Relevant bibliographies by topics / Transient Ischemic Dilation

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  1. Journal articles
  2. Dissertations / Theses
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  4. Conference papers

Academic literature on the topic 'Transient Ischemic Dilation'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Transient Ischemic Dilation"

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Hardebeck, Charles J. "Transient ischemic dilation." Journal of the American College of Cardiology 44, no. 1 (July 2004): 211–12. http://dx.doi.org/10.1016/j.jacc.2004.04.003.

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Petretta, Mario, Wanda Acampa, Stefania Daniele, Maria Piera Petretta, Monica Plaitano, and Alberto Cuocolo. "Transient Ischemic Dilation in Patients With Diabetes Mellitus." Circulation: Cardiovascular Imaging 6, no. 6 (November 2013): 908–15. http://dx.doi.org/10.1161/circimaging.113.000497.

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FISHER, T. "Transient ischemic dilation identifies risk of subsequent cardiac events." Journal of Nuclear Cardiology 4, no. 1 (February 1997): S106. http://dx.doi.org/10.1016/s1071-3581(97)91535-x.

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Lisko, John Cyril, Nicola Nicoloff, Brandon Mikolich, Amitha Dhingra, and J. Ronald Mikolich. "TRANSIENT ISCHEMIC DILATION: A NEW PERSPECTIVE UTILIZING CARDIAC MRI." Journal of the American College of Cardiology 65, no. 10 (March 2015): A1316. http://dx.doi.org/10.1016/s0735-1097(15)61316-x.

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Gorshkova, O. P., M. V. Lensman, A. I. Artem'eva, and D. P. Dvoretsky. "Dynamics of pial vessels reactivity after brief cerebral ischemia." Regional blood circulation and microcirculation 14, no. 1 (March 30, 2015): 74–78. http://dx.doi.org/10.24884/1682-6655-2015-14-1-74-78.

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Cerebral blood vessel reactivity is one of the main determinants of final outcome of brain ischemia. Most of studies on the vascular mechanisms of ischemic brain injury, however, focus on the acute changes within ischemic period or several hours after it. Dilatatory capacity of cerebral arterioles (perfusion reservoir) is considered as an important factor of brain perfusion elevation in critical situations.The aim of the present study was to examine the pial vessel reactivity in response to hypercapnia in rats, subjected to transient global cerebral ischemia, at 7, 14 and 21 days after ischemia. Materials and methods. Transient global cerebral ischemia was induced in anesthetized Wistar rats by bilateral common carotid artery occlusion for 12 min with simultaneous controlled hypotension to 45±3 mm Hg, followed by blood reinfusion and recovery from anesthesia. Three different groups of rats were re-anesthetized at 7, 14 or 21 days after ischemia and subjected to microvascular reactivity studies using in vivo video microscopy. Hypercapnia was caused by i.v. injection of acetazolamide. The changes in diameter of pial arteries and veins in response to hypercapnia were measured. Results and discussion. Global cerebral ischemia led to marked decrease in pial vessels (both arteries and veins) reactivity in response to hypercapnia, caused by i.v. injection of acetazolamide. In intact rats, i.v. injection of acetazolamide led to pial arteries dilation and pial veins constriction; in animals subjected to ischemia-reperfuion. the numbers of dilated large arteries and constricted small veins were much less, as well as the extent of arterial dilation. Reactivity changes were observed in all time points studied. Conclusions. Thus, transient global cerebral ischemia cause marked and long lasting (3 weeks) decrease in pial vessel reactivity in response to hypercapnia.
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Lester, Davis, Stephanie El-Hajj, Ayman A. Farag, Pradeep Bhambhvani, Lindsey Tauxe, Jaekyeong Heo, Ami E. Iskandrian, and Fadi G. Hage. "Prognostic value of transient ischemic dilation with regadenoson myocardial perfusion imaging." Journal of Nuclear Cardiology 23, no. 5 (October 21, 2015): 1147–55. http://dx.doi.org/10.1007/s12350-015-0272-7.

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Slomka, Piotr J., Daniel S. Berman, and Guido Germano. "Normal limits for transient ischemic dilation with 99mTc myocardial perfusion SPECT protocols." Journal of Nuclear Cardiology 24, no. 5 (June 28, 2016): 1709–11. http://dx.doi.org/10.1007/s12350-016-0582-4.

