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Journal articles on the topic 'Transient Neonatal Zinc Deficiency'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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1

Krieger, I., B. E. Alpern, and S. C. Cunnane. "Transient neonatal zinc deficiency." American Journal of Clinical Nutrition 43, no. 6 (1986): 955–58. http://dx.doi.org/10.1093/ajcn/43.6.955.

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2

Carroll, H. F. "Transient neonatal zinc deficiency." American Journal of Clinical Nutrition 45, no. 2 (1987): 496–97. http://dx.doi.org/10.1093/ajcn/45.2.496.

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3

Golan, Yarden, Naoya Itsumura, Fabian Glaser, Bluma Berman, Taiho Kambe, and Yehuda G. Assaraf. "Molecular Basis of Transient Neonatal Zinc Deficiency." Journal of Biological Chemistry 291, no. 26 (2016): 13546–59. http://dx.doi.org/10.1074/jbc.m116.732693.

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4

Golan, Yarden, Taiho Kambe, and Yehuda G. Assaraf. "The role of the zinc transporter SLC30A2/ZnT2 in transient neonatal zinc deficiency." Metallomics 9, no. 10 (2017): 1352–66. http://dx.doi.org/10.1039/c7mt00162b.

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Transient neonatal zinc deficiency (TNZD) results from loss of function mutations in theSLC30A2/ZnT2gene. Nursing mothers harboring this defective zinc transporter produce zinc-deficient milk. Consequently, their exclusively breastfed infants develop severe zinc deficiency. The present review summarizes our current knowledge onSLC30A2/ZnT2gene mutations and highlights the molecular mechanisms underlying this zinc deficiency. We further propose novel approaches for the early diagnosis and prevention of TNZD.

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5

Watson, Lauren, David Cartwright, Luke A. Jardine, et al. "Transient neonatal zinc deficiency in exclusively breastfed preterm infants." Journal of Paediatrics and Child Health 54, no. 3 (2017): 319–22. http://dx.doi.org/10.1111/jpc.13780.

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6

Golan, Yarden, Adrian Lehvy, Guy Horev, and Yehuda G. Assaraf. "High proportion of transient neonatal zinc deficiency causing alleles in the general population." Journal of Cellular and Molecular Medicine 23, no. 2 (2018): 828–40. http://dx.doi.org/10.1111/jcmm.13982.

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7

Itsumura, Naoya, Yoshie Kibihara, Kazuhisa Fukue, et al. "Novel mutations in SLC30A2 involved in the pathogenesis of transient neonatal zinc deficiency." Pediatric Research 80, no. 4 (2016): 586–94. http://dx.doi.org/10.1038/pr.2016.108.

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8

Liew, Hui M., Colin W. Tan, Clement K. M. Ho, Jade N. Chee, and Mark J. A. Koh. "Transient Neonatal Zinc Deficiency Caused by a Novel Mutation in the SLC30A2 Gene." Pediatric Dermatology 34, no. 2 (2017): e104-e105. http://dx.doi.org/10.1111/pde.13065.

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9

Lova Navarro, Miguel, Ángel Vera Casaño, Carmen Benito López, et al. "Transient Neonatal Zinc Deficiency Due to a New Autosomal Dominant Mutation in GeneSLC30A2(ZnT-2)." Pediatric Dermatology 31, no. 2 (2014): 251–52. http://dx.doi.org/10.1111/pde.12257.

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10

Kasana, Shakhenabat, Jamila Din, and Wolfgang Maret. "Genetic causes and gene–nutrient interactions in mammalian zinc deficiencies: Acrodermatitis enteropathica and transient neonatal zinc deficiency as examples." Journal of Trace Elements in Medicine and Biology 29 (January 2015): 47–62. http://dx.doi.org/10.1016/j.jtemb.2014.10.003.

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11

Lasry, Inbal, Young Ah Seo, Hadas Ityel, et al. "A Dominant Negative Heterozygous G87R Mutation in the Zinc Transporter, ZnT-2 (SLC30A2), Results in Transient Neonatal Zinc Deficiency." Journal of Biological Chemistry 287, no. 35 (2012): 29348–61. http://dx.doi.org/10.1074/jbc.m112.368159.

