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Relevant bibliographies by topics / Type 2 myocardial infarction / Dissertations / Theses

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Dissertations / Theses on the topic 'Type 2 myocardial infarction'

Author: Grafiati

Published: 28 September 2022

Last updated: 31 July 2025

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1

Adachi, Yuichiro. "Angiotensin 2 type 2 receptor deficiency exacerbates heart failure and reduces survival after acute myocardial infarction in mice." Kyoto University, 2006. http://hdl.handle.net/2433/144310.

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2

Putot, Alain. "Approche épidémiologique des infarctus du myocarde de type 2 : Etiologies, caractéristiques, traitements et pronostic." Thesis, Bourgogne Franche-Comté, 2020. http://www.theses.fr/2020UBFCI001.

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Introduction : L’infarctus du myocarde (IDM) de type 2 a été récemment défini par un déséquilibre entre apports et besoins du myocarde en oxygène, en l’absence de processus athérothrombotique. L’objectif de ce travail était d’en décrire les principales étiologies, ainsi que les caractéristiques épidémiologiques, cliniques et pronostiques.Méthode : Les données des patients présentant un IDM de type 2 ont été recueillies à partir de l’observatoire des infarctus de Cote d’Or (RICO). Un travail complémentaire a analysé les données rétrospectives du service des urgences du Centre Hospitalier Univer

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3

Reed, Grant William. "Associations Between Cardiac Troponin, Mechanism of Myocardial Injury, and Long-Term Mortality After Non-Cardiac Vascular Surgery." Case Western Reserve University School of Graduate Studies / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=case1491571890479287.

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4

Yao, Coffy-Akpolet. "Nouvelles approches épidémiologiques des infarctus du myocarde de type 2 : vers une prise en charge personnalisée." Electronic Thesis or Diss., Bourgogne Franche-Comté, 2023. http://www.theses.fr/2023UBFCI011.

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Introduction : l’infarctus du myocarde (IDM) de type 2, qui résulte d’un déséquilibre entre les apports et les besoins en oxygène du myocarde en l’absence d’événement athéro-thrombotique, est une entité mal connue. L’objectif de ce travail était de préciser certains aspects épidémiologiques et diagnostiques de l’IDM de type 2, notamment la place de la maladie coronaire, et d’évaluer la pertinence clinique et pronostique d’une nouvelle classification des IDM proposée par de LemosMéthodes : A partir de la base de données de l’Observatoire des Infarctus de la Côte d’Or (RICO), nous avons analysé

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5

Grzesiak, Aleksandra [Verfasser]. "Angiotensin II type 2 receptor stimulation : a novel options of therapeutic interference with the renin-angiotensin system in myocardial infarction? / Aleksandra Grzesiak." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2014. http://d-nb.info/1052529879/34.

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6

Lauer, Dilyara [Verfasser]. "Prevention of cardiac remodeling after experimental myocardial infarction. Role of the angiotensin II type 2 receptor stimulation and modulation of MMP/TIMP axis / Dilyara Lauer." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2016. http://d-nb.info/1119803527/34.

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7

Davidson, Melissa Anne. "A Pharmacovigilance Approach for Assessing Cardiovascular, Osteological, and Carcinogenic Risk Associated with Thiazolidinedione Drugs Used in the Treatment of Type 2 Diabetes Mellitus." Thesis, Université d'Ottawa / University of Ottawa, 2018. http://hdl.handle.net/10393/38062.

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Diabetes is a chronic and debilitating disease that affects nearly half a billion people worldwide with the vast majority of diabetics suffering from Type 2 diabetes mellitus (T2DM), a disease characterized by insulin insensitivity that often requires pharmacotherapy to effectively maintain target blood sugar levels. The thiazolidinedione (TZD) class of drugs consists of oral hypoglycaemic agents used alone or in combination with other antidiabetic drugs to treat T2DM. The drugs within this class, which include rosiglitazone and pioglitazone, were originally heralded as providing novel first a

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8

Wu, Chiung-Jung. "Promoting self-management for patients with type 2 diabetes following a critical cardiac event." Thesis, Queensland University of Technology, 2007. https://eprints.qut.edu.au/16465/1/Chiung-Jung_Wu_Thesis.pdf.

