Relevant bibliographies by topics / Urolithiasis. Mechanism. Randall's plaque

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Academic literature on the topic 'Urolithiasis. Mechanism. Randall's plaque'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Urolithiasis. Mechanism. Randall's plaque"

1

Naveen, Maledavar Priyanka Patil* Kshitij Lele Sneha Kakade. "Urolithiasis: Advances in Pathophysiology, Microbiome Role, Immune Pathways, and Evolving Diagnostic and Therapeutic Strategies for Kidney Stone Formation and Management." International Journal of Pharmaceutical Sciences 3, no. 5 (2025): 3396–411. https://doi.org/10.5281/zenodo.15473206.

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Urolithiasis, or kidney stone disease, is a multifactorial disorder characterized by crystalline deposits in the urinary system. Its global prevalence has risen significantly over the past two decades, making it a major public health concern. The etiology involves genetic, metabolic, dietary, and environmental factors. Kidney stones, including calcium oxalate, uric acid, struvite, and cystine stones, form through distinct mechanisms. The stone formation process includes supersaturation, nucleation, crystal growth, aggregation, and retention in the kidneys. Hormonal influences, particularly sex
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2

Yamgar, Priyanka Vitthal, Vikram V. Nimbalkar, and Hemant J. Pagar. "Urolithiasis: Mechanism of stone formation, diagnostic modalities and treatment protocols." IP International Journal of Comprehensive and Advanced Pharmacology 9, no. 4 (2024): 227–35. http://dx.doi.org/10.18231/j.ijcaap.2024.034.

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Kidney stone disease, also known as nephrolithiasis or urolithiasis, represents one of the oldest documented medical conditions in human history. Despite longstanding awareness of the processes leading to stone formation and evolution, the precise underlying mechanisms remain largely elusive. Recent technological advancements have spurred numerous innovations and surgical techniques for treating kidney stones. Research suggests that five distinct mechanisms, including crystallization and urine supersaturation, contribute to kidney stone development. Randall's plaques, specifically, play a pivo
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3

Alelign, Tilahun, and Beyene Petros. "Kidney Stone Disease: An Update on Current Concepts." Advances in Urology 2018 (2018): 1–12. http://dx.doi.org/10.1155/2018/3068365.

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Kidney stone disease is a crystal concretion formed usually within the kidneys. It is an increasing urological disorder of human health, affecting about 12% of the world population. It has been associated with an increased risk of end-stage renal failure. The etiology of kidney stone is multifactorial. The most common type of kidney stone is calcium oxalate formed at Randall’s plaque on the renal papillary surfaces. The mechanism of stone formation is a complex process which results from several physicochemical events including supersaturation, nucleation, growth, aggregation, and retention of
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4

Emirgaev, Zaur K., Ruslan N. Tagirov, Nair S. Tagirov, Andrei G. Vasiliev, and Ruslan N. Tagirov. "Metabolic disorders and androgen deficiency in the pathogenesis of urolithiasis." Pediatrician (St. Petersburg) 15, no. 1 (2024): 65–78. http://dx.doi.org/10.17816/ped15165-78.

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This review summarizes and critically analyzes current data on the pathogenesis of urolithiasis (urolithiasis, nephrolithiasis). Emphasis is placed on such issues as: mechanisms of urinary stone formation; risk factors for stone formation; the role of oxidative stress; the chemical composition of renal stones (and especially oxalates); the role of Randall’s plaques, osteopontin, uromodulin (Tamm–Horsfall protein), α-enolase; and the mechanism of stone formation in the collecting ducts. Insufficiently studied issues of microbiota influence — (a) kidney and urinary tract and (b) gastrointestinal
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5

Evan, Andrew P., Fredric L. Coe, James E. Lingeman, et al. "Mechanism of Formation of Human Calcium Oxalate Renal Stones on Randall's Plaque." Anatomical Record: Advances in Integrative Anatomy and Evolutionary Biology 290, no. 10 (2007): 1315–23. http://dx.doi.org/10.1002/ar.20580.

