Relevant bibliographies by topics / Transports paracellulaire

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Academic literature on the topic 'Transports paracellulaire'

Author: Grafiati
Published: 4 June 2021
Last updated: 11 February 2022

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Journal articles on the topic "Transports paracellulaire"

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Van Itallie, Christina M., and James M. Anderson. "CLAUDINS AND EPITHELIAL PARACELLULAR TRANSPORT." Annual Review of Physiology 68, no. 1 (2006): 403–29. http://dx.doi.org/10.1146/annurev.physiol.68.040104.131404.

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Knöpfel, Thomas, Nina Himmerkus, Dorothee Günzel, Markus Bleich, Nati Hernando, and Carsten A. Wagner. "Paracellular transport of phosphate along the intestine." American Journal of Physiology-Gastrointestinal and Liver Physiology 317, no. 2 (2019): G233—G241. http://dx.doi.org/10.1152/ajpgi.00032.2019.

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Inorganic phosphate (Pi) is crucial for many biological functions, such as energy metabolism, signal transduction, and pH buffering. Efficient systems must exist to ensure sufficient supply for the body of Pi from diet. Previous experiments in humans and rodents suggest that two pathways for the absorption of Pi exist, an active transcellular Pi transport and a second paracellular pathway. Whereas the identity, role, and regulation of active Pi transport have been extensively studied, much less is known about the properties of the paracellular pathway. In Ussing chamber experiments, we charact
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TYSNES, KRISTOFFER R., and LUCY J. ROBERTSON. "Investigation of effects ofGiardia duodenalison transcellular and paracellular transport in enterocytes usingin vitroUssing chamber experiments." Parasitology 142, no. 5 (2014): 691–97. http://dx.doi.org/10.1017/s0031182014001772.

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SUMMARYThe mechanisms by which different genotypes ofGiardia duodenalisresult in different symptoms remain unresolved. In particular, we lack detailed knowledge on which transport mechanisms (transcellular or paracellular) are affected by differentGiardiaisolates. Using horse radish peroxidase (HRP) and creatinine as transcellular and paracellular probes, respectively, we developed a robust assay that can be used with an Ussing chamber to investigate epithelial transport, as well as short-circuit current as an indicator of net ion transport. We investigated 2Giardiaisolates, both Assemblage A,
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Thongon, Narongrit, La-iad Nakkrasae, Jirawan Thongbunchoo, Nateetip Krishnamra, and Narattaphol Charoenphandhu. "Prolactin stimulates transepithelial calcium transport and modulates paracellular permselectivity in Caco-2 monolayer: mediation by PKC and ROCK pathways." American Journal of Physiology-Cell Physiology 294, no. 5 (2008): C1158—C1168. http://dx.doi.org/10.1152/ajpcell.00020.2008.

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Prolactin (PRL) was previously demonstrated to rapidly enhance calcium absorption in rat duodenum and the intestine-like Caco-2 monolayer. However, its mechanism was not completely understood. Here, we investigated nongenomic effects of PRL on the transepithelial calcium transport and paracellular permselectivity in the Caco-2 monolayer by Ussing chamber technique. PRL increased the transcellular and paracellular calcium fluxes and paracellular calcium permeability within 60 min after exposure but decreased the transepithelial resistance of the monolayer. The effects of PRL could not be inhibi
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García, Néstor H., Carla R. Ramsey, and Franklyn G. Knox. "Understanding the Role of Paracellular Transport in the Proximal Tubule." Physiology 13, no. 1 (1998): 38–43. http://dx.doi.org/10.1152/physiologyonline.1998.13.1.38.

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Fluid and solute reabsorption by the proximal tubule is the result of both transcellular and paracellular flux. The role of transcellular transport has been extensively studied, but the importance of paracellular flux has not been as thoroughly investigated. The purpose of this review is to update concepts about the contribution of paracellular transport for reabsorption by the proximal tubule.
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Angelow, Susanne, and Alan SL Yu. "Claudins and paracellular transport: an update." Current Opinion in Nephrology and Hypertension 16, no. 5 (2007): 459–64. http://dx.doi.org/10.1097/mnh.0b013e32820ac97d.

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Montalbetti, Nicolas, Anna C. Rued, Dennis R. Clayton, et al. "Increased urothelial paracellular transport promotes cystitis." American Journal of Physiology-Renal Physiology 309, no. 12 (2015): F1070—F1081. http://dx.doi.org/10.1152/ajprenal.00200.2015.

