Relevant bibliographies by topics / Tumor necrosis factor. Septische shock

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Tumor necrosis factor. Septische shock'

Author: Grafiati
Published: 4 June 2021
Last updated: 8 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Tumor necrosis factor. Septische shock.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Tumor necrosis factor. Septische shock"

1

Baud, L., J. Cadranel, G. Offenstadt, L. Luquel, B. Guidet, and P. Amstutz. "Tumor Necrosis Factor and Septic Shock." Critical Care Medicine 18, no. 3 (1990): 349. http://dx.doi.org/10.1097/00003246-199003000-00034.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Murphey, E. D., and D. L. Traber. "TUMOR NECROSIS FACTOR PRETREATMENT ATTENUATES ENDOTOXIN-INDUCED SHOCK." Shock 9, Supplement (1998): 32. http://dx.doi.org/10.1097/00024382-199806001-00107.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

MATUSCHAK, GEORGE M., KARI E. LAMPRECH, and ANDREW J. LECHNER. "Pentoxifylline Inhibits Tumor Necrosis Factor Production in Septic Shock." Journal of Interferon Research 14, no. 5 (1994): 293–95. http://dx.doi.org/10.1089/jir.1994.14.293.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Meldrum, Daniel R. "Tumor necrosis factor in the heart." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 274, no. 3 (1998): R577—R595. http://dx.doi.org/10.1152/ajpregu.1998.274.3.r577.

Full text
Abstract:
The heart is a tumor necrosis factor (TNF)-producing organ. Both myocardial macrophages and cardiac myocytes themselves synthesize TNF. Accumulating evidence indicates that myocardial TNF is an autocrine contributor to myocardial dysfunction and cardiomyocyte death in ischemia-reperfusion injury, sepsis, chronic heart failure, viral myocarditis, and cardiac allograft rejection. Indeed, locally (vs. systemically) produced TNF contributes to postischemic myocardial dysfunction via direct depression of contractility and induction of myocyte apoptosis. Lipopolysaccharide or ischemia-reperfusion ac
APA, Harvard, Vancouver, ISO, and other styles
5

Appoloni, O., E. Dupont, M. Vandercruys, M. Andriens, J. Duchateau, and Jean-Louis Vincent. "Association of tumor necrosis factor-2 allele with plasma tumor necrosis factor-alpha levels and mortality from septic shock." American Journal of Medicine 110, no. 6 (2001): 486–88. http://dx.doi.org/10.1016/s0002-9343(01)00656-8.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Huys, Liesbeth, Filip Van Hauwermeiren, Lien Dejager, et al. "Type I interferon drives tumor necrosis factor–induced lethal shock." Journal of Experimental Medicine 206, no. 9 (2009): 1873–82. http://dx.doi.org/10.1084/jem.20090213.

Full text
Abstract:
Tumor necrosis factor (TNF) is reputed to have very powerful antitumor effects, but it is also a strong proinflammatory cytokine. Injection of TNF in humans and mice leads to a systemic inflammatory response syndrome with major effects on liver and bowels. TNF is also a central mediator in several inflammatory diseases. We report that type I interferons (IFNs) are essential mediators of the lethal response to TNF. Mice deficient in the IFN-α receptor 1 (IFNAR-1) or in IFN-β are remarkably resistant to TNF-induced hypothermia and death. After TNF injection, IFNAR-1−/− mice produced less IL-6, h
APA, Harvard, Vancouver, ISO, and other styles
7

Dellinger, R. Phillip. "Tumor necrosis factor in septic shock and multiple system trauma." Critical Care Medicine 25, no. 11 (1997): 1771–73. http://dx.doi.org/10.1097/00003246-199711000-00005.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Rhee, Peter, Kenneth Waxman, Lisa Clark, et al. "Tumor necrosis factor and monocytes are released during hemorrhagic shock." Resuscitation 25, no. 3 (1993): 249–55. http://dx.doi.org/10.1016/0300-9572(93)90122-7.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Teppo, A. M., and C. P. Maury. "Radioimmunoassay of tumor necrosis factor in serum." Clinical Chemistry 33, no. 11 (1987): 2024–27. http://dx.doi.org/10.1093/clinchem/33.11.2024.

