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Books on the topic 'Unipolar depressive disorder'

Author: Grafiati

Published: 4 June 2021

Last updated: 11 February 2022

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M, Goodyer Ian, ed. Unipolar depression: A lifespan perspective. Oxford University Press, 2003.

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Prendes-Alvarez, Stefania, Alan F. Schatzberg, and Charles B. Nemeroff. Pharmacological Treatments for Unipolar Depression. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780199342211.003.0011.

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Major depressive disorder is a chronic syndrome associated with high mortality (secondary to suicide and increased risk for heart disease, stroke, and other serious diseases). It is one of the most common medical disorders affecting adults in the world today. In the United States, the lifetime prevalence of major depression is 16.7% for adults. The average age of onset is 32 years, and women are 70% more likely to develop depression than men. Neither the core requisite symptoms for the diagnosis of a major depressive episode nor the required duration of at least 2 weeks has changed from DSM-IV to DSM-5. This chapter discusses the main issues surrounding the treatment of major depressive disorder, such as suicidality and goals of treatment, and provides information about all treatment options approved by the U.S. Food & Drug Administration. Drugs are categorized by their mechanisms of action.

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Grisaru, Nimrod, Bella Chudakov, Alex Kaptsan, Alona Shaldubina, Julia Applebaum, and R. H. Belmaker. TMS in bipolar disorder. Edited by Charles M. Epstein, Eric M. Wassermann, and Ulf Ziemann. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780198568926.013.0041.

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This article reviews the existing animal and human literature on the clinical potential of transcranial magnetic stimulation (TMS) in mania and bipolar depression, and discusses potential future directions for this work. Studies of TMS in depression and normal volunteers suggested lateral specificity of TMS-induced mood effects. Clinical trials to compare left versus right prefrontal TMS in mania have been developed. Studies to understand the effect of TMS in bipolar depression have been undertaken. The results show efficacy similar to that for unipolar depression. But this does not provide support for the concept of TMS as an anti-bipolar, or mood-stabilizing, treatment. The utility of TMS as prophylaxis for subsequent manic or depressive episodes has not been reported in bipolar disorder. More work is needed to clarify the risk of mood switch, and the potential of TMS as prophylaxis against future manic or depressive episodes.

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Karen, Bellenir, ed. Depression sourcebook: Basic consumer health information about unipolar depression, bipolar disorder, postpartum depression, seasonal affective disorder, and other types of depression in children, adolescents, women, men, the elderly, and other selected populations ... Omnigraphics, 2002.

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Barlow, David H., Todd J. Farchione, Shannon Sauer-Zavala, et al. Unified Protocol for Transdiagnostic Treatment of Emotional Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med-psych/9780190685973.001.0001.

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The Unified Protocol (UP) for Transdiagnostic Treatment of Emotional Disorders: Therapist Guide is a treatment programv applicable to all anxiety and unipolar depressive disorders and potentially other disorders with strong emotional components (e.g., eating disorders, borderline personality disorder). The UP for the Transdiagnostic Treatment of Emotional Disorders addresses neuroticism by targeting the aversive, avoidant reactions to emotions that, while providing relief in the short term, increase the likelihood of future negative emotions and maintains disorder symptoms. The strategies included in this treatment are largely based on common principles found in existing empirically supported psychological treatments—namely, fostering mindful emotion awareness, reevaluating automatic cognitive appraisals, changing action tendencies associated with the disordered emotions, and utilizing emotion exposure procedures. The focus of these core skills has been adjusted to specifically address core negative responses to emotional experiences.

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Bertocci, Michele A., and Mary L. Phillips. Neuroimaging of Depression. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0025.

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This chapter illustrates the historical progression, methodological approaches, and current neurobiological understanding of depression, the first leading cause of mental and behavioral disorder disability in the United States. We describe and position, in relation to depressive symptoms, the complex abnormalities that depressed adults and youth show concerning neural function during tasks and at rest, structural abnormalities, as well as key neurotransmitter, neuroreceptor, and metabolic abnormalities that have been examined in the literature. We also describe newer findings and methods such as differentiating between unipolar and bipolar depression and applying machine learning to individual prediction. Finally, we provide suggestions for future study.

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The Unified Protocol for Transdiagnostic Treatment of Emotional Disorders: Workbook was developed to help people who are struggling with intense emotions like anxiety, sadness, anger, and guilt. A person may have an emotional disorder when his or her emotions are so overwhelming that they get in the way of moving forward in life. Although emotions affect our lives in different ways, there are three features that often occur across emotional disorders. These are (a) frequent, strong emotions; (b) negative reactions to emotions; and (c) avoidance of emotions. The goal of this workbook is to change the way that people with emotional disorders respond to their emotions when they occur. This treatment program is applicable to all anxiety and unipolar depressive disorders and potentially other disorders with strong emotional components. The strategies included in this treatment are largely based on common principles found in existing empirically supported psychological treatments.

