Relevant bibliographies by topics / Tumor migration assay

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Tumor migration assay'

Author: Grafiati
Published: 4 June 2025
Last updated: 1 August 2025

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Tumor migration assay.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Tumor migration assay"

1

Deryugina, E. I., and M. A. Bourdon. "Tenascin mediates human glioma cell migration and modulates cell migration on fibronectin." Journal of Cell Science 109, no. 3 (1996): 643–52. http://dx.doi.org/10.1242/jcs.109.3.643.

Full text
Abstract:
The role of tenascin in mediating tumor cell migration was studied using two cell migration models. In migration/invasion Transwell assays U251.3 glioma cells rapidly migrated through the 8 mu m pore size membranes onto tenascin- and fibronectin-coated surfaces. In this assay the number of cells migrating onto tenascin was 52.2 +/- 9.6% greater than on fibronectin within 4 hours. To assess cell migration rates and cell morphology, U251.3 migration was examined in a two-dimension spheroid outgrowth assay. The radial distance migrated by U251.3 cells from tumor spheroids was found to be 53.8 +/-
APA, Harvard, Vancouver, ISO, and other styles
2

Liang, Junhui, Wenjing Sun, Hui Song, et al. "NOL6 promotes the proliferation and migration of endometrial cancer cells by regulating TWIST1 expression." Epigenomics 13, no. 19 (2021): 1571–85. http://dx.doi.org/10.2217/epi-2021-0218.

Full text
Abstract:
Aim: To investigate the role and function of NOL6, a protein related to ribosome biogenesis, in endometrial cancer. Methods: Methyl thiazolyl tetrazolium assay, colony formation assay, flow cytometry apoptosis assay, transwell assay and wound healing assays were carried out for evaluating cell proliferation, migration and apoptosis. Immunohistochemistry, western blot and tumor xenograft assays were carried out for detecting the level of protein expression and tumor formation. Results: We demonstrated that NOL6 is overexpressed in endometrial cancer and promotes cell proliferation and migration
APA, Harvard, Vancouver, ISO, and other styles
3

Johnston, Adrian, Zeqi Wan, Tina Chen, et al. "Abstract 4710: Novel 3D cytotoxicity assay to assess the impact of chimeric antigen receptor (CAR) domain design on the tumor infiltration and cytotoxicity efficacy of CAR T-cell therapies for solid tumors." Cancer Research 83, no. 7_Supplement (2023): 4710. http://dx.doi.org/10.1158/1538-7445.am2023-4710.

Full text
Abstract:
Abstract CAR T-cell therapy is a popular topic of discussion in and out of the scientific community. Already with multiple therapies FDA-approved for certain blood cancers, focus has rightly shifted toward FDA approval for solid tumor indications. Yet, advancing past clinical trials to FDA approval has yet to occur and has proved challenging. Of course, multiple factors are at play as to why, but we aim to use-in house expertise to elucidate one, namely CAR T-cell tumor infiltration and tissue migration and surveillance efficacy. Migration is a central parameter ignored in traditional 2D cytot
APA, Harvard, Vancouver, ISO, and other styles
4

Yoon, Jinsoo, Christopher R. Parish, and Lucy A. Coupland. "A Rapid and Accurate Bioluminescence-Based Migration Assay Permitting Analysis of Tumor Cell/Stromal Cell Interactions." Methods and Protocols 3, no. 1 (2020): 10. http://dx.doi.org/10.3390/mps3010010.

Full text
Abstract:
Bioluminescent tumor cell lines are used extensively in vivo to monitor tumor growth and metastasis but rarely used in vitro to follow tumor cell behavior. Tumor cell migration is frequently studied in vitro using transwell assays, however, current methods do not permit the co-incubation of tumor cells with different stromal cell types for analysis of the effects of intercellular cross-talk on tumor cell migration. We describe a novel migration assay using bioluminescent tumor cell lines that is rapid, accurate, and permits the study of the effects of tumor cell-stromal cell interactions on tu
APA, Harvard, Vancouver, ISO, and other styles
5

Koizumi, Shinichiro, Makoto Horikawa, Taisuke Yamamoto, et al. "ET-5 POTENT BYSTANDER EFFECT IN SUICIDE GENE THERAPY USING TK-EXPRESSING STEM CELLS FROM HUMAN EXFOLIATED DECIDUOUS TEETH." Neuro-Oncology Advances 4, Supplement_3 (2022): iii5. http://dx.doi.org/10.1093/noajnl/vdac167.017.

