Relevant bibliographies by topics / Uterus – Physiology

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Reports

Academic literature on the topic 'Uterus – Physiology'

Author: Grafiati
Published: 4 June 2021
Last updated: 31 January 2023

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Journal articles on the topic "Uterus – Physiology"

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Lammers, Wim J. E. P., H. Mirghani, B. Stephen, S. Dhanasekaran, A. Wahab, M. A. H. Al Sultan, and F. Abazer. "Patterns of electrical propagation in the intact pregnant guinea pig uterus." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 294, no. 3 (March 2008): R919—R928. http://dx.doi.org/10.1152/ajpregu.00704.2007.

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Previous studies have reported on propagation of individual spikes in isolated segments of the pregnant uterus, but there is no information on patterns of spike propagation in the intact organ. There is also no information on propagation of myometrial burst. The aim of this study was to record, at high resolution, patterns of propagation of electrical activities in the pregnant uterus. Sixteen timed-pregnant guinea pigs were euthanized at term, and their uteruses isolated. Fetuses were removed and replaced by an equal amount of Tyrode. A 240-electrode array was positioned at various locations along the organ, all signals were recorded simultaneously, and the electrical propagations were reconstructed. In the intact pregnant uterus at term, spikes propagated with high velocity in longitudinal (6.8 ± 2.4 cm/s) and slower velocity in circular direction (2.8 ± 1.0 cm/s; P < 0.01). Direction of propagation and frequency of activity were highly variable but showed similar patterns at the ovary or cervical end and along the anterior, posterior, and antimesometrial borders. Along mesometrium, spike propagation was sparse and fractionated. Migration of burst (0.6 ± 0.4 cm/s) was significantly much slower than that of individual spikes ( P < 0.001). Initial burst activity was located at variable locations along the ovarial end of the antimesometrial border, while the latest excitation occurred at the cervical end (1.2 ± 0.9 min). In conclusion, high resolution electrical mapping of the intact pregnant uterus reveals fundamental properties in spatial and temporal patterns of spike and burst propagation that determine the contraction of the organ.
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Tabb, TN, RE Garfield, and G. Thilander. "Physiology of myometrial function: intercellular coupling and its role in uterine contractility." Fetal and Maternal Medicine Review 3, no. 2 (July 1991): 169–83. http://dx.doi.org/10.1017/s096553950000053x.

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The mammalian uterus is composed of a preponderance of small smooth muscle cells usually aligned in two layers. The number of muscle cells in the human uterus at term is estimated at 200 billion, each minute fusiform cell measuring about 5–10μm in diameter and about 200μm in length. The main function of the uterus is to harbour the developing fetus during pregnancy and then to contract vigorously during labour to expel the products of conception. In order for the uterus to contract rhythmically and forcefully, a mechanism must exist to allow interaction between muscle cells in order to achieve synchronous activity. Phasic or cyclical patterns of contractile activity of the uterus cannot be accounted for by stimulation or inhibition from the nervous or endocrine systems. Since myometrial cells are dependent upon action potentials for their contractile processes, some system must be present between the muscle cells for the propagation of action potentials between them. The observation that gap junctions occur in large numbers between myometrial cells during parturition is thought to be significant in this regard and they are considered to play an essential role in parturition and in the control and co-ordination of uterine contractility. In this brief review, we will discuss the role of gap junctions in the modulation of myometrial contractility and the mechanisms that regulate their synthesis and permeability.
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Levy, Anat, Yoram Yagil, Michael Bursztyn, Ronit Barkalifa, Shimon Scharf, and Chana Yagil. "ACE2 expression and activity are enhanced during pregnancy." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 295, no. 6 (December 2008): R1953—R1961. http://dx.doi.org/10.1152/ajpregu.90592.2008.

