Relevant bibliographies by topics / Tumor vascularization

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Tumor vascularization'

Author: Grafiati
Published: 4 June 2021
Last updated: 8 February 2022

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Journal articles on the topic "Tumor vascularization"

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Zhang, Li-Zhi, Chang-Qing Zhang, Zhen-Yu Yan, Qing-Cheng Yang, Yao Jiang, and Bing-Fang Zeng. "Tumor-initiating cells and tumor vascularization." Pediatric Blood & Cancer 56, no. 3 (November 8, 2010): 335–40. http://dx.doi.org/10.1002/pbc.22886.

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Brossa, Alessia, Lola Buono, Sofia Fallo, Alessandra Fiorio Pla, Luca Munaron, and Benedetta Bussolati. "Alternative Strategies to Inhibit Tumor Vascularization." International Journal of Molecular Sciences 20, no. 24 (December 7, 2019): 6180. http://dx.doi.org/10.3390/ijms20246180.

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Endothelial cells present in tumors show different origin, phenotype, and genotype with respect to the normal counterpart. Various mechanisms of intra-tumor vasculogenesis sustain the complexity of tumor vasculature, which can be further modified by signals deriving from the tumor microenvironment. As a result, resistance to anti-VEGF therapy and activation of compensatory pathways remain a challenge in the treatment of cancer patients, revealing the need to explore alternative strategies to the classical anti-angiogenic drugs. In this review, we will describe some alternative strategies to inhibit tumor vascularization, including targeting of antigens and signaling pathways overexpressed by tumor endothelial cells, the development of endothelial vaccinations, and the use of extracellular vesicles. In addition, anti-angiogenic drugs with normalizing effects on tumor vessels will be discussed. Finally, we will present the concept of endothelial demesenchymalization as an alternative approach to restore normal endothelial cell phenotype.
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RAJORIA, SHILPI, ROBERT SURIANO, YUSHAN L. WILSON, ANDREA L. GEORGE, JAN GELIEBTER, STIMSON P. SCHANTZ, and RAJ K. TIWARI. "Estradiol-mediated tumor neo-vascularization." Oncology Letters 2, no. 3 (March 21, 2011): 453–57. http://dx.doi.org/10.3892/ol.2011.283.

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Auguste, Patrick, Sylvie Lemiere, Fréderic Larrieu-Lahargue, and Andreas Bikfalvi. "Molecular mechanisms of tumor vascularization." Critical Reviews in Oncology/Hematology 54, no. 1 (April 2005): 53–61. http://dx.doi.org/10.1016/j.critrevonc.2004.11.006.

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Ribatti, Domenico, and Francesco Pezzella. "Overview on the Different Patterns of Tumor Vascularization." Cells 10, no. 3 (March 13, 2021): 639. http://dx.doi.org/10.3390/cells10030639.

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Angiogenesis is a crucial event in the physiological processes of embryogenesis and wound healing. During malignant transformation, dysregulation of angiogenesis leads to the formation of a vascular network of tumor-associated capillaries promoting survival and proliferation of the tumor cells. Starting with the hypothesis formulated by Judah Folkman that tumor growth is angiogenesis-dependent, this area of research has a solid scientific foundation and inhibition of angiogenesis is a major area of therapeutic development for the treatment of cancer. Over this period numerous authors published data of vascularization of tumors, which attributed the cause of neo-vascularization to various factors including inflammation, release of angiogenic cytokines, vasodilatation, and increased tumor metabolism. More recently, it has been demonstrated that tumor vasculature is not necessarily derived by endothelial cell proliferation and sprouting of new capillaries, but alternative vascularization mechanisms have been described, namely vascular co-option and vasculogenic mimicry. In this article, we have analyzed the mechanisms involved in tumor vascularization in association with classical angiogenesis, including post-natal vasculogenesis, intussusceptive microvascular growth, vascular co-option, and vasculogenic mimicry. We have also discussed the role of these alternative mechanism in resistance to anti-angiogenic therapy and potential therapeutic approaches to overcome resistance.
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Lejeune, Ferdy J. "Improving drug penetration in tumors by targeting tumor vascularization." Targeted Oncology 1, no. 2 (April 2006): 90–96. http://dx.doi.org/10.1007/s11523-006-0019-0.

