, 2007.<br>Advisor: Dr. Richard J. Paul Title from electronic thesis title page (viewed Nov. 23, 2007). Includes abstract. Keywords: transgenic mice; hypertension; vascular smooth muscle; Na+-Ca2+ exchanger Includes bibliographical references.

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10

Tanaka, Tomohiro. "Skeletal muscle AMP-activated protein kinase phosphorylation parallels metabolic phenotype in leptin transgenic mice under dietary modification." Kyoto University, 2006. http://hdl.handle.net/2433/143868.

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11

PRITCHARD, TRACY J. "Expression and Function of the Na ⁺-K ⁺ATPase α-Isoforms in Smooth Muscle: Evidence from Transgenic Mice". University of Cincinnati / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1186672962.

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12

Lindsey, Madison L. "The Impact of FoxO1 Overexpression on the Regulation of CD36 in Skeletal Muscle." University of Toledo / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1525425389232742.

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13

Latvanlehto, A. (Anne). "Type XIII collagen:organization of the mouse gene, generation of three genetically engineered mouse lines by homologous recombination, and biochemical studies on the molecular properties of the type XIII collagen protein." Doctoral thesis, University of Oulu, 2004. http://urn.fi/urn:isbn:9514275934.

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Abstract Genomic clones covering the entire mouse type XIII collagen gene (Col13a1) were isolated, and the complete exon-intron organization was characterized. The gene was found to be about 135 kb in size and to locate in the mouse chromosome 10. Comparison of gene structures and promoter regions between man and mouse indicated high conservation between the two species. In order to understand the biological function of type XIII collagen, a mouse line that expresses type XIII collagen with replacement of the cytosolic and transmembrane domains by a short, non-descript sequence was generated

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14

Dimoulas, Peter Michael. "The fast-start and sprinting ability, and the effects of growth hormone (GH) upregulation on the muscle functioning of GH-transgenic coho salmon." Thesis, University of British Columbia, 2009. http://hdl.handle.net/2429/7321.

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If GH-transgenic coho salmon escaped into the natural environment would their performance during predator-prey encounters or spawning migrations be different from their wild conspecifics? We examined fast-start and sprinting performance to infer their prospective ability to evade predatory strikes and chasing predators, to chase prey, and to complete spawning migrations. Similarly, we addressed the effects of GH upregulation on muscle functioning. Fast-starts are rapid escape events, and are a summation of short-term behavior, white muscle intrinsic properties, musculoskeletal linking, and

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15

Chalothorn, Dan. "CELLULAR TRAFFICKING PROPERTIES AND PHYSIOLOGICAL FUNCTIONS OF THE á1-ADRENOCEPTOR SUBTYPES." UKnowledge, 2003. http://uknowledge.uky.edu/gradschool_diss/409.

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The 1-adrenoceptors (1-ARs) serve as an interface between the sympathetic nervous system and the cardiovascular system where they are mediators of systemic arterial blood pressure, initiators of positive inotropy, and regulators of cellular growth responses. There are three subtypes: 1A-, 1B-, and 1D-ARs. This dissertation research investigated the trafficking properties of the 1-ARs at the cellular level as well as physiological relevance of the 1-ARs at the tissue level. In vitro studies using transiently transfected 1-AR/GFP subtypes revealed distinct basal localization patterns and differe

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16

Eklund, L. (Lauri). "Genetic studies of collagen types XV and XVIII:type XV collagen deficiency in mice results in skeletal myopathy and cardiovascular defects, while the homologous endostatin precursor type XVIII collagen is needed for normal development of the eye." Doctoral thesis, University of Oulu, 2001. http://urn.fi/urn:isbn:9514265793.

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Abstract Overlapping genomic clones coding for the α1 chain of mouse type XV collagen (Col15a1) were isolated. The gene was found to be 110 kb in length and to contain 40 exons. Analysis of the proximal 5'-flanking region showed properties characteristic of a housekeeping gene promoter, and functional analysis identified cis-acting elements for both positive and negative regulation of Col15a1 gene expression. The general exon-intron pattern of the mouse Col15a1 gene was found to be highly similar to that of its human homologue, and comparison of 5'-flanking sequences indicated four co

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17

Sokolow, Sophie. "Les souris déficientes pour les échangeurs sodium-calcium (NCX1 et NCX3): deux modèles murins pour l'étude de leurs rôles pysiologiques in vivo ;Implication de NCX3 dans la fonction neuromusculaire." Doctoral thesis, Universite Libre de Bruxelles, 2004. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/211196.

