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Dissertations / Theses on the topic 'Traumatic brain injury'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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1

Force, Lisa Marie. "Traumatic brain injury and acidosis /." view abstract or download text of file, 2006. http://hdl.handle.net/1794/3913.

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2

Singh, Rajiv K. "Depression after traumatic brain injury." Thesis, University of Sheffield, 2017. http://etheses.whiterose.ac.uk/18730/.

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Background Depression is known to be common after traumatic brain injury (TBI) and associated with worse functional and psychosocial outcomes. However, there remains considerable uncertainty over the exact prevalence of the condition. Aims The aim of this study was to accurately assess the prevalence of post TBI depression and its changes over a period of one year. The associated demographic and injury features were also examined for possible association with risk of depression in the hope that those with higher susceptibility to depression may be identified. Methods The study population was a

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3

Perel, Pablo Andraes. "Prognosis in traumatic brain injury." Thesis, London School of Hygiene and Tropical Medicine (University of London), 2009. http://researchonline.lshtm.ac.uk/1635515/.

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Introduction: The general purpose of this thesis was to study prognosis in traumatic brain injury (TBI) patients, with the aim of providing useful and practical information in clinical practice and clinical research. The specific objectives were: to develop and validate practical prognostic models for TBI patients and to assess the validity of the Modified Oxford Handicap Scale (mOHS) for predicting disability at six months. Methods: A survey was first conducted to understand the importance of prognostic information among physicians. A systematic review of prognostic models for TBI patients wa

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4

Rowe, Rachel K. "POST-TRAUMATIC SLEEP FOLLOWING DIFFUSE TRAUMATIC BRAIN INJURY." UKnowledge, 2013. http://uknowledge.uky.edu/neurobio_etds/7.

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Traumatic brain injury (TBI) is a major cause of death and disability throughout the world with few pharmacological treatments available for individuals who suffer from neurological morbidities associated with TBI. Cellular and molecular pathological processes initiated at the time of injury develop into neurological impairments, with chronic sleep disorders (insomnia, hypersomnolence) being among the somatic, cognitive and emotional neurological impairments. Immediately post-injury, TBI patients report excessive daytime sleepiness, however, discordant opinions suggest that individuals should

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5

Keller, Kristen Jo. "Challenges to Secondary Brain Injury Prevention in Severe Traumatic Brain Injury." Diss., The University of Arizona, 2014. http://hdl.handle.net/10150/338712.

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BACKGROUND/AIMS: Inconsistency in the use of secondary brain injury prevention guidelines among US trauma centers after severe traumatic brain injury is prevalent in many literature sources. However, this phenomenon has not been thoroughly studied. The purpose of this DNP project is to identify the key barriers and challenges in compliance to the evidence-based guidelines for secondary brain injury prevention. DESIGN: An exploratory, emergent design was used to collect descriptive qualitative data through the use of a survey. SETTING: Six Phoenix Metropolitan Level 1 trauma centers. PARTICIPA

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6

Al-Hasani, Omer Hussain. "Traumatic brain injury with particular reference to diffuse traumatic axonal injury subpopulations." Thesis, University of Edinburgh, 2011. http://hdl.handle.net/1842/5569.

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Traumatic brain injury (TBI) remains an important cause of morbidity and mortality within society. TBI may result in both focal and diffuse brain injury. Diffuse traumatic axonal injury (TAI) is an important pathological substrate of TBI, and can be associated with a range of clinical states, ranging from concussion through to death, the clinical severity being associated with a number of factors related to the injury. A retrospective study was conducted using 406 cases with TBI, from the archive of the Academic Department of Pathology (Neuropathology) University of Edinburgh, during the perio

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7

Malhotra, Rajiv. "GENE EXPRESSION FOLLOWING TRAUMATIC BRAIN INJURY." VCU Scholars Compass, 1998. http://scholarscompass.vcu.edu/etd/5082.

