Relevant bibliographies by topics / Troponíny

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Troponíny'

Author: Grafiati
Published: 5 June 2025
Last updated: 15 July 2025

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Journal articles on the topic "Troponíny"

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Tokarčíková, Katarína, Beáta Bolerázska, Tomáš Elexa, and Štefan Hauks. "Laboratórna dynamika akútneho infarktu myokardu / Laboratory Dynamics of Acute Myocardial Infarction." Laboratórna Diagnostika XXVII, no. 2/2022 (2022): 188–95. https://doi.org/10.5281/zenodo.7224731.

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<strong>S&Uacute;HRN</strong> Diagnostika ak&uacute;tneho infarktu myokardu v priebehu niekoľk&yacute;ch desiatok rokov dosiahla veľk&yacute; pokrok predov&scaron;etk&yacute;m vďaka rozvoju v oblasti vy&scaron;etrovac&iacute;ch a laborat&oacute;rnych met&oacute;d. V s&uacute;časnosti sa na potvrden&iacute; diagn&oacute;zy ak&uacute;tneho infarktu myokardu v&yacute;znamn&yacute;m sp&ocirc;sobom podieľa preuk&aacute;zanie laborat&oacute;rnej dynamiky kardiomarkerov. Preferovan&yacute;mi biomarkermi na pos&uacute;denie myokardi&aacute;lneho po&scaron;kodenia s&uacute; tropon&iacute;n T a&nbsp;tro
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Arifulin, Eugene A., and Eugene V. Sheval. "Non-Canonical Localization of Cardiac Troponins: Expanding Functions or Causing Pathologies?" International Journal of Molecular Sciences 25, no. 6 (2024): 3117. http://dx.doi.org/10.3390/ijms25063117.

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The troponin complex—consisting of three subunits: troponin C (TnC), cardiac troponin I (cTnI) and cardiac troponin T (cTnT)—plays a key role in the regulation of myocardial contraction. Troponins are preferentially localized in the cytoplasm and bind to myofibrils. However, numerous, albeit scattered, studies have shown the presence of troponins in the nuclei of muscle cells. There is increasing evidence that the nuclear localization of troponins may be functionally important, making troponins an important nuclear player in the pathogenesis of various diseases including cancer and myopathies.
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Labugger, Ralf, Jeremy A. Simpson, Michelle Quick, et al. "Strategy for Analysis of Cardiac Troponins in Biological Samples with a Combination of Affinity Chromatography and Mass Spectrometry." Clinical Chemistry 49, no. 6 (2003): 873–79. http://dx.doi.org/10.1373/49.6.873.

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Abstract Background: Cardiac troponins are modified during ischemic injury and are found as a heterogeneous mixture in blood of patients with cardiovascular diseases. We present a strategy to isolate cardiac troponins from human biological material, by use of affinity chromatography, and to provide samples ready for direct analysis by mass spectrometry. Methods: Cardiac troponins were isolated from human left ventricular tissue by affinity chromatography. Isolated troponins were either eluted and analyzed by Western blot or enzymatically digested while bound to affinity beads. The resulting pe
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Chaulin, A. M., O. E. Abashina, and D. V. Duplyakov. "High-sensitivity cardiac troponins: detection and central analytical characteristics." Cardiovascular Therapy and Prevention 20, no. 2 (2021): 2590. http://dx.doi.org/10.15829/1728-8800-2021-2590.

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Recently, there have been some important changes in the laboratory diagnosis of patients with acute coronary syndrome, due to the introduction into routine practice of new high- and ultra-sensitive techniques for detection of myocardial damage biomarkers — cardiac troponins. Each method for cardiac troponins’ detection, among the existing wide variety of troponin immunoassays, has different analytical characteristics and allows the detection of different concentrations of troponins in the same patient. With an increasing number of companies developing high-sensitivity troponin immunoassays rec
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Toledo, João Gabriel Assis, Ícaro Morais de Paula Kashima, Wilkes de Oliveira, Wendel Mattos Pompilho, Cristiano Guilherme Alves de Oliveira, and Juliano Gomes Barreto. "Avaliação dos níveis séricos de troponinas I em pacientes com doença renal crônica." Revista Remecs - Revista Multidisciplinar de Estudos Cientí­ficos em Saúde 7, no. 12 (2022): 32–42. http://dx.doi.org/10.24281/rremecs2022.7.12.32-42.