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Grunstein, Jeremy, Joseph J. Masbad, Reed Hickey, Frank Giordano, and Randall S. Johnson. "Isoforms of Vascular Endothelial Growth Factor Act in a Coordinate Fashion To Recruit and Expand Tumor Vasculature." Molecular and Cellular Biology 20, no. 19 (October 1, 2000): 7282–91. http://dx.doi.org/10.1128/mcb.20.19.7282-7291.2000.

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ABSTRACT Vascular endothelial growth factor (VEGF) is an essential regulator of vascularization. It is expressed as several splice variants; the major forms contain 120 amino acids, 164 amino acids, and 188 amino acids. We utilized transformed cells nullizygous for VEGF to specifically express each of these isoforms in isolation, in order to determine the role of each in tumorigenic neo-vascularization. We found that only the intermediate isoform, VEGF164, could fully rescue tumor growth; VEGF120 partially rescued tumor growth, and VEGF188 failed completely to rescue tumor expansion. Surprisingly, the vascular density of VEGF188 isoform-expressing tumors is significantly greater than that of wild-type VEGF cells and the other isoform-specific tumors. The failure of the hypervascular VEGF188-expressing tumors to grow may be due to inadequate perfusion of the massive number of microvessels in these tumors; three-dimensional imaging of the tumorigenic vasculature indicated little or no recruitment of the peripheral vasculature. This demonstrates that the VEGF isoforms perform unique functions which together enable tumorigenic vascularization.
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Dock, W., F. Grabenwöger, V. Metz, K. Eibenberger, and M. T. Farrés. "Tumor vascularization: assessment with duplex sonography." Radiology 181, no. 1 (October 1991): 241–44. http://dx.doi.org/10.1148/radiology.181.1.1887039.

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Conejo-Garcia, Jose R., Ronald J. Buckanovich, Fabian Benencia, Maria C. Courreges, Stephen C. Rubin, Richard G. Carroll, and George Coukos. "Vascular leukocytes contribute to tumor vascularization." Blood 105, no. 2 (January 15, 2005): 679–81. http://dx.doi.org/10.1182/blood-2004-05-1906.

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Abstract There is no proof that hematopoietic cells contribute significantly to vasculogenesis in postnatal life. Here we report a novel leukocyte subset within ovarian carcinoma that coexpresses endothelial and dendritic cell markers. Fluorescence-activated cell sorter (FACS) analysis identified a high frequency of VE-cadherin+ CD45+ leukocytes (39% of host cells) in 10 of 10 solid tumors evaluated. This population represented less than 1% of nontumor cells in ascites and peripheral blood. At the protein level, more than 86% of these cells expressed the endothelial markers P1H12, CD34, and CD31 and leukocyte markers CD11c and major histocompatibility complex (MHC) class II. At the mRNA level, we detected TEM1, TEM7, and Thy-1, specific markers of angiogenic endothelium. Finally, this population has the capacity to generate functional blood vessels in vivo. Because of its mixed phenotype, we named this population vascular leukocytes (VLCs). Our data provide an important link between hematopoietic endothelial precursors and vascular development in postnatal life and a possible novel therapeutic target.
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Belotti, Dorina, Denise Pinessi, and Giulia Taraboletti. "Alternative Vascularization Mechanisms in Tumor Resistance to Therapy." Cancers 13, no. 8 (April 15, 2021): 1912. http://dx.doi.org/10.3390/cancers13081912.

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Blood vessels in tumors are formed through a variety of different mechanisms, each generating vessels with peculiar structural, molecular, and functional properties. This heterogeneity has a major impact on tumor response or resistance to antineoplastic therapies and is now emerging as a promising target for strategies to prevent drug resistance and improve the distribution and efficacy of antineoplastic treatments. This review presents evidence of how different mechanisms of tumor vessel formation (vasculogenesis, glomeruloid proliferation, intussusceptive angiogenesis, vasculogenic mimicry, and vessel co-option) affect tumor responses to antiangiogenic and antineoplastic therapies, but also how therapies can promote alternative mechanisms of vessel formation, contributing to tumor recurrence, malignant progression, and acquired drug resistance. We discuss the possibility of tailoring treatment strategies to overcome vasculature-mediated drug resistance or to improve drug distribution and efficacy.
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Dissertations / Theses on the topic "Tumor vascularization"

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D'Amico, Anthony V. "Analysis of mammalian tumor vascularization in the development of a therapy to prevent metastasis." Thesis, Massachusetts Institute of Technology, 1986. http://hdl.handle.net/1721.1/33470.

