Relevant bibliographies by topics / Uterine corpus endometrial carcinoma

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Uterine corpus endometrial carcinoma'

Author: Grafiati
Published: 7 June 2025
Last updated: 16 July 2025

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Journal articles on the topic "Uterine corpus endometrial carcinoma"

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Abu-Rustum, Nadeem, Catheryn Yashar, Rebecca Arend, et al. "Uterine Neoplasms, Version 1.2023, NCCN Clinical Practice Guidelines in Oncology." Journal of the National Comprehensive Cancer Network 21, no. 2 (2023): 181–209. http://dx.doi.org/10.6004/jnccn.2023.0006.

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Adenocarcinoma of the endometrium (also known as endometrial cancer, or more broadly as uterine cancer or carcinoma of the uterine corpus) is the most common malignancy of the female genital tract in the United States. It is estimated that 65,950 new uterine cancer cases will have occurred in 2022, with 12,550 deaths resulting from the disease. Endometrial carcinoma includes pure endometrioid cancer and carcinomas with high-risk endometrial histology (including uterine serous carcinoma, clear cell carcinoma, carcinosarcoma [also known as malignant mixed Müllerian tumor], and undifferentiated/dedifferentiated carcinoma). Stromal or mesenchymal sarcomas are uncommon subtypes accounting for approximately 3% of all uterine cancers. This selection from the NCCN Guidelines for Uterine Neoplasms focuses on the diagnosis, staging, and management of pure endometrioid carcinoma. The complete version of the NCCN Guidelines for Uterine Neoplasms is available online at NCCN.org.
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Caesar Muhammad Wijaya, Puja Agung Antonius, Syamel Muhammad, and Mutia Paramadita Anugrah. "Uterine Corpus Endometrial Carcinoma in Young Women." Andalas Obstetrics And Gynecology Journal 9, no. 1 (2025): 161–66. https://doi.org/10.25077/aoj.9.1.161-166.2025.

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Introduction : Endometrial carcinoma is a malignant epithelial tumor that forms in the uterus's inner lining, or endometrium. The average age at diagnosis is 61 years, with cases diagnosed after 50 years more than 90%. Endometrial carcinoma is rare in young women, usually defined as occurring under age 50 or menopause, with rare cases occurring under age 40. Case Report : A 35-year-old woman nulliparous complaints of abdominal pain since 3 months ago and complained of weight loss. Based on the anamnesis, the patient is not married. On physical examination, revealed an area of firmness in the suprapubic region associated with pain on palpation. On ultrasound examination, the uterus was found to be anteflexed, with an inhomogeneous appearance, and a solid cystic mass along the uterus. Adnexa measuring 10.02 x 10.20 x 12.02 cm, vascular scale 4, not clearly defined. The patient was diagnosed with suspected uterine corpus carcinoma. Result : In this patient, a total hysterectomy and bilateral salpingo-oophorectomy were performed because the patient did not want fertility. Conclussion : In young women who want to preserve fertility, conservative management is often implemented. But, when fertility-sparing treatment is not considered, the rate of recurrence and progression is considerably low in this case. Ultimately, although fertility-sparing treatments are an attractive alternative to surgery resulting in permanent loss of fertility, unfortunately, they can only be applied in a subset of cases. The standard surgical procedure recommended is a total extra-fascial hysterectomy with bilateral salpingo-oophorectomy. Keywords: Uterine Corpus Endometrial Carcinoma, Young Women
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Pundir, Swati, Manish Kumar, Usha Joshi, and Sheela Chaudhari. "Clinico-Radiological and Histopathological Study of Endometrial Lesions in Patients Presenting with Abnormal Uterine Bleeding." Journal of Medical Sciences and Health 10, no. 2 (2024): 142–47. http://dx.doi.org/10.46347/jmsh.v10.i2.24.316.

