Relevant bibliographies by topics / Translational silencing

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Translational silencing'

Author: Grafiati
Published: 28 June 2021
Last updated: 29 July 2025

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Journal articles on the topic "Translational silencing"

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Sampath, Prabha, Barsanjit Mazumder, Vasudevan Seshadri, and Paul L. Fox. "Transcript-Selective Translational Silencing by Gamma Interferon Is Directed by a Novel Structural Element in the Ceruloplasmin mRNA 3′ Untranslated Region." Molecular and Cellular Biology 23, no. 5 (2003): 1509–19. http://dx.doi.org/10.1128/mcb.23.5.1509-1519.2003.

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ABSTRACT Transcript-selective translational control of eukaryotic gene expression is often directed by a structural element in the 3′ untranslated region (3′-UTR) of the mRNA. In the case of ceruloplasmin (Cp), induced synthesis of the protein by gamma interferon (IFN-γ) in U937 monocytic cells is halted by a delayed translational silencing mechanism requiring the binding of a cytosolic inhibitor to the Cp 3′-UTR. Silencing requires the essential elements of mRNA circularization, i.e., eukaryotic initiation factor 4G, poly(A)-binding protein, and poly(A) tail. We here determined the minimal si
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Baez, María Verónica, Luciana Luchelli, Darío Maschi, et al. "Smaug1 mRNA-silencing foci respond to NMDA and modulate synapse formation." Journal of Cell Biology 195, no. 7 (2011): 1141–57. http://dx.doi.org/10.1083/jcb.201108159.

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Mammalian Smaug1/Samd4A is a translational repressor. Here we show that Smaug1 forms mRNA-silencing foci located at postsynapses of hippocampal neurons. These structures, which we have named S-foci, are distinct from P-bodies, stress granules, or other neuronal RNA granules hitherto described, and are the first described mRNA-silencing foci specific to neurons. RNA binding was not required for aggregation, which indicates that S-foci formation is not a consequence of mRNA silencing. N-methyl-d-aspartic acid (NMDA) receptor stimulation provoked a rapid and reversible disassembly of S-foci, tran
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Mazumder, Barsanjit, Vasudevan Seshadri, Hiroaki Imataka, Nahum Sonenberg, and Paul L. Fox. "Translational Silencing of Ceruloplasmin Requires the Essential Elements of mRNA Circularization: Poly(A) Tail, Poly(A)-Binding Protein, and Eukaryotic Translation Initiation Factor 4G." Molecular and Cellular Biology 21, no. 19 (2001): 6440–49. http://dx.doi.org/10.1128/mcb.21.19.6440-6449.2001.

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ABSTRACT Ceruloplasmin (Cp) is a glycoprotein secreted by the liver and monocytic cells and probably plays roles in inflammation and iron metabolism. We showed previously that gamma interferon (IFN-γ) induced Cp synthesis by human U937 monocytic cells but that the synthesis was subsequently halted by a transcript-specific translational silencing mechanism involving the binding of a cytosolic factor(s) to the Cp mRNA 3′ untranslated region (UTR). To investigate how protein interactions at the Cp 3′-UTR inhibit translation initiation at the distant 5′ end, we considered the “closed-loop” model o
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Chapat, Clément, Seyed Mehdi Jafarnejad, Edna Matta-Camacho, et al. "Cap-binding protein 4EHP effects translation silencing by microRNAs." Proceedings of the National Academy of Sciences 114, no. 21 (2017): 5425–30. http://dx.doi.org/10.1073/pnas.1701488114.

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MicroRNAs (miRNAs) play critical roles in a broad variety of biological processes by inhibiting translation initiation and by destabilizing target mRNAs. The CCR4–NOT complex effects miRNA-mediated silencing, at least in part through interactions with 4E-T (eIF4E transporter) protein, but the precise mechanism is unknown. Here we show that the cap-binding eIF4E-homologous protein 4EHP is an integral component of the miRNA-mediated silencing machinery. We demonstrate that the cap-binding activity of 4EHP contributes to the translational silencing by miRNAs through the CCR4–NOT complex. Our resu
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Vyas, Keyur, Sujan Chaudhuri, Douglas W. Leaman, et al. "Genome-Wide Polysome Profiling Reveals an Inflammation-Responsive Posttranscriptional Operon in Gamma Interferon-Activated Monocytes." Molecular and Cellular Biology 29, no. 2 (2008): 458–70. http://dx.doi.org/10.1128/mcb.00824-08.