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12

Li, Zhongtao, Jiayue Wang, Yuan Yang, and Sheng Wang. "A novel homozygous mutation p.E88K in maternal SLC30A2 gene as a cause of transient neonatal zinc deficiency." Experimental Dermatology 29, no. 6 (2020): 556–61. http://dx.doi.org/10.1111/exd.14099.

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13

Chowanadisai, Winyoo, Bo Lönnerdal, and Shannon L. Kelleher. "Identification of a Mutation in SLC30A2 (ZnT-2) in Women with Low Milk Zinc Concentration That Results in Transient Neonatal Zinc Deficiency." Journal of Biological Chemistry 281, no. 51 (2006): 39699–707. http://dx.doi.org/10.1074/jbc.m605821200.

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14

Miletta, Maria Consolata, Andreas Bieri, Kristin Kernland, et al. "Transient Neonatal Zinc Deficiency Caused by a Heterozygous G87R Mutation in theZinc Transporter ZnT-2 (SLC30A2)Gene in the Mother Highlighting the Importance of Zn2+for Normal Growth and Development." International Journal of Endocrinology 2013 (2013): 1–8. http://dx.doi.org/10.1155/2013/259189.

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Suboptimal dietary zinc (Zn2+) intake is increasingly appreciated as an important public health issue. Zn2+is an essential mineral, and infants are particularly vulnerable to Zn2+deficiency, as they require large amounts of Zn2+for their normal growth and development. Although term infants are born with an important hepatic Zn2+storage, adequate Zn2+nutrition of infants mostly depends on breast milk or formula feeding, which contains an adequate amount of Zn2+to meet the infants’ requirements. An exclusively breast-fed 6 months old infant suffering from Zn2+deficiency caused by an autosomal do

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15

Nishi, Y., S. Hatano, K. Aihara, A. Fujie, and M. Kihara. "Transient partial growth hormone deficiency due to zinc deficiency." Journal of the American College of Nutrition 8, no. 2 (1989): 93–97. http://dx.doi.org/10.1080/07315724.1989.10720283.

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16

Kim, Mi Ock, Sun Young Park, Oh Soo Kwon, et al. "Three Cases of Transient Symptomatic Zinc Deficiency." Korean Journal of Pediatric Gastroenterology and Nutrition 2, no. 1 (1999): 123. http://dx.doi.org/10.5223/kjpgn.1999.2.1.123.

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17

Minutillo, C., P. J. Pemberton, M. L. Willoughby, P. J. Price, and L. R. Marshall. "Neonatal purpura fulminans and transient protein C deficiency." Archives of Disease in Childhood 65, no. 5 (1990): 561–62. http://dx.doi.org/10.1136/adc.65.5.561-b.

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18

MacDonald, P. D., I. D. Walker, P. Galea, and L. G. Alroomi. "Acquired transient protein C deficiency in neonatal cardiac failure." Archives of Disease in Childhood 65, no. 1 (1990): 158. http://dx.doi.org/10.1136/adc.65.1.158-b.

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19

Stapleton, Karen M., Edward O'Loughlin, and John P. Relic. "Transient zinc deficiency in a breast-fed premature infant." Australasian Journal of Dermatology 36, no. 3 (1995): 157–59. http://dx.doi.org/10.1111/j.1440-0960.1995.tb00959.x.

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20

Crisóstomo, Mafalda, Mafalda Casinhas Santos, Ermelindo Tavares, and Florbela Cunha. "Transient symptomatic zinc deficiency in an exclusively breastfed infant." BMJ Case Reports 14, no. 6 (2021): e241754. http://dx.doi.org/10.1136/bcr-2021-241754.