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Type 2 diabetes is a global health problem. Evidence indicates that type 2 diabetes can lead to serious complications, such as a cardiac event, which usually require critical nursing care. Patients with type 2 diabetes and with a history of cardiac disease are at greater risk of a further cardiac event requiring readmission to hospital. Evidence indicates that improved diabetes management assists patients with type 2 diabetes to manage their condition efficiently, reduces risks of a further cardiac event, and therefore reduces hospitalisations. However, there is limited information foun

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9

Wu, Chiung-Jung. "Promoting self-management for patients with type 2 diabetes following a critical cardiac event." Queensland University of Technology, 2007. http://eprints.qut.edu.au/16465/.

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Type 2 diabetes is a global health problem. Evidence indicates that type 2 diabetes can lead to serious complications, such as a cardiac event, which usually require critical nursing care. Patients with type 2 diabetes and with a history of cardiac disease are at greater risk of a further cardiac event requiring readmission to hospital. Evidence indicates that improved diabetes management assists patients with type 2 diabetes to manage their condition efficiently, reduces risks of a further cardiac event, and therefore reduces hospitalisations. However, there is limited information foun

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10

Curato, Caterina. "Identification of a non-cytotoxic and IL-10- producing CD8+AT2R+ T lymphocyte population in response to ischemic heart injury." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2011. http://dx.doi.org/10.18452/16362.

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Neuere Untersuchungen legen eine kardioprotektive Rolle für den Angiotensin AT2-Rezeptor nahe, welcher die Postinfarkt-Entzündungsreaktion vermindert, wobei der zelluläre Mechanismus noch wenig verstanden ist. Das Ziel dieser Arbeit war es deshalb, die potentielle Rolle des AT2-Rezeptors in der zellulären Immunantwort auf ischemische Herzverletzungen zu ergründen. Sieben Tage nach myokardialem Infarkt in Ratten wurde der AT2-Rezeptor mittels Immunfluoreszenzfärbung von Gewebeschnitten in einer CD8 T-Zellfraktion detektiert, die das Peri-Infarkt-Myokard infiltiert hatte. Wir haben eine Methode

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11

Jacquin, Laurent. "Déséquilibre d’oxygénation et lésions myocardiques aiguës : approche clinique en service d’accueil des urgences." Thesis, Lyon, 2021. https://n2t.net/ark:/47881/m6736qrr.

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Dans ce travail de thèse, nous nous sommes intéressés en première partie aux critères de déséquilibre d’oxygénation impliqués dans la survenue d’un infarctus de type 2. Nous avons exploré chez 610 patients l’association entre les paramètres de ces critères et la survenue de lésions myocardiques aiguës, et d’infarctus de type 2, ainsi que la relation entre ces paramètres et l’extension de l’atteinte du myocarde. Nos résultats ne montraient pas de lien entre l’amplitude du déséquilibre d’oxygénation et la survenue de lésions myocardiques aiguës. Il n’y avait également pas de corrélation avec l’i

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12

Herrera, Comoglio Nelly. "Évenements cardiovasculaires majeurs et mortalité en patients traités avec hypoglycémiants non insuliniques." Thesis, Bordeaux, 2019. http://www.theses.fr/2019BORD0355.

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Le diabète mellitus Type 2 (DMT2) est une maladie chronique et progressive causée par multiples facteurs. Plus de 422 millions de personnes dans le monde ont du diabète; la maladie a un profond impact social et économique. La maladie cardiovasculaire est la cause principale de la morbidité et la mortalité chez les patients diabétiques, qui ont des taux de mortalité plus élevées que la population non-diabétique. La définition de la DMT2 est basée sur ses manifestations métaboliques – surtout celles de glucose sanguin – qui servent comme marqueurs du contrôle et de l’évolution de la maladie. Cep

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13

Cediel, Calderón Germán Eduardo. "Papel de la troponina i como biomarcador pronóstico en pacientes atendidos en los servicios de urgencias sin diagnóstico de síndrome coronario agudo." Doctoral thesis, Universitat Rovira i Virgili, 2017. http://hdl.handle.net/10803/435687.

