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Books on the topic 'Tumor Formation'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Yosef, Shiloh, and SpringerLink (Online service), eds. The DNA Damage Response: Implications on Cancer Formation and Treatment. Dordrecht: Springer Netherlands, 2009.

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2

Gogichadze, G. K. Karyogamic theory of cancer cell formation from the view of the XXI century. Hauppauge, N.Y: Nova Science Publishers, 2009.

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3

T, Gogichadze, ed. Karyogamic theory of cancer cell formation from the view of the XXI century. New York: Nova Biomedical Books, 2010.

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4

Vásquez Rodríguez, Fernando, ed. La tutoría de la investigación. Bogotá. Colombia: Universidad de La Salle. Ediciones Unisalle, 2019. http://dx.doi.org/10.19052/9789585486331.

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Esta obra es de gran utilidad para los noveles tutores, quienes, en la mayoría de los casos, comienzan su labor apenas con los referentes de un profesor que los acompañó en su trabajo de grado, pero sin contar con una fuente de consulta en la que logren profundizar o capacitarse en este nuevo rol. A lo largo de las páginas de este texto se podrán apreciar y distinguir las características, los alcances, las dificultades y el itinerario formativo de un tutor; una labor académica que empieza en las vicisitudes de la conformación de un equipo de investigación, pasando por las extensas jornadas de diálogo, revisión y señalamiento de compromisos, hasta llegar a la supervisión formal y de contenido del trabajo final.

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5

Reader, Jocelyn, Sarah Lynam, Amy Harper, Gautam Rao, Maya Matheny, and Dana M. Roque. Ovarian Tumor Microenvironment and Innate Immune Recognition. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0004.

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Ovarian adenocarcinoma is typified by detection at late stages with dissemination of cancer cells into the peritoneal cavity and frequent acquisition of chemoresistance. A number of studies show the importance of the tumor microenvironment and innate immune recognition in tumor progression. Ovarian cancer cells can regulate the composition of their stroma to promote the formation of ascitic fluid rich in cytokines and bioactive lipids such as PGE2, and to stimulate the differentiation of stromal cells into a pro-tumoral phenotype. In response, cancer-associated fibroblasts, cancer-associated mesenchymal stem cells, tumor-associated macrophages, and other peritoneal cells can act through direct and indirect mechanisms to regulate tumor growth, chemoresistance via alteration of class III β‎ tubulin, angiogenesis and dissemination. This chapter deciphers the current knowledge about the role of stromal cells, associated secreted factors, and the immune system on tumor progression. This suggests that targeting the microenvironment holds great potential to improve the prognosis of patients with ovarian adenocarcinoma.

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6

Hatef, Jeffrey, and Russell R. Lonser. Hemangioblastoma. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0007.

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Hemangioblastomas are benign central nervous system tumors that are found primarily (99%) in the cerebellum, brainstem, and spinal cord. They can occur sporadically (67% of cases) or in the context of the familial neoplasia syndrome, von Hippel-Lindau disease (VHL; 33%). These lesions often remain quiescent or grow in a saltatory pattern. When these tumors cause signs or symptoms, the signs or symptoms are often associated with peritumoral cyst formation. Whether the tumor occurs sporadically or in the context of VHL, complete resection is the treatment of choice when necessary. This chapter describes the clinical, imaging, and treatment features of this neoplasm.

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7

Malyshev, Igor. Immunity, Tumors and Aging : The Role of HSP70: The Role of HSP70. Springer, 2013.

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8

Jordan, Nerissa. Non-metastatic neurological manifestations of malignancy. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0238.

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Neurological complications of systemic malignancy are frequent. They may reflect direct local effects of the tumour; CNS infection; side effects of chemotherapy or radiotherapy; nutritional or metabolic derangements; or a paraneoplastic syndrome. The paraneoplastic neurological syndromes are a group of disorders associated with a malignancy outside the nervous system. The pathophysiology is immune-mediated, with the tumour’s expression of neuronal proteins invoking antibody formation, which in turn results in neurological symptoms. This chapter will mainly focus on these syndromes.

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9

(Editor), James H. Finke, and Ronald M. Bukowski (Editor), eds. Cancer Immunotherapy at the Crossroads: How Tumors Evade Immunity and What Can Be Done (Current Clinical Oncology). Humana Press, 2003.

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10

Zhang, Quan. Temperature modulated aflatoxin B1 hepatic disposition, and formation and persistence of DNA adducts in rainbow trout. 1992.

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11

1944-, Finke James H., and Bukowski Ronald M, eds. Cancer immunotherapy at the crossroads: How tumors evade immunity and what can be done. Totowa, N.J: Humana Press, 2004.

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12

1944-, Finke James H., and Bukowski Ronald M, eds. Cancer immunotherapy at the crossroads: How tumors evade immunity and what can be done. Totowa, N.J: Humana Press, 2004.

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13

Blanshard, Alastair J. L. Mahaffy and Wilde. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198789260.003.0002.

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This chapter explores the relationship between Wilde and his Trinity College Classics tutor, John Pentland Mahaffy. This complex relationship played a vital part in the formation of Wilde’s distinctive version of Hellenism. It was a troubled relationship. Wilde and Mahaffy disagreed about politics, sexuality, religion, the course of Greek history, and the role and function of classical education. Examining these points of disagreement brings into clear relief a number of important contours in Wilde’s attitudes to the Greeks. Mahaffy, through his life and writing, provoked Wilde into defining precisely what the Greeks meant to Wilde, how they might be studied, and their value to contemporary society.

