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Relevant bibliographies by topics / Ulcere gastroduodenal

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Academic literature on the topic 'Ulcere gastroduodenal'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Ulcere gastroduodenal"

1

Vainshtein, S. G., Z. I. Shust, M. N. Zhukovsky, and Ya N. Fedoriv. "Local treatment of gastroduodenal ulcers." Kazan medical journal 67, no. 6 (November 15, 1986): 403–4. http://dx.doi.org/10.17816/kazmj70743.

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In spite of the established ideas about peptic ulcer as a general disease of the whole organism, there is a continuous flow of publications in which local treatment of ulcers of the mucous membrane of the stomach and duodenum is recommended. With the development of fiber optics, it became possible to target the ulcers, activating reparation processes in them. Adhesives and film-forming compositions MK-6, Lifusol, Gastrovol etc. were used to cover the ulcers.

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2

Xu, Fang, Cai-ling Mai, and Qian Zhu. "Association of TLR-9 polymorphisms with the development of gastroduodenal ulcer: A hospital-based study in a Chinese cohort." European Journal of Inflammation 16 (January 1, 2018): 1721727X1875726. http://dx.doi.org/10.1177/1721727x18757262.

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Helicobacter pylori infections have been one of the major factors associated with gastroduodenal ulcer. Toll-like receptors (TLRs) of human recognize mycobacterium-induced immune response and protect subjects from disease pathogenesis. Variants in TLR genes are believed to influence immune responses to H. pylori and clinical severity. TLR-9 polymorphisms have been associated with susceptibility to gastroduodenal ulcer in different populations. In this study, we investigate the role of common TLR-9 variants in susceptibility/resistance to the development of gastroduodenal ulcer in a Chinese cohort. The present hospital-based case–control study enrolled 580 patients with abdominal discomfort, and based on endoscopic investigation, the patients were categorized into (1) gastric ulcer (n = 154), (2) duodenal ulcer (n = 70), (3) gastric and duodenal ulcers (n = 25), (4) gastritis (n = 195), and (5) healthy stomach (n = 136). A total of 520 healthy controls from similar geographical areas were enrolled as controls. TLR-9 (C-1237T, C-1486T, and G+2848A) polymorphisms were genotyped by polymerase chain reaction-restriction fragment length polymorphism (PCR-RFLP). Plasma levels of tumor necrosis factor (TNF)-α, interleukin (IL)-6, and IL-1β in both healthy controls and patients were quantified by enzyme-linked immunosorbent assay (ELISA). Furthermore, messenger RNA (mRNA) expressions of TNF-α, IL-6, and IL-1β in biopsy tissues were quantified by real-time polymerase chain reaction (RT-PCR). The prevalence of TLR-9+2848 heterozygotes (CT) was significantly higher in gastroduodenal ulcer patients compared to healthy controls. Sub-categorization of patients revealed higher prevalence of heterozygotes of TLR-9 C+2848T and C-1486T polymorphisms in patients with gastric ulcer (GU), duodenal ulcer (DU), and those with both gastric and duodenal ulcers (GDU) when compared to controls. Patients displayed higher plasma cytokine levels than healthy controls. TLR-9 polymorphisms (C+2848T and C-1486T) correlated with altered cytokine expression in biopsy tissues and their plasma levels. In conclusion, TLR-9 (C+2848T and C-1486T) polymorphisms are associated with gastroduodenal ulcer and correlated with altered cytokine levels.

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Ashcroft, Darren M., Stephen R. Chapman, Wendy K. Clark, and David S. Millson. "Upper Gastroduodenal Ulceration in Arthritis Patients Treated with Celecoxib." Annals of Pharmacotherapy 35, no. 7-8 (July 2001): 829–34. http://dx.doi.org/10.1345/aph.10382.