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En els últims anys, l'ús generalitzat de la troponina en els serveis d'urgències, ha permès la seva detecció en pacients amb diagnòstics clínics diferents a la síndrome coronària aguda (SCA). L'objectiu d'aquesta tesi va ser establir el valor pronòstic d'una troponina I (cTnI) elevada en el seguiment dels pacients atesos en un servei d'urgències i que no són diagnosticats de SCA, estudiant també, el seu valor pronòstic en els pacients donats d'alta directament des d´urgències. També ens plantegem identificar aquells pacients amb diagnòstic d'infart de miocardi (IM) tipus 2 i injúria miocàrdic

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14

McSweeney, Sara Jane. "11β-hydroxysteroid dehydrogenase type 1 : a new therapeutic target post-myocardial infarction?" Thesis, University of Edinburgh, 2010. http://hdl.handle.net/1842/4833.

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Glucocorticoids can reduce infarct size when given immediately after myocardial infarction (MI) but are detrimental when administration is continued into the post-infarct healing phase. A number of experimental studies have shown that reduction of infarct expansion by enhancing blood supply to the infarct border reduces remodelling and improves heart function post-MI. Previous experiments from this laboratory have shown that mice unable to locally regenerate corticosterone due to deficiency in 11β-hydroxysteroid dehydrogenase type 1 (11HSD1) have an enhanced angiogenic response during myocardi

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15

Oranzie, Marlon. "Electrochemical Aptasensing of B-Type Natriuretic Peptide-A Biomarker for Myocardial Infarction." University of the Western Cape, 2019. http://hdl.handle.net/11394/7707.

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>Magister Scientiae - MSc<br>infarction (MI) affects many parts of the western world and in South Africa alone it is estimated that MI is responsible for 1 in 6 deaths (17.3%). Traditional diagnostic methods for MI include an electrocardiograms and blood tests. The problem with these diagnostic methods are that they are time consuming, require large sample volumes, expensive equipment and complicated machinery. To achieve early detection of MI the discovery of specific, sensitive and reliable biomarkers are required. Brain natriuretic peptide (BNP) has been identified as a reliable biomarker f

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16

Gianelli, Dorothy M. "Comparison study between Type A and Type B individuals' compliance to cardiac rehabilitation following myocardial infarction /." Staten Island, N.Y. : [s.n.], 1987. http://library.wagner.edu/theses/nursing/1987/thesis_nur_1987_giane_compa.pdf.

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17

Chan, Cangel Pui Yee. "A superior early myocardial infarction marker : human heart-type fatty acid-binding protein /." View Abstract or Full-Text, 2002. http://library.ust.hk/cgi/db/thesis.pl?CHEM%202002%20CHAN.

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Thesis (Ph. D.)--Hong Kong University of Science and Technology, 2002.<br>Includes bibliographical references (leaves 139-166). Also available in electronic version. Access restricted to campus users.

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18

Groves, Robert W. "A study of the effect of cotreatment of taprostene (CG 4203), a novel stabilized prostacyclin analogue, with saruplase, a gene technologically produced unglycosylated single chain urokinase-type plasminogen activator (r-scuPA), in thrombolysis in vivo." Thesis, University of Bath, 1990. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.278267.

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19

Luther, Daniel J. "The role of type VI collagen in cardiac remodeling following myocardial infarction in mice." Thesis, Kent State University, 2014. http://pqdtopen.proquest.com/#viewpdf?dispub=3618869.

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<p> Cardiac remodeling is a dynamic process largely propagated by cardiac fibroblasts (CFs), the critical mediators of wound repair. Following myocardial infarction (MI), this process is accelerated resulting in aberrant structural changes to the heart. Investigation into the fibrotic responses of the heart to injury have focused on collagens type I and III however, we have uncovered a novel role for type VI collagen (Col6). Here, we report the effects of the deletion of Col6 from the myocardium during post-MI wound repair and demonstrate that <i>Col6<sup>-/-</sup></i> mice are resistant to

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20

Luther, Daniel J. "The Role of Type VI Collagen In Cardiac Remodeling Following Myocardial Infarction In Mice." Kent State University / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=kent1376067347.