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In the current study, we investigated the expression and activity of ACE2 during pregnancy in normotensive and hypertensive rats, focusing on the relative contribution of the uterus and the placentas, the kidney serving as a reference. We used the Sabra rat model of salt-sensitive hypertension. We confirmed a systemic vasodilatory state during the third trimester of pregnancy, as evidenced by a reduction in blood pressure, both in normotensive and hypertensive rats. At the time that blood pressure was reduced, ACE2 was expressed abundantly in the reproductive organs. The relative levels of ACE2 mRNA in the pregnant animal were placenta > kidneys ≥ uterus and of ACE2 activity kidney > placenta > uterus. In the uterus and the placenta, ACE2 expression was unaffected by strain, salt-loading, or the level of blood pressure. ACE2 activity in the uterus of the nonpregnant rat was not affected by any of these variables either, but during pregnancy increased in salt-loaded animals. When estimating the total contribution of the uterus to ACE2 mRNA and activity during pregnancy, we found that the amount of ACE2 mRNA increased in both strains irrespective of diet, but that ACE2 activity increased only in salt-loaded animals. We further estimated the relative total contribution of the uterus, placentas, and kidneys to ACE2 expression and activity during pregnancy by adjusting for mass and number of organs and found that the placentas were the major contributors, followed by the kidney and the uterus. We conclude that during pregnancy, the placentas, in particular, but also the uterus, constitute important sources of ACE2, in addition to its normal production in the kidney, leading to an estimated twofold increase in total ACE2 activity. These data are consistent the hypothesis that transient ACE2 overexpression and increased activity during pregnancy may be important in modulating systemic, as well as local hemodynamics in the uteroplacental unit.
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Bulat, R., M. S. Kannan, and R. E. Garfield. "Studies of the innervation of rabbit myometrium and cervix." Canadian Journal of Physiology and Pharmacology 67, no. 8 (August 1, 1989): 837–44. http://dx.doi.org/10.1139/y89-131.

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We characterized the innervation of isolated circular and longitudinal-oriented muscle strips from the nulliparous rabbit uterus and cervix by field stimulation (FS). FS with increasing frequency (2.5–50 pps) and voltage (2.5–70 V) caused graded increases in isometric contraction with no relaxation or inhibition of spontaneous activity. Tetrodotoxin(TTX, 3.1 × 10−6 M) significantly reduced the FS response by 75% in all strips at higher stimulus frequencies. Contractile responses to FS were also significantly inhibited by atropine (3.5 × 10−6 M) in circular uterus and in longitudinal cervix. Guanethidine (5 × 10−6 M) reduced the response in all strips, as did phentolamine (3.6 × 10−6 M) in longitudinal uterus and circular cervix. Propranolol (3.9 × 10−6 M) did not significantly change the response in longitudinal uterus or circular cervix. In longitudinal uterus, combined guanethidine and atropine produced significant inhibition, but not statistically different from either drug alone. Similar results were seen in circular uterus. Electron microscopy and glyoxylic acid histofluorescence indicate that both blood vessels and smooth muscle in rabbit uterus are supplied with adrenergic nerves. The results suggest the presence of TTX-sensitive adrenergic and cholinergic excitatory innervation of rabbit uterus and cervix.Key words: uterus, myometrium, cervix, adrenergic innervation.
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Maruyama, Tetsuo, Hirotaka Masuda, Masanori Ono, Takashi Kajitani, and Yasunori Yoshimura. "Human uterine stem/progenitor cells: their possible role in uterine physiology and pathology." REPRODUCTION 140, no. 1 (July 2010): 11–22. http://dx.doi.org/10.1530/rep-09-0438.