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ABSTRACT We previously showed that ribosomal protein L13a is required for translational silencing of gamma interferon (IFN-γ)-induced ceruloplasmin (Cp) synthesis in monocytes. This silencing also requires the presence of the GAIT (IFN-gamma activated inhibitor of translation) element in the 3′ untranslated region (UTR) of Cp mRNA. Considering that Cp is an inflammatory protein, we hypothesized that this mechanism may have evolved to silence a family of proinflammatory proteins, of which Cp is just one member. To identify the other mRNAs that are targets for this silencing, we performed a geno
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Huarte, Joaquin, André Stutz, Marcia L. O'Connell, et al. "Transient translational silencing by reversible mRNA deadenylation." Cell 69, no. 6 (1992): 1021–30. http://dx.doi.org/10.1016/0092-8674(92)90620-r.

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Nair, Asha P. K., Hans H. Hirsch, Marco Colombi, and Christoph Moroni. "Cyclosporin A Promotes Translational Silencing of Autocrine Interleukin-3 via Ribosome-Associated Deadenylation." Molecular and Cellular Biology 19, no. 1 (1999): 889–98. http://dx.doi.org/10.1128/mcb.19.1.889.

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ABSTRACT Translation is regulated predominantly by an interplay betweencis elements at the 3′ and 5′ ends of mRNAs andtrans-acting proteins. Cyclosporin A (CsA), a calcineurin antagonist and blocker of interleukin-2 (IL-2) transcription in T cells, was found to inhibit translation of IL-3 mRNA in autocrine mast cell tumor lines. The mechanism involved ribosome-associated poly(A) shortening and required an intact AU-rich element in the 3′ untranslated region. FK506, another calcineurin inhibitor, shared the effect. The translational inhibition by CsA was specific to oncogenically induced lympho
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Stutz, A., J. Huarte, P. Gubler, B. Conne, D. Belin, and J. D. Vassalli. "In vivo antisense oligodeoxynucleotide mapping reveals masked regulatory elements in an mRNA dormant in mouse oocytes." Molecular and Cellular Biology 17, no. 4 (1997): 1759–67. http://dx.doi.org/10.1128/mcb.17.4.1759.

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In mouse oocytes, tissue-type plasminogen activator (tPA) mRNA is under translational control. The newly transcribed mRNA undergoes deadenylation and translational silencing in growing oocytes, while readenylation and translation occur during meiotic maturation. To localize regulatory elements controlling tPA mRNA expression, we identified regions of the endogenous transcript protected from hybridization with injected antisense oligodeoxynucleotides. Most of the targeted sequences in either the 5' untranslated region (5'UTR), coding region, or 3'UTR were accessible to hybridization, as reveale
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Zekri, Latifa, Eric Huntzinger, Susanne Heimstädt, and Elisa Izaurralde. "The Silencing Domain of GW182 Interacts with PABPC1 To Promote Translational Repression and Degradation of MicroRNA Targets and Is Required for Target Release." Molecular and Cellular Biology 29, no. 23 (2009): 6220–31. http://dx.doi.org/10.1128/mcb.01081-09.

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ABSTRACT GW182 family proteins are essential in animal cells for microRNA (miRNA)-mediated gene silencing, yet the molecular mechanism that allows GW182 to promote translational repression and mRNA decay remains largely unknown. Previous studies showed that while the GW182 N-terminal domain interacts with Argonaute proteins, translational repression and degradation of miRNA targets are promoted by a bipartite silencing domain comprising the GW182 middle and C-terminal regions. Here we show that the GW182 C-terminal region is required for GW182 to release silenced mRNPs; moreover, GW182 dissoci
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Ostareck-Lederer, Antje, Dirk H. Ostareck, Christophe Cans, et al. "c-Src-Mediated Phosphorylation of hnRNP K Drives Translational Activation of Specifically Silenced mRNAs." Molecular and Cellular Biology 22, no. 13 (2002): 4535–43. http://dx.doi.org/10.1128/mcb.22.13.4535-4543.2002.

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ABSTRACT hnRNPK and hnRNP E1/E2 mediate translational silencing of cellular and viral mRNAs in a differentiation-dependent way by binding to specific regulatory sequences. The translation of 15-lipoxygenase (LOX) mRNA in erythroid precursor cells and of the L2 mRNA of human papilloma virus type 16 (HPV-16) in squamous epithelial cells is silenced when either of these cells is immature and is activated in maturing cells by unknown mechanisms. Here we address the question of how the silenced mRNA can be translationally activated. We show that hnRNP K and the c-Src kinase specifically interact wi
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Dissertations / Theses on the topic "Translational silencing"

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Kapasi, Purvi. "An Insight into GAIT Complex Mediated Translational Silencing." Cleveland State University / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=csu1232567504.

