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Journal articles on the topic 'Tubuloreticular inclusions'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Katzberg, H. D., and D. G. Munoz. "Tubuloreticular inclusions in inclusion body myositis." Clinical Neuropathology 29, no. 07 (2010): 262–66. http://dx.doi.org/10.5414/npp29262.

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2

Zirkin, Richard M., Gilbert E. Herman, and Edna Elfont. "Tubuloreticular inclusions in colonic mucosa." Human Pathology 19, no. 3 (1988): 370–71. http://dx.doi.org/10.1016/s0046-8177(88)80535-5.

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3

Harb, Joseph M., Jerome V. Murphy, and Thomas T. Tang. "Cerebral Tubuloreticular Inclusions In Reye's Syndrome." Proceedings, annual meeting, Electron Microscopy Society of America 43 (August 1985): 664–65. http://dx.doi.org/10.1017/s0424820100120011.

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IntroductionTubuloreticular inclusions (TRI) were first reported in patients with herpesvirus encephalitis or systemic lupus erythematosus (SLE). TRI are abnormal subcellular structures which consist of fine irregularly branched, anastomosing tubules within cisternae of endoplasmic reticulum. TRI have also been reported in blood lymphoreticular cells, vascular endothelial cells, mesenchymal cells, and epithelial cells in a variety of clinical conditions which include SLE and other autoimmune diseases, congenital immunodeficiency, lymphoproliferative disorders, neoplasms, neurodegenerative diso

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4

Willicombe, M., C. Roufosse, L. Moran, J. Moss, D. Taube, and T. Cook. "Significance of Tubuloreticular Inclusions in Renal Allografts." Transplantation 98 (July 2014): 529. http://dx.doi.org/10.1097/00007890-201407151-01779.

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5

Madewell, B. R., and R. J. Munn. "Tubuloreticular inclusions in equine connective tissue neoplasms." Journal of Comparative Pathology 100, no. 4 (1989): 449–52. http://dx.doi.org/10.1016/0021-9975(89)90011-x.

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6

Nossent, Johannes, Warren Raymond, Milica Ognjenovic, Alexandra Kang, Aron Chakera, and Daniel Wong. "The importance of tubuloreticular inclusions in lupus nephritis." Pathology 51, no. 7 (2019): 727–32. http://dx.doi.org/10.1016/j.pathol.2019.07.007.

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7

Gaillard, François, Sophie Ismael, Aurélie Sannier, et al. "Tubuloreticular inclusions in COVID-19–related collapsing glomerulopathy." Kidney International 98, no. 1 (2020): 241. http://dx.doi.org/10.1016/j.kint.2020.04.022.

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8

Lee, Jee Youn, Seung Hwan Song, Yu Seun Kim, et al. "Tubuloreticular inclusions in peritubular capillaries of renal allografts." Pathology - Research and Practice 213, no. 9 (2017): 1185–90. http://dx.doi.org/10.1016/j.prp.2017.06.009.

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9

Lee, Chang-Joo, Kwang-Sun Suh, Kyung-Hee Kim, Yoon-Kyung Chang, Ki-Ryang Na, and Kang-Wook Lee. "The Clinicopathologic Significance of Endothelial Tubuloreticular Inclusions in Glomerular Diseases." Ultrastructural Pathology 37, no. 6 (2013): 386–94. http://dx.doi.org/10.3109/01913123.2013.814738.

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10

PEDERSEN, COURT, THOMAS HORN, JETTE JUNGE, SVEN HAAHR, and JENS OLE NIELSEN. "Tubuloreticular inclusions in skin biopsies from patients with HIV infection." APMIS 97, no. 1-6 (1989): 249–52. http://dx.doi.org/10.1111/j.1699-0463.1989.tb00784.x.

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11

Squire, Jeremy, Venita Jay, Laurence Becker, and Jim Rutka. "MYCN AMPLIFICATION AND TUBULORETICULAR INCLUSIONS IN A PEDIATRIC MALIGNANT GLIOMA." Journal of Neuropathology and Experimental Neurology 52, no. 3 (1993): 322. http://dx.doi.org/10.1097/00005072-199305000-00249.

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12

El-Sayegh, Suzanne, Geovani Faddoul, Ninad Parekh, Chetana Rondla, and Elie El-Charabaty. "Collapsing Focal Segmental Glomerulosclerosis and Tubuloreticular Inclusions in Lupus – A Case Report." OALib 01, no. 01 (2014): 1–4. http://dx.doi.org/10.4236/oalib.preprints.1200013.