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The human uterus mainly consists of the endometrium and the outer smooth muscle layer termed the myometrium. The uterus harbours the exceptional and remarkable regenerative ability responsible for cyclical regeneration and remodelling throughout the reproductive life. The uterus must swiftly and cooperatively enlarge to hold the growing foetus during pregnancy. Furthermore, the endometrium, in particular the functionalis layer, must also regenerate, differentiate and regress with each menstrual cycle under hormonal control. Endometrial regeneration from the basal layer is thought to contribute to replacement of the functionalis layer followed by its slough off during menses and parturition. These morphological and functional features of human endometrium can be reproduced in murine models in which severely immunodeficient mice are xenotransplanted with dispersed human endometrial cells under the kidney capsule. The uterine myometrium possesses the similar plasticity of the endometrium. This is demonstrated by multiple cycles of pregnancy-induced enlargement and regression after parturition. It is likely that regeneration and remodelling in the female reproductive tract are achieved presumably through endometrial and myometrial stem cell systems. Recent evidence now supports the existence of these stem cell systems in humans. Here, we will review our current understanding of uterine stem/progenitor cells. We also propose a novel hypothetical model in which stem cell activities explain the physiological remodelling and regeneration of the human uterus and the pathogenesis of gynaecological diseases such as endometriosis.
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Nakamura, Takahiro J., Michael T. Sellix, Michael Menaker, and Gene D. Block. "Estrogen directly modulates circadian rhythms of PER2 expression in the uterus." American Journal of Physiology-Endocrinology and Metabolism 295, no. 5 (November 2008): E1025—E1031. http://dx.doi.org/10.1152/ajpendo.90392.2008.

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Fluctuations in circulating estrogen and progesterone levels associated with the estrous cycle alter circadian rhythms of physiology and behavior in female rodents. Endogenously applied estrogen shortens the period of the locomotor activity rhythm in rodents. We recently found that estrogen implants affect Period ( Per) gene expression in the suprachiasmatic nucleus (SCN; central clock) and uterus of rats in vivo. To explore whether estrogen directly influences the circadian clock in the SCN and/or tissues of the reproductive system, we examined the effects of 17β-estradiol (E2) on PER2::LUCIFERASE (PER2::LUC) expression in tissue explant cultures from ovariectomized PER2::LUC knockin mice. E2 applied to explanted cultures shortened the period of rhythmic PER2::LUC expression in the uterus but did not change the period of PER2::LUC expression in the SCN. Raloxifene, a selective estrogen receptor modulator and known E2 antagonist in uterine tissues, attenuated the effect of E2 on the period of the PER2::LUC rhythm in the uterus. These data indicate that estrogen directly affects the timing of the molecular clock in the uterus via an estrogen receptor-mediated response.
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van Doorn, Marieke B., Frederik K. Lotgering, and Henk CS Wallenburg. "Physiology and practical implications of dynamic exercise in pregnancy." Fetal and Maternal Medicine Review 3, no. 1 (January 1991): 11–28. http://dx.doi.org/10.1017/s0965539500000413.

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In pregnancy the uterus and its contents constitute a growing mass of tissue with an increasing need for oxygen and substrates. Muscle activity is associated with an even more dramatic increase in metabolic demands. One might question if the physiological adaptations are adequate to provide for the combined demands of exercise and pregnancy.
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Wegener, Jörg W., Matthias Lee, and Franz Hofmann. "Hypothyroidism does not affect the dihydropyridine sensitivity of precontracted murine uterus." Canadian Journal of Physiology and Pharmacology 81, no. 9 (September 1, 2003): 890–93. http://dx.doi.org/10.1139/y03-090.

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Thyroid hormones are known to influence various processes of cell differentiation. Recently, it was reported that hypothyroidism reduces the sensitivity to Ca2+-channel antagonists in the rat uterus. We examined the sensitivity to dihydropyridines of the uterus from mice that had reduced thyroid hormone levels. Isradipine relaxed with the same potency precontracted uterine muscle strips from control and hypothyroid mice, independently from a pseudo-pregnant state. These results demonstrate that hypothyroidism does not change dihydropyridine sensitivity (i.e., the pattern of Ca2+-channel expression) in the murine uterus.Key words: uterus, smooth muscle, Ca2+ channel, isradipine.
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Gosden, Roger G. "Ovary and uterus transplantation." REPRODUCTION 136, no. 6 (December 2008): 671–80. http://dx.doi.org/10.1530/rep-08-0099.