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21

Bandyopadhyay, Somnath. "Changes in the mouse left ventricular transcriptome after myocardial infarction." Laramie, Wyo. : [University of Wyoming], 2006. http://proquest.umi.com/pqdweb?did=1212778701&sid=1&Fmt=2&clientId=18949&RQT=309&VName=PQD.

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22

Keele, Jacque Anne. "Regulation of arginine metabolism following acute myocardial infarction in mice." Laramie, Wyo. : University of Wyoming, 2008. http://proquest.umi.com/pqdweb?did=1707917171&sid=1&Fmt=2&clientId=18949&RQT=309&VName=PQD.

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23

Miwa, Senri. "Spaciotemporal alteration of 8-hydroxy-2'-deoxyguanosine levels in cardiomyocytes after myocardial infarction in rats." Kyoto University, 2002. http://hdl.handle.net/2433/149330.

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24

Stevens, Victoria E. "Personality type and ways of coping : a study of female spouses of post myocardial infarction patients /." Access Digital Full Text version, 1992. http://pocketknowledge.tc.columbia.edu/home.php/bybib/11228313.

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Thesis (Ed.D.)--Teachers College, Columbia University, 1992.<br>Includes tables. Typescript; issued also on microfilm. Sponsor: Marilyn Rawnsley. Dissertation Committee: John P. Allegrante. Includes bibliographical references (leaves 135-141).

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25

Morrice, K. W. "An investigation into the potential utility of Intermedin (adrenomedullin-2) as a biomarker of myocardial ischaemia or infarction." Thesis, Queen's University Belfast, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.580099.

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Introduction: Elevated blood levels of Cardiac Troponin (cTnT) are the gold standard for diagnosis of acute coronary syndrome (ACS). However cTnT is not detectable for 3-4 hours after symptom onset; an early marker of myocardial ischaemia is therefore desirable. The peptide Calcitonin Gene-Related Peptide (CGRP) is released from nerve endings during myocardial ischaemia and has cardioprotective effects. Intermedin (IMD), from the same family, is elevated in myocardial infarction and oxidative stress in animal studies. Methods: I enrolled 81 patients with chest pain, suspected to have acute cor

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26

Dutra, Luiz Antonio [UNESP]. "Planejamento, síntese e avaliação farmacológica de novos compostos 1,2,5-oxadiazol-2-n-óxido úteis como preventivos de aterotrombose." Universidade Estadual Paulista (UNESP), 2013. http://hdl.handle.net/11449/108423.

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Made available in DSpace on 2014-08-13T14:50:36Z (GMT). No. of bitstreams: 0 Previous issue date: 2013-11-29Bitstream added on 2014-08-13T18:01:10Z : No. of bitstreams: 1 000736321_20141129.pdf: 359412 bytes, checksum: 2b097cc17fda8e667e01f3cd49b99769 (MD5) Bitstreams deleted on 2014-12-01T14:49:47Z: 000736321_20141129.pdf,Bitstream added on 2014-12-01T14:50:25Z : No. of bitstreams: 1 000736321.pdf: 4184687 bytes, checksum: 6844804819f4dc3ee8faded627f4a83d (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>Doenças cardiovasculares como infarto do miocárdio e acidente

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Dutra, Luiz Antonio. "Planejamento, síntese e avaliação farmacológica de novos compostos 1,2,5-oxadiazol-2-n-óxido úteis como preventivos de aterotrombose /." Araraquara, 2013. http://hdl.handle.net/11449/108423.