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Danner, Johannes [Verfasser], and Gunter [Akademischer Betreuer] Meister. "Regulation of gene silencing: From microRNA biogenesis to post-translational modifications of TNRC6 complexes / Johannes Danner ; Betreuer: Gunter Meister." Regensburg : Universitätsbibliothek Regensburg, 2017. http://d-nb.info/1172970572/34.

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Chiang, Jen-Chieh. "Dosage Compensation of Trisomy 21 and Its Implications for Hematopoietic Pathogenesis in Down Syndrome." eScholarship@UMMS, 2011. http://escholarship.umassmed.edu/gsbs_diss/931.

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Down Syndrome (DS), the most common aneuploidy seen in live-borns, is caused by trisomy for chromosome 21. DS imposes high risks for multiple health issues involving various systems of the body. The genetic complexity of trisomy 21 and natural variation between all individuals has impeded understanding of the specific cell pathologies and pathways involved. In addition, chromosomal disorders have been considered outside the hopeful progress in gene therapies for single-gene disorders. Here we test the feasibility of correcting imbalanced expression of genes across an extra chromosome by expres
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Chiang, Jen-Chieh. "Dosage Compensation of Trisomy 21 and Its Implications for Hematopoietic Pathogenesis in Down Syndrome." eScholarship@UMMS, 2017. https://escholarship.umassmed.edu/gsbs_diss/931.

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Down Syndrome (DS), the most common aneuploidy seen in live-borns, is caused by trisomy for chromosome 21. DS imposes high risks for multiple health issues involving various systems of the body. The genetic complexity of trisomy 21 and natural variation between all individuals has impeded understanding of the specific cell pathologies and pathways involved. In addition, chromosomal disorders have been considered outside the hopeful progress in gene therapies for single-gene disorders. Here we test the feasibility of correcting imbalanced expression of genes across an extra chromosome by expres
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Bartlett, Danielle. "PAX3 expression, protein modifications and downstream target gene profiling in melanocytes and melanoma cells." Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2013. https://ro.ecu.edu.au/theses/924.

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PAX3 is a transcription factor. It plays a major role in the development of melanocytes in the embryo. As a result of alternative splicing, the gene gives rise to eight different transcripts which encode proteins that have differing structures and are therefore likely to activate different downstream target genes. The presence of post-translational modifications has also been shown to alter the functions of the proteins. PAX3 regulates the maintenance of undifferentiated melanoblasts and mediates pathways involved in proliferation, migration and survival. It has been shown to be expressed in m
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Floris, Maïna. "Etude des régulations post-transcriptionnelles en réponse à la lumière chez Arabidopsis thaliana." Thesis, Aix-Marseille, 2013. http://www.theses.fr/2013AIXM4005.

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Ce travail de thèse porte sur l’étude des régulations post-transcriptionnelles en réponse à la lumière chez A.thaliana. Nous avons étudié deux systèmes de réponse à la lumière, la régulation traductionnelle des antennes photosynthétiques (Lhc) et la régulation de la voie des anthocyanes par le RNA silencing permettant la photoprotection. Dans une première partie nôtre approche a permis de montrer que la lumière a un impact sur le niveau de traduction global. De plus nous avons pu mettre en évidence que certaines Lhc sont régulées de façon traductionnelles en réponse à la lumière. Cette régulat
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Guidi, Mònica. "Micro RNA-Mediated regulation of the full-length and truncated isoforms of human neurotrophic tyrosine kinase receptor type 3 (NTRK 3)." Doctoral thesis, Universitat Pompeu Fabra, 2009. http://hdl.handle.net/10803/7114.

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Neurotrophins and their receptors are key molecules in the development of the<br/>nervous system. Neurotrophin-3 binds preferentially to its high-affinity receptor<br/>NTRK3, which exists in two major isoforms in humans, the full-length kinaseactive<br/>form (150 kDa) and a truncated non-catalytic form (50 kDa). The two<br/>variants show different 3'UTR regions, indicating that they might be differentially<br/>regulated at the post-transcriptional level. In this work we explore how<br/>microRNAs take part in the regulation of full-length and truncated NTRK3,<br/>demonstrating that the two isof
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Lettrich, Patrik. "Translační iniciační faktory proteinové rodiny 4E a jejich vliv na regulaci genové exprese." Master's thesis, 2021. http://www.nusl.cz/ntk/nusl-446448.

