Relevant bibliographies by topics / Tumor initiating cells (CICs)

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers
  5. Reports

Academic literature on the topic 'Tumor initiating cells (CICs)'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Tumor initiating cells (CICs)"

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Ravindran, Shilpa, Saad Rasool, and Cristina Maccalli. "The Cross Talk between Cancer Stem Cells/Cancer Initiating Cells and Tumor Microenvironment: The Missing Piece of the Puzzle for the Efficient Targeting of these Cells with Immunotherapy." Cancer Microenvironment 12, no. 2-3 (2019): 133–48. http://dx.doi.org/10.1007/s12307-019-00233-1.

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AbstractCancer Stem Cells/Cancer Initiating Cells (CSCs/CICs) is a rare sub-population within a tumor that is responsible for tumor formation, progression and resistance to therapies. The interaction between CSCs/CICs and tumor microenvironment (TME) can sustain “stemness” properties and promote their survival and plasticity. This cross-talk is also pivotal in regulating and modulating CSC/CIC properties. This review will provide an overview of the mechanisms underlying the mutual interaction between CSCs/CICs and TME. Particular focus will be dedicated to the immunological profile of CSCs/CIC
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Tomei, Sara, Ola Ibnaof, Shilpa Ravindran, Soldano Ferrone, and Cristina Maccalli. "Cancer Stem Cells Are Possible Key Players in Regulating Anti-Tumor Immune Responses: The Role of Immunomodulating Molecules and MicroRNAs." Cancers 13, no. 7 (2021): 1674. http://dx.doi.org/10.3390/cancers13071674.

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Cancer cells endowed with stemness properties and representing a rare population of cells within malignant lesions have been isolated from tumors with different histological origins. These cells, denominated as cancer stem cells (CSCs) or cancer initiating cells (CICs), are responsible for tumor initiation, progression and resistance to therapies, including immunotherapy. The dynamic crosstalk of CSCs/CICs with the tumor microenvironment orchestrates their fate and plasticity as well as their immunogenicity. CSCs/CICs, as observed in multiple studies, display either the aberrant expression of
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Lotti, Fiorenza, Awad M. Jarrar, Rish K. Pai, et al. "Chemotherapy activates cancer-associated fibroblasts to maintain colorectal cancer-initiating cells by IL-17A." Journal of Experimental Medicine 210, no. 13 (2013): 2851–72. http://dx.doi.org/10.1084/jem.20131195.

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Many solid cancers display cellular hierarchies with self-renewing, tumorigenic stemlike cells, or cancer-initiating cells (CICs) at the apex. Whereas CICs often exhibit relative resistance to conventional cancer therapies, they also receive critical maintenance cues from supportive stromal elements that also respond to cytotoxic therapies. To interrogate the interplay between chemotherapy and CICs, we investigated cellular heterogeneity in human colorectal cancers. Colorectal CICs were resistant to conventional chemotherapy in cell-autonomous assays, but CIC chemoresistance was also increased
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Ghatak, Shibnath, Vincent C. Hascall, Roger R. Markwald, and Suniti Misra. "FOLFOX Therapy Induces Feedback Upregulation of CD44v6 through YB-1 to Maintain Stemness in Colon Initiating Cells." International Journal of Molecular Sciences 22, no. 2 (2021): 753. http://dx.doi.org/10.3390/ijms22020753.

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Cancer initiating cells (CICs) drive tumor formation and drug-resistance, but how they develop drug-resistance characteristics is not well understood. In this study, we demonstrate that chemotherapeutic agent FOLFOX, commonly used for drug-resistant/metastatic colorectal cancer (CRC) treatment, induces overexpression of CD44v6, MDR1, and oncogenic transcription/translation factor Y-box-binding protein-1 (YB-1). Our study revealed that CD44v6, a receptor for hyaluronan, increased the YB-1 expression through PGE2/EP1-mTOR pathway. Deleting CD44v6, and YB-1 by the CRISPR/Cas9 system attenuates th
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Holgado, Esther, Maria Cortes Semperes, Olga Pernia, et al. "Insulin analogues to stimulate cell growth in human cancer initiating cells (CICs)." Journal of Clinical Oncology 31, no. 15_suppl (2013): 1575. http://dx.doi.org/10.1200/jco.2013.31.15_suppl.1575.

