Academic literature on the topic 'Trauma shock'

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Journal articles on the topic "Trauma shock"

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Scherer, Priscilla. "Shock Trauma." American Journal of Nursing 89, no. 11 (November 1989): 1440. http://dx.doi.org/10.2307/3426144.

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Willard, Louise A. "Shock Trauma!" Journal of Christian Nursing 10, no. 3 (1993): 26–29. http://dx.doi.org/10.1097/00005217-199310030-00009.

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SCHERER, PRISCILLA. "SHOCK TRAUMA." AJN, American Journal of Nursing 89, no. 11 (November 1989): 1440–45. http://dx.doi.org/10.1097/00000446-198911000-00016.

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Bhubaneswari, T. S., and Ajeet Singh. "Irony as Trauma & Trauma as Irony in Claude Lanzmann’s Shoah." Contemporary Research: An Interdisciplinary Academic Journal 3, no. 1 (December 31, 2019): 67–71. http://dx.doi.org/10.3126/craiaj.v3i1.27492.

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Claude Lanzmann makes use of what this essay posits as metairony which dramatizes the shocks of the acting out of the trauma of the Holocaust. The film director makes the survivors and witnesses and the viewers to become retraumatized and to relive the past. By so doing, the traumatized mind can cope with the trauma because acting out helps the reflective consciousness to prevent itself from being overwhelmed by shock, in Walter Benjamin’s assumption, by reproducing shock, that is, by seizing upon each traumatic moment and parrying it - in effect, by responding to violence with violence. Testimonies in Shoah break the boundary between the experience of shock and experience as shock.
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Armstrong, Bruce, and Julia Carpenter. "Shock trauma Baltimore, USA." Intensive and Critical Care Nursing 11, no. 3 (June 1995): 151–56. http://dx.doi.org/10.1016/s0964-3397(95)80657-1.

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Dillworth, Judy, and Julie Mill Strange. "Shock Trauma Care Plans." American Journal of Nursing 89, no. 1 (January 1989): 140. http://dx.doi.org/10.2307/3471034.

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Dutton, Richard P. "SHOCK AND TRAUMA ANESTHESIA." Anesthesiology Clinics of North America 17, no. 1 (March 1999): 83–95. http://dx.doi.org/10.1016/s0889-8537(05)70080-8.

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Norris, H. Thomas. "Trauma, Sepsis, and Shock." American Journal of Surgical Pathology 13, no. 6 (June 1989): 530. http://dx.doi.org/10.1097/00000478-198906000-00019.

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Ohnishi, Mitsuo. "Shock Wave Trauma Research." Impact 2019, no. 3 (March 22, 2019): 76–78. http://dx.doi.org/10.21820/23987073.2019.3.76.

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DILLWORTH, JUDY. "Shock Trauma Care Plans." AJN, American Journal of Nursing 89, no. 1 (January 1989): 140. http://dx.doi.org/10.1097/00000446-198901000-00036.

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Dissertations / Theses on the topic "Trauma shock"

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Nandra, Kiran Kaur. "Novel therapeutic approaches for experimental trauma-haemorrhage." Thesis, Queen Mary, University of London, 2013. http://qmro.qmul.ac.uk/xmlui/handle/123456789/8470.

