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Books on the topic 'Type 2/genetics'

Author: Grafiati

Published: 2 June 2025

Last updated: 31 July 2025

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1

Gloyn, Anna L., and Mark I. McCarthy. Genetics in diabetes: Type 2 diabetes and related traits. Karger, 2014.

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2

Florez, Jose C., ed. The Genetics of Type 2 Diabetes and Related Traits. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-01574-3.

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3

A, Hitman Graham, ed. Type 2 diabetes: Prediction and prevention. J. Wiley, 1999.

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4

1951-, Nelkin Barry D., ed. Genetic mechanisms in multiple endocrine neoplasia type 2. Chapman & Hall, 1996.

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5

Watson, Carolyn Joyce. Molecular genetic investigations in Type 2 neurofibromatosis. University of Manchester, 1996.

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6

Nelkin, Barry D. Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2. Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-662-21948-5.

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7

G, Leslie R. D., ed. Causes of diabetes: Genetic and environmental factors. J. Wiley, 1993.

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8

Milan, Potmesil, and Kohn Kurt W, eds. DNA topoisomerases in cancer. Oxford University Press, 1991.

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9

Flavin, Nora. Cloning and characterisation of the bovine activin receptor type II gene (ActRII): Its localisation to chromosome 2 (BTA2) by somatic cell genetic analysis and the genotyping of an associated microsatelltie UCD2. University College Dublin, 1996.

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10

S, Liju Samuel. Early Onset Type 2 Diabetes Genetics Associations. Quadry, Fatima, 2023.

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11

Hitman, Graham A. Type 2 Diabetes: Prediction and Prevention. Wiley & Sons, Incorporated, John, 2007.

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12

Hitman, Graham A. Type 2 Diabetes: Prediction and Prevention. Wiley & Sons, Incorporated, John, 2000.

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13

Florez, Jose C. Genetics of Type 2 Diabetes and Related Traits: Biology, Physiology and Translation. Springer London, Limited, 2016.

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14

Florez, Jose C. The Genetics of Type 2 Diabetes and Related Traits: Biology, Physiology and Translation. Springer, 2018.

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15

Florez, Jose C. The Genetics of Type 2 Diabetes and Related Traits: Biology, Physiology and Translation. Springer, 2016.

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16

Kan, Carol, and Ma-Li Wong. Genetics. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198789284.003.0004.

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An association between type 2 diabetes mellitus (T2DM) and depression has been reported in epidemiological studies. Finding a genetic overlap between T2DM and depression will provide evidence to support a common biological pathway to both disorders. Genetic correlations observed from twin studies indicate that a small magnitude of the variance in liability can be attributed to genetic factors. However, no genetic overlap has been observed between T2DM and depression in genome-wide association studies using both the polygenic score and the linkage disequilibrium score regression approaches. Cla

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17

Nielsen, David A., Dmitri Proudnikov, and Mary Jeanne Kreek. The Genetics of Impulsivity. Edited by Jon E. Grant and Marc N. Potenza. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780195389715.013.0080.

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Impulsivity is a complex trait that varies across healthy individuals, although when excessive, it is generally regarded as dysfunctional. Impulsive behavior may lead to initiation of drug addiction that interferes with inhibitory controls, which may in turn result in facilitation of the individual’s impulsive acts. Although environmental factors play a considerable role in impulsive behavior, a body of evidence collected in twin studies suggests that about 45% of the variance in impulsivity is accounted for by genetic factors. Genetic variants studied in association with impulsivity include t

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18

Genetic analysis of complex traits: Proceedings of Genetic Analysis Workshop 5, held at Chantilly, France, September 2-5, 1987. Liss, 1989.

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19

(Editor), F. M. Matschinsky, ed. Molecular Pathogenesis of Modys: American Diabetes Association Symposium. Scottsdale, Ariz., Usa, November 6-8, 1998 (Frontiers in Diabetes). S. Karger Publishers (USA), 2000.

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20

Nelkin, Barry D. Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2. Springer London, Limited, 2013.

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21

Nelkin, Barry D. Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2. Springer Berlin / Heidelberg, 2013.