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Changes in the urothelial barrier are observed in patients with cystitis, but whether this leads to inflammation or occurs in response to it is currently unknown. To determine whether urothelial barrier dysfunction is sufficient to promote cystitis, we employed in situ adenoviral transduction to selectively overexpress the pore-forming tight junction-associated protein claudin-2 (CLDN-2). As expected, the expression of CLDN-2 in the umbrella cells increased the permeability of the paracellular route toward ions, but not to large organic molecules. In vivo studies of bladder function revealed h
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Ayala-Torres, Carlos, Susanne M. Krug, Jörg D. Schulzke, Rita Rosenthal, and Michael Fromm. "Tricellulin Effect on Paracellular Water Transport." International Journal of Molecular Sciences 20, no. 22 (2019): 5700. http://dx.doi.org/10.3390/ijms20225700.

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In epithelia, large amounts of water pass by transcellular and paracellular pathways, driven by the osmotic gradient built up by the movement of solutes. The transcellular pathway has been molecularly characterized by the discovery of aquaporin membrane channels. Unlike this, the existence of a paracellular pathway for water through the tight junctions (TJ) was discussed controversially for many years until two molecular components of paracellular water transport, claudin-2 and claudin-15, were identified. A main protein of the tricellular TJ (tTJ), tricellulin, was shown to be downregulated i
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Hou, Jianghui. "Paracellular transport in the collecting duct." Current Opinion in Nephrology and Hypertension 25, no. 5 (2016): 424–28. http://dx.doi.org/10.1097/mnh.0000000000000253.

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Venn, Alexander A., Coralie Bernardet, Apolline Chabenat, Eric Tambutté, and Sylvie Tambutté. "Paracellular transport to the coral calcifying medium: effects of environmental parameters." Journal of Experimental Biology 223, no. 17 (2020): jeb227074. http://dx.doi.org/10.1242/jeb.227074.

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ABSTRACTCoral calcification relies on the transport of ions and molecules to the extracellular calcifying medium (ECM). Little is known about paracellular transport (via intercellular junctions) in corals and other marine calcifiers. Here, we investigated whether the permeability of the paracellular pathway varied in different environmental conditions in the coral Stylophora pistillata. Using the fluorescent dye calcein, we characterised the dynamics of calcein influx from seawater to the ECM and showed that increases in paracellular permeability (leakiness) induced by hyperosmotic treatment c
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Dissertations / Theses on the topic "Transports paracellulaire"

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Tria, Scherrine. "Novel in vitro models for pathogen detection based on organic transistors integrated with living cells." Phd thesis, Ecole Nationale Supérieure des Mines de Saint-Etienne, 2013. http://tel.archives-ouvertes.fr/tel-00972057.

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In biological systems, different tissues have evolved to form a barrier. An example is the intestinal epithelium, consisting of a single layer of cells lining the wall of the stomach and colon. It restricts the passage of harmful chemicals or pathogens from the light into the tissue, while selectively absorbing the most nutrients, electrolytes and water are necessary for the host. Tight junctions are structures which limit the passage of the material through the space between the cells. The ability to measure the paracellular and transcellular transport is of vital importance because it provid
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Rasmussen, Julia E. Anderson James M. "Claudins and regulation of the paracellular transport system." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2006. http://dc.lib.unc.edu/u?/etd,330.

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Thesis (M.S.)--University of North Carolina at Chapel Hill, 2006.<br>Title from electronic title page (viewed Oct. 10, 2007). "... in partial fulfillment of the requirements for the degree of Master of Science in the Department of Cell and Molecular Physiology." Discipline: Cell and Molecular Physiology; Department/School: Medicine.
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Chittchang, Montakarn Johnston Thomas P. "Effect of secondary structure on paracellular transport of polypeptides." Diss., UMK access, 2004.

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Thesis (Ph. D.)--School of Pharmacy and Dept. of Chemistry. University of Missouri--Kansas City, 2004.<br>"A dissertation in pharmaceutical sciences and chemistry." Advisor: Thomas P. Johnston. Typescript. Vita. Description based on contents viewed Feb. 23, 2006; title from "catalog record" of the print edition. Includes bibliographical references (leaves 202-223). Online version of the print edition.
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Ramakrishnan, Suresh Krishna. "Studies on renal paracellular transport in health and disease." Paris 6, 2013. http://www.theses.fr/2013PA066297.

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Le récepteur du calcium (CaSR), exprimé par les cellules parathyroïdiennes, contrôle la calcémie en régulant la sécrétion d’hormone parathyroïdienne (PTH). Afin de comprendre le rôle extra-parathyroïdien du CaSR, nous avons utilisé un modèle de rat thyro-parathyroïdectomisé, recevant de manière prolongée une dose fixe de PTH exogène. Ainsi, l’inhibition chronique du CaSR était associée à une augmentation de la reabsorption tubulaire rénale du calcium et à une augmentation de la calcémie ionisée. . Par ailleurs, nous avons démontré que le CaSR était uniquement exprimé dans la branche large asce
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Collares, Buzato Carla Beatriz. "Modulation of paracellular permeability and intercellular junctions in cultured epithelia." Thesis, University of Newcastle Upon Tyne, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.283019.