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A 3-month-old, full term female infant, adequate for gestational age, and exclusively breastfed, was admitted with a 10 day history of generalised scaling erythematous dermatitis, affecting the face (perinasal, nasolabial folds and periauricular), acral and intertriginous areas, with irritability and failure to thrive. Her mother had been treated with isoniazid since the third trimester because of family contact with tuberculosis. Based on a diagnosis of suspected impetiginised eczema, the infant was treated with flucloxacillin and prednisolone, and maternal isoniazid was suspended, with no im

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21

Tang, Tina, and Joseph M. Lam. "Unique presentation of transient zinc deficiency from low maternal breast milk zinc levels." Pediatric Dermatology 35, no. 2 (2017): 255–56. http://dx.doi.org/10.1111/pde.13349.

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22

Kiechl-Kohlendorfer, Ursula, Franz-Martin Fink, and Elisabeth Steichen-Gersdorf. "Transient Symptomatic Zinc Deficiency in a Breast-fed Preterm Infant." Pediatric Dermatology 24, no. 5 (2007): 536–40. http://dx.doi.org/10.1111/j.1525-1470.2007.00512.x.

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23

Sibileva, E. N. "Seasonal Features of Transient Hyperthyrotropinemia in newborns." Problems of Endocrinology 50, no. 5 (2004): 11–14. http://dx.doi.org/10.14341/probl11512.

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The results of a 5-year neonatal screening of thyrotropic hormone (TTH) in 51,222 neonatal infants were analyzed to evaluate the severity of goiter endemia in the Arkhangelsk Region that is an area of mild and moderate iodine deficiency and a high-latitude region. The analysis established the following regularity: the spread of elevated TTH levels > 5 m U/l in neonatal infants was much higher in summer months. The fluctuating pattern of transient hyperthyrotropinemia was found in the contrast seasons. The magnitude of these fluctuations depended on the climatic and geographic area where the

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24

Kolevská, J., V. Brunclík, and L. Bartošová. "Neonatal Transient Hypothyroidism in Two Whippet Puppies." Acta Veterinaria Brno 76, no. 2 (2007): 277–82. http://dx.doi.org/10.2754/avb200776020277.

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The work describes a case of neonatal transient hypothyroidism in two whippet puppies aged 10 weeks. The condition was caused by iodine deficiency either in the diet solely or in combination with a partial peroxidase defect. Goitre developed in the puppies, fed giblets and poultry meat. Laboratory examinations in both puppies showed symptoms of hypothyroidism: non-regenerative anaemia, hypercholesterolaemia, significant decrease in the concentration of total thyroxine and increased concentration of thyrotropin. Ultrasonographic examination demonstrated enlarged thyroid glands. Cytological exam

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25

Lee, Min Geol, Kyung Tae Hong, and Jin Ju Kim. "Transient symptomatic zinc deficiency in a full-term breast-fed infant." Journal of the American Academy of Dermatology 23, no. 2 (1990): 375–79. http://dx.doi.org/10.1016/0190-9622(90)70226-8.

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26

Yusrawati, Yusrawati, Defrin Defrin, and Hudila Rifa Karmia. "Neonatal Growth, Neurotrophine, Zinc, and Ferritin Concentration in Normal and Iron Deficience Pregnancy: An Observational Analitic Study." Open Access Macedonian Journal of Medical Sciences 7, no. 7 (2019): 1114–18. http://dx.doi.org/10.3889/oamjms.2019.202.

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BACKGROUND: Anemia in pregnancy was one of the national problems. Insufficient iron deposits before pregnancy and inadequate iron intake during pregnancy could lead to iron deficiency anaemia in pregnancy, followed by iron deficiency in neonates. AIM: This study aimed to assess the molecular relationship of maternal iron deficiency with the function of the neonatal central nervous system to know the cognitive aspects of learning ability of children. METHODS: This study was an observational analytic study with cross-sectional design underwent in RSUP Dr M. Djamil Padang, RSI Ibnu Si

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27

El-Maghraby, Mamdouh M., and Ahmed E. Mahmoud. "Clinical, hematological, and biochemical studies on hypozincemia in neonatal calves in Egypt." Veterinary World 14, no. 2 (2021): 314–18. http://dx.doi.org/10.14202/vetworld.2021.314-318.

