Relevant bibliographies by topics / Trinucleotides

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Academic literature on the topic 'Trinucleotides'

Author: Grafiati
Published: 3 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Trinucleotides"

1

Michel, Christian J., and Giuseppe Pirillo. "Strong Trinucleotide Circular Codes." International Journal of Combinatorics 2011 (August 25, 2011): 1–14. http://dx.doi.org/10.1155/2011/659567.

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Recently, we identified a hierarchy relation between trinucleotide comma-free codes and trinucleotide circular codes (see our previous works). Here, we extend our hierarchy with two new classes of codes, called DLD and LDL codes, which are stronger than the comma-free codes. We also prove that no circular code with 20 trinucleotides is a DLD code and that a circular code with 20 trinucleotides is comma-free if and only if it is a LDL code. Finally, we point out the possible role of the symmetric group ∑4 in the mathematical study of trinucleotide circular codes.
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Suchsland, Ruth, Bettina Appel, Matthäus Janczyk, and Sabine Müller. "Solid Phase Assembly of Fully Protected Trinucleotide Building Blocks for Codon-Based Gene Synthesis." Applied Sciences 9, no. 11 (2019): 2199. http://dx.doi.org/10.3390/app9112199.

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The use of pre-formed trinucleotides, representing codons of the 20 canonical amino acids, for oligonucleotide-directed mutagenesis offers the advantage of controlled randomization and generation of “smart libraries”. We here present a method for the preparation of fully protected trinucleotides on solid phase. The key issue of our strategy is the linkage of the starting nucleoside to the solid support via a traceless disulfide linker. Upon trinucleotide assembly, the disulfide bridge is cleaved under reducing conditions, and the fully protected trinucleotide is released with a terminal 3′-OH
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Benard, Emmanuel, and Christian J. Michel. "Transition and Transversion on the Common Trinucleotide Circular Code." Computational Biology Journal 2013 (May 29, 2013): 1–10. http://dx.doi.org/10.1155/2013/795418.

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In 1996, a trinucleotide circular code which is maximum, self-complementary, and , called , was identified statistically on a large gene population of eukaryotes and prokaryotes (Arquès and Michel (1996)). Transition and transversions I and II are classical molecular evolution processes. A comprehensive computer analysis of these three evolution processes in the code shows some new results; in particular (i) transversion I on the 2nd position of any subset of trinucleotides of generates trinucleotide circular codes which are always and (ii) transversion II on the three positions of any subset
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Suchsland, Ruth, Bettina Appel, Pasi Virta, and Sabine Müller. "Synthesis of fully protected trinucleotide building blocks on a disulphide-linked soluble support." RSC Advances 11, no. 7 (2021): 3892–96. http://dx.doi.org/10.1039/d0ra10941j.

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Fully protected trinucleotides are synthesized on a tetrapodal soluble support using a disulphide linkage that upon reductive cleavage allows release of the trinucleotide with free 3′-OH group for further conversion to a phosphoramidite.
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McClellan, James, and Sarah Newbury. "Inexpressible trinucleotides." Nature 379, no. 6564 (1996): 396. http://dx.doi.org/10.1038/379396a0.

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Piwowar, Monika, Jan Meus, Piotr Piwowar, Zdzisław Wiśniowski, Justyna Stefaniak, and Irena Roterman. "Tandemly repeated trinucleotides - comparative analysis." Acta Biochimica Polonica 53, no. 2 (2006): 279–87. http://dx.doi.org/10.18388/abp.2006_3340.

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Characteristics of 64 possible tandem trinucleotide repeats (TSSR) from Homo sapiens (hs), Mus musculus (mm) and Rattus norvegicus (rn) genomes are presented. Comparative analysis of TSSR frequency depending on their repetitiveness and similarity of the TSSR length distributions is shown. Comparative analysis of TSSR sequence motifs and association between type of motif and its length (n) using rho-coefficient method (quantitatively measuring the association between variables in contingency tables) is presented. These analyses were carried out in the context of neurodegenerative diseases based
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Polyansky, Anton A., Mathias Kreuter, John D. Sutherland, and Bojan Zagrovic. "Direct interplay between stereochemistry and conformational preferences in aminoacylated oligoribonucleotides." Nucleic Acids Research 47, no. 21 (2019): 11077–89. http://dx.doi.org/10.1093/nar/gkz902.