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Orientador: Jean Leandro dos Santos<br>Banca: Leoberto Costa Tavares<br>Banca: Cíntia Duarte de Freitas Milagre<br>Resumo: Doenças cardiovasculares como infarto do miocárdio e acidente vascular encefálico ainda representam a principal causa de morte no Brasil. A aterosclerose é uma doença progressiva e silenciosa classificada como fator de risco para o desenvolvimento de doenças cardiovasculares. É caracterizada pelo aumento dos níveis de colesterol no plasma os quais são oxidados por radicais livres originando a lipoproteína de baixa densidade oxidada (LDLox). A fagocitose de LDLox por macróf

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28

Bena, Stefania. "Expression and function of the formyl peptide receptor 2 in experimental myocardial infarct." Thesis, Queen Mary, University of London, 2014. http://qmro.qmul.ac.uk/xmlui/handle/123456789/7905.

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In Acute Myocardial Infarction (AMI), inflammation is a prerequisite for healing but it can paradoxically extend tissue injury; hence it needs to be modulated. Here, we investigated the role of the pro resolving GPCR FPR2/ALX and its agonist Annexin A1 (AnxA1) in AMI using mice lacking of the Fpr2/3 genes and with an in-frame GFP gene ‘knocked-in’. We developed protocols aimed to determine GFP expression as an indication of Fpr2 gene activity. Also, the Left Anterior Descending Coronary Artery of male Fpr2/3 KO and littermate controls (WT) was occluded for 30min and re-opened for 90min. At the

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29

Frewen, Sharon H. "The design and evaluation of a short-term group psychotherapy model for survivors of a first myocardial infarction." Thesis, Rhodes University, 2005. http://hdl.handle.net/10962/d1015041.

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There is extensive evidence that the rehabilitation of individuals with coronary heart disease needs to include psychological components to complement the exercise and dietary recommendations that are normally provided. However, psychological aspects have not been integrated into medical care in South Africa to any significant degree. Psychological interventions overseas have included the modification of the Type A behaviour pattern, stress management, cognitive restructuring, relaxation techniques, improved communication skills, the identification and expression of emotions, and emotional sup

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30

Condén, Emelie. "Type D Personality : Psychometric Properties of the DS14 and Associations with Ill Health and Coronary Heart Disease in General and Clinical Populations." Doctoral thesis, Uppsala universitet, Institutionen för neurovetenskap, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-233041.

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Type D personality, or distressed personality, refers to the joint tendency to experience negative emotions and to inhibit self-expression in social interactions. The overall aims of this thesis were to examine the impact of Type D personality on adolescents’ self-perceived health, to examine the factorial and temporal stability of the Type D personality construct DS14, and to clarify whether type D personality is an independent risk factor for recurrent myocardial infarction and all-cause mortality among patients with myocardial infarction. The prevalence of Type D personality in the adolesce

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31

Dean, Stephanie A. "Regulation of angiotensin converting enzyme and angiotensin II type 1 receptor by 17beta-estradiol in female rats: Implications following experimental myocardial infarction." Thesis, University of Ottawa (Canada), 2005. http://hdl.handle.net/10393/26883.

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The present studies tested the hypothesis that 17beta-estradiol (E2) downregulates ACE and AT1R in several tissues important to cardiovascular regulation, including the brain and heart, and that this downregulation attenuates the progression of LV dysfunction following myocardial infarction (MI). In Experiment 1, female Wistar rats were randomized into one of four groups: (1) sham-ovariectomized (OVX); (2) OVX+vehicle (veh); (3) OVX+E2 replacement at physiological levels and (4) OVX+high E2. Five weeks following OVX, ACE and ATIR were increased 15-90% in the heart, several cardiovascular nucle

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32

Hodson, Aimee Elizabeth. "Insulin Treatment Increases Myocardial Ceramide Accumulation and Disrupts Cardiometabolic Function." BYU ScholarsArchive, 2016. https://scholarsarchive.byu.edu/etd/5954.

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Prevalence of diabetes, especially type 2 diabetes mellitus (T2DM) is increasing worldwide. Millions of people are already affected by T2DM and estimates predict over half a billion people will likely be suffering from the disease by 2030. T2DM is associated with an increased risk of developing cardiovascular disease. Cardiovascular dysfunction is the leading cause of mortality among type 2 diabetics. Treatment for T2DM has changed over time. Though it was once known as insulin independent, a large portion of type 2 diabetics are now treated with insulin injections. However, type 2 diabetics t

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33

Whittington, H. J. "The influence of age and type 2 diabetes on cardioprotective interventions against myocardial ischaemia-reperfusion injury." Thesis, University College London (University of London), 2013. http://discovery.ucl.ac.uk/1402476/.