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OBJECTIVE: To evaluate the comparative incidence of endoscopic gastroduodenal ulcers in patients with rheumatoid arthritis or osteoarthritis treated with celecoxib. DESIGN: Quantitative systematic review of randomized controlled trials. SUBJECTS: Patients (n = 4632) with rheumatoid arthritis or osteoarthritis reported in five trials. MAIN OUTCOME MEASURES: Rate ratios, rate differences, and the number needed to harm were calculated for the incidence of endoscopically documented gastroduodenal ulcers. RESULTS: Pooled rate ratios (RRs) relative to placebo for endoscopic ulcers at 12 weeks were 1.96 (95% CI 0.85 to 4.55) for celecoxib 100 mg twice daily and 2.35 (95% CI 1.02 to 5.38) for celecoxib 200 mg twice daily. There was no significant difference in gastroduodenal ulcers at 12 weeks between celecoxib 200 mg twice daily and celecoxib 100 mg twice daily; the corresponding pooled RR was 1.21 (95% CI 0.62 to 2.38). In contrast, celecoxib 200 mg twice daily was associated with a significantly lower rate of gastroduodenal ulcers than was naproxen 500 mg twice daily at 12 weeks (RR 0.24; 95% CI 0.17 to 0.33). On average, for every seven patients treated with naproxen, one more had an endoscopic ulcer than if they were treated with celecoxib. Celecoxib 200 mg twice daily also had a significantly lower risk of endoscopic ulcers than did either modified-release diclofenac 75 mg twice daily at 24 weeks (RR 0.24; 95% CI 0.11 to 0.52) or ibuprofen 800 mg three × daily at 12 weeks (RR 0.30; 95% CI 0.20 to 0.46). CONCLUSIONS: Endoscopic studies have shown that celecoxib, at a wide range of doses, is associated with a lower incidence of gastroduodenal ulcers than are diclofenac, ibuprofen, or naproxen. The incidence rates of gastroduodenal ulcers associated with celecoxib were similar, although not equivalent, to placebo. Head-to-head comparisons suggest that, at the wide range of doses studied (100–800 mg/d), there are no dose-related increases in endoscopic gastroduodenal ulcers with celecoxib. The results of longer term comparative trials of celecoxib based on clinical outcomes are needed to determine celecoxib's ultimate risk—benefit profile.

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4

Melnyk, I. V., and S. B. Soliev. "The original scale prediction of recurrence risk of gastroduodenal ulcer bleeding." Experimental and Clinical Gastroenterology, no. 2 (April 7, 2021): 72–78. http://dx.doi.org/10.31146/1682-8658-ecg-186-2-72-78.

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Research goal - to examine the effectiveness of active-individualized tactics in treatment of chronic gastric and duodenal ulcers. Material and methods. Analysis of treatment of 251 patients with chronic gastric and duodenal ulcer bleeding was conducted. Duodenal ulcer bleeding was present in 202 cases (80,5%), gastric ulcers were the reason of bleeding in 49 cases (19,5%). Treatment conducted according to principles of active-individualized tactics. Results. The provided characteristics and the acquired results were presented according to the fundamental components of active-individualized tactics in treatment of gastric and duodenal ulcers. Diagnostic and treatment abilities of endoscopy were determined, an unique scale of bleeding relapse risk was presented, the time limits and main indications for surgery were explained,, the role of bleeding relapse was emphasized as crucial in forming the main results of treatment of patients with bleeding gastric and duodenal ulcers. Conclusion. At present time, active-individualized tactics should be used for treatment of gastric and duodenal ulcer bleeding.

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Santos-Rancaño, Rocio, Esteban Martín Antona, and José Vicente Méndez Montero. "A Challenging Case of a Large Gastroduodenal Artery Pseudoaneurysm after Surgery of a Peptic Ulcer." Case Reports in Surgery 2015 (2015): 1–3. http://dx.doi.org/10.1155/2015/370937.