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Background: Abnormal uterine bleeding [AUB] is one of the most common presentations among women of all age groups. It is ‘Bleeding from the uterine corpus that is abnormal in volume, regularity or timing for the majority of last 6 months. Common pathologies include hormonal imbalance patterns, atrophic endometrium, endometritis, endometrial polyp, endometrial hyperplasia and endometrial carcinoma. Material & Methods: All specimens of endometrial curettage, biopsy and hysterectomy received in department of pathology GMC, Haldwani from January 2020 to September 2021 , have been included in the study. Specimens were fixed in 10% formalin and processed routinely. Clinical details and endometrial thickness by ultrasonography were recorded and analysed. Microscopic examination was performed after routine and special stains. Results: Out of a total of 103 cases, abnormal uterine bleeding was frequently observed in the age group 40 – 49 years, followed by 50 – 59 years. Most common presenting complaint was menorrhagia followed by post menopausal bleeding. Most common histopathological finding was proliferative phase endometrium followed by secretory phase endometrium. The range of endometrial thickness recorded in the proliferative phase was mostly 4-9 mm followed by the secretory phase with 9 – 15 mm. Two cases of endometrial carcinoma were seen after 5th decade onward. All cases of endometrial hyperplasia and carcinoma had an endometrial thickness of more than 15 mm. Conclusion: AUB may be the only presenting complaint in patients with malignant or pre-malignant endometrial lesions. Histopathology helps to reach a definitive diagnosis crucial for management. Keywords: Abnormal uterine bleeding, Endometrial sampling, Endometrial thickness, Endometrial hyperplasia, Endometrial carcinoma
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Deolet, Ellen, Jo Van Dorpe, and Koen Van de Vijver. "Mesonephric-Like Adenocarcinoma of the Endometrium: Diagnostic Advances to Spot This Wolf in Sheep’s Clothing. A Review of the Literature." Journal of Clinical Medicine 10, no. 4 (2021): 698. http://dx.doi.org/10.3390/jcm10040698.

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Mesonephric-like adenocarcinoma is a recently described rare neoplasm occurring in the uterine corpus and ovary. This under-recognized subtype of carcinoma can be very challenging to diagnose. In mesonephric adenocarcinoma a variety of growth patterns can be present within the same tumor, as a result of which they can be misinterpreted and diagnosed as low-grade endometrioid adenocarcinoma, clear cell carcinoma, or even serous carcinoma and carcinosarcoma. We report a case of mesonephric-like adenocarcinoma misdiagnosed as a low-grade endometrioid endometrial adenocarcinoma that had an early local recurrence and metastasized to the liver and the lungs. Histopathological, immunohistochemical and molecular analysis were performed and compared to published literature, providing a comprehensive overview of the current knowledge. Databases (Pubmed, Web of Science, Google Scholar) were searched with a combination of the following search terms: mesonephric-like, mesonephric, adenocarcinoma, carcinoma, uterine body, uterine corpus, endometrium. Mesonephric-like adenocarcinoma is a difficult-to-diagnose entity. Advanced diagnostics, including improved morphologic, immunohistochemical and molecular knowledge can help develop new therapeutic strategies against this specific subtype of endometrial cancer with an aggressive clinical behavior.
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Magriples, U., F. Naftolin, P. E. Schwartz, and M. L. Carcangiu. "High-grade endometrial carcinoma in tamoxifen-treated breast cancer patients." Journal of Clinical Oncology 11, no. 3 (1993): 485–90. http://dx.doi.org/10.1200/jco.1993.11.3.485.

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PURPOSE Several reports have associated tamoxifen administration with endometrial carcinoma. A retrospective study of the histologic features of uterine cancer in patients with a history of breast carcinoma was undertaken to determine the effect of treatment with tamoxifen. MATERIALS AND METHODS A computer search of the Yale-New Haven Hospital Tumor Registry from 1980 to 1990 identified 53 patients with a history of breast carcinoma who subsequently developed a malignant tumor of the uterine corpus. RESULTS Fifteen patients received tamoxifen for breast carcinoma and 38 did not. The mean ages of the two groups were not significantly different. The mean interval between detection of breast and endometrial cancers was 5 years in the tamoxifen group and 12 years in the nontreated group (P = .0023). Sixty-seven percent of patients in the tamoxifen group had poorly differentiated endometrioid carcinomas (including adenosquamous carcinoma) or carcinomas associated with poor outcome (eg, uterine papillary serous carcinoma, clear-cell carcinoma, or mixed müllerian tumor), as compared with 24% in the nontreated group (P = .03). Patients in the tamoxifen group were much more likely to die of endometrial cancer (33.3% v 2.6% of the nontreated group, P = .005). CONCLUSION From this retrospective study, it appears that women receiving tamoxifen as treatment for breast cancer who subsequently develop uterine cancer are at risk for high-grade endometrial cancers that have a poor prognosis. These findings also indicate that tamoxifen-associated uterine cancers may have a different basis from those associated with steroidal estrogen treatment.
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Chandwani, Teena V., Dharitri M. Bhat, and Archana H. Deshpande. "Histological spectrum and diagnostic challenges in endometrial carcinoma of the uterus." International Journal of Reproduction, Contraception, Obstetrics and Gynecology 12, no. 6 (2023): 1830–37. http://dx.doi.org/10.18203/2320-1770.ijrcog20231564.