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The translation represents one of the most crucial processes in the cell. That is why it is often targeted by various regulations. Its initiation phase has a particularly important role in regulatory processes. Initiation of translation usually starts by recognition and binding of canonical eukaryotic initiation factor 4E1 (eIF4E1) to the methylguanosine cap present on the 5' end of the majority of eukaryotic mRNA. The family of 4E translation initiation factors contains two more members - eIF4E2 and eIF4E3. Those two proteins can bind cap structure as well which predetermines it to function i
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Books on the topic "Translational silencing"

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S, Cho-Chung Yoon, Gewirtz A. M, Stein Cy A, and New York Academy of Sciences., eds. Therapeutic oligonucleotides: Transcriptional and translational strategies for silencing gene expression. New York Academy of Sciences, 2005.

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Rhoads, Robert E. MiRNA regulation of the translational machinery. Edited by SpringerLink (Online service). Springer, 2009.

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Letelier-Ruz, Elias. Silence =: Silencio. The Muses' Company, 1992.

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Domínguez Prieto, Juan Miguel, 1963-, ed. Antología viva y confidente de la inspiración: Los poetas del silencio : edición cronológica de textos. AdamaRamada Ediciones, 2006.

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Stein, Cy A., and A. M. Gewirtz. Therapeutic Oligonucleotides: Transcriptional and Translational Strategies for Silencing Gene Expression (Annals of the New York Academy of Sciences). Blackwell Publishing Limited, 2005.

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Cho-Chung, Yoon S., Cy A. Stein, Cooley's Anemia Symposium 2005 Lake Buen, Elliott P. Vichinsky, and NIH SYMPOSIUM ON THERAPEUTIC OLIGONUCLEO. Therapeutic Oligonucleotides: Transcriptional and translational Strategies for Silencing Gene Expression (Annals of the New York Academy of Sciences). New York Academy of Sciences, 2006.

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Storyshares. Silencio (Silence - Translation). Story Share, Inc., 2023.

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Storyshares. Silencio (Silence - Translation). Story Share, Inc., 2023.

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Simons, Margaret A. The Silencing of Simone de Beauvoir. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190608811.003.0004.

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This chapter, first published in 1983, initially breaks the news of the scandal of the first English translation of Le deuxième sexe to the English speaking world. Through a painstaking comparative reading of the Parshley translation, published by Knopf, alongside the original French, the chapter reveals the abridgment and editing of the original text with no indication of specific cuts in the text. It shows that Parshley’s version of The Second Sex exhibits a sexist pattern of selection that reduces the impact of Beauvoir’s discussions of women’s history; drastically reduces the number of ref
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Lubenix te ch'aben: Se ha cansado el silencio. Escritores en Lenguas Indígenas, 2011.

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Book chapters on the topic "Translational silencing"

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Paro, Renato, Ueli Grossniklaus, Raffaella Santoro, and Anton Wutz. "RNA-Based Mechanisms of Gene Silencing." In Introduction to Epigenetics. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-68670-3_6.

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AbstractAlthough epigenetic states are typically associated with DNA-methylation and posttranslational histone modifications, RNAs often play an important role in their regulation. Specific examples have already been discussed in the context of dosage compensation (see book ► Chap. 10.1007/978-3-030-68670-3_4 of Wutz) and genomic imprinting (see book ► Chap. 10.1007/978-3-030-68670-3_5 of Grossniklaus). In this Chapter, we will take a closer look at a particular class of RNAs implicated in gene silencing. Although the focus will lie on RNA-based silencing mechanisms in plants, many of its comp
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Gargantini, Pablo R., César G. Prucca, and Hugo D. Luján. "Post-transcriptional Gene Silencing and Translation in Giardia." In Giardia. Springer Vienna, 2011. http://dx.doi.org/10.1007/978-3-7091-0198-8_15.

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Pantuchowicz, Agnieszka. "On Gender Silencing in Translation: A Case Study in Poland." In Language and Literature in a Glocal World. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8468-3_8.

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Pager, Cara T., Karen A. Wehner, Gabriele Fuchs, and Peter Sarnow. "Chapter 5 MicroRNA-Mediated Gene Silencing." In Progress in Molecular Biology and Translational Science. Elsevier, 2009. http://dx.doi.org/10.1016/s1877-1173(09)90005-9.