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Cao, Suyi, Liang-Chao Wang, Herman Kwansa, Richard J. Roman, David R. Harder, and Raymond C. Koehler. "Endothelin rather than 20-HETE contributes to loss of pial arteriolar dilation during focal cerebral ischemia with and without polymeric hemoglobin transfusion." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 296, no. 5 (May 2009): R1412—R1418. http://dx.doi.org/10.1152/ajpregu.00003.2009.

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Partial exchange transfusion with a cell-free hemoglobin (Hb) polymer during transient middle cerebral artery occlusion (MCAO) reduces infarct volume but fails to increase blood flow, as might be expected with the induced decrease in hematocrit. In ischemic brain, endothelin antagonists are known to produce vasodilation. In nonischemic brain, pial arterioles constrict after Hb exchange transfusion, and the constriction is blocked by an inhibitor of 20-HETE synthesis. We tested the hypothesis that a 20-HETE synthesis inhibitor and an endothelin A receptor antagonist increase pial arteriolar dilation after Hb exchange transfusion during MCAO. Pial arteriolar diameter was measured in the ischemic border region of the distal MCA border region through closed cranial windows in anesthetized rats subjected to the filament model of MCAO. During 2 h of MCAO, pial arteriolar dilation gradually subsided from 37 ± 3 to 7 ± 5% (±SE). Compared with residual dilation at 2 h of MCAO with vehicle superfusion (14 ± 3%), loss of dilation was not prevented by superfusion of a 20-HETE synthesis inhibitor (21 ± 5%), partial Hb exchange transfusion (7 ± 5%) that decreased hematocrit to 23%, or a combination of the two (5 ± 5%). However, loss of dilation was prevented by superfusion of an endothelin A receptor antagonist with (35 ± 4%) or without (32 ± 5%) Hb transfusion. Pial artery constriction during reperfusion was attenuated by HET0016 alone and by BQ610 with or without Hb transfusion. Systemic administration of the endothelin antagonist during prolonged MCAO increased blood flow in the border region. Thus loss of pial arteriolar dilation in the ischemic border region during prolonged MCAO depends on endothelin A receptor activation, and this effect was independent of the presence of cell-free Hb polymers in the plasma. In contrast to previous work in nonischemic brain, inhibition of oxygen-dependent 20-HETE synthesis does not significantly influence the pial arteriolar response to polymeric Hb exchange transfusion during focal ischemia.
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Hida, Satoshi, Taishiro Chikamori, Hirokazu Tanaka, Yuko Igarashi, Chie Shiba, Yasuhiro Usui, Tsuguhisa Hatano, and Akira Yamashina. "POST-ISCHEMIC MYOCARDIAL STUNNING IS SUPERIOR TO TRANSIENT ISCHEMIC DILATION TO DETECT MULTI-VESSEL CORONARY ARTERY DISEASE." Journal of the American College of Cardiology 57, no. 14 (April 2011): E794. http://dx.doi.org/10.1016/s0735-1097(11)60794-8.

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Sciagrà, Roberto. "Transient ischemic dilation in hypertrophic cardiomyopathy: A complex sign in a complex disease." Journal of Nuclear Cardiology 27, no. 6 (December 13, 2018): 2044–47. http://dx.doi.org/10.1007/s12350-018-01567-x.

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Dissertations / Theses on the topic "Transient Ischemic Dilation"

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Graber, Taylor. "Imaging for Chest Pain Assessment: An Algorithmic Approach Using Noninvasive Modalities to Define Medical vs. Interventional Treatment." Thesis, The University of Arizona, 2017. http://hdl.handle.net/10150/623439.