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13

Haas, Mark, Andrew J. Aronson, and Sharon M. Bartosh. "Progressive postinfectious glomerulonephritis with multiple tubuloreticular inclusions in an HIV-negative patient." American Journal of Kidney Diseases 30, no. 5 (1997): 725–28. http://dx.doi.org/10.1016/s0272-6386(97)90502-3.

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14

Kuyama, Jun, Yoshio Kanayama, Shuichi Katagiri, Toshiharu Tamaki, Takeshi Yonezawa, and Seiichiro Tarui. "Tubuloreticular Inclusions and Paired Cisternae Induced in Human Lymphocytes Cultured withStaphylococcus AureusCowan 1." Ultrastructural Pathology 8, no. 2-3 (1985): 155–63. http://dx.doi.org/10.3109/01913128509142149.

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15

Willicombe, Michelle, Jill Moss, Linda Moran, et al. "Tubuloreticular Inclusions in Renal Allografts Associate with Viral Infections and Donor-Specific Antibodies." Journal of the American Society of Nephrology 27, no. 7 (2015): 2188–95. http://dx.doi.org/10.1681/asn.2015050478.

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16

Alpers, Charles E., Sami Harawi, and Helmut G. Rennke. "Focal Glomerulosclerosis With Tubuloreticular Inclusions: Possible Predictive Value for Acquired Immunodeficiency Syndrome (AIDS)." American Journal of Kidney Diseases 12, no. 3 (1988): 240–42. http://dx.doi.org/10.1016/s0272-6386(88)80129-x.

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17

Lane, R. J. M., K. A. McLean, J. Moss, and D. F. Woodrow. "Myopathy in HIV infection: the role of zidovudine and the significance of tubuloreticular inclusions." Neuropathology and Applied Neurobiology 19, no. 5 (1993): 406–13. http://dx.doi.org/10.1111/j.1365-2990.1993.tb00462.x.

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18

Feldman, Dorothy, Richard M. Hoar, Wendell H. Niemann, Thomas Valentine, Mark Cukierski, and Andrew G. Hendrickx. "Tubuloreticular inclusions in placental chorionic villi of rhesus monkeys after maternal treatment with interferon." American Journal of Obstetrics and Gynecology 155, no. 2 (1986): 413–24. http://dx.doi.org/10.1016/0002-9378(86)90844-6.

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19

Sidhu, Gurdip S., and Nicholas D. Cassai. "Aids: Tubuloreticular Structures (Trs), Cylindrical Confronting Cisternae (Ccc), and Related Alterations." Microscopy and Microanalysis 5, S2 (1999): 1092–93. http://dx.doi.org/10.1017/s143192760001878x.

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TRS and CCC are endoplasmic reticulum membrane-derived structures seen in HIV-infected individuals in a variety of cell types. TRS (synonym: tubuloreticular inclusions) are 24-25 nm in diameter, branching tubules which are short or long and associated with the granular or smooth endoplasmic reticulum, the Golgi complex, the perinuclear cistern, and annulate lamellae (Fig. 1-3, 8). The tubules are noticeably more stretched out in Kaposi's sarcoma endothelial cells, presumably by an increase of matrix material within the reticulum sac (Fig. 3). TRS consist of membranous components, including pol

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20

Kuyama, Jun, Yoshio Kanayama, Hajime Mizutani, et al. "Formation of Tubuloreticular Inclusions in Mitogen-Stimulated Human Lymphocyte Cultures by Endogenous or Exogenous Alpha Interferon." Ultrastructural Pathology 10, no. 1 (1986): 77–85. http://dx.doi.org/10.3109/01913128609015565.

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21

Yang, A. H., B. S. Lin, K. L. Kuo, C. C. Chang, Y. Y. Ng, and W. C. Yang. "The clinicopathological implications of endothelial tubuloreticular inclusions found in glomeruli having histopathology of idiopathic membranous nephropathy." Nephrology Dialysis Transplantation 24, no. 11 (2009): 3419–25. http://dx.doi.org/10.1093/ndt/gfp288.