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Background and Aim: Zinc has a wide spectrum of biological activities and its deficiency has been related to various dysfunctions. This study aimed to clarify the clinical, hematological, and biochemical changes in Holstein dairy calves with naturally occurring hypozincemia before and after treatment. Materials and Methods: This study was carried out on 25 Holstein dairy calves <1 month of age in the El-Salhya Dairy Farm, Al-Sharqiya Province, Egypt. Calves were born from apparent healthy dams without any clinical signs of zinc deficiency. They were divided into two groups. The first group

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28

Yamada, Saori, Susumu Tanaka, Soju Seki, and Mikihiko Kogo. "Membrane excitabilities in neonatal rat mesencephalic trigeminal neurons under dietary zinc deficiency." Journal of Oral Science 63, no. 3 (2021): 242–46. http://dx.doi.org/10.2334/josnusd.20-0566.

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29

Omran, Ahmed, Heba Mousa, Mohamed Osama Abdalla, and Osama Zekry. "Maternal and neonatal vitamin D deficiency and transient tachypnea of the newborn in full term neonates." Journal of Perinatal Medicine 46, no. 9 (2018): 1057–60. http://dx.doi.org/10.1515/jpm-2017-0280.

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Abstract Aim: To investigate the association between maternal and neonatal serum 25-hydroxyvitamin D (25-OHD) levels and development of transient tachypnea of the newborn (TTN) in full term infants. Methods: This was a prospective case-control study carried out on 30 neonates with TTN and their mothers and 30 control neonates and their mothers. Levels of 25-OHD were measured in maternal and neonatal blood samples that were obtained in the first 12–24 h of postnatal age. Results: Both maternal and neonatal 25-OHD levels in the TTN group were significantly lower compared to the control group (P=

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30

Li, Li‐Ting, Zhong‐Die Li, Ye Yang, et al. "ABCB11 deficiency presenting as transient neonatal cholestasis: Correlation with genotypes and BSEP expression." Liver International 40, no. 11 (2020): 2788–96. http://dx.doi.org/10.1111/liv.14642.

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31

D’Amico, Giovanna, Corinne De Laet, Guillaume Smits, et al. "Acquired Zinc Deficiency Mimicking Acrodermatitis Enteropathica in a Breast-Fed Premature Infant." Pediatric Reports 13, no. 3 (2021): 444–49. http://dx.doi.org/10.3390/pediatric13030051.

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We present a case of a transient acquired zinc deficiency in a breast-fed, 4-month-old-male prematurely born infant, with acrodermatitis enteropathica-like symptoms such as crusted, eroded, erythemato-squamous eruption in periorificial and acral patterns. The laboratory investigations showed low zinc levels in the infant’s and the mother’s serum and in the mother’s milk; genetic analysis did not show any mutation in the SLC39A4 gene, involved in acrodermatitis enteropathica. Acquired zinc deficiency is often found in premature infants because of their increased requirement, the low serum and m

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32

Ahmed, Shabbeer, and Avula Kanthi Sagar. "Comparison of maternal serum and neonatal cord blood levels of zinc in relation to birth weight and period of gestation." International Journal of Contemporary Pediatrics 8, no. 8 (2021): 1349. http://dx.doi.org/10.18203/2349-3291.ijcp20212880.

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Background: Women are at increased risk of zinc deficiency during pregnancy because of high fetal requirements for zinc. Severe maternal zinc deficiency has been associated with poor fetal growth, spontaneous abortion and congenital malformations (i.e., anencephaly), whereas milder forms of zinc deficiency have been associated with low birth weight (LBW), intrauterine growth retardation, and preterm delivery. However, the research relating maternal zinc status and birth weight has not produced consistent results. This study has been undertaken to confirm the association between maternal serum

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33

Glover, M. T., and D. J. Atherton. "Transient Zinc Deficiency in Two Full-term Breast-fed Siblings Associated with Low Maternal Breast Milk Zinc Concentration." Pediatric Dermatology 5, no. 1 (1988): 10–13. http://dx.doi.org/10.1111/j.1525-1470.1988.tb00877.x.