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Ovarian and uterine transplantation are procedures gaining more attention again because of potential applications in respectively fertility preservation for cancer and other patients and, more tentatively, women with uterine agenesis or hysterectomy. Cryopreservation of tissue slices, and possibly whole organs, is providing opportunities for banking ovaries for indefinite periods before transplanting them back to restore fertility. The natural plasticity of this organ facilitates grafting to different sites where they can be revascularized and rapidly restore the normal physiology of secretion and ovulation. Ischemic damage is a chief limitation because many follicles are lost, at least in avascular grafts, and functional longevity is reduced. Nevertheless, grafts of young ovarian tissue, even after cryopreservation, can be highly fertile in laboratory rodents and, in humans, autografts have functioned for up to 3 years before needing replacement. Transplantation by vascular anastomosis provides potentially longer function but it is technically much more demanding and riskier for the recipient. It is the only practicable method with the uterus, and has enabled successful pregnancies in several species, but not yet in humans. Contrary to claims made many years ago, neither organ is privileged immunologically, and allografts become rapidly rejected except in hosts whose immune system is deficient or suppressed pharmacologically. All in all, transplantation of these organs, especially the ovary, provides a broad platform of opportunities for research and new applications in reproductive medicine and conservation biology.
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Sohan, Karen, Rebecca Wiggins, and Peter Soothill. "Cervical physiology in pregnancy and labour." Fetal and Maternal Medicine Review 11, no. 3 (August 1999): 135–41. http://dx.doi.org/10.1017/s0965539599000339.

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The uterine cervix is a remarkable structure, which plays an essential role in pregnancy. During the development of the conceptus within the uterus, the cervix usually remains firmly closed to ensure that the developing fetus attains an appropriate degree of maturity to permit extra-uterine survival. On the other hand, it prepares for labour and birth, by undergoing a process of effacement, whereby the substance of the cervix shortens and thins out. During labour, it must be stretched and dilated to a sufficient diameter, usually about 10cm at term, to allow the successful passage of the fetus through the birth canal.
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Dissertations / Theses on the topic "Uterus – Physiology"

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Zhao, Hang, and 趙航. "Melatonin receptors in the rat uterus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2000. http://hub.hku.hk/bib/B31241384.

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Chatdarong, Kaywalee. "Reproductive physiology of the female cat : with special reference to cervical patency, sperm distribution and hysterography /." Uppsala : Dept. of Obstetrics and Gynaecology, Swedish Univ. of Agricultural Sciences, 2003. http://epsilon.slu.se/v162.pdf.

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MacKintosh, Sian Bethan Patricia. "Development of 3D cultural models of epithelial and stromal cells to study the pathophysiology of the bovine endometrium." Thesis, Royal Veterinary College (University of London), 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572141.

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Dunlap, Kathrin Anson. "The role of ovine betaretroviruses in uteroplacental function." [College Station, Tex. : Texas A&M University, 2006. http://hdl.handle.net/1969.1/ETD-TAMU-1850.

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Lin, Chunling. "The role of oestrogens in the growth and induction of progesterone receptors in the mammary gland and uterus of pigs." Thesis, University of Reading, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.480562.

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Bulawa, Lillith. "The Effects of Total Body Proton Irradiation on Mouse Myometrium." Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/honors/548.