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Haemorrhagic shock (HS) is commonly associated with trauma. Severe haemorrhage causes hypoperfusion of tissues resulting in a global ischaemic state, and resuscitation is performed to restore circulating volume. However, the return of oxygen to ischaemic tissues causes the induction of a systemic inflammatory response, which contributes to cell death leading to organ failure. In trauma patients, failure of more than four organs is linked to certain mortality, highlighting the need for interventions that may reduce or prevent the deterioration in organ function. The aim of this thesis was to investigate the effect of therapeutic approaches on the organ injury and dysfunction induced by HS. Briefly, male Wistar rats were subjected to haemorrhage by withdrawal of blood to reduce the mean arterial pressure to 35 ± 5 mmHg for 90 min. Followed by resuscitation with 20 ml/kg Ringer’s lactate for 10 min and 50% of the shed blood for 50 min. Organ function was determined 4 h after the onset of resuscitation. This model was used to investigate the effect of three different interventions on the organ injury and dysfunction induced. In the first study, administration of bone marrow-derived mononuclear cells (BMMNCs) upon resuscitation resulted in (1) significant attenuation of the organ injury and dysfunction associated with HS, and (2) restoration of the activation of the Akt pro-survival pathway. It is possible that these beneficial effects are mediated by paracrine mediators secreted by BMMNCs, which modulate this pathway, however injection of large numbers of cells is not practical in the acute setting of trauma. Therefore, in the next study erythropoietin (EPO) was used as a daily pre-treatment for three days prior to the induction of haemorrhage, as EPO is a known stimulus of endothelial progenitor cell (EPC) mobilisation. EPO pre-treatment resulted in (1) significant attenuation of the organ injury and dysfunction associated with HS, (2) mobilisation of EPCs (CD34+/flk-1+), and (3) activation of the Akt pro-survival pathway with enhanced activation of eNOS. However, when used clinically EPO is associated with an increased risk of thrombotic events, therefore in the final study a non-erythropoietic analogue of EPO was investigated. Treatment with pyroglutamate helix B surface peptide (pHBSP) resulted in (1) significant attenuation of the organ injury and dysfunction associated with HS, and (2) activation of the Akt pro-survival pathway with enhanced activation of both eNOS and STAT3. Additionally, late pHBSP treatment, up to 60 min after the onset of resuscitation, exerted the highest degree of protection. The findings of this thesis support the view that modulation of the Akt pro-survival pathway is a potential therapeutic target in the treatment of the ischaemia-reperfusion injury associated with severe haemorrhage and resuscitation.
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Talving, Peep. "Aspects of hepatoduodenal trauma and fluid therapy in hemorrhagic shock /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-740-5/.

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Butler, Christopher. "Spectatorial Shock and Carnal Consumption: (Re)envisaging Historical Trauma in New French Extremity." Scholar Commons, 2013. http://scholarcommons.usf.edu/etd/4648.

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New French Extremity films are violent, transgressive, and break many social taboos in their narratives. However, this genre's directors are intelligent and construct these films with clues to France's past and how it still has implications in the present. This thesis was written to point out how New French Extremity films offer spectators the potential to reincorporate traumatic moments in French history by juxtaposing them against present day social, political, and economic ideologies. The purpose for this course of study was to investigate historical encounters that are present in New French Extremity filmmaking, something that has yet to be addressed by other scholars in any great detail. The general approach taken was to use Walter Benjamin's theory of allegory to secure connections between the past and present and illustrate how they could be interpreted by the film's spectators. The outcome of this research indicates how a spectator can potentially change his or her relationship with history and work towards reassessing his or her relationship with the present under certain social, political, or economic structures.
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Crosthwaite, Paul James. "Shock waves : temporality, history and trauma in the postmodernist response to World War II." Thesis, University of Newcastle Upon Tyne, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.435632.

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Harland, Katie. "The 'gentle shock of mild surprise' : Wordsworth's poetry as the missed experience of trauma theory." Thesis, University of Newcastle Upon Tyne, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.500947.

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Since the 1990s, trauma theory has occupied an important position in the field of literary criticism. This thesis looks at the writings of Geoffrey Hartman, Paul de Man and Cathy Caruth, three critics whose work has impacted on the field of trauma theory, as a means of exploring specifically how an early interest in Wordsworth s writings led them to an explicit or implicit interest in trauma and its effects. The final chapter proposes that Wordsworth's poetry has a unique capacity to convey the aspects of trauma that theoretical language cannot express.
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Alexander, Geoffrey C. "The Effect of Adding Drag-Reducing Polymers to Resuscitation Fluid During Hemorrhagic Shock on Skeletal Muscle Microcirculation." VCU Scholars Compass, 2006. http://scholarscompass.vcu.edu/etd/1522.