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The pathology which results from traumatic brain injury (TBI) have long been believed to be immediate and irreversible. However, recently it has been shown that, although the primary effects are virtually unavoidable, the secondary effects manifest themselves through biochemical processes set in motion at the time of the injury. These events are frequently mediated through the process of excitotoxicity, which results from a widespread release of excitatory neurotransmitters. These neurotransmitters go on to activate both ionotropic and metabotropic receptors. The signal transduction initiated

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8

Davies, Suzanne. "Personality change following traumatic brain injury." Thesis, University of Birmingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.397519.

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This thesis is submitted in partial fulfilment of the requirements for the degree of Clip. Psy, D. _ at the School of Psychology, University of Birmingham. It represents both the clinical work and research conducted over the course of the clinical training. Volume I contains the research components which are concerned with examining factors affecting adjustment following traumatic brain injury (TBI). The literature review explores-3 0 papers-written since 1998t hat examine the factors-purported to - affect the development of depression after a TBI. The empirical paper examines personality chan

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9

Macqueen, Ruth. "Masculine identity after traumatic brain injury." Thesis, University of East Anglia, 2016. https://ueaeprints.uea.ac.uk/60949/.

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Background: Men are twice as likely to experience a Traumatic Brain Injury (TBI) as women suggesting that aspects of masculine identity play an important role in how people acquire their brain injury. Research also suggests that masculine identity influences how people manage their health experiences. Masculine identity may therefore be an important consideration for neuropsychological therapy and rehabilitation more generally particularly because part of the process of rehabilitation concerns helping individuals with their sense of self. This research aimed to explore men’s experiences of mas

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10

Helmy, Adel Ezzat. "Neuro-inflammation in traumatic brain injury." Thesis, University of Cambridge, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.610114.

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11

Nagulavancha, Sruthi. "Traumatic brain injury options web application." Kansas State University, 2010. http://hdl.handle.net/2097/4626.

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Master of Science<br>Department of Computing and Information Sciences<br>Daniel A. Andresen<br>According to the Division of Injury Response, Centers for Disease Control and Prevention, approximately 1.4 million Americans sustain a traumatic brain injury each year. The aim of the project is to create a web interface to link survivors, family members, and caregivers of individuals suffering from traumatic brain injuries (TBI) to potentially helpful agencies or service centers within their local communities. Often the TBI service centers located in the remote places are difficult to trace hence t

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12

LeMay, Carrie C., and Jill D. Stinson. "Sex Offenders With Traumatic Brain Injury." Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/etsu-works/7906.

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13

Osterstock, Guillaume. "Hypothalamic defaults after traumatic brain injury." Thesis, Montpellier 1, 2012. http://www.theses.fr/2012MON1T017/document.

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Les travaux de cette thèse ont porté sur le contrôle des neurones à GHRH dans des conditions physiologiques et pathologiques. Le but étant de caractériser les mécanismes cellulaires et moléculaires impliqués dans le fonctionnement ou dérégulations du réseau de neurones à GHRH. Ces neurones sont les principaux stimulateurs de la libération de l’hormone de croissance (GH). Nous avons d’abord montré que l’axe de la croissance et de l’appétit peuvent être régulés indépendamment au niveau de l’hypothamus. En effet, la ghréline, seule hormone produite par le tractus gastro-intestinal et connue pour

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14

Moazzez, Lesko Mehdi. "Prognosis in traumatic brain injury (TBI)." Thesis, University of Manchester, 2011. https://www.research.manchester.ac.uk/portal/en/theses/prognosis-in-traumatic-brain-injury-tbi(8b69e340-7ecd-4890-9746-863089bf55f5).html.

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Introduction: Prognosis in Traumatic Brain Injury (TBI) can be made using prognostic models (the IMPACT and CRASH models) or brain injury biomarkers (S100B). Current prognostic models are derived from historic datasets recruited from heterogeneous countries in terms of trauma care and for the purpose of clinical trials. Objective: To construct a prognostic model suitable for British trauma care, to compare the prognostic performance of prognostic models with S100B and to assess the combination of prognosticators from the constructed models with S100B. Methods: A dataset of 802 TBI cases from t

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15

Arietta, Luca. "Clinical Data Mining: Traumatic Brain Injury." Master's thesis, Alma Mater Studiorum - Università di Bologna, 2012. http://amslaurea.unibo.it/3897/.