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1575 Background: Conflicting epidemiologic studies have suggested insulin and their analogues currently used in the management of type 2 diabetes to be involved in tumor progression and development. Surprisingly, time of exposure to insulin analogs reported in those retrospective studies is too short for be a carcinogen. We hypothesized that differential affinity that insulin analogs display for insulin receptor and IGF-1R at CICs may account for this possibility. [AspB10]insulin (X10) is a long-acting insulin analog that displays greater affinity for IGF1R than either the long-acting insulin
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Kato, S., M. F. Liberona, J. Cerda-Infante, et al. "Simvastatin interferes with cancer ‘stem-cell’ plasticity reducing metastasis in ovarian cancer." Endocrine-Related Cancer 25, no. 10 (2018): 821–36. http://dx.doi.org/10.1530/erc-18-0132.

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Cell plasticity of ‘stem-like’ cancer-initiating cells (CICs) is a hallmark of cancer, allowing metastasis and cancer progression. Here, we studied whether simvastatin, a lipophilic statin, could impair the metastatic potential of CICs in high-grade serous ovarian cancer (HGS-ovC), the most lethal among the gynecologic malignancies. qPCR, immunoblotting and immunohistochemistry were used to assess simvastatin effects on proteins involved in stemness and epithelial-mesenchymal cell plasticity (EMT). Its effects on tumor growth and metastasis were evaluated using different models (e.g., spheroid
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Wang, Dongqing, Haitao Zhu, Yanfang Liu, et al. "The Low Chamber Pancreatic Cancer Cells Had Stem-Like Characteristics in Modified Transwell System: Is It a Novel Method to Identify and Enrich Cancer Stem-Like Cells?" BioMed Research International 2014 (2014): 1–10. http://dx.doi.org/10.1155/2014/760303.

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Cancer stem cells (CSCs) or cancer-initiating cells (CICs) play an important role in tumor initiation, progression, metastasis, chemoresistance, and recurrence. It is important to construct an effective method to identify and isolate CSCs for biotherapy of cancer. During the past years, many researchers had paid more attention to it; however, this method was still on seeking. Therefore, compared to the former methods that were used to isolate the cancer stem cell, in the present study, we tried to use modified transwell system to isolate and enrich CSCs from human pancreatic cancer cell lines
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Li, Lingyu, Jiuwei Cui, Chang Wang, et al. "Adoptive transfer of NK cells in combination with chemotherapy to improve outcomes of patients with locally advanced colon carcinoma." Journal of Clinical Oncology 35, no. 15_suppl (2017): e15038-e15038. http://dx.doi.org/10.1200/jco.2017.35.15_suppl.e15038.

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e15038 Background: The prognosis of advanced colon cancer (CC) patients remains disappointing, partly due to their greater proportion of CC-initiating cells (CICs), which is responsible for cancer drug-resistance and immune escape. Immunotherapies by harnessing the immune system to eliminate tumors have attracted broad attention. This study was to detect whether chemotherapy could enhance cytotoxicity of natural killer (NK) cells to CC cells (CCs), especially for CICs in vitro, and further evaluate the efficacy and safety of NK-cell therapy combined with chemotherapy in patients with local adv
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Ishigaki, Tomohiro, Seiichiro Kobayashi, Nobuhiro Ohno, et al. "Comprehensive Analysis of Surface Antigens on Adult T-Cell Leukemia/Lymphoma (ATL) Cells and Search for ATL-Initiating Cell Markers." Blood 124, no. 21 (2014): 1674. http://dx.doi.org/10.1182/blood.v124.21.1674.1674.

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Abstract Adult T-cell leukemia/lymphoma (ATL) is highly aggressive malignancy caused by human T-cell leukemia virus type 1 (HTLV1). Intensive combination chemotherapy can initially reduce ATL cells, but relapses are common. Today many kinds of tumors are considered to be organized in a hierarchy of heterogeneous cell populations, with only a small proportion of cancer initiating cells (CICs) capable of sustaining tumor formation and growth. Even if most of tumor cells are killed, remaining chemo-resistant CICs can be causes of relapses. However, CICs of ATL have not been identified yet. Our fi
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Chang, Ching-Wen, Chien-Chih Chen, Meng-Ju Wu, et al. "Active Component ofAntrodia cinnamomeaMycelia Targeting Head and Neck Cancer Initiating Cells through Exaggerated Autophagic Cell Death." Evidence-Based Complementary and Alternative Medicine 2013 (2013): 1–15. http://dx.doi.org/10.1155/2013/946451.