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22

Ismail, Khalida, Andreas Barthel, Stefan R. Bornstein, and Julio Licinio, eds. Depression and Type 2 Diabetes. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198789284.001.0001.

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Type 2 diabetes is predicted to affect between 10% and 25% of the world population in the next 20 years. Depression is a common comorbid condition in those affected with type 2 diabetes, and the combination of these conditions is associated with a poorer prognosis, including earlier mortality. Genetic and epigenetic predisposition and overlap of risk factors related to our modern lifestyle seem to drive the shared biology of diabetes and depression. This book aims to provide an understanding of the sequelae of events leading to the frequent comorbidity of diabetes and depression. This book pro

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23

Sayer, John A. Autosomal dominant tubule-interstitial kidney disease, including medullary cystic disease. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0318_update_001.

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The term medullary cystic kidney disease (MCKD) describes a group of autosomal dominantly inherited renal disorders. The term MCKD is used interchangeably with other terms, most commonly autosomal dominant interstitial kidney disease, and now may be distinguished using a molecular genetic diagnosis into at least three types. These include MCKD type 1, MCKD type 2 (also known now as uromodulin-associated kidney disease), and REN-associated kidney disease. Each of these types have phenotypic overlap but with a few distinguishing features. MCKD typically leads to end-stage renal failure between 3

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24

Neurofibromatosis. Exon Publications, 2024. https://doi.org/10.36255/neurofibromatosis.

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Neurofibromatosis is a group of genetic disorders that cause tumors to form on nerves throughout the body, leading to a range of physical, neurological, and cosmetic challenges. This article provides a comprehensive guide to understanding Neurofibromatosis, its causes, and how it is managed. It begins by explaining the condition and its three main types: Neurofibromatosis type 1 (NF1), Neurofibromatosis type 2 (NF2), and Schwannomatosis, each with unique symptoms and complications. The article explores the genetic basis of the disorder, detailing the role of mutations in the NF1, NF2, SMARCB1,

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25

Struys, Eduard A., Marjo S. van der Knaap, and Gajja S. Salomons. 2-Hydroxyglutaric Acidurias. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0022.

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The group of congenital diseases related to 2-hydroxyglutaric acid metabolism consists of five disease entities: D-2-hydroxyglutaric aciduria Type I (D2HGA-I), D-2-hydroxyglutaric aciduria Type II (D2HGA-II), metaphyseal chondromatosis with D-2-hydroxyglutaric aciduria (MC-HGA), L-2-hydroxyglutaric aciduria (L2HGA), and combined D,L-2-hydroxyglutaric aciduria (DL2HGA). Overall, the clinical presentation varies from early onset with mostly lethal neonatal encephalopathy in DL2HGA to slowly progressive mental and motor delay with white matter abnormalities, with an insidious and sometimes adult

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26

Hebenstreit, Daniel. Signal Transduction and Gene Activation by STAT6: Gene Regulation Processes Involved in Type-2 Immune Polarisation. Simon & Schuster, 2008.

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27

Dalbeth, Nicola. Genetic basis of hyperuricaemia and gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198748311.003.0004.

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Owing to the different means of ascertaining prevalence between studies, it is difficult to compare prevalence across countries. Country-specific studies that collect data with the same methodology show that the prevalence of gout is increasing. Factors that influence the prevalence of gout are inherited genetic factors and environmental exposures. Some foods that increase serum urate levels and trigger acute gouty arthritis are risk factors—red meat and beer are the best established, but seafood and sugar-sweetened beverages also increase serum urate levels and are strong anecdotal triggers o

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28

Rabinow, Paul. French DNA: Trouble in Purgatory. University Of Chicago Press, 2002.

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29

Rabinow, Paul. French DNA: Trouble in Purgatory. University Of Chicago Press, 1999.

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30

Sanai, Nader. Low-Grade Gliomas. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696696.003.0027.

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Low grade gliomas encompass multiple histologic diagnoses of primary brain tumors, most commonly grade 2 oligodendroglioma and grade 2 astrocytoma. This chapter presents a case of a patient with a left temporal low grade glioma who presented with seizures, which are a common presenting symptom for this tumor type. Management of patients with a newly diagnosed low grade glioma typically begins with maximal safe surgical resection for surgically accessible tumors. Surgical planning may involve functional imaging such as with fMRI. Genetic and molecular markers help distinguish subtypes of low gr

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31

Understanding Type 2 Diabetes: Fewer Highs, Fewer Lows, Better Health. EMPower, 2017.