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Il trauma cranico é tra le piú importanti patologie traumatiche. Ogni anno 250 pazienti ogni 100.000 abitanti vengono ricoverati in Italia per un trauma cranico. La mortalitá é di circa 17 casi per 100.000 abitanti per anno. L’Italia si trova in piena “media” Europea considerando l’incidenza media in Europa di 232 casi per 100.000 abitanti ed una mortalitá di 15 casi per 100.000 abitanti. Degli studi hanno indicato come una terapia anticoagulante é uno dei principali fattori di rischio di evolutiviá di una lesione emorragica. Al contrario della terapia anticoagulante, il rischio emorra

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16

Akin, Faith W. "Vestibular Evaluation of Traumatic Brain Injury." Digital Commons @ East Tennessee State University, 2009. https://dc.etsu.edu/etsu-works/2448.

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17

Yin, Terry. "Neuroprotective strategies for traumatic brain injury." Diss., University of Iowa, 2015. https://ir.uiowa.edu/etd/1811.

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Traumatic brain injury (TBI) causes life-debilitating conditions. While patient survival after a TBI has improved, the outlook for quality of life after TBI currently remains poor. In order to address this problem, there is a significant unmet need for new therapeutic options to prevent progression of deficits associated with TBI. To this end, we investigated two strategies to combat the deleterious affect of TBI. First, we targeted cerebral acidosis associated with TBI by testing whether disruption of acid sensing ion channel 1a (ASIC1a) in CNS, or buffering acidosis with sodium bicarbonate,

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18

Savicki, Laura Elizabeth. "Collaborative referencing in traumatic brain injury." Thesis, University of Iowa, 2012. https://ir.uiowa.edu/etd/2977.

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Traumatic brain injury (TBI) is a global health epidemic that has deleterious consequences for the individuals with the brain injury, their families, and society. The development and validation of effective treatments is imperative. The current study was inspired by Ylvisaker's collaborative intervention approach with individuals with TBI and draws on a line of work by Duff and colleagues (e.g., Duff et al., 2006; Gupta et al., 2011) documenting patterns of spared and impaired learning abilities in individuals with various

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Akin, Faith W., and Owen D. Murnane. "Vestibular Consequences of Mild Traumatic Brain Injury (Blast Injury)." Digital Commons @ East Tennessee State University, 2008. https://dc.etsu.edu/etsu-works/1940.

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20

Lalani, Sanam Jivani. "Effects of Traumatic Brain Injury on Pediatric Brain Volume." BYU ScholarsArchive, 2018. https://scholarsarchive.byu.edu/etd/6924.

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This study investigated the effects of lesion presence within larger brain networks (e.g., default mode network (DMN), salience network (SN), and mentalizing network (MN)) in the chronic phase of a pediatric traumatic brain injury (TBI) and the effect on social function. We compared children with a TBI to children with an orthopedic injury (OI) with three different aims. The first aim was to determine whether network volume differed by group (e.g., TBI vs. OI). Second, investigate if lesion presence in a sub component region of the network resulted in total network volume loss for that network

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21

LaRoux, Charlene I. 1979. "Executive function deficits in traumatic brain injury." Thesis, University of Oregon, 2010. http://hdl.handle.net/1794/11063.

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xii, 98 p. : ill. (some col.)<br>The short and long term pathophysiology of traumatic brain injury (TBI) has not been fully elucidated. Individuals recently suffering a mild TBI (mTBI) or having a history of TBI frequently suffer deficits in their ability to maintain and allocate attention within and between tasks. This dissertation examines the influence of mild and chronic TBI on performance of task switching. We employed spatial and numerical task switching paradigms to assess the behavioral deficits in mTBI, and we used an internally generated switching and an externally cued switching tas

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22

Hånell, Anders. "Plasticity and Inflammation following Traumatic Brain Injury." Doctoral thesis, Uppsala universitet, Neurokirurgi, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-146551.

