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Dissertations / Theses on the topic 'Tumar'

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Published: 18 May 2024

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1

Tumar, Iyad [Verfasser]. "Resource Management of Disruption Tolerant Networks / Iyad Tumar." Aachen : Shaker, 2011. http://d-nb.info/1081884959/34.

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2

Das, Sanjib Kumar. "Characterization of Tumar infiltrating lymphocytes in marine sarcoma and their role in curbing malignancy." Thesis, University of North Bengal, 1997. http://hdl.handle.net/123456789/1006.

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3

Svanberg, Frida, and Timothy Shen. "EN FÖRÄNDRAD IDENTITET : Sexualitetens påverkan i samband med bröstcancer. En litteraturöversikt." Thesis, Högskolan i Skövde, Institutionen för hälsa och lärande, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-10471.

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Bakgrund: I Sverige drabbas 8000 kvinnor av bröstcancer varje år. Dessa kvinnor utsätts för både kroppsliga och emotionella biverkningar och förändringar. Syfte: Syftet med denna litteraturöversikt är att belysa hur kvinnor som drabbas av bröstcancer upplever att deras sexualitet påverkas av sjukdom och behandling. Metod: Metoden var en litteraturstudie. Resultat: Ett huvudtema framkom en förändrad identitet; och fem subteman: kroppsliga förändringar, känna sig okvinnlig, relationens betydelse, kommunikationens betydelse för att komma vidare och sjukvårdens betydelse. Slutsats: Resultatet visa
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4

Butler, Savannah E. "TUMOR-INTRINSIC INFLAMMATORY PATHWAYS ASSOCIATED WITH TUMOR DORMANCY AND RECURRENCE." VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/4753.

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The successful treatment of breast cancer is limited due to a fraction of tumor cells escaping drug-treatment by entering a dormant state, only to relapse years or decades later at distant sites. Host-driven chronic inflammatory cells such as M2 macrophages play an important role in tumorigenesis, but the role of tumor-intrinsic inflammatory signaling involved in tumor dormancy and recurrence is unknown. We sought to determine the role of tumor-intrinsic inflammatory pathways in mouse mammary carcinoma cells (MMC) treated with Adriamycin (ADR), a clinically relevant chemotherapeutic drug. We f
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5

Casiraghi, Nicola. "Quantitative analyses to study tumor clones dynamics and tumor heterogeneity." Doctoral thesis, Università degli studi di Trento, 2017. https://hdl.handle.net/11572/368498.

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Prostate cancer is a highly heterogeneous disease and its manifestations can vary from indolent localized tumor to widespread metastases. This heterogeneity is also observed at the molecular level both inter- and intra-patient. Intra-patient heterogeneity in the clinical setting of men with castration resistant prostate cancer (CRPC) might be informative in terms of treatment decision. Here I present analytical work on two approaches relevant to the characterization of intra-patient heterogeneity and applied to unpublished CRPC patients sequencing data. The first is based on the genome wide in
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Casiraghi, Nicola. "Quantitative analyses to study tumor clones dynamics and tumor heterogeneity." Doctoral thesis, University of Trento, 2017. http://eprints-phd.biblio.unitn.it/2626/2/Disclaimer_Casiraghi.pdf.

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Prostate cancer is a highly heterogeneous disease and its manifestations can vary from indolent localized tumor to widespread metastases. This heterogeneity is also observed at the molecular level both inter- and intra-patient. Intra-patient heterogeneity in the clinical setting of men with castration resistant prostate cancer (CRPC) might be informative in terms of treatment decision. Here I present analytical work on two approaches relevant to the characterization of intra-patient heterogeneity and applied to unpublished CRPC patients sequencing data. The first is based on the genome wide in
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7

Diego, Ángeles Pedro, Ximena Vega, and José Palacios. "Tumor mucoso apendicular." Asociación Colombiana de Cirugía, 2016. http://hdl.handle.net/10757/615473.