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34

Hermeziu, Bogdan, Damien Sanlaville, Muriel Girard, Claude L??onard, Stanislas Lyonnet, and Emmanuel Jacquemin. "Heterozygous Bile Salt Export Pump Deficiency: A Possible Genetic Predisposition to Transient Neonatal Cholestasis." Journal of Pediatric Gastroenterology and Nutrition 42, no. 1 (2006): 114–16. http://dx.doi.org/10.1097/01.mpg.0000184429.34001.68.

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35

Adnan, Chaliza, I. Wayan Dharma Artana, Ketut Suarta, I. Gusti Lanang Sidiartha, I. Wayan Gustawan, and Ni Putu Veny Kartika Yantie. "Serum zinc level and prognosis of neonatal sepsis." Paediatrica Indonesiana 60, no. 2 (2020): 37–42. http://dx.doi.org/10.14238/pi60.2.2020.37-42.

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Background The prognosis of neonatal sepsis can be influenced by various risk factors, one of which is a deficiency of zinc micronutrient substances. Objective To assess for a potential association between serum zinc level and prognosis of infants with early-onset neonatal sepsis (EONS). Methods This prospective cohort study was done in neonates with clinical EONS from September 2017 until December 2018. Serum zinc level was measured on the first day of diagnosis and prognosis was assessed on the fourth day. The association between serum zinc levels and prognosis of EONS was analyz

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36

Adnan, Chaliza, I. Wayan Dharma Artana, Ketut Suarta, I. Gusti Lanang Sidiartha, I. Wayan Gustawan, and Ni Putu Veny Kartika Yantie. "Serum zinc level and prognosis of neonatal sepsis." Paediatrica Indonesiana 60, no. 2 (2020): 61–66. http://dx.doi.org/10.14238/pi60.2.2020.61-66.

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Background The prognosis of neonatal sepsis can be influenced by various risk factors, one of which is a deficiency of zinc micronutrient substances. Objective To assess for a potential association between serum zinc level and prognosis of infants with early-onset neonatal sepsis (EONS). Methods This prospective cohort study was done in neonates with clinical EONS from September 2017 until December 2018. Serum zinc level was measured on the first day of diagnosis and prognosis was assessed on the fourth day. The association between serum zinc levels and prognosis of EONS was analyz

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37

Sinclair, Graham, Sylvia Stockler-Ipsiroglu, Gabriella Horvath, and Momen Almomen. "Pyruvate Carboxylase Deficiency Type C: A Rare Cause of Acute Transient Flaccid Paralysis with Ketoacidosis." Neuropediatrics 49, no. 06 (2018): 369–72. http://dx.doi.org/10.1055/s-0038-1667171.

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AbstractPyruvate carboxylase (PC) is a biotin-containing enzyme that is responsible for the adenosine triphosphate-dependent carboxylation of pyruvate to oxaloacetate, a key intermediate in the tricarboxylic acid cycle. PC deficiency (OMIM 266150) is a rare autosomal recessive metabolic disease, causing elevation of pyruvate, lactate, and alanine. Three types of PC deficiency have been described in the literature; A, B, and C. Type A PC deficiency, also called infantile or North American type, is characterized by infantile onset acidosis, failure to thrive, and developmental delay. The second

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38

El Fékih, Nadia, Kharfi Monia, Sebastien Schmitt, Iméne Dorbani, Sébastien Küry, and Mohamed R. Kamoun. "Transient symptomatic zinc deficiency in a breast-fed infant: Relevance of a genetic study." Nutrition 27, no. 10 (2011): 1087–89. http://dx.doi.org/10.1016/j.nut.2011.06.002.

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39

Bye, A. M. E., A. Goodfellow, and D. J. Atherton. "Transient Zinc Deficiency in a Full-Term Breast-Fed Infant of Normal Birth Weight." Pediatric Dermatology 2, no. 4 (1985): 308–11. http://dx.doi.org/10.1111/j.1525-1470.1985.tb00471.x.

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40

Nyholm Kyvsgaard, Julie, Christina Ellervik, Emilie Bundgaard Lindkvist, et al. "Perinatal Whole Blood Zinc Status and Cytokines, Adipokines, and Other Immune Response Proteins." Nutrients 11, no. 9 (2019): 1980. http://dx.doi.org/10.3390/nu11091980.