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J, Judd Sandra, ed. Depression sourcebook: Basic consumer health information about unipolar depression, bipolar disorder, dysthymia, seasonal affective disorder, postpartum depression, and other depressive disorders, including facts about populations at special risk, coexisting medical conditions, symptoms, treatment options, and suicide prevention; along with statistical data, a glossary of related terms, and a directory of resources for additional help and information. 2nd ed. Omnigraphics, 2008.

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A spectrum approach to mood disorders: Not fully bipolar but not unipolar : practical management. 2016.

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Dallaspezia, Sara, and Francesco Benedetti. Sleep in other psychiatric disorders. Edited by Sudhansu Chokroverty, Luigi Ferini-Strambi, and Christopher Kennard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199682003.003.0048.

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There are complex relationships among sleep, sleep disorders, and psychiatric illnesses: not only can sleep abnormalities be symptoms of psychiatric disorders, but also some sleep disorders increase the risks of developing episodes of psychiatric disorders. During the past few decades, a number of sleep investigations have been performed in patients with the aim of identifying specific sleep patterns associated with psychiatric disorders. Although the majority of these studies have focused on major unipolar depression, sleep abnormalities have also been reported in other psychiatric disorders, such as bipolar disorder, schizophrenia, alcohol-related and drug abuse disorders, borderline personality disorder, and eating disorders.

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Bellenir, Karen. Depression Sourcebook: Basic Consumer Health Information About Unipolar Depression, Bipolar Disorder, Postpartum Depression, Seasonal Affective Disorder, and Other Types of (Health Reference Series). Omnigraphics, 2002.

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Yes You Can!: Commitment and Strategies for Overcoming Bipolar and Unipolar Depression. Regent Press, 2000.

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Parker, Gordon, and Amelia Paterson. Should the bipolar disorders be modelled dimensionally or categorically? Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198748625.003.0002.

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Historically, there have been categorical models of bipolar disorder and dimensional models of bipolar disorder. This chapter seeks to outline the history of these models as well as some recent supporting research. The models are evaluated in two ways; how well they reflect the underlying nature of bipolar disorder, and how useful they are to the patient and to the clinician in undertaking treatment decisions. The dimensional model posits that depression and bipolar lie on a continuum with pure unipolar depression at one end, bipolar disorder at the other, and some experience of highs without diagnosable (hypo)mania in-between. The categorical model posits that depression and bipolar are entirely separate conditions and that bipolar I and II are separate conditions. It is the position of the authors that the categorical model is a better reflection of the underlying nature of bipolar disorder and has proved more useful in clinical practice.

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DeRubeis, Robert J., and Daniel R. Strunk, eds. The Oxford Handbook of Mood Disorders. Oxford University Press, 2015. http://dx.doi.org/10.1093/oxfordhb/9780199973965.001.0001.

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Mood disorders are a pressing societal problem, with depression alone now constituting a leading cause of disability in Western Europe and the United States. In the most comprehensive volume of its kind, the Oxford Handbook of Mood Disorders provides detailed coverage of the characterization, understanding, and treatment of mood disorders. Chapters are written by the world’s leading experts in their respective areas. The Handbook provides coverage of unipolar depression, bipolar disorder, and variants of these disorders. Current approaches to classifying the mood disorders are reviewed, and contemporary controversies are placed in historical context. Chapter authors offer a variety of approaches to understanding the heterogeneity of the experiences of those who meet criteria for mood disorders, both within and across cultures. The role of genetic and environmental risk factors as well as premorbid personality and cognitive processes in the development of mood pathology are detailed. Interpersonal, neurobiological, and psychological factors also receive detailed consideration. The volume reviews mood disorders in special populations (e.g., postpartum and seasonal mood disorders) as well as common comorbidities (e.g., anxiety, substance use disorders). Somatic and psychosocial treatment approaches receive in-depth coverage with chapters that describe and review empirical evidence regarding each of the most influential treatment approaches. The depth and breadth offered by the Oxford Handbook of Mood Disorders make it an invaluable resource for clinicians and researchers, as well as for scholars and students.

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Rucker, James J. H., and Peter McGuffin. Copy Number Variation in Neuropsychiatric Disorders. Edited by Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.005.

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It has long been known that the human genome is subject to deletion and duplication of genetic material by various molecular mechanisms. Until recently, such events were assumed to be relatively rare phenomena. It is now known that submicroscopic deletions or duplications calledcopy number variants(CNVs) are a major source of genomic variation. Rare CNVs (defined as occurring in less than 1 percent of the population) have been implicated in schizophrenia and autism. Measured in terms of odds ratios, individual CNVs have been shown to have large effects, some increasing the risk of disorder several-fold. But they are incompletely penetrant, no one CNV is either necessary or sufficient to cause the disorder. The findings are less clear-cut with bipolar disorder but, here, too, rare CNVs probably play a role. In unipolar depression, initial evidence suggests an overall increase in rare CNVs that disrupt exons, the coding regions of genes.

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