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The background of the thesis is based on the conflicting results between bench and bedside regarding the susceptibility to myocardial infarction with old age and diabetes. In laboratories all over the world, strategies have been developed to protect the myocardium from this insult, including the use of ischaemic conditioning (short periods of ischaemia and reperfusion prior to or following lethal ischaemia) or the use of a variety of pharmacological agents. However, surprisingly, translating these effective cardioprotective treatments into the clinic has proved problematic. The main issue seem

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34

Geil, Dominik. "Charakterisierung der Ca 2+ -Transportaktivität des sarkoplasmatischen Retikulums nach experimentellem Myokardinfarkt." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 1998. http://dx.doi.org/10.18452/14393.

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Zur Charakterisierung der myokardialen SR Ca2+-Transportaktivität nach experimentellem Myokardinfarkt wurde 10-15 Wochen alten Ratten die linke Koronararterie unterbunden und sechs Wochen später die Funktion und Expression der Ca2+-ATPase des sarkoplasmatischen Retikulums untersucht. Ein Teil der Tiere wurde mit dem Carnitin-Palmitoyl-Transferase-1- Inhibitor Etomoxir behandelt, der die mitochondrale Oxidation von langkettigen Fettsäuren hemmt und dadurch verstärkt Glukoseoxidation bewirkt. Diese Stoffwechselumstellung sollte die Schädigung der myokardialen Funktion und die g

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35

Messina, Julia Antoinette. "Molecular Localization of Hypoxia Inducible Factor-1-Alpha in Post-Ischemic Myocardium Following in Vivo Prolyl-4 Hydroxylase-2 Gene Silencing." VCU Scholars Compass, 2006. http://hdl.handle.net/10156/2197.

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36

Anderson, Donna. "Social support, quality of life and impact of illness on quality of life following myocardial infarction of coronary by-pass surgery /." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://proquest.umi.com/pqdweb?did=737023791&sid=4&Fmt=2&clientId=9268&RQT=309&VName=PQD.

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37

Govind, Satish C. "Myocardial Effects of Type 2 Diabetes, Co-morbidities, and Changing Loading Conditions : a Clinical Study by Tissue Velocity Echocardiography." Doctoral thesis, Stockholm : Skolan för teknik och hälsa, Kungliga Tekniska högskolan, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-4359.

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38

Kakimoto, Yu. "Sorbin and SH3 Domain-containing Protein 2 Is Released from Infarcted Heart in Very Early Phase: Proteomic Analysis of Cardiac Tissues from Patients." Kyoto University, 2014. http://hdl.handle.net/2433/188650.

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39

Das, Rajiv. "Risk assessment and quality of care of patients with redefined acute myocardial infarction : a second evaluation of methods and management of acute coronary events study : EMMACE-2." Thesis, University of Leeds, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.416317.

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40

Hobbs, Daniel. "PATHOGENIC ROLE OF PHOSPHODIESTERASE TYPE 5 UPREGULATION IN CARDIAC ISCHEMIA/REPERFUSION INJURY." VCU Scholars Compass, 2010. http://scholarscompass.vcu.edu/etd/106.

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Phosphodiesterase Type 5 (PDE5) inhibitors are cardioprotective against ischemia/reperfusion (I/R) injury. However, it remains uncertain if I/R affects PDE5. We hypothesized that generation of reactive oxygen species (ROS) during I/R leads to upregulation of PDE5, which contributes to pathological changes following acute myocardial infarction (AMI). Adult male ICR mice were subjected to 30 minutes of in vivo or ex vivo I/R. To examine the role of ROS, a subset of hearts were perfused with 100 µM hydrogen peroxide (H2O2). Expression and activity of PDE5, pPDE5, and cGMP-dependent protein k

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41

Reyes, Levy Austin. "New mechanisms in nitric oxide synthase related endothelial dysfunction in the isolated heart." The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1338346540.