Full text
Abstract:
Abstract Introduction We investigated HSVTK/GCV suicide gene therapy for malignant glioma, and demonstrated the migration ability and antitumor effect of various tissue-derived pluripotent stem cells. In recent years, stem cells from human exfoliated deciduous teeth (SHED), which have excellent ethical and self-renewal ability, have attracted attention, especially in regenerative medicine. In this study, using SHEDTK transfected with TK, we examined the migration ability and antitumor effect against malignant glioma and metastasis models. Methods In vitro assay: Using Matrigel chamber, the mig
APA, Harvard, Vancouver, ISO, and other styles
6

Chen, Yingying, Yuqiang Zhang, Wei Song, Ying Zhang, Xiu Dong, and Mingqi Tan. "Ginsenoside Rh2 Inhibits Migration of Lung Cancer Cells under Hypoxia via mir-491." Anti-Cancer Agents in Medicinal Chemistry 19, no. 13 (2019): 1633–41. http://dx.doi.org/10.2174/1871520619666190704165205.

Full text
Abstract:
Background: Ginsenoside Rh2 (Rh2), which is extracted from ginseng, exerts antitumor activity. Here we would like to study the role of Rh2 on hypoxia-induced migration in lung adenocarcinoma. Methods: Lung adenocarcinoma A549 and H1299 cells were cultured in 1% O2 condition to mimic the hypoxic tumor microenvironment. The migrations of cancer cells were measured by transwell assay and scratch assay. Results: Rh2 could inhibit hypoxia-induced A549 and H1299 cell migration via increase of mir-491 expression. Further, mir-491 antisense oligonucleotide could repress hypoxia-induced migration and t
APA, Harvard, Vancouver, ISO, and other styles
7

Wan, Huimin, Tingting Lin, Mengtian Shan, Jingjing Lu та Zhongliang Guo. "LINC00491 Facilitates Tumor Progression of Lung Adenocarcinoma via Wnt/β-Catenin-Signaling Pathway by Regulating MTSS1 Ubiquitination". Cells 11, № 23 (2022): 3737. http://dx.doi.org/10.3390/cells11233737.

Full text
Abstract:
Background: Long non-coding RNAs have been reported to be involved in tumorigenesis and progression through different regulatory mechanisms. It has been reported that aberrantly expressed long non-coding RNA LINC00491 promotes malignancy in multiple tumors, while the role of LINC00491 in lung adenocarcinoma (LUAD) is little reported and the mechanism for regulating tumor progression has not been elucidated. Methods: RNA sequencing and the TCGA database were combined to screen differentially expressed lncRNAs that facilitate tumor progression. The expression level of LINC00491 was examined in L
APA, Harvard, Vancouver, ISO, and other styles
8

Guo, Kai, Lei Song, Jianyong Chang, Peicheng Cao та Qi Liu. "AEBP1 Promotes Glioblastoma Progression and Activates the Classical NF-κB Pathway". Behavioural Neurology 2020 (6 листопада 2020): 1–10. http://dx.doi.org/10.1155/2020/8890452.

Full text
Abstract:
Objective. Our study was aimed at investigating the mechanistic consequences of the upregulation of adipocyte enhancer-binding protein 1 (AEBP1) in glioblastoma (GBM). Methods. The expression of AEBP1 in GBM was assessed by bioinformatics analysis and qRT-PCR; the effects of AEBP1 on GBM cell proliferation, migration, invasion, and tumor growth in vitro and in vivo were detected by a CCK-8 assay, colony formation assay, scratch assay, Transwell assay, and subcutaneous tumor formation, respectively. The activation of related signaling pathways was monitored using western blot. Results. Tumor-re
APA, Harvard, Vancouver, ISO, and other styles
9

Noh, Jinok, Jinyeong Yu, Wootak Kim, Aran Park та Ki-Sook Park. "Bone Marrow-Derived Mesenchymal Stem Cells Migrate toward Hormone-Insensitive Prostate Tumor Cells Expressing TGF-β via N-Cadherin". Biomedicines 9, № 11 (2021): 1572. http://dx.doi.org/10.3390/biomedicines9111572.