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22

Singh, Gautam Kumar, Sandeep Arora, Debdeep Mitra, Pankaj Das, Vikram Singh, and Akanksha Gupta. "Dive Deep into Trophic Ulcer; Glomerulonephritis is Still a Complication in Hansen'S Disease." International Journal of Mycobacteriology 10, no. 2 (2021): 199–201. http://dx.doi.org/10.4103/ijmy.ijmy_41_21.

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Hansen's disease is one of the ancient skin diseases known to humankind, still foxes even trained physician leading to delay in its diagnosis and unusual health burden. India followed by Brazil constitutes the highest number in newly diagnosed cases. Even though the World Health Organization and individual country have done much to contain the spread of leprosy, the findings of systemic complications, grade 2 deformity, and childhood leprosy reflect some shortcomings of the worldwide public health program. Renal involvement, particularly glomerulonephritis, is a known common systemic complicat

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23

Howell, D. N., and L. A. Szczech. "Contributions of Microscopy to the Diagnosis and Investigation of Aids-Associated Renal Disease." Microscopy and Microanalysis 5, S2 (1999): 1094–95. http://dx.doi.org/10.1017/s1431927600018791.

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Microscopy has had a major role in the analysis of renal disorders associated with human immunodeficiency virus (HIV) infection, both as a diagnostic method and as a means of studying pathogenic mechanisms. In the diagnostic realm, microscopic analysis of renal tissue obtained at biopsy and autopsy is a mainstay for the detection of a wide range of glomerular, vascular, and tubulointerstitial diseases. As an investigative tool, microscopy has made an important, albeit somewhat controversial, contribution to our understanding of the pathogenesis of at least one HIV-associated renal lesion.A var

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24

Elmaghrabi, Ayah, Elizabeth Brown, Ei Khin, Jared Hassler, and Allen R. Hendricks. "Tubuloreticular Inclusions in the Absence of Systemic Lupus Erythematosus and HIV Infection: A Report of Three Pediatric Cases." Case Reports in Nephrology and Dialysis 7, no. 2 (2017): 91–101. http://dx.doi.org/10.1159/000477661.

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Tubuloreticular inclusions (TRIs) are subcellular structures located within the cisternae of endoplasmic reticulum. Formation of TRIs has been linked to the exposure of excess interferon (IFN), either from endogenous or exogenous sources. In renal disease, TRIs have been most commonly associated with systemic lupus erythematosus (SLE), and human immunodeficiency virus-associated nephropathy (HIVAN). Case reports of patients with renal biopsies showing TRIs without underlying SLE or HIV are infrequent in adults, and to our knowledge none have been reported in children. We report 3 pediatric cas

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25

Price, George, and Lizardo Cerezo. "Clq nepropathy: A case report of a newly described glomerularopathy." Proceedings, annual meeting, Electron Microscopy Society of America 45 (August 1987): 618–19. http://dx.doi.org/10.1017/s0424820100127529.

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A distinctly new pathologic entity that causes steroid resistant nephrotic syndrome has been described by Jennette and Hipp. Clq nephropathy gets its name from the strong positivity of Clq localized by immunofluorescence. Reactions for C3, IgG and IgM are present, with a mild reaction for IgA occuring in some cases.The ultrastructural features of Clq glomerular nephropathy (Clq GN) and systemic lupus erythmatosis glomerular nephropathy (SLE GN) are strikingly similar in that they both demonstrate mesangial cell proliferative activity, mesangial dense deposits, and subendothelial or subepitheli

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26

Noemi, Esparza Martín. "Renal-limited lupus-like glomerulonephritis." Archives of Renal Diseases and Management 3, no. 2 (2017): 048. https://doi.org/10.17352/2455-5495.000029.

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In  the  setting  of  an  IgG-dominant  immune  complex-mediated  glomerulonephritis,  there  are  multiple  pathological fi ndings that strongly suggest the diagnosis of lupus nephritis (LN) including “full-house” immunofluorescence staining for IgG,  IgM,  IgA,  C3  and  C1,  extraglomerular  immune  deposits,  combined mesangial, subendothelial and subepithelial immune deposits   and   the   presence   of   endothelial &n

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27

Marquart, Karl-Horst. "Occurrence of Tubuloreticular Structures and Intracisternal Paracrystalline Inclusions in Endothelial Cells of Tissue from Different Epidemiological Types of Kaposi's Sarcoma." Ultrastructural Pathology 29, no. 2 (2005): 85–93. http://dx.doi.org/10.1080/01913120590912205.