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Traumatic Brain Injury (TBI) mainly affects young persons in traffic accidents and the elderly in fall accidents. Improvements in the clinical management have significantly improved the outcome following TBI but survivors still suffer from depression, memory problems, personality changes, epilepsy and fatigue. The initial injury starts a series of events that give rise to a secondary injury process and despite several clinical trials there is no drug available for clinical use that targets secondary brain injury mechanisms. Some recovery of function is seen during the first months following in

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23

Van, der Merwe Jó-Marié. "Family needs following adult traumatic brain injury." Thesis, University of Port Elizabeth, 2004. http://hdl.handle.net/10948/335.

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Traumatic brain injury (TBI) represents a significant and growing type of disability in South Africa. Coping with the impact of traumatic brain injury is one of the most difficult tasks that can confront a family, and family members experience a wide range of needs as the injured person progresses through rehabilitation. In South Africa, research on family needs following traumatic brain injury has thus far been neglected and rehabilitation resources are sadly lacking. For this reason it is necessary to accumulate knowledge about these families’ needs so as to assist with the planning of futur

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24

Crawford, Maria Anne, and n/a. "Speed of retrieval after traumatic brain injury." University of Otago. Department of Psychology, 2005. http://adt.otago.ac.nz./public/adt-NZDU20060830.115029.

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Although it is well established that persons with traumatic brain injury (TBI) experience word retrieval difficulties, the underlying cause of these deficits is not known. Difficulties with word retrieval have negative social implications as they can impact on the ability to converse with others. The overarching goal of this dissertation was to determine the underlying cause of problems with word retrieval after TBI. To test word retrieval in this dissertation, participants were given a series of word fluency tasks and the speed of word generation was measured. In addition to measuring inter

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25

Kruijk, Jelle de. "Mild traumatic brain injury intervention and prognosis /." [Maastricht] : Maastricht : UPM, Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 2001. http://arno.unimaas.nl/show.cgi?fid=7622.

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26

Israelsson, Charlotte. "Molecular Characterization of Experimental Traumatic Brain Injury." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7087.

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27

Johnson, Victoria E. "Progressive neuopathological changes following traumatic brain injury." Thesis, University of Glasgow, 2012. http://theses.gla.ac.uk/3162/.

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Whilst the acute effects of traumatic brain injury (TBI) may be devastating, growing evidence suggests TBI may initiate long-term neurodegenerative processes. Indeed, an epidemiological association between TBI and Alzheimer’s disease (AD) has been demonstrated. Here the link between TBI and chronic neurodegeneration is explored by examining associated pathologies in material from patients surviving TBI for varying intervals. Furthermore, the Aβ clearing enzyme, neprilysin, has emerged as important with regards to Aβ dynamics in AD and thus may be relevant to Aβ-plaque formation in TBI. Its inf

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28

Craddock, Holly L. "Nursing interventions utilized in traumatic brain injury." Honors in the Major Thesis, University of Central Florida, 1998. http://digital.library.ucf.edu/cdm/ref/collection/ETH/id/27.

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This item is only available in print in the UCF Libraries. If this is your Honors Thesis, you can help us make it available online for use by researchers around the world by following the instructions on the distribution consent form at http://library.ucf.edu/Systems/DigitalInitiatives/DigitalCollections/InternetDistributionConsentAgreementForm.pdf You may also contact the project coordinator, Kerri Bottorff, at kerri.bottorff@ucf.edu for more information.<br>Bachelors<br>Health and Public Affairs<br>Nursing

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29

Bradshaw, Douglas Robert Saunders. "Linear wave propagation in traumatic brain injury." Thesis, University of Southampton, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341646.

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30

Williams, Claire. "Emotion deficit disorders following traumatic brain injury." Thesis, Swansea University, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.678429.

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31

Herbert, Camilla. "Grief and loss following traumatic brain injury." Thesis, University of Sheffield, 1998. http://etheses.whiterose.ac.uk/10277/.

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Literature Review: The literature review summarises the main theoretical models of grief and illustrates how these have developed from intra- to inter-personal models and from clinical to psychosocial models of bereavement. The paper explores the concepts of pathological, anticipatory, and disenfranchised grief, and attempts to identify aspects that are relevant to grief in contexts other than bereavement. Finally an attempt is made to integrate themes from the literature that are applicable to the experience of loss amongst relatives of traumatically brain-injured patients. Some useful concep

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32

Khadr, Sophie N. "Neuroendocrine consequences of childhood traumatic brain injury." Thesis, University of Edinburgh, 2010. http://hdl.handle.net/1842/29194.