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42

Hoxha, M. "THE POTENTIAL THERAPEUTIC ROLE OF MONTELUKAST AND NEW HYBRID AGENTS, TXA2 ANTAGONIST-COX-2 INHIBITORS IN CARDIOVASCULAR EVENTS." Doctoral thesis, Università degli Studi di Milano, 2016. http://hdl.handle.net/2434/347148.

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The main target of my PhD research project was to explore new alternatives to reduce the cardiovascular (CV) risk targeting the arachidonic acid metabolites. Hence, I have been part of two different projects, one studying multitarget compounds with balanced COXIB and TP receptor antagonist properties, and the other evaluating the potential role of a leukotriene (LT) antagonist drug such as montelukast in improving the CV outcome. In reference to the first project, new multitarget compounds were synthesized at the University of Turin, by substituting the carboxylic function of Lumiracoxib.

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43

Conti, Filipe Fernandes. "Efeitos do treinamento físico na cardiomiopatia e neuropatia autonômica associadas ao diabetes tipo 2 em fêmeas." Universidade Nove de Julho, 2016. http://bibliotecatede.uninove.br/handle/tede/1837.

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Submitted by Nadir Basilio (nadirsb@uninove.br) on 2018-07-16T21:15:58Z No. of bitstreams: 1 Filipe Fernandes Conti.pdf: 2117259 bytes, checksum: 21aef2931e9bc68c1df23685f3d8200a (MD5)<br>Made available in DSpace on 2018-07-16T21:15:58Z (GMT). No. of bitstreams: 1 Filipe Fernandes Conti.pdf: 2117259 bytes, checksum: 21aef2931e9bc68c1df23685f3d8200a (MD5) Previous issue date: 2016-12-15<br>Diabetic patients frequently present cardiomyopathy and autonomic neuropathy, which increases mortality risk. On the other hand, exercise training has been suggested as a non-pharmacological tool in the pre

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Wagner, Claudia Karin. "Kardioprotektion durch Postkonditionierung gesunder Rattenherzen sowie von Herzen mit kardiovaskulären Risikofaktoren: Charakterisierung der Signaltransduktion unter besonderer Betrachtung von PI3-K/Akt, mTOR, ERK1/2 und GSK-3ß." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2008. http://nbn-resolving.de/urn:nbn:de:bsz:14-ds-1226858602601-68237.

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In den ersten Versuchsreihen der hier vorliegenden Arbeit bestätigte sich, dass im in vitro Modell die klassische ischämische Präkonditionierung kardioprotektiv wirkt. Die Präkonditionierung bewirkte eine Infarktgrößenreduktion um 54 %; dies wird durch Literaturangaben bestätigt. Die Postkonditionierung dagegen, trotz drei verschiedener Postkonditionierungsprotokolle, ist am isoliert perfundierten Rattenherzen nicht protektiv. Im in vivo Rattenherz-Modell wurden die Präkonditionierung und die klinisch relevantere Postkonditionierung gegenüberstellend untersucht. Hier zeigte sich, dass die 3 Re

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Addisu, Anteneh. "Natriuretic peptides as a humoral link between the heart and the gastrointestinal system." [Tampa, Fla] : University of South Florida, 2008. http://purl.fcla.edu/usf/dc/et/SFE0002406.

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Gunter, Bryan R., Kristen A. Butler, Rick L. Wallace, Steven M. Smith, and Sam Harirforoosh. "NSAIDs-Induced Cardio- and Cerebro-Vascular Adverse Events: a Meta-analysis." Digital Commons @ East Tennessee State University, 2017. https://doi.org/10.1111/jcpt.12484.