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The boundaries of human space exploration continue to expand with new technology and discoveries making it even more important to investigate the effects of space on biological systems. Although humans have explored space in small increments, reproductive studies must be conducted to determine if stable short- or long-term residences for humans can exist in space. This study explored the effects of whole-body proton radiation on uterine smooth muscle known as the myometrium. Two types of mice utilized in this study were C57BL/6 and B6.129S6Cybbtm1Din/J NOX2 knockout mice. C57BL/6 mice are standard laboratory mice that were used to represent the wildtype treatment group (N=18). The B6.129S6Cybbtm1Din/J NOX2 knockout mice have the NADPH Oxidase 2 gene shut off and represented the NOX2 Knockout treatment group (N=18). A third treatment group was made up of half of the C57BL/6 mice and were fed apocynin (N=18). Apocynin has been shown to inhibit NAPDH oxidase production in mice. NADPH Oxidase 2 is involved in the production of deleterious Reactive Oxygen Species (ROS); thus, apocynin should reduce the production of ROS in mice exposed to radiation. Different doses of radiation (0Gy, 0.5Gy, and 2.0Gy) were applied to the myometrium creating three different treatment subgroups within each mouse strain. The mice received 250 MeV protons at an approximate dose rate of 70cGy/ minute. Myometrium tissue was obtained one week following the radiation treatment. The uteri were removed, embedded, sectioned, and stained in hematoxylin and eosin solution. Thickness was determined by taking five measurements each of the outer longitudinal layer length, the inner circular layer length and the total length of both layers of the myometrium for three individual pieces of tissue for each animal. A one-way analysis of variance (ANOVA) was used to determine statistical differences between the groups and subgroups. Wildtype control mice exposed to 2.0Gy (N=5) of radiation had the thickest outer longitudinal layers compared to wildtype mice exposed to 0Gy (N=5) and 0.5Gy (N=6) (p=0.005, p=0). In the apocynin fed and Knockout treatment groups, the subgroups exposed to 0Gy had the thickest layers compared to their respective subgroups exposed to 0.5Gy and 2.0Gy. The apocynin fed mice exposed to 0Gy (N=6) outer longitudinal layer was statistically significantly thicker than the apocynin-fed mice exposed to 0.5Gy (p=0.004; N=6). The inner circular layer of the apocynin-fed mice exposed to 0.5Gy was statistically significantly thicker than the apocynin-fed mice exposed to 2.0Gy (p=0.001; N=6). Amongst the treatment groups, the wildtype control versus the apocynin fed mice exposed to 0Gy showed the apocynin-fed group to have the thicker outer longitudinal layer (p=0.003) and combined layers (p=0.001). Overall, the knockout group showed no statistical difference when compared to the wildtype control group. Further studies are necessary to reduce the possible confounding effect of the estrous cycle in the mice. The different phases of the mice estrus cycle may inadvertently affect the mouse uterine thickness due to the fluctuations in hormones. This study will add to the limited research regarding the female reproductive system in hopes of expanding the knowledge needed to actualize space colonization.
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Fedorka, Carleigh Elizabeth. "AN INVESTIGATION INTO SPECIFIC SEMINAL PLASMA PROTEINS AND THEIR EFFECT ON THE INNATE IMMUNE RESPONSE TO BREEDING IN THE MARE." UKnowledge, 2017. http://uknowledge.uky.edu/gluck_etds/29.

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The mare experiences a transient innate immune response to breeding, the resolution of which is crucial for optimal fertility. The majority of mares are able to modulate this inflammation in a timely fashion, but a subpopulation exists which fail to do so and are considered susceptible to persistent breeding-induced endometritis (PBIE). Seminal plasma has been shown to modulate aspects of this inflammation. Recently, two seminal plasma proteins have garnered interest for their immune modulating properties: cysteine-rich secretory protein-3 (CRISP-3) and lactoferrin. These proteins have been found to alter the binding between sperm and neutrophils based on sperm viability in vitro, but minimal work has evaluated their effect on endometrial mRNA expression of cytokines and inflammation in response to breeding. Experiments were performed to analyze the expression of equine CRISP-3. Found to be primarily synthesized in the ampulla of the vas deferens and to a lesser extent in the vesicular gland, CRISP-3 expression was only seen in the postpubertal stallion. Due to the effect of sperm viability on protein function in vitro, varying sperm populations were analyzed for their effect on gene expression in the uterus. It was determined that viable sperm suppressed the gene expression of the inflammatory modulating cytokine interleukin-6 (IL-6) in comparison to dead sperm. Next, the effect of CRISP-3 and lactoferrin on endometrial gene expression in the normal and susceptible mare was investigated. Neither protein had a significant effect on the mRNA expression of inflammatory cytokines in the normal mares at six hours post-breeding. In contrast, lactoferrin was found to significantly suppress the expression of the pro-inflammatory cytokine tumor necrosis factor (TNF)-α in susceptible mares. Due to this, lactoferrin was further analyzed as an immunomodulant for the treatment of PBIE. Susceptible mares were infused with varying doses of lactoferrin at six hours post-breeding. Although not in a dose-dependent fashion, lactoferrin was found to decrease both fluid retention and neutrophil migration, in addition to suppressing the expression of the pro-inflammatory cytokine interferon gamma (IFNγ) and increasing the gene expression of the anti-inflammatory cytokine interleukin-1 receptor antagonist (IL-1RN). In conclusion, CRISP-3 expression occurs in secretory aspects of the male reproductive tract, and appears to be up regulated after sexual maturation. Viability of spermatozoa affects the immune response to breeding and should be taken into consideration for experimental design and interpretation of data. The seminal plasma proteins CRISP-3 and lactoferrin have minimal effect on endometrial gene expression in normal mares, but lactoferrin suppresses the expression of TNF in susceptible mares. Finally, lactoferrin was found to function as a potent anti-inflammatory for the persistent inflammation seen in susceptible mares when administered post-breeding. This protein should be further investigated as a potential therapeutic for the treatment of persistent breeding-induced endometritis.
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Lesage, Audrey. "Rôles de FOXL2 dans la physiologie endométriale chez les bovins." Thesis, Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLS422/document.

