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1

Greenberg, Mitchell D., and Francis Hutchins. Uterine fibroids. W.B. Saunders, 1995.

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2

Segars, James. Fibroids. John Wiley & Sons, 2013.

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3

A, Brosens I., ed. Uterine leiomyomata: Pathogenesis and management. Taylor & Francis, 2006.

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4

Darlene, Dixon, Kohn Michael C, and National Institute of Environmental Health Sciences., eds. Advances in uterine leiomyoma research: Mathematical modeling in environmental health studies. U.S. Dept. of Health & Human Services, Public Health Service, National Institutes of Health, National Institute of Environmental Health Sciences, 2000.

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5

Meera, Viswanathan, RTI International-University of North Carolina Evidence-based Practice Center., and United States. Agency for Healthcare Research and Quality., eds. Management of uterine fibroids: An update of the evidence. U.S. Dept. of Health and Human Services, Public Health Service, Agency for Healthcare Research and Quality, 2007.

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6

Greig, Lloyd B. 100 questions and answers about uterine fibroids. Jones and Bartlett Publishers, 2010.

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7

Taylor, Carolyn V. The expression of cell adhesion molecules in normal human uterus and uterine leiomyoma. National Library of Canada, 1994.

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8

R, Rolland, Chadha Dev R, and Willemsen, Wilhelmus Nicolass Petrus, 1947-, eds. Gonadotropin down-regulation in gynecological practice: Proceedings of an international symposium held at the University of Nijmegen, the Netherlands, April 25 and 26, 1986. Liss, 1986.

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9

Stewart, Elizabeth A. Uterine Fibroids: The Complete Guide. Johns Hopkins University Press, 2007.

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10

Brosens, Ivo. Uterine Leiomyomas: Pathogenesis and Management. Informa Healthcare, 2005.

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11

(Editor), I. A. Brosens, and B. Lunenfeld (Editor), eds. Pathogenesis and Medical Management of Uterine Fibroids. Informa Healthcare, 1999.

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12

Uterine artery embolization and gynecologic embolotherapy. Lippincott Williams & Wilkins, 2005.

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13

Greig, Lloyd B. 100 Questions and Answers about Uterine Fibroids. Jones & Bartlett Learning, LLC, 2010.

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14

Dionne, Carla. Sex, Lies, and the Truth about Uterine Fibroids. Avery, 2001.

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15

The treatment of fibroids. Hill of Content, 2000.

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16

Sex, lies and the truth about uterine fibroids: A journey from diagnosis to treatment to renewed good health. Avery, 2001.

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17

Skilling, Johanna, and Eileen Hoffman. Fibroids: The Complete Guide to Taking Charge of Your Physical, Emotional and Sexual Well-Being. Hachette Books, 2009.

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18

Skilling, Johanna. Fibroids: The Complete Guide to Taking Charge of Your Physical, Emotional and Sexual Well-Being. Marlowe & Company, 2006.

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19

Skilling, Johanna. Fibroids: The Complete Guide to Taking Charge of Your Physical, Emotional, and Sexual Well-Being. Marlowe & Company, 2000.

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20

Congenital Uterine Anomalies: Education for Patients and the Public. Exon Publications, 2025. https://doi.org/10.36255/congenital-uterine-anomalies-patient-public-education.

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Congenital uterine anomalies are birth-related abnormalities in the structure of the uterus that may impact menstrual health, fertility, or pregnancy outcomes. These conditions arise during fetal development when the uterus does not form correctly. Some women may have no symptoms at all, while others discover the condition during evaluation for infertility or recurrent pregnancy loss. This article explains in clear language what congenital uterine anomalies are and outlines the different types, such as septate, bicornuate, and didelphys uterus. It describes how these structural differences occ
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21

Gottlieb, Samantha D. Not Quite a Cancer Vaccine: Selling HPV and Cervical Cancer. Rutgers University Press, 2018.

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22

Gottlieb, Samantha D. Not Quite a Cancer Vaccine: Selling HPV and Cervical Cancer. Rutgers University Press, 2017.

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23

Not Quite a Cancer Vaccine: Selling HPV and Cervical Cancer. Rutgers University Press, 2018.

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24

Hakim, Alan J., and Rodney Grahame. Hypermobility syndromes. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0159.

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Hypermobility-related syndromes constitute a family of heritable disorders of connective tissue (HDCT) that derive from abnormalities affecting genes that encode for the connective tissue matrix proteins such as collagen, fibrillin, and tenascin. They range from such commonplace though poorly recognized conditions such as the joint hypermobility syndrome (JHS) to the better-known, if more rare, eponymous syndromes such as Marfan's syndrome (MFS) and the different types of the Ehlers-Danlos syndrome (EDS). The more common presentations are with skin pathology (bruising, scaring), joint or spina
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