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Los tumores mucosos apendiculares tienen baja incidencia y comúnmente se diagnostican en el estudio anatomo-patólogico después de la apendicectomía. Se reporta el caso de una mujer de 41 años de edad, con un cuadro clínico de ocho meses de evolución, caracterizado por dolor abdominal de tipo opresivo, difuso y de gran intensidad en el hemiabdomen inferior, acompañado de náuseas. Después de cinco meses de iniciado este cuadro clínico, se evidenció una masa en la fosa iliaca derecha; el dolor se agudizó e intensificó, y las náuseas continuaron, por lo cual fue remitida al hospital. En los
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8

Petty, Aaron. "Novel MIG-7 expression increases tumor cell invasion and tumor progression." Online access for everyone, 2008. http://www.dissertations.wsu.edu/Thesis/Spring2008/a_petty_040908.pdf.

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9

Anderson, Jeff. "Genetic Analysis Of Specialized Tumor Associated Macrophages And Tumor Associated Fibroblast." The Ohio State University, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=osu1228083946.

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10

Liu, Jian. "POLYMER MODIFICATION OF FULLERENE FOR PHOTODYNAMIC TUMOR THERAPY AND TUMOR IMAGING." 京都大学 (Kyoto University), 2010. http://hdl.handle.net/2433/120886.

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11

Quang, Ly. "Photosensitizing effects of M-Tetrahydroxypheylchlorin on human tumor xenografts : correlation with sensitizer uptake, tumor doubling time and tumor histology /." [S.l.] : [s.n.], 1999. http://www.ub.unibe.ch/content/bibliotheken_sammlungen/sondersammlungen/dissen_bestellformular/index_ger.html.

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12

Kim, Jungho. "Involvement of WT1 tumor suppressor gene in desmoplastic small round cell tumor." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0034/NQ64587.pdf.

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13

Kim, Jungho 1964. "Involvement of WT1 tumor suppressor gene in desmoplastic small round cell tumor." Thesis, McGill University, 1999. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=36621.

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The transformation process involves genetic changes to cellular protooncogenes and tumor suppressor genes. The consequences of these genetic alterations are to confer a growth advantage to the cell. Three genetic mechanisms activate oncogenes or inactivate tumor suppressor genes in human neoplasms: (i) mutations, (ii) gene amplification, and (iii) chromosomal rearrangements. In many human cancers, tumor-specific chromosomal rearrangements are known to create chimeric products with the ability to transform cells. The EWS/WT1 protein is such a fusion product, resulting from a t(11;22) chromosoma
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14

Guarino, Brianna D. "Role of the tumor microenvironment on mechanosensitive TRPV4 channels and tumor angiogenesis." NEOMED Integrated Pharmaceutical Medicine / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ne2mh1619702863516646.

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15

SACHDEVA, MOHIT. "MiR-145 is a tumor suppressor in both tumor progression and metastasis." OpenSIUC, 2010. https://opensiuc.lib.siu.edu/dissertations/206.

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MicroRNAs (miRNAs) are non-coding small RNAs that regulate gene expression at the post-transcriptional level by interacting with the 3'-untranslated region (3'-UTR) of a target gene. Our previous studies indicate that miR-145 is downregulated in breast and colon cancer; moreover, it functions as a tumor suppressor capable of inhibiting tumor cell growth both in vitro and in vivo. In this study, we show that a putative tumor suppressor, miR-145, is regulated through the phosphoinositide-3 kinase (PI-3K)/Akt and p53 pathways. Importantly, p53 transcriptionally induces the expression of miR-145 b
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16

Chaddad, Hassan. "Development of vascularized tumor spheroids mimicking the tumor environment : angiogenesis and hypoxia." Thesis, Strasbourg, 2019. http://www.theses.fr/2019STRAJ001.