Full text
Abstract:
The prostate tumor microenvironment plays important roles in the metastasis and hormone-insensitive re-growth of tumor cells. Bone marrow-derived mesenchymal stem cells (BM-MSCs) are recruited into prostate tumors to facilitate tumor microenvironment formation. However, the specific intrinsic molecules mediating BM-MSCs’ migration to prostate tumors are unknown. BM-MSCs’ migration toward a conditioned medium (CM) of hormone-insensitive (PC3 and DU145) or hormone-sensitive (LNCaP) prostate tumor cells was investigated using a three-dimensional cell migration assay and a transwell migration assa
APA, Harvard, Vancouver, ISO, and other styles
10

Jin, Yang, Li Lv, Shu-Xiang Ning, Ji-Hong Wang, and Rong Xiao. "The Anti-tumor Activity and Mechanisms of rLj-RGD3 on Human Laryngeal Squamous Carcinoma Hep2 Cells." Anti-Cancer Agents in Medicinal Chemistry 19, no. 17 (2020): 2108–19. http://dx.doi.org/10.2174/1871520619666191022160024.

Full text
Abstract:
Background: Laryngeal Squamous Cell Carcinoma (LSCC) is a malignant epithelial tumor with poor prognosis and its incidence rate increased recently. rLj-RGD3, a recombinant protein cloned from the buccal gland of Lampetra japonica, contains three RGD motifs that could bind to integrins on the tumor cells. Methods: MTT assay was used to detect the inhibitory rate of viability. Giemsa’s staining assay was used to observe the morphological changes of cells. Hoechst 33258 and TUNEL staining assay, DNA ladder assay were used to examine the apoptotic. Western blot assay was applied to detect the chan
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Tumor migration assay"

1

Essex, Rachel R. "Determining the Effects of CD151 and β1 on Tumor Cell Adhesion and Migration". UKnowledge, 2015. http://uknowledge.uky.edu/cme_etds/56.

Full text
Abstract:
Previous studies have shown that the upregulation of CD151 and β1 is associated with poor prognosis in many cancers such as breast cancer. Studies have provided evidence that these proteins are associated with the adhesion and migration of tumor cells. In this study, a microfluidic flow chamber was utilized to determine how CD151 and β1 affected the firm and initial adhesion of metastatic breast cancer cells to a planar endothelial monolayer under shear stress. This system mimicked the adhesion of metastatic breast cancer cells to the endothelial cells of the circulatory system. CD151 and β1 i
APA, Harvard, Vancouver, ISO, and other styles
2

Staneva, Ralitza. "Dynamique des cellules cancéreuses dans le coeur tumoral et rôle de la matrice extracellulaire dans l'invasion métastatique Cancer cells in the tumor core exhibit spatially coordinated migration patterns A new biomimetic assay reveals the temporal role of matrix stiffening in cancer cell invasion." Thesis, Sorbonne Paris Cité, 2018. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=1974&f=14542.

Full text
Abstract:
Le développement de carcinomes est un processus graduel, pendant lequel l'accumulation de modifications génétiques promeut la prolifération et la différenciation aberrante des cellules épithéliales. Le processus métastatique est la cause majeure de décès dus au cancer. Dans le cancer du côlon, vingt-cinq pourcent des patients présentent déjà des métastases au diagnostic de la maladie, et 25-35% en développent à la progression de la maladie. La colonisation métastatique d'organes distants requiert la réalisation d'une série complexe d'événements. L'acquisition d'un phénotype migratoire par les
APA, Harvard, Vancouver, ISO, and other styles
3

Vittadello, Sean T. "Mathematical models for cell migration and proliferation informed by visualisation of the cell cycle." Thesis, Queensland University of Technology, 2020. https://eprints.qut.edu.au/204074/1/Sean_Vittadello_Thesis.pdf.