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Troponinas são marcadores biológicos de lesão cardíaca, sendo um dos mais precisos. Nos eventos de Infarto do Agudodo Miocárdio os níveis de troponina tende-se a alterar de 3 a 6 horas após seu início, chegando ao pico em até 12 horas, permanecendo alterado de 7 a 14 dias. No entanto, há relatos de resultados “falsopositivos” em pacientes portadores de lesão renal. O presente trabalho teve como objetivo avaliar os níveis de troponina I em pacientes com doença renal crônica sem evidências clínicas e/ou laboratoriais de lesão miocárdica. O presente trabalho foi realizado na Universidade Iguaçu (
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Zamaraeva, D. V., I. I. Trunina, A. S. Sharykin, and I. M. Osmanov. "CAUSES OF CARDIAC TROPONINS ELEVATING IN CHILDREN WITH CARDIOVASCULAR PATHOLOGY AND ITS VALUE IN CLINICAL PRACTICE." Pediatria. Journal named after G.N. Speransky 100, no. 1 (2021): 119–24. http://dx.doi.org/10.24110/0031-403x-2021-100-1-119-124.

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Troponin I has been successfully used in the diagnosis of acute myocardial infarction in adults. According to recent studies the field of cardiac troponins was widely developed. Troponin I elevated in patients with heart failure, tachyarrhythmias, myocarditis, pulmonary hypertension, intoxications of cardiotoxic drugs, sepsis and other similar conditions. Most literature reviews represent information of using cardiac troponins only among adults. Pediatric publications contain much less information. There are no reference values and practical recommendations for troponin I in children. The aim
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Muhammed, Raman, and Hussein Sinjari. "Assessment of cardiac troponins status in patients on regular hemodialysis." Advanced Medical Journal 10, no. 1 (2025): 44–49. https://doi.org/10.56056/amj.2025.316.

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Background and objectives: heart disease is a significant contributor to both fatalities and health issues among patients undergoing regular hemodialysis. Cardiac Troponins serve as sensitive indicators of heart muscle injury, playing a vital role in diagnosing acute coronary syndrome. The aim of this study is to assess cardiac troponins status in an Iraqi cohort of asymptomatic Hemodialysis patients. Methods: One-hundred-thirty-six asymptomatic hemodialysis patients who visited the hemodialysis center at the Hawler Teaching Hospital in Erbil, Iraq, between August 2022 and May 2023 participate
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Jabor, A., Z. Kubíček, J. Komrsková, et al. "Comparison of troponin I (Abbott, Beckman Coulter, Siemens) and troponin T (Roche) high-sensitivity measurement results." Klinická biochemie a metabolismus 27, no. 2 (2019): 53–60. https://doi.org/10.61568/kbm.2019.009.

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Chaulin, Aleksey M. "Cardiac Troponins Metabolism: From Biochemical Mechanisms to Clinical Practice (Literature Review)." International Journal of Molecular Sciences 22, no. 20 (2021): 10928. http://dx.doi.org/10.3390/ijms222010928.

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The metabolic processes of endo- and exogenous compounds play an important role in diagnosing and treating patients since many metabolites are laboratory biomarkers and/or targets for therapeutic agents. Cardiac troponins are one of the most critical biomarkers to diagnose cardiovascular diseases, including acute myocardial infarction. The study of troponin metabolism is of great interest as it opens up new possibilities for optimizing laboratory diagnostics. This article discusses in detail the key stages of the cardiac troponins metabolism, in particular the mechanisms of release from a heal
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Junaid, Mahmood Alam, Salman Amna, Noureen Shazia, and Sultana Ishrat. "Comparative precision analysis of cardiac Tropinin I (cTnI) on two immunoassay instruments, Roche Cobas e411 and Beckman Coulter Access 2." Chemistry Research Journal 3, no. 6 (2018): 58–62. https://doi.org/10.5281/zenodo.13760149.