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Background: Endometrial carcinoma is the most common type of malignancy of the uterine corpus accounting for 95% of all primary malignancies. Epidemiological studies have proved the role of unopposed estrogen as an important factor in pathogenesis of endometrial carcinoma. Pathologists play an important role not only in the histological confirmation of the diagnosis, but in subtyping, grading and staging of the tumor. Current study is undertaken to study the histological spectrum in endometrial carcinomas. Methods: Present study includes histologically confirmed cases of endometrial carcinoma of uterine corpus over a period of 24 months (July2020 to June 2022) in a tertiary health care center in central India. After gross examination, and standard sectioning, all these tumors were subtyped on histology and grading, staging was done as per WHO and FIGO recommendations. In cases with diagnostic dispute and overlapping features, IHC markers were applied. Results: Amongst total 30 confirmed cases of endometrial carcinoma, endometrioid type was most common (25) followed by villoglandular and serous type of adenocarcinoma. Architectural and nuclear grading was done in all cases. Staging could be done in hysterectomy specimens only. Difficulties encountered while grading and staging are discussed. Conclusions: Endometrial carcinoma was the most common type of malignancy. Other types were villoglandular and serous adenocarcinoma. Various additional histological features were also observed. Grade I tumors were commonly seen. Depth of myometrial invasion was measured in hysterectomy specimens. Various diagnostic challenges encountered are discussed.
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Mandic, Aljosa, Bojana Gutic, Tatjana Kapicl-Ivkovic, Ljiljana Segedi-Mladenovic, and Mihaela Mocko-Kacanski. "Clinical and histopathological characteristics in patients with postmenopausal bleeding." Archive of Oncology 21, no. 1 (2013): 5–10. http://dx.doi.org/10.2298/aoo1301005m.

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Background: Incidence of endometrial carcinoma in Vojvodina is 15-20/100 000. In 75% cases, endometrial carcinoma is diagnosed in postmenopausal period. In 90 % of patients, the first clinical sign is postmenopausal bleeding. The aim of the study was to investigate clinical and histopathological characteristics in patients with postmenopausal bleeding. Methods: The study included 122 patients with postmenopausal bleeding. All of these patients underwent gynecological examination and vaginal ultrasound. We obtained materials for histopathological analysis by fractionate explorative curettage. Once we had definitive histopathological findings, we divided patients in two groups A (endometrial carcinoma) and B (benign changes). Results: We confirmed significant statistical differences between examined group A and B, including age (64.49 compared with 58.81 years), postmenopausal period (13.67 instead 9.11 years), and length of uterine corpus (6.41 instead 5.25 cm). Conclusion: Elderly women with longer postmenopausal interval and postmenopausal bleeding had increased risk for endometrial carcinoma. Measurement of endometrial thickness by transvaginal ultrasound appeared to be insufficient parameter for differentiating the benign from the malignant changes of endometrium. Patients with endometrial carcinoma had significantly longer corpus of uterus comparing to patients with benign changes. Body mass index was not found to be significant risk factor in development of endometrial carcinoma in the examined groups. Obesity was diagnosed in both groups, suggesting that increased body mass index is a risk factor for development of pathological changes in endometrium, which could lead to postmenopausal bleeding.
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Li, Yingqiang, Jie Zhou, Jing Zhang, Ming Liu, Shifu Chen, and Yaqiong Liu. "Cancer-associated fibroblasts in uterine corpus endometrial carcinoma." Journal of Clinical Oncology 40, no. 16_suppl (2022): e17618-e17618. http://dx.doi.org/10.1200/jco.2022.40.16_suppl.e17618.