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Le microenvironnement tumoral, l'angiogenèse tumorale et l'hypoxie jouent un rôle crucial dans la progression tumorale et le développement de thérapies de nombreux cancers. Les limites de pénétration des médicaments, les phénomènes de résistance aux anti-cancéreux, la vascularisation de la tumeur et l’hypoxie sont tous des paramètres influençant les effets du médicament. La culture cellulaire 3D permet de créer un microenvironnement qui imite l’architecture et la fonction des tissus in vivo. L’expression de gènes et de protéines modifiée par l’environnement 3D est une autre caractéristique qui
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17

ZONARI, ERIKA. "Tumor infiltrating myeloid cells: modulators of tumor microenvironment and novel therapeutic targets." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2012. http://hdl.handle.net/10281/29814.

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Activation of a productive immune response requires transient upregulation of miR155 in the hematopoietic compartment. In order to investigate miR-155 in the context of tumor-associated immune responses, we stably knocked down (KD) miR-155 in the myeloid compartment of MMTV-PyMT mice (PyMT), a mouse model of spontaneous breast carcinogenesis that closely mimics tumor-host interactions seen in humans. Notably, myeloid cell specific miR-155 KD significantly accelerated tumor growth, as reflected by increased tumor mass and more pronounced secondary hematopoietic changes, i.e. leukocytosis and an
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18

Vianna, Karina Costa Maia. "Tumor estomal gastrointestinal (GIST)." reponame:Repositório Institucional da UFPR, 2012. http://hdl.handle.net/1884/26630.

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Resumo: Introdução: Os tumores estromais gastrointestinais (GIST) são neoplasias raras que se originam das células intersticiais de Cajal .A última década foi de grande avanço com o esclarecimento dos mecanismos moleculares, seguido da terapia molecular, que propiciaram um grande aumento na sobrevida. Objetivo: Avaliar a experiência do Hospital de Clínicas de Curitiba no tratamento do GIST localizado e avançado, com análise das características clínicas e anatomo-patológicas e uso do imatinibe. Metodologia: Estudo retrospectivo de 32 pacientes com diagnóstico de GIST por imunohistoquímica, c-Ki
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19

Devine, Raymond David. "Tumor Induced Cardiovascular Dysfunction." The Ohio State University, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=osu1448969937.

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20

Ylikorkala, Antti. "The LKB1 tumor suppressor." Helsinki : University of Helsinki, 2001. http://ethesis.helsinki.fi/julkaisut/laa/haart/vk/ylikorkala/.

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21

Salmenkivi, Kaisa. "Tumor markers in pheochromocytomas." Helsinki : University of Helsinki, 2003. http://ethesis.helsinki.fi/julkaisut/laa/haart/vk/salmenkivi/.

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22

Costa, Alexandra Fontes da. "Análise imunoistoquímica das proteínas maspin, p63 e bcl2 em tumor odontogênico queratocístico, cisto dentígero e ameloblastoma." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/23/23141/tde-13082007-162555/.

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Os cistos e tumores odontogênicos sempre tiveram grande importância dentro da Odontologia, seja pela grande prevalência clínica seja pelo grande acometimento do indivíduo afetado pela lesão. A nova classificação da Organização Mundial de Saúde trouxe a mudança de categoria do queratocisto, que recebe agora a nômina de tumor odontogênico queratocístico, e que figura não mais na categoria de cisto odontogênico de desenvolvimento, mas sim de tumor odontogênico. Certa precipitação nessa mudança levou alguns autores a sugerirem a necessidade de estudos que esclareçam as características clínicas e h
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23

Bassani, Nicklas. "New targets in tumor angiogenesis to block tumor re-growth and therapeutic resistance." Doctoral thesis, Universitat de Barcelona, 2017. http://hdl.handle.net/10803/462953.

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Antiangiogenic drugs are used clinically for treatment of different types of cancers and in the case of renal cell carcinoma (RCC) are now standard first-line treatments (Rini, 2009). Nevertheless, these agents mainly serve to stabilize the disease but are not able to eliminate all tumor cells and resistance eventually develops concomitant with progression (Kerber and Folkman, 2002). Using different orthoxenograft mouse models of RCC we confirmed that inhibitors of the VEGF pathway have a therapeutic window of effectiveness but unfortunately adaptation and tumor relapse always occur. Several
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24

Lammerts, Ellen. "Tumor stroma in anaplastic thyroid carcinoma interstitial collagen and tumor interstitial fluid pressure /." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2001. http://publications.uu.se/theses/91-554-5198-5/.