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We report a 48-year-old man in whom a chronic postbulbar duodenal ulcer destroyed much of the back wall of the duodenum and gastroduodenal artery causing pseudoaneurysm. The lesion was found and evaluated by contrast-enhanced computed tomography (that revealed a large pseudoaneurysm of 83 mm×75 mm in diameter) and by angiography and then treated with transcatheter embolization leading to a complete resolution of the lesion. The case is rare and important for several reasons. First, we demonstrate that pseudoaneurysm of the gastroduodenal artery caused by a duodenal ulcer can occur and present a diagnostic challenge (as far as we know, only three cases have been reported previously in the literature). Second, this case report focuses on the importance of ligation of the gastroduodenal artery when bleeding of peptic ulcers occurs. Additionally, we present an overview of the relevant literature.

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Spivakovskiy, Yu M., A. Yu Spivakovsky, Yu V. Chernenkov, O. V. Volkova, I. V. Goremykina, and A. K. Dushanova. "The case of atypical course of gastric ulcer in pediatric practice." Experimental and Clinical Gastroenterology, no. 11 (November 20, 2019): 93–99. http://dx.doi.org/10.31146/1682-8658-ecg-171-11-93-99.

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In the article, the authors present brief information about gastric ulcer and duodenal ulcer, its epidemiology, diagnostic capabilities, approaches to the treatment and prevention of complications. In addition, there are data on the diagnosis and verification of symptomatic gastroduodenal ulcers. Using the example of a clinical case of a complicated symptomatic ulcer in a child, the authors describe the difficulties encountered at the stages of diagnosis, treatment, and follow-up at high risk of complications.

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Zherlov, G. K., A. P. Koshel, V. M. Vorobiyov, and Yu D. Yermolayev. "Язвенная болезнь желудка и двенадцатиперстной кишки: выбор тактики при кровотечении." Bulletin of Siberian Medicine 5, no. 1 (March 30, 2006): 76–80. http://dx.doi.org/10.20538/1682-0363-2006-1-76-80.

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Ulcerative gastroduodenal bleedings remain the actual problem of emergency surgery because of quite frequency and high mortality. The analysis of treatment results in 782 patients was conducted. There were 239 patients with gastric ulcers, 488 patients with duodenal ulcers, 37 patients with combined ulcers and 18 patients with bleeding peptic ulcers of gastroenteranastomosis. The choice of surgical policy was based on emergency fibrogastroscopy findings. According to these findings the locality and severity of bleeding were defined using J. Forrest scale. The treatment began in intensive care with obligatory endoscopic control "by the program". Emergency operations were performed in 5,9% cases and urgent operations were performed in 16.4% cases of all patients who suffered with bleeding. There were 21,7% patients with operative treatment in remote and planned order. Postoperative mortality was 3,8%. Postoperative complications were in 12,2% patients. There were consider that the treatment of ulcerative gastroduodenal bleedings should be based on the individual active and waiting policy concluding in somatic patient's condition, duration of ulcerative anamnesis, presence of gastroduodenal bleedings in the past, ulcer location, and bleeding intensity (using J. Forrest scale) and effectiveness of conservative hemostasis.

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Wong, Grace L. H., Louis H. S. Lau, Jessica Y. L. Ching, Yee-Kit Tse, Rachel H. Y. Ling, Vincent W. S. Wong, Philip W. Y. Chiu, James Y. W. Lau, and Francis K. L. Chan. "Prevention of recurrent idiopathic gastroduodenal ulcer bleeding: a double-blind, randomised trial." Gut 69, no. 4 (June 22, 2019): 652–57. http://dx.doi.org/10.1136/gutjnl-2019-318715.