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e17618 Background: The general role of cancer-associated fibroblasts (CAFs) and their invasive characteristics in Uterine Corpus Endometrial Carcinoma (UCEC) remain unknown. CAFs serve as an important component of the tumor microenvironment and promote tumorigenesis and cancer aggressiveness. Methods: UCEC mRNA data were downloaded from TCGA cBioPortal ( http://www.cbioportal.org/ ). CAF levels were estimated by three algorithms: MCPCOUNTER, XCELL, and EPIC, so three CAF scores were gotten and ranked. Then, the data of CAF scores in each cohort were divided into quarters, and we labeled the sample by upper quartile and lower quartile with “high”, “medium”, and “low”. To mitigate the possible classification error from each estimation method, we defined samples as “high” by at least two methods consistently labeling “high”. Samples labeled with “low” by at least two methods were assigned to the consensual low CAF group. The high and low group were analysis using GSEA software, Survival analysis was performed using the R package “survival”. Results: There were 129, 118 and 280 samples in the high, low and middle CAFs group, respectively. GSEA results showed extensive gene expression differences between the high and low CAF groups. 193 gene sets were significantly enriched at nominal p value < 0.01 in the low group, 869 gene sets were significantly enriched at nominal p value < 0.01 in the high group. In the low group, obvious differences, included lim mammary stem cell up, reactome degradation of the extracellular matrix, bertucci medullary vs ductal breast cancer dn etc. are enriched in the high group. xu akt1 targets 6hr, OUELLET (Gene Set) ovarian cancer invasive vs lmp up etc. are enriched in the low group. OS and PFS survival analysis showed there was no significant difference between these two groups. Conclusions: Our study analyzed the relationship between CAFs with UCEC, CAFs were associated with an immunosuppressive microenvironment. Larger transcriptomic alterations occurred in UCECs with high CAF infiltration, and with AKT, ALCALA APOPTOSIS and SPLICEOSOME pathways prominent in this process. But Survival analysis showed no difference between the two groups.
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Hiratsuka, Daiki, Takehiro Tsukazaki, Kenbun Sone, Kazuaki Neriishi, and Kimihiro Takechi. "A Case of Nonpuerperal Uterine Inversion Caused by Cervical Cancer." Case Reports in Obstetrics and Gynecology 2022 (January 31, 2022): 1–5. http://dx.doi.org/10.1155/2022/1630192.

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Uterine inversion is a rare puerperal event in the third stage of labor. Nonpuerperal uterine inversion is even rarer and is mainly caused by uterine fibroids, uterine sarcoma, or endometrial cancer. This is the first report of uterine inversion caused by cervical cancer. A 67-year-old woman presented with a 10 cm pelvic mass. Contrast-enhanced magnetic resonance imaging revealed uterine inversion, which was preoperatively diagnosed to be caused by endometrial cancer and was treated using an extended abdominal hysterectomy. Postoperative histopathological examination revealed that the primary tumor was a squamous cell carcinoma with coexistent high-grade squamous intraepithelial lesions and small-cell neuroendocrine carcinoma. Immunostaining was diffusely positive for p16 and negative for estrogen receptors. The postoperative diagnosis was cervical squamous cell carcinoma. Our observations suggested that cervical carcinoma can cause uterine inversion by invading the corpus.
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Westin, Shannon N., Robin A. Lacour, Diana L. Urbauer, et al. "Carcinoma of the Lower Uterine Segment: A Newly Described Association With Lynch Syndrome." Journal of Clinical Oncology 26, no. 36 (2008): 5965–71. http://dx.doi.org/10.1200/jco.2008.18.6296.

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Purpose Endometrial carcinoma in the lower uterine segment (LUS) is a poorly described cancer that can be clinically confused with endocervical carcinoma. We performed a case-comparison study to document the clinicopathologic characteristics of LUS tumors and their association with risk factors for endometrial cancer. Patients and Methods The clinical records and pathology reports from women who underwent hysterectomy at our institution for endometrial or endocervical adenocarcinoma over an 11-year interval were reviewed. The LUS group consisted of women with endometrial tumors that clearly originated between the lower uterine corpus and the upper endocervix. Immunohistochemistry and microsatellite instability and MLH1 methylation assays were performed. Results Thirty-five (3.5%) of 1,009 women had endometrial carcinoma of the LUS. Compared with patients with corpus tumors, LUS patients were younger, had higher stage tumors, and had more invasive tumors. Preoperative diagnosis of the LUS tumors more frequently included the possibility of endocervical adenocarcinoma. Seventy-three percent of the LUS tumors had an immunohistochemical expression pattern typical of conventional endometrioid adenocarcinoma. Ten (29%) of 35 women with LUS tumors were confirmed to have Lynch syndrome or were strongly suspected to have Lynch syndrome on the basis of tissue-based molecular assays. Conclusion The prevalence of Lynch syndrome in patients with LUS endometrial carcinoma (29%) is much greater than that of the general endometrial cancer patient population (1.8%) or in endometrial cancer patients younger than age 50 years (8% to 9%). On the basis of our results, the possibility of Lynch syndrome should be considered in women with LUS tumors.
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Dissertations / Theses on the topic "Uterine corpus endometrial carcinoma"