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<em>Background</em>: In recent years, exchanging from one company brand or technology to another, for In Vitro Diagnostics, is becoming an important concern for clinical laboratories, when providing services for cardiac biomarkers such as Troponins. <em>Aim:</em> In present study we demonstrated comparative analytical correlation precision compatibility of cTnI on two immunoassay instruments, Cobas e411 and Beckman Coulter Access 2. <em>Materials and Methods:</em> Blood samples were collected from 75 patients and 25 healthy individuals in heparinized tubes. The patients were divided into three
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Dissertations / Theses on the topic "Troponíny"

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Malnic, Bettina. "Expressão do complexo troponina em E. coli e mapeamento dos domínios funcionais da troponina T." Universidade de São Paulo, 1995. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-03042012-121955/.

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A contração muscular esquelética é regulada pelo complexo troponina/tropomiosina de maneira dependente de Ca2+. O complexo troponina consiste de três subunidades: a troponina C (TnC), a troponina I (TnI) e a troponina T (TnT). A troponina C é a subunidade que liga Ca2+, a TnI é a subunidade inibitória e a TnT liga-se fortemente à tropomiosina. A TnI e a TnT são altamente insolúveis a baixas forças iônicas, a não ser que estejam complexadas com a TnC. O complexo troponina pode ser reconstituído \"in vitro\" a partir das subunidades isoladas simplesmente misturando-se as subunidades em razões eq
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Kuster, Nils. "Biomarqueurs des risques cardiaque et métabolique." Thesis, Montpellier, 2016. http://www.theses.fr/2016MONTT049/document.

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Il existe des interactions profondes entre les fonctions rénales et cardiaques. Un dysfonctionnement de l'un ou l'autre de ces deux organes entraîne un dysfonctionnement du second. Ces interactions entre coeur et rein sont regroupées sous le terme de syndromes cardio-rénaux. Les biomarqueurs sont, par définition des indicateurs objectivement mesurés d'un processus biologique normal, d'un processus pathologique ou d'une réponse pharmacologique à une intervention thérapeutique. Les marqueurs biologiques dont très utiles à l'exploration des phénomènes pathologiques cardiaques et rénaux. En pratiq
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Akodad, Mariama. "Marqueurs pronostiques biologiques et morphologiques du TAVI à l’ère de l’évolution des pratiques." Thesis, Montpellier, 2019. http://www.theses.fr/2019MONTT061.

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La prise en charge du rétrécissement aortique a connu une véritable révolution avec l’avènement du remplacement valvulaire aortique percutané (TAVI). Cette technique, s’adressant initialement à des patients à haut risque chirurgical, a été étendue à des patients à moindre risque du fait de l’amélioration des résultats et s’est accompagnée, au fil des années, d’une simplification de la procédure. Cependant, la sélection rigoureuse des patients en amont de la procédure reste la clé du succès de cette technique. Les facteurs cliniques et échographiques sont insuffisants pour permettre une évaluat
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Michima, Lilian Emy dos Santos. "Influência do exercício físico prolongado sobre a concentração sérica de troponina I cardíaca e sobre a função cardíaca em cavalos de enduro." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/10/10136/tde-13122007-143355/.

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Com o objetivo de avaliar se o exercício físico prolongado causa alterações miocárdicas em eqüinos de enduro e se estas alterações cardíacas têm influência no desempenho dos animais durante as provas, avaliaram-se 30 cavalos, divididos em três grupos de dez animais cada, sendo G1 composto por animais que percorreram distâncias acima de 100 km, G2 por animais que percorreram distâncias menores de 100 km e G3 por animais desqualificados por alterações metabólicas. Os animais foram avaliados em três momentos distintos, T0 (pré-exercício, em repouso), T1 (entre 30 e 60 minutos após o exercício) e
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Silva, Gabriela Venturini da. "Identificação de alvos protéicos com potencial diagnóstico e prognóstico em doença arterial coronária." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/5/5166/tde-31072012-111044/.