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32

Understanding Type 2 Diabetes: Fewer Highs Fewer Lows Better Health. ReadHowYouWant.com, Limited, 2013.

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33

Understanding Type 2 Diabetes: Fewer Highs, Fewer Lows, Better Health. Exisle Publishing Pty Limited, 2014.

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34

Understanding Type 2 Diabetes: Fewer Highs, Fewer Lows, Better Health. Exisle Publishing Limited, 2013.

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35

Understanding Type 2 Diabetes: Fewer Highs, Fewer Lows, Better Health. Exisle Publishing Pty Limited, 2017.

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36

Sandler, Karen. Eternity [3 1/2 Disk, HTML]. Hard Shell Word Factory, 1998.

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37

Diabetes. Exon Publication, 2024. http://dx.doi.org/10.36255/diabetes.

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Diabetes is a comprehensive guide that explains the causes, symptoms, diagnosis, and treatment options for this common metabolic condition. The article starts with an abstract and introduction, offering a detailed overview of what diabetes is and how it affects the body’s ability to regulate blood sugar. It covers the three main types of diabetes—Type 1, Type 2, and gestational diabetes—providing insights into the risk factors, genetic influences, and environmental triggers for each. The article also discusses how diabetes is diagnosed and the importance of managing the condition through medic

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38

Taylor, Jennie W., and Scott R. Plotkin. Familial CNS Tumor Syndromes. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0135.

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Neurofibromatosis type 2 (NF2) is a genetic disorder caused by constitutional mutations in the NF2 tumor-suppressor gene. Bilateral vestibular schwannomas are the hallmark of the syndrome, though meningiomas, ependymomas, and other peripheral schwannomas are common. Inheritance is autosomal dominant and de novo mutations are found in about 50% of patients. Standard treatment for symptomatic tumors is surgery. Radiation therapy may be considered for progressive tumors that are not surgically accessible, but secondary cancers after radiation have been reported. Retrospective studies suggest that

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39

Gnudi, Luigi, Giorgio Gentile, and Piero Ruggenenti. The patient with diabetes mellitus. Edited by Giuseppe Remuzzi. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0149_update_001.

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About one third of patients with type 1 diabetes develop diabetic nephropathy long-term (usually not before at least 10 years of diabetes), though this proportion is falling as standards of care have risen. Nephropathy is strongly associated with other microvascular complications of diabetes, so that some degree of retinopathy is to be expected, and evidence of neuropathy is common. Patients with type 2 diabetes are equally susceptible, but this is an older group in which vascular disease and other pathologies are also more likely. The rise in type 2 diabetes accounts for diabetes being the mo

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40

Gestational Diabetes: Education for Patients and the Public. Exon Publications, 2025. https://doi.org/10.36255/gestational-diabetes.

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Gestational diabetes is a condition that develops during pregnancy when the body cannot regulate blood sugar levels properly. This article provides a detailed guide to help expecting mothers, families, and the general public understand gestational diabetes, its causes, symptoms, diagnosis, treatment, and management. The article begins with an introduction to the condition, explaining how hormonal changes during pregnancy can interfere with insulin function. It then explores the prevalence of gestational diabetes, highlighting risk factors such as obesity, genetics, and age, with references to

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41

Bhopal, Raj S. Epidemic of Cardiovascular Disease and Diabetes. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780198833246.001.0001.

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Coronary heart disease (CHD) and stroke, collectively cardiovascular disease (CVD), are caused by narrowing and blockage of the arteries supplying the heart and brain, respectively. In type 2 diabetes (DM<sub>2</sub>) insulin is insufficient to maintain normal blood glucose. South Asians have high susceptibility to these diseases. Drawing upon the scientific literature and discussions with 22 internationally recognized scholars, this book focuses on causal explanations and their implications for prevention and research. Genetically based hypotheses are considered together with the developmenta

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42

Roman, Eve, Alexandra Smith, and Lorelei Mucci. Leukemias. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190676827.003.0028.