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Previous studies have shown an increase in survival when a minute amount of drag-reducing polymers were added to a resuscitation fluid. The purpose of this investigation was to examine the effect of adding a minute amount of the drag-reducing polymer polyethylene glycol to a resuscitation fluid, on the microcirculation of skeletal muscle during a volume-controlled hemorrhage model. The spinotrapezius muscle in twelve male Sprague Dawley rats was exteriorized for microvascular measurements of the arterioles. The diameters of the three levels of arterioles, interstitial fluid PO2, and RBC velocity in the feed arteriole were measured. Flow in the feed arteriole was calculated using the diameter and RBC velocity. Heart rate, mean arterial pressure, respiratory rate, arterial blood gases, arterial blood electrolytes, and arterial blood metabolites were measured. No significant physiological differences were observed between the DRP group and the Control group.
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Moore, Kathryn M. "Identification of Early Markers of Occult Tissue Hypoperfusion in Patients with Multiple Trauma Injuries." UKnowledge, 2016. http://uknowledge.uky.edu/nursing_etds/27.

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Injury is a global health problem and in the United States is the leading cause of death for persons aged 1 – 44 years. The primary causes of trauma related death are head injury and hemorrhage; hemorrhagic shock is difficult to recognize in the first hours after trauma. Identification of specific and optimal criteria upon which to base effective triage decisions for trauma patients has been an elusive goal for decades. The purpose of this dissertation was to identify measures available in the prehospital phase of care and in the Emergency Department that should be included for a more comprehensive definition of the trauma patient who will require trauma center care to better allocate trauma care and resources available. The first paper is a critical review of early physiologic markers of occult tissue hypoperfuson in which we examine markers of cardiovascular function and markers of tissue perfusion. In this review, we found surrogate measures of tissue perfusion include shock index as a measure of hemodynamic stability and acid-base indicators as measures of tissue oxygenation. This review guides the variable selection for the research study. The second paper is a report of a study conducted to examine shock index calculated from the first available prehospital vital signs and first available emergency department vital signs as a predictor of mortality within 48-hours in trauma compared to the Injury Severity Score. Shock index can be calculated in real-time during the course of treatment and provides continuous input into the ever changing condition of the patient. Injury severity score is calculated once, at the time of hospital discharge and is used primarily as a marker for comparison of injury severity in research and quality measures of trauma care. The study consisted of 516,156 trauma patient data reported to the National Trauma Data Bank (NTDB) in 2009. The results revealed SI as calculated in both the pre-hospital phase of care by Emergency Medical Services and in the Emergency Department to be significant independent predictors of mortality within forty-eight hours from trauma injuries. The third paper is a report of a study conducted to examine potential markers of occult tissue hypoperfusion within forty-eight hours of injury. The variables included four major variable categories, physiologic measures, anatomic measures, injury severity and presence of reported comorbid illness. The variable most predictive of death from trauma related injuries within forty-eight hours was the need for intubation. The findings from this dissertation provide further evidence of the value of multiple physiologic markers in early recognition of occult tissue hypoperfusion. Data from neither the review of the literature nor the two data-based studies are sufficient to identify a brief, accurate, easily used clinical instrument. Further work is needed to develop a clinically useful instrument to identify the occult tissue hypoperfusion in the trauma patient.
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DeBorde, Alisa M. "Seeing Trauma: The Known and the Hidden in Nineteenth-Century Literature." Scholar Commons, 2018. http://scholarcommons.usf.edu/etd/7141.