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Head and neck squamous cell carcinoma (HNSCC) is a highly lethal cancer. Previously, we identify head and neck cancer initiating cells (HN-CICs), which are highly tumorigenic and resistant to conventional therapy. Therefore, development of drug candidates that effectively target HN-CICs would benefit future head and neck cancer therapy. In this study, we first successfully screened for an active component, named YMGKI-1, from natural products ofAntrodia cinnamomeaMycelia (ACM), which can target the stemness properties of HNSCC. Treatment of YMGKI-1 significantly downregulated the aldehyde dehy
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Dissertations / Theses on the topic "Tumor initiating cells (CICs)"

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Scalabrini, Luiza Coimbra. "O papel da quinase Aurora A na biologia das células iniciadoras de turmor pulmonares com mutação em KRAS." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-11042017-082202/.

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Mutações ativadoras no gene KRAS são prevalentes em cancer de pulmão e a as vias de sinalização de RAS estão aumentadas em células iniciadoras de tumor (CITs), que são definidas como células autorrenováveis capazes de iniciar a formação tumoral, sustentar o crescimento tumoral e promover a disseminação tumoral. Entretanto, terapias direcionadas a RAS não foram efetivas até hoje e a identificação de alvos de KRAS que contribuam para o fenótipo oncogênico é necessária. Como a quinase Aurora A (AURKA) já foi implicada, tanto na oncogênese induzida por KRAS, quanto em promover a função das CITs, n
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Schlitter, Anne-Marie [Verfasser]. "CD57high neuroblastoma cells have characteristics of tumor-initiating cells / Anne-Marie Schlitter." Ulm : Universität Ulm. Medizinische Fakultät, 2013. http://d-nb.info/1030045453/34.

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Chau, Wing-ka, and 周穎嘉. "Characterization of ovarian tumor-initiating cells and mechanisms of chemoresistance." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/197834.

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Chemoresistance remains a major clinical obstacle to effective management of ovarian cancer. Cancer stem cells (or tumor-initiating cells, TICs) have been discovered recently, and have played a pivotal role in changing the view of cancer development; however, the molecular mechanisms by which these cells escape conventional therapies remain elusive. In this study, TICs were isolated from ovarian cancer cells as tumor spheres with specific stem properties under TIC-selective conditions. Unlike non-TICs, TICs strongly express stem cell factor (SCF) and c-Kit. Blocking SCF-c-Kit by SCF neutralizi
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Ishibashi, Tomoko [Verfasser], and Martin [Akademischer Betreuer] Jechlinger. "Identification and Characterization of Tumor Initiating Cells in Various Mouse Mammary Tumor Models / Tomoko Ishibashi ; Betreuer: Martin Jechlinger." Heidelberg : Universitätsbibliothek Heidelberg, 2016. http://d-nb.info/1180608070/34.

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Maekawa, Hisatsugu. "A Chemosensitivity Study of Colorectal Cancer Using Xenografts of Patient-Derived Tumor Initiating Cells." Kyoto University, 2018. http://hdl.handle.net/2433/235985.

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Anderson, Angela S. "Characterization of Metabolic Differences in Benign, Slow Developing and Tumor Initiating Ovarian Cancers." Diss., Virginia Tech, 2013. http://hdl.handle.net/10919/50812.

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Ovarian cancer is known as the "silent killer," due to its late diagnosis and frequent recurrence after initial treatment.  Finding a new way to diagnose and treat ovarian cancer in conjunction with current therapies is paramount.  By capitalizing on metabolic changes that occur during cancer progression, interventions can be developed.  The Nobel laureate Otto Warburg is credited with discovering an altered metabolic state within cancer cells known as the Warburg effect.  In the Warburg effect, cancer cells participate in an increased rate of aerobic glycolysis with an excess secretion of lac
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Rieger, Megan Elizabeth. "Transcription Cofactor LBH is a Direct Target of the Oncogenic WNT Pathway with an Important Role in Breast Cancer." Scholarly Repository, 2010. http://scholarlyrepository.miami.edu/oa_dissertations/659.