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L'implantation est une étape cruciale de la gestation et du développement post-natal chez les mammifères. L'implantation est définie comme l'établissement d'interactions cellulaires et permanentes entre un endomètre réceptif et un embryon compétent et synchronisé. L'endomètre est un capteur (sensor) biologique de qualité embryonnaire qui conduit la trajectoire de développement du conceptus jusqu'à terme. Les données préalables de notre équipe et d'autres ont suggéré que FOXL2 - un facteur de transcription clé pour l'établissement et le maintien de la fonction ovarienne – pourrait avoir des rôles biologiques majeurs dans le développement et les fonctions de l'endomètre chez les mammifères. L'objectif de mon travail était de comprendre dans quelle mesure FOXL2 contribue à la régulation de la fonction endométriale chez les bovins laitiers. Nous avons d'abord évalué les conséquences des variations du métabolisme maternel sur l'expression endométriale de FOXL2 et une sélection de gènes candidats. Notre étude a mis en évidence que, chez les femelles Holstein primipares taries immédiatement après le vêlage, l'expression de FOXL2 est augmentée alors que l'expression des enzymes antioxydantes est diminuée dans l'endomètre lors de l'implantation. Grâce à un modèle in vitro de cultures primaires de cellules endométriales bovines (fibroblastes et cellules épithéliales glandulaires) surexprimant transitoirement FOXL2, nous avons pu montrer que l'expression des gènes codant pour les enzymes antioxydantes n’est pas dépendante de FOXL2. Sur la base de notre modèle expérimental in vitro, les profils d'expression des gènes ont ensuite été déterminés à l'aide d'un oligoarray bovin non commercial. L’analyse des données a révélé une variation de l’identité des gènes cible de FOXL2 en fonction du type cellulaire considéré. Comme dans l'ovaire, FOXL2 régule des gènes liés à "la réponse immunitaire", "l’apoptose" et "la détermination du sexe". Nos résultats ont également mis en évidence la régulation par FOXL2 de fonctions spécifiques de l'endomètre, telles que «la réponse à l'interféron de type I» et «la modification de matrice extracellulaire». En somme, nos données mettent en lumière le rôle de FOXL2 dans la régulation de la physiologie endométriale bovine. Ses fonctions biologiques mériteraient d'être analysées et comparées chez d'autres espèces de mammifères
Implantation is a critical milestone ensuring a successful pregnancy and normal post-natal development in mammals. Implantation is defined as the establishment of cellular and permanent interactions between a receptive endometrium and a competent and synchronised embryo. Endometrium has been proposed to be a biological sensor of embryo quality that drives the developmental trajectory of the conceptus until term. Previous data from our team and others have suggested major biological roles for FOXL2 – a key transcription factor for the establishment and maintenance of ovarian function- in the development and functions of the mammalian endometrium. The aim of my work was to provide new insights on the contribution of FOXL2 to the regulation of the endometrial function in dairy cattle. We first evaluated the consequences of variations in maternal metabolism on the endometrial expression of FOXL2 and a selection of candidate genes. Our data demonstrated that, in Holstein primiparous females dried immediately after parturition, FOXL2 expression was increased whereas antioxidant enzymes expression was decreased in the endometrium at implantation. Using an in vitro model of primary cultures of bovine endometrial cells (fibroblasts and glandular epithelial cells) transiently overexpressing FOXL2, expression of genes encoding antioxidant enzymes did not appear to be FOXL2 dependant. Based on our in vitro experimental model, gene expression profiles were then determined using a bovine custom oligoarray. Data analyses unveil differences in FOXL2-regulated genes according to endometrial cell origin. As in the ovary, FOXL2 regulated sets of genes related to "immune response", "apoptosis" and "sex determination". Our results also highlighted regulation of endometrium-specific genes by FOXL2 including “response to type I interferon” and “extracellular matrix modification”. Altogether our data support the involvement of FOXL2 in the regulation of bovine endometrial physiology that deserves to be analyzed in other mammalian species
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Stjernholm, Ylva. "Endocrine and neuronal interactions in human cervical ripening /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19981009stje.