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Thesis (Ph. D.)--Massachusetts Institute of Technology, Dept. of Nuclear Engineering, 1986.
MICROFICHE COPY AVAILABLE IN ARCHIVES AND SCIENCE
Bibliography: leaves 120-123.
by Anthony V. D'Amico.
Ph.D.
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Kim, Jung-Hyun. "Conjugated linoleic acid modulation of mammary tumor growth and vascularization through alteration of the eicosanoid pathway /." For electronic version search Digital dissertations database. Restricted to UC campuses. Access is free to UC campus dissertations, 2005. http://uclibs.org/PID/11984.

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Brunner, Carlos Henrique Maciel. "Avaliação do comportamento vascular do tumor de Ehrlich na forma sólida em camundongos submetidos à eletroquimioterapia com bleomicina." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-26012016-160046/.

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A eletroquimioterapia (EQT) é uma modalidade de tratamento recente que se baseia na associação de quimioterápicos potencializados pela eletroporação. Possui indicação para neoplasias sólidas de origens histológicas distintas, apresentando baixa morbidade e elevada eficiência. A ação da EQT ocorre em múltiplos sítios, tanto envolvendo a quebra da molécula de DNA, quanto exercendo efeito sobre a vasculatura tumoral. No presente estudo buscou-se maior compreensão dos eventos vasculares, avaliando-se qualitativamente e quantitativamente, com auxílio de marcação imunológica, com fator VIII e VEGF-A, a vascularização do tumor de Ehrlich implantado na forma sólida em camundongos, não tratados e submetidos à EQT com bleomicina, após sete dias de tratamento. No intuito de melhor elucidar os fenômenos vasculares, também foi investigado o efeito do resveratrol associado à EQT. O resveratrol, presente em vegetais como as uvas, possui efeitos de inibição do HIF1-α, reconhecida proteina que estimula a angiogênese em condições de hipoxia tumoral. Os animais submetidos à quimioterapia com bleomicina não apresentaram redução de volume tumoral, ao contrário dos que sofreram EQT com o mesmo fármaco. Evidenciou-se maior densidade microvascular tumoral em animais tratados com quimioterapia, quando comparados aos não tratados e aos submetidos à EQT. O tratamento com resveratrol diminuiu a expressão de VEGF-A e obteve efeito mais pronunciado quando associado à EQT com bleomicina. Através dos fenômenos pesquisados pôde-se evidenciar que a EQT com bleomicina foi efetiva na redução do volume do tumor de Ehrlich e que houve redução da atividade proliferativa assim como da densidade microvascular tumoral. Também observou-se que o resveratrol, ainda mais quando associado à EQT com bleomicina, reduz a proliferação tumoral e a expressão de VEGF-A
The electrochemotherapy (EQT) is a new treatment modality based on the association of chemotherapy potentiated by electroporation. Has indication for solid neoplasms of histological distinct origins, presenting low morbidity and high efficiency. The action of the EQT occurs in multiple sites, both involving the breakage of the DNA molecule, as having an effect on the tumor vasculature. The present study aimed at better understanding of vascular events, evaluating qualitatively and quantitatively, using immune labeling, with factor VIII and VEGF-A, the vascularization of the Ehrlich tumor implanted in solid form in mice, untreated and submitted to EQT with bleomycin, after seven days of treatment. In order to better elucidate the vascular phenomena, was also investigated the effect of resveratrol associated with EQT. The resveratrol present in plants such as grapes, has inhibitory effects of HIF1-α, a protein that is recognized to stimulates angiogenesis in tumor hypoxia. The animals submitted to chemotherapy with bleomycin showed no reduction of tumor volume, unlike those who suffered EQT with the same drug. It was evidenced increased microvessel density tumor in animals treated with chemotherapy, when compared to untreated and those submitted to EQT. The treatment with resveratrol decreased the expression of VEGF-A and obtained effect was more pronounced when associated to the EQT with bleomycin. This research can prove that the EQT with bleomycin was effective in reducing the volume of Ehrlich tumor and that there was a reduction of proliferative activity as well as of microvascular density tumor. Also it was observed that the resveratrol, even more when associated with EQT with bleomycin, reduces the tumor proliferation and the expression of VEGF-A
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McCandless, John Richard 1954. "Alpha-6 beta-1 and alpha-6 beta-4 integrin expression and the vascularization of human prostate tumor xenografts." Thesis, The University of Arizona, 1997. http://hdl.handle.net/10150/278603.