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(1) Background: Zinc is an essential micronutrient and zinc deficiency is associated with immune dysfunction. The neonatal immune system is immature, and therefore an optimal neonatal zinc status may be important. The aim of this study was to investigate the possible association between neonatal whole blood (WB)-Zinc content and several immune markers. (2) Methods: In total, 398 healthy newborns (199 who later developed type 1 diabetes and 199 controls) from the Danish Newborn Screening Biobank had neonatal dried blood spots (NDBS) analyzed for WB-Zinc content and (i) cytokines: Interleukin (I

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41

Secchi, Luciana A. de A., Juliana F. Mazzeu, Mara Santos Córdoba, Íris Ferrari, Helton Estrela Ramos, and Francisco de Assis Rocha Neves. "Transient neonatal hypothyroidism in a boy with unbalanced translocation t(8;16)." Arquivos Brasileiros de Endocrinologia & Metabologia 56, no. 8 (2012): 564–69. http://dx.doi.org/10.1590/s0004-27302012000800017.

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Genetic defects resulting in deficiency of thyroid hormone synthesis can be found in about 10% of the patients with permanent congenital hypothyroidism, but the identification of genetic abnormalities in association with the transient form of the disease is extremely rare. We report the case of a boy with transient neonatal hypothyroidism that was undiagnosed in the neonatal screening, associated with extrathyroid malformations and mental retardation. The boy carries an unbalanced translocation t(8;16), and his maternal uncle had a similar phenotype. Chromosomal analysis defined the patient's

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42

Barruscotti, Stefania, Camilla Vassallo, Chiara Giorgini, et al. "Transient symptomatic zinc deficiency in a breast‐fed African infant: case report and literature review." International Journal of Dermatology 58, no. 8 (2018): 963–65. http://dx.doi.org/10.1111/ijd.14206.

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43

Dassoni, Federica, Zerihun Abebe, Federica Ricceri, Aldo Morrone, Cristiana Albertin, and Bernard Naafs. "High Frequency of Symptomatic Zinc Deficiency in Infants in Northern Ethiopia." Dermatology Research and Practice 2014 (2014): 1–5. http://dx.doi.org/10.1155/2014/719701.

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Background. Zinc deficiency occurs in infants when its demand exceeds its supply. It presents with cutaneous signs which, in severe cases, are associated with diarrhea, alopecia, and irritability. Genetic and acquired forms of zinc deficiency have been reported and often overlap clinical features. Malnutrition, prematurity, malabsorption syndromes, and burns may cause an increased demand for zinc.Methods. Cases of acquired transient infantile zinc deficiency (TIZD) observed during a period of 3 years at Ayder Referral Hospital of Mekelle, Northern Ethiopia, are reported here. Since no sophisti

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44

Azizi, Fereidoun, Mohamed Afkhami, Aezam Sarshar, and Mahtalat Nafarabadi. "Effects of transient neonatal Hyperthyrotropinemia on Intellectual quotient and psychomotor performance." International Journal for Vitamin and Nutrition Research 71, no. 1 (2001): 70–73. http://dx.doi.org/10.1024/0300-9831.71.1.70.

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Transient neonatal hyperthyrotropinemia (TNH) occurs frequently in areas of iodine deficiency. To evaluate the effect of TNH in intellectual function and psychomotor performance, a historical cohohrt study was performed in 9 years old children with documented TNH at birth. 18 children with TNH who had been born in Mahdieh Hospital were studied at age 9 and compared to 19 matcheal children born at the same time, but having normal thyroid function at birth. Global intelligence (IQ) and psychomotor performance were evaluated with Raven and Bender-Gestalt tests, respectively. Total serum T4 and T3

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45

Chowanadisai, Winyoo, Shannon L. Kelleher, and Bo Lönnerdal. "Zinc Deficiency Is Associated with Increased Brain Zinc Import and LIV-1 Expression and Decreased ZnT-1 Expression in Neonatal Rats." Journal of Nutrition 135, no. 5 (2005): 1002–7. http://dx.doi.org/10.1093/jn/135.5.1002.