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6

Zubarev, Vadim A., Juliya M. Zabrodskaya, Anatoly I. Arkhangel'sky, Marlen E. Topuzov, and Iliya V. Dovzhansky. "THE ROLE OF DISORGANIZATION OF CONNECTIVE TISSUE AND BASEMENT MEMBRANE OF RENAL TUBULAR EPITHELIUM IN THE PATHOGENESIS OF UROLITHIASIS." Morphological newsletter 28, no. 2 (2020): 78–81. http://dx.doi.org/10.20340/mv-mn.2020.28(2):78-81.

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According to a number of researchers, pathological changes in the structures of the renal papilla play a key role in the pathogenesis of urolithiasis. According to the results of existing studies, destructive processes in collagen are characterized by a change in the length, thickness of the fibers and their orientation in space. Collagen disorganization has enzymatic and non-enzymatic mechanisms. The aim of the study was to study the frequency and features of localization of Randall's plaques and morphological study of the state of connective tissue and the basement membrane of the epithelium
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7

Rajput, Divyanshu, Tanpreet Kaur Badwal, Shreya Gupta, and Rajendra Kumar. "An insight on mechanism and management of kidney stones and its recurrence." Pharmaspire 15, no. 04 (2023): 283–95. http://dx.doi.org/10.56933/pharmaspire.2023.15143.

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Kidney stones (calculi) comprise mineral concretions which can form in both renal calyces and pelvis and can be free floating or associated with the renal papillae. It is an emerging urological condition that affects around 12% of the world’s population. Globally, the prevalence and recurrence of kidney stone disease are increasing, and there are limited effective treatment options. I manifest a brief general overview and then concentrate on risk factors, pathophysiology, and medical treatment of kidney stones. The major component of most stones is calcium oxalate, and many of them develop on
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8

Mohod, Priya. "Homeopathy as a saviour for urolithiasis: A narrative review shading light on pathophysiology of renal stones and homeopathy drugs." Journal of Preventive Medicine and Holistic Health 8, no. 2 (2023): 57–65. http://dx.doi.org/10.18231/j.jpmhh.2022.013.

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Kidney stone disease is a crystal concretion formed generally within the kidneys. It is an accumulative urological disorder of human health, affecting about 12% of the world population. Higher risk of kidney failure which has been associated with end stage renal failure. Calcium oxalate, which forms at Randall's plaque on the renal papillary surfaces, is the most common type of kidney stone. Stone formation is highly prevalent, with rates of up to 14.8% and increasing, and a recurrence rate of up to 50% within the first 5 years of the initial stone incident. The formation of the stone is a com
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9

Bergsland, Kristin J., Elaine M. Worcester, and Fredric L. Coe. "Role of proximal tubule in the hypocalciuric response to thiazide of patients with idiopathic hypercalciuria." American Journal of Physiology-Renal Physiology 305, no. 4 (2013): F592—F599. http://dx.doi.org/10.1152/ajprenal.00116.2013.

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The most common metabolic abnormality found in calcium (Ca) kidney stone formers is idiopathic hypercalciuria (IH). Using endogenous lithium (Li) clearance, we previously showed that in IH, there is decreased proximal tubule sodium absorption, and increased delivery of Ca into the distal nephron. Distal Ca reabsorption may facilitate the formation of Randall's plaque (RP) by washdown of excess Ca through the vasa recta toward the papillary tip. Elevated Ca excretion leads to increased urinary supersaturation (SS) with respect to calcium oxalate (CaOx) and calcium phosphate (CaP), providing the
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10

Patil, Ruchita S., Azam Z. Shaikh, Akash S. Jain, Divakar R. Patil, Sameer R. Shaikh, and S. P. Pawar. "Kidney Stone: Mechanism of Formation and its Prevention A Review." Asian Journal of Pharmaceutical Research, December 23, 2024, 381–86. https://doi.org/10.52711/2231-5691.2024.00060.

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One of the earliest recognized medical disorders, kidney stone disease still has unanswered questions on how it starts and progresses. The precise mechanisms behind the production of stones remain enigmatic, despite major technological improvements in recent decades leading to a variety of theories and surgical techniques. Three different fundamental pathways for kidney stone production have been identified by research, including observations made by the authors and other organizations. Intrarenal crystal precipitation is primarily caused by urinary supersaturation and crystallization. Randall
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