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Objectives: 1) To determine the prevalence, aetiology and clinical significance of pituitary dysfunction after moderate or severe childhood traumatic brain injury (TBI); and 2) to examine its impact on quality of life (QoL) and body composition. Subjects: Retrospective observational study of 33 survivors of accidental TBI (27 males) and two of inflicted TBI (both males). Accidental TBI group: mean (SD) age at study was 13.4y (3.7y) and interval since injury, 4.1y (1.6y). King's Outcome Scale for Childhood Head Injury (KOSCHI) rating: 15 good recovery, 16 moderate disability, 2 severe disabilit

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33

Trenchard, Sian Olivia. "Traumatic brain injury in a paediatric population." Thesis, University of Manchester, 2013. https://www.research.manchester.ac.uk/portal/en/theses/traumatic-brain-injury-in-a-paediatric-population(cf299afa-75ff-4a58-9684-6b332d25715e).html.

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This thesis examined neuropsychological and psychological outcomes following paediatric traumatic brain injury (TBI). The introductory chapter provides an overview of the paediatric TBI literature, giving definitions of key terms and concepts and providing a description of the epidemiology of childhood head injury. Key models relevant to paediatric TBI are introduced, including developmental neurological, cognitive and psychological perspectives. This is followed by a discussion of factors pertinent to outcome after TBI, followed by a description of outcomes relating to cognitive, behavioural,

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34

Donnelly, Joseph. "Intracranial monitoring after severe traumatic brain injury." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/271422.

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Intracranial monitoring after severe traumatic brain injury offers the possibility for early detection and amelioration of physiological insults. In this thesis, I explore cerebral insults due raised intracranial pressure, decreased cerebral perfusion pressure and impaired cerebral pressure reactivity after traumatic brain injury. In chapter 2, the importance of intracranial pressure, cerebral perfusion pressure and pressure reactivity in regulating the cerebral circulation is elucidated along with a summary of the existing evidence supporting intracranial monitoring in traumatic brain injury.

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35

Walk, Alexandra Elizabeth. "Traumatic Brain Injury: Teacher Knowledge and Skills." University of Dayton / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=dayton1312572728.

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36

Kinkela, Jessica H. "Diagnosis Threat in Mild Traumatic Brain Injury." Ohio University / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1223597555.

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37

Szabó, Borbála L. "Families of adolescents with traumatic brain injury /." The Ohio State University, 2000. http://rave.ohiolink.edu/etdc/view?acc_num=osu1488196234911166.

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38

Wagner, Michael R. "The Nitroxidative Response to Traumatic Brain Injury." Ohio University / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1584179902783766.

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39

Thomasz, Tennille Amanda. "Investigations of Friendship Following Traumatic Brain Injury." Thesis, The University of Sydney, 2016. http://hdl.handle.net/2123/14916.

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This research is comprised of two studies, both conducted with the overarching aim of investigating friendships following traumatic brain injury (TBI). The first investigation was a survey study. Sixty-eight speech pathologists who work with clients who have sustained a TBI were surveyed. The survey aimed to answer specific queries surrounding the current perspective of speech pathologists when working with friends, the reason that speech pathologists work with friends, barriers to working with friends and why speech pathologists perceive that some friendships succeed post TBI. Responses were

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40

Akin, Faith W. "Symposium: Vestibular Related Traumatic Brain Injury (TBI)." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etsu-works/2428.

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41

Akin, Faith W., and Owen D. Murnane. "Vestibular Consequences of Mild Traumatic Brain Injury." Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/etsu-works/1928.

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42

MacMillan, Pamela Jo. "Vulnerability to disability following traumatic brain injury." W&M ScholarWorks, 1999. https://scholarworks.wm.edu/etd/1550154124.

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43

PISCHIUTTA, FRANCESCA. "Mesenchymal stromal cells for traumatic brain injury." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2014. http://hdl.handle.net/10281/52353.