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Bessette, Paul Henry. "Engineering and physiology of disulfide bond isomerization in Escherichia coli /." Full text (PDF) from UMI/Dissertation Abstracts International, 2000. http://wwwlib.umi.com/cr/utexas/fullcit?p9995165.

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Books on the topic "Uterus – Physiology"

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T, Chard, and Grudzinskas J. G, eds. The uterus. Cambridge: Cambridge University Press, 1994.

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Gil, Nam Hong, and SpringerLink (Online service), eds. Biomechanics of the Gravid Human Uterus. Berlin, Heidelberg: Springer-Verlag Berlin Heidelberg, 2011.

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E, Garfield Robert, and Tabb Thomas N, eds. Control of uterine contractility. Boca Raton: CRC Press, 1994.

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E, Carsten Mary, and Miller Jordan D, eds. Uterine function, molecular and cellular aspects. New York: Plenum Press, 1990.

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Foos, Laurie. Ex utero. London: Review, 1997.

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Foos, Laurie. Ex utero. Minneapolis: Coffee House Press, 1995.

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Foos, Laurie. Ex utero. San Diego: Harcourt Brace & Co., 1996.

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A, Lavia Lynn, ed. Cellular signals controlling uterine function. New York: Plenum Press, 1991.

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T, Chard, and Grudzinskas J. G, eds. Uterus. Cambridge University Press, 1994.

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Wynn, Ralph M. Biology of the Uterus. Springer, 2012.

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Book chapters on the topic "Uterus – Physiology"

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Greiss, Frank C., and James C. Rose. "Vascular Physiology of the Nonpregnant Uterus." In Biology of the Uterus, 69–87. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5589-2_5.

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Moulton, Bruce C., Kamil C. Akcali, Thomas F. Ogle, Thomas L. Brown, Joan Motz, and Sohaib A. Khan. "Control of Apoptosis in the Uterus during Decidualization." In Cell Death in Reproductive Physiology, 48–66. New York, NY: Springer New York, 1997. http://dx.doi.org/10.1007/978-1-4612-1944-6_5.

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Jashnani, Kusum D., and Meherrituja V. Palve. "Normal Physiologic Changes of Gravid Uterus." In Maternal Mortality - Lessons Learnt from Autopsy, 149–53. Singapore: Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-3420-9_29.

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"Physiology of Pregnancy." In Advances in Medical Diagnosis, Treatment, and Care, 24–59. IGI Global, 2021. http://dx.doi.org/10.4018/978-1-7998-4357-3.ch002.

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Pregnancy is associated with profound anatomical, physiological, biochemical, and endocrine changes that affect multiple organs and systems. One fertilized egg cell implanted in the lining of uterus initiates countless bodily changes. Secretion of ovarian hormones increases greatly. The bone marrow produces more RBCs and blood volume increases. The heart enlarges slightly to handle an extra supply of blood and shifts its position as uterus enlarges with the growing fetus. Such changes are necessary to help women adapt to the pregnant state and to support the growth and survival of the fetus. Such anatomical and physiological changes can also create confusion during a pregnant woman's clinical review. Likewise, changes in the biochemistry of blood during pregnancy may cause difficulties in interpreting tests. Hence, there is need to understand the deviation from normal anatomical, physiological, biochemical, and endocrine changes occurring during pregnancy so as to plan appropriate strategies for better maternal and fetal health.
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Patnaik, Arati. "Physiology and Molecular Biology of Myometrium and Cervix during Pregnancy and Labour." In The Uterus Manual, 21. Jaypee Brothers Medical Publishers (P) Ltd., 2009. http://dx.doi.org/10.5005/jp/books/10984_3.