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28

Schänzer, Anne, Leonie Rager, Iris Dahlhaus, et al. "Morphological Characteristics of Idiopathic Inflammatory Myopathies in Juvenile Patients." Cells 11, no. 1 (2021): 109. http://dx.doi.org/10.3390/cells11010109.

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Background: In juvenile idiopathic inflammatory myopathies (IIMs), morphological characteristic features of distinct subgroups are not well defined. New treatment strategies require a precise diagnosis of the subgroups in IIM, and, therefore, knowledge about the pathomorphology of juvenile IIMs is warranted. Methods: Muscle biopsies from 15 patients (median age 8 (range 3–17) years, 73% female) with IIM and seven controls were analyzed by standard methods, immunohistochemistry, and transmission electron microscopy (TEM). Detailed clinical and laboratory data were accessed retrospectively. Resu

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29

Jay, Venita, James Rutka, Laurence E. Becker, and Jeremy Squire. "Pediatric malignant glioma with tubuloreticular inclusions and MYCN amplification. Report of a case with immunohistochemical, ultrastructural, flow cytometric, karyotypic, and southern blot analysis." Cancer 73, no. 7 (1994): 1987–93. http://dx.doi.org/10.1002/1097-0142(19940401)73:7<1987::aid-cncr2820730734>3.0.co;2-j.

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30

Sathi, Satyanand, Alok Sharma, Anil Kumar Garg, et al. "An Interesting Case of Nonlupus Full-House Nephropathy." Case Reports in Nephrology 2021 (December 28, 2021): 1–4. http://dx.doi.org/10.1155/2021/9043003.

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Full-house immunofluorescence and endothelial tubuloreticular inclusions are known as characteristic features of lupus nephritis. However, both features are not pathognomonic for lupus nephritis. A kidney biopsy specimen showing full-house immunofluorescence pattern in the absence of autoantibodies and classical clinical features of Systemic Lupus Erythematosus (SLE) is now considered as nonlupus full-house nephropathy (FHN). Nonlupus FHN may be idiopathic or due to other disease processes known as secondary nonlupus FHN. Here, we report the case of a 36-year-old female who presented with neph

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Jain, Akshat, Jaiswal Vidyaagar, and Ravi Raghvan. "Sickle Cell Disease with Dermatomyositis - a Rare and Complex Comorbidity." Blood 138, Supplement 1 (2021): 4174. http://dx.doi.org/10.1182/blood-2021-148878.

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Abstract Introduction and Case Presentation - A 4 yrs old female with sickle cell disease (SCD) and intermittent asthma presented with polyarthralgia predominantly involving bilateral hip and knee joints and became non-ambulatory over a course of 2 months. She developed chronic facial swelling, and a pruritic erythematous rash involving face, extensor surface of the hand and the right knee with significant weight loss. Physical examination was significant for heliotropic rash on the eyelids, Also present were Gottron's papules and macules on both hands and right knee along with right leg tende

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32

Bromfield, M., R. McQuillan, R. John, and C. Avila-Casado. "The significance of tubuloreticular inclusions as a marker of systemic stimulation by interferons in a case of focal and segmental glomerulosclerosis associated with cytomegalovirus (CMV) infection." Clinical Kidney Journal 7, no. 2 (2014): 174–78. http://dx.doi.org/10.1093/ckj/sft156.

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33

Kudose, Satoru, Dominick Santoriello, Andrew S. Bomback, M. Barry Stokes, Vivette D. D’Agati, and Glen S. Markowitz. "Sensitivity and Specificity of Pathologic Findings to Diagnose Lupus Nephritis." Clinical Journal of the American Society of Nephrology 14, no. 11 (2019): 1605–15. http://dx.doi.org/10.2215/cjn.01570219.

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Background and objectivesIn 2012, the Systemic Lupus International Collaborating Clinics proposed that lupus nephritis, in the presence of positive ANA or anti-dsDNA antibody, is sufficient to diagnose SLE. However, this “stand-alone” kidney biopsy criterion is problematic because the ISN/RPS classification does not specifically define lupus nephritis. We investigated the combination of pathologic features with optimal sensitivity and specificity for the diagnosis of lupus nephritis.Design, setting, participants, & measurementsThree hundred consecutive biopsies with lupus nephritis and 560

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34

Bu, Lihong, Bo Ye, Anne M. Kouri, and Youngki Kim. "Diagnostic Utility of Galactose-Deficient Immunoglobulin A1 Immunostaining in the Differentiation of Lupus Nephritis and Immunoglobulin A Nephropathy." Glomerular Diseases 1, no. 1 (2021): 34–39. http://dx.doi.org/10.1159/000511056.