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The multiple pathological cascades activated after traumatic brain injury (TBI) and their extended nature offer the possibility for therapeutic interventions possibly affecting multiple injury mechanisms simultaneously. Mesenchymal stromal cell (MSC) therapy matches this need, being a bioreactor of a variety of molecules able to interact and modify the injured brain microenvironment. Compared to autologous MSCs, bank stored GMP-graded allogenic MSCs appear to be a realistic choice for TBI in a translational perspective, due to the need of delivering cell therapy in the acute phase of the path

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44

Sauerbeck, Andrew David. "TRICHLOROETHYLENE EXPOSURE AND TRAUMATIC BRAIN INJURY INTERACT AND PRODUCE DUAL INJURY BASED PATHOLOGY AND PIOGLITAZONE CAN ATTENUATE DEFICITS FOLLOWING TRAUMATIC BRAIN INJURY." UKnowledge, 2011. http://uknowledge.uky.edu/gradschool_diss/133.

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The development of Parkinson's disease (PD) in humans has been linked to genetic and environmental factors for many years. However, finding common single insults which can produce pathology in humans has proved difficult. Exposure to trichloroethylene (TCE) or traumatic brain injury (TBI) has been shown to be linked to PD and it has also been proposed that multiple insults may be needed for disease development. The present studies show that exposure to TCE prior to a TBI can result in pathology similar to early PD and that the interaction of both insults is required for impairment in behaviora

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45

Carter-Allison, Samantha Natalie. "Diagnosis threat and injury beliefs after mid traumatic brain injury." Thesis, King's College London (University of London), 2015. https://kclpure.kcl.ac.uk/portal/en/theses/diagnosis-threat-and-injury-beliefs-after-mid-traumatic-brain-injury(c6ba3d52-13d9-46ea-aeee-d34ed2e43943).html.

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Background: Diagnosis threat is a form of stereotype threat, where individuals with a history of mild traumatic brain injury (mTBI) have shown performance decrements on cognitive tasks, owing to negative expectancies around cognitive ability elicited by cues in the environment. This study systematically reviews experimental studies to gauge the presence/absence of an effect of diagnosis threat on neuropsychological task performance in mTBI. It also investigates whether methodological variation and methodological quality contribute to variation in study findings. Method: A systematic search of

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46

Rossouw, Joanne Courtney. "Incidence of traumatic brain injury, prevalence of dysphagia, and factors predicting health outcomes following traumatic brain injury in adults." Master's thesis, University of Cape Town, 2015. http://hdl.handle.net/11427/16646.

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Includes bibliographical references<br>South Africa has a high incidence of injury-related disorders, such as traumatic brain injury (TBI) as a result of motor vehicle accidents and assault. Dysphagia is a common sequela of TBI, which may result in malnutrition or aspiration pneumonia. There is limited epidemiological data available for TBI and dysphagia in South Africa which is important for health care planning. There is also inadequate literature reporting predictive factors for dysphagia and health outcomes of patients with TBI and swallowing disorders for the South African context, which

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47

Sacho, Raphael Hillel. "Brain temperature, inflammation and outcome after severe traumatic brain injury." Thesis, University of Manchester, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.503675.

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48

Powell, Janet M. "Effectiveness of comprehensive inpatient rehabilitation following traumatic brain injury /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/10320.

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49

Reid, Louise Marie. "Traumatic brain injury, post-traumatic stress disorder symptom reporting and attentional bias : unravelling the misidentification of post-traumatic stress disorder in people with a traumatic brain injury." Thesis, University of Glasgow, 2009. http://theses.gla.ac.uk/1221/.

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Background: Post-traumatic stress disorder (PTSD) can occur following a traumatic event that has led to moderate to severe traumatic brain injury (TBI) even when there is little or no memory for the event. The incidence of PTSD is higher when diagnosed by self-report questionnaires compared to structured clinical interview. Previous studies suggest PTSD can be misdiagnosed in a significant proportion of cases and the incidence is in fact low. To explore this issue further there is a need to not only understand whether there are differences between cases that do and do not fulfill symptom crite

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50

Bellander, Bo-Michael. "On the role of complement activation following traumatic brain injury /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-929-3/.

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