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Abstract To address the structural and dynamical consequences of amino-acid attachment at 2′- or 3′-hydroxyls of the terminal ribose in oligoribonucleotides, we have performed an extensive set of molecular dynamics simulations of model aminoacylated RNA trinucleotides. Our simulations suggest that 3′-modified trinucleotides exhibit higher solvent exposure of the aminoacylester bond and may be more susceptible to hydrolysis than their 2′ counterparts. Moreover, we observe an invariant adoption of well-defined collapsed and extended conformations for both stereoisomers. We show that the average
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Houlston, C. E., M. Cummings, H. Lindsay, S. Pradhan, and R. L. P. Adams. "DNA substrate specificity of pea DNA methylase." Biochemical Journal 293, no. 3 (1993): 617–24. http://dx.doi.org/10.1042/bj2930617.

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DNA methylase, present in low-salt extracts of nuclei prepared from Pisum sativum shoot tips, methylates model DNA substrates containing CNG trinucleotides or CI dinucleotides only. The binding to the hemimethylated trinucleotide substrates is very much stronger and more persistent than the binding to the unmethylated substrates or to the hemimethylated dinucleotide substrate. When the DNA concentration is limiting, the rate of methyl-group transfer with the hemimethylated CNG substrate is much greater than that with the unmethylated CNG. However, the Vmax. is similar for the two CNG substrate
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Fimmel, Elena, Christian J. Michel, and Lutz Strüngmann. "n -Nucleotide circular codes in graph theory." Philosophical Transactions of the Royal Society A: Mathematical, Physical and Engineering Sciences 374, no. 2063 (2016): 20150058. http://dx.doi.org/10.1098/rsta.2015.0058.

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The circular code theory proposes that genes are constituted of two trinucleotide codes: the classical genetic code with 61 trinucleotides for coding the 20 amino acids (except the three stop codons { TAA , TAG , TGA }) and a circular code based on 20 trinucleotides for retrieving, maintaining and synchronizing the reading frame. It relies on two main results: the identification of a maximal C 3 self-complementary trinucleotide circular code X in genes of bacteria, eukaryotes, plasmids and viruses (Michel 2015 J. Theor. Biol. 380, 156–177. ( doi:10.1016/j.jtbi.2015.04.009 ); Arquès & Miche
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Suchsland, Ruth, Bettina Appel, and Sabine Müller. "Preparation of trinucleotide phosphoramidites as synthons for the synthesis of gene libraries." Beilstein Journal of Organic Chemistry 14 (February 13, 2018): 397–406. http://dx.doi.org/10.3762/bjoc.14.28.

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The preparation of protein libraries is a key issue in protein engineering and biotechnology. Such libraries can be prepared by a variety of methods, starting from the respective gene library. The challenge in gene library preparation is to achieve controlled total or partial randomization at any predefined number and position of codons of a given gene, in order to obtain a library with a maximum number of potentially successful candidates. This purpose is best achieved by the usage of trinucleotide synthons for codon-based gene synthesis. We here review the strategies for the preparation of f
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Dissertations / Theses on the topic "Trinucleotides"

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Devys, Didier. "Etude moleculaire de deux maladies dues a des expansions de trinucleotides." Université Louis Pasteur (Strasbourg) (1971-2008), 1996. http://www.theses.fr/1996STR13030.

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Depuis 1991, des expansions de trinucleotides ont ete retrouvees pour 10 maladies genetiques, alors que ce type de mutation etait inconnu jusqu'alors. Leur mode de transmission presente des caracteristiques tres inhabituelles: biais de transmission parentale des formes les plus severes, augmentation au cours des generations successives du risque de developper la maladie (x fragile) ou de la severite et precocite des manifestations cliniques (anticipation dans la maladie de steinert ou la choree de huntingon). Le syndrome de l'x fragile est du a une expansion instable d'une repetition de trinuc
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Chen, Jinquan. "Femtosecond Transient Absorption Study of Excited-State Dynamics in DNA Model Systems:Thymine-dimer Containing Trinucleotides, Alternate Nucleobases,and Modified Backbone Dinucleosides." The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1343762303.

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LIA, ANNE-SOPHIE. "Dystrophie myotonique de steinert : du developpement foetal humain a un modele transgenique murin - mecanismes et consequences de l'instabilite des trinucleotides ctg repetes." Paris 7, 1998. http://www.theses.fr/1998PA077249.