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Limb-Bud and Heart (LBH) is a novel key transcriptional regulator of vertebrate development. However, the molecular mechanisms upstream of LBH and its role in adult development are unknown. Here we show that in epithelial development, LBH expression is tightly controlled by Wnt signaling. LBH is transcriptionally induced by the canonical Wnt pathway, as evident by the presence of functional TCF/LEF binding sites in the LBH locus and rapid beta-catenin-dependent upregulation of endogenous LBH by Wnt3a. In contrast, LBH induction by Wnt/beta-catenin signaling is inhibited by Wnt7a, which in
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Noudomi, Seishiro. "CD146 is a novel marker for highly tumorigenic cells and a potential therapeutic target in malignant rhabdoid tumor." Kyoto University, 2016. http://hdl.handle.net/2433/217140.

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Shafiee, Hadi. "Marker-Free Isolation and Enrichment of Rare Cell Types Including Tumor Initiating Cells through Contactless Dielectrophoresis." Diss., Virginia Tech, 2010. http://hdl.handle.net/10919/77262.

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Microfluidics has found numerous applications ranging from the life sciences industries for pharmaceuticals and biomedicine (drug design, delivery and detection, diagnostic devices) to industrial applications of combinational synthesis (such as rapid analysis and high throughput screening). Among all these, one of the intriguing exploitation of microfluidics or micro total analysis systems (µTAS) is the separation of circulating tumor cells (CTCs) from body fluids. Cancer cells spread from the initial site of a tumor by first invading the surrounding tissue, then by entering the blood or lymph
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Ramm, Paul [Verfasser], and Hans Robert [Akademischer Betreuer] Kalbitzer. "High field 1H-NMR spectroscopy on cell suspensions of neural progenitor cells and brain tumor-initiating cells / Paul Ramm. Betreuer: Hans Robert Kalbitzer." Regensburg : Universitätsbibliothek Regensburg, 2011. http://d-nb.info/1023398796/34.

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Book chapters on the topic "Tumor initiating cells (CICs)"

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Machida, Keigo. "Tumor-Initiating Stem-Like Cells." In Cancer Stem Cells. John Wiley & Sons, 2014. http://dx.doi.org/10.1002/9781118356203.ch27.

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Kameda-Smith, Michelle M., Minomi K. Subapanditha, Sabra K. Salim, Chitra Venugopal, and Sheila K. Singh. "Differentiation of Brain Tumor Initiating Cells." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8805-1_8.

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Bapat, Sharmila A. "Tumor-Initiating Cells in Ovarian Cancer." In Cell Biology of the Ovary. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-7941-2_5.

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Toh, Tan Boon, Yuk Kien Chong, Beng Ti Ang, and Carol Tang. "Glioblastoma Multiforme: Cryopreservation of Brain Tumor-Initiating Cells (Method)." In Tumors of the Central Nervous System, Volume 4. Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-1706-0_10.

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Boiko, Alexander D. "Isolation of Melanoma Tumor-Initiating Cells from Surgical Tissues." In Methods in Molecular Biology. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-62703-227-8_16.

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Mori, Federica, Valeria Canu, Laura Lorenzon, Alfredo Garofalo, Giovanni Blandino, and Sabrina Strano. "Cancer Gastric Chemoprevention: Isolation of Gastric Tumor-Initiating Cells." In Methods in Molecular Biology. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-3191-0_12.

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Ishii, Hideshi. "The Role of the Tumor Suppressor Fhit in Cancer-Initiating Cells." In Regulatory Networks in Stem Cells. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-227-8_36.

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Mulholland, David J., and Hong Wu. "Genetic and Signaling Pathway Regulations of Tumor-Initiating Cells of the Prostate." In Stem Cells and Prostate Cancer. Springer New York, 2013. http://dx.doi.org/10.1007/978-1-4614-6498-3_5.

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Tang, Kwan Ho, Stephanie Ma, and Xin-Yuan Guan. "Liver Tumor-Initiating Cells/Cancer Stem Cells: Past Studies, Current Status, and Future Perspectives." In Advances in Cancer Stem Cell Biology. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-0809-3_11.