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ObjectivePatients with a history of Helicobacter pylori-negative idiopathic bleeding ulcers have a considerable risk of recurrent ulcer complications. We hypothesised that a proton pump inhibitor (lansoprazole) is superior to a histamine 2 receptor antagonist (famotidine) for the prevention of recurrent ulcer bleeding in such patients.DesignIn this industry-independent, double-blind, randomised trial, we recruited patients with a history of idiopathic bleeding ulcers. After ulcer healing, we randomly assigned (1:1) patients to receive oral lansoprazole 30 mg or famotidine 40 mg daily for 24 months. The primary endpoint was recurrent upper GI bleeding within 24 months, analysed in the intention-to-treat population as determined by an independent adjudication committee.ResultsBetween 2010 and 2018, we enrolled 228 patients (114 patients in each study group). Recurrent upper GI bleeding occurred in one patient receiving lansoprazole (duodenal ulcer) and three receiving famotidine (two gastric ulcers and one duodenal ulcer). The cumulative incidence of recurrent upper GI bleeding in 24 months was 0.88% (95% CI 0.08% to 4.37%) in the lansoprazole arm and 2.63% (95% CI 0.71% to 6.91%) in the famotidine arm (p=0.313; crude HR 0.33, 95% CI 0.03 to 3.16, p=0.336). None of the patients who rebled used aspirin, non-steroidal anti-inflammatory drugs or other antithrombotic drugs.ConclusionThis 2-year, double-blind randomised trial showed that among patients with a history of H. pylori-negative idiopathic ulcer bleeding, recurrent bleeding rates were comparable between users of lansoprazole and famotidine, although a small difference in efficacy cannot be excluded.Trial registration numberNCT01180179; Results.

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Pokydko, M. I., T. V. Formanchuk, O. V. Goncharenko, A. M. Formanchuk, and V. A. Katsal. "The complex treatment of patients with perforated gastroduodenal ulcers." Reports of Vinnytsia National Medical University 22, no. 3 (September 28, 2018): 479–84. http://dx.doi.org/10.31393/reports-vnmedical-2018-22(3)-17.

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To improve the immediate results of surgical treatment of patients with perforated gastroduodenal ulcers using minimally invasive correction and an integrated treatment. The results of 253 patients with perforated gastroduodenal ulcers who were treated from 2013 to 2018 were analyzed. Chronic ulcers were found in 82 (32%) cases among all gastric ulcers, acute ones constituted 42 (16%) cases. Chronic duodenal ulcers reached 94 (37%), acute ones constituted 35 (14%). The diagnostic laparoscopy was performed in 13 (5%) patients, in 8 (3%) patients the perforated hole was sutured laparoscopically and in 5 (2%) patients the conversion was performed. Abdominocentesis with preoperative abdominal cavity drainage was carried out in 45(18%) patients. Through this drainage after exudate evacuation 200 ml of dekasan solution was injected. In 119 cultures of abdominal exudates with determinate pathogenic microflora Enterococcus faecalis dominated - 31 patients (26%). Among the postoperative complications the failure of intestinal sutures was observed in 4 (1.5%) patients. Postoperative mortality was 11.5%. Progression of multiple organ failure became the cause of mortality in 17 (59%) patients. Thus, the time from the moment of perforation till the moment of surgery must not be the sole criterion in the choice of method of operative intervention, and it is insufficient as an absolute indicator for the possibility of the radical surgical procedures (gastric resection). Undoubtedly laparoscopic suturing of the perforated gastroduodenal ulcer has advantages over laparotomy.

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10

Negovan, Anca, Mihaela Iancu, Valeriu Moldovan, Septimiu Voidazan, Simona Bataga, Monica Pantea, Kinga Sarkany, Cristina Tatar, Simona Mocan, and Claudia Banescu. "Clinical Risk Factors for Gastroduodenal Ulcer in Romanian Low-Dose Aspirin Consumers." Gastroenterology Research and Practice 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/7230626.