Full text
Abstract:
Cell migration and proliferation are essential for normal physiological processes, however their misregulation contributes to pathologies including cancer. In this thesis we develop and analyse new mathematical models of cell migration and proliferation, based on new experimental studies that provide visualisation of cell cycle progression, to improve understanding of the migration and proliferation of cells. In particular, we investigate cell migration as a function of cell cycle dynamics, normally-hidden cell synchronisation in cellular assays, whether cell migration and proliferation are mu
APA, Harvard, Vancouver, ISO, and other styles
4

Roy, Joannie. "Fonctionnalisation de substrats pour l'étude des phénotypes de migration cellulaire." Thèse, 2017. http://hdl.handle.net/1866/20274.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Tumor migration assay"

1

Mobilio, Daniel, Agata M. Kieliszek, Chitra Venugopal, Sheila K. Singh, and Shawn C. Chafe. "Efficient Migration Assay for Brain Tumor Stem Cells." In Methods in Molecular Biology. Springer US, 2025. https://doi.org/10.1007/978-1-0716-4654-0_11.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Vinci, Maria, Carol Box, Miriam Zimmermann, and Suzanne A. Eccles. "Tumor Spheroid-Based Migration Assays for Evaluation of Therapeutic Agents." In Target Identification and Validation in Drug Discovery. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-311-4_16.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Zimmermann, Miriam, Carol Box, and Suzanne A. Eccles. "Two-Dimensional vs. Three-Dimensional In Vitro Tumor Migration and Invasion Assays." In Target Identification and Validation in Drug Discovery. Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-311-4_15.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Dey, Abhinav, and David J. Sharp. "In Vitro Screening Assay for Activators of T-Cell Migration to Solid Tumors." In Methods in Molecular Biology. Springer US, 2022. http://dx.doi.org/10.1007/978-1-0716-1952-0_11.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Centonze, Giorgia, Jennifer Chapelle, Costanza Angelini, et al. "The Scaffold Protein p140Cap as a Molecular Hub for Limiting Cancer Progression: A New Paradigm in Neuroblastoma." In Pheochromocytoma, Paraganglioma and Neuroblastoma. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96383.

Full text
Abstract:
Neuroblastoma, the most common extra-cranial pediatric solid tumor, is responsible for 9–15% of all pediatric cancer deaths. Its intrinsic heterogeneity makes it difficult to successfully treat, resulting in overall survival of 50% for half of the patients. Here we analyze the role in neuroblastoma of the adaptor protein p140Cap, encoded by the SRCIN1 gene. RNA-Seq profiles of a large cohort of neuroblastoma patients show that SRCIN1 mRNA levels are an independent risk factor inversely correlated to disease aggressiveness. In high-risk patients, SRCIN1 was frequently altered by hemizygous dele
APA, Harvard, Vancouver, ISO, and other styles
6

Van Damme, Jo, Jean-Pierre Lenaerts, and Sofie Struyf. "Assays for leucocyte migration." In Cytokine Cell Biology. Oxford University PressOxford, 2000. http://dx.doi.org/10.1093/oso/9780199638604.003.0006.

Full text
Abstract:
Abstract In view of the recent developments in cytokine research, there has been a revived interest in chemotaxis assays. Indeed, within a short time a number of novel chemotactic cytokines (chemokines) have been identified. These low molecular weight proteins are different from other cytokines such as interleukin-1 (IL-1) and tumour necrosis factor (TNF), previously reported to be chemotactic for leukocytes. Chemokines belong to a large family of small proteins that exert their chemotactic activity different leucocytic cell types (1-4). Like several other chemotactic substances, such as formy
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Tumor migration assay"

1

"Impact of Heparan Sulphate Binding Domain of Chemokine CCL21 to Migration of Breast Cancer Cells." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2020. http://dx.doi.org/10.29117/quarfe.2020.0132.

Full text
Abstract:
Lymph node metastasis constitutes a key event in breast cancer progression. Chemokines are small proteins, which can promote metastatic spread by inducing cancer cell migration and invasion. Chemokine function is dependant upon their binding to both cell surface heparan sulphate (HS) molecules and to their specific receptor. Our group has demonstrated a significant increase in chemokine receptor CCR7 expression in cancerous breast epithelia compared to healthy controls. This study is designed to test the hypothesis that a non-HS binding forms of chemokine CCL21 can disrupt the normal response
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the extended bibliographies on the topic 'Tumor migration assay' for particular source types:
Journal articles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography