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Books on the topic 'Urinary system disease'

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Published: 4 June 2021
Last updated: 7 February 2022

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1

Thomas, Robert (Robert H.)., ed. Renal and urinary systems. 4th ed. Edinburgh: Elsevier, 2013.

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2

Field, Michael. The renal system. 2nd ed. Edinburgh: Churchill Livingstone, 2010.

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3

Field, Michael J. The renal system. Edinburgh: Harcourt Publishers, 2001.

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4

Field, Michael J. The renal system. Edinburgh: Churchill Livingstone, 2001.

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5

A, Pollock Carol, and Harris, David C. (David Charles), 1953-, eds. The renal system: Basic science and clinical conditions. 2nd ed. Edinburgh: Churchill Livingstone/Elsevier, 2010.

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6

Field, Michael. The renal system: [basic science and clinical conditions]. Edinburgh: Harcourt Publishers, 2001.

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7

M, Hutson John, and Smith E. Durham, eds. Congenital anomalies of the urinary and genital tracts. Oxford: Isis Medical Media, 1996.

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8

A, O'Callaghan C., ed. The renal system at a glance. 2nd ed. Malden, Mass: Blackwell Pub., 2005.

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9

The renal system at a glance. 3rd ed. Chichester, West Sussex: Wiley-Blackwell, 2009.

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10

K, Wong Newton W., and ebrary Inc, eds. The renal system explained: An illustrated core text. Nottingham: Nottingham University Press, 2009.

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11

Anne, McGinn Kerry, ed. The ostomy book: Living comfortably with colostomies, ileostomies, and urostomies. Boulder, CO: Bull Pub., 2008.

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12

Mullen, Barbara Dorr. The ostomy book: Living comfortably with colostomies, ileostomies, and urostomies. Palo Alto, Calif: Bull Pub. Co., 1992.

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13

Rao, Satish S. C. Disorders of the Pelvic floor and Anorectum. Philadelphia, Pa: Saunders, 2008.

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14

Pathobiology of the Aging Mouse: General Aspects, Endocrine System, Blood and Lymphoid System, Respiratory System, Urinary System, Cardiovascular System, and Reproductive System. Not Avail, 1996.

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15

Pelvic Organ Dysfunction In Neurological Disease Clinical Management And Rehabilitation. Cambridge University Press, 2010.

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16

J, Droller Michael, ed. Surgical management of urologic disease: An anatomic approach. St. Louis: Mosby Year Book, 1992.

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17

Carlyle, Jones Thomas, Mohr U, and Hunt Ronald Duncan, eds. Urinary system. Berlin: Springer-Verlag, 1986.

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18

Urinary system. 2nd ed. Berlin: Springer, 1998.

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19

Jones, Thomas C., Ulrich Mohr, and Gordon C. Hard. Urinary System. Springer, 2011.

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20

Jones, Thomas C., and Wolfram Clauß. Urinary System. Springer, 2012.

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21

The Urinary System (Human Body Systems). Greenwood Press, 2004.

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22

Yanhua, Ge, and Tong Yanling, eds. Jie shi bing jian kang yi ji. Changchun: Jilin ke xue ji shu chu ban she, 2004.

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23

Huiying, Zhao, ed. Xin nao xue guan ji bing jian kang yi ji. Changchun: Jilin ke xue ji shu chu ban she, 2004.

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24

(Firm), Medicode, ed. Urinary system. Salt Lake City, UT: Medicode, 1995.

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25

1930-, Schofield P. F., and Lupton E. W. 1946-, eds. The Causation and clinical management of pelvic radiation disease. London: Springer-Verlag, 1989.

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26

Heyns, Chris. Tuberculosis and parasitic infestations involving the urogenital system. Edited by Rob Pickard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0006.

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Urogenital tuberculosis is caused by Mycobacterium tuberculosis, which evokes a granulomatous tissue reaction leading to caseous necrosis, fibrosis, and eventual calcification. It most commonly presents as cystitis with sterile pyuria but can show many other symptoms and signs requiring a high index of suspicion to make the diagnosis. Schistosomiasis (Bilharzia) affecting the urinary tract is caused by the flatworm Schistosoma haematobium. Humans are infested by contact with fresh water harbouring the intermediate snail host. Echinococcosis (hydatid disease), is caused by the tapeworm Echinococcus granulosis or multilocularis. Human infection results from close contact with the parasite host (usually dogs and sheep). Filariasis, caused by the roundworm Wuchereria bancrofti, is transmitted by mosquito bite
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27

Bowker, Lesley K., James D. Price, Ku Shah, and Sarah C. Smith. Infection and immunity. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0024.

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This chapter provides information on the ageing immune system, an overview of infection in older people, antibiotic use in older patients, meticillin-resistant Staphylococcus aureus (MRSA), disease caused by MRSA, Clostridium difficile-associated diarrhoea, near-patient urine tests, asymptomatic bacteriuria, urinary tract infection, treatment of urinary tract infection, recurrent urinary tract infection, and varicella-zoster infection.
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28

E, Riddle Janet T., and Stewart Elizabeth M, eds. The Digestive system and the urinary system. Edinburgh [Scotland]: Churchill Livingstone, 1989.

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29

Renal and Urinary System (Crash Course - US). Mosby, 1998.

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30

Renal and Urinary Systems (Crash Course). 2nd ed. C.V. Mosby, 2003.

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31

Panicker, Jalesh N., and Clare J. Fowler. Non-traumatic neurourology. Edited by Christopher R. Chapple. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0046.

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This chapter reviews bladder disturbances in non-traumatic neurological conditions and provides an approach to its evaluation and management. The pattern of bladder dysfunction depends upon the level of neurological localisation and accordingly, lesions can be suprapontine, infrapontine/suprasacral (spinal), or infrasacral. The importance of the frontal lobes for bladder control has been confirmed and vascular disease or tumour can result in incontinence. There is better understanding about the very different urological profile of the two sometimes confused conditions, multiple system atrophy and Parkinson’s disease. Guidelines for the management of lower urinary tract dysfunction in multiple sclerosis are reviewed. Lower urinary tract (LUT) dysfunction is common in neurological disease and its importance to patient health and quality of life is now widely recognized.
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32

Urinary System (Monographs of Pathology of Laboratory Animals). Springer-Verlag, 1987.

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33

Netter Collection of Medical Illustrations - Urinary System: Volume 5. Elsevier - Health Sciences Division, 2012.

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34

Crash Course Renal and Urinary System Updated Print + EBook Edition. Elsevier - Health Sciences Division, 2015.

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35

Davey, Patrick, and David Sprigings, eds. Diagnosis and Treatment in Internal Medicine. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.001.0001.

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Diagnosis and Treatment in Internal Medicine is a new textbook, written by experts in their field, that provides succinct and authoritative guidance across the breadth of internal medicine. Diagnosis is the bedrock of management, and so how to reach a differential diagnosis of symptoms or presenting problems is a major element of the book. There is also comprehensive coverage of disorders of the body systems, including psychological aspects and palliative care. Chapters are structured so that key information can rapidly be found. Doctors need a broad perspective on health and its promotion, and there are sections addressing nutrition, lifestyle and prevention of disease. Diagnosis and Treatment in Internal Medicine is the ideal reference for doctors early in their careers in hospital medicine or primary care, and senior medical students. Sections of the book: • The approach to the patient • Assessment of symptoms and presenting problems • Cardiovascular disorders • Respiratory disorders • Intensive care medicine • Disorders of the kidney and urinary tract • Diabetes mellitus and endocrine disorders • Gastro-intestinal disorders • Disorders of the liver • Neurological disorders • Disorders of the skin • Disorders of the musculoskeletal system • Haematological disorders • Disorders of the immune system • Infectious diseases • Nutrition and its disorders • Lifestyle and environmental causes of disease • Prevention of disease • Screening for disease
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36

Barsoum, Rashad S. Schistosomiasis. Edited by Vivekanand Jha. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0194_update_001.

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The urinary system is the primary target of Schistosoma haematobium infection, which leads to granuloma formation in the lower urinary tract that heals with fibrosis and calcification. While the early lesions may be associated with distressing acute or subacute symptoms, it is the late lesions that constitute the main clinical impact of schistosomiasis. The latter include chronic cystitis, ureteric fibrosis, ureterovesical obstruction or reflux which may lead to chronic pyelonephritis. Secondary bacterial infection and bladder cancer are the main secondary sequelae of urinary schistosomiasis.The kidneys are also a secondary target of S. mansoni infection, attributed to the systemic immune response to the parasite. Specific immune complexes are responsible for early, often asymptomatic, possibly reversible, mesangioproliferative lesions which are categorized as ‘class I’. Subsequent classes (II–VI) display different histopathology, more serious clinical disease, and confounding pathogenic factors. Class II lesions are encountered in patients with concomitant salmonellosis; they are typically exudative and associated with acute-onset nephrotic syndrome. Classes III (mesangiocapillary glomerulonephritis) and IV (focal segmental sclerosis) are progressive forms of glomerular disease associated with significant hepatic pathology. They are usually associated with immunoglobulin A deposits which seem to have a significant pathogenic role. Class V (amyloidosis) occurs with long-standing active infection with either S. haematobium or S. mansoni. Class VI is seen in patients with concomitant HCV infection, where the pathology is a mix of schistosomal and cryoglobulinaemic lesions, as well as amyloidosis which seems to be accelerated by the confounded pathogenesis.Early schistosomal lesions, particularly those of the lower urinary tract, respond to antiparasitic treatment. Late urological lesions may need surgery or endoscopic interventions. As a rule, glomerular lesions do not respond to treatment with the exception of class II where dual antiparasitic and antibiotic therapy is usually curative. Patients with end-stage kidney disease may constitute specific, yet not insurmountable technical and logistic problems in dialysis or transplantation. Recurrence after transplantation is rare.
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37

Neligan, Patrick J., and John G. Laffey. Obstetric physiology and special considerations in ICU. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0365.

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Pregnant patients constitute less than 1% of intensive care unit admissions, and fewer than 1% of obstetric patients become critically ill. Critical illness may result from pregnancy-specific diseases, diseases that pregnancy predisposes to, or are co-incidental to pregnancy. The presence of a second patient—the foetus—may necessitate adjustments to therapeutic and supportive strategies. However, the foetus is generally robust despite maternal illness. The physiological changes of pregnancy are significant, but may delay the diagnosis of critical illness, requiring modifications to standard management approaches. These include increases in minute ventilation, resulting in a ‘low normal’ PaCO2, a reduction in mean arterial pressure, but increased heart rate, low serum creatinine, relative hypoglycaemia, relative leukocytosis, and reduced lower oesophageal sphincter tone. Pre-eclampsia is a disease of the uteroplacental unit that results in abnormal maternal physiology. Pregnant women are at risk for acute respiratory distress syndrome, due to gastropulmonary aspiration and increased risk of community-acquired pneumonia, sepsis, principally of the genito-urinary system, and thromboembolic disease.
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38

Sobel, Jack D. Genito-urinary fungal infections. Edited by Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum, and Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0027.

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The predominant fungal causes of genitourinary disease are Candida spp.; other fungal genera are uncommon pathogens in both sexes. Vulvovaginal candidiasis affects millions of women worldwide—and includes acute sporadic, recurrent, and chronic syndromes—and considerable progress has been made in understanding its pathophysiology and hence the best therapy. Therapeutic options are still limited, however, and misdiagnosis is common. In contrast, urinary tract candidiasis reflects an entirely different pathogenesis and clinical expression affecting a predominantly hospital-based older population. Candida organisms are extremely difficult to eradicate from often complicated urinary tract infections. Non-Candida fungal species reach the kidney and prostate by the bloodstream rather than the ascending route taken by Candida spp. In women, not infrequently, there is simultaneous lower genital tract and urinary tract infection, requiring attention to both systems.
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39

Teoh, Eugene, and Michael J. Weston. Computed tomography. Edited by Christopher G. Winearls. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0014.

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Computed tomography (CT) has increased in use exponentially for the assessment of patients with renal tract pathology. This has been promoted by the availability of multidetector thin-slice CT so that intravenous urography has been superseded by CT urography. The latter may be considered as a ‘one-stop’ imaging investigation for haematuria, with increased detection of both urinary tract cancers and urolithiasis. Multiplanar reformats are made possible with the use of thin slices, allowing clear delineation of other pathologies such as urinary tract injury. In the transplant recipient, protocols have been developed for the assessment of more immediate complications such as thrombotic and stenotic disease. During follow-up, CT continues to inform the management of post-transplant lymphoproliferative disorder and other immunosuppressant-related complications. Unenhanced CT of the urinary tract has established its role in assessment of patients with renal colic, with the ability to detect pathology outside of the urinary tract. Renal CT has been developed for the characterization of renal masses, accompanied by the now well-established Bosniak renal cyst classification system. As the usefulness of CT increases, clear awareness of safety issues has to be maintained. These include the administration of intravenous iodinated contrast medium in higher-risk patient groups, particularly those with renal impairment. The radiation burden that comes with CT poses an added risk to the patient that should not be ignored. This necessitates clear referral guidelines for its use, which should be applied in careful balance with the global assessment of the patient.
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40

World Health Organization (WHO). Diseases of the Kidney, the Lower Urinary Tract & the Male Genital System Cioms (International Nomenclature of Diseases). World Health Organization, 1992.

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41

International Nomenclature of Diseases: Diseases of the Kidney, the Lower Urinary Tract, and the Male Genital System. The Stationery Office Books (Agencies), 1992.

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42

Wiles, Kate, Kate Bramham, and Catherine Nelson-Piercy. Kidney disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198713333.003.0044.

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This chapter describes the physiological adaptations to pregnancy in women with and without renal disease, reports pregnancy outcomes in women with both acute kidney injury and chronic kidney disease, and discusses a management strategy for antenatal and peripartum care. Acute kidney injury (AKI) is difficult to define in pregnancy because of the physiological increase in glomerular filtration. A normal creatinine can mask renal injury in pregnancy. This chapter considers important causes of AKI in pregnancy including pre-eclampsia, HELLP syndrome, thrombotic microangiopathy, acute fatty liver of pregnancy, systemic lupus erythematosus, urinary tract infection, and obstruction. The trend in the developed world for delaying pregnancy and the increasing prevalence of obesity mean that greater numbers of pregnancies will be complicated by chronic kidney disease. Maternal and fetal complications increase with worsening prepregnancy renal function including the development of pre-eclampsia, fetal growth restriction, premature delivery, and fetal loss. Prepregnancy counselling and the intrapartum management for women with lupus nephritis, immunoglobulin A nephropathy, polycystic kidney disease, and diabetic nephropathy are discussed. Renal replacement therapies in pregnancy including both dialysis and renal transplantation are considered, and practical guidance on renal biopsy, anaesthesia, and the pharmacology of renal disease in pregnancy is offered.
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43

Herbal formularies for health professionals: Digestion and elimination, including the gastrointestinal system, liver and gallbladder, urinary system, and the skin. 2018.

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44

Urinary Tract And Kidney Diseases And Disorders Sourcebook: Basic Consumer Health Information About the Urinary System, Including the Bladder, Urethra, ... Reference Series) (Health Reference Series). 2nd ed. Omnigraphics, Inc., 2005.

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45

Said, Neveen, and Dan Theodorescu. Molecular biology of bladder cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0071.

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Bladder cancer is the most common malignancy involving the urinary system, caused primarily by tobacco use and exposure to industrial chemicals with an estimated 73,510 patients affected and 14,880 deaths in 2012. This chapter will summarize what is known about the most common molecular derangements in human bladder cancer. It will focus on the function and biological/clinical relevance of these genes in models of urothelial cancer and in patients with this disease. It is not meant as a comprehensive review of all the functions of the aforementioned genes in normal physiology or other cancer types. Furthermore, the selection of what genes/pathways are described is by necessity empirical and so we apologize to any author whose work was not described or quoted.
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46

(Editor), Richard Naftalin, ed. One Stop Doc Renal and Urinary System and Electrolyte Balance (One Stop Doc). A Hodder Arnold Publication, 2005.

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47

Daudon, Michel, and Paul Jungers. Cystine stones. Edited by Mark E. De Broe. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0203_update_001.

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Cystinuria, an autosomal recessive disease (estimated at 1:7000 births worldwide), results from the defective reabsorption of cystine and dibasic amino acids (also ornithine, arginine, lysine, COAL) by epithelial cells of renal proximal tubules, leading to an abnormally high urinary excretion of these amino acids. Due to the poor solubility of cystine at the usual urine pH, formation of cystine crystals and stones ensues. Incidence of homozygotes is estimated at 1 in 7000 births worldwide, but is lower in European countries and much higher in populations with frequent consanguinity. Cystine stones represent 1–2% of all stones in adults and 5–8% in paediatric patients, with an equal distribution between males and females.Cystinuria is caused by inactivating mutations in the gene SLC3A1 or SLC7A9, both encoding proteins contributing to the function of the heterodimeric transport system of cystine.Cystine nephrolithiasis may present in infants, most frequently in adolescents or young adults, sometimes later. Cystine calculi are weakly radio-opaque. Stone analysis using infrared spectroscopy (or X-ray diffraction) allows immediate and accurate diagnosis. Urinary amino acid chromatography quantifies urinary cystine excretion, needed to define the therapeutic strategy.Urological treatment of cystine stones currently uses extracorporeal stone wave lithotripsy or flexible ureterorenoscopy with Holmium laser, that is, minimally invasive techniques. However, as cystine stones are highly recurrent, preventive therapy is essential.Medical treatment combines reduced methionine and sodium intake, to lower cystine excretion; hyperdiuresis (> 3 L/day) to reduce cystine concentration; and active alkalinization preferably using potassium citrate (40–80 mEq/day) to increase cystine solubility by rising urine pH up to 7.5–8. If these measures are insufficient to prevent recurrent stone formation, a thiol derivative (D-penicillamine or tiopronin), which converts cystine into a more soluble disulphide, should be added. Close monitoring and adherence of the patient to the therapeutic programme are needed to ensure life-long compliance, the key for successful prevention in the long term.
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48

Stephens, F., and F. d. Stephens. Congenital Anomalies of The Urinary and Genital Tracts. Taylor & Francis, 1996.

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49

R, Sant Grannum, ed. Pathophysiologic principles of urology. Boston: Blackwell Scientific Publications, 1994.

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50

Wilson, John W., and Lynn L. Estes. Infectious Syndromes in Adults. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190696924.003.0005.

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This section contains tables and text covering an exhaustive group of infectious syndromes including respiratory tract infections, infective endocarditis, intravascular catheter-related infections, central nervous system infections, urinary tract infections, soft-tissue infections, osteomyelitis, gastrointestinal infections, tick-borne infections, tuberculosis, sexually transmitted diseases, HIV, hepatitis, and fungal and zoonotic infections. Vaccination schedules, travel medicine, and bioterrorism are also reviewed.
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