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Trauma as an official diagnosis first entered the DSM in 1980 and literary theorists began employing the term to discuss literature not too long after. Since the 1990s, theorists have largely focused on twentieth-century trauma literature with Holocaust and Modernist texts garnering much of the critical interest. Yet, Victorian life was also marked by trauma-causing events. From railway catastrophes, to industrial accidents, to premature deaths, and infectious diseases, Victorians reckoned with wounds to the mind through their lived experience. Trauma scholars who do work with nineteenth-century texts, with few exceptions, consider trauma in terms of its modern theories. While the work of Cathy Caruth, Shoshanna Felman, Ann Whitehead, E. Anne Kaplan, Dominick LaCapra, and Judith Herman has stimulated important discussions about trauma literature, their development of the concept of trauma rarely reaches further back than Freud. Victorian configurations of the mind and its response to psychical wounding have much to offer to the current discussion of literary trauma. This dissertation presents a study of Victorian literary texts through current theories of trauma juxtaposed with nineteenth-century formulations of the concept. The analysis offers three main points: one, to identify instances of trauma in nineteenth century texts that would otherwise go unnoticed; two, to situate texts within the cultural and historic milieu of their publication and to consider how literary conventions and forms indicative of the nineteenth-century serve to represent the effects and symptoms of trauma, and three, as a result of seeing trauma in the texts, to challenge common readings of Victorian literary characters, images, and forms.
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Cosser, Ramírez Carlos Arturo. "Características clínicas y epidemiológicas en los pacientes que ingresaron a la Unidad de Shok [i.e. Shock] Trauma del Hospital Nacional Dos de Mayo, 2001-2002." Bachelor's thesis, Universidad Nacional Mayor de San Marcos, 2005. https://hdl.handle.net/20.500.12672/1901.

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OBJETIVO: Describir las características clínicas y epidemiológicas en los pacientes que ingresaron a la Unidad de Shock Trauma del Hospital Nacional Dos de Mayo durante los años Enero 2001- Diciembre 2002. DISEÑO: Es un estudio retrospectivo descriptivo, tipo serie de casos, realizado mediante la revisión de historias clínicas, lo cual nos permitió analizar las características de los pacientes que ingresaron a esta Unidad durante este periodo. MATERIALES Y MÉTODOS: Se revisaron los libros de registro y las historias clínicas de los pacientes que ingresaron a esta Unidad durante el periodo ya establecido, los datos se llenaron en fichas prefabricadas. En ellos se registraron 1549 pacientes de los cuales, cumplieron con los criterios de inclusión 1424, 724 pacientes del 2001 y 700 pacientes del 2002. RESULTADOS: Del estudio descriptivo se obtuvo que el 67,7% de pacientes fueron varones, existiendo una relación hombres a mujeres de 2:1, y el 54,1% de pacientes tenían entre 18 y 60 años. El 82,5% de pacientes ingresaron al servicio de shock trauma por el servicio de Emergencia. Se realizaron 1694 procedimientos de los cuales el 36,1% fueron Intubaciones Endotraqueales. En este periodo se realizaron 1742 Exámenes Auxiliares de los cuales el 15% fueron TAC cerebrales. La causa más frecuente de ingreso a la Unidad de Shock Trauma es el Traumatismo (27,3%), siendo el Traumatismo Encéfalo Craneano el más importante, seguido por la Insuficiencia Respiratoria Aguda. La principal causa de mortalidad fue la Insuficiencia Respiratoria Aguda con un 21%. El 41,57% de pacientes permaneció en la Unidad por un periodo comprendido entre 1 y 6 horas. El 42,71% de pacientes queda en hospitalización. En el análisis estadístico se encontró relación estadísticamente significativa entre el diagnóstico de traumatismo con la edad (p = 0,004) y con el sexo masculino; relación entre la Insuficiencia Respiratoria Aguda y la edad (p = 0.045). CONCLUSIONES: La Unidad de Shock Trauma atendió en este periodo el 1,7% de la población que acudió a la Emergencia del Hospital, el traumatismo y la insuficiencia respiratoria fueron las causas más frecuentes.
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Labruto, Fausto. "Modifications of cardiovascular response to ischemia and trauma /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-379-5/.

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Books on the topic "Trauma shock"

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Faist, Eugen, John L. Ninnemann, and Douglas R. Green, eds. Immune Consequences of Trauma, Shock, and Sepsis. Berlin, Heidelberg: Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73468-7.