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Growth and metastasis of tumors appear to be dependent on the ability of tumor cells to recruit blood vessels. Integrins are a class of cell adhesion molecules that may have a role in angiogenesis. In this study the effect of the expression of two integrins, α6β1 and α6β4, on microvessel density in human prostate tumor xenografts in SCID mice was evaluated. Five methods (one-person count, two-person count, digital analysis of immunostained tissues, and digital analysis of vascular corrosion casts) were used to measure microvessel density. Results indicate that alpha6 integrin expression correlates negatively with tumor vessel density. and with tumor cell proliferation but not the extent of the tumor burden. β4 integrin expression does not appear to affect tumor vessel density, tumor cell proliferation, nor tumor burden. Comparison of methods of quantitation suggest that computer-assisted vessel counting may offer advantages over optical counting or computer-assisted area measurement.
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Agarwal, Pranay. "Multiscale Biomaterials for Cell and Tissue Engineering." The Ohio State University, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1482945107612275.

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Orme, Michelle Elaine. "The vascularization of solid tumours : mathematical models of tumour angiogenesis and vascular tumour growth." Thesis, University of Bath, 1996. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.362238.

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Colombel, Virginie. "Synthèse et étude de l’activité biologique de nouveaux analogues du N-acétylcolchinol." Thesis, Lyon 1, 2009. http://www.theses.fr/2009LYO10294/document.

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Le N-acétylcolchinol est un composé hémi-synthétique connu pour inhiber la polymérisation de la tubuline en microtubules. Il a montré une activité prometteuse en tant qu’agent ciblant la vascularisation tumorale, cependant, sa cardiotoxicité a conduit à l’arrêt des essais cliniques en phase I. Cette thèse porte sur la synthèse et l'évaluation biologique de nouveaux allocolchicinoïdes, composés analogues du N-acétylcolchinol. Dans un premier temps, une nouvelle voie de synthèse permettant l’accès, de façon racémique, au squelette dibenzoxépine de ces molécules a été mise au point. Elle comprend notamment trois étapes clés, un couplage de Suzuki-Miyaura, une addition de Grignard et une cyclodéshydratation effectuée en présence d’un acide de Brønsted. Par la suite, trois séries d’allocolchicinoïdes de structures variées, que ce soit au niveau du cycle médian oxépine ou des substituants présents sur les noyaux benzéniques, ont été synthétisées. L’activité sur tubuline de la plupart de ces molécules a été évaluée, ce qui a conduit à une rationalisation des relations structure-activité
N-acetylcolchinol is a semi-synthetic inhibitor of the polymerization of tubulin into microtubules, that showed promising activity as vascular-disrupting agent. However, its toxicity evidenced in phase I clinical trials precluded its further development. This thesis describes the synthesis and biological evaluation of new allocolchicinoids, analogues of N-acetylcolchinol.A racemic synthesis of the dibenzoxepine framework of these compounds was first established. A Suzuki-Miyaura coupling, a Grignard addition and a Brønsted acid-mediated cyclodehydration constituted the key steps of the strategy. Then, three different series of dibenzoxepines have been synthesized, which differ by the nature of the substituent on the oxepine medium ring and on phenyl rings. These new dibenzoxepines were tested against the inhibition of microtubule assembly, leading to a structure-activity relationship study
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Kornfeld, Sophie. "La vascularisation tumorale : une cible thérapeutique des acides gras polyinsaturés n-3 pour sensibiliser les tumeurs mammaires aux traitements anticancéreux." Thesis, Tours, 2011. http://www.theses.fr/2011TOUR3308/document.