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Em todo o mundo, milhões de pacientes são atendidos em emergências por apresentarem dor torácica de início aguda, mas apenas uma parcela deve-se a síndrome coronariana aguda (SCA). Em situações como essa é de extrema importância distinguir quando a dor torácica é devido à isquemia do miocárdio, pois esta é de alto risco e o início do tratamento deve ser imediato. Novos biomarcadores são necessários para auxiliar no diagnóstico e conduta clínica a ser tomada diante de situações de emergência como esta. Recentemente a quantificação de troponinas através de ensaios ultrassensíveis tem sido amplam
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Fairhead, Giles. "The interactions of Troponin T with Troponin C, Troponin I and Tropomyosin." Thesis, University of Birmingham, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.422010.

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Santos, Andre Luis Fernandes dos. "Dosagem sérica da enzima creatinafosfoquinase-isoenzima MB (CK-MB) e de troponina I (cTnI) de cães eletrocardiograficamente normais e naqueles com desníveis (infra e supra) do segmento ST, utilizando ensaio imunométrico por quimioluminescência." Universidade de São Paulo, 2005. http://www.teses.usp.br/teses/disponiveis/10/10136/tde-09042008-131819/.

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Ao contrário do homem, as cardiopatias de natureza hipóxica/isquêmica são pouco relatadas nos cães. Raros são os relatos de infarto agudo do miocárdio (IAM) nesses espécimes; entretanto, existem achados eletrocardiográficos que indicam hipóxia/isquemia miocárdica, como os desníveis (infra e supra) do segmento ST. Com o intuito de constatar algum dano nas células do miocárdio em condições de má perfusão, utilizaram-se 38 cães, dos quais 20 com traçados eletrocardiográficos normais e 18 com desníveis (infra e supra) do segmento ST, na derivação DII, velocidade de 50 mm/s e sensibilidade N. Nos a
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González, Del Hoyo Maria Isabel. "Utilidad de la determinación de troponina cardíaca para la predicción de mortalidad y eventos Cardiovasculares en pacientes atendidos en los." Doctoral thesis, Universitat Rovira i Virgili, 2020. http://hdl.handle.net/10803/669817.

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La troponina cardíaca és un marcador específic de dany miocàrdic i la seva elevació s'associa a un pronòstic advers en pacients amb una síndrome coronària aguda. En analogia, s’estableix la hipòtesi de què en pacients sense un esdeveniment coronari, indicaria també un risc augmentat d'esdeveniments cardiovasculars. Per tant, l'objectiu d'aquesta tesi doctoral va ser avaluar la capacitat de la troponina cardíaca per predir esdeveniments adversos en pacients atesos a Urgències amb diferents escenaris clínics. Es tracta de quatre estudis realitzats en pacients visitats al servei d’Urgències de
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Uzuelli, Juliana Alves. "Alterações das concentrações plasmáticas de troponina I e de metaloproteinases 2 e 9 da matriz extracelular após embolia aguda em cães." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/17/17133/tde-17082008-163352/.

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O diagnóstico da tromboembolia pulmonar aguda (EPA) e a avaliação da gravidade desta condição é desafiador. Enquanto as concentrações de troponina I cardíaca (TI) já estão bem estabelecidas quanto ao risco de estratificação, não há estudos prévios que tenham examinado se há alguma relação linear entre as concentrações de TI cardíaca e a gravidade da EPA. Além disso, as metaloproteinases (MMPs) da matriz extracelular estão envolvidas na fisiopatologia da EPA. Entretanto, é desconhecido se o aumento da atividade gelatinolítica das MMPs após a EPA reflete a gravidade desta condição. Nós examinamo
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Farah, Chuck Shaker. "Funções estruturais e regulatórias das regiões N- e C-terminal da troponina I." Universidade de São Paulo, 1994. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-08082012-152838/.

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O complexo troponina-tropomiosina regula a contração muscular esquelética e cardíaca. A ligação do cálcio nos sítios regulatórios localizados no domínio N-terminal da troponina C (TnC) induz uma mudança conformacional que remove a ação inibitória da troponina I (TnI) e inicia a contração muscular. Nós usamos fragmentos recombinantes da TnI e uma série de mutantes da TnC para estudar as interações estruturais e regulatórias das diferentes regiões da TnI com os domínios da TnC, TnT e actina-tropomiosina. Nossos resultados indicam que a TnI é organizada em regiões que apresentam funções estrutura
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Books on the topic "Troponíny"

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Calvert, Melanie Jane. The interactions of cardiac and skeletal troponin I and troponin C. University of Birmingham, 2000.