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Leukemias are a diverse group of acute and chronic haematological malignancies, that account for 2% to 3% of cancers globally. Recent advances in molecular biology and therapy have transformed the landscape for several leukemia subtypes changing some, but by no means all, from rapidly fatal diseases to treatable conditions with a good prognosis. In general, however, this progress has not been matched by new aetiological insights. Albeit accounting for a relatively small proportion, genetic predisposition syndromes such as neurofibromatosis, Li-Fraumeni and Trisomy 21, have the biggest impact i

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43

Metabolic Syndrome: Education for Patients and the Public. Exon Publications, 2025. https://doi.org/10.36255/metabolic-syndrome-patient-public-education.

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Metabolic syndrome is a group of health conditions that happen together and increase the risk of heart disease, stroke, and type 2 diabetes. This article offers a complete explanation of what metabolic syndrome is, how it is diagnosed, and why it matters.It begins with a definition of the condition and moves into the five key risk factors: abdominal obesity, high blood pressure, high blood sugar, high triglycerides, and low HDL cholesterol. The article explains how these factors interact and why they are used together to identify the syndrome. It continues by discussing the causes of metabolic

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44

Dalbeth, Nicola. Gout. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0141.

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Gout is a common and treatable disorder of purine metabolism. Gout typically presents as recurrent self-limiting episodes of severe inflammatory arthritis affecting the foot. In the presence of persistent hyperuricaemia, tophi, chronic synovitis, and joint damage may develop. Diagnosis of gout is confirmed by identification of monosodium urate (MSU) crystals using polarizing light microscopy. Hyperuricaemia is the central biochemical cause of gout. Genetic variants in certain renal tubular urate transporters including SLC2A9 and ABCG2, and dietary factors including intake of high-purine meats

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45

Dalbeth, Nicola. Gout. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0141_update_003.

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Gout is a common and treatable disorder of purine metabolism. Gout typically presents as recurrent self-limiting episodes of severe inflammatory arthritis affecting the foot. In the presence of persistent hyperuricaemia, tophi, chronic synovitis, and joint damage may develop. Diagnosis of gout is confirmed by identification of monosodium urate (MSU) crystals using polarizing light microscopy. Hyperuricaemia is the central biochemical cause of gout. Genetic variants in certain renal tubular urate transporters including SLC2A9 and ABCG2, and dietary factors including intake of high-purine meats

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46

Waldek, Stephen. Fabry disease. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0335_update_001.

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Fabry disease is a rare X-linked disorder of glycosphingolipid metabolism caused by a deficiency of the lysosomal acid hydrolase enzyme, alpha-galactosidase A. The resulting accumulation of substrate, mostly globotriaosylceramide, leads to a progressive, multiorgan disease affecting predominantly the kidneys, skin, heart, and nervous system. It is one of over 50 lysosomal storage diseases. It is typically diagnosed in young men after many years of ‘acral pain’ syndrome, when the diagnosis is made through identification of characteristic abnormalities of skin, kidney or heart, or of other organ

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47

Kendrisic, Mirjana, and Borislava Pujic. Endocrine and autoimmune disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0047.

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Advanced maternal age and increasing numbers of women of childbearing age with endocrine and autoimmune disorders have become the challenge for both anaesthetists and obstetricians. Genetic studies have provided new insight into underlying causes of endocrine disorders and prenatal prediction of inheritance. The expression of endocrine disease may influence the interpretation of diagnostic laboratory testing during pregnancy. Better understanding of the pathophysiological mechanisms enables new therapeutic approaches which can compromise pregnancy outcome. Although only a small number of drugs

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48

Report of the African Regional Workshop on Development of a Global Information System for Farmed Types of Aquatic Genetic Resources (Incorporating a Review of Strategic Priorities for a Global Plan of Action), Addis Ababa, Ethiopia, 2–4 December 2019. FAO, 2021. http://dx.doi.org/10.4060/cb2343en.

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49

Atwood, Margaret Eleanor. Maddaddam #2: The Year Of The Flood - Little Brown. Virago Press Ltd, 2013.

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