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25

To, Kit-yan, and 杜潔欣. "Beta-catenin signaling in the interactions of tumor cells and the tumor microenvironment." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2015. http://hdl.handle.net/10722/211113.

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26

Payne, Kyle K. "Immunotherapy of Cancer: Reprogramming Tumor/Immune Cellular Crosstalk to Improve Anti-Tumor Efficacy." VCU Scholars Compass, 2015. http://scholarscompass.vcu.edu/etd/3939.

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Immunotherapy of cancer has been shown to be promising in prolonging patient survival. However, complete elimination of cancer and life-long relapse-free survival remain to be major challenge for anti-cancer therapeutics. We have previously reported that ex vivo reprogramming of tumor-sensitized immune cells by bryostatin 1/ionomycin (B/I) and the gamma-chain (γ-c) cytokines IL-2, IL-7, and IL-15 resulted in the generation of memory T cells as well as CD25+ NKT cells and CD25+ NK cells. Adoptive cellular therapy (ACT) utilizing these reprogrammed immune cells protected FVBN202 mice from tumor
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27

Pearce, Janina V. "The Role of Tumor and Tumor Microenvironment on Breast Cancer-Associated Adipocyte Plasticity." VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/5933.

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Cancer-associated cachexia is a condition defined by a sustained net-negative energy imbalance. Although the different types of adipose tissue – white, beige, and brown – have been implicated in contributing to cancer-associated cachexia, the mechanisms of these maladaptive changes and their impact on whole-body energy expenditure have not been fully elucidated. Using breast cancer as our model, we demonstrate white adipose tissue browning in murine and human breast cancer; furthermore, we demonstrate that this effect is extremely localized and takes place early in tumor progression. We utiliz
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28

D'ORIA, CLAUDIA. "ORGANOID MODELS TO STUDY THE CROSSTALK BETWEEN TUMOR CELLS AND TUMOR INFILITRATING LYMPHOCYTES." Doctoral thesis, Università degli Studi di Milano, 2020. http://hdl.handle.net/2434/700588.

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Recent advances in 3D culture technology allow embryonic and adult mammalian stem cells to generate organoids in vitro, which reflect key structural and functional properties of organs they originate (Clevers H., Cell, 2016). For this reason, they represent a powerful tool to study human physiological and pathological processes, in particular to investigate complex processes like tumorigenesis and tumor growth, resembling the in vivo mechanisms. In particular, in tumor contest neoplastic cells activate several strategies to escape from immunesurveillance, such as the recruitment of immune cell
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29

Carter, Ashley. "AN EXAMINATION OF OBESITY IN PEDIATRIC BRAIN TUMOR SURVIVORS: FOOD FOR THOUGHT." Thesis, The University of Arizona, 2015. http://hdl.handle.net/10150/528118.

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A Thesis submitted to The University of Arizona College of Medicine - Phoenix in partial fulfillment of the requirements for the Degree of Doctor of Medicine.<br>Background: Great strides have been made in childhood cancer treatment efficacy over the past two decades leading to improved survival rates, and now attention is being directed toward identifying and understanding complications that affect many of these patients as they reach adulthood. Obesity is a well‐recognized late effect that has many potential long‐term consequences some of which include cardiovascular disease, type II diabete
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30

Kasnitz, Nadine [Verfasser], and Manfred [Akademischer Betreuer] Rohde. "Tumor-associated neutrophils during Pseudomonas aeruginosa-mediated tumor therapy / Nadine Kasnitz ; Betreuer: Manfred Rohde." Braunschweig : Technische Universität Braunschweig, 2016. http://d-nb.info/1175819042/34.

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31

Szot, Christopher Sang. "A three-dimensional in vitro tumor model representative of the in vivo tumor microenvironment." Diss., Virginia Tech, 2013. http://hdl.handle.net/10919/49597.

