Academic literature on the topic 'Urethral pressure'

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Journal articles on the topic "Urethral pressure"

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Prantil, Rachelle L., Ron J. Jankowski, Yasuhiro Kaiho, et al. "Ex vivo biomechanical properties of the female urethra in a rat model of birth trauma." American Journal of Physiology-Renal Physiology 292, no. 4 (2007): F1229—F1237. http://dx.doi.org/10.1152/ajprenal.00292.2006.

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Stress urinary incontinence (SUI) is the involuntary release of urine during sudden increases in abdominal pressures. SUI is common in women after vaginal delivery or pelvic trauma and may alter the biomechanical properties of the urethra. Thus we hypothesize that injury due to vaginal distension (VD) decreases urethral basal tone and passive stiffness. This study aimed to assess the biomechanical properties of the urethra after VD in the baseline state, where basal muscle tone and extracellular matrix (ECM) are present, and in the passive state, where inactive muscle and ECM are present. Fema
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Prantil-Baun, Rachelle, William C. de Groat, Minoru Miyazato, Michael B. Chancellor, Naoki Yoshimura, and David A. Vorp. "Ex vivo biomechanical, functional, and immunohistochemical alterations of adrenergic responses in the female urethra in a rat model of birth trauma." American Journal of Physiology-Renal Physiology 299, no. 2 (2010): F316—F324. http://dx.doi.org/10.1152/ajprenal.00299.2009.

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Birth trauma and pelvic injury have been implicated in the etiology of stress urinary incontinence (SUI). This study aimed to assess changes in the biomechanical properties and adrenergic-evoked contractile responses of the rat urethra after simulated birth trauma induced by vaginal distension (VD). Urethras were isolated 4 days after VD and evaluated in our established ex vivo urethral testing system that utilized a laser micrometer to measure the urethral outer diameter at proximal, middle, and distal positions. Segments were precontracted with phenylephrine (PE) and then exposed to intralum
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Le Feber, Joost, Els Van Asselt, and Ron Van Mastrigt. "Neurophysiological modeling of voiding in rats: urethral nerve response to urethral pressure and flow." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 274, no. 5 (1998): R1473—R1481. http://dx.doi.org/10.1152/ajpregu.1998.274.5.r1473.

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In male urethan-anesthetized rats, activity was measured in nerves that run over the proximal urethra. The urethral nerve response to stepwise urethral perfusion could be described by a four-parameter model (fit error <6%). At the onset of perfusion, the urethra was closed and the pressure increased with the infused volume. The nerve activity (NA) increased linearly with this inserted volume to a maximum (NAmax), which was proportional to the instantaneous pressure. The duration of this first episode (δ t) was inversely proportional to the perfusion rate. After infusion of a fixed volume, t
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Ruiz, Sonia, Miguel Virseda-Chamorro, Fabian Queissert, Andrés López, Ignacio Arance, and Javier C. Angulo. "The Mode of Action of Adjustable Transobturator Male System (ATOMS): Intraoperative Urethral Pressure Measurements." Uro 1, no. 2 (2021): 45–53. http://dx.doi.org/10.3390/uro1020007.

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(1) Background: The Adjustable Transobturator Male System (ATOMS) device is increasingly used to treat post-prostatectomy incontinence as it enhances residual urinary sphincteric function and allows continence recovery or improvement by dorsal compression of the bulbar urethra through a fixed transobturator mesh passage. The mode of action and the profile of the patients with best results are not totally understood. (2) Methods: Intraoperative urethral pressure measurements at different filling levels of the ATOMS device show increased urethral resistance and enhanced residual sphincteric acti
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Souza, Ana Beatriz Gomes de, Haylton Jorge Suaid, Carla Adelino Suaid, Silvio Tucci Jr, Adauto José Cologna, and Antonio Carlos Pereira Martins. "Comparison of two experimental models of urodynamic evaluation in female rats." Acta Cirurgica Brasileira 23, suppl 1 (2008): 59–65. http://dx.doi.org/10.1590/s0102-86502008000700011.