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Vaskivuo, T. (Tommi). "Regulation of apoptosis in the female reproductive system." Doctoral thesis, University of Oulu, 2002. http://urn.fi/urn:isbn:9514266676.

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Abstract Apoptosis is a genetically programmed mechanism for a multicellular organism to remove cells that are unnecessary, or potentially harmful. The female reproductive system is characterised by a high rate of cellular proliferation. At the same time, apoptosis is also abundant during the normal physiological function of the ovary and endometrium. More than half of the 7 million oocytes that are produced during human ovarian development are deleted before birth and only about 400 oocytes reach the stage of ovulation during the female fertile lifespan. The fate of the non-ovulatory follicles is atresia, occurring through the mechanism of apoptosis. The endometrium goes through radical renewal processes during each menstrual cycle. Apoptosis has been suggested to participate in the regulation of endometrial cellular homeostasis. Errors in this mechanism can result in endometrial diseases such as hyperplasia and cancer. In this work, apoptosis and its regulation were studied in the human fetal and adult ovary, normal endometrium and endometrial pathologies. In fetal ovaries, apoptosis was already abundantly present in oocytes at 13 weeks of gestation. The maximum rate of apoptosis was seen between the 14th and 20th weeks, after which apoptosis decreased towards term. Ovarian Bcl-2 expression was detected in early fetal life during weeks 13 and 14. Bax expression was observed throughout the studied period, from week 13 to 40. The expression of transcription factor GATA-4, which is linked to follicular survival, was localised to the granulosa cells and was high in early fetal life and decreased somewhat towards term. In adult life apoptosis was located in the granulosa cells of the growing follicles. In ovarian biopsies from women homozygous for the inactivating C566T mutation of the FSH receptor, apoptosis or GATA-4 expression was not detected. During corpus luteum regression a peak in apoptosis was detected 10 - 12 days after the LH surge, and was preceded by an increase in 17HSD type 1 and TNF-α expression. During normal menstrual cycles, the highest rate of apoptosis was observed in the menstrual endometrium. This increase in apoptosis was preceded by a decreased Bcl-2/Bax ratio. In endometrial hyperplasia, the rate of apoptosis was similar to that seen during normal proliferation of the endometrium, but an apparent increase was observed in grade II endometrial carcinoma. In grade III carcinoma, the rate of apoptosis was lower than in grade II carcinoma but higher than in hyperplasia. These results indicate that apoptosis is the mechanism behind the substantial oocyte demise during ovarian development. During adult life, apoptosis was mainly localised to the granulosa cells of the growing follicles which do not reach the stage of a dominant follicle. In ovaries where FSH action is abolished, folliculogenesis was impaired and ovarian apoptosis was negligible. Apoptosis is also the underlying mechanism of corpus luteum regression. In the endometrium, apoptosis has a role in rejuvenating the endometrium for growth during the next endometrial cycle and in regulating cellular homeostasis.
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Taylor, Sarah Elizabeth. "Investigation of Protein Phosphatase 2A A-alpha Subunit Mutation as a Disease Driver in High-Grade Endometrial Carcinoma." Case Western Reserve University School of Graduate Studies / OhioLINK, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1567791544641051.

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Li, Wen-Wei, and 李文瑋. "Study of uterine 24p3 protein induced apoptosis in endometrial carcinoma cell line." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/63424306367863830409.

