Log in

Relevant bibliographies by topics / Urobilinogen

Contents

Journal articles
Dissertations / Theses
Book chapters
Conference papers

Academic literature on the topic 'Urobilinogen'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Urobilinogen.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Urobilinogen"

1

Qadir, Muhammad Imran, Muhammad Asad, and Maleeha Azhar. "Interaction of Urobilinogen with Body Sweating." Asian Journal of Basic Science & Research 02, no. 01 (2020): 16–18. http://dx.doi.org/10.38177/ajbsr.2020.2102.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Schrumpf, A. "The transformation of urobilinogen to urobiline." Acta Medica Scandinavica 79, S50 (April 24, 2009): 264–66. http://dx.doi.org/10.1111/j.0954-6820.1932.tb14564.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Kotal, P., and J. Fevery. "Urobilinogen-i is a major derivative of bilirubin in bile of homozygous Gunn rats." Biochemical Journal 268, no. 1 (May 15, 1990): 181–85. http://dx.doi.org/10.1042/bj2680181.

Full text

Abstract:

Gunn rats lack bilirubin UDP-glycosyltransferases, but diazo-negative derivatives of bilirubin have been described in their bile. In order to investigate this alternative disposal of bilirubin, crude bile samples from Gunn and Wistar rats were directly analysed by h.p.l.c. Besides bilirubin (in Gunn rats) or its glycosides (in Wistar rats), two major compounds were detected. A yellow one corresponded to the previously documented vitamin B-2 and was equally prominent in Gunn rats or Wistar-rat bile. The other compound was colourless, but on standing in contact with air it was spontaneously oxidized to a pinkish-yellow pigment. It was far more prominent in Gunn-rat bile. Analysis of bile obtained after intravenous injection of [14C]bilirubin to Gunn rats demonstrated that this compound was highly labelled. Freezing and thawing of the bile resulted in the formation of a series of diazo-negative derivatives, demonstrating that the original compound was quite labile. Spectral (adsorption and fluorescent) and chromatographic (h.p.l.c., t.l.c. and paper chromatography) analysis of the oxidized form of the labelled compound allowed its identification as urobilin-i. The colourless compound secreted in bile was urobilinogen-i. Administration of neomycin and bacitracin to Gunn rats or gut resection suppressed the biliary excretion of urobilinogen and thus confirmed its intestinal origin. Urobilinogen seems thus to represent the major bilirubin derivative present in Gunn-rat bile. Its breakdown products might represent the so-far-unidentified diazo-negative polar bilirubin derivatives. Since only a small amount of bilirubin is present in Gunn-rat bile, the urobilinogen formed in the intestinal lumen seems to be derived from bilirubin reaching the gut via routes other than the biliary one.

APA, Harvard, Vancouver, ISO, and other styles

4

Qadir, Muhammad Imran, and Asma Rasheed. "UROBILINOGEN INFLUENCE ON THE LIKELINESS OF PINEAPPLE." Journal of Bio Innovation 09, no. 04 (July 1, 2020): 481–84. http://dx.doi.org/10.46344/jbino.2020.v09i04.07.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

WITH, TORBEN K. "The Urobilinogen Tolerance Test as Functional Liver Test." Acta Medica Scandinavica 125, no. 6 (April 24, 2009): 588–99. http://dx.doi.org/10.1111/j.0954-6820.1946.tb09277.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Ding, Yu, Junmin Zhao, Gangsheng Liu, Yinglong Li, Jiang Jiang, Yun Meng, Tingting Xu, and Kaifeng Wu. "Total Bilirubin Predicts Severe Progression of Diabetic Retinopathy and the Possible Causal Mechanism." Journal of Diabetes Research 2020 (August 3, 2020): 1–7. http://dx.doi.org/10.1155/2020/7219852.