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The inability to accurately reproduce the complexities of the in vivo tumor microenvironment with reductionist-based two-dimensional in vitro cell culture models has been a notable deterrent in identifying therapeutic agents that reliably translate to in vivo animal and human clinical trials. In an effort to address this, a growing number of three-dimensional (3D) in vitro tumor models capable of mimicking specific tumorigenic processes have emerged within the last decade. This concept stems from the understanding that cells cultured within 3D in vitro matrices have the ability to acquire phen
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32

Senkowski, Wojciech. "High-throughput screening using multicellular tumor spheroids to reveal and exploit tumor-specific vulnerabilities." Doctoral thesis, Uppsala universitet, Cancerfarmakologi och beräkningsmedicin, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-320598.

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High-throughput drug screening (HTS) in live cells is often a vital part of the preclinical anticancer drug discovery process. So far, two-dimensional (2D) monolayer cell cultures have been the most prevalent model in HTS endeavors. However, 2D cell cultures often fail to recapitulate the complex microenvironments of in vivo tumors. Monolayer cultures are highly proliferative and generally do not contain quiescent cells, thought to be one of the main reasons for the anticancer therapy failure in clinic. Thus, there is a need for in vitro cellular models that would increase predictive value of
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33

Zhang, Xiaomeng. "MRI OF TUMOR pH AND PERFUSION." Diss., The University of Arizona, 2010. http://hdl.handle.net/10150/195293.

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In the early 1920s, Otto Warburg demonstrated that tumor cells have a capacity to convert glucose and other substrates into lactic acid instead of CO2 and water, even under aerobic conditions. Consequently, Warburg assumed that the intracellular pH (pHi) of tumor was acidic. However, later studies have shown that maintenance of pHi within a pH range of 7.0-7.2 is necessary for normal cellular proliferation and that the extracellular pH (pHe) is partially acidic in solid tumors. A low pHe may be an important factor inducing invasive behavior in tumor cells. Research into causes and consequence
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34

Vitaliti, Alessandra. "Inhibition of tumor induced angiogenesis /." [S.l.] : [s.n.], 1999. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=13409.

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35

Barendsz-Janson, A. F. "Predictive models of tumor angiogenesis." [Maastricht : Maastricht : Universiteit Maastricht] ; University Library, Maastricht University [Host], 1998. http://arno.unimaas.nl/show.cgi?fid=8524.

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36

Hellebrekers, Debby Maria Elisabeth Ida. "Epigenetic regulation of tumor angiogenesis." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Universiteit Maastricht [host], 2006. http://arno.unimaas.nl/show.cgi?fid=5612.

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37

Sheikholvaezin, Ali. "Recombinant antibodies and tumor targeting." Doctoral thesis, Umeå : Univ, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-875.

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38

Fujioka, Kaoru. "Centrosome aberrations and tumor development /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-627-8/.

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39

Weens, William. "Mathematical modeling of liver tumor." Phd thesis, Université Pierre et Marie Curie - Paris VI, 2012. http://tel.archives-ouvertes.fr/tel-00779177.

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Comme démontre récemment pour la régénération du foie après un dommage cause par intoxication, l'organisation et les processus de croissance peuvent être systématiquement analyses par un protocole d'expériences, d'analyse d'images et de modélisation [43]. Les auteurs de [43] ont quantitativement caractérise l'architecture des lobules du foie, l'unité fonctionnelle fondamentale qui constitue le foie, et en ont conçu un modèle mathématique capable de prévoir un mécanisme jusqu'alors inconnu de division ordonnée des cellules. La prédiction du modèle fut ensuite validée expérimentalement. Dans ce
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40

Mercier, Laurence. "Ultrasound-guided brain tumor resection." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=107629.

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Malignant gliomas are the most common type of primary brain tumors in adults. Contrarily to brain metastases that have clear borders, malignant gliomas are poorly circumscribed lesions because they diffusely infiltrate the brain parenchyma. When possible, standard treatment of gliomas includes surgical resection, though surgeons unintentionally leave behind some of the tumor more than 50% of the time. Two factors are mainly responsible for this situation. First, most current neuronavigation systems are based on preoperative images; these systems become less accurate as the surgery progresses
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41

Weiß, Jakob. "Regulation Tumor-infiltrierender dendritischer Zellen." Diss., Ludwig-Maximilians-Universität München, 2014. http://nbn-resolving.de/urn:nbn:de:bvb:19-178105.