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<b><i>Background:</i></b> Renal biopsy plays an important role in the establishment of the diagnosis and the management of patients with lupus nephritis. Immunoglobulin A (IgA) nephropathy rarely has been reported in kidney biopsy of lupus patients. Lupus nephritis and IgA nephropathy can be readily diagnosed on renal biopsy when the classic patterns are present. However, atypical patterns can become a diagnostic challenge. Galactose-deficient IgA1 (Gd-IgA1) is a key element in the pathogenesis of primary IgA nephropathy. Glomerular Gd-IgA1 deposits, detected by immunof

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35

Stephens, Edward B., Chunqiao Tian, Zhuang Li, Opendra Narayan, and Vincent H. Gattone. "Rhesus Macaques Infected with Macrophage-Tropic Simian Immunodeficiency Virus (SIVmacR71/17E) Exhibit Extensive Focal Segmental and Global Glomerulosclerosis." Journal of Virology 72, no. 11 (1998): 8820–32. http://dx.doi.org/10.1128/jvi.72.11.8820-8832.1998.

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ABSTRACT We previously showed that inoculation of rhesus macaques with molecularly cloned lymphocytetropic simian immunodeficiency virus (SIVmac239) results in SIV-associated nephropathy (SIVAN) and that the glomerulosclerotic lesions were associated with the selection of macrophagetropic (M-tropic) variants (V. H. Gattone et al., AIDS Res. Hum. Retroviruses 14:1163–1180, 1998). In the present study, seven rhesus macaques were inoculated with M-tropic SIVmacR71/17E, and the renal pathology was examined at necropsy. All SIVmacR71/17E-infected macaques developed AIDS, and most developed other sy

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36

Errabelli, Praveen, Maulik Lathiya, Neeharik Mareedu, and Loren P. Herrera Hernandez. "Crescentic Glomerulonephritis Possibly Caused by COVID-19 Infection." Journal of Clinical Medicine 14, no. 10 (2025): 3302. https://doi.org/10.3390/jcm14103302.

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Background: The COVID-19 (coronavirus disease 2019) pandemic has presented a serious challenge to nephrologists, since it can lead to severe kidney injury in the form of acute tubular necrosis, with many patients requiring renal replacement therapy. This is predominantly seen in people who develop severe respiratory manifestations like ARDS (acute respiratory distress syndrome) from the viral infection, a cytokine storm or septic shock with unstable hemodynamics. It also presents with various glomerular injuries, mainly collapsing glomerulopathy in high-risk APOL1 (Apolipoprotein L1) genotype

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37

Yasin, Shireena A., Erdal Sag, Katie Arnold, et al. "Tubuloreticular inclusions in juvenile dermatomyositis: a diagnostically useful marker?" Pediatric Rheumatology 12, S1 (2014). http://dx.doi.org/10.1186/1546-0096-12-s1-p91.

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38

Gleeson, Sarah, Jack Beadle, Linda Moran, Ted Fitzgerald, Candice Roufosse, and Michelle Willicombe. "#6395 TUBULORETICULAR INCLUSIONS: A NEW PROGNOSTIC BIOMARKER IN KIDNEY TRANSPLANTATION." Nephrology Dialysis Transplantation 38, Supplement_1 (2023). http://dx.doi.org/10.1093/ndt/gfad063b_6395.

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Abstract Background and Aims Tubuloreticular inclusions (TRIs) seen on electron microscopy (EM) are classically associated with lupus nephritis (LN) and systemic viral infections in native biopsies. Traditionally a marker for enhanced type I interferon expression, little is known about their significance post-transplant. We aimed to look at a large cohort of transplant biopsies showing TRIs to investigate associations and outcomes. Method A retrospective analysis was performed on two prospective databases; an in-centre transplant registry and a histopathology database holding data on all kidne

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39

Tranesh, Ghassan. "Clinicopathologic Correlation of 60 Cases of Lupus Nephritis with Tubuloreticular Inclusions." American Journal of Clinical Pathology 146, suppl_1 (2016). http://dx.doi.org/10.1093/ajcp/aqw161.010.