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Austin, Emily Jane. Characterisation of the interaction sites between Troponin T and Troponin C of the Troponin complex from human cardiac muscle. University of Birmingham, 1999.

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Swanwick, Richard Stephen. Mapping the interactions of the C-terminus of rabbit skeletal troponin T with troponin C and tropomyosin. University of Birmingham, 2003.

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Sabry, Mohamed Abdalla. Development transitions of the contractile regulatory proteins, troponin I and troponin T, in striated muscles: An immunochemical study. University of Birmingham, 1992.

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Cummins, Bernadette. Diagnostic studies using cardiac troponin-1 in myocardial infarction. University of Birmingham, 1987.

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Al-Hillawi, Eman. Characterisation of the regulatory function of cardiac troponin-I. University of Birmingham, 1996.

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Razzaq, Tahir M. Structural functional studies on troponin T probed by protein engineering. University of Birmingham, 1996.

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Jennings, Fiona. A comparison of ward based and laboratory based cardiac troponin measurements. The Author], 2003.

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Probst, Julia. Kardiales Troponin I im Serum des Hundes: Referenzbereich und Untersuchungen an Patienten. Hieronymus, 2002.

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Lennox, Richard J. An evaluation of serum troponin-T as a prognostic indicator in unstable angina. The Author], 1994.

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Book chapters on the topic "Troponíny"

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Augustine, Christopher J., and Eric E. Harrison. "Troponins." In Managing Cardiovascular Risk In Elective Total Joint Arthroplasty. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-26415-3_9.

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Lackner, K. J., and D. Peetz. "Troponin." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_3124.

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Lackner, K. J., and D. Peetz. "Troponin." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2018. http://dx.doi.org/10.1007/978-3-662-49054-9_3124-1.

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Gusev, Nikolai B. "Troponin." In Encyclopedia of Metalloproteins. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-1533-6_56.

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van Balen, J. A. M., A. A. Demeulemeester, M. Frölich, et al. "Troponine I." In Memoboek. Bohn Stafleu van Loghum, 2012. http://dx.doi.org/10.1007/978-90-313-9129-5_120.

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van Balen, J. A. M., A. A. Demeulemeester, M. Frölich, et al. "Troponine T." In Memoboek. Bohn Stafleu van Loghum, 2012. http://dx.doi.org/10.1007/978-90-313-9129-5_121.

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Heilmeyer, Ludwig M. G., Karin Lohmann, Silke U. Reiffert, and Kornelia Jaquet. "Cardiac Troponin." In Interacting Protein Domains. Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60848-3_41.

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Varga, Anita. "Cardiac Troponin." In Interpretation of Equine Laboratory Diagnostics. John Wiley & Sons, Inc., 2017. http://dx.doi.org/10.1002/9781118922798.ch12.

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Mahmud, Zabed, and Peter M. Hwang. "Cardiac Troponin Complex: Cardiac Troponin C (TNNC1), Cardiac Troponin I (TNNI3), and Cardiac Troponin T (TNNT2)." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101901.

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Mahmud, Zabed, and Peter M. Hwang. "Cardiac Troponin Complex: Cardiac Troponin C (TNNC1), Cardiac Troponin I (TNNI3), and Cardiac Troponin T (TNNT2)." In Encyclopedia of Signaling Molecules. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101901-1.

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Conference papers on the topic "Troponíny"

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Chiang, Chang-Yue, Chien-Hsing Chen, Chien-Tsung Wang, Chin-Wei Wu, and Hsing-Yu Chiang. "Trace Determination of Cardiac Troponin I Using a Label-Free Fiber Optical Localized Surface Plasmon Resonance Biosensors." In 3D Image Acquisition and Display: Technology, Perception and Applications. Optica Publishing Group, 2024. http://dx.doi.org/10.1364/3d.2024.jm4a.10.