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A Thesis submitted to The University of Arizona College of Medicine - Phoenix in partial fulfillment of the requirements for the Degree of Doctor of Medicine.
To analyze the roles of CCTA, MPI, and CC to formulate a sequential clinical algorithm to use in patients with chest pain, risk factors for CAD, and an abnormal EKG. The goals of the study are to streamline and refine workup, to decrease radiation exposure to patients, and to contain costs. 39 patients underwent CCTA, MPI, and CC within 30 months of each other. CCTA was used to categorize mild, moderate, or severe CAD. MPI used SSS, SDS, TID, and formal reading to define mild, moderate, or severe physiologic ischemia. CC and coronary intervention cine films were analyzed to define and treat anatomical CAD medically or by intervention. Results: There was strong correlation between CCTA, CC, and treatment type (p<0.0001). CCTA was able to stratify all patients with mild or severe ischemia to appropriate treatment groups, and to reduce the need for MPI. With moderate ischemia from CCTA, the additional use of MPI could have reduced the need for 16/18 (89%) patients who underwent CC to undergo further testing. No patients with mild or moderate CAD by CCTA, followed by mild to moderate physiologic ischemia by MPI, needed CC or intervention. 37/39 patients (95%) could have avoided one or more tests using our algorithm. CCTA followed by MPI may be used in symptomatic patients with risk factors for CAD and an abnormal EKG to stratify mild and moderate CAD, and to thereby avoid cardiac catheterization. Our algorithm could lead to savings in healthcare expenditures, save patients from unnecessary invasive procedures, decrease radiation exposure, and total cost.
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Fadiji, Isaac Olusola. "Prognostic value of transient ischemic dilatation (TID) in patients with normal single photon emission computed tomography (SPECT) myocardial perfusion imaging (MPI)." Thesis, 2016. https://hdl.handle.net/10539/24218.

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A research report submitted to the Faculty of Health Sciences, University of the Witwatersrand. In fulfilment of the requirements for the degree of Master of Medicine In the branch of Nuclear Medicine April 2016
Objectives: This study evaluated the clinical significance of an isolated transient ischemic dilatation (TID) in the otherwise normal or relatively normal single photon emission computed tomography (SPECT) myocardial perfusion imaging (MPI) in determining the risk of future cardiac events. Background: the prognostic value of TID in patients with otherwise normal SPECT MPI has been a subject of controversy among clinicians. Therefore, there is no consensus on how best to manage this patient group of patients. Methods: From a database of 4000 consecutive patients who underwent stress-rest MPI studies over a 13 year period (2000 -2013) at department of Nuclear Medicine, University of the Witwatersrand academic hospital, Johannesburg. 123 patients without known cardiac history of coronary artery disease and had normal or relatively normal SPECT MPI but TID > 1.21 were identified (study group 1). 41 patients, from the study group 1, with valid telephone contacts and hospital records were interviewed to determine the prevalence of cardiac events. The images were retrieved, reviewed and re-processed by a 4th year Nuclear Medicine registrar to ensure they were rightly called. Both the telephone interviewer and the imaging analyst were blinded to the patients’ clinical data. Results: The prevalence of TID in a normal or relative normal SPECT MPI was 4%. There were 9 (21.9%) cases of angina, 2 (4.9%) cases of revascularization and 4 (9.8%) cases of myocardial infarction. Other independent predictor of cardiac events were; diabetes, summed stress score and summed rest score. Conclusion: TID in the setting of normal or relatively normal SPECT MPI may signify a prognosis that is not good and may be a predictor of future cardiac events.
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Book chapters on the topic "Transient Ischemic Dilation"

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Ghesani, Munir, Nasrin Ghesani, E. DePuey, Amir Kashefi, and Yi Zhang. "CASE 79: LAD AND RCA ISCHEMIA; TRANSIENT ISCHEMIC DILATATION (TID)." In Nuclear Medicine: A Case-Based Approach, 106. Jaypee Brothers Medical Publishers (P) Ltd., 2016. http://dx.doi.org/10.5005/jp/books/12735_81.

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Conference papers on the topic "Transient Ischemic Dilation"

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Woo, Jonghye, Piotr J. Slomka, Ryo Nakazato, Balaji K. Tamarappoo, James K. Min, Guido Germano, Daniel S. Berman, and Damini Dey. "Feasibility of determining myocardial transient ischemic dilation from cardiac CT by automated stress/rest registration." In SPIE Medical Imaging, edited by David R. Haynor and Sébastien Ourselin. SPIE, 2012. http://dx.doi.org/10.1117/12.911472.

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Journal articles
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