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碩士<br>國立臺灣大學<br>生化科學研究所<br>95<br>24p3 protein, a lipocalin, is one of the secretory proteins during mouse estrous cycle. According to the data of our laboratory, reveals that 24p3 protein induces apoptosis of human endometrial carcinoma cell line via releasing cytochrome c from mitochondria and activating caspases. These results suggest that 24p3 protein plays an unique role in female reproduction. To elucidate the mechanism of 24p3 protein induced apoptosis in reproduction system, mice uterine 24p3 protein and human endometrial cell line (RL95-2) were used in this study. After treatment of RL95-2 cells, 24p3 protein creates an oxidative intracellular environment that may trigger the changes of MAPK activity and elevation of intracellular calcium via NAPDH oxidase. These data suggest that MAPK and calcium may be correlated to cell death. Without significant changes in mRNA levels of p53, bcl-2, and bax could be observed in 24p3 protein-treated cells. These findings revealed that the eliciting of cell physiological response by 24p3 protein in a novel pathway.
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Books on the topic "Uterine corpus endometrial carcinoma"

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Malpica, Anais. Biopsy interpretation of the uterine cervix and corpus. Wolters Kluwer/Lippincott Williams & Wilkins, 2010.

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(Editor), H. Kuramoto, and M. Nishida (Editor), eds. Cell and Molecular Biology of Endometrial Carcinoma. Springer, 2003.

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Carton, James. Gynaecological pathology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759584.003.0012.

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This chapter covers gynaecological pathology and includes vulval skin diseases, benign vulval tumours, vulval carcinoma, vaginal infections, vaginal tumours, cervical carcinoma, cervical screening, endometriosis, endometrial carcinoma, uterine leiomyomas (fibroids), uterine leiomyosarcoma, functional ovarian cysts, benign non-epithelial ovarian tumours, benign epithelial ovarian tumours, borderline epithelial ovarian tumours, ovarian carcinomas, pelvic inflammatory disease, ectopic pregnancy, polycystic ovarian syndrome, hydatidiform mole, and pre-eclampsia.
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Book chapters on the topic "Uterine corpus endometrial carcinoma"

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Habiba, Marwan, and Giuseppe Benagiano. "Adenomyosis and Endometrial Carcinoma." In Uterine Adenomyosis. Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-13012-5_10.

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Chitrathara, K. "Ovarian Preservation in Endometrial Carcinoma." In Uterine Cancer. Springer India, 2015. http://dx.doi.org/10.1007/978-81-322-1892-0_17.

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Rekhi, Bharat, Kedar K. Deodhar, and Santosh Menon. "Pathology of Endometrial Hyperplasia and Carcinoma." In Uterine Cancer. Springer India, 2015. http://dx.doi.org/10.1007/978-81-322-1892-0_10.

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Warrier, Arun. "Systemic Treatment of Advanced Endometrial Carcinoma." In Uterine Cancer. Springer India, 2015. http://dx.doi.org/10.1007/978-81-322-1892-0_28.

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Palacios, Jose, and Paola Dal Cin. "Molecular Pathology and Cytogenetics of Endometrial Carcinoma, Carcinosarcoma, and Uterine Sarcomas." In Uterine Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-044-1_5.

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Rajanbabu, Anupama. "Molecular Pathology and Cytogenetics of Endometrial Carcinoma, Carcinosarcoma, and Uterine Sarcomas." In Uterine Cancer. Springer India, 2015. http://dx.doi.org/10.1007/978-81-322-1892-0_2.

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Lessey, Bruce A., Aleksandr E. Vendrov, and Lingwen Yuan. "Endometrial Cancer Cells as Models to Study Uterine Receptivity." In Cell and Molecular Biology of Endometrial Carcinoma. Springer Japan, 2003. http://dx.doi.org/10.1007/978-4-431-53981-0_19.

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Fujimoto, Jiro, Ikumi Aoki, Hiroshi Toyoki, Sufia Khatun, Eriko Sato, and Teruhiko Tamaya. "Sex Steroid-Dependent and -Independent Angiogenesis in Uterine Endometrial Cancers." In Cell and Molecular Biology of Endometrial Carcinoma. Springer Japan, 2003. http://dx.doi.org/10.1007/978-4-431-53981-0_11.

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Palacios, Jose, and Paola Dal Cin. "Molecular Pathology and Cytogenetics of Endometrial Carcinoma, Carcinosarcoma, and Uterine Sarcomas." In Current Clinical Oncology. Springer International Publishing, 2015. http://dx.doi.org/10.1007/7631_2015_6.

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Palacios, Jose, and Paola Dal Cin. "Erratum to: Molecular Pathology and Cytogenetics of Endometrial Carcinoma, Carcinosarcoma, and Uterine Sarcomas." In Current Clinical Oncology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/7631_2018_3.