Full text

Abstract:

Early detection and treatment are key to delaying the progression of diabetic retinopathy (DR), avoiding loss of vision, and reducing the burden of advanced disease. Our study is aimed at determining if total bilirubin has a predictive value for DR progression and exploring the potential mechanism involved in this pathogenesis. A total of 540 patients with nonproliferative diabetic retinopathy (NPDR) were enrolled between July 2014 and September 2016 and assigned into a progression group (N=67) or a stable group (N=473) based on the occurrence of diabetic macular edema (DME), vitreous hemorrhage, retinal detachment, or other conditions that may cause severe loss of vision following a telephonic interview in August 2019. After further communication, 108 patients consented to an outpatient consultation between September and November 2019. Our findings suggest the following: (1) TBIL were significant independent predictors of DR progression (HR: 0.70, 95% CI: 0.54–0.89, p=0.006). (2) Examination of outpatients indicated that compared to stable group patients, progression group patients had more components of urobilinogen and LPS but a lower concentration of TBIL. The relationship between bilirubin and severe DR was statistically significant after adjusting for sex, age, diabetes duration, type of diabetes, FPG, and HbA1c (OR: 0.70, 95% CI: 0.912–0.986, p=0.016). The addition of serum LPS and/or urobilinogen attenuated this association. This study concludes that total bilirubin predicts an increased risk of severe DR progression. Decreasing bilirubin might be attributed to the increased levels of LPS and urobilinogen, which may indicate that the change of bilirubin levels is secondary to intestinal flora disorder and/or intestinal barrier destruction. Further prospective investigations are necessary to explore the causal associations for flora disorder, intestinal barrier destruction, and DR.

APA, Harvard, Vancouver, ISO, and other styles

7

NAKAMURA, Takashi, Katsuyuki SATO, Mitsuo AKIBA, and Masao OHNISHI. "Urobilinogen, as a Bile Pigment Metabolite, Has an Antioxidant Function." Journal of Oleo Science 55, no. 4 (2006): 191–97. http://dx.doi.org/10.5650/jos.55.191.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Walthour, Amy, and Paul Dassow. "How should a positive urine urobilinogen dipstick test be evaluated?" Evidence-Based Practice 17, no. 12 (December 2014): E1. http://dx.doi.org/10.1097/01.ebp.0000540865.30949.f3.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Cheah, S. H., S. L. Ch'Ng, R. Husain, and M. T. Duncan. "Effects of fasting during Ramadan on urinary excretion in Malaysian Muslims." British Journal of Nutrition 63, no. 2 (March 1990): 329–37. http://dx.doi.org/10.1079/bjn19900119.

Full text

Abstract:

Urine analysis was conducted on male Muslims before, during and after Ramadan. Various changes in urine volume, osmolality, total solute, sodium, potassium, titratable acidity and urea in response to altered feeding and activity regimens were found. There were no detectable levels of ketones, protein, glucose, urobilinogen and haemoglobin. It was concluded that the body adapted to fasting during Ramadan and that there were no adverse effects on renal function.

APA, Harvard, Vancouver, ISO, and other styles

10

Chindarkar, Nandkishor S., Landen L. Rentmeester, Binh T. Ly, and Robert L. Fitzgerald. "Black urine due to urobilinogen in a patient with alcoholic pellagra." Clinical Biochemistry 47, no. 12 (August 2014): 1132–35. http://dx.doi.org/10.1016/j.clinbiochem.2014.03.016.