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Krtolica, Ana. "Role of Microenvironment in Regulating Stem Cell and Tumor Initiating Cancer Cell Behavior and Its Potential Therapeutic Implications." In Stem Cells and Cancer Stem Cells, Volume 11. Springer Netherlands, 2013. http://dx.doi.org/10.1007/978-94-007-7329-5_14.

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Conference papers on the topic "Tumor initiating cells (CICs)"

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Massagué, Joan. "Abstract IA006: Metastasis initiating cells and ecosystems." In Abstracts: AACR Virtual Special Conference: The Evolving Tumor Microenvironment in Cancer Progression: Mechanisms and Emerging Therapeutic Opportunities; in association with the Tumor Microenvironment (TME) Working Group; January 11-12, 2021. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.tme21-ia006.

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Emmink, Benjamin L., Winan J. van Houdt, Robert G. Vries, et al. "Abstract 4294: Differentiated colorectal cancer cells protect tumor-initiating cells from irinotecan." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-4294.

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Miao, Yuxuan. "Abstract 1491: Immune resistance emerges from tumor-initiating stem cells." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-1491.

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Conard, Sara E., Aaron Ebbs, and Albert S. Baldwin. "Abstract 2877: IKK-mediated signaling controls prostate tumor initiating cells." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-2877.

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Bansal, Nitu, Neil Campbell, Daniel Medina, Robert DiPaola, Joseph R. Bertino, and Hatem E. Sabaawy. "Abstract 4279: Targeting Bmi1 in human prostate tumor initiating cells." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-4279.

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Miao, Yuxuan, and Elaine Fuchs. "Abstract A093: Immune resistance emerges from tumor-initiating stem cells." In Abstracts: Fourth CRI-CIMT-EATI-AACR International Cancer Immunotherapy Conference: Translating Science into Survival; September 30 - October 3, 2018; New York, NY. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/2326-6074.cricimteatiaacr18-a093.

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Miao, Yuxuan. "Abstract 1491: Immune resistance emerges from tumor-initiating stem cells." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-1491.

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Yu, F., J. Wu, C. Gong, F. Su, and E. Song. "Let-7 Inverts the Chemoresistance of Breast Tumor-Initiating Cells." In Abstracts: Thirty-Second Annual CTRC‐AACR San Antonio Breast Cancer Symposium‐‐ Dec 10‐13, 2009; San Antonio, TX. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.sabcs-09-1136.

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Gruber, Martina, Florian Handle, and Zoran Culig. "Abstract 2001: Transcriptional integrator p300 in human prostate tumor-initiating cells." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-2001.

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White, Margaret E., Emma Viktoria Marie Hyddmark, Ali Zarezadeh, et al. "Abstract 1188: Forced induction of differentiation in osteosarcoma tumor initiating cells." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-1188.

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Reports on the topic "Tumor initiating cells (CICs)"

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Drake, Charles G. Immunological Targeting of Tumor Initiating Prostate Cancer Cells. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada613701.

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Nie, Daotai. Targeting Tumor Oct4 to Deplete Prostate Tumor and Metastasis Initiating Cells. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada613791.

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Baldwin, Albert S. Promotion of Tumor-Initiating Cells in Primary and Recurrent Breast Tumors. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada613713.

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Baldwin, Albert S. Promotion of Tumor-Initiating Cells in Primary and Recurrent Breast Tumors. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada596410.

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Marsden, Carolyn. Mesenchymal Stem Cells in the Bone Marrow Provide a Supportive Niche for Early Disseminated Breast Tumor-Initiating Cells. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada552881.

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Rahal, Omar M., and Rosalia C. Simmen. Dietary Regulation of PTEN Signaling and Mammary Tumor Initiating Cells: Implications for Breast Cancer Prevention. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada576870.

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Rahal, Omar. Dietary Regulation of PTEN Signaling and Mammary Tumor Initiating Cells: Implications for Breast Cancer Prevention. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada547234.

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Behbod, Fariba. Laser Raman Tweezer Spectroscopy for the Molecular and Functional Characterization of Single Live Mouse Mammary Tumor-Initiating Cells. Defense Technical Information Center, 2012. http://dx.doi.org/10.21236/ada562457.

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