Relevant bibliographies by topics / Tubular injuries

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
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Academic literature on the topic 'Tubular injuries'

Author: Grafiati
Published: 22 June 2024
Last updated: 31 July 2025

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Journal articles on the topic "Tubular injuries"

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Kato, Takashi, Man Hagiyama, Yasutoshi Takashima, Azusa Yoneshige, and Akihiko Ito. "Cell adhesion molecule-1 shedding induces apoptosis of renal epithelial cells and exacerbates human nephropathies." American Journal of Physiology-Renal Physiology 314, no. 3 (2018): F388—F398. http://dx.doi.org/10.1152/ajprenal.00385.2017.

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Chronic kidney disease (CKD) is an important problem throughout the world, associated with the increase of blood urea nitrogen (BUN) and serum creatinine (sCre) and with renal tubular injuries. It is crucial to elucidate the molecular mechanisms of renal injuries to identify the new therapeutics and early diagnostic methods. We focused on cell adhesion molecule-1 (CADM1) protein. CADM1, its isoform SP4, is expressed in the epithelial cells of various tissues, including renal distal tubules, localized on the lateral cell membrane, mediates cell-cell adhesion via trans-homophilic binding, and in
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Guan, Yu, Daisuke Nakano, Lei Li, et al. "Protease-Activated Receptor 1 Contributes to Microcirculation Failure and Tubular Damage in Renal Ischemia-Reperfusion Injury in Mice." BioMed Research International 2021 (February 23, 2021): 1–8. http://dx.doi.org/10.1155/2021/6665714.

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Ischemia-reperfusion- (IR-) induced kidney injury is difficult to avoid during renal transplantation and robot-assisted partial nephrectomy. Renal IR injury is characterized by tubular damage, microcirculation failure, and inflammation, which coordinately augment renal injury; however, no specific treatment is available for these conditions. Protease-activated receptor-1 (PAR-1) and its ligand, thrombin, are involved in coagulation and were shown to be associated with epithelial cell injury. Here, we hypothesized that PAR-1 exaggerated renal IR-induced tubular cell damage and microcirculation
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Hosohata, Keiko, Denan Jin, Shinji Takai, and Kazunori Iwanaga. "Vanin-1 in Renal Pelvic Urine Reflects Kidney Injury in a Rat Model of Hydronephrosis." International Journal of Molecular Sciences 19, no. 10 (2018): 3186. http://dx.doi.org/10.3390/ijms19103186.

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Urinary tract obstruction and the subsequent development of hydronephrosis can cause kidney injuries, which results in chronic kidney disease. Although it is important to detect kidney injuries at an early stage, new biomarkers of hydronephrosis have not been identified. In this study, we examined whether vanin-1 could be a potential biomarker for hydronephrosis. Male Sprague-Dawley rats were subjected to unilateral ureteral obstruction (UUO). On day 7 after UUO, when the histopathological renal tubular injuries became obvious, the vanin-1 level in the renal pelvic urine was significantly high
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Huang, Liang-Ti, and Chung-Ming Chen. "Kidney Injuries and Evolution of Chronic Kidney Diseases Due to Neonatal Hyperoxia Exposure Based on Animal Studies." International Journal of Molecular Sciences 23, no. 15 (2022): 8492. http://dx.doi.org/10.3390/ijms23158492.

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Preterm birth interrupts the development and maturation of the kidneys during the critical growth period. The kidneys can also exhibit structural defects and functional impairment due to hyperoxia, as demonstrated by various animal studies. Furthermore, hyperoxia during nephrogenesis impairs renal tubular development and induces glomerular and tubular injuries, which manifest as renal corpuscle enlargement, renal tubular necrosis, interstitial inflammation, and kidney fibrosis. Preterm birth along with hyperoxia exposure induces a pathological predisposition to chronic kidney disease. Hyperoxi
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Gosling, Peter, and Anne J. Sutcliffe. "Proteinuria following Trauma." Annals of Clinical Biochemistry: International Journal of Laboratory Medicine 23, no. 6 (1986): 681–85. http://dx.doi.org/10.1177/000456328602300610.