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Wutz, Anton. "RNA-Mediated Silencing Mechanisms in Mammalian Cells." In Progress in Molecular Biology and Translational Science. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-387685-0.00011-1.

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Cheunsuchon, Pornsuk, Yunli Zhou, Xun Zhang, et al. "Silencing of the ImprintedDLK1-MEG3Locus in Human Clinically Non-Functioning Pituitary Adenomas." In BASIC/TRANSLATIONAL - Pituitary Biology & Tumorigenesis. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p2.p1-391.

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Song, Sang-houn, Kyong-ha So, Yutaka Suzuki, et al. "Analysis of the Expression Profile in 3T3-L1 Adipocytes by shRNA-Mediated Silencing of Adipogenin Gene." In BASIC/TRANSLATIONAL - Endocrine Influence on Adipose Tissue. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part3.p2.p2-426.

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Blackmore, Julia Kristine, Vaishali Chaubal, Sudipan Karmakar, and Carolyn Louise Smith. "The Silencing Mediator of Retinoic Acid and Thyroid Hormone Receptor (SMRT) Coregulator Promotes Breast Carcinogenesis through Multiple Cellular Pathways." In BASIC/TRANSLATIONAL - Hormones & Breast Cancer. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part1.p3.p1-59.

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Garber, Kathryn B., Daniel Gruskin,, and Stephen T. Warren. "FMR1 and the Fragile X Syndrome." In Inborn Errors Of Development. Oxford University PressNew York, NY, 2008. http://dx.doi.org/10.1093/oso/9780195306910.003.0126.

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Abstract Fragile X syndrome (FXS) is an X-linked dominant disorder with reduced penetrance whose primary manifestation is moderate-to- severe mental retardation. It is most often caused by the transcriptional silencing of the FMR1 gene due to an expansion of a CGG-repeat found in the 5’ untranslated region (UTR). This allelic class, with greater than 200 CGG-repeats, is referred to as the full mutation. Normal alleles have up to 54 repeats with the 30 repeats being most common. Intermediate between normal and full mutation alleles are the premutation alleles with 55–200 repeats. Premutation al
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Craig, Nancy L., Rachel Green, Carol Greider, Gisela Storz, Cynthia Wolberger, and Orna Cohen-Fix. "Regulation of transcription." In Molecular Biology. Oxford University Press, 2021. http://dx.doi.org/10.1093/hesc/9780198788652.003.0009.

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This chapter describes the regulation of transcription. A variety of mechanisms are used to repress or activate transcription initiation in bacteria. Transcription can be repressed by blocking the binding of RNA polymerase (Trp repressor, cI). Transcription can be activated by recruiting the polymerase holoenzyme (CAP), stimulating open complex formation (CAP, NtrC), or altering the structure of promoter DNA (MerR). Meanwhile, eukaryotic genes are regulated by co-activator and co-repressor complexes, which are recruited to the DNA by sequence-specific DNA-binding proteins. These complexes typi
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Conference papers on the topic "Translational silencing"

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Leibowitz-Amit, Raya, Liron Zehavi, Dror Avni, and Yehezkel Sidi. "Abstract A17: miRNA silencing in malignant melanoma: Mechanisms, regulation, and potential implications." In Abstracts: AACR International Conference on Translational Cancer Medicine--; Mar 21–24, 2010; Amsterdam, The Netherlands. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1078-0432.tcme10-a17.

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Samanta, R., J. Dunning, A. Taylor, et al. "Severe Respiratory Failure in Adult Influenza Infection Is Characterised by Mechanisms Relating to Pulmonary Endothelial Leak and Interferon Gamma Induced Translational Silencing." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a5288.

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Reports on the topic "Translational silencing"

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Meir, Shimon, Michael Reid, Cai-Zhong Jiang, Amnon Lers, and Sonia Philosoph-Hadas. Molecular Studies of Postharvest Leaf and Flower Abscission. United States Department of Agriculture, 2005. http://dx.doi.org/10.32747/2005.7696523.bard.

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Original objectives: Understanding the regulation of abscission competence by exploring the nature and function of auxin-related gene expression changes in the leaf and pedicelAZs of tomato (as a model system), was the main goal of the previously submitted proposal. We proposed to achieve this goal by using microarray GeneChip analysis, to identify potential target genes for functional analysis by virus-induced gene silencing (VIGS). To increase the potential of accomplishing the objectives of the previously submitted proposal, we were asked by BARD to show feasibility for the use of these two
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