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What is known and objective: Although non-steroidal anti-inflammatory drugs (NSAIDs) have been studied in randomized, controlled trials and meta-analyses in an effort to determine their cardiovascular (CV) risks, no consensus has been reached. These studies continue to raise questions, including whether cyclooxygenase-2 (COX-2) selectivity plays a role in conferring CV risk. We performed a meta-analysis of current literature to determine whether COX-2 selectivity leads to an increased CV risk. Methods: We utilized randomized, controlled trials and prospective cohort studies. We selected eight

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Rezende, Paulo Cury. "Expressão do precondicionamento isquêmico em pacientes com diabetes mellitus tipo 2 e doença arterial coronariana." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/5/5131/tde-06012016-135102/.

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Introdução: Acredita-se que o diabetes mellitus possa alterar mecanismos celulares do miocárdio tornando-o mais sensível a um insulto isquêmico, e que esta menor resistência do miocárdio isquêmico induzido pelo diabetes possa ser um dos motivos para o pior prognóstico observado em pacientes com doença arterial coronariana e diabetes. Um dos principais mecanismos adaptativos protetores do miocárdio é o precondicionamento isquêmico, sendo este desencadeado por curtos períodos de isquemia seguidos por reperfusão e que tornam o tecido mais resistente a um insulto isquêmico grave e prolongado. Em h

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Philouze, Clothilde. "Dépistage des altérations précoces de la fonction régionale myocardique par échocardiographie de stress et effet d’une intervention par supplémentation en vitamine D3 dans le diabète de type 2 : approche translationnelle." Thesis, Avignon, 2018. http://www.theses.fr/2018AVIG0346/document.

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Le diabète est aujourd’hui la 7ème cause de mortalité dans le monde. Dans cette population, la cardiomyopathie diabétique est la principale cause de morbi-mortalité. Le développement de cette complication débutant dès l’apparition du diabète, un dépistage et une prise en charge précoces de cette pathologie sont donc de première importance et sont les deux objectifs visés par ces travaux. La première étude que nous avons réalisée a permis de démontrer l’utilité d’une évaluation compréhensive de la fonction régionale myocardique gauche par 2D speckle-tracking imaging, en conditions de stress à l

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Bakhta, Oussama. "Métabolites circulants induits par le conditionnement ischémique à distance et mécanismes cardioprotecteurs de la colchicine à la phase aigue de l'infarctus du myocarde Metabolic Signature of Remote Ischemic Preconditioning Involving a Cocktail of Amino Acids and Biogenic Amines Cardioprotective role of colchicine against inflammatory injury in a rat 2 model of acute myocardial infarction." Thesis, Angers, 2017. http://www.theses.fr/2017ANGE0089.

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La reperfusion précoce à la phase aiguë de l’infarctus du myocarde améliore le pronostic des patients. Elle induit cependant une partie des lésions d’ischémie-reperfusion (I/R) liées à l’activation de mécanismes métaboliques et inflammatoires. Le conditionnement ischémique à distance (RIC) d’une part et les approches pharmacologiques d’autre part, constituent un espoir dans la prévention des lésions de reperfusion contre lesquelles nous ne disposons pas de traitement validé chez l’homme. L’objectif de cette thèse était d’explorer ces stratégies de cardioprotection à la phase aiguë de l’infarct

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Labbé, Vincent. "Risques thrombotiques et hémodynamiques chez les patients hospitalisés en réanimation présentant une fibrillation atriale de novo au cours d’un sepsis : caractérisation, stratification et stratégies thérapeutiques." Electronic Thesis or Diss., Sorbonne université, 2023. https://accesdistant.sorbonne-universite.fr/login?url=https://theses-intra.sorbonne-universite.fr/2023SORUS556.pdf.

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Objectifs Les patients hospitalisés en réanimation pour un sepsis sont à haut risque d’accidents thrombotiques (AT) intéressant l’ensemble des circulations (grande, coronaire, petite). Nous souhaitons étudier ce risque thrombotique lors du sepsis (i) au sein de la grande circulation chez les patients présentant une fibrillation atriale de novo (FAN), (ii) au sein de la circulation coronaire chez les patients présentant un infarctus du myocarde (IDM) et (iii) au sein de la petite circulation chez les patients COVID-19 sévère. Par ailleurs, si le risque d’AT pose la question de l’intérêt de la t

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