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Conference papers on the topic "Uterine corpus endometrial carcinoma"

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Mercioni, Marina Adriana, and Stefan Holban. "Uterine Corpus Endometrial Carcinoma Prediction from Genomic Analysis with Machine Learning." In 2024 International Conference on Development and Application Systems (DAS). IEEE, 2024. http://dx.doi.org/10.1109/das61944.2024.10541297.

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Gupta, Bindiya, Shalini Rajaram, Sandhya Jain, Neerja Goel, and Naveen Tanwar. "Collision tumor of endometrial stromal sarcoma and squamous cell cancer: A rare entity." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685363.

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A collision tumor is defined by the presence of two separate tumors in one organ on gross, microscopic, and immunohistochemical studies and they should be distinguished from malignant mullerian mixed tumors. A 60 year old lady P8L8 presented with blood stained vaginal discharge and post menopausal bleeding. Examination revealed a 1 x 2 cm cervical growth which was reported as squamous cell carcinoma cervix. Imaging revealed myohyperplasia with normal uterine cavity. The patient underwent Type III radical hysterectomy, bilateral salphingo-oophorectomy and bilateral pelvic lymphadenectomy. The uterine corpus revealed 5 cm growth in uterine cavity which was reported as high grade endometrial stromal sarcoma and the cervical growth was non keratinising squamous cell carcinoma infiltrating the former. The lymph nodes, parametria and vaginal cuff were free of tumor. The patient was referred for adjuvant chemotherapy and radiation therapy.
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Zuo, Mengqi. "Correlation between Differential Expression of m6A and Prognosis of Uterine Corpus Endometrial Carcinoma." In The International Conference on Biomedical Engineering and Bioinformatics. SCITEPRESS - Science and Technology Publications, 2022. http://dx.doi.org/10.5220/0011375000003443.

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Wang, Tongxin, Weijia Lu, Fan Yang, et al. "Microsatellite Instability Prediction of Uterine Corpus Endometrial Carcinoma Based on H&E Histology Whole-Slide Imaging." In 2020 IEEE 17th International Symposium on Biomedical Imaging (ISBI). IEEE, 2020. http://dx.doi.org/10.1109/isbi45749.2020.9098647.

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Ben Safta, I., H. Mansouri, O. Jaidane, et al. "402 Clear cell carcinoma of the uterine corpus." In IGCS Annual 2019 Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-igcs.402.

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Zhang, Jiayu, Pei Kuang, Fujian Wan, Yingjie Zhang, Wenhua Zhang, and Xiaoyu Zhang. "Effects of CLDN9 on Proliferation of Uterine Corpus Endometrioid Carcinoma Cells." In ICBBT 2023: 2023 15th International Conference on Bioinformatics and Biomedical Technology. ACM, 2023. http://dx.doi.org/10.1145/3608164.3608183.

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Lekhi, Anshika, Rahul Manchanda, Nidhi Jain, Sravani Chithra, and Hena Kausar. "Presentation of endometrial carcinoma in young women." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685342.

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Background: Endometrial carcinoma is a disease of older postmenopausal women, and is relatively uncommon in patients younger than 40 years. Endometrial carcinomas in this age group may be familial, associated with Lynch syndrome, or sporadic. Patient usually has increased exposure to estrogen. In 2%–14% of cases, it occurs in young patients (less than 40 years of age) who are eager to preserve their fertility. Its treatment includes hysterectomy, bilateral salpingo-oophorectomy and pelvic lymphadenectomy and in some cases, radiation therapy. Prevention of fertility is major challenge encountered in such cases. Aim: To present a case of young woman with endometrial carcinoma and through it to review the literature of its presentation and management in such groups. Case: We report a case of endometrial cancer in a 35-year-old woman with previous 3 cesarean treated for abnormal uterine bleeding and cared for in our department. Conclusion: Most endometrial carcinomas presenting in this young age are associated with estrogen excess. Pathologically they are usually low-grade endometrioid carcinomas with lower stage and are associated with favorable clinical outcomes. With this case the authors emphasize the need of endometrial reckoning in young females with abnormal bleeding before starting any medical treatment. Also highlighting the management options in such cases where fertility preservation holds challenge.
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Moiseenko, T., N. Chernikova, M. Adamyan, E. Nepomnyashchaya, and Y. Poryvaev. "EP495 Influence of comorbid estrogen-dependent uterine pathology on morphological characteristics of endometrial carcinoma." In ESGO Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2019. http://dx.doi.org/10.1136/ijgc-2019-esgo.554.