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Scalea, Thomas M., ed. The Shock Trauma Manual of Operative Techniques. New York, NY: Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2371-7.

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Scalea, Thomas M., ed. The Shock Trauma Manual of Operative Techniques. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-27596-9.

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Weil, Max Harry. Diagnosisand treatment of shock. 2nd ed. Baltimore, Md., USA: Williams & Wilkins, 1993.

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Faist, Eugen, Jonathan L. Meakins, and Friedrich W. Schildberg, eds. Host Defense Dysfunction in Trauma, Shock and Sepsis. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8.

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Fischer, Hubert. A colour atlas of trauma pathology. London: Wolfe, 1991.

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Goodall, Jane, and Christopher Lee. Trauma and public memory. Houndmills, Basingstoke, Hampshire: Palgrave Macmillan, 2015.

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1944-, LaPierre Aline, ed. Connection, our deepest desire and greatest fear: The neuroaffective relational model for healing developmental trauma. Berkeley, Calif: North Atlantic Books, 2012.

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1966-, Turner Lee, and Hodgetts T. J, eds. Trauma rules 2: Incorporating military trauma rules. 2nd ed. Malden, Mass: Blackwell Pub., 2006.

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Derek, Burke, and Porter Keith M, eds. Key topics in trauma. Oxford, England: BIOS Scientific Publishers, 1997.

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Book chapters on the topic "Trauma shock"

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Driscoll, Peter, Carl Gwinnutt, Terry Brown, and Olive Goodall. "Shock." In Trauma Resuscitation, 102–38. London: Macmillan Education UK, 1993. http://dx.doi.org/10.1007/978-1-349-12158-8_5.

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Sridhar, Srilata Kavita. "Shock." In Encyclopedia of Trauma Care, 1478–84. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-29613-0_262.

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Aydin, Ani, and Khanjan H. Nagarsheth. "Spinal Shock." In Encyclopedia of Trauma Care, 1516–17. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-29613-0_152.

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Bettega, Ana Luísa, Phillipe Abreu, Wagner Herbert Sobottka, Adonis Nasr, and Antonio Marttos. "Management of Shock." In The Trauma Golden Hour, 39–44. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-26443-7_8.

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Berry, Stepheny. "Shock Management in Trauma." In Encyclopedia of Trauma Care, 1484–88. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-29613-0_505.

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Schlag, G., H. Redl, S. Bahrami, J. Davies, P. Smuts, and I. Marzi. "Trauma and Cytokines." In Shock, Sepsis, and Organ Failure, 128–62. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77420-1_6.

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Novelli, G. P., and A. Di Filippo. "Haemorrhagic shock: pathophysiology and treatment." In Trauma Operative Procedures, 55–63. Milano: Springer Milan, 1999. http://dx.doi.org/10.1007/978-88-470-2151-8_6.

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Zhao, Yan, and Yuan-Guo Zhou. "Heat Shock Protein 90 in Severe Trauma." In Heat Shock Proteins, 533–45. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-23158-3_24.

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Li, Baiqiang, and Haichen Sun. "Acute Coagulopathy of Trauma-Shock." In Advanced Trauma and Surgery, 137–44. Singapore: Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-2425-2_9.

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Syring, Rebecca S. "Shock in the Trauma Patient." In Manual of Trauma Management in the Dog and Cat, 19–45. West Sussex, UK: John Wiley & Sons, Ltd., 2013. http://dx.doi.org/10.1002/9781118785645.ch3.

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Conference papers on the topic "Trauma shock"

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Endradita, Galih, Ahmad Yudianto, Ria Kumala, and Muhammad Afiful Jauhani. "Clinical Forensics in Electric Shock Trauma: A Case Study." In International Conference on Law, Economics and Health (ICLEH 2020). Paris, France: Atlantis Press, 2020. http://dx.doi.org/10.2991/aebmr.k.200513.022.