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Thirteen trauma patients admitted to a major injuries unit were classified according to their injury severity. Urinary excretion of total protein, albumin and gamma glutamyl transpeptidase (GGT) activity were assessed over the following 6 days. All patients showed an initial glomerular and tubular proteinuria during the first 24 h which subsided by the second post-trauma day. The excretion of total protein and albumin was positively correlated with injury severity. Those patients with the severest injuries showed a marked recurrent total proteinuria around days 3 to 4 post-trauma which exhibit
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Duffy, Patrick, Seán McMahon, Xi Wang та ін. "Synthetic bioresorbable poly-α-hydroxyesters as peripheral nerve guidance conduits; a review of material properties, design strategies and their efficacy to date". Biomaterials Science 7, № 12 (2019): 4912–43. http://dx.doi.org/10.1039/c9bm00246d.

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Watanabe, Toru. "Kidney and Urinary Tract Involvement in Kawasaki Disease." International Journal of Pediatrics 2013 (2013): 1–8. http://dx.doi.org/10.1155/2013/831834.

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Kawasaki disease (KD) is a systemic vasculitis and can develop multiple organ injuries including kidney and urinary tract involvement. These disorders include pyuria, prerenal acute kidney injury (AKI), renal AKI caused by tubulointerstitial nephritis (TIN), hemolytic uremic syndrome (HUS), and immune-complex mediated nephropathy, renal AKI associated with either Kawasaki disease shock syndrome or unknown causes, acute nephritic syndrome (ANS), nephrotic syndrome (NS), renal tubular abnormalities, renal abnormalities in imaging studies, and renal artery lesions (aneurysms and stenosis). Pyuria
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Futrakul, Narisa, and P. Futrakul. "Renal microvascular and tubular injuries in type II diabetic nephropathy." Kidney International 74, no. 3 (2008): 390. http://dx.doi.org/10.1038/ki.2008.173.

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Taneda, Sekiko, Kazuho Honda, Kimiko Tomidokoro, Kenta Uto, Kosaku Nitta, and Hideaki Oda. "Eicosapentaenoic acid restores diabetic tubular injury through regulating oxidative stress and mitochondrial apoptosis." American Journal of Physiology-Renal Physiology 299, no. 6 (2010): F1451—F1461. http://dx.doi.org/10.1152/ajprenal.00637.2009.

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The present study was designed to elucidate a possible mechanism of hyperglycemia-induced tubular injury and to examine a therapeutic potential of dietary eicosapentaenoic acid (EPA) for the prevention of diabetic kidney disease. Utilizing streptozotocin-induced diabetic mice, the extents of albuminuria and histological injuries were monitored at 2 wk after diabetic induction. Reactive oxygen species (ROS) production, apoptosis, and hypoxia in the kidney were evaluated by immunohistochemistry and Western blotting. An in vitro study was performed using rat proximal tubular cells (NRK-52E) to co
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Xiong, Mingxia, Lei Jiang, Yang Zhou та ін. "The miR-200 family regulates TGF-β1-induced renal tubular epithelial to mesenchymal transition through Smad pathway by targeting ZEB1 and ZEB2 expression". American Journal of Physiology-Renal Physiology 302, № 3 (2012): F369—F379. http://dx.doi.org/10.1152/ajprenal.00268.2011.

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Most chronic kidney injuries inevitably progress to irreversible renal fibrosis. Tubular epithelial-to-mesenchymal transition (EMT) is recognized to play pivotal roles in the process of renal fibrosis. However, a comprehensive understanding of the pathogenesis of renal scar formation and progression remains an urgent task for renal researchers. The endogenously produced microRNAs (miRNAs), proved to play important roles in gene regulation, probably regulate most genes involved in EMT. In this study, we applied microarray analysis to investigate the expression profiles of miRNA in murine inters
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Dissertations / Theses on the topic "Tubular injuries"

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RUEGG, CHARLES EDWARD. "MECHANISMS UNDERLYING REGIOSELECTIVE ACUTE TUBULAR NECROSIS OF RENAL PROXIMAL TUBULAR SEGMENTS." Diss., The University of Arizona, 1987. http://hdl.handle.net/10150/184162.