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Les acides gras polyinsaturés n-3 (acide docosahexaènoïque, DHA et acide eicosapentaènoïque, EPA) sensibilisent les tumeurs mammaires aux agents anticancéreux. Cette sensibilisation implique la régulation de la vascularisation tumorale. En effet, un régime nutritionnel EPA/DHA, associé à une chimiothérapie par le Docétaxel (Taxane) diminue la quantité de vascularisation (effet anti-angiogénique). Une amélioration de la qualité vasculaire est aussi observée par une diminution de la pression du liquide interstitiel, paramètre décrit comme un frein à la délivrance des drogues dans les tumeurs. Cette diminution est associée à une extravasation plus importante du bleu d’Evans, suggérant une meilleure distribution des agents anticancéreux au sein des tumeurs. L’effet antiangiogénique du DHA implique une diminution de la voie de signalisation VEGF/eNOS/NO. Ainsi, l’activation de la NO synthase endothéliale (eNOS) est diminuée dans des cellules endothéliales en culture et dans les tumeurs mammaires. Nos résultats suggèrent que l’apport d'acides gras EPA/DHA aux patients au cours de la chimiothérapie pourrait être une nouvelle approche thérapeutique pour normaliser la vascularisation tumorale et améliorer l’efficacité des traitements anticancéreux
Polyunsaturated fatty acids n-3 (docosahexaenoic acid, DHA and eicosapentaenoic acid, EPA) sensitize mammary tumors to anticancer drugs. This sensitization involves the regulation of tumor vasculature. Indeed, a nutritional diet with EPA / DHA, in combination with taxane chemotherapy (docetaxel) decreases the vascular quantity (anti-angiogenic effect). An improvement of vascular quality is also observed by a decrease of interstitial fluid pressure, a parameter described as a barrier to drug delivery in tumors. This decrease improves extravasation of Evans blue, suggesting a better distribution of anticancer agents in tumors. The antiangiogenic effect of DHA involves a decrease of signaling pathway VEGF / eNOS / NO. Thus, activation of endothelial NO synthase (eNOS) is decreased in endothelial cells in culture and in mammary tumors. Our results suggest that intake of fatty acids EPA / DHA to patients during chemotherapy could be a new therapeutic approach to normalize tumor vasculature and improve the efficacy of cancer treatments
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Maurin, Mathieu. "Nanoparticules fluorescentes à base de Pluronic : application à l'imagerie intravitale de la vascularisation par microscopie à deux photons et au transport de molécules." Phd thesis, Université de Grenoble, 2011. http://tel.archives-ouvertes.fr/tel-00593468.

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Les chromophores classiques ne sont pas toujours efficaces en absorption à deux photons. Leur faible efficacité nécessite l'utilisation de fortes puissances laser et de grandes concentrations en colorants. Dans ce sens, la microscopie à deux photons in vivo requière le développement de nouvelles stratégies de marquage utilisant des chromophores spécialement dédiés à la microscopie à deux photons. Dans le cadre de collaborations avec des chimistes spécialisés dans la synthèse de molécules à forte section efficace d'absorption à deux photons, différents chromophores ont été synthétisés. Ces molécules organiques sont souvent hydrophobes et ne sont pas utilisables directement pour les applications en biologie. Le travail effectuer ici a consisté à encapsuler ces molécules dans des micelles de copolymères biocompatibles, les Pluronic. Les Pluronic sont des matériaux pouvant s'auto assembler en milieu aqueux sous forme de micelles et permettent de solubiliser des composés hydrophobes. Cette stratégie est déjà utilisé pour permettre de transporter différents composés hydrophobes dans les organismes vivants et a été utilisée ici pour transporter des chromophores ultrasensibles à deux photons dans le sang de manière à imager la vascularisation in vivo.
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CHIU, PO-SHENG, and 邱柏升. "Photoacoustic Imaging of Tumor Vascularization on Nude Mice." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/54558119038172030168.