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This work proposes a carboxyl-graphene-oxide-based fiber optic localized surface plasmon resonance biosensor which employed anti-Cardiac Troponin I (anti-cTnI) as the recognition element to detect the cTnI-protein in 10 min with a limit of detection (LOD) of 5.8 pM, which meets the acceptable LOD for clinical testing of 0.04 ng/mL.
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Puspasari, Ira, Tati L. R. Mengko, Agung W. Setiawan, Miftah Pramudyo, Nobuo Watanabe, and Trio Adiono. "Enhanced Phonocardiogram Analysis Through Troponin Test for the Myocardial Injury Detection." In 2024 International Conference on TVET Excellence & Development (ICTeD). IEEE, 2024. https://doi.org/10.1109/icted62334.2024.10844650.

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Souza, M., X. Hu, and X. Zhang. "AVALIAÇÃO DE UMA NOVA PLATAFORMA DE IMUNOENSAIO RÁPIDO: SUPERFLEX." In Resumos do 54º Congresso Brasileiro de Patologia Clínica/Medicina Laboratorial. Zeppelini Editorial e Comunicação, 2022. http://dx.doi.org/10.5327/1516-3180.140s1.6837.

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Objetivo: Avaliar o desempenho clínico do ensaio SuperFlex cTnl (troponina I). Método: Avaliação do sexo específico da imprecisão total (%CV) no percentil 99 de acordo com o CLSI EP05 e o CLSI EP17-A2; calcular a porcentagem de homens e mulheres saudáveis com concentrações de troponina I iguais ou superiores ao LoD de acordo com a Diretriz(1); avaliar a precisão diagnóstica do ensaio cTnl de acordo com o CLSI EP12-A2. Conclusão: O CV% dividido por sexo e p99th foram 7,15% a 27 ng/l (homens; n = 308) e 7,35% a 24 ng/l (mulheres; n = 312). A taxa de detecção de troponina I (cTnI) de todos os ind
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Gunda, Naga Siva Kumar, and Sushanta K. Mitra. "Microfluidic Based Biosensor for Detection of Cardiac Markers." In ASME 2013 Fluids Engineering Division Summer Meeting. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/fedsm2013-16270.

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Myocardial Infarction (MI) occurs when the blood flow to the heart is blocked. It is major threat to human kind. Current laboratory and ELISA tests are expensive, time consuming, and are not very sensitive. Biosensors can play an important role in the diagnosis of MI without relying on hospital visits. Therefore, researchers are focusing to develop rapid, hand-held, inexpensive biosensors for detecting cardiac markers. In the present study, one of the cardiac markers (Troponin T) is detected using microfluidic based biosensor. Troponin T (cTnT) releases in to the blood serum within 4–6 h after
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Ferreira, Carlos Eduardo dos Santos, Adriana Caschera Leme, Marieli Martins Moreno, Simone Rodrigues Gomes, and Thais Cristine Rodrigues Leonel. "Analytical and clinical performance evaluation of high-sensitivity Cardiac Troponin I (hs-cTnI) measurement through point of care." In Resumos do 56º Congresso Brasileiro de Patologia Clínica/Medicina Laboratorial. Zeppelini Editorial e Comunicação, 2024. https://doi.org/10.5327/1516-3180.142s1.12196.

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Objective: Troponins are the preferred biomarkers for screening patients with chest pain, directing appropriate interventions based on the 0-hour/1-hour algorithm. The Atellica® vTLi POC analyzer, using Magnotech™ Technology, quantifies hs-cTnI levels from a small sample, facilitating prompt evaluation and management of chest pain patients. This study aimed to validate the Atellica® vTLi analyzer for hs-cTnI measurement in patients with suspected acute myocardial infarction (AMI) by comparing its performance against established reference methodologies. Method: The study was conducted in two ph
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Sousa, Beatriz Machado Brandão, Ninivi Daniely Farias Santos, Larissa Medrado Mendes Cavalcante Oliveira, Rômulo Eduardo Leite da Silva, and Jocelia Martins Cavalcante Dantas. "VALOR PROGNÓSTICO DA TROPONINA I EM PACIENTES ACOMETIDOS PELA COVID-19 SEM INFARTO." In 1° Congresso Sul Maranhense de Cardiologia. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/csmc/10.