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Schenkman, Kenneth A., Wayne A. Ciesielski, Eileen M. Bulger, David J. Carlbom, Jeremy M. Shaver, Dana M. Fisk, Kellie L. Sheehan, et al. "Noninvasive Measurement Of Muscle Oxygenation Identifies Shock In Trauma Patients." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3775.

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Reilly, Matthew A., Daniel Sherwood, Richard Watson, Walt Gray, William E. Sponsel, Kimberly Thoe, Brian Lund, and Randolph Glickman. "Manifestations of Primary Blast-Induced Ocular Trauma." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14546.

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Prevalence of ocular trauma in battlefield injury has increased significantly in recent years. The majority of such injuries are due to improvised explosive devices. While impact-induced injuries are relatively well characterized, little is known regarding the effects of primary blast. We exposed porcine eyes to a simulated blast using a shock tube to correlate the blast overpressure with various trauma manifestations.
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Bolander, Richard, Cynthia Bir, and Pamela VandeVord. "Intracranial Pressure Measurement Within the Rat Skull is Sensitive to Shock Wave Intensity and Weight of the Specimen." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53590.

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Blast associated injuries have been quantified into different classes based on the type of trauma that they create [1]. Of these types of trauma, the neuropathology invoked by shock wave exposure is the most ambiguous [1]. The properties associated with shock wave exposure have lead to multiple hypothesized mechanisms for brain trauma including: acceleration-based damage, a thoracic squeeze resulting in pressure pulses to the brain, or transference of energy from the shock wave into the brain via the skull [2, 3].
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Laksari, Kaveh, Kurosh Darvish, and Keyanoush Sadeghipour. "Shock Wave as a Mechanism of Injury in Soft Tissues." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53823.

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The aim of this study is to investigate the propagation of shock waves and self-preserving waves in soft tissues such as aorta and brain as a mechanism of injury in high rate loading conditions as seen in blunt trauma and blast-induced trauma (BIT). It is shown that such phenomena can only be seen in nonlinear viscoelastic materials and the existing linear and quasi-linear models predict only decaying waves. Based on the results of this study, it is shown that when studying such high-rate loading conditions as in a blast, it is critical to consider the discontinuities predicted in strain and stress in certain realistic initial conditions to accurately determine the extent of soft tissue injuries.
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Rahbar, Elaheh, Bryan A. Cotton, John B. Holcomb, and Charles E. Wade. "Reduced Plasma Oncotic Pressure is Indicative of Injury Severity in Trauma Patients." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14539.

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Trauma is the leading cause of death in people aged 1–44 years old, with hemorrhagic shock accounting for nearly half of these deaths. These fatalities often occur early, within 24 hours, but many are deemed potentially preventable with appropriate resuscitation. 1–2 The term “appropriate resuscitation” is somewhat ambiguous and subjective because of the lack of a standardized resuscitation protocol. We attribute this deficiency to our rudimentary understanding of the mechanisms of hemorrhagic shock and resuscitation.
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Luo, Haibiao, Robin O. Cleveland, and James C. Williams. "Shock Wave Lithotripters With Broad Focus Result in Greater Stress in Human Kidney Stones: Numerical Simulation." In ASME 2008 3rd Frontiers in Biomedical Devices Conference. ASMEDC, 2008. http://dx.doi.org/10.1115/biomed2008-38093.

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Shock wave lithotripsy (SWL) has been used to treat kidney stones for decades. However, there is growing recognition that shock waves induces trauma to kidney tissue [1, 2]. The poor understanding of stone comminution mechanisms means that the design of new lithotripters is principally a practice of empiricism [3]. A mechanistic understanding of stone comminution would provide a criterion to develop new lithotripsy systems. In this work, a three-dimensional finite-difference time-domain (FDTD) solution to the linear elastic equations was employed [4] to investigate the stress and displacement fields of kidney stones subject to lithotripsy shock waves. The kidney stone models were obtained from micro-computed tomography images (resolution of 20 μm) and have diameters from 2 mm to 5 mm.
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Levinson, Andrew T., Andrew H. Stephen, Michael D. Connolly, Kenneth A. Lynch, Amy C. Palmisciano, Richard N. Read, Daithi S. Heffernan, and Mitchell M. Levy. "Microcirculatory Abnormalities As A Predictor Of Outcomes In Critically Ill Patients With Trauma Compared To Patients With Septic Shock." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a4583.