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Background. Aspirin use for cardiovascular or cancer prevention is limited due to its gastrointestinal side effects.Objective. Our prospective, observational case-control study aims to identify the predictive factors for ulcers in low-dose aspirin consumers (75–325 mg/day).Methods. The study included patients who underwent an upper digestive endoscopy and took low-dose aspirin treatment.Results. We recruited 51 patients with ulcer (ulcer group) and 108 patients with no mucosal lesions (control group). In univariate analysis, factors significantly associated with ulcers were male gender (p=0.001), anticoagulants (p=0.029), nonsteroidal anti-inflammatory drugs (p=0.013), heart failure (p=0.007), liver (p=0.011) or cerebrovascular disease (p=0.004), diabetes mellitus (p=0.043), ulcer history (p=0.044), and alcohol consumption (p=0.018), but notHelicobacter pyloriinfection (p=0.2). According to our multivariate regression analysis results, history of peptic ulcer (OR 3.07, 95% CI 1.06–8.86), cotreatment with NSAIDs (OR 8, 95% CI 2.09–30.58) or anticoagulants (OR 4.85, 95% CI 1.33–17.68), male gender (OR 5.2, 95% CI 1.77–15.34), and stroke (OR 7.27, 95% CI 1.40–37.74) remained predictors for ulcer on endoscopy.Conclusions. Concomitant use of NSAIDs or anticoagulants, comorbidities (cerebrovascular disease), and male gender are the most important independent risk factors for ulcer on endoscopy in low-dose aspirin consumers, in a population with a high prevalence ofH. pyloriinfection.

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Dissertations / Theses on the topic "Ulcere gastroduodenal"

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AUCOUTURIER, JEAN-PAUL. "Quels sont les ulceres qui saignent ? etude tranversale d'une serie hospitaliere de 310 patients consecutifs ayant un ulcere gastrique ou duodenal." Reims, 1989. http://www.theses.fr/1989REIMM087.

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Minault, Sophie. "Incidence de l'ulcere gastrique et duodenal sur une base de population urbaine : bilan d'un an d'enregistrement sur la commune de reims en 1988." Reims, 1989. http://www.theses.fr/1989REIMM080.

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CABIANCA, ROCHE BEATRICE. "Campylobacter pylori et ulcerations gastro-duodenales chez le jeune adulte : a propos de 45 observations." Bordeaux 2, 1988. http://www.theses.fr/1988BOR25312.

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Simon, Jean-Philippe. "Etude prospective de la prevalence de campylobacter pylori apres intervention pour ulcere gastroduodenal." Nancy 1, 1988. http://www.theses.fr/1988NAN11004.

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Roux, Philippe. "Hemostase endoscopique des ulceres gastro-duodenaux : a propos d'une experimentation animale." Clermont-Ferrand 1, 1989. http://www.theses.fr/1989CLF13817.

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KARAM, RAJA. "Perforation en peritoine libre d'un ulcere du collet d'une hernie hiatale : a propos d'une observation." Angers, 1988. http://www.theses.fr/1988ANGE1032.

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GEORGEAC, CONSTANTIN. "Etude du traitement chirurgical des ulceres gastro-duodenaux hemorragiques au c. H. U. D'angers." Angers, 1991. http://www.theses.fr/1991ANGE1021.

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8

PEROUX, JEAN-LUC. "Helicobacter pylori et pathologie gastroduodenale en nouvelle-caledonie." Nice, 1992. http://www.theses.fr/1992NICE6551.

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9

LASBASSES, CLAUDINE. "Prevention des ulceres gastro-duodenaux de stress : activite comparee du sucralfate et de la cimetidine : a propos de quatre-vingt-dix-sept cas dans un service de reanimation polyvalente." Bordeaux 2, 1988. http://www.theses.fr/1988BOR25165.

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RUDELLI, ALAIN. "Helicobacter pylori, gastrite et maladie ulcereuse chez l'adulte jeune." Amiens, 1991. http://www.theses.fr/1991AMIEM059.

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Books on the topic "Ulcere gastroduodenal"

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Francis, Mégraud, and Lamouliatte Hervé, eds. Gastroduodenal pathology and Campylobacter pylori: Proceedings of the First Meeting of the European Campylobacter Pylori Study Group, held in Bordeaux, France, 7-8 October 1988. Amsterdam: Excerpta Medica, 1989.

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2

Helicobacter Pylori & Gastroduodenal Disease. 2nd ed. Blackwell Science, 1992.

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J, Rathbone B., Heatley Richard Val, and Rathbone B. J, eds. Helicobacter pylori and gastroduodenal disease. 2nd ed. London: Blackwell Scientific, 1992.