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Tavelin, Staffan. "New Approaches to Studies of Paracellular Drug Transport in Intestinal Epithelial Cell Monolayers." Doctoral thesis, Uppsala University, Department of Pharmacy, 2003. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-3388.

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<p>Studies of intestinal drug permeability have traditionally been performed in the colon-derived Caco-2 cell model. However, the permeability of these cell monolayers resembles that of the colon rather than that of the small intestine, which is the major site of drug absorption following oral administration. One aim of this thesis was therefore to develop a new cell culture model that mimics the permeability of the small intestine. 2/4/A1 cells are conditionally immortalized with a temperature sensitive mutant of SV40T. These cells proliferate and form multilayers at 33°C. At cultivation temp
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Noach, Arthur Bernard Joseph. "Enhancement of paracellular drug transport across epithelia : in vitro and in vivo studies /." Leiden : Leiden University, Leiden/Amsterdam Center for Drug Research, Division of Pharmacology, 1994. http://www.gbv.de/dms/bs/toc/183781511.pdf.

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Krishan, Mansi. "Enhanced Intranasal Delivery of Gemcitabine to the Central Nervous System." University of Cincinnati / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1384850749.

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Marais, Etienne Barend. "Permeation of excised intestinal tissue by insulin released from Eudragit® L100/Trimethyl chitosan chloride microspheres /E.B. Marais." Thesis, North-West University, 2013. http://hdl.handle.net/10394/9676.

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The purpose of this research project was to develop and characterise matrix type microspheres prepared from Eudragit® L100, containing insulin as model peptide drug as well as an absorption enhancer, N-trimethyl chitosan chloride (TMC), to improve intestinal absorption via the paracellular route. Insulin loaded microspheres were prepared using a single water in oil emulsification/evaporation method in accordance with a fractional factorial design (23) and subsequently characterised in terms of morphology as well as internal structure. Also, insulin and TMC loading were determined using a high
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Khan, Ambreen Ayaz. "The design of novel nano-sized polyanion-polycation complexes for oral protein delivery." Thesis, University of Hertfordshire, 2014. http://hdl.handle.net/2299/13773.

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Introduction Oral delivery of proteins faces numerous challenges due to their enzymatic susceptibility and instability in the gastrointestinal tract. In recent years, the polyelectrolyte complexes have been explored for their ability to complex protein and protect them against chemical and enzymatic degradation. However, most of the conventional binary polyelectrolyte complexes (PECs) are formed by polycations which are associated with toxicity and non-specific bio-interactions. The aim of this thesis was to prepare a series of ternary polyelectrolyte complexes (APECs) by introduction of a pol
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Books on the topic "Transports paracellulaire"

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Bockenhauer, Detlef, and Robert Kleta. Approach to the patient with renal Fanconi syndrome, glycosuria, or aminoaciduria. Edited by Robert Unwin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0041_update_001.

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Up to 80% of filtered salt and water is returned back into the circulation in the proximal tubule. Several solutes, such as phosphate, glucose, low-molecular weight proteins, and amino acids are exclusively reabsorbed in this segment, so their appearance in urine is a sign of proximal tubular dysfunction. An entire orchestra of specialized apical and basolateral transporters, as well as paracellular molecules, mediate this reabsorption. Defects in proximal tubular function can be isolated (e.g. isolated renal glycosuria, aminoacidurias, or hypophosphataemic rickets) or generalized. In the latt
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Houillier, Pascal. Magnesium homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0027.

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Magnesium is critically important in the process of energy release. Although most magnesium is stored outside the extracellular fluid compartment, the regulated concentration appears in blood. Urinary magnesium excretion can decrease rapidly to low values when magnesium entry rate into the extracellular fluid volume is low, which has several important implications: cell and bone magnesium do not play a major role in the defence of blood magnesium concentration; while a major role is played by the kidney and especially the renal tubule, which adapts to match the urinary magnesium excretion and
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Book chapters on the topic "Transports paracellulaire"

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Larsen, Erik Hviid, and Jens Nørkær Sørensen. "Stationary and Nonstationary Ion and Water Flux Interactions in Kidney Proximal Tubule: Mathematical Analysis of Isosmotic Transport by a Minimalistic Model." In Reviews of Physiology, Biochemistry and Pharmacology. Springer International Publishing, 2019. http://dx.doi.org/10.1007/112_2019_16.