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14

Nockles, Peter. Pre-Tractarian Oxford. Edited by Stewart J. Brown, Peter Nockles, and James Pereiro. Oxford University Press, 2017. http://dx.doi.org/10.1093/oxfordhb/9780199580187.013.6.

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The roots of and context for the genesis of the Oxford Movement can be traced to the intellectual and spiritual formation of its leaders, protagonists, and disciples provided within the milieu of the University of Oxford and notably Oriel College. The influence of the Oriel Noetics was crucial but that of John Keble and Hurrell Froude on the impressionable John Henry Newman and others was no less significant. A parting of the ways between those who would become Tractarians and the Noetics, first evident in the Peel election of 1829, was reflected in Oriel’s tutor dispute of 1830, giving a foretaste of the deepening divisions of the 1830s discussed in a later chapter.

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15

Panjwani, Imranali. Challenges in Teaching Islamic Studies in Western Universities. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190677565.003.0009.

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When teaching Islam to undergraduates, the question of pedagogy is crucial. Modules must be designed to capture the breadth of the religion, including ethics, spirituality, worldview, role of holy figures, history, scientific disciplines, cultural formations, and contemporary developments. Although Western universities should be commended for introducing Islamic Studies to undergraduates, they streamline Islam to the extent that it is reduced to Islamic history. This means Islam’s intellectual tradition is seen as a contribution of the past rather than a living contribution for current human problems. In this chapter, I will share the challenges I faced as a tutor in Islamic Studies at King’s College London within the context of two pedagogical issues: (1) how Islamic Studies modules could be designed more effectively and (2) how effective learning environments can be created for undergraduate students of Islamic Studies.

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16

Sieper, Joachim. Ankylosing spondylitis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0113.

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Ankylosing spondylitis (AS) is a chronic inflammatory disease predominantly of the sacroiliac joint (SIJ) and the spine. It starts normally in the second decade of life and has a slight male predominance. The prevalence is between 0.2 and 0.8% and is strongly dependent on the prevalence of HLA B27 in a given population. For the diagnosis of AS, the presence of radiographic sacroiliitis is mandatory. However, radiographs do not detect active inflammation but only structural bony damage. Most recently new classification criteria for axial spondyloarthritis (SpA) have been developed by the Assessement of Spondylo-Arthritis international Society (ASAS) which cover AS but also the earlier form of non-radiographic axial SpA. MRI has become an important new tool for the detection of subchondral bone marrow inflammation in SIJ and spine and has become increasingly important for an early diagnosis. HLA B27 plays a central role in the pathogenesis but its exact interaction with the immune system has not yet been clarified. Besides pain and stiffness in the axial skeleton patients suffer also from periods of peripheral arthritis, enthesitis, and uveitis. New bone formation as a reaction to inflammation and subsequent ankylosis of the spine determine long-term outcome in a subgroup of patients. Currently only non-steroidal anti-inflammatory drugs (NSAIDs) and tumour necrosis factor (TNF) blockers have been proven to be effective in the medical treatment of axial SpA, and international ASAS recommendations for the structured management of axial SpA have been published based on these two types of drugs. Conventional disease-modifying anti-rheumatic drugs (DMARDs) such as methotrexate are not effective.

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17

Sieper, Joachim. Axial spondyloarthropathies. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0113_update_003.

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Axial spondyloarthritis (axSpA) is a chronic inflammatory disease predominantly of the sacroiliac joint (SIJ) and the spine. It starts normally in the second decade of life and has a slight male predominance. The prevalence is between 0.2% and 0.8% and is strongly dependent on the prevalence of HLA-B27 in a given population. AxSpA can be split in patients with radiographic axSpA (also termed ankylosing spondylitis (AS)) and in patients with non-radiographic axSpA (nr-axSpA). For the diagnosis of AS, the presence of radiographic sacroiliitis is mandatory. However, radiographs do not detect active inflammation but only structural bony damage. Most recently new classification criteria for axSpA have been developed by the Assessment of Spondylo-Arthritis International Society (ASAS) which cover AS but also the earlier form of nr-axSpA. MRI has become an important new tool for the detection of subchondral bone marrow inflammation in SIJ and spine and has become increasingly important for an early diagnosis. HLA-B27 plays a central role in the pathogenesis but its exact interaction with the immune system has not yet been clarified. Besides pain and stiffness in the axial skeleton patients suffer also from periods of peripheral arthritis, enthesitis, and uveitis. New bone formation as a reaction to inflammation and subsequent ankylosis of the spine determine long-term outcome in a subgroup of patients. Currently only non-steroidal anti-inflammatory drugs (NSAIDs) and tumour necrosis factor (TNF) blockers have been proven to be effective in the medical treatment of axial SpA, and international ASAS recommendations for the structured management of axial SpA have been published based on these two types of drugs. Conventional disease-modifying anti-rheumatic drugs (DMARDs) such as methotrexate are not effective.

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18

Training the Reading Team: A Guide for Supervisors of a Volunteer Tutoring Program. International Reading Association, 1999.

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