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Grimm, Matthew V., Karim H. Muci-Küchler, Brandon J. Hinz, and Shawn M. Walsh. "Comparison of Numerical and Experimental Results of Small Scale Compressed Gas Blast Experiments Involving a Surrogate Head Form." In ASME 2012 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/imece2012-87663.

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Exposure to a shock wave from an explosive blast can result in injury to the human body even if external signs of trauma are not present. Gaining a better understanding of the mechanisms contributing to those injuries can result in the design of better personal protective equipment (PPE). Compressed gas blast experiments can be conveniently used to explore the mechanical response of PPE systems and instrumented surrogate head forms to blast loading scenarios in a laboratory environment. Likewise, numerical simulations can be used to study relevant field variables related to the compressed gas blast and its effects on the target. In this regard, experimental data is needed to validate simulation results. This paper presents an experiment that uses a small scale compressed gas blast generator to explore the pressure distribution around a surrogate head form due to blast loading. The compressed gas blast generator is an open-end shock tube which creates a shock wave when the diaphragm that separates the high pressure and low pressure (ambient air) regions ruptures. The overpressures on selected locations of the surrogate head form were measured with piezoelectric pressure sensors and the data was processed to obtain positive phase durations and positive phase impulses. The surrogate head form was positioned off-axis from the exit of the compressed gas blast generator to preclude the discharge flow from affecting the overpressure measurements. A three-dimensional Coupled Eulerian-Lagrangian (CEL) model of the experiment described above was prepared in Abaqus/Explicit. Selected numerical and experimental results were compared and there was good agreement between them.
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10

Gao, Jian, Sean Connell, Riyi Shi, and Jun Chen. "Blast-Induced Neurotrauma: Characterizing the Blast Wave Impact and Tissue Deformation." In ASME 2010 3rd Joint US-European Fluids Engineering Summer Meeting collocated with 8th International Conference on Nanochannels, Microchannels, and Minichannels. ASMEDC, 2010. http://dx.doi.org/10.1115/fedsm-icnmm2010-30747.

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Primary blast injury, caused by exposure to the primary pressure wave emitted from explosive ordnance, is a common trauma associated with modern warfare activities. The central nervous system is particularly vulnerable to primary blast injury, which is responsible for many of the war related casualties and mortalities. An ex vivo model system is developed to introduce a blast wave, generated from a shock tube, directly to spinal cord tissue sample. A high-speed shadowgraphy is utilized to visualize the development of the blast wave and its interaction with the tissue samples. The surface deformation of the tissues is also measured for the analysis of internal stress and possible damage occurred in the tissue sample. Understanding the temporal development of the blast-tissue interaction provides valuable input for characterizing and modeling blast-induced neurotrauma. Particularly, tracking the sample surface deformation over time provides realistic boundary conditions for numerically simulating the injury and understanding the temporal development of stress.
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Reports on the topic "Trauma shock"

1

Tweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality,Apoptosis, Inflammation and Mitochondrial Dysfunction. Fort Belvoir, VA: Defense Technical Information Center, December 2013. http://dx.doi.org/10.21236/ada612817.

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2

Tweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality, Apoptosis, Inflammation and Mitochondrial Dysfunction. Fort Belvoir, VA: Defense Technical Information Center, December 2012. http://dx.doi.org/10.21236/ada612818.

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3

Tweardy, David J. Prevention of Trauma/Hemorrhagic Shock-Induced Mortality, Apoptosis, Inflammation and Mitochondrial Dysfunction Using IL-6 as a Resuscitation Adjuvant. Fort Belvoir, VA: Defense Technical Information Center, December 2011. http://dx.doi.org/10.21236/ada612819.

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