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42

Chowdury, Simon. "Retroviral targeting to tumor antigens :." Thesis, University College London (University of London), 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.411165.

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43

Mathis, Robert Austin. "Intra-tumor heterogeneity and evolution." Thesis, Massachusetts Institute of Technology, 2017. http://hdl.handle.net/1721.1/113432.

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Thesis: Ph. D., Massachusetts Institute of Technology, Department of Biology, 2017.<br>This electronic version was submitted by the student author. The certified thesis is available in the Institute Archives and Special Collections.<br>Cataloged from student-submitted PDF version of thesis.<br>Includes bibliographical references.<br>Although the treatment of cancer is a major focus of biomedical research, many cancers are extremely hard to treat. Tumors likely resist treatment because each tumor is heterogeneous, and can evolve. Although tumor evolution has long been appreciated, it remains i
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44

Graff, Christilyn Paula. "Antibody engineering for tumor immunotherapy." Thesis, Massachusetts Institute of Technology, 2002. http://hdl.handle.net/1721.1/29279.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Dept. of Chemical Engineering, 2002.<br>Vita.<br>Includes bibliographical references (leaves 130-140).<br>Antibodies have been used as cancer therapeutics for several decades. One area in which this therapy may be improved is the retention time of antibody in the tumor relative to normal tissue. In this Thesis, we have attempted to elucidate the mechanisms that are most influential to improving antibodies as cancer therapeutics. Carcinoembryonic antigen (CEA) has long been identified as a tumor-associated antigen. CEA is also quite stable
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45

Valladão, Maria Luiza de Castro Ramos 1977. "Remodelamento do tumor venéreo transmissível." [s.n.], 2007. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308603.

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Orientador: Konradin Metze<br>Tese (doutorado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-20T00:01:11Z (GMT). No. of bitstreams: 1 Valladao_MariaLuizadeCastroRamos_D.pdf: 10270983 bytes, checksum: 4313e15091d949287ba12830e233437d (MD5) Previous issue date: 2012<br>Resumo: O tumor venéreo transmissível canino (TVTC) é transmitido por meio da transplantação de células neoplásicas. O sulfato de vincristina é o agente quimioterápico mais utilizado no tratamento do TVTC. O objetivo deste estudo foi comparar as alterações morfológicas
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46

Dennie, Joëlle 1970. "NMR imaging of tumor angiogenesis." Thesis, Massachusetts Institute of Technology, 1997. http://hdl.handle.net/1721.1/43595.

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Thesis (S.M.)--Massachusetts Institute of Technology, Dept. of Nuclear Engineering, 1997.<br>Includes bibliographical references (leaves 66-69).<br>Cancer remains a major medical problem accounting for over 500,000 deaths in the US annually. A common feature of most human tumors is their ability to induce the proliferation of new blood vessels, i.e. angiogenesis. Considerable evidence now exists which demonstrates that these tumor vessels are associated with a distinct range of morphological and physiological properties which are not present in normal tissue vasculature. Several studies now do
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47

Raman, Sundaresan. "Phenotypical Analysis of Tumor Microenvironment." The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1354712085.

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48

Tuijnder, Marcel. "Biological models of tumor reversion." Paris 7, 2005. http://www.theses.fr/2005PA077085.

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49

Cahlin, Christian. "Cyclooxygenase activity and tumor progression /." Göteborg : Departments of Surgery and Transplantation, Institute of Clinical Sciences at Sahlgrenska Academy, University of Gothenburg, 2008. http://hdl.handle.net/2077/18198.

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50

Åkerman, Maria E. "Targeting and inhibiting tumor angiogenesis /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2005. http://wwwlib.umi.com/cr/ucsd/fullcit?p3166405.

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