Academic literature on the topic 'Transport neuronal'

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Journal articles on the topic "Transport neuronal"

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Sammler, Esther, Stefan Titz, and Sheriar Hormuzdi. "Neuronal chloride transport tuning." Lancet 385 (February 2015): S85. http://dx.doi.org/10.1016/s0140-6736(15)60400-7.

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JODAL, M. "Neuronal influence on intestinal transport." Journal of Internal Medicine 228, S732 (1990): 125–32. http://dx.doi.org/10.1111/j.1365-2796.1990.tb01484.x.

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Benaïssa, Ibtissem. "Analogie du transport neuronal au transport électronique en nanotechnologie." Journal of Renewable Energies 12, no. 1 (2023): 9–28. http://dx.doi.org/10.54966/jreen.v12i1.115.

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Le système nerveux est formé de deux types de cellules: les cellules gliales et les neurones. Les astrocytes, comme la plupart des cellules gliales, ont longtemps été considérés essentiellement pour leur rôle de support et d’entretien du tissu nerveux. Mais, de plus en plus d’évidences plaident en faveur d’une implication beaucoup plus importante des astrocytes dans la communication nerveuse. Les astrocytes sont couplés les uns aux autres par des ‘gap-jonctions’ à travers lesquels peuvent circuler divers métabolites. C’est par ces jonctions que les astrocytes évacuent vers les capillaires, le
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MENZIKOV, SERGEY A. "NEURONAL MULTIFUNCTIONAL ATPase." Biophysical Reviews and Letters 08, no. 03n04 (2013): 213–27. http://dx.doi.org/10.1142/s1793048013300065.

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Here, we review the properties of a suggested mechanism for a neural ATPase complex based on our recent experimental findings. The mechanism represents a multifunctional ATPase: an enzyme that is a chloride pump and a GABA receptor. This enables new views on the ways Cl - channel transports anions and its regulation by the intra- and extracellular ions and molecules (in particular by glucose, ATP, [Formula: see text]). The hydrolytic activity of this GABA A-coupled ATPase provides the [Formula: see text] transport process the energy and determines a certain direction of ions flux across neuron
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Kaye, D. M., S. D. Wiviott, L. Kobzik, R. A. Kelly, and T. W. Smith. "S-nitrosothiols inhibit neuronal norepinephrine transport." American Journal of Physiology-Heart and Circulatory Physiology 272, no. 2 (1997): H875—H883. http://dx.doi.org/10.1152/ajpheart.1997.272.2.h875.

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Although it has been recently shown that nitric oxide (NO) and its congeners (NO(x)), including nitrosothiols, may modify catecholamine turnover in the brain, it is not known whether NO(x) affect norepinephrine (NE) uptake by sympathetic neurons. The nitrosothiol NO donor S-nitroso-acetylpenicillamine (SNAP, 100 microM for 1 h) elicited a concentration-dependent reduction in desipramine-sensitive [3H]NE uptake into PC-12 cells (66 +/- 3%; P < 0.01) or cultured rat superior cervical ganglia (74 +/- 5%; P < 0.001), whereas desipramine-insensitive [3H]NE uptake was unaffected, indicating a
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Perry, Rotem Ben-Tov, and Mike Fainzilber. "Nuclear transport factors in neuronal function." Seminars in Cell & Developmental Biology 20, no. 5 (2009): 600–606. http://dx.doi.org/10.1016/j.semcdb.2009.04.014.

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Stiess, Michael, and Frank Bradke. "Neuronal transport: myosins pull the ER." Nature Cell Biology 13, no. 1 (2010): 10–11. http://dx.doi.org/10.1038/ncb2147.

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Staff, N. P., E. E. Benarroch, and C. J. Klein. "Neuronal intracellular transport and neurodegenerative disease." Neurology 76, no. 11 (2011): 1015–20. http://dx.doi.org/10.1212/wnl.0b013e31821103f7.

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Brenner, S. R., N. P. Staff, E. E. Benarroch, and C. J. Klein. "Neuronal intracellular transport and neurodegenerative disease." Neurology 77, no. 21 (2011): 1932. http://dx.doi.org/10.1212/wnl.0b013e318239bf96.