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40

Alkhalidi, Hisham. "The Role of Electron Microscopy in the Assessment of Dermatomyositis: A Retrospective Pilot Study on Skeletal Muscle Biopsies." March 13, 2013. https://doi.org/10.5281/zenodo.7936.

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Aims: To assess the contribution of electron microscopy in the process of muscle biopsies evaluation for dermatomyositis. Study Design: Retrospective review of muscle biopsy cases. Place and Duration of Study: Pathology Department of King Khalid University Hospital, King Saud University, Riyadh, Saudi Arabia from January 2008 to January 2012. Methodology: Samples from cases suspected to have dermatomyositis were reviewed for light and ultrastructural morphological examination. Tubuloreticular inclusions (TRI) were considered present if these undulating tubules were detected in the endothelial

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41

Koszutski, Tomasz, Łukasz Mielańczyk, Grażyna Kucharska, et al. "Presence of tubuloreticular inclusions in ultrastructural studies of renal biopsies in children with lupus nephropathy – one-center preliminary study." Central European Journal of Immunology, 2023. http://dx.doi.org/10.5114/ceji.2023.125237.

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42

Theodoropoulou, Eleni, Eleni Chelioti, George Christofilidis, Kostas Palamaris, Marina Papadaki, and Harikleia Gakiopoulou. "#2984 SLE associated fibrillary glomerulonephritis vs SLE nephritis with fibrils: can a straightforward distinction always be made?" Nephrology Dialysis Transplantation 39, Supplement_1 (2024). http://dx.doi.org/10.1093/ndt/gfae069.1283.

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Abstract Background and Aims Fibrillary glomerulonephritis (FGN) is an uncommon but serious kidney disease with unclear aetiology, variable prognosis, and a not well-established treatment. FGN has been associated with autoimmune diseases including SLE. Other aetiologies comprise diabetes, malignancies, HCV and chronic infections. Our goal is to discuss the pathologic and clinical differential diagnosis conundrums that a patient with a suspected SLE presented. Method We present the case of a 53-year-old woman evaluated for proteinuria (1 g/24 h), and glomerular haematuria. Renal function was no

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43

Xing, Changhong, Jaya Trivedi, Nicole Bitencourt, Dennis K. Burns, Joan S. Reisch, and Chunyu Cai. "Myxovirus resistance protein A (MxA) expression in myositides: Sarcoplasmic expression is common in both dermatomyositis and lupus myositis." Muscle & Nerve, February 19, 2024. http://dx.doi.org/10.1002/mus.28066.

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AbstractIntroduction/AimsMyxovirus resistance protein A (MxA) is a type I interferon (IFN1) pathway activation marker and MxA sarcoplasmic expression is currently recognized as a highly specific marker for dermatomyositis (DM). However, we have frequently observed endothelial tubuloreticular inclusions (TRI), another surrogate IFN1 activation marker, in a variety of overlap myositides. The aim of this study was to examine MxA expression in those myositides.MethodsWe retrospectively performed MxA immunostaining on a wide range of myositides.ResultsMxA sarcoplasmic expression was present in DM (

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44

Jacob, Anju, Shameer M. Habeeb, Leal Herlitz, et al. "Case Report: CMV-Associated Congenital Nephrotic Syndrome." Frontiers in Pediatrics 8 (November 27, 2020). http://dx.doi.org/10.3389/fped.2020.580178.

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Background: Congenital nephrotic syndrome, historically defined by the onset of large proteinuria during the first 3 months of life, is a rare clinical disorder, generally with poor outcome. It is caused by pathogenic variants in genes associated with this syndrome or by fetal infections disrupting podocyte and/or glomerular basement membrane integrity. Here we describe an infant with congenital CMV infection and nephrotic syndrome that failed to respond to targeted antiviral therapy. Case and literature survey highlight the importance of the “tetrad” of clinical, virologic, histologic, and ge

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45

"COLLAPSING GLOMERULOPATHY WITH TUBULORETICULAR INCLUSION IN HIV NEGATIVE PATIENT." American Journal of Kidney Diseases 63, no. 5 (2014): B88. http://dx.doi.org/10.1053/j.ajkd.2014.01.295.

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46

"10: Tubuloreticular Inclusion Bodies (TRIs) in a Case of Primary Focal Segemental Glomerulosclerosis (FSGS)." American Journal of Kidney Diseases 53, no. 4 (2009): B25. http://dx.doi.org/10.1053/j.ajkd.2009.01.031.

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