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AbstractOur mathematical model of epithelial transport (Larsen et al. Acta Physiol. 195:171–186, 2009) is extended by equations for currents and conductance of apical SGLT2. With independent variables of the physiological parameter space, the model reproduces intracellular solute concentrations, ion and water fluxes, and electrophysiology of proximal convoluted tubule. The following were shown: Water flux is given by active Na+ flux into lateral spaces, while osmolarity of absorbed fluid depends on osmotic permeability of apical membranes. Following aquaporin “knock-out,” water uptake is not reduced but redirected to the paracellular pathway. Reported decrease in epithelial water uptake in aquaporin-1 knock-out mouse is caused by downregulation of active Na+ absorption. Luminal glucose stimulates Na+ uptake by instantaneous depolarization-induced pump activity (“cross-talk”) and delayed stimulation because of slow rise in intracellular [Na+]. Rate of fluid absorption and flux of active K+ absorption would have to be attuned at epithelial cell level for the [K+] of the absorbate being in the physiological range of interstitial [K+]. Following unilateral osmotic perturbation, time course of water fluxes between intraepithelial compartments provides physical explanation for the transepithelial osmotic permeability being orders of magnitude smaller than cell membranes’ osmotic permeability. Fluid absorption is always hyperosmotic to bath. Deviation from isosmotic absorption is increased in presence of glucose contrasting experimental studies showing isosmotic transport being independent of glucose uptake. For achieving isosmotic transport, the cost of Na+ recirculation is predicted to be but a few percent of the energy consumption of Na+/K+ pumps.
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Shachar-Hill, Bruria, and Adrian E. Hill. "Paracellular fluid transport by epithelia." In International Review of Cytology. Elsevier, 2002. http://dx.doi.org/10.1016/s0074-7696(02)15014-5.

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Knoers, Nine V. A. M., and Elena N. Levtchenko. "Disorders of tubular electrolyte handling." In Oxford Textbook of Medicine, edited by John D. Firth. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0506.

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Glycosuria—glucose reabsorption in the proximal tubule is carried out by two different pairs of apical Na<sup>+</sup>-dependent (SGLT1 and -2) and basolateral Na<sup>+</sup>-independent (GLUT1 and -2) glucose transporters. Abnormalities in renal glucose transport can be seen in association with other defects of proximal tubular transport. Familial renal glycosuria is a rare autosomal recessive condition caused by mutations in the SGLT2-encoding gene, SLC5A2. Phosphate-handling disorders—the plasma concentration of inorganic phosphate depends on the balance between intestinal absorption, renal excretion, and the internal contribution from bone. Changes of serum phosphate levels can be caused by numerous inherited and acquired conditions. Disorders associated with increased urinary phosphate excretion and low serum phosphate levels produce symptoms that mainly affect the bones: rickets in children and osteomalacia in adults. Magnesium-handling disorders—normal plasma magnesium concentration is achieved by variation of urinary magnesium excretion in response to altered uptake by the intestine. The main site of magnesium absorption is the small bowel, via paracellular simple diffusion at high intraluminal concentrations, and via active transcellular uptake through the magnesium channel TRPM6 at low concentrations. Regulation and fine-tuning of serum magnesium concentration occurs primarily in the kidney. Genetic disorders of magnesium handling include Gitelman’s syndrome. Aminoaciduria and renal Fanconi’s syndrome—most amino acids (except for tryptophan, which is protein bound) are freely filtered by the glomerulus, after which 95 to 99.9% are reabsorbed in the proximal tubules by apical Na<sup>+</sup>-dependent cotransporters and Na<sup>+</sup>-independent cotransporters. Aminoaciduria is defined as urinary excretion of more than 5% of the filtered load of an amino acid. Renal Fanconi’s syndrome is characterized by a generalized defect of both Na<sup>+</sup>-coupled and receptor-mediated proximal tubular transport.
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"Implications of Transport via the Paracellular Pathway on Drug Development." In Tight Junctions. CRC Press, 2001. http://dx.doi.org/10.1201/9781420038538-33.

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Smith, Philip, and Chao-Pin Lee. "Implications of Transport via the Paracellular Pathway on Drug Development." In Tight Junctions. CRC Press, 2001. http://dx.doi.org/10.1201/9781420038538.ch31.

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Yen, Wan-Ching, and Vincent H. L. Lee. "Paracellular transport of a proteolytically labile pentapeptide across the colonic and other intestinal segments of the albino rabbit: implications for peptide drug design." In Advances in Drug Delivery Systems, 6. Elsevier, 1994. http://dx.doi.org/10.1016/b978-0-444-82027-3.50014-6.

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Conference papers on the topic "Transports paracellulaire"

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Chandrasena, AR, G. Ho, L. Araujo, Y. Mao, J. Pan, and JA Frank. "Regulation of Paracellular Transport by Differential Claudin Expression in Alveolar Epithelial Cells." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a3560.

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