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46

Chowanadisai, Winyoo, Shannon L. Kelleher, and Bo Lönnerdal. "Maternal zinc deficiency reduces NMDA receptor expression in neonatal rat brain, which persists into early adulthood." Journal of Neurochemistry 94, no. 2 (2005): 510–19. http://dx.doi.org/10.1111/j.1471-4159.2005.03246.x.

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47

Braslavsky, Débora, Ana Keselman, Marcela Galoppo, et al. "Neonatal cholestasis in congenital pituitary hormone deficiency and isolated hypocortisolism: characterization of liver dysfunction and follow-up." Arquivos Brasileiros de Endocrinologia & Metabologia 55, no. 8 (2011): 622–27. http://dx.doi.org/10.1590/s0004-27302011000800017.

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INTRODUCTION: Neonatal cholestasis due to endocrine diseases is infrequent and poorly reco-gnized. Referral to the pediatric endocrinologist is delayed. OBJECTIVE: We characterized cholestasis in infants with congenital pituitary hormone deficiencies (CPHD), and its resolution after hormone replacement therapy (HRT). SUBJECTS AND METHODS: Sixteen patients (12 males) were included; eleven with CPHD, and five with isolated central hypocortisolism. RESULTS: Onset of cholestasis occurred at a median age of 18 days of life (range 2-120). Ten and nine patients had elevated transaminases and γGT, res

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48

Ducarme, Guillaume, Lucie Planche, Emeric Abet, Valérie Desroys du Roure, and Amélie Ducet-Boiffard. "A Prospective Study of Association of Micronutrients Deficiencies during Pregnancy and Neonatal Outcome among Women after Bariatric Surgery." Journal of Clinical Medicine 10, no. 2 (2021): 204. http://dx.doi.org/10.3390/jcm10020204.

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Little is known about the association of micronutrients deficiencies during pregnancy and neonatal outcome among women after Roux-en-Y gastric bypass (RYGB) or sleeve gastrectomy (SG). We present a prospective study of 87 consecutive women with a history of RYGB (n = 37) or SG (n = 50) who underwent complete and regular clinical and biological nutritional assessments during pregnancy. Data on maternal characteristics, biological nutritional parameters, antenatal management, and perinatal outcome were collected. The objective was to evaluate serum levels of micronutrients at the second trimeste

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49

Ducarme, Guillaume, Lucie Planche, Emeric Abet, Valérie Desroys du Roure, and Amélie Ducet-Boiffard. "A Prospective Study of Association of Micronutrients Deficiencies during Pregnancy and Neonatal Outcome among Women after Bariatric Surgery." Journal of Clinical Medicine 10, no. 2 (2021): 204. http://dx.doi.org/10.3390/jcm10020204.

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Little is known about the association of micronutrients deficiencies during pregnancy and neonatal outcome among women after Roux-en-Y gastric bypass (RYGB) or sleeve gastrectomy (SG). We present a prospective study of 87 consecutive women with a history of RYGB (n = 37) or SG (n = 50) who underwent complete and regular clinical and biological nutritional assessments during pregnancy. Data on maternal characteristics, biological nutritional parameters, antenatal management, and perinatal outcome were collected. The objective was to evaluate serum levels of micronutrients at the second trimeste

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Huizinga, TW, RW Kuijpers, M. Kleijer, et al. "Maternal genomic neutrophil FcRIII deficiency leading to neonatal isoimmune neutropenia [see comments]." Blood 76, no. 10 (1990): 1927–32. http://dx.doi.org/10.1182/blood.v76.10.1927.1927.

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Abstract:

Abstract The healthy mother of a child with transient immune neutropenia was found to be “NA-null.” The mother's neutrophils did not react with anti- NA1 and anti-NA2 antibodies (polyclonal human alloantibodies and mouse monoclonal antibodies). A healthy donor was discovered during routine neutrophil antigen typing whose neutrophils were also “NA-null.” This NA-phenotype was due to the absence of FcRIII (CD16 antigen) on neutrophils as demonstrated with anti-FcRIII monoclonal antibodies. The neutrophils of these two individuals were not able to bind dimeric immunoglobulin G. However, their cel

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