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Guyer, Christopher, Sree Rajesh, and Mary E. Connor. "Anatomy and Physiology of the Uterus." In Diagnostic and Operative Hysteroscopy, 6–19. Cambridge University Press, 2020. http://dx.doi.org/10.1017/9781316276020.002.

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Jilla, Manju. "Physiology: The Uterus, Cervix and Practical Aspect." In Mastering the Techniques in Hysteroscopy, 34. Jaypee Brothers Medical Publishers (P) Ltd., 2017. http://dx.doi.org/10.5005/jp/books/13032_5.

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"Normal anatomy and physiology of the uterus." In Handbook of Outpatient Hysteroscopy, 19–27. CRC Press, 2005. http://dx.doi.org/10.1201/b13545-7.

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COUSE, J., S. HEWITT, and K. KORACH. "Steroid Receptors in the Ovary and Uterus." In Knobil and Neill's Physiology of Reproduction, 593–678. Elsevier, 2006. http://dx.doi.org/10.1016/b978-012515400-0/50020-8.

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Binder, April K., Wipawee Winuthayanon, Sylvia C. Hewitt, John F. Couse, and Kenneth S. Korach. "Steroid Receptors in the Uterus and Ovary." In Knobil and Neill's Physiology of Reproduction, 1099–193. Elsevier, 2015. http://dx.doi.org/10.1016/b978-0-12-397175-3.00025-9.

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Reports on the topic "Uterus – Physiology"

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Bazer, Fuller W., Arieh Gertler, and Elisha Gootwine. Role of Placental Lactogen in Sheep. United States Department of Agriculture, January 2001. http://dx.doi.org/10.32747/2001.7574339.bard.

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Central problems in sheep and dairy cattle production are reproductive failure due to embryonic/fetal mortality and low birth weights, especially in prolific breeds, and reduced milk yields which adversely affect neonatal survival and economy of production. The sheep placenta expresses lactogenic (ovine placental lactogen, oPL) and somatogenic (ovine placental growth hormone, oGH) hormones. Our research has focused on the biological roles of oPL and oGH in function of the uterine endometrium during gestation and the mammary gland during pregnancy and lactation. Major conclusions were that: ( 1 ) immunization of prepubertal ewes against oPL resulted in increased birth weights of their lambs and their milk production during lactation; (2) neither oPL nor oGH had an antiluteolytic effect on uterine endometrium to affect lifespan of the corpus luteum; (3) only sequential exposure of the progesterone stimulated uterus to oIFNt and oPL or oGH increased endometrial gland proliferation and secretory protein gene expression; (4) oPL signals through a homodimer of ovine prolactin receptor (PRL-R) and heterodimer of oPRL-R and growth hormone receptor (GH-R); (5) exogenous recombinant oPL and oGH stimulated mammogenesis and milk yield during lactation; and (6) mutation of oPL and oGH was used to define specific biological effects and a rational basis for design of a specific receptor agonists or antagonists. This project was very productive in elucidating basic biological effects of oPL and oGH on intracellular signal transduction pathways, uterine development and secretory function, as well as mammogenesis and lactogenesis. We determined that immunization of prepubertal ewes against roPL increased birth weights of their lambs, especially those born as twins and triplets, as well as enhanced lactational performance. These studies significantly extended our knowledge of uterine and fetal-placental physiology and provided a foundation for new strategies to enhance reproductive and lactation efficiency. Based on these results, the major achievements were: 1) creation of a practical and cost effective management tool for producers to increase reproductive performance, neonatal survival, and milk yield of ewes in commercial flocks; and 2) define, for the first time, biological effects of oPL on endometrial functions and gene expression by uterine gland epithelium.
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