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Nous avons généré des souris déficientes pour les gènes codant pour les échangeurs Na/Ca de type I (NCX1) et de type III (NCX3) afin d'étudier, in vivo, le rôle de ces deux protéines.<p>L‘analyse phénotypique des souris adultes totalement déficientes pour le gène Ncx1 (Ncx1-/-) n'a pu être menée étant donné que ces souris décèdent au cours du développement embryonnaire.<p>Les souris déficientes pour le gène Ncx3 (Ncx3-/-) sont viables et fertiles. Nous avons analysé l'effet de l'inactivation du gène Ncx3 dans le muscle squelettique et plus particulièrement au niveau de la jonction neuromuscula

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18

Zhao, Guisheng. "PARACRINE/AUTOCRINE ACTIONS OF INSULIN-LIKE GROWTH FACTOR I (IGF-I) IN TRANSGENIC MICE: EFFECTS OF IGF-I IN BONE AND SMOOTH MUSCLE CELLS IN VIVO." Cincinnati, Ohio : University of Cincinnati, 2001. http://www.ohiolink.edu/etd/view.cgi?ucin983477992.

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19

O'Leary, Debra Alison. "Characterisation of gene structure and function of the ETS transcription factor Gabpα in mouse". Monash University, Centre for Functional Genomics and Human Disease, 2003. http://arrow.monash.edu.au/hdl/1959.1/9445.

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20

Hazama, Masaaki. "Different regulatory sequences are required for parvalbumin gene expression in skeletal muscles and neuronal cells of transgenic mice." Kyoto University, 2002. http://hdl.handle.net/2433/149726.

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21

Jonuschies, J. "Characterisation of lentiviral transgene expression in muscle precursor cells : towards a potential therapy for Duchenne Muscular Dystrophy." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1362850/.

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Duchenne Muscular Dystrophy is an X-linked genetic disorder characterised by progressive muscle degeneration due to the absence of functional dystrophin protein. Damaged muscle fibres are initially regenerated by satellite cells, the principal muscle-resident stem cells, which give rise to committed progenitor cells that differentiate and fuse with the damaged muscle fibre to introduce new functional myonuclei. Satellite cells also self-renew to replenish the stem cell pool. Autologous transplantation of genetically-corrected satellite cells presents an attractive strategy to introduce a funct

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22

Miner, Jeffrey H. "Factors regulating skeletal muscle development : cell culture and transgenic mouse studies." Thesis, 1991. https://thesis.library.caltech.edu/2918/1/Miner_jh_1991.pdf.

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The final steps of vertebrate skeletal muscle development involve withdrawal of determined muscle precursors from the cell cycle, expression of muscle-specific genes encoding myofibril components, and cell fusion to form terminally differentiated multinucleated myotubes. A great deal of this process has been adapted to and studied in cell culture for decades. While much has been learned about how a cell's environment influences differentiation decisions and what those decisions involve, only in the last few years has progress been made in understanding how the observed drastic changes in gene

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23

Hsiao, Chung-Der, and 蕭崇德. "Establishment and Application of Zebrafish Transgenic Lines with Skeletal-Muscle Specific or Germ-Cell Predominant Expression of Transgenes." Thesis, 2002. http://ndltd.ncl.edu.tw/handle/31318466294335771109.

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博士<br>國立臺灣大學<br>漁業科學研究所<br>91<br>Mosaic expression of transgenes in the F0 generation severely hinders the study of transient expression in transgenic fish. Although many approaches have been tried to overcome these limitations, they were only marginally successful at reducing mosaicism in the F0 generation. Thus, we need a simple and effective method for enhancing uniform expression of transgenes in the F0 generation, and preventing the silencing and unstable transmission of transgenes in subsequent generations of transgenic fish. Inclusion of inverted terminal repeats (ITRs) from adeno-assoc

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24

Suzuki, Shana T. N. "Effects of enhanced muscle growth by myostatin propeptide trangene and dietary fat content on gene expression of adiponectin, adiponectin receptors, PPAR-α and PPAR-γ". Thesis, 2007. http://hdl.handle.net/10125/20774.

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25

Chin, Hui-Yen, and 陳韋燕. "Functional Studies of Transgenic Zebrafish With Giant Grouper's Growth Hormone Specific Expression In Muscle." Thesis, 2012. http://ndltd.ncl.edu.tw/handle/08218691888654651099.

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碩士<br>國立臺灣海洋大學<br>水產養殖學系<br>100<br>Muscle-specific transgenic zebrafish over expressing giant grouper’s growth hormone was established to study the autocrine and paracrine effect of this hormone in vertebrate system. Growth enhancement was observed in the transgenic zebrafish analyzed by phenotype and gene expression analysis. The transgenic zebrafish showed an increase in body weight up to 92% and total body length up to 31% as compared to control. In addition, hypertrophy was observed in the muscle myofibers with an increase from 39-76% in the myofiber area of 3-month old transgenic zebrafi

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26

Liao, Chia-Hsuan, and 廖嘉瑄. "Molecular Mechanisms Study of Myocyte Hypertrophy in Transgenic Zebrafish with Muscle-specific Overexpression Progranulin Gene." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/95086138860018719576.

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碩士<br>臺灣大學<br>微生物與生化學研究所<br>95<br>Progranulin (Pgrn) was considered as a family of epithelial tissue growth factor because of the potential functions displayed in the regulation of development, wound healing and progression of several cancer types. In our previous study, we found hepatic pgrn and IGF-1 was co-induced with growth hormone (GH) administration. As well known that GH-IGF1 axis plays a critical role in regulating of somatic growth and the majority of fish body is muscle tissue. Therefore, we attempt to prove the function of pgrn in zebrafish by muscle-specific over-expression approa

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Liao, Chia-Hsuan. "Molecular Mechanisms Study of Myocyte Hypertrophy in Transgenic Zebrafish with Muscle-specific Overexpression Progranulin Gene." 2007. http://www.cetd.com.tw/ec/thesisdetail.aspx?etdun=U0001-2807200714500000.

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28

Huang, Shih-Chin, and 黃士晉. "Establishment of transgenic fluorescent fish expressing novel Taiwan Acropora coral fluorescent proteins by teleost muscle-specific promoter." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/42628840754249314760.

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碩士<br>國立臺灣海洋大學<br>水產養殖學系<br>98<br>Transgenic zebrafish lines strongly expressing Taiwan Acropora coral pink fluorescent protein TcFP-11 and cyan fluorescent protein TcFP-13 were established, respectively under the control of a novel zebrafish muscle-specific ckmb promoter/enhancer by high efficient Tol2 transposon system. Firstly, the transgenic wild-type and white zebrafish lines expressing Taiwan Acropora coral pink fluorescent protein TcFP-11 and cyan fluorescent protein TcFP-13 were established and stably inherited to F4 offspring, respectively by well-known zebrafish mylz2 promoter. It pr

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29

Chen, Yun-Qu, and 陳韻曲. "Establishing transgenic zebrafish models, which overexpress spermine oxidase in skeletal muscle and brain, to study the effects of polyamine catabolism in muscle hypertrophy and the hypoxic oxidative stress of fish." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/ntv292.

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碩士<br>國立臺灣海洋大學<br>生命科學暨生物科技學系<br>106<br>Spermine oxidase (SMO), which catalyzed oxidative catabolism of spermine and also produce reactive oxygen species (ROS). According to previous studies in mice, SMO plays important physiological role in skeletal muscle fiber differentiation and pathological role in brain damage under hypoxia stress. However, the function of SMO and polyamines oxidative catabolism in fish has never been reported. Here, we used molecular genetic approach to clarify the relationship between the expression level of SMO and the hypoxia tolerance in zebrafish. By using Tol2 tra

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30

Iou-Cheng and 張佑誠. "Identification of the differentially expressed genes and proteins in the muscle and testis of transgenic mice containing untranslated CAG trinucleotide repeats expansion." Thesis, 2005. http://ndltd.ncl.edu.tw/handle/88093418826894288888.

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碩士<br>中山醫學大學<br>醫學研究所<br>93<br>Among human trinucleotide repeat disorders, the most frequent triplets found to be expanded are CAG and its complementary sequence, CTG. CAG repeats are almost always found on coding regions whereas CTG expansions are located in untranslated regions (UTRs). Previously we made transgenic mice expressing a muscle-specific transcript with (CAG)200 inserted in the 3’-UTR of EGFP gene. Mice express expanded CAG repeat exhibited abnormal muscle cell morphology, low muscle activity and decreased reproduction ability. In this study, we investigated the effects of CAGn ex

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31

Fernando, Shannon M. "Myocyte Androgen Receptor Modulates Body Composition and Metabolic Parameters." Thesis, 2010. http://hdl.handle.net/1807/25577.

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Androgens (such as testosterone) have been shown to increase lean body mass and reduce fat body mass in men through activation of androgen receptors (AR). While this suggests a potential clinical use for androgens, attempts at utilization of this class of hormones as a therapeutic are limited by side effects due to indiscriminate AR activation in various tissues. Thus, a greater understanding of the tissues and cells involved in promoting these changes would be beneficial. Here we show that selective overexpression of AR in muscle cells of transgenic (HSA-AR) rodents both increases lean muscle

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