Full text
Abstract:
Abstract We present a double-antibody radioimmunoassay for determination of the concentration of tumor necrosis factor (TNF) in serum. TNF in serum competes with a fixed amount of 125I-labeled TNF for the binding sites of specific rabbit antibodies. The bound TNF is precipitated with Sepharose-bound anti-rabbit IgG, then centrifuged, and the radioactivity of the pellets is counted. The detection limit of the assay is 7 ng/L (B0-3 SD). Bound radioactivity in the range of 10% to 90% of the B0 counts corresponds to TNF concentrations of 26 to 10,000 ng/L. Of 40 sera from healthy subjects, 21 (53%
APA, Harvard, Vancouver, ISO, and other styles
10

Dinata, Khrisanti, Ari L. Runtunuwu, Jose M. Mandei, and Julius H. Lolombulan. "Correlation between tumor necrosis factor-alpha and septic shock in children." Paediatrica Indonesiana 53, no. 1 (2013): 1. http://dx.doi.org/10.14238/pi53.1.2013.1-5.

Full text
Abstract:
Background The crucial role cytokines play in the pathophysiologyof sepsis is widely accepted. Infection stimulates the productionof cytokines in various cell types. Tumor necrosis factor-alpha(TNF-a) is one of the most extensively investigated cytokines inexperimental and clinical sepsis. Tumor necrosis factor-alpha hasbeen shown to mediate lethality in experimental sepsis.Objective To evaluate for a possible correlation between TNF-alevel and septic shock in children.Methods This cross-sectional study was conducted in Manadofrom June to September 2011. A total of 40 patients with arecent dia
APA, Harvard, Vancouver, ISO, and other styles
More sources

Dissertations / Theses on the topic "Tumor necrosis factor. Septische shock"

1

Engelberts, Ingeborg. "Tumor necrosis factor during sepsis king of cytokines? /." Maastricht : Maastricht : Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 1994. http://arno.unimaas.nl/show.cgi?fid=6955.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Ayub, Qasim. "Prevention of endotoxic shock in mice using anti-tumor necrosis factor-alpha monoclonal antibody." Thesis, University of North Texas, 1991. https://digital.library.unt.edu/ark:/67531/metadc798464/.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Labruto, Fausto. "Modifications of cardiovascular response to ischemia and trauma /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-379-5/.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Baumann, Cory W. "Heat Shock Protein 70 Regulates Tumor Necrosis Factor-Alpha and Myogenin in Skeletal Muscle Following Chemical-Induced Injury." 2015. http://scholarworks.gsu.edu/kin_health_diss/12.

Full text
Abstract:
Skeletal muscle injury results in functional deficits that can take several weeks to fully recover. Ultimate recovery of function is dependent on the muscle’s ability to regenerate, a highly coordinated process that involves transient muscle inflammation and the replacement of damaged myofibers. Instrumental in the inflammatory response, is the pro-inflammatory cytokine TNF-α. Expression of TNF-α is thought to be regulated, in part, by the stress sensing 70 kDa heat shock protein (Hsp70). However, it remains unclear how Hsp70 alters TNF-α following injury, and if so, how these changes affect s
APA, Harvard, Vancouver, ISO, and other styles
5

Tsai, Ming-Che, та 蔡明哲. "Part IMLC601, a Traditional Chinese Medicine attenuates traumatic brain injury in rats by reducing early microglial expression of tumor necrosis factor-αPart IIHigh-altitude pulmonary edema can be prevented by heat shock protein-70-mediated hyperbaric oxygen preconditioning". Thesis, 2015. http://ndltd.ncl.edu.tw/handle/q4c3vn.

Full text
Abstract:
博士<br>中山醫學大學<br>醫學研究所<br>103<br>Part I Objective: Traumatic brain injury (TBI) causes increased release of several mediators from injured and dead cells and elicits microglial activation. Activated microglia change morphology, migrate to injury sites, and release tumor necrosis factor-alpha (TNF-α) and others. In this study we used a controlled fluid percussion injury model of TBI in the rat to determine whether immediate treatment with MLC601, a traditional Chinese medicine, would affect microglial activation and improve recovery. MLC was chosen for this study because it is beneficial in trea
APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Tumor necrosis factor. Septische shock"

1

Lowry, Stephen F., Kimberly J. VanZee, Craig S. Rock, et al. "Tumor Necrosis Factor as a Mediator of Sepsis." In Shock, Sepsis, and Organ Failure. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77420-1_1.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Cohen, J. "Clinical Role of Tumor Necrosis Factor in Septic Shock." In Update in Intensive Care and Emergency Medicine. Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84423-2_30.