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The convoluted (CPT) and straight (SPT) portions of the renal proximal tubule are susceptible to injury by a wide variety of chemical agents. These agents often affect the CPT or SPT selectively by proposed mechanisms usually attributed to tubular concentration, blood flow delivery patterns and tubuloglomerular feedback responses within the intact kidney. The innate cellular responses to chemical exposures remain virtually unexplored. Hence, the basic goal of this research was to develop an in vitro system that was conducive to examining the innate cellular differences in susceptibility betwee
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SILBER, PAUL MICHAEL. "EARLY INDICATION AND PATHOGENESIS OF RENAL PROXIMAL TUBULE INJURY (ENZYMURIA)." Diss., The University of Arizona, 1987. http://hdl.handle.net/10150/184097.

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It is well known that a variety of toxicants can cause damage to the renal proximal tubule. However, the early pathogenesis of these deleterious interactions between a toxicant and this region of the nephron remain poorly understood. Thus, the purpose of this research was to attempt to answer three interrelated questions. First, what are the earliest changes in kidney function and structure after administration of tubule toxicants in vivo? Secondly, how do these structural/functional alterations change over time? Finally, are certain indicators of renal "dysfunction" more sensitive then other
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Rubira, Cláudio José [UNESP]. "Efetividade de antibióticos em pacientes com trauma de tórax submetidos à toracostomia tubular fechada: revisão sistemática e metanálise." Universidade Estadual Paulista (UNESP), 2008. http://hdl.handle.net/11449/91400.

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Made available in DSpace on 2014-06-11T19:25:19Z (GMT). No. of bitstreams: 0 Previous issue date: 2008-05-09Bitstream added on 2014-06-13T19:52:57Z : No. of bitstreams: 1 rubira_cj_me_botfm.pdf: 645645 bytes, checksum: e75d94d4917e2b0afc20438d49f14b0f (MD5)<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)<br>Cerca de um terço dos traumas de tórax necessitam de hospitalização, e a grande maioria destes pacientes é tratada com toracostomia tubular fechada. A principal morbidade associada a este procedimento é o empiema pleural. Devido aos possíveis índices de morbidade e
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Rubira, Cláudio José. "Efetividade de antibióticos em pacientes com trauma de tórax submetidos à toracostomia tubular fechada : revisão sistemática e metanálise /." Botucatu : [s.n.], 2008. http://hdl.handle.net/11449/91400.

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Resumo: Cerca de um terço dos traumas de tórax necessitam de hospitalização, e a grande maioria destes pacientes é tratada com toracostomia tubular fechada. A principal morbidade associada a este procedimento é o empiema pleural. Devido aos possíveis índices de morbidade e mortalidade relacionados ao empiema pós- traumático e as implicações do uso irracional de antibióticos, acreditamos ser relevante a realização desta revisão sistemática. Objetivos: Avaliar a efetividade da administração de antibiótico na redução de empiema em pacientes com toracostomia tubular devido a trauma. Métodos: Revis
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Niasse, Aïssata. "Rôle de STAT5 dans l’épithélium rénal." Electronic Thesis or Diss., Sorbonne université, 2020. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2020SORUS441.pdf.

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Au cours des maladies rénales, les facteurs tissulaires et en particulier épithéliaux sont capables de moduler les lésions rénales ainsi que le pronostic de la maladie. La voie de signalisation JAK/STAT (Janus kinase / Signal transducers and activators of transcription), classiquement décrite dans les cellules immunitaires, a été décrite récemment dans les cellules rénales intrinsèques. Pour la première fois, nous montrons ici l'activation intraépithéliale de novo de STAT5 chez l'Homme et au cours des lésions glomérulaires et tubulaires in vivo. De plus, la délétion de STAT5 dans l'épithélium
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Books on the topic "Tubular injuries"

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Turner, Neil. Mechanisms of progression of chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0136.

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Three major hypotheses attempt to explain progressive kidney disease following diverse diseases and injuries. To varying degrees they can explain the observed risk factors for progression and the ability of interventions to lower risk. The hyperfiltration hypothesis argues that progression is due to stress on residual nephrons leading to injury and damage to remaining glomeruli. The toxicity of proteinuria hypothesis proposes that serum proteins or bound substances are toxic to tubular or tubulointerstitial cells. This sets up cycles of damage which lead to tubulointerstitial scarring. The pod
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Henderson, Lorna K., Brian J. Nankivell, and Jeremy R. Chapman. Chronic allograft dysfunction. Edited by Jeremy R. Chapman. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0286.