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PURPOSE: Urodynamic studies in small animals can be performed through urethral sounding or cystostomy. OBJECTIVE: To compare the two methods of urodynamic evaluation in female rats. METHODS: Ten female rats weighing on average 250g, under anesthesia with urethane (1,25 mg/kg) were submitted in three repeats to an urethal catheter of 0,64 mm in external diameter for cystometric measurements of vesicle pressure(VP1) and contraction time (CT1). The catheter was extracted at a constant velocity of 0.05 cm/minute until complete exteriorization and determinations of maximal urethral pressure (UP1) a
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Julia-Guilloteau, V., P. Denys, J. Bernabé, et al. "Urethral closure mechanisms during sneezing-induced stress in anesthetized female cats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 293, no. 3 (2007): R1357—R1367. http://dx.doi.org/10.1152/ajpregu.00003.2007.

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During stress-induced increase in abdominal pressure, urinary continence is maintained by urethral closure mechanisms. Active urethral response has been studied in dogs and rats. Such an active urethral response is also believed to occur in humans during stress events. We aimed to investigate urethral closure mechanisms during sneezing in cats. Urethral pressures along the urethra (UP1–UP4), with microtip transducer catheters with UP4 positioned in the distal urethra where the external urethral sphincter (EUS) is located, and intravesical pressure were measured, and abdominal wall, anal sphinc
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Kamo, Izumi, Tracy W. Cannon, Deirdre A. Conway, et al. "The role of bladder-to-urethral reflexes in urinary continence mechanisms in rats." American Journal of Physiology-Renal Physiology 287, no. 3 (2004): F434—F441. http://dx.doi.org/10.1152/ajprenal.00038.2004.

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Urethral closure mechanisms during passive increments in intravesicular pressure (Pves) were investigated using microtip transducer catheters in urethane-anesthetized female rats. After a block of reflex bladder contractions by spinal cord transection at T8-T9, abruptly raising Pves to 20, 40, or 60 cmH2O for 2 min induced a bladder pressure-dependent contractile response in a restricted portion of the middle urethra (12.5–15 mm from the urethral orifice) that was abolished by cutting the pelvic nerves bilaterally. In pelvic nerve-intact rats, the bilateral transection of either the pudendal n
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Mackiewicz, Agnieszka G., Tomasz Klekiel, Jagoda Kurowiak, Tomasz Piasecki, and Romuald Bedzinski. "Determination of Stent Load Conditions in New Zealand White Rabbit Urethra." Journal of Functional Biomaterials 11, no. 4 (2020): 70. http://dx.doi.org/10.3390/jfb11040070.

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Background: Frequency of urethral stenosis makes it necessary to develop new innovative methods of treating this disease. This pathology most often occurs in men and manifests itself in painful urination, reduced urine flow, or total urinary retention. This is a condition that requires immediate medical intervention. Methods: Experimental tests were carried out on a rabbit in order to determine the changes of pressure in the urethra system and to estimate the velocity of urine flow. For this purpose, a measuring system was proposed to measure the pressure of a fluid-filled urethra. A fluorosco
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Kamo, Izumi, Kazumasa Torimoto, Michael B. Chancellor, William C. de Groat, and Naoki Yoshimura. "Urethral closure mechanisms under sneeze-induced stress condition in rats: a new animal model for evaluation of stress urinary incontinence." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 285, no. 2 (2003): R356—R365. http://dx.doi.org/10.1152/ajpregu.00010.2003.

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The urethral closure mechanism under a stress condition induced by sneezing was investigated in urethane-anesthetized female rats. During sneezing, while the responses measured by microtip transducer catheters in the proximal and middle parts of the urethra increased, the response in the proximal urethra was almost negligible when the bladder response was subtracted from the urethral response or when the abdomen was opened. In contrast, the response in the middle urethra during sneezing was still observed after subtracting the bladder response or after opening the abdomen. These responses in t
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Kamo, Izumi, and Tadatoshi Hashimoto. "Involvement of reflex urethral closure mechanisms in urethral resistance under momentary stress condition induced by electrical stimulation of rat abdomen." American Journal of Physiology-Renal Physiology 293, no. 3 (2007): F920—F926. http://dx.doi.org/10.1152/ajprenal.00466.2006.

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A novel method for evaluating the urethral resistance during abrupt elevation of abdominal pressure was developed in spinalized female rats under urethane anesthesia. Electrical stimulation of abdominal muscles for 1 s induced increases in both the intra-abdominal and the intravesical pressure in a stimulus-dependent manner, and the bladder response was almost lost when the abdomen was opened. The lowest intravesical pressure during electrical stimulation that induced fluid leakage from the urethral orifice (leak point pressure) and the maximal intravesical pressure without urine leakage below
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Dissertations / Theses on the topic "Urethral pressure"

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Balog, Brian Michael. "Brain-Derived Neurotrophic Factor Mediates Recovery from Stress Urinary Incontinence." University of Akron / OhioLINK, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=akron1602113592326106.

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Kwong-Pang and 徐廣鵬. "Changes of Urethral Pressure Profilometry in Pubovaginal Sling and Modified Burch Colposuspension Procedures." Thesis, 2005. http://ndltd.ncl.edu.tw/handle/81462699089571568324.

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碩士<br>中山醫學大學<br>醫學研究所<br>93<br>OBJECTIVE: To evaluate the pre-operative and post-operative difference effects on the urethral pressure profilometry in pubovaginal slingplasty and modified Burch colposuspension procedures. DESIGN: This is a descriptive study of urodynamic parameter and urethral pressure profilometry before and after operation. All the patients were followed at least 24 months after surgery. METHODS: A total of 63 patients with successful anti-incontinence surgery were recruited to this study. Thirty-eight women had received a modified Burch colposuspension (MBC) before 199
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MADILL, STEPHANIE. "Differences in pelvic floor muscle activation and functional output between women with and without stress urinary incontinence." Thesis, 2009. http://hdl.handle.net/1974/5185.

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Introduction: The primary purpose of this research was to determine whether women with stress urinary incontinence (SUI) demonstrate pelvic floor muscle (PFM) strength or endurance deficits and/or changes in the motor control patterns used during maximum voluntary PFM contractions (PFM MVCs) and coughing. A secondary purpose was to determine the effect of age on these parameters. Methods: After first validating the use of vaginal pressure to study the functional output of the PFMs, three studies were carried out to address these objectives. In two studies vaginal pressure and PFM and abdominal
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Books on the topic "Urethral pressure"

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Frenier, Susan L. Urethral pressure response to alpha adrenergic agonist and antagonist drugs in the normal male cat. 1990.

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Marks, Steven L. The effects of phenoxybenzamine and acepromazine maleate on urethral pressure profiles of anesthetized healthy male cats. 1993.

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Daly, Donna, and Christopher Chapple. Anatomy, neurophysiology, and pharmacological control mechanisms of the bladder. Edited by Christopher R. Chapple. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0034.

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The lower urinary tract has two main functions; the collection and low pressure storage of urine and periodical controlled elimination of urine at an appropriate time. In order to achieve continence during bladder filling and storage and produce efficient and effective bladder emptying, there is accurate coordination between opening and closing of the urethral sphincters and contraction of the detrusor smooth muscle. The process of micturition has two phases: the storage/filling phase and the voiding phase. The analogy for the transition between these two phases has been described as an on-off
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Jones, Julie Ellis, and Hashim Hashim. Urodynamics. Edited by Christopher R. Chapple. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0035.

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Urodynamic studies encompass several tests to investigate the hydrodynamics of the lower urinary tract during bladder storage/filling and voiding. These tests include bladder diaries, free uroflowmetry, and post-void residuals, filling cystometry, voiding pressure/flow studies, urethral pressure profiles, leak point pressures, videourodynamics, ambulatory urodynamics, and electromyography. The tests are performed after formulating a urodynamics question to objectively observe lower urinary tract function and dysfunction with the idea of choosing an appropriate treatment for the pathology. Inva
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Lee, Olivia T., Jennifer N. Wu, Frederick J. Meyers, and Christopher P. Evans. Genitourinary aspects of palliative care. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0084.

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Genitourinary tract diseases in the palliative care setting most commonly involve urinary tract obstruction, intractable bleeding, fistulae, and bladder-associated pain. Sources of obstruction in the lower urinary tract include benign prostatic hyperplasia, invasive prostate or bladder cancer, urethral stricture, or bladder neck contracture. Upper tract obstruction includes intraluminal or extraluminal blockage of the renal collecting system and ureters, such as transitional cell carcinoma, fibroepithelial polyps, stricture, stones, pelvic or retroperitoneal malignancy, fibrosis, or prior radi
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Book chapters on the topic "Urethral pressure"

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Sand, Peter K., and Donald R. Ostergard. "Urethral Closure Pressure Profiles." In Urodynamics and the Evaluation of Female Incontinence. Springer London, 1995. http://dx.doi.org/10.1007/978-1-4471-2109-1_15.

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Sand, Peter K., and Donald R. Ostergard. "Augmenting Urethral Closure Pressure Profiles." In Urodynamics and the Evaluation of Female Incontinence. Springer London, 1995. http://dx.doi.org/10.1007/978-1-4471-2109-1_16.

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Wen, Jian Guo. "Cystometry, Pressure Flow Study and Urethral Pressure Measurement." In Urodynamics, Neurourology and Pelvic Floor Dysfunctions. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-42193-3_8.

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Versi, E. "Relevance of Urethral Pressure Profilometry To Date." In Micturition. Springer London, 1990. http://dx.doi.org/10.1007/978-1-4471-1780-3_6.

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van Geelen, J. M. "The value of the urethral pressure profile." In Practical Aspects of Urinary Incontinence. Springer Netherlands, 1986. http://dx.doi.org/10.1007/978-94-009-4237-0_4.

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Sand, Peter K., and Donald R. Ostergard. "Dynamic Urethral Closure Pressure Profiles: Valsalva and Cough Profiles." In Urodynamics and the Evaluation of Female Incontinence. Springer London, 1995. http://dx.doi.org/10.1007/978-1-4471-2109-1_17.

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Sand, Peter K., and Donald R. Ostergard. "Pelvic Floor Stimulation: Effect on Urethral Closure Pressure Profile." In Urodynamics and the Evaluation of Female Incontinence. Springer London, 1995. http://dx.doi.org/10.1007/978-1-4471-2109-1_40.

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Sand, Peter K., and Donald R. Ostergard. "The Effect of Pudendal Block on the Urethral Closure Pressure Profile." In Urodynamics and the Evaluation of Female Incontinence. Springer London, 1995. http://dx.doi.org/10.1007/978-1-4471-2109-1_42.

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Sand, Peter K., and Donald R. Ostergard. "The Effect of Retropubic Urethropexy on the Resting Urethral Closure Pressure Profile." In Urodynamics and the Evaluation of Female Incontinence. Springer London, 1995. http://dx.doi.org/10.1007/978-1-4471-2109-1_49.

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Sand, Peter K., and Donald R. Ostergard. "The Effect of Retropubic Urethropexy on the Dynamic Urethral Closure Pressure Profile." In Urodynamics and the Evaluation of Female Incontinence. Springer London, 1995. http://dx.doi.org/10.1007/978-1-4471-2109-1_50.

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Conference papers on the topic "Urethral pressure"

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Ahmadi, Mahdi, Rajesh Rajamani, Gerald Timm, and A. S. Sezen. "Distributed pressure sensors for a urethral catheter." In 2015 37th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2015. http://dx.doi.org/10.1109/embc.2015.7320154.

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Klunder, Mario, Ronny Feuer, Bastian Amend, et al. "Eliminating pulse-induced artifacts in Urethral Pressure data." In 2015 37th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2015. http://dx.doi.org/10.1109/embc.2015.7318968.

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Ahmadi, M., Y. Zhang, R. Rajamani, G. Timm, and A. S. Sezen. "A Super-Capacitive Pressure Sensor for a Urethral Catheter." In 2018 40th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2018. http://dx.doi.org/10.1109/embc.2018.8513064.

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Klunder, Mario, Oliver Sawodny, and Ronny Feuer. "Increasing spatial resolution through automated catheter rotation in high definition urethral pressure profilometry." In 2016 IEEE International Conference on Automation Science and Engineering (CASE). IEEE, 2016. http://dx.doi.org/10.1109/coase.2016.7743377.

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Spirka, Thomas, Kimberly Kenton, Linda Brubaker, and Margot Damaser. "Pathway to Finite Element Analysis of Stress Urinary Incontinence Mechanics." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53050.

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Stress urinary incontinence (SUI), a condition that affects mainly women, is characterized by the involuntary leakage of urine caused by an increase in abdominal pressure in the absence of a bladder contraction that raises the vesical (bladder) pressure to a level that exceeds the urethral closure pressure.
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Cargill, Robert S., Kevin K. Toosi, and Edward J. Macarak. "Mechanical Properties of the Fetal Bovine Bladder Lamina Propria and Their Correlation With Changes in Extracellular Matrix." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193131.

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The urinary bladder is an organ whose purpose is to store urine at low pressure and periodically expel it. This system normally operates at relatively low pressure to protect the kidneys from the deleterious effects of increased pressure. In certain pathologies, this organ can be subject to a decrease in compliance (“stiffening”) and an increase of the storage pressure which causes higher back pressure on the kidney and ultimately results in kidney damage if untreated. Clinically, these pathologies are exemplified in disorders such as myelomeningocele, posterior urethral valves, dysfunctional
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Prantil, Rachelle L., Ronald J. Jankowski, Kazumasa Torimoto, William C. de Groat, Michael B. Chancellor, and David A. Vorp. "Effects of Diabetes Mellitus on the Biomechanical Properties of the Female Rat Urethra in the Passive State." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43009.

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Patients with diabetes mellitus (DM) suffer impaired lower urinary tract dysfunctions. The purpose of the current study was to evaluate the effects of DM on the passive biomechanical properties of the female rat urethra. DM was induced by injection of streptozotocin. Urethras were excised and mounted in an ex-vivo testing system. EDTA was added to the bath to inactivate smooth muscle. Continuous outer diameter measurements were made at proximal, middle, and distal portions of the urethra with a laser micrometer during stepwise increases of static, intraurethral pressure (0 to 20 mmHg). Complia
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Champaigne, Kevin D., Sarette N. Jenderny, and Jiro Nagatomi. "Electrophysiological Investigation of Hydrostatic Pressure Mechanotransduction by Urothelial Cell Lines." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53518.

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The urothelium is the epithelial lining of the ureters, urinary bladder, and urethra. Recent discoveries have suggested that in addition to providing a barrier function to urine, the urothelium actively participates in sensory functions related to thermal, chemical, and mechanical stimuli, and releases chemical signals in response[1]. In addition to a sensitivity to cell membrane stretch caused by wall tension upon bladder filling, in vitro studies by our group have shown that urothelial cells may be sensitive to hydrostatic pressure directly without requiring membrane stretching [2]. Specific
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Bertram, Christopher D., Nicholas K. Truong, and Stephen D. Hall. "PIV Experiments to Measure the Velocity Field Just Downstream of an Oscillating Collapsible Tube." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176287.

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Almost all vascular conduits in the human body, being flexible, collapse elastically to a non-circular cross-section when the external pressure sufficiently exceeds the internal. Examples include the brachial artery (sphygmomanometry), veins (everyday manoeuvres), pulmonary airways (forced expiration), pulmonary capillaries (zones 1 and 2), and the urethra (micturition). Venous collapse is involved in regulation of venous return, which in turn regulates cardiac output.
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Reports on the topic "Urethral pressure"

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Zhu, Zhihong, Yue Zhuo, Haitao Jin, Boyu Wu, and Zhijie Li. Chinese Medicine Therapies for Neurogenic Bladder after Spinal Cord Injury: A protocol for systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.8.0084.

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Neurogenic bladder (NB), a refractory disease, is characterized by voiding dysfunction of bladder and/or urethra, and spinal cord injury (SCI) is a common cause. Chinese medicine therapies have been applied extensively in the treatment of neurogenic bladder, especially in China, and the results are promising but varying. Thus, the aim of this work is to assess the efficacy and safety of various Chinese medicine therapies for neurogenic bladder after spinal cord injury. Condition being studied: Chinese medicine therapies; Neurogenic bladder after spinal cord injury. Main outcome(s): The primary
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