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INTRODUÇÃO: A infecção pela Síndrome Respiratória Aguda Grave por Coronavírus 2 (SARS-CoV-2) geralmente se manifesta por sintomas de acometimento respiratório, mas também pode ter repercussões extrapulmonares, como complicações cardíacas. Por outro lado, biomarcadores são entidades mensuráveis que podem apontar a existência ou gravidade de certas doenças. Nesse sentido, a alteração dos níveis de troponina I, um biomarcador de lesão cardíaca, torna-se relevante na avaliação de pacientes acometidos pela COVID-19. OBJETIVO: Investigar a relação da troponina I com prognósticos de COVID-19 e fatore
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Morozova, E. P., V. V. Krasitskaya, and L. A. Frank. "DEVELOPMENT OF A LATERAL FLOW APTASENSOR FOR RAPID ANALYSIS OF THE CARDIAC MARKER TROPONIN I." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-91.

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This work demonstrated the fundamental possibility of using aptamers as an alternative to antibodies for performing lateral flow analysis of the cardiac marker Troponin I. Detection of Troponin I in a buffer was carried out using two test systems containing aptamers and antibodies.
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Reser, Marcelo Rodrigues, and Cristine da Rocha. "ALTERAÇÕES DOS MARCADORES CARDÍACOS EM CRIANÇAS DIAGNOSTICADAS COM COVID-19." In I Congresso Brasileiro de Bioquímica Humana On-line. Revista Multidisciplinar em Saúde, 2021. http://dx.doi.org/10.51161/rems/652.

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Introdução: A doença por coronavírus 2019 (COVID-19) é uma infecção causada pelo SARS-CoV-2, potencialmente grave, de elevada transmissibilidade e de distribuição global. A transmissão ocorre principalmente por meio de gotículas respiratórias ou contato com objetos ou superfícies contaminados. Os sintomas clínicos mais comuns vão desde anosmia, ageusia, coriza, diarreia, febre, mialgia, fadiga e cefaleia nos casos leves, acrescentando tosse e febre persistentes nos casos moderados até Síndrome Respiratória Aguda Grave. Aproximadamente 15% dos infectados requerem suporte de oxigênio e cerca de
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Nefedova, V. V., O. I. Kechko, A. M. Matyushenko, and D. I. Levitsky. "MOLECULAR MECHANISMS OF TROPONIN COMPLEX AND TROPOMYOSIN INTERACTION." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-197.

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Studies of the molecular mechanisms of muscle contraction are again becoming extremely relevant. The cornerstone of this kind of research is the establishment of a detailed mechanism of molecular rearrangements in the troponin-tropomyosin system underlying the regulation of muscle contraction in norm and pathology. The aim of this work was to establish the parameters of the interaction between troponin and tropomyosin and the assembly mechanism of this complex.
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Zur, M., S. Congrete, and D. Datta. "Admission Troponin and Rise in Troponin and Outcome inIntermediate Risk (Submassive) Pulmonary Embolism." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a1804.

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Reports on the topic "Troponíny"

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Viksna, Ludmila, Oksana Kolesova, Aleksandrs Kolesovs, Ieva Vanaga, and Seda Arutjunana. Clinical characteristics of COVID-19 patients (Latvia, Spring 2020). Rīga Stradiņš University, 2020. http://dx.doi.org/10.25143/fk2/hnmlhh.

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Data include following variables: Demographics, epidemiological history, comorbidities, diagnosis, complications, and symptoms on admission to the hospital. Also, body’s temperature and SpO2. Blood cells: white cells count (WBC), neutrophils (Neu), lymphocytes (Ly), eosinophils (Eo) and monocytes (Mo), percentages of segmented and banded neutrophils, erythrocytes (RBC), platelet count (PLT), hemoglobin (Hb), and hematocrit (HCT); Inflammatory indicators: erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP); Tissue damage indicators: alanine aminotransferase (ALT), lactate dehydrog
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