Full text
APA, Harvard, Vancouver, ISO, and other styles
3

Engelmann, H., D. Aderka, Y. Nophar, et al. "Soluble and Cell Surface Receptors for Tumor Necrosis Factor." In Host Defense Dysfunction in Trauma, Shock and Sepsis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_76.

Full text
APA, Harvard, Vancouver, ISO, and other styles
4

Rothe, G., W. Kellermann, J. Briegel, B. Schaerer, and G. Valet. "Activation of Neutrophils by Tumor Necrosis Factor-α During Sepsis." In Host Defense Dysfunction in Trauma, Shock and Sepsis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_94.

Full text
APA, Harvard, Vancouver, ISO, and other styles
5

Lowry, S. F. "Tumor Necrosis Factor and Other Cytokines as Mediators of Clinical Sepsis." In Host Defense Dysfunction in Trauma, Shock and Sepsis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_27.

Full text
APA, Harvard, Vancouver, ISO, and other styles
6

Mealy, K., B. G. Robinson, J. A. Majzoub, and D. W. Wilmore. "Effects of Tumor Necrosis Factor on the Hypothalamic-Pituitary-Adrenal Axis." In Host Defense Dysfunction in Trauma, Shock and Sepsis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_87.

Full text
APA, Harvard, Vancouver, ISO, and other styles
7

Norton, Jeffrey A., and H. Richard Alexander. "Tumor Necrosis Factor-α and Interleukin-1 Desensitization: Clinical Utility and Possible Mechanisms." In Shock, Sepsis, and Organ Failure. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77420-1_13.

Full text
APA, Harvard, Vancouver, ISO, and other styles
8

Hosford, D., and P. Braquet. "Potential Role for Platelet-Activating Factor and Tumor Necrosis Factor in the Immune Impairments in Shock and Trauma." In Immune Consequences of Trauma, Shock, and Sepsis. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-73468-7_40.

Full text
APA, Harvard, Vancouver, ISO, and other styles
9

Stadler, J., B. G. Bentz, J. M. Langrehr, et al. "Tumor Necrosis Factor Alpha Inhibits Hepatocyte Mitochondrial Respiration and Induces Release of Cytoplasmic Enzymes." In Host Defense Dysfunction in Trauma, Shock and Sepsis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_67.

Full text
APA, Harvard, Vancouver, ISO, and other styles
10

Barke, R. A., P. S. Brady, and L. J. Brady. "Modulation of Hepatic Mitochondrial Fat Oxidation and Hepatic Gene Transcription by Tumor Necrosis Factor." In Host Defense Dysfunction in Trauma, Shock and Sepsis. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77405-8_68.

Full text
APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Tumor necrosis factor. Septische shock"

1

Qiu, Ping, Xizhong Cui, Junfeng Sun, Judith A. Welsh, Charles Natanson, and Peter Q. Eichacker. "Selective Tumor Necrosis Factor Inhibitors' Effect On Survival In Septic Shock: A Meta-Analysis." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6002.

Full text
APA, Harvard, Vancouver, ISO, and other styles
2

Stenson, E., E. Killeen, JC Cohen, and SI Shah. "100% Hyperoxia at Birth Results in Increased Levels of Heat Shock Protein 27 (HSP27) and Decreased Levels of Tumor Necrosis Factor Alpha (TNF-alpha), Toll-Like Receptor 4 (TLR-4), Thyroid Transcription Factor (TTF-1), and Interleukins 6 and 10 (IL-6,10) in the Lungs of Adult Sprague-Dawley Rats." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a4112.

Full text
APA, Harvard, Vancouver, ISO, and other styles
You might also be interested in the extended bibliographies on the topic 'Tumor necrosis factor. Septische shock' for particular source types:
Journal articles
We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!

To the bibliography