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Kozawa, Eito, Kaiji Inoue, Saki Tuchihashi, et al. "EP136/#225 Evaluation of uterine endometrial carcinoma histological grades using magnetic resonance imaging texture analyses." In IGCS 2023 Annual Meeting Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/ijgc-2023-igcs.232.

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10

Qureshi, Sabuhi. "Study of PTEN immunohistochemical expression in endometrial hyperplasia." In 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685337.

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Objective: The incidence of endometrial hyperplasia &amp; carcinoma is increasing in developing nations. Newer techniques are being tried to recognise endometrial hyperplasia. One of these is tumor suppressor gene phosphatase &amp; tensin homologue (PTEN). It is frequently inactivated i.e turned off in endometrial hyperplasia lesions. This is an early event in endometrial tumorigenesis that may occur in response to known endocrine risk factors &amp; offers an informative immunohistochemical marker for premalignant disease. The present study was planned to study PTEN immunohistochemical expression in endometrial hyperplasia. Methods: Women of &gt;40 years of age presenting with abnormal uterine bleeding in the OPD of OBGYN Department of KG Medical University underwent endometrial biopsy. The histopathology of the biopsy tissue was done in department of Pathology of KG Medical University. The cases of endometrial hyperplasia were studied for PTEN immunohistochemical expression. Results: 168 women of &gt;40 years of age with abnormal uterine bleeding underwent endometrial biopsy. 50 women were diagnosed as endometrial hyperplasia. Of these, PTEN evaluation was done in 27 cases. Loss of PTEN expression was found in 11 cases (40.74%) of endometrial hyperplasia. Loss of PTEN expression was more in complex hyperplasia with atypia (66.66%) as compared to simple hyperplasia without atypia (29.4%). Conclusion: There is positive correlation between loss of PTEN expression and grade of morphological differentiation of hyperplasia.
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Reports on the topic "Uterine corpus endometrial carcinoma"

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Bazer, Fuller W., Arieh Gertler, and Elisha Gootwine. Role of Placental Lactogen in Sheep. United States Department of Agriculture, 2001. http://dx.doi.org/10.32747/2001.7574339.bard.

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Central problems in sheep and dairy cattle production are reproductive failure due to embryonic/fetal mortality and low birth weights, especially in prolific breeds, and reduced milk yields which adversely affect neonatal survival and economy of production. The sheep placenta expresses lactogenic (ovine placental lactogen, oPL) and somatogenic (ovine placental growth hormone, oGH) hormones. Our research has focused on the biological roles of oPL and oGH in function of the uterine endometrium during gestation and the mammary gland during pregnancy and lactation. Major conclusions were that: ( 1 ) immunization of prepubertal ewes against oPL resulted in increased birth weights of their lambs and their milk production during lactation; (2) neither oPL nor oGH had an antiluteolytic effect on uterine endometrium to affect lifespan of the corpus luteum; (3) only sequential exposure of the progesterone stimulated uterus to oIFNt and oPL or oGH increased endometrial gland proliferation and secretory protein gene expression; (4) oPL signals through a homodimer of ovine prolactin receptor (PRL-R) and heterodimer of oPRL-R and growth hormone receptor (GH-R); (5) exogenous recombinant oPL and oGH stimulated mammogenesis and milk yield during lactation; and (6) mutation of oPL and oGH was used to define specific biological effects and a rational basis for design of a specific receptor agonists or antagonists. This project was very productive in elucidating basic biological effects of oPL and oGH on intracellular signal transduction pathways, uterine development and secretory function, as well as mammogenesis and lactogenesis. We determined that immunization of prepubertal ewes against roPL increased birth weights of their lambs, especially those born as twins and triplets, as well as enhanced lactational performance. These studies significantly extended our knowledge of uterine and fetal-placental physiology and provided a foundation for new strategies to enhance reproductive and lactation efficiency. Based on these results, the major achievements were: 1) creation of a practical and cost effective management tool for producers to increase reproductive performance, neonatal survival, and milk yield of ewes in commercial flocks; and 2) define, for the first time, biological effects of oPL on endometrial functions and gene expression by uterine gland epithelium.
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