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Despite improvements in short-term renal allograft survival, long-term survival has not appreciably changed. Excepting death with a functioning graft, most late graft loss results from chronic allograft dysfunction. Immune and non-immune-mediated injuries contribute to graft dysfunction over time, ultimately leading to a non-specific and irreversible histological end-point of fibrosis, tubular atrophy, and glomerulosclerosis. Screening and early identification of pathology is crucial to allow timely intervention in order to prevent permanent nephron damage and graft loss. This chapter outlines
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Book chapters on the topic "Tubular injuries"

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Trump, Benjamin F., and Irene K. Berezesky. "Ion Deregulation in Injured Proximal Tubule Epithelial Cells." In Nephrotoxicity. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4757-2040-2_113.

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Picken, M. M. "Monoclonal Gammopathies: Glomerular and Tubular Injuries." In Pathobiology of Human Disease. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-12-386456-7.05411-3.

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Parilovsky, Оlexander, and Ivan Yatsenko. "FORENSIC VETERINARY CHARACTERISTICS OF FRACTURES, FRACTURE DISLOCATIONS, DISLOCATIONS AND SUBLUXATIONS OF THE BONES IN THE ANIMAL SKELETONS QUALIFIED AS SEVERE INJURIES." In Priority areas for development of scientific research: domestic and foreign experience. Publishing House “Baltija Publishing”, 2021. http://dx.doi.org/10.30525/978-9934-26-049-0-42.

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The article presents forensic veterinary characteristics of fractures, fracture dislocations, dislocations and subluxations of skeletal bones in animals classified as severe injuries, including open and closed fractures of the upper and lower jaws, which lead to inability to receive normal food and water after healing; fracture or fracture dislocation of one or more thoracic or lumbar vertebrae along with spinal cord dysfunction or in the presence of clinically established severe shock; fracture of the dorsal or ventral arch of the first cervical vertebra; fractures and fracture dislocations o
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Harris, Kevin P. G. "Proteinuria: Implications for Progression and Management." In Mechanisms and Clinical Management of Chronic Renal Failure. Oxford University PressNew York, NY, 2000. http://dx.doi.org/10.1093/oso/9780192629333.003.0005.

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Abstract It is a well established observation from both human and animal studies of renal disease, that once a degree of renal damage has occurred then the progression of kidney failure is inexorable, even if the original injurious process has resolved [1]. Such observations have led to the hypothesis that following renal damage of any aetiology a maladaptive response occurs in the remaining nephrons which results in their eventual destruction by a common pathogenic mechanism. In support of this, the histological appearance of the kidney in end-stage renal disease is similar whatever the initi
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ware, J. Anthony. "Cellular Mechanisms of Angiogenesis." In Angiogenesis And Cardiovascular Disease. Oxford University PressNew York, NY, 1999. http://dx.doi.org/10.1093/oso/9780195112351.003.0002.

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Abstract Alterations in endothelial cell function occur not only as part of new vessel formation, but also wound healing. Small injuries to the endothelial cell monolayer heal by migration of adjacent endothelial cells into the wound; after they reattach to the underlying matrix, they then reenter the cell cycle and divide to replace the denuded cells. In angiogenesis, the first step under most circumstances is dissolution of the underlying matrix, brought about by release of proteases (e.g., collagenases and plasminogen activator) from the stimulated endothelial cells themselves or from cells
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Conference papers on the topic "Tubular injuries"

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Hassan, T. F., M. Hussain, Khaled Al Rumaithi, et al. "Offline Tubulars Bucking Service with Triplex Catwalks." In ADIPEC. SPE, 2024. http://dx.doi.org/10.2118/222112-ms.

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Abstract Significant operational drilling hazards and incidents while handling and running the drilling tubulars such as drops, hand injuries, line of fire and working at height, moreover the drilling tripping speed repeatedly started to comprise the drilling efficiency and the planned well delivery. The newly introduced offline bucking service and triplex catwalks technology is a progressive shift towards an efficient well construction operation by revolutionizing tubular handling as a significant improvement to maintain safe work environment, reduce the CO2 emission, well delivery optimizati
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Mercier, Melina, Corin Shirley, Shelby Stafford, et al. "Fluidic Flexible Matrix Composites for Volume Management in Prosthetic Sockets." In ASME 2014 Conference on Smart Materials, Adaptive Structures and Intelligent Systems. American Society of Mechanical Engineers, 2014. http://dx.doi.org/10.1115/smasis2014-7706.