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碩士
國立陽明大學
醫學工程研究所
95
Abstract The accumulated evidence show out that angiogenesis plays an important role in tumor cell proliferation. At the early development of the tumor, the tumor cells are often balanced between positive and negative regulators of angiogenesis, and this condition persist for a certain period of time, from months to years in human body, in a vascular quiescent status. When the balance was broken, the tumor starts to grow rapidly. Tumor neovascularization relates to the tumor growth, invading, and metastasis. On the other hand, blocking the blood supply of the tumors is one of the most efficient methods for tumor treatment. Therefore, it is an important issue to detect/monitoring tumor neovascularization in clinics, in order to detect the tumor as well as conduct tumor therapy. In addition, the monitoring of tumor neovascularization has been applied for estimating the prognosis of tumors and the diagnosis of cancer therapy in clinics. Photoacoustic imaging (PAI) has been shown to have higher spatial resolution and optical contrast than conventional ultrasound imaging due to the high contrast absorption of blood on green and near infrared (NIR) light. We conducted in-vivo PAI of tumor blood vessels – monitoring 3-24 days development of tumor after the implant of the tumor cells on the back of nude mice. We observed that the PAI signals increased with the blood vessel developments and the growth of the tumor. This study shows that PAI is suitable for monitoring of the development of blood vessels near the superficial tumors. These results demonstrate the usefulness of the PAI for tumor angiogenesis monitoring in small animals. After detecting tumor angiogenesis, we will lose the relationship between tumor structure and tumor visualization. Therefore, we fused ultrasound image with PAI for clearly identifying vessels position of the tumor. Furthermore, clinical ultrasound Doppler was used to verify vessel exists which were detected by PAI.
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Books on the topic "Tumor vascularization"

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Ribatti, Domenico, and Francesco Pezzella. Tumor Vascularization. Elsevier Science & Technology Books, 2020.

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Ribatti, Domenico, and Francesco Pezzella. Tumor Vascularization. Elsevier Science & Technology, 2020.

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Tumor Vascularization. Elsevier, 2020. http://dx.doi.org/10.1016/c2019-0-00115-x.

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Book chapters on the topic "Tumor vascularization"

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Hatzikirou, Haralambos, Arnaud Chauvière, John Lowengrub, J. De Groot, and Vittorio Cristini. "Effect of Vascularization on Glioma Tumor Growth." In Modeling Tumor Vasculature, 237–59. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4614-0052-3_10.

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Gullino, Pietro M. "Angiogenesis, Tumor Vascularization, and Potential Interference with Tumor Growth." In Biological Responses in Cancer, 1–20. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-1236-9_1.

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Zhang, Huafeng, and Gregg L. Semenza. "von Hippel-Lindau Tumor Suppressor, Hypoxia-Inducible Factor-1, and Tumor Vascularization." In Cancer Genome and Tumor Microenvironment, 119–32. New York, NY: Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-0711-0_6.

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Ribba, Benjamin, Floriane Lignet, and Luigi Preziosi. "Computational Models of Vascularization and Therapy in Tumor Growth." In Mechanical and Chemical Signaling in Angiogenesis, 227–46. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-642-30856-7_11.

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Pietras, Alexander, A. Sofie Johnsson, and Sven Påhlman. "The HIF-2α-Driven Pseudo-Hypoxic Phenotype in Tumor Aggressiveness, Differentiation, and Vascularization." In Current Topics in Microbiology and Immunology, 1–20. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/82_2010_72.

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Seitz, Rüdiger J., and Wolfgang Wechsler. "Vascularization of human cerebral gliomas: a lectin-cytochemical and morphometric study." In Biology of Brain Tumour, 131–37. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2297-9_18.

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Delides, C. S., Efi Protopapa, and L. Revesz. "Vascularization and Curability of Certain Human Tumors, a Practical Approach." In Angiogenesis in Health and Disease, 369. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3358-0_41.

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Ranieri, Girolamo, Annamaria Catino, Vittorio Mattioli, Vito Fazio, Gennaro Gadaleta Caldarola, and Cosmo Damiano Gadaleta. "Targeting Tumour Vascularization from Bench to Bedside: Suggestions for Combination with Hyperthermia." In Cancer Microenvironment and Therapeutic Implications, 203–19. Dordrecht: Springer Netherlands, 2009. http://dx.doi.org/10.1007/978-1-4020-9576-4_11.