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4

(Editor), Francis Megraud, and Herve Lamouliatte (Editor), eds. Gastroduodenal Pathology and Campylobacter Pylori (International congress series). Elsevier, 1989.

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5

A, Rostom, and Canadian Coordinating Office for Health Technology Assessment., eds. Gastroduodenal ulcers associated with the use of non-steroidal anti-inflammatory drugs: A systematic review of preventive pharmacological interventions. Ottawa: Canadian Coordinating Office for Health Technology Assessment, 2004.

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Book chapters on the topic "Ulcere gastroduodenal"

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Mirabella, Antonino, Massimo Lupo, Fausto Di Marco, and Vincenzo Mandalà. "Gastroduodenal Ulcer." In The Role of Laparoscopy in Emergency Abdominal Surgery, 11–31. Milano: Springer Milan, 2012. http://dx.doi.org/10.1007/978-88-470-2327-7_2.

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Seelig, M. H., C. Behr, E. L. Zurmeyer, and K. Schönleben. "Laparoskopische Notfallchirurgie bei perforierten gastroduodenalen Ulcera / Laparoscopic Emergency Surgery for Perforated Gastroduodenal Ulcers." In Deutsche Gesellschaft für Chirurgie, 268–71. Berlin, Heidelberg: Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-56458-1_81.

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Mirabella, Antonino, Massimo Lupo, Ferdinando Agresta, Stefano Mandalà, Gabriele Anania, Mario Campli, and Kjetil Soreide. "Perforated Gastroduodenal Ulcer." In Emergency Laparoscopy, 79–101. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-29620-3_7.

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4

Schumpelick, V., and G. Winkeltau. "Bleeding Gastroduodenal Ulcer." In Die Chirurgie und ihre Spezialgebiete Eine Symbiose, 632–33. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-95662-1_260.

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Anania, Gabriele, Alberto Campagnaro, Francesco Marchetti, Giuseppe Resta, and Roberto Cirocchi. "Perforated Gastroduodenal Ulcer." In Emergency laparoscopic surgery in the elderly and frail patient, 129–39. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-79990-8_13.

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Soreide, Kjetil. "Perforated Gastroduodenal Ulcer (PGDU)." In Resources for Optimal Care of Emergency Surgery, 141–44. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-49363-9_20.

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Rathbone, B. J., J. I. Wyatt, and R. V. Heatley. "Bacteria and gastroduodenal inflammation." In Advances in Peptic Ulcer Pathogenesis, 101–19. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1245-8_5.

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Sellers, L. A., and A. Allen. "Mucus and gastroduodenal mucosal protection." In Advances in Peptic Ulcer Pathogenesis, 121–44. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1245-8_6.

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O’Morain, C., and R. Collins. "Helicobacter pylori and Duodenal Ulcer." In Helicobacter pylori and Gastroduodenal Pathology, 136–41. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77486-7_24.

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Crampton, J. R., and W. D. W. Rees. "Gastroduodenal bicarbonate secretion: its contribution to mucosal defence." In Advances in Peptic Ulcer Pathogenesis, 145–62. Dordrecht: Springer Netherlands, 1988. http://dx.doi.org/10.1007/978-94-009-1245-8_7.

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Conference papers on the topic "Ulcere gastroduodenal"

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Popovichenko, N. V., A. A. Rogovoy, V. V. Ogarko, and V. N. Zalesskiy. "Use of monofiber optics for laser endoscopic biostimulation therapy of gastroduodenal ulcers." In Conference on Lasers and Electro-Optics. Washington, D.C.: OSA, 1986. http://dx.doi.org/10.1364/cleo.1986.tuk19.

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Shapkin, U. G., C. V. Kapralov, A. A. Gogolev, V. V. Lychagov, and V. V. Tuchin. "An endoscopic laser Doppler flowmetry of a gastroduodenal mucosa at bleeding ulcer." In SPIE Proceedings, edited by Valery V. Tuchin. SPIE, 2006. http://dx.doi.org/10.1117/12.697333.

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