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La dystrophie myotonique de steinert (dm), maladie autosomique dominante, est la dystrophie musculaire la plus frequente de l'adulte. Une aggravation et une precocite des symptomes au cours des generations successives est observee (phenomene d'anticipation). Le defaut moleculaire correspond a l'amplification d'un trinucleotide repete ctg localise dans la partie 3' non traduite du gene dmpk, codant une serine-threonine kinase. Cette these a pour objectif la comprehension des mecanismes et des consequences de l'instabilite des repetitions ctg dans la dm. Dans une premiere partie, nous avons etud
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Yuan, Qiu-Ping. "Trinucleotide repeats and neuropsychiatric phenotypes /." Stockholm : [Karolinska institutets bibl.], 2001. http://diss.kib.ki.se/2001/91-7349-058-X/.

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Worthey, Elizabeth Anabel. "The evolution of trinucleotide repeats." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.426078.

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Gadgil, Rujuta Yashodhan. "Instability at Trinucleotide Repeat DNAs." Wright State University / OhioLINK, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204.

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Zajac, Pawel. "Parallel target selection by trinucleotide threading." Doctoral thesis, KTH, Genteknologi, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-11284.

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DNA is the code for all life. Via intermediary RNA the information encoded by the genome is relayed to proteins executing the various functions in a cell. Together, this repertoire of inherently linked biological macromolecules determines all characteristics and features of a cell. Technological advancements during the last decades have enabled the pursuit of novel types of studies and the investigation of the cell and its constituents at a progressively higher level of detail. This has shed light on numerous cellular processes and on the underpinnings of several diseases. For the majority of
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Schmidt, Kristina H. "CTG trinucleotide repeat instability in Escherichia coli." Thesis, University of Edinburgh, 1999. http://hdl.handle.net/1842/14353.

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In order to identify cellular factors that affect trinucleotide repeat stability, changes in the length of a (CTG)<sub>43</sub> repeat were studied over 140 generations in wild-type <i>Escherichia coli</i> and in strains that are deficient in post-replicative mismatch repair, secondary structure repair and homologous recombination. It is shown that (CTG)<sub>43</sub> inserted into pUC18 expands and contracts in wild-type <i>E. coli</i> in an orientation-dependent manner that is unaffected by transcription. In cells deficient in post-replicative mismatch repair (CTG)<sub>43</sub> repeat instabi
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Zahra, Rabaab. "CAG.CTG trinucleotide repeat instability in the E.coli chromosome." Thesis, University of Edinburgh, 2006. http://hdl.handle.net/1842/11667.

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In order to identify the molecular basis of genetic instability, a polymerization-independent strategy is developed to generate expanded repeat arrays. The repeat tracts are integrated in the 5’end of <i>lacZ</i> gene in the <i>Escherichia coli</i> chromosome. Using this model system, instability is studied in wild type <i>E. coli</i> and in strains deficient in cellular pathways such as DNA repair, replication and recombination. The work demonstrates that instability (expansion and contraction) in wild type cells is length and orientation dependent. Longer tracts are more unstable than shorte
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Darlow, John M. J. M. "Secondary structure in trinucleotide repeat DNA in vivo." Thesis, University of Edinburgh, 1999. http://hdl.handle.net/1842/13566.

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By inserting different numbers of different trinucleotides into the centre of a long palindrome it has been possible to investigate their tendencies to form hairpins <I>in vivo</I> in any particular alignment and with odd or even numbers of repeat units in the hairpin. It is shown that with d(CAG)<SUP>.</SUP>d(CTG) repeat tracts there is a markedly greater tendency to form hairpins with even numbers of repeat units than with odd numbers whereas d(GAC)<SUP>.</SUP>d(GTC) repeats (which are rare, short, and have not been found to expand) show no such alternation despite having the same base compo
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Books on the topic "Trinucleotides"

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Richard, Guy-Franck, ed. Trinucleotide Repeats. Springer New York, 2020. http://dx.doi.org/10.1007/978-1-4939-9784-8.

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Kohwi, Yoshinori. Trinucleotide Repeat Protocols. Humana Press, 2004. http://dx.doi.org/10.1385/1592598048.

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Kohwi, Yoshinori, and Cynthia T. McMurray, eds. Trinucleotide Repeat Protocols. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-411-1.

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Kohwi, Yoshinori, and Cynthia T. McMurray. Trinucleotide repeat protocols. 2nd ed. Humana Press, 2013.

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Yoshinori, Kohwi, ed. Trinucleotide repeat protocols. Humana Press, 2004.

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Oostra, Ben A., ed. Trinucleotide Diseases and Instability. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-540-69680-3.

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1946-, Oostra Ben A., ed. Trinucleotide diseases and instability. Springer, 1998.

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Oliver, Quarrell, ed. Juvenile Huntington's disease: (and other trinucleotide repeat disorders). Oxford University Press, 2009.