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Konerding, M. A., E. Fait, A. Gaumann, Ch Dimitropoulou, and W. Malkusch. "The Vascularization of Experimental and Human Primary Tumors: Comparative Morphometric and Morphologic Studies." In Angiogenesis, 429–47. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4757-9185-3_40.

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Ribatti, Domenico, and Francesco Pezzella. "Sprouting and nonsprouting angiogenesis in tumors." In Tumor Vascularization, 1–13. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-819494-2.00001-8.

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Conference papers on the topic "Tumor vascularization"

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Chiu, Bson, Shu-Wei Huang, and Huihua Kenny Chiang. "Photoacoustic imaging of tumor vascularization on nude mice." In Biomedical Optics (BiOS) 2007, edited by Alexander A. Oraevsky and Lihong V. Wang. SPIE, 2007. http://dx.doi.org/10.1117/12.700255.

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Wiemker, Rafael, Thomas Bülow, Roland Opfer, Sven Kabus, and Ekta Dharaiya. "Intuitive parameter-free visualization of tumor vascularization using rotating connectivity projections." In Medical Imaging, edited by Xiaoping P. Hu and Anne V. Clough. SPIE, 2008. http://dx.doi.org/10.1117/12.765333.

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Burrell, Kelly, Shahrzad Jalali, Soroush Larjani, Richard Hill, and Gelareh Zadeh. "Abstract 3475: Tumor stage dependent contribution of bone marrow derived cells to tumor neo-vascularization." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-3475.

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Dencks, Stefanie, Tatjana Opacic, Marion Piepenbrock, Fabian Kiessling, and Georg Schmitz. "Determination of adequate measurement times for super-resolution characterization of tumor vascularization." In 2017 IEEE International Ultrasonics Symposium (IUS). IEEE, 2017. http://dx.doi.org/10.1109/ultsym.2017.8092020.

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Dencks, Stefanie, Marion Piepenbrock, Georg Schmitz, Tatjana Opacic, and Fabian Kiessling. "Determination of adequate measurement times for super-resolution characterization of tumor vascularization." In 2017 IEEE International Ultrasonics Symposium (IUS). IEEE, 2017. http://dx.doi.org/10.1109/ultsym.2017.8092351.

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Heijblom, Michelle, Daniele Piras, Frank M. van den Engh, Joost M. Klaase, Mariël Brinkhuis, Wiendelt Steenbergen, and Srirang Manohar. "Photoacoustic imaging of breast tumor vascularization: a comparison with MRI and histopathology." In European Conferences on Biomedical Optics, edited by Vasilis Ntziachristos and Charles P. Lin. SPIE, 2013. http://dx.doi.org/10.1117/12.2033305.

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Nakamura, Mitsuhiro, Satoru Kyo, Xivzhi Zhang, Masahiro Takakura, Yoshiko Maida, Yasunari Mizumoto, Yukiko Bono, Toshiyuki Sasagawa, and Masaki Inoue. "Abstract 5199: Tumor vascularization by endothelial differentiation of endometrial cancer stem cells." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5199.

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Shakhar, Guy, Yoav Manaster, and Tali Feferman. "Abstract PR04: Intravital imaging of cytolytic activity reveals how poor tumor vascularization and hypoxia limit tumor rejection." In Abstracts: AACR Special Conference: Tumor Angiogenesis and Vascular Normalization: Bench to Bedside to Biomarkers; March 5-8, 2015; Orlando, FL. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-8514.tumang15-pr04.

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Bucharskaya, A. B., G. N. Maslyakova, M. L. Chekhonatskaya, N. B. Zakharova, G. S. Terentyuk, N. A. Navolokin, B. N. Khlebtsov, et al. "The evaluation of tumor vascularization as a prognostic factor of plasmonic phothothermal therapy efficiency." In 2020 International Conference Laser Optics (ICLO). IEEE, 2020. http://dx.doi.org/10.1109/iclo48556.2020.9285670.

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Nakamura, Mitsuhiro, Xiuzhi Zhang, Masahiro Takakura, Yoshiko Maida, Yasunari Mizumoto, Yukiko Bono, Toshiyuki Sasagawa, and Satoru Kyo. "Abstract 4892: Hypoxia enhances tumor vascularization by endothelial differentiation of endometrial cancer stem cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-4892.

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