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Bakshi, Rachit, Shuchi Mittal, Zhixiang Liao та Clemens R. Scherzer. "A Feed-Forward Circuit of EndogenousPGC-1αandEstrogen Related Receptor αRegulates the Neuronal Electron Transport Chain". Parkinson's Disease 2016 (2016): 1–9. http://dx.doi.org/10.1155/2016/2405176.

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Peroxisome proliferator-activated receptor γcoactivator 1α(PGC-1α) is a central regulator of cellular and mitochondrial metabolism. Cellular bioenergetics are critically important in “energy-guzzling” neurons, but the components and wiring of the transcriptional circuit through whichPGC-1αregulates the neuronal electron transport chain have not been established. This information may be vital for restoring neuronal bioenergetics gene expression that is compromised during incipient Parkinson’s neuropathology and in aging-dependent brain diseases. Here we delineate a neuronal transcriptional circ
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Dissertations / Theses on the topic "Transport neuronal"

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MacAskill, A. F. N. "Control of neuronal mitochondrial transport." Thesis, University College London (University of London), 2010. http://discovery.ucl.ac.uk/19495/.

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Synapses consume large amounts of energy, and energy supply to synaptic sites is critical for their proper function. Most energy in the brain is supplied by mitochondria, organelles efficient at utilising oxygen and substrates such as glucose and pyruvate to produce cellular energy in the form of ATP. Due to the large size of many neurons - which precludes the rapid diffusion of ATP from one side of the cell to the other - mitochondria must be positioned close to activated synaptic sites. There must therefore be transport pathways that allow mitochondria to move throughout the cell. As pattern
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Mahato, Deependra. "Mutation of Polaris, an Intraflagellar Transport Protein, Shortens Neuronal Cilia." Thesis, University of North Texas, 2005. https://digital.library.unt.edu/ark:/67531/metadc4856/.

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Primary cilia are non-motile organelles having 9+0 microtubules that project from the basal body of the cell. While the main purpose of motile cilia in mammalian cells is to move fluid or mucus over the cell surface, the purpose of primary cilia has remained elusive for the most part. Primary cilia are shortened in the kidney tubules of Tg737orpk mice, which have polycystic kidney disease due to ciliary defects. The product of the Tg737 gene is polaris, which is directly involved in a microtubule-dependent transport process called intraflagellar transport (IFT). In order to determine the imp
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Chen, Liang. "Single molecule and single particle studies of neuronal axonal transport /." May be available electronically:, 2009. http://proquest.umi.com/login?COPT=REJTPTU1MTUmSU5UPTAmVkVSPTI=&clientId=12498.

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Johnson, Christopher M. "Investigating the Slow Axonal Transport of Neurofilaments: A Precursor for Optimal Neuronal Signaling." Ohio University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1452018547.

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Pekergin, Mehmet Ferhan. "Optimisation combinatoire par le calcul neuronal et parallelisme optimal." Paris 5, 1992. http://www.theses.fr/1992PA05S017.

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Cette these constituee de deux parties aborde l'efficacite du parallelisme suivant deux approches complementaires. Dans la premiere partie l'architecture neuronale est consideree comme une architecture massivement parallele sans contrainte de communication et deux methodes fondees sur le calcul neuronal appliquees a l'optimisation combinatoire sont etudiees. La methodologie de l'optimisation par les reseaux de neurones aleatoires est developpee et une methode deterministe utilisant des reseaux dynamiques est proposee pour le probleme du stable maximum. Dans le second travail l'approche de la m
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Coats, Charles Jason. "Development of primary neuronal culture of embryonic rabbit dorsal root ganglia for microfluidic chamber analysis of axon mediated neuronal spread of Bovine Herpesvirus type 1." Thesis, Manhattan, Kan. : Kansas State University, 2010. http://hdl.handle.net/2097/4115.

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Li, Yunyun [Verfasser], and Peter [Akademischer Betreuer] Hänggi. "Noise assisted transport in artificial channels and neuronal membranes / Yunyun Li. Betreuer: Peter Hänggi." Augsburg : Universität Augsburg, 2011. http://d-nb.info/1077700296/34.

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Copp, Steven Wesley. "Enzymatic regulation of skeletal muscle oxygen transport: novel roles for neuronal nitric oxide synthase." Diss., Kansas State University, 2013. http://hdl.handle.net/2097/15512.

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Doctor of Philosophy<br>Department of Anatomy and Physiology<br>Timothy I. Musch<br>Nitric oxide (NO) is synthesized via distinct NO synthase (NOS) enzymes and constitutes an essential cardiovascular signaling molecule. Whereas important vasomotor contributions of endothelial NOS (eNOS) have been well-described, the specific vasomotor contributions of nNOS-derived NO in healthy subjects during exercise are unknown. The purpose of this dissertation is to test the global hypothesis that nNOS-derived NO is a critical regulator of exercising skeletal muscle vascular control. Specifically, we ut
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Naudon, Laurent. "Recherche d'une participation du transporteur neuronal de la dopamine et du transporteur vésiculaire à l'adaptation neuronale." Rouen, 1994. http://www.theses.fr/1994ROUES066.

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L'ensemble des résultats que nous avons obtenus semblent indiquer que le transporteur neuronal de la dopamine et le transporteur vésiculaire des monoamines, malgré leurs rôles essentiels dans la transmission synaptique, ne participent que faiblement à l'adaptation des neurones dopaminergiques
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Daoust, Alexia. "IRM du manganèse (MEMRI) : couplage à l'imagerie chimique par microsonde synchrotron pour optimiser l'imagerie fonctionnelle du transport neuronal." Phd thesis, Université de Grenoble, 2012. http://tel.archives-ouvertes.fr/tel-00770158.

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Résumé Le manganèse (Mn2+) est un élément essentiel du corps humain. Ses propriétés paramagnétiques permettent son utilisation comme agent de contraste pour l'IRM (Mn-MRI ou MEMRI). Analogue du calcium (Ca2+), il pénètre les neurones essentiellement par les canaux calciques. Il est ensuite transporté le long des microtubules jusqu'aux synapses où il est libéré, puis capturé par les autres neurones. Ainsi, il peut rendre compte du transport axonal antérograde et rétrograde. L'approche MEMRI peut ainsi apporter des informations uniques sur la connectivité fonctionnelle cérébrale. Toutefois, deux
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Books on the topic "Transport neuronal"

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Gribkoff, Valentin K. Structure, function, and modulation of neuronal voltage-gated ion channels. Wiley, 2009.

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1955-, Gonzalez-Lima Francisco, ed. Cytochrome oxidase in neuronal metabolism and Alzheimer's disease. Plenum Press, 1998.

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Veli, Himanen, Nijkamp Peter, Reggiani Aura, and Raitio Juha, eds. Neural networks in transport applications. Ashgate, 1998.

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A, Lappi Douglas, ed. Suicide transport and immunolesioning. R.G. Landes, 1994.

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Murdoch, Ritchie J., Keynes R. D, and Bolis Liana, eds. Ion channels in neural membranes: Proceedings of the 11th International Conference on Biological Membranes held at Crans-sur-Sierre, Switzerland, June 10-14, 1985. A.R. Liss, 1986.

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J, Alvarez-Leefmans F., Russell John M. 1942-, and International Brain Research Organization. Congress, eds. Chloride channels and carriers in nerve, muscle, and glial cells. Plenum Press, 1990.

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Suter, Daniel M., and Kyle E. Miller, eds. Neuronal Mechanics and Transport. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-823-8.

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Neuronal cytoskeleton: Morphogenesis, transport and synaptic transmission. Japan Scientific Societies Press, 1993.

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Elam, John. Axonal Transport in Neuronal Growth and Regeneration. Springer, 2013.

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Elam, John. Axonal Transport in Neuronal Growth and Regeneration. Springer London, Limited, 2013.

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Book chapters on the topic "Transport neuronal"

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Knight, Adam L., Yanmin Chen, Tao Sun, and Zu-Hang Sheng. "Neuronal Mitochondrial Transport." In The Functions, Disease-Related Dysfunctions, and Therapeutic Targeting of Neuronal Mitochondria. John Wiley & Sons, Inc, 2015. http://dx.doi.org/10.1002/9781119017127.ch7.

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Voelzmann, André, and Natalia Sanchez-Soriano. "Drosophila Primary Neuronal Cultures as a Useful Cellular Model to Study and Image Axonal Transport." In Methods in Molecular Biology. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-1990-2_23.

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AbstractThe use of primary neuronal cultures generated from Drosophila tissue provides a powerful model for studies of transport mechanisms. Cultured fly neurons provide similarly detailed subcellular resolution and applicability of pharmacology or fluorescent dyes as mammalian primary neurons. As an experimental advantage for the mechanistic dissection of transport, fly primary neurons can be combined with the fast and highly efficient combinatorial genetics of Drosophila, and genetic tools for the manipulation of virtually every fly gene are readily available. This strategy can be performed
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Atkin, Talia A., Andrew F. MacAskill, and Josef T. Kittler. "Neuronal Mitochondrial Transport and Dysfunction." In Mitochondrial Dysfunction in Neurodegenerative Disorders. Springer London, 2011. http://dx.doi.org/10.1007/978-0-85729-701-3_10.

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Stephenson, F. Anne, and Kieran Brickley. "Mechanisms of Neuronal Mitochondrial Transport." In Folding for the Synapse. Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-7061-9_6.

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Deitmer, Joachim W. "pH regulation and acid/base-mediated transport in glial cells." In Glial ⇔ Neuronal Signaling. Springer US, 2004. http://dx.doi.org/10.1007/978-1-4020-7937-5_10.

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Ledeen, R. W., D. A. Aquino, M. Sbaschnig-Agler, C. M. Gammon, and K. K. Vaswani. "Fundamentals of Neuronal Transport of Gangliosides. Functional Implications." In Gangliosides and Modulation of Neuronal Functions. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71932-5_21.

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Caputto, R., B. L. Caputto, M. S. Domowicz, and S. C. Kivatinitz. "Gangliosides: Biosynthesis, Transport and Location in the Plasma Membrane." In Gangliosides and Modulation of Neuronal Functions. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71932-5_20.

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Pichiule, P., J. C. Chavez, R. J. Przybylski, and J. C. LaManna. "Increase of Neuronal Nitric Oxide Synthase during Chronic Hypoxia." In Oxygen Transport to Tissue XX. Springer US, 1998. http://dx.doi.org/10.1007/978-1-4615-4863-8_37.

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Karpova, Anna, Maria Andres-Alonso, Katarzyna M. Grochowska, and Michael R. Kreutz. "Protein Transport from Synapse-to-Nucleus and the Regulation of Gene Expression." In Transcriptional Regulation by Neuronal Activity. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-68550-7_3.

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Miller, Richard J. "Modulation and Functions of Neuronal Ca2+ Permeable Channels." In Calcium Transport and Intracellular Calcium Homeostasis. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-83977-1_1.

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Conference papers on the topic "Transport neuronal"

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Maria, Stavrou, Heffernan Áine, Carter Roderick, et al. "Astrocytes control motor neuronal axonal transport deficits in C9ORF72 ALS." In ABN 2024 annual meeting abstracts. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/jnnp-2024-abn.165.

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Zhu, Liang, and Robert Flower. "Role of Vasomotion in Control of Retina Edema in Diabetic Retinopathy: Quantification of Fluid Transport Through Retinal Capillaries." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-189507.

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Diabetic retinopathy refers to diabetes-related complications in the retina, It is a progressive disease and its symptoms in the eyes can vary from non-vision threatening to vision loss, and it can lead to permanent damage to the neuronal retinal tissue. The irreversible nature of the damage suggests that prevention of diabetes by eliminating risk factors and early screening are the cornerstone of relevant treatment to stop or limit visual damage in those patients.
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Smith, Jennifer A., Gregoriy Smiyun, Leslie Wilson, Stuart Feinstein, and Mary Ann Jordan. "Abstract 5294: Inhibition of mitochondrial transport in neuronal cells by microtubule-targeting drugs eribulin, ixabepilone, paclitaxel, and vincristine." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-5294.

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Lee, Sung Jin, Jingjing Sun, Michael King, Huikai Xie, and Malisa Sarntinoranont. "Viscoelastic Property Changes of Acute Rat Brain Tissue Slices as a Function of Cell Viability." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53909.

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Changes in mechanical properties within brain tissues after losses in cell viability have not been well investigated. Lack of oxygen and nutrient transport can induce hypoxic neuronal injury and increase cell membrane permeability, and cell membranes and matrix components can lose their structural and mechanical integrity. These physical changes may have an effect on mechanical properties of brain tissue [1]. In this study, the viscoelastic behavior of two anatomical regions (cerebral cortex and hippocampus) in acute rat brain tissue slices were measured as a function of cell viability using i
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Stork, Larissa Rosa, Lucca Stephani Ribeiro, Izabella Savergnini Deprá, Luísa D’Ávila Camargo, and Maria Angélica Santos Novaes. "Tau protein and its role in Alzheimer’s disease physiopathology: a literature review." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.132.

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Background: Alzheimer’s disease (AD) is a neurodegenerative disorder characterized by a double proteinopathy: deposition of amyloid-β into plaques and hyperphosphorylation of Tau protein. Objectives: To understand the genetic and molecular aspects of Tau protein and its relationship with Alzheimer’s disease. Methods: We conducted a systematic literature search using Pubmed/ MEDLINE and ClinicalKey databases, applying the descriptors: “Alzheimer Disease” AND “Tau proteins’’ AND Tauopathies, during July and August of 2020. The inclusion criteria were English and Portuguese articles published bet
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Brooks, Joseph Bruno Bidin. "De novo variant in the MAPK8IP3 gene in the differential diagnosis of global development delay. Case report." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.181.

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Context: The global development delay has a high prevalence and heterogeneity in the world population. With the advancement of technology and detection of pathogenic variants detected by sequencing the exome, genes related to global developmental delay could be identified and collaborate for further clinical clarification. Among the studied genes, the MAPK8IP3 gene, became an attractive candidate due to its performance in neuronal axonal transport in vertebrates and invertebrates. This case report was approved by the Ethics Committee of Universidade Metropolitana de Santos. Case Report: The pr
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Garrido Rodriguez, Maria Concepcion. "Análisis de la calidad del servicio en el transporte público mediante redes neuronales artificiales." In CIT2016. Congreso de Ingeniería del Transporte. Universitat Politècnica València, 2016. http://dx.doi.org/10.4995/cit2016.2016.4120.

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El éxito de un servicio de transporte público reside en su capacidad para captar nuevos pasajeros y de fidelizar el uso de los actuales. De ahí que las administraciones y los gestores de transporte se preocupen cada vez más por conocer cuál es la calidad percibida por los usuarios. Aunque existen numerosos métodos matemáticos que ya han sido utilizados para analizar la calidad del servicio en el transporte público, es necesario seguir avanzando en el estudio de nuevas técnicas que sean válidas para su estudio. En esta ponencia se aplica la técnica de las redes neuronales artificiales al estudi
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Cruz, Ortiz G. A., and Pavel Vorobiev. "PERFORMANCE SIMULATION OF A AI-BASED CONTROLLER FOR SOLAR TRACKERS." In Intelligent transport systems. Russian University of Transport, 2024. http://dx.doi.org/10.30932/9785002446094-2024-181-184.

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Solar trackers are a useful tool to increase the energy production of solar panels by orienting them towards the sun. In these devices it is possible to implement intelligent functions such as protection against strong gusts of wind, assisted cleaning with rain or dew, optimization of energy production with cloudy skies and proactive maintenance with issue resolution. In recent years, machine learning has gained great popularity due to its applicability in a wide variety of areas for non-trivial solving algorithms. In this work, machine learning features were implemented in a solar tracker pro
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Mudrakola, Harsha V., Chengbiao Wu, Kai Zhang, and Bianxiao Cui. "Single Molecule Imaging of Axonal Transport in Live Neurons." In Laser Science. OSA, 2009. http://dx.doi.org/10.1364/ls.2009.lsthb3.

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Yang, Chun-Lin, Nandan Shettigar, and C. Steve Suh. "A Proposition for Describing Real-World Network Dynamics." In ASME 2021 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/imece2021-73360.

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Abstract This study presents a proposition for describing the dynamics of real-world networks under the general framework of complex networks. Outward behaviors of complex networks are the manifestation of the coupled dynamics at the macroscopic level and the individual dynamics at the microscopic level. At the macroscopic level a law of coupling governs the interactions of network constituents. At the microscopic level, the dynamics of individual constituent is defined by energy that follows normal distribution. Constituent dynamics are bounded by physical constraints. Consequently, network d
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