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Pinheiro, Philip Mark. A study of RNA trinucleotide repeats involved in myotonic dystrophy. University of Portsmouth, School of Biological Sciences, 1999.

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Montgomery, Samantha Erin. Structural characterization of slipped trinucleotide repeats and their processing in human cell extracts. National Library of Canada, 2002.

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Book chapters on the topic "Trinucleotides"

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Pirillo, Giuseppe. "A Characterization for a Set of Trinucleotides to be a Circular Code." In Determinism, Holism, and Complexity. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4757-4947-2_22.

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Cooper, Arthur J. L., and John P. Blass. "Trinucleotide-Expansion Diseases." In Neurochemical Mechanisms in Disease. Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-7104-3_11.

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den Dunnen, Wilfred. "Trinucleotide repeat disorders." In Greenfield's Neuropathology 10e Set, 10th ed. CRC Press, 2024. http://dx.doi.org/10.1201/9781003389699-37.

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Kooy, R. Frank, Ben A. Oostra, and Patrick J. Willems. "The Fragile X Syndrome and Other Fragile Site Disorders." In Trinucleotide Diseases and Instability. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-540-69680-3_1.

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MacDonald, Marcy E. "Molecular Genetics of Huntington’s Disease." In Trinucleotide Diseases and Instability. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-540-69680-3_2.

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Waring, J. D., and R. G. Korneluk. "Myotonic Dystrophy." In Trinucleotide Diseases and Instability. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-540-69680-3_3.

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Wells, Robert D., Albino Bacolla, and Richard P. Bowater. "Instabilities of Triplet Repeats: Factors and Mechanisms." In Trinucleotide Diseases and Instability. Springer Berlin Heidelberg, 1998. http://dx.doi.org/10.1007/978-3-540-69680-3_4.

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Clark, H. Brent. "Introduction to Trinucleotide Repeat Diseases." In Neurodegeneration: The Molecular Pathology of Dementia and Movement Disorders. Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444341256.ch25.

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Tsuji, Shoji. "Scope of Trinucleotide Repeat Disorders." In Pathogenesis of Neurodegenerative Disorders. Humana Press, 2001. https://doi.org/10.1007/978-1-59259-106-0_4.

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Faux, Noel. "Single Amino Acid and Trinucleotide Repeats." In Advances in Experimental Medicine and Biology. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-5434-2_3.

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Conference papers on the topic "Trinucleotides"

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Liang, Yupu, Dina Sokol, and Sarah Zelikovitz. "Clustering Tandem Repeats via Trinucleotides." In 2012 IEEE 12th International Conference on Data Mining Workshops. IEEE, 2012. http://dx.doi.org/10.1109/icdmw.2012.57.

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Rocha Filho, Juliano Henrique, Beatriz Brasil Braga, Kristine Leão Alarcão, and Maria Teresa Aires Cabral Dias. "Clinical Findings of Type 3 Spinocerebellar Ataxia." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.198.

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Background: Spinocerebellar ataxias (SCAs) are a genetically heterogeneous group of progressive autosomal disorders of dominant inheritance with a gradual degeneration of the cerebellum and related pathways [1]. This leads to a movement disorder, loss of balance and coordination, accompanied by slurred speech [2]. Among the approximately 40 types of SCA, the spinocerebellar ataxia type 3 (SCA3), also known as Machado-Joseph disease, is the most clinically heterogeneous [3]. It involves the cerebellar, pyramidal, extrapyramidal, motor neuron and oculomotor systems [2]. Objectives: Understand th
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Yagodkin, Andrey, Alex Azhayev, Jarkko Roivainen, et al. "Improved synthesis of trinucleotide phosphoramidites for generation of randomized oligonucleotide libraries." In XIVth Symposium on Chemistry of Nucleic Acid Components. Institute of Organic Chemistry and Biochemistry, Academy of Sciences of the Czech Republic, 2008. http://dx.doi.org/10.1135/css200810309.

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Forras, Patricia Vanda, Sibylle Wilfling, Anne Mrochen, et al. "F70 Early manifestation and rapid symptom progression in a case of homozygous trinucleotide expansion in the TBP-gene in autosomal dominant spinocerebellar ataxia type 17." In EHDN 2022 Plenary Meeting, Bologna, Italy, Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jnnp-2022-ehdn.161.

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Reports on the topic "Trinucleotides"

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Ozcelik, Hilmi. Investigation of Trinucleotide Repeat Expansion in Familial Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada405233.

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