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Persons with transfemoral and transtibial protheses experience changes in the volume of their residual limb during the course of the day. These changes in volume unavoidably lead to changes in quality of fit of the prosthesis, skin irritations, and soft tissue injuries. The associated pain and discomfort can become debilitating by reducing one’s ability to perform daily activities. While significant advancements have been made in prostheses, the undesirable pain and discomfort that occurs due to the volume change is still a major challenge that needs to be solved. The goal of this program is t
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Omar, Tarek A., Nabih E. Bedewi, and Azim Eskandarian. "Significant Severity Reduction of Side-Impact Injuries by Using ITS Airbags: FE Simulation and Severity Analysis." In ASME 2002 International Mechanical Engineering Congress and Exposition. ASMEDC, 2002. http://dx.doi.org/10.1115/imece2002-39083.

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The Inflatable Tubular Structure (ITS) airbag is a potentially life-saving device that has been implemented recently in some luxury vehicles. Its main objective is to provide head protection for the front seat occupants against upper side-interior car components. In a previous research conducted by the authors, a nonlinear Finite Element (FE) model for the ITS-airbag system was successfully developed and tested. In the current research, the developed ITS model is combined with a full-scale FE vehicle model and 50th percentile side-impact dummy (SID) model. The combined model is then used to co
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Weinmann, Denise, Peter P. Pott, and Max B. Scha ̈fer. "Steerable Tip Enables Expanding Robot to Pass Sigmoid Curve." In The Hamlyn Symposium on Medical Robotics. The Hamlyn Centre Imperial College London, 2024. http://dx.doi.org/10.31256/hsmr2024.55.

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Colonoscopy is a powerful tool for the early detection of colorectal diseases. State-of-the-art colonoscopy, yet effective, often elicits discomfort among individuals, deterring some from undergoing this crucial diagnostic procedure. In addition, advancing the colonoscope up to the ileocecal valve demands a high level of expertise of the physician. The length of the colon requires the colonoscope to have a certain bending stiffness to resist buckling and looping during insertion, leading to substantial contact forces exerted on the colon wall. This contributes to patient discomfort and the ris
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GUO, Da, Jian-min WANG, and Jian-ming OUYANG. "Injured Human Kidney Proximal Tubular Epithelial Cells Modulate Nucleation and Growth of Calcium Oxalate Crystals." In 2nd International Conference on Biomedical and Biological Engineering 2017 (BBE 2017). Atlantis Press, 2017. http://dx.doi.org/10.2991/bbe-17.2017.27.

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Bieber, Malte, Sarah Menzel, Anja Lena Thiebes, et al. "Viability of coaxial atomization for disintegration of cell solutions in cell spray applications." In ILASS2017 - 28th European Conference on Liquid Atomization and Spray Systems. Universitat Politècnica València, 2017. http://dx.doi.org/10.4995/ilass2017.2017.4609.

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Treating Leukemia with intravenous stem cell transplantation represents a well-established therapy technique. For applications, that require high local cell concentrations, transplantation by conventional intravenous injection is less potent, due to cell distribution with blood circulation. Instead, spraying them directly onto the injured or diseased area shows promising results in various applications, e.g. superficial treatment of topographically challenging wounds, in situ seeding of cells on implants, deposition of cells in tubular organs for stem cell therapy. The present work aims for a
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Reports on the topic "Tubular injuries"

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Goel, Dr Divanshu, and Dr Manjeet Singh. HYBRID EXTERNAL FIXATION FOR PROXIMAL TIBIAL FRACTURES. World Wide Journals, 2023. http://dx.doi.org/10.36106/ijar/1505336.

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Introduction: Intraarticular fractures of the tibial plateau and periarticular fractures of the proximal tibia, caused by high energy trauma pose a therapeutic dilemma. Such fractures are associated with extensive soft tissue damage with or without compound injury. The management of such high velocity injuries become a challenge to the trauma surgeons. The goals of these periarticular fractures management are 1. Restoration of joint congruity by anatomic reduction 2. Stable xation of fractures thus allowing early movements 3. Proper care of injured soft tissues. In earlier days uniplanar exte
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