Full text

APA, Harvard, Vancouver, ISO, and other styles

More sources

Dissertations / Theses on the topic "Urobilinogen"

1

HAMAJIMA, NOBUYUKI, KENJI WAKAI, EMI MORITA, MARIKO NAITO, ASAHI HISHIDA, SAYO KAWAI, RIEKO OKADA, et al. "ASSOCIATION OF UGT1A1 GLY71ARG WITH URINE UROBILINOGEN." Nagoya University School of Medicine, 2011. http://hdl.handle.net/2237/14913.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Urobilinogen"

1

Kumar, Vijay, and Kiran Dip Gill. "Qualitative Test for Bile Pigments and Urobilinogen in Urine." In Basic Concepts in Clinical Biochemistry: A Practical Guide, 123–27. Singapore: Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-8186-6_31.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Pape, Puja T., Victoria J. A. Sharp, and Jessica Rockafellow. "Urine Dipstick: An Approach to Glucosuria, Ketonuria, pH, Specific Gravity, Bilirubin and Urobilinogen – Undeniable Chemistry." In Urine Tests, 117–41. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-29138-9_7.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Free, Alfred H., and Helen M. Free. "Urobilinogen." In Urinalysis in Clinical Laboratory Practice, 77–83. CRC Press, 2018. http://dx.doi.org/10.1201/9781351077460-15.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

"14.3 Bilirubin und Urobilinogen." In Taschenlehrbuch Klinische Chemie und Hämatologie, edited by Klaus Dörner. Stuttgart: Georg Thieme Verlag, 2009. http://dx.doi.org/10.1055/b-0034-12801.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

"14.3 Bilirubin und Urobilinogen." In Taschenlehrbuch Klinische Chemie und Hämatologie, edited by Klaus Dörner. Stuttgart: Georg Thieme Verlag, 2013. http://dx.doi.org/10.1055/b-0034-12952.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Thompson, Richard. "Jaundice." In Oxford Textbook of Medicine, 2444–51. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199204854.003.1520_update_002.

Full text

Abstract:

All haem molecules are degraded in macrophages by haem oxygenase to biliverdin, and thence by biliverdin reductase to bilirubin, which is selectively removed by hepatocytes from sinusoidal blood and then conjugated, mainly by one of the two specific isoforms of the microsomal enzyme UDP-glucuronyl (glucuronate-glucuronosyl) transferase, chiefly with two glucuronic acid moieties. Conjugated bilirubin is excreted into the bile by the anionic conjugate transporter protein (MRP2), but in many liver diseases it refluxes back into blood and—since it is water soluble and less firmly bound to albumin than unconjugated bilirubin—about 1% is filtered across the glomerular membrane and darkens the urine (choluria). In the distal intestine conjugated bilirubin is deconjugated and reduced to a series of uro- and stercobilinogens that give the normal colour to faeces. Some colourless urobilinogen is normally absorbed from the colon and undergoes an enterohepatic circulation, with a small amount being excreted in urine. If this biliary excretion is impaired in liver disease, or increased in haemolysis, then excess urobilinogen is excreted in urine, where it is easily detected by routine clinical ‘stix’....

APA, Harvard, Vancouver, ISO, and other styles

7

Farne, Hugo, Edward Norris-Cervetto, and James Warbrick-Smith. "Jaundice." In Oxford Cases in Medicine and Surgery. Oxford University Press, 2015. http://dx.doi.org/10.1093/oso/9780198716228.003.0020.

Full text

Abstract:

The metabolism of bilirubin in humans is summarized in Figure 14.1 and can be divided into three sequential steps: 1 Production of unconjugated bilirubin. Red blood cells are broken down by macrophages (mainly in the spleen), which degrade haemoglobin into iron and unconjugated (water insoluble) bilirubin. The iron is stored inside transferrin proteins. Unconjugated bilirubin travels to the liver bound to albumin. In disease, unconjugated bilirubin can be produced by haemolysis of red cells intravascularly, rather than in the spleen. 2 Conjugation of bilirubin. Liver hepatocytes uptake unconjugated bilirubin and conjugate it to glucuronate, thus making water soluble, conjugated bilirubin. 3 Excretion of bilirubin. Once conjugated, bilirubin is secreted into the bile canaliculi. Conjugated bilirubin flows with bile down the bile ducts and into the duodenum. Inside the bowel, conjugated bilirubin is metabolized by bacteria into colourless products (urobilinogen, stercobilinogen). Some of these can be reabsorbed by the gut and excreted via the kidneys, but the vast majority are oxidized in the gut into coloured pigments (urobilin, stercobilin) which give faeces their brown colour. Consequently, if there is complete obstruction of the bile ducts there will be no flow of conjugated bilirubin into the gut, no conversion into urobilinogen, and therefore not even a trace of urobilinogen in the urine. The terminology is confusing because different people mean different things. If you are going to use this terminology, make sure that you and your colleagues agree on the definitions. Nonetheless, this is what people usually mean: • Prehepatic jaundice: this refers to jaundice caused by an excessive production of bilirubin. Remember that bilirubin is produced by the breakdown of haemoglobin in the blood vessels or the spleen, hence the term prehepatic. • Hepatic jaundice: for some people, this means any jaundice due to pathology in the liver (anatomically), such as points 3, 4, and 5 in Figure 14.1, and can thus include problems with hepatocytes (e.g. hepatitis) or with the bile canaliculi (e.g. primary sclerosing cholangitis, PSC).

APA, Harvard, Vancouver, ISO, and other styles

8

Johnson, Colin, and Mark Wright. "Diseases of the gallbladder and biliary tree." In Oxford Textbook of Medicine, edited by Jack Satsangi, 3196–208. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198746690.003.0334.

Full text

Abstract:

Diseases of the gallbladder and bile ducts are common, with gallstones and their complications being most frequent. Less common are biliary strictures, usually malignant, which are caused by adenocarcinomas of the pancreas, bile ducts, ampulla of Vater, and gallbladder. Rarely encountered are sclerosing cholangitis and a variety of congenital disorders. Disorders of the biliary system include gallstones, which cause biliary colic and cholecystitis by obstruction of the cystic duct, and bile duct obstruction (cholestasis), with jaundice, dark urine, and pale stools, itching, and sometimes constant right hypochondrial pain. Fevers and rigors may indicate bacterial infection of the biliary tract (cholangitis), which frequently accompanies partial obstruction. Weight loss may be due to fat malabsorption but can also be caused by malignancy. Prolonged biliary obstruction leads to skin changes of increased pigmentation (due to melanin) and cholesterol deposition (xanthelasma and xanthoma). Biliary cirrhosis can cause portal venous hypertension and liver cell failure. Disorders of the biliary system generally give rise to the biochemical picture of cholestasis: the serum (conjugated) bilirubin concentration may be normal or raised; serum alkaline phosphatase, γ‎-glutamyl transferase, and bile acids are elevated; serum transaminases show only modest elevation. Bilirubinuria is present, with the disappearance of urobilinogen from the urine indicating complete biliary obstruction. Imaging is critical in the diagnosis of biliary disease, initially by ultrasonography, with CT and MRI in more complicated cases. However, these investigations sometimes provide insufficient anatomical detail for diagnosis or planning of treatment, and further imaging with the cholangiographic techniques of magnetic resonance cholangiopancreatography, endoscopic retrograde cholangiopancreatography (ERCP) or percutaneous transhepatic cholangiography (PTC) are required. ERCP and PTC can be used to place biliary stents.

APA, Harvard, Vancouver, ISO, and other styles

Conference papers on the topic "Urobilinogen"

1

Paglinawan, Arnold C., Febus Reidj G. Cruz, Leonardo D. Valiente, Jesus Paolo T. Mendoza, Arnold M. Chanliongco, Jerome B. Torres, and Rachelle Geleen S. Tungol. "Measurement of Specific Gravity, Urobilinogen, Blood, Protein and pH Level of Urine Samples Using Raspberry Pi based Portable Urine Test Strip Analyzer." In ICBET 2020: 2020 10th International Conference on Biomedical Engineering and Technology. New York, NY, USA: ACM, 2020. http://dx.